One of the most painful but easy-to-treat dental emergencies is a dry socket.
• Dry socket symptoms are experienced after a tooth extraction.
• This condition requires follow-up care by the doctor who performed the surgery, an oral surgeon or a dentist who is familiar with how to treat it.
For more information, contact :-
Dr Sachdeva's Dental Aesthetic And Implant Institute,
I 101, Ashok Vihar Phase 1, Delhi- 110052
Contact us at
• Phone : +919818894041,01142464041
• Our Websites:
• www.sachdevadentalcare.com
• www.dentalclinicindelhi.com
• www.dentalimplantindia.co.in
• www.dentalcoursesdelhi.com
• www.facialaestheticsdelhi.com
#drysocket #management #thirdmolarextraction #extractioncomplications
Fibro-osseous lesions of the jaws
Fibrous dysplasia
Cemento-osseous dysplasia
Focal cemento-osseous dysplasia
Periapical cemento-osseous dysplasia
Florid cemento-osseous dysplasia
Ossifying fibroma
Juvenile aggressive ossifying fibroma
Cherubism
Fibro-osseous lesions (FOL) are characterized by replacement of normal bone architecture by collagen fibers and fibroblasts containing calcified tissue.
They include a wide variety of lesions of developmental, dysplastic and neoplastic origins with clinical and radiographic presentation and behavior.
Because of the histological similarities between diverse diseases, proper diagnosis requires correlation of history, clinical and radiographic findings.Fibrous Dysplasia
2. Reactive (dysplastic lesions arising in the tooth-bearing area (presumably of periodontal origin).
a. Periapical cemento-osseous dysplasia
b. Focal cemento-osseous dysplasia
c. Florid cemento-osseous dysplasia
3. Fibro-osseous neoplasms (widely designated as cementifying fibroma, ossifying fibroma or cemento-ossifying fibroma.Bone dysplasias
a. Fibrous dyspla i. Monostoticii. Polyostotic
iii. Polyostotic with endocrinopathy (McCune-Albright)
iv Osteofibrous dysplasia
b. Osteitis deformansc. Pagetoid heritable bone dysplasias of childhood
d. Segmental odontomaxillary dysplasia
2. Cemento-osseous dysplasias
a. Focal cemento-osseous dysplasia b. Florid cemento-osseous dysplasia
3.Inflammatory/reactive processes
a. Focal sclerosing osteomyelitisb. Diffuse sclerosing osteomyelitis
c. Proliferative periostitis
4. Metabolic Disease: hyperparathyroidism
5. Neoplastic lesions (Ossifying fibromas)
a. Ossifying fibromab. Hyperparathyroidism jaw lesion syndrome
c. Juvenile ossifying fibroma i. Trabecular typeii. Psammomatoid type
d. Gigantiform cementomas
One of the most painful but easy-to-treat dental emergencies is a dry socket.
• Dry socket symptoms are experienced after a tooth extraction.
• This condition requires follow-up care by the doctor who performed the surgery, an oral surgeon or a dentist who is familiar with how to treat it.
For more information, contact :-
Dr Sachdeva's Dental Aesthetic And Implant Institute,
I 101, Ashok Vihar Phase 1, Delhi- 110052
Contact us at
• Phone : +919818894041,01142464041
• Our Websites:
• www.sachdevadentalcare.com
• www.dentalclinicindelhi.com
• www.dentalimplantindia.co.in
• www.dentalcoursesdelhi.com
• www.facialaestheticsdelhi.com
#drysocket #management #thirdmolarextraction #extractioncomplications
Fibro-osseous lesions of the jaws
Fibrous dysplasia
Cemento-osseous dysplasia
Focal cemento-osseous dysplasia
Periapical cemento-osseous dysplasia
Florid cemento-osseous dysplasia
Ossifying fibroma
Juvenile aggressive ossifying fibroma
Cherubism
Fibro-osseous lesions (FOL) are characterized by replacement of normal bone architecture by collagen fibers and fibroblasts containing calcified tissue.
They include a wide variety of lesions of developmental, dysplastic and neoplastic origins with clinical and radiographic presentation and behavior.
