POLYHYDRAMNIOS
Presented By:
SNEHLATA PARASHAR
POLYHYDRAMNIOS
INTRODUCTION
DEFINITION
INCIDENCE
ETIOLOGY
CLINICAL TYPES
INVESTIGATIONS
COMPLICATIONS
MANAGEMENT
INTRODUCTION
Amniotic fluid normally increase in amount
throughout pregnancy from a few milliliters to a liter at
38th week. The fluid is not static ; the water and
solutes in it are change every few hours. There are
two chief abnormalities of amniotic fluid.
 Polyhydramnios
 Oligohydramnios
Polyhydramnios is a medical condition excess
of amniotic fluid in the amniotic sac. There are 2
clinical varieties of polyhydramnios.
 Chronic
 Acute
The opposite to Polyhydramnios is Oligohydramnios a
deficiency of amniotic fluid.
DEFINITION
Polyhydramnios is defined as a state where liquor
amnii exceeds 1500 to 2000 ml.
Clinical Definition: The excessive accumulation
of liquor amnii causing discomfort to the patient or when
a imaging help needed to substantiate the clinical
diagnosis of the lie and presentation of foetus.
The incidence varies from 1-2% of the cases. it is
more common of multipare than the premigravidae. it
occurs 1 in 1000 pregnancies.
INCIDENCE
ETIOLOGY
 Fetal Anomalies: Congenital fetal malformation is
associated with polyhydramnios in about 20% cases.
 Anencephaly
 Open Spina Bifida
 Esophageal or Duodenal Atresia
 Facial Cleft
 Hydrops Fetalis
 Placenta: Chorioangioma of the placenta.
 Multiple Pregnancies: Hydromnios is more
common in uniovular twins, usually affecting the
second sac.
 Maternal:
 Diabetis
 Cardiac or Renal Disease
CLINICAL TYPES
Depending on the rapidly of onset, hydramnios may
be:
 Chronic: It is the commonest onset in insidious
taking few weeks.
 Acute: It is extremely rare acutely on pre-existing
chronic variety. The chronic variety is 10 times
commoner than the acute one.
CHRONIC POLYHYDRAMNIOS
 Symptoms:
 Dyspnoea.
 Palpitation.
 Oedema of legs.
 Signs:
 The patient may be dyspnoea state in the lying down
position.
 Evidence of pre-eclampsia (edema, hypertension &
protenuria) may be present.
ABDOMINAL EXAMINATION
 Inspection:
 Abdominal marked enlarge.
 The skin is tense shiny with large straiae.
 Palpation:
 Height of the uterus is more than the period of
amenorrhoea.
 Girth of the abdomen round the umbilicus more than the
normal.
 Foetal parts can’t be well defined: so also the
presentation or the position.
 Auscultation:
 Foetal heart sound is not heard easily.
INVESTIGATION
 USG:
 Amniotic fluid index is more than 25cm.
 To note the lie and presentation of the foetus.
 To diagnose any foetal congenital malformation.
COMPLICATION
 Maternal:
 During Pregnancy:
 Pre eclampsia.
 Malpresentation.
 Pre rupture of the membrane.
 Pre term labour.
 During Labour:
 Early rupture of the membrane.
 Cord prolaps.
 Increase operative delivery due to malpresentation.
 Puerperium:
 Sub involuation
 Increase puerperial morbidity due to infection resulting from
increase operative interpherence and blood loss.
 Foetal Complication:
 Foetal death are mostly due to prematurity & congenital
abnormalities.
MANAGEMENT
 Principles Of Management:
 To relieve the symptoms.
 To find out the causes.
 To avoid and to deal with the complication.
 Supportive Therapy:
 Bed rest, treatment associate with condition like
preeclampsia.
 Investigation are done to exclude congenital foetal
malformation.
 Further management depends on
 Response to treatment.
 Period of gestation.
 Presence of foetal malformation.
 Associated complicating factors.
 Response to treatment is good means pregnancy is
continued.
 Unresponsive:
 Pregnancy less than 37 weeks – Amniocentesis
 Pregnancy more than 37 week – Induction of labour is done.
 Amniocentesis, termination of pregnancy.
 During Labour:
 Usual management is followed.
