Recommended
More Related Content
Similar to pneumothorax-sagargandhi-160911062559.pptx
Similar to pneumothorax-sagargandhi-160911062559.pptx (20)
More from JuniorDoc1
Recently uploaded
Recently uploaded (20)
pneumothorax-sagargandhi-160911062559.pptx
- 1. Pneumothorax Dr. Sagar Gandhi Resident, Dept. Of Pulmonary Medicine GMCH 1
- 2. Introduction The term pneumothorax was first coined by Itard, a student of Laennec, in 1803 Laennec described the clinical picture of pneumothorax in 1819 He described most pneumothoraces as occurring in patients with pulmonary tuberculosis, although he recognised that pneumothoraces also occurred in otherwise healthy lungs, a condition he described as “pneumothorax simple” 2
- 3. Introduction 3 The modern description of primary spontaneous pneumothorax occurring in otherwise healthy people was provided by Kjaergard in 1932 Primary pneumothorax remains a significant global problem The incidence is 18-28/100 000 per year for men and 1.2-6/100 000 per year for women
- 5. What is pneumothorax 5 Pneumothorax is defined as presence of air or gas in the pleural space (OR) air between the lung and chest wall, in other term, air between the visceral pleura and the parietal pleura
- 6. Blebs and bullae are also known as emphysema-like changes (ELCs) The probable cause of pneumothorax is rupture of an apical bleb or bulla Because the compliance of blebs or bullae in the apices is lower compared with that of similar lesions situated in the lower parts of the lungs Pathophysiology 6
- 7. 7 It is often hard to assess whether bullae are the site of leakage, and where the site of rupture of the visceral pleura is Smoking causes a 9-fold increase in the relative risk of a pneumothorax in females A 22-fold increase in male smokers With a dose-response relationship between the number of cigarettes smoked per day and occurrence of Pathophysiology
- 8. Mechanism In normal people, the pressure in pleural space is negative during the entire respiratory cycle. Two opposite forces result in negative pressure in pleural space. (outward pull of the chest wall and elastic recoil of the lung) The negative pressure will be disappeared if any communication develops . 8
- 9. When a communication develops between an alveolus or other intrapulmonary air space and pleural space, air will flow into the pleural space until there is no longer a pressure difference or the communication is sealed 9
- 10. Pathophysiology 10 Negative pressure eliminated The lung recoil-& lung-volume decrease V/Q low –anatomic shunt hypoxia Positive pressure ◦ Compress blood vessels and heart ◦ Decreased cardiac output ◦ Impaired venous return ◦ Hypotension Result in ◦ A decrease in vital capacity ◦ A decrease in PaO2
- 12. Primary spontaneous pneumothorax It occurs in young healthy individuals without underlying lung disease. It is due to rupture of apical sub-pleural bleb or bullae Predisposing factors: Smoking. Tall, thin male. Airway inflammation (distal) Structural abnormalities of bronchial tree Genetic contribution 12
- 13. Secondary spontaneous pneumothorax: It is seen in pt with underlying lung disease. 13 INFECTION: TB Acute bacterial pneumonia COPD Obstructive lung disease ILD Fibrosis Eosinophilic granuloma Sarcoidosis Lymphangioleiomyomatosis.. Malignancy: Primary lung carcinoma Complication of chemotherapy Connective tissue disease Scleroderma Marfans syndrome Catamenial pneumothorax Pulmonary infarct Wegener’s granulomatosis…
- 14. 14
- 16. Clinical manifestation 16 Dyspnea Chest pain ( pleuritic) Uncommon manifestation Cough Hemoptysis Orthopnea Cyanosis Mod tachycardia
- 17. Traumatic pneumothorax 17 Accidental trauma:(non-iatrogenic) Blunt trauma: with fracture ribs. Penetrating trauma: stab wound or shot gun injury. Iatrogenic : Positive pressure ventilation: Alveolar rupture interstitial emphysema pneumothorax. Interventional procedures: Biopsy, thoraco-centesis, CVP line,trachestomy etc..