Because of the histological similarities between diverse diseases, proper diagnosis requires correlation of history, clinical and radiographic findings.Fibrous Dysplasia
2. Reactive (dysplastic lesions arising in the tooth-bearing area (presumably of periodontal origin).
a. Periapical cemento-osseous dysplasia
b. Focal cemento-osseous dysplasia
c. Florid cemento-osseous dysplasia
3. Fibro-osseous neoplasms (widely designated as cementifying fibroma, ossifying fibroma or cemento-ossifying fibroma.Bone dysplasias
a. Fibrous dyspla i. Monostoticii. Polyostotic
iii. Polyostotic with endocrinopathy (McCune-Albright)
iv Osteofibrous dysplasia
b. Osteitis deformansc. Pagetoid heritable bone dysplasias of childhood
d. Segmental odontomaxillary dysplasia
2. Cemento-osseous dysplasias
a. Focal cemento-osseous dysplasia b. Florid cemento-osseous dysplasia
3.Inflammatory/reactive processes
a. Focal sclerosing osteomyelitisb. Diffuse sclerosing osteomyelitis
c. Proliferative periostitis
4. Metabolic Disease: hyperparathyroidism
5. Neoplastic lesions (Ossifying fibromas)
a. Ossifying fibromab. Hyperparathyroidism jaw lesion syndrome
c. Juvenile ossifying fibroma i. Trabecular typeii. Psammomatoid type
d. Gigantiform cementomas
Odontogenic keratocyst (OKC) is the cyst arising from the cell rests of dental lamina. It can occur anywhere in the jaw, but commonly seen in the posterior part of the mandible. Radiographically, most OKCs are unilocular when presented at the periapex and can be mistaken for radicular or lateral periodontal cyst.
an overview of muscle pain disorder which regularly create some discomfort for patient to live a normal life as well as to the doctor regarding diagnosis of the problem.
Brief notes on the inflammation of Alveolar bone that surrounds a tooth that has recently been extracted. It occurs as a complication of tooth extraction.
Odontogenic keratocyst (OKC) is the cyst arising from the cell rests of dental lamina. It can occur anywhere in the jaw, but commonly seen in the posterior part of the mandible. Radiographically, most OKCs are unilocular when presented at the periapex and can be mistaken for radicular or lateral periodontal cyst.
an overview of muscle pain disorder which regularly create some discomfort for patient to live a normal life as well as to the doctor regarding diagnosis of the problem.
Brief notes on the inflammation of Alveolar bone that surrounds a tooth that has recently been extracted. It occurs as a complication of tooth extraction.
osteomyelitis of jaw bones / dental implant courses by Indian dental academy Indian dental academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
Acute and Chronic Osteomyelitis - Infection of BoneRahul Singh
Acute and Chronic Osteomyelitis - Infection of Bone
http://essentialinspiration4u.blogspot.com
Osteomyelitis is defined as an acute or chronic inflammatory process of bone, bone marrow and its structure secondary to infection with micro organisms.
Duration , Mechanism & Host response.
Duration - Acute / Subacute / Chronic
Mechanism - Heamatogenous (tonsil , lungs , ear/ GIT) - Exogenous (injection , open fractures)
Host response - Pyogenic / Granulomatous
Introduction of bacteria from :
Outside through a wound or continuity from a neighboring soft tissue infection
Hematogenous spread from a pre existing focus (most common route of infection)
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
Imaging features of acute and chronic osteomyelitis are described in this PPT. Infective arthritis along with fungal infections of soft tissue are also covered very well. Special emphasis is given on tubercular infection of bone.
Dr, Kathirvel Gopalakrishnan
M.D.S (OMFS)
Presentation on osteomyelitis of jaw which helps for a quick refresh.
Classification, management described in detail for easy understanding of the subject.
India Clinical Trials Market: Industry Size and Growth Trends [2030] Analyzed...Kumar Satyam
According to TechSci Research report, "India Clinical Trials Market- By Region, Competition, Forecast & Opportunities, 2030F," the India Clinical Trials Market was valued at USD 2.05 billion in 2024 and is projected to grow at a compound annual growth rate (CAGR) of 8.64% through 2030. The market is driven by a variety of factors, making India an attractive destination for pharmaceutical companies and researchers. India's vast and diverse patient population, cost-effective operational environment, and a large pool of skilled medical professionals contribute significantly to the market's growth. Additionally, increasing government support in streamlining regulations and the growing prevalence of lifestyle diseases further propel the clinical trials market.