 If the uterine contration become sluggish, oxytocin
infusion may be started.
 To prevent post partum haemorrhage.
 IV administraion of methergin.
POLYHYDRAMINOS

POLYHYDRAMINOS

  • 1.
  • 2.
  • 3.
    INTRODUCTION Amniotic fluid normallyincrease in amount throughout pregnancy from a few milliliters to a liter at 38th week. The fluid is not static ; the water and solutes in it are change every few hours. There are two chief abnormalities of amniotic fluid.  Polyhydramnios  Oligohydramnios Polyhydramnios is a medical condition excess of amniotic fluid in the amniotic sac. There are 2 clinical varieties of polyhydramnios.  Chronic  Acute The opposite to Polyhydramnios is Oligohydramnios a deficiency of amniotic fluid.
  • 4.
    DEFINITION Polyhydramnios is definedas a state where liquor amnii exceeds 1500 to 2000 ml. Clinical Definition: The excessive accumulation of liquor amnii causing discomfort to the patient or when a imaging help needed to substantiate the clinical diagnosis of the lie and presentation of foetus. The incidence varies from 1-2% of the cases. it is more common of multipare than the premigravidae. it occurs 1 in 1000 pregnancies. INCIDENCE
  • 5.
    ETIOLOGY  Fetal Anomalies:Congenital fetal malformation is associated with polyhydramnios in about 20% cases.  Anencephaly  Open Spina Bifida  Esophageal or Duodenal Atresia  Facial Cleft  Hydrops Fetalis  Placenta: Chorioangioma of the placenta.  Multiple Pregnancies: Hydromnios is more common in uniovular twins, usually affecting the second sac.  Maternal:  Diabetis  Cardiac or Renal Disease
  • 6.
    CLINICAL TYPES Depending onthe rapidly of onset, hydramnios may be:  Chronic: It is the commonest onset in insidious taking few weeks.  Acute: It is extremely rare acutely on pre-existing chronic variety. The chronic variety is 10 times commoner than the acute one.
  • 7.
    CHRONIC POLYHYDRAMNIOS  Symptoms: Dyspnoea.  Palpitation.  Oedema of legs.  Signs:  The patient may be dyspnoea state in the lying down position.  Evidence of pre-eclampsia (edema, hypertension & protenuria) may be present.
  • 8.
    ABDOMINAL EXAMINATION  Inspection: Abdominal marked enlarge.  The skin is tense shiny with large straiae.  Palpation:  Height of the uterus is more than the period of amenorrhoea.  Girth of the abdomen round the umbilicus more than the normal.  Foetal parts can’t be well defined: so also the presentation or the position.  Auscultation:  Foetal heart sound is not heard easily.
  • 9.
    INVESTIGATION  USG:  Amnioticfluid index is more than 25cm.  To note the lie and presentation of the foetus.  To diagnose any foetal congenital malformation.
  • 10.
    COMPLICATION  Maternal:  DuringPregnancy:  Pre eclampsia.  Malpresentation.  Pre rupture of the membrane.  Pre term labour.  During Labour:  Early rupture of the membrane.  Cord prolaps.  Increase operative delivery due to malpresentation.  Puerperium:  Sub involuation  Increase puerperial morbidity due to infection resulting from increase operative interpherence and blood loss.  Foetal Complication:  Foetal death are mostly due to prematurity & congenital abnormalities.
  • 11.
    MANAGEMENT  Principles OfManagement:  To relieve the symptoms.  To find out the causes.  To avoid and to deal with the complication.  Supportive Therapy:  Bed rest, treatment associate with condition like preeclampsia.  Investigation are done to exclude congenital foetal malformation.  Further management depends on  Response to treatment.  Period of gestation.  Presence of foetal malformation.  Associated complicating factors.
  • 12.
     Response totreatment is good means pregnancy is continued.  Unresponsive:  Pregnancy less than 37 weeks – Amniocentesis  Pregnancy more than 37 week – Induction of labour is done.  Amniocentesis, termination of pregnancy.  During Labour:  Usual management is followed.  If the uterine contration become sluggish, oxytocin infusion may be started.  To prevent post partum haemorrhage.  IV administraion of methergin.