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Dr V.RAMKUMAR 
CONSULTANT DENTAL& FACIOMAXILLARY SURGEON 
REG NO; 4118 TAMILNADU-INDIA(ASIA)
INTRODUCTION 
 Osteomyelitis is an extensive inflammation of 
a bone. 
 Involves the cancellous portion, bone marrow, 
cortex, and periosteum. 
 Because of the benefit of antibiotic Therapy 
osteomyelitis is -no longer a common, severe 
infection producing serious systemic involvement.
CLASSIFICATION 
Osteomyelitis may occur in several forms: 
 Suppurative osteomyelitis 
 Acute 
 Chronic 
 Nonsuppurative osteomyelitis 
 Chronic sclerosing 
• Focal 
• Diffuse 
 Garre’s Osteomyslitis 
 Osteomyelitis accompanying systemic diseases
SUPPURATIVE OSTEOMYELITIS 
Osteomyelitis In Infants (osteomyelltis neonaterum) 
The microorganisms are believed to enter 
wounds made during delivery when the finger is 
inserted into the child's mouth and the mucosa 
scratched or later through injuries of the mucosa 
made by sucking an object.
Four- year – old child with acute osteomyelitis of the 
mandible
Clinical Findings 
 Ostemyelitis in infants may have a sudden 
onset and run an acute course. 
 Such cases are associated with a severe 
constitutional reaction 
 High fever 
 Rapid pulse, 
 Vomiting, delirium, and prostration.
 In chronic course - slow onset, with slight 
fever, and moderate pain. 
 The local signs are swelling of the face, 
 Edema of the eyelids, 
 Subperiosteal abscesses that develop on 
the alveolar mucosa and palate, 
 Sinustracts draining pus. 
Treatment 
 Antibiotics should be given intravenously
Acute suppurative Ostsomyelitls 
 Acute, osteomyelitis in adults Involves the 
mandible more often than the maxilla. 
 the various vulnerable parts of the jaw may 
succumb, especially the alveolar process, the 
angle of the jaw, the posterior part of the 
ramus and the coronoid process.
Large sequestrum in a patient with extensive 
osteomyelitis of the mandible
 The onset of acute osteomyelitis is 
accompanied by fever and chills, rapid 
pulse and respiration, and sometimes 
nausea and vomiting. Dehydration and 
acidosis may accompany the toxemia, and 
albuminuria is a frequent finding.
The white cell count may show an 
appreciable leukocytosis. 
In addition, there is a decided "shift to left. 
Toxemia is indicated by the presence of 
immature cells.
Acute osteomyelitis causing considerable bone 
destruction and resulting in a radiolucent appearance 
of the ramus
RADIOGRAPHIC FINDINGS 
 Enlargement of the marrow spaces. 
 Later the cortex becomes involved, 
 Forms osteolytic channels 
 Surround dead pieces of bone, or sequestrum. 
 Larger radiolucent areas denote active destruction of 
bone.
Treatment 
 Complete bed rest, 
 a high-protein and high caloric diet, 
 Adequate multivitamins. 
Dehydration - administration of intravenous 
solutions.
 Blood transfusions when RBC count is low. 
 Analgesics for pain. 
Antibiotic therapy. 
Penicillin - immediate drug of choice.
Chronic Suppurative Osteomyelitis 
Chronic osteomyelitis, results from infection 
by subvirulent organisms. 
The failure to drain the pus results in 
accumulation of pus and 
consequent elevation of 
periosteum from the bone. 
The subperiosteal blood vessels are 
stretched, breaks resulting in ischemia.
 Due to ischemia, cortical bone becomes 
devitalized 
Such a devitalized piece of bone appears 
sclerosed and becomes a foreign body known 
as sequestrum. 
 The mandibular premolar regions are 
mostly involved. 
This is due to the thrombosis of the inferior 
dental vessels exerting its pressure on the 
inferior dental canal.
Chronic osteomyelitis – large sequestrum
Clinical Features 
 Similar to those of acute osteomyelitis. 
 The pain is less severe 
 The temperature is still elevated 
 Leukocytosis is only slightly greater 
than normal.
Teeth may not be loose or sore, so that 
mastication is at least possible. 
Acute exacerbations of the chronic stage 
may occur periodically. 
 The suppuration may perforate the bone 
to form a fistulous tract 
 This form should be treated on the same 
principles as its acute counterpart.
Radiographic findings 
The sequestrum often appears 
radiopaque, 
 separated by a zone of radiolucency, - 
“moth eaten appearance” 
 A layer of subperiosteal new bone 
formation – “involucrum” 
 Appears as a linear laminated opacity, 
parallel to the cortical surface.
Chronic osteomyelitis –– MMaannddiibbllee –– IIIIII mmoollaarr 
rreeggiioonn
Chronic osteomyelitis – Mandible (Ramus).
SEQUESTRUM
TREATMENT 
 Emperical antibiotic therapy 
 Sequestrectomy and curettage 
 Saucerization – cleaning up the bone cavity 
rendering the cavity broad.
 Partial or total removal of cortex – known as 
decortiication 
 Cavity is packed with iodoform or whitehead’s 
varnish
NON SUPPURATIVE OSTEOMYELITIS 
CHRONIC FOCAL SCLEROSING OSTEOMYELITIS 
(Condensing Osteitis) 
 Chronic focal scelrosing osteomyelitis is an unusual 
reaction of bone to infection, 
 Occurrs in instances of extremely high tissue 
resistance 
 In cases of a low – grade infection.
Clinical Features 
 Exclusively in young persons 
 before the age of 20 years. 