Growing Prevalence of Lifestyle Diseases
The rising incidence of lifestyle diseases such as diabetes, cardiovascular diseases, and cancer is a major trend driving the clinical trials market in India. These conditions necessitate the development and testing of new treatment methods, creating a robust demand for clinical trials. The increasing burden of these diseases highlights the need for innovative therapies and underscores the importance of India as a key player in global clinical research.
Health Education on prevention of hypertensionRadhika kulvi
Hypertension is a chronic condition of concern due to its role in the causation of coronary heart diseases. Hypertension is a worldwide epidemic and important risk factor for coronary artery disease, stroke and renal diseases. Blood pressure is the force exerted by the blood against the walls of the blood vessels and is sufficient to maintain tissue perfusion during activity and rest. Hypertension is sustained elevation of BP. In adults, HTN exists when systolic blood pressure is equal to or greater than 140mmHg or diastolic BP is equal to or greater than 90mmHg. The
Global launch of the Healthy Ageing and Prevention Index 2nd wave – alongside...ILC- UK
The Healthy Ageing and Prevention Index is an online tool created by ILC that ranks countries on six metrics including, life span, health span, work span, income, environmental performance, and happiness. The Index helps us understand how well countries have adapted to longevity and inform decision makers on what must be done to maximise the economic benefits that comes with living well for longer.
Alongside the 77th World Health Assembly in Geneva on 28 May 2024, we launched the second version of our Index, allowing us to track progress and give new insights into what needs to be done to keep populations healthier for longer.
The speakers included:
Professor Orazio Schillaci, Minister of Health, Italy
Dr Hans Groth, Chairman of the Board, World Demographic & Ageing Forum
Professor Ilona Kickbusch, Founder and Chair, Global Health Centre, Geneva Graduate Institute and co-chair, World Health Summit Council
Dr Natasha Azzopardi Muscat, Director, Country Health Policies and Systems Division, World Health Organisation EURO
Dr Marta Lomazzi, Executive Manager, World Federation of Public Health Associations
Dr Shyam Bishen, Head, Centre for Health and Healthcare and Member of the Executive Committee, World Economic Forum
Dr Karin Tegmark Wisell, Director General, Public Health Agency of Sweden
CHAPTER 1 SEMESTER V - ROLE OF PEADIATRIC NURSE.pdfSachin Sharma
Pediatric nurses play a vital role in the health and well-being of children. Their responsibilities are wide-ranging, and their objectives can be categorized into several key areas:
1. Direct Patient Care:
Objective: Provide comprehensive and compassionate care to infants, children, and adolescents in various healthcare settings (hospitals, clinics, etc.).
This includes tasks like:
Monitoring vital signs and physical condition.
Administering medications and treatments.
Performing procedures as directed by doctors.
Assisting with daily living activities (bathing, feeding).
Providing emotional support and pain management.
2. Health Promotion and Education:
Objective: Promote healthy behaviors and educate children, families, and communities about preventive healthcare.
This includes tasks like:
Administering vaccinations.
Providing education on nutrition, hygiene, and development.
Offering breastfeeding and childbirth support.
Counseling families on safety and injury prevention.
3. Collaboration and Advocacy:
Objective: Collaborate effectively with doctors, social workers, therapists, and other healthcare professionals to ensure coordinated care for children.
Objective: Advocate for the rights and best interests of their patients, especially when children cannot speak for themselves.
This includes tasks like:
Communicating effectively with healthcare teams.
Identifying and addressing potential risks to child welfare.
Educating families about their child's condition and treatment options.
4. Professional Development and Research:
Objective: Stay up-to-date on the latest advancements in pediatric healthcare through continuing education and research.
Objective: Contribute to improving the quality of care for children by participating in research initiatives.
This includes tasks like:
Attending workshops and conferences on pediatric nursing.
Participating in clinical trials related to child health.
Implementing evidence-based practices into their daily routines.
By fulfilling these objectives, pediatric nurses play a crucial role in ensuring the optimal health and well-being of children throughout all stages of their development.
The Importance of Community Nursing Care.pdfAD Healthcare
NDIS and Community 24/7 Nursing Care is a specific type of support that may be provided under the NDIS for individuals with complex medical needs who require ongoing nursing care in a community setting, such as their home or a supported accommodation facility.