- 18. clinical type of PNX Pneumothorax Closed pneumothorax Open pneumothorax Tension pneumothorax 18
- 19. Closed Open pneumothorax pneumothorax 19 Tension pneumothorax Is sealed is open The pleural tear The pleural tear The pleural tear act as a ball & valve mechanism The pleural cavity pressure is < the atmospheric pressure The pleural cavity pressure is = the atmospheric pressure The pleural cavity pressure is > the atmospheric pressure
- 20. Clinical manifestation 20 Tension pneumothorax ◦ Distressed with rapid labored respiration ◦ Cyanosis ◦ Marked tachycardia Patient who suddenly deteriorate clinically, be suspected in the patient with ◦ Mechanical ventilation ◦ Cardiopulmonary resuscitation
- 21. Physical examination 21 ◦ Depend on size of pneumothorax ◦ The vital signs usually normal ◦ Unilateral Chest movements ◦ The trachea may be shifted toward the contralateral side if the pneumothorax is large ◦ Tactile fremitus is absent ◦ The percussion note is hyperresonant ◦ The breath sounds are reduced or absent on the affected side ◦ The lower edge of the liver may be shifted inferiorly with a right-side pneumothorax
- 23. Pneumothorax Erect Small pneumothorax Apical lucency Visceral pleural line Large pneumothorax Apical lucency (>2cm in width) Visceral pleural line Tension pneumothorax Lung collapse Mediastinal shift Low flat diaphragm Supine Deep Costophrenic sulcus Lucent Cardiophrenic sulcus Sharp Mediastinal contour Double diaphragm
- 24. Radiological manifestation cxray in erect position cxray in supine position 24
- 27. BTS guideline(1993) ◦ Small ◦ Moderate ◦ large BTS guideline(2003) ◦ Lung margin to chest wall ◦ small<2cm ◦ large≥2cm ACCP guideline ◦ Lung apex to chest top ◦ Small <3cm ◦ large≥3cm Estimation of pneumothorax volume 27
- 28. 28
- 29. Quantification of the size The simple method to estimate the size Small, a visible rim of < 2 cm between the lung margin and the chest wall Large, a visible rim of ≥2 cm between the lung margin and chest Light index Measure transverse Diameters of lung and Compare it with diameter hemithorax wall 29 2 Hemithorax (HT) Lung (L) 9
- 30. 30
- 31. CT scanning 31 It is recommended in difficult cases such as patients in whom the lungs are obscured by overlying surgical emphysema To differentiate a pneumothorax from suspected bulla in complex cystic lung disease
- 32. CT can diagnose easily pneumothroax
- 33. CT can diagnose easily pneumothroax
- 35. U/S in pneumothorax Classical belief lung not optimal for U/S. Ultrasound found to be more sensitive than CXR in diagnosis of pneumothorax.
- 36. U/S signs of pneumothorax Loss of lung sliding. Loss of comet tails. loss of seashore sign (M mode). Stratosphere sign or bar code sign(M mode).
- 37. MANAGMENT 37 Goals ◦ To promote lung expansion ◦ To eliminate the pathogenesis/cause ◦ To decrease pneumothorax recurrence Treatment options according to ◦ Classification of pneumothorax ◦ Pathogenesis ◦ The extension of lung collapse ◦ Severity of disease ◦ Complication and concomitant underlying diseases
- 38. 38
- 39. 39
- 40. 40
- 41. TREATMENT OPTIONS FOR PSP AND SSP Observation O2 treatment Simple aspiration Small catheter aspiration Chest tube drainage/ICD Thoracoscopy (VAT with blebectomy & 41 VAT with pleurectomy) Open (axillary) thoracotomy
- 42. Observation - PSP 42 Small, closed mildly symptomatic spontaneous pneumothorax do not require hospital admission It should be stressed to patient that they should be return directly to hospital in the event of developing breathlessness.