Defecation
Normal defecation begins with movement in the left colon, moving stool toward the anus. When stool reaches the rectum, the distention causes relaxation of the internal sphincter and an awareness of the need to defecate. At the time of defecation, the external sphincter relaxes, and abdominal muscles contract, increasing intrarectal pressure and forcing the stool out
The Valsalva maneuver exerts pressure to expel faeces through a voluntary contraction of the abdominal muscles while maintaining forced expiration against a closed airway. Patients with cardiovascular disease, glaucoma, increased intracranial pressure, or a new surgical wound are at greater risk for cardiac dysrhythmias and elevated blood pressure with the Valsalva maneuver and need to avoid straining to pass the stool.
Normal defecation is painless, resulting in passage of soft, formed stool
CONSTIPATION
Constipation is a symptom, not a disease. Improper diet, reduced fluid intake, lack of exercise, and certain medications can cause constipation. For example, patients receiving opiates for pain after surgery often require a stool softener or laxative to prevent constipation. The signs of constipation include infrequent bowel movements (less than every 3 days), difficulty passing stools, excessive straining, inability to defecate at will, and hard feaces
IMPACTION
Fecal impaction results from unrelieved constipation. It is a collection of hardened feces wedged in the rectum that a person cannot expel. In cases of severe impaction the mass extends up into the sigmoid colon.
DIARRHEA
Diarrhea is an increase in the number of stools and the passage of liquid, unformed feces. It is associated with disorders affecting digestion, absorption, and secretion in the GI tract. Intestinal contents pass through the small and large intestine too quickly to allow for the usual absorption of fluid and nutrients. Irritation within the colon results in increased mucus secretion. As a result, feces become watery, and the patient is unable to control the urge to defecate. Normally an anal bag is safe and effective in long-term treatment of patients with fecal incontinence at home, in hospice, or in the hospital. Fecal incontinence is expensive and a potentially dangerous condition in terms of contamination and risk of skin ulceration
HEMORRHOIDS
Hemorrhoids are dilated, engorged veins in the lining of the rectum. They are either external or internal.
FLATULENCE
As gas accumulates in the lumen of the intestines, the bowel wall stretches and distends (flatulence). It is a common cause of abdominal fullness, pain, and cramping. Normally intestinal gas escapes through the mouth (belching) or the anus (passing of flatus)
FECAL INCONTINENCE
Fecal incontinence is the inability to control passage of feces and gas from the anus. Incontinence harms a patient’s body image
PREPARATION AND GIVING OF LAXATIVESACCORDING TO POTTER AND PERRY,
An enema is the instillation of a solution into the rectum and sig
Leading the Way in Nephrology: Dr. David Greene's Work with Stem Cells for Ki...Dr. David Greene Arizona
As we watch Dr. Greene's continued efforts and research in Arizona, it's clear that stem cell therapy holds a promising key to unlocking new doors in the treatment of kidney disease. With each study and trial, we step closer to a world where kidney disease is no longer a life sentence but a treatable condition, thanks to pioneers like Dr. David Greene.
2. ▪ The word “osteomyelitis” originates from the ancient Greek
words osteon (bone) and muelinos (marrow) and means
infection of medullary portion of the bone.
3. ▪ It define as the inflammation of bone and its marrow contents.(Shafer,7th
edition)
▪ It is an acute or chronic inflammatory process in the medullary spaces or
cortical surfaces of the bone that extends away from the initial site of
involvement (Neville,3rd edition)
DEFINITION
4. ▪ According to anatomic location of infectious process
• Intramedullary
• Subperiosteal
• Periosteal
CLASSIFICATION
5. ▪ According to duration and severity
1.Acute —it occurs initial infection with the micro-organism
2.Chronic—it can be primary or secondary
a.Primary -virulence of the microorganism is low and the host
resistance is high. This type is not preceded by an episode of
acute symptoms
b.Secondary-it is secondary to incompletely treated acute
osteomyelitis
6. ▪ Depending upon the presence or absence of
suppuration
A.SUPPURATIVE OSTEOMYELITIS
1.Acute suppurative osteomyelitis
2.Chronic suppurative osteomyelitis
a. Primary not followed by acute phase
b. secondary followed by acute phase
3.Infantile osteomyelitis
10. Clinical Staging of Osteomyelitis
1.Initial stage—spontaneous pain (localized).
2.Acute stage (suppurative stage)—in this stage, there is severe
pain, soreness and looseness of the involved teeth
▪ Early acute stage—progressive sensitivity of the adjacent teeth
to percussion and pain in the involved side of jaw.
▪ Late acute stage—paresthesia or anesthesia of the lip region
supplied by the mental nerve. Other systemic symptoms can
occur.
11. 3.Osteonecrotic stage—diminished spontaneous pain,abscess
formation and pus discharge
4.Sequestrum stage—lack of symptoms sequestrum formation visible
on the radiograph.
SEQUESTRUM(lysed or remained bone)
INVOLCRUM( new bone formation due to new blood vessels)
CLOCAE (channels formed in order to discharge pus from sequestra)
12. Occurrance
▪ Sex—it is more common in men, than women.
▪ Site-May involve either maxilla or the mandible .
Osteomyelitis in maxilla :
Rare occurrance due to-
- Extensive blood supply and significant collaterals
- Porous nature of membranous bones
- Thin cortical plates
- Abundant medullary spaces
13. Osteomyelitis in mandible
▪ An important factor in establishment of osteomyelitis in
mandible is compromise of blood supply
▪ Blood supply
- Primary supply – by inferior alveolar artery, except coronoid
(temporalis vessels)
- Secondary supply – periosteal supply
▪ Venous drainage – upwards via inferior alveolar vein to
pharyngeal plexus
▪ Downwards to external jugular veins
14. Clinical features
▪ Acute
▪ Initial symptoms—it has rapid onset and course. Patient
complaint of severe pain, paresthesia or anesthesia of the
mental nerve.
▪ Initial signs—at this stage, the process is truly intra-medullary,
therefore swelling is absent, teeth are notmobile and fistulae
are not present.
15. ▪ Late symptoms—there is deep intense pain, anorexia,malaise,
fever, and regional lymphadenopathy. Patient also complain of
soreness of involved teeth which become loose within 10 to 14
days. There is also fetid oral odor.
▪ Late signs—pus exudates around the gingival sulcus or
through mucosal and cutaneous fistula. There is firm cellulitis
of cheek and abscess formation with localized warmth and
tenderness on palpation. The patient feels toxic and
dehydrated.
16. ▪ Chronic
▪ Onset—it has insidious onset with slight pain, slow increase in
jaw size and a gradual development of sequestra without
fistula.
▪ Symptoms—it is painless unless there is an acute or sub-acute
exacerbation.
▪ Necrotic bone—in some cases, necrotic bone may be visible
inside the oral cavity
17. ▪ Sinus—intraorally and extraorally sinus developed
intermittently and drains small amount of pus and then
gradually heals. Sinus extends from medullary bone, through
cortical plate, to mucous membrane or skin. Sinus may be at
a considerable distance from the offending infection.
▪ Signs—local tenderness and swelling develop over the bone in
the area of abscess
▪ Lymph nodes—regional lymphadenopathy is present
18. Radiographic features
▪ Acute- develops at least after one to two weeks .At this time
diffuse lytic changes in the bone begin to appear .Individual
trabeculae become fuzzy and indistinct and radiolucent areas
begin to appear
▪ Chronic –
1.Radiodensity—single or multiple radiolucencies of variable
sizes are seen.
2.Margins—irregular outline and poorly defined borders.
3.Moth eaten appearance
4. Sequestra
19. 4.Teeth— the roots of the teeth may undergo external
resorption and the lamina dura may become less
apparent as it blends with surrounding granular
sclerotic bone
5.Fistula tract—fistula tracts may appear on the radiograph as
radiolucent bands transversing the body of the jaw and
penetrating the cortical plates.
6.Joint involvement—in patients with extensive chronic
osteomyelitis, the disease may spread to mandibular condyle
and joint, resulting in septic arthritis
7.Pathological fracture
20.
21. Diagnosis
▪ Clinical diagnosis—pain, swelling, fever is present.
▪ Radiological diagnosis—loss of lamina dura, saucer shaped
destruction, sequestrum formation and motheaten
appearance will give clue to the diagnosis.