- 43. Observation - SSP 43 Observation alone is only recommend in patients with small SSP of less than 1 cm depth or isolated apical pneumothorax in asymptomatic patients Hospitalization is recommended in these cases All other cases will require active intervention (aspiration or chest drain insertion)
- 44. Marked breathlessness in a patient with a small (<2 cm) PSP may develop tension pneumothorax Observation is inappropriate and active intervention is required If a patient is hospitalised for observation, supplemental high flow (10 l/min) oxygen should be given where feasible. Observation - PSP or SSP 44
- 45. O2 TREATMENT-- PSP or SSP Inhalation of high concentration of oxygen may reduce the total pressure of gases in pleural capillaries by reducing the partial pressure of nitrogen This should increase the pressure gradient between the pleural capillaries and the pleural cavity Thereby increasing absorption of air from the pleural cavity 45
- 46. The rate of resolution/reabsorption of spontaneous pneumothorax is 1.25 – 1.8% of volume of hemithorax every 24 hours The addition of high flow oxygen therapy has been shown to result in a 4-fold increase in the rate of pneumothorax reabsorption during the periods of oxygen supplementation 46
- 47. Simple aspiration 47 Simple aspiration is recommended as first line treatment for all PSP requiring intervention Simple aspiration is less likely to succeed in secondary pneumothoraces and in this situation, is only recommended as an initial treatment in small (<2 cm) pneumothoraces in minimally breathless patients under the age of 50 years Patients should be admitted to hospital and observed for at least 24 hours before discharge.
- 48. Repeated aspiration is reasonable for primary pneumothorax when the first aspiration has been unsuccessful A volume of < 2.5 L has been aspirated on the first attempt The aspiration can be done by needle or catheter 48 Catheter aspiration
- 49. Intercostal tube drainage INDICATIONS ◦ Unstable pneumothorax ◦ Severe dyspnea ◦ Large lung collapse ◦ Open or tension pneumothorax ◦ Recurrent pneumothorax ◦ Simple aspiration or catheter aspiration drainage is unsuccessful in controlling symptoms
- 50. 50 50 The site of chest tube insertion is in the Triangle of safety midaxillary line of fourth and sixth intercostal Intercostal tube drainage
- 51. Trocar tube thoracostomy Insertion of trocar into the pleural space. 51
- 52. Trocar tube thoracostomy Insertion of the chest tube through the trocar 52
- 53. 5 Operative tube thoracostomy The physician’s index finger is used to enlarge the opening and to explore the pleural space Placement of chest tube Intrapleurally using large hemostat
- 54. Observation of drainage 54 No bubble released ◦ The lung re-expansion ◦ The chest tube is obstructed by secretion or blood clot ◦ The chest tube shift to chest wall, the hole of the chest tube is located in the chest wall If the lung re expanded, removing the chest tube 24 hours after re expansion. Otherwise, the chest tube will be inserted again or regulated the position.
- 55. Complications of intercostal tube drainage 55 Penetration of major organs ◦ Lung, stomach, spleen, liver, heart and great vessels ◦ It occurs more commonly when a sharp metal trocar is inappropriately applied Pleural infection ◦ Empyema, the rate of 1% Surgical emphysema ◦ Subcutaneous emphysema
- 56. Chemical pleurodesis 56 Goals ◦ To prevent pneumothorax recurrence ◦ To produce inflammation of pleura and adhesions Indications ◦ Persist air leak and repeated pneumothorax ◦ Bilateral pneumothorax ◦ Complicated with bullae ◦ Lung dysfunction, not tolerate to operation
- 57. Chemical pleurodesis Sclerosing agents ◦ Tetracycline ◦ Doxycycline ◦ Talc ◦ Erythromycin ◦ 10% povidine iodine The instillation of sclerosing agents into the pleural space lead to an aseptic inflammation with dense adhesions. 57