▪ Laboratory diagnosis—the medullary spaces are filled with
inflammatory exudate that may or may not progress to the
actual formation of pus. The inflammatory cells are chiefly
neutrophilic polymorphonuclear leukocytes, but may show
occasional lymphocytes and plasma cells.
▪ Investigation to be carried out in osteomyelitis—investigation
like gram staining, culture and sensitivity,WBC count and
complete hemogram, blood sugar,Mantoux test, radiographs,
scintigraphy and computerized tomography.
22. Differential diagnosis
▪ Paget’s disease—it affects multiple bones and the complete
involvement of individual bone.
▪ Eosinophilic granuloma—margins are better than osteomyelitis
and have no evidence of bone sclerosis
23. Management
The goal of definitive therapy is to attenuate and eradicate
the proliferating pathogenic microorganisms and to
support healing. This is accomplished by removing
pathogenic supporative debris, providing regional stability
and disrupting pathophysiology barriers while re-
establishing vascular permeability to the infected area
24. 1.Incision and drainage
2.Irrigation and debridement of necrotic areas
3.Empiric therapy:
▪ Regimen I—aqueous penicillin 2 million units, IV, 4hourly plus
oxacillin 1 gm, IV, 4 hourly.
▪ Regimen II—if the patient is asymptomatic after 48 to72 hours,
then penicillin V 500 mg, 6 hourly and dicloxacillin 250 mg, 4
hourly, for an additional 2 to 4 weeks
25. 4. Extraction—extraction of carious teeth with periapical
infection, should be done. It should be carried out to remove
the source of infection from the oral cavity.
5.• Supportive therapy:
Adequate rehydration —patients is suffering from osteomyelitis
required adequate rehydration in the form of fluids.
Rich nutritional diet —rich nutritional diet should be given.
Vitamin therapy—multivitamin supplements should be given.
27. CHRONIC FOCAL SCELEROSING OSTEOMYELITIS
(Condensing osteitis)
mild bacterial infection
through carious tooth
bone
Proliferation as infection act as
stimulant rather than irritant
Clinical features
1.Children>young adults>older
individual
2.Mandibulat first molar most
commonly affected.
3.Mild pain associated with an infected
pulp
Radiographic features
1.Well circumscribed radio-opaque
mass of sclerotic bone surrounding
and extending below the apex of
roots.
2.Intact lamina dura with widening
of PDL space
28. CHRONIC DIFFUSE SCLEROSING OSTEOMYELITIS
▪ Proliferative reaction of bone to a low
grade infection.
▪ Portal of entry is diffuse periodontal
disease
Clinical features
1.Most common in older individual
with especially in edentulous
mandibular jaws or edentulous
areas and does not excihibit any
gender predominance.
2.On acute exacerbation results in
vague pain ,unpleasant taste and
mild suppuration ,many times with
the spontaneous formation od a
fistula opening onto the mucosal
surface to establish drainage.
Radiographic features
1.Diffuse patchy,sclerosis of bone
(cotton wool appearance)
2.Sometimes bilateral
involvement
3.Occationally involvement of
both maxilla and mandible of
same patient.
4.Border between the sclerosed
bone and normal bone is
indistinct.
29. CHRONIC OSTEOMYELITIS WITH PROLIFERATIVE PERIOSTITIS
(Garre’s chronic nonsuppurative sclerosing osteitis ,periostitis
ossificans)
▪ Focal gross thickening of the periosteum with peripheral reactive bone formation resulting
from mild irritation or infection
Clinical features
1.Young person before the age of 25 years
2.Most frequently involves anterior surface of tibia
3.In jaws more common in mandible of children and young adults
(most cases occur in bicuspid and molar region)
4.Patient complains of toothache or pain in the jaw and a bony hard swelling on the outer
surface of the jaw.This mass is usually of several week duration.
5.This occur as a result of overlying soft tissue infection or cellulitis subsequently involving
periosteum
30. Radiographic features
1.Often reveals an carious tooth opposite the hard bony mass
2.Occlusal radiograph shows a focal overgrowth of bone on the
outer surface of the cortex ,which may be described as
duplication of the cortical layer of bone .
3.This mass of bone is smooth and rather well calcified and may
itself show a thin but definite cortical layer.