- 58. Methods ◦ Via chest tube or by surgical mean ◦ Administration of intrapleural local anaesthesia, 200 – 300 mg lidocaine intrapleurally injection ◦ Agents diluted by 60 – 100 ml saline ◦ Injected to pleural space ◦ Clamp the tube 4hours ◦ Drainage again ◦ Observed by chest X-ray film, if air of pleural space is absorbed, remove the chest tube ◦ If pneumothorax still exist, repeated pleurodesis. Chemical pleurodesis 58
- 59. 59 Side effect ◦ Chest pain ◦ Fever ◦ Dyspnea ◦ Acute respiratory distress syndrome ◦ Acute respiratory failure Chemical pleurodesis 59
- 60. Surgical treatment 60 Indications ◦ No response to medical treatment ◦ Persistant air leak ◦ Hemopneumothorax ◦ Bilateral pneumothoraces ◦ Recurrent pneumothorax ◦ Tension pneumothorax failed to drainage ◦ Thickened pleura making lung unable to reexpand ◦ Multiple blebs or bullae
- 61. Thoracoscopy- VATs 61 It is being increasingly used in ot with recurrent psp-ssp Recent studies show VATsit may be the procedure of choice and it has very less complication when compare to other open surgery Pleural belbs causes pneumothorax which can be treated by a method called endostapling or suturefollowed by pleurodeisis
- 62. Axillary (open) thoracotomy Transaxillary minithoracotomy is preferred .when VATsis not available Open thotacotomy with suturing of blebs and pleural abrasion is done Recurrence High risk professions B/L or tension pneumothorax INDICATIONS 62
- 63. Complications of pneumothorax Recurrence of spontaneous pneumothorax Tension pneumothorax Hydropneumothorax Encysted pneumothorax Failure of expansion of the collapsed lung Re-expansion pulmonary edema Broncho-pleural fistula Pneumomediastinum
- 64. Recurrence of spontaneous pneumothorax 50% on the same side. 15% on the contralateral side. More common in secondary spontaneous pneumothorax.
- 65. Tension pneumothorax It is life threatening condition. The pleural pressure is more than the atmospheric pressure. Radiological manifestations of large pneumothorax Mediastinal shift, Flattening of the hemidiaphragm & Lung collapse. Associated with clinical manifestations of circulatory collapse (tachycardia, hypotension & sweating). It is more common with Positive pressure ventilation & Traumatic pneumothorax.
- 67. Hydropneumothorax Due to rupture of pleural adhesions. Bronchopleural fistula.
- 68. Encysted pneumothorax Due to pleural adhesions.
- 69. Failure of re-expansion of the collapsed lung Due to pleural adhesions. Or tracheobronchial injury.
- 70. Mediastinal and subcutaneous emphysema ◦ Alveoli rupture, the air enter into pulmonary interstitial and then goes into mediastinal and subcutaneous tissues. ◦ After aspiration or intercostal chest tube insertion, the air enters the subcutaneous by the needle hole or incision – sur ◦ Physical exam gical emphysema – crepitus is present. 70 Subcutaneous emphysema
- 71. Re-expansion pulmonary edema Due to rapid re-expansion of collapsed lung.
- 72. Case discussion.. 72 Ibrahim sheikh 62 yr male Ex auto driver Chr bidi smoker 40 yrs 1 katta/ day left 1 yr Chr alcoholic 40 yrs 60 ml/day left 2 yrs Non tobacco chewer
- 73. Shortness of breath 4yr ^1 day Cough dry 4 days Chest pain 2 days No fever/loa/low/pe Kco oad on mdi 4 yrs No ho ptb/dm/htn/ihd 73
- 74. Ho admission to our dept Rt pneumothorax 23 dec 2015 –icd+ pleurodeisis 16 jan 2016 – icd + pleurodeisis 26 jan 2016 – icd c failure to expand 74
- 75. 2d echo No RWMA lvef 60% Mod TR Mild PAH PASP 50 mmhg Hb 9.9 tlc 7000 p-85 l-12 spo2- 94 c RA Abg/lft/kft/serostatus/bsl/---wnl 75
- 76. Serial xrays 76
- 77. 1ST XRAY 23 DEC 2015 Pneumotx rt 77
- 78. ICD INSERTED 78
- 79. PLERODEISIS 1 79
- 80. ICD REMOVED 80
- 81. 16 MARCH PNEUMO 2ND TIME 81
- 84. 26 TH MARCH 3RD PNEUMOTX 84
- 85. 3RD ICD 85
- 86. INTERMITTENT PLEURODESIS C BETADINE 10 % C INCENTIVE SPIROMETRY 86
- 87. LATEST X RAY 87
- 88. THANK YOU 88