 Tooth most commonly is the mandibular 
first molar. 
 Mild pain associated with an infected 
pulp.
RADIOGRAPHIC FINDINGS 
 Well – circumscribed radiopaque mass of 
sclerotic bone 
 surrounds and extends below the apex of 
one or both roots 
 The entire root outline is nearly always visible
Radiograph shows sclerotic sequestrum
 An important feature in distinguishing it from 
the benign cementoblastoma 
Radiopacity stands out in distinct contrast to 
the trabeculation of the normal bone. 
 This is basically a reaction of bone to a mild 
bacterial infection.
TREATMENT 
The tooth is treated endodontically or extracted. 
 The sclerotic bone constituting the osteomyelitis 
is not attached to the tooth. 
Remains after the tooth is removed.
Chronic Diffuse sclerosing Osteomyelitis 
Chronic diffuse sclerosing osteomyelitis is a 
condition analogous to the focal form of the 
disease. 
 Represents a proliferative reaction of the bone. 
 Entry for the infection is through diffuse 
periodontal disease.
CLINICAL FEATURES 
 May occur at any age 
 Most common in older persons. 
Especially in edentulous mandibular jaws or 
edentulous areas 
Presents no clinical indications of its presence
Exposure of the necrotic bone – Intraoral 
view of exposed bone
An acute exacerbation of the dormant chronic 
infection 
 Spontaneous formation of a fistula opening 
onto the mucosal surface to establish drainage 
 vague pain 
 bad taste in the mouth.
RADIOGRAPHIC FEATURES 
 Diffuse sclerosis of bone. 
 Radiopaque lesion may be extensive - bilateral 
 The border between the sclerosis and the 
normal bone is often indistinct.
TREATMENT 
 Lesion is usually too extensive to be removed 
surgically, 
 Yet it frequently undergoes acute exacerbations 
Antibiotic administration.
If a tooth is present in one of these sclerotic 
areas and must be extracted the probability must 
be recognized.
Garre's Osteomyelitis Of The Mandible 
chronic osteomyelitis with proliferative periostitis, 
Periostitis Ossificans 
 Nonsuppurative process in which there is 
 Peripheral sub periosteal bone deposition 
 Caused by mild irritation and infection.
 Affects children and young adults 
 Generally involves the mandible. 
 The infectious process localizes in the 
periosteum
A patient, aged 10 years, with 
Garre’s osteomyelitis of the 
mandible
 Spreads only slightly into the interior of 
the bone. 
 Bony thickening is visible in the 
radiograph. 
 Garre’s osteomyelitis should be 
distinguished from infantile cortical 
hyperostosis, or Caffey's disease.
 Caffey believed this disease to be of 
infectious origin 
 Because of the accompanying high fever 
 Elevated sedimentation rate. 
The disease is selflimiting and eventually 
regresses.
Radiograph shows extensive proliferation of 
subperiosteal bone
TREATMENT 
 Unusually removal of the infected tooth 
 Curettage of the socket are curative. 
Surgery should be done only if there is 
obvious facial asymmetry after at least a 6- 
month waiting period.
Osteomyelitis associated with systemic 
diseases 
 In addition to nonspecific forms of osteomyelitis, 
there are several specific types that accompany 
certain systemic diseases. 
 They include tuberculosis, actinomycosis and 
syphilis.
TREATMENT 
 Generally, treatment involves management of 
the systemic pathosis, as well as local forms 
of therapy. 
 Osteomyelitis of the mandible also has been 
reported as a complication of sickle cell 
anemia.
Osteomyelitis

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Osteomyelitis

  • 1. Dr V.RAMKUMAR CONSULTANT DENTAL& FACIOMAXILLARY SURGEON REG NO; 4118 TAMILNADU-INDIA(ASIA)
  • 2. INTRODUCTION  Osteomyelitis is an extensive inflammation of a bone.  Involves the cancellous portion, bone marrow, cortex, and periosteum.  Because of the benefit of antibiotic Therapy osteomyelitis is -no longer a common, severe infection producing serious systemic involvement.
  • 3. CLASSIFICATION Osteomyelitis may occur in several forms:  Suppurative osteomyelitis  Acute  Chronic  Nonsuppurative osteomyelitis  Chronic sclerosing • Focal • Diffuse  Garre’s Osteomyslitis  Osteomyelitis accompanying systemic diseases
  • 4. SUPPURATIVE OSTEOMYELITIS Osteomyelitis In Infants (osteomyelltis neonaterum) The microorganisms are believed to enter wounds made during delivery when the finger is inserted into the child's mouth and the mucosa scratched or later through injuries of the mucosa made by sucking an object.
  • 5. Four- year – old child with acute osteomyelitis of the mandible
  • 6. Clinical Findings  Ostemyelitis in infants may have a sudden onset and run an acute course.  Such cases are associated with a severe constitutional reaction  High fever  Rapid pulse,  Vomiting, delirium, and prostration.
  • 7.  In chronic course - slow onset, with slight fever, and moderate pain.  The local signs are swelling of the face,  Edema of the eyelids,  Subperiosteal abscesses that develop on the alveolar mucosa and palate,  Sinustracts draining pus. Treatment  Antibiotics should be given intravenously
  • 8. Acute suppurative Ostsomyelitls  Acute, osteomyelitis in adults Involves the mandible more often than the maxilla.  the various vulnerable parts of the jaw may succumb, especially the alveolar process, the angle of the jaw, the posterior part of the ramus and the coronoid process.
  • 9. Large sequestrum in a patient with extensive osteomyelitis of the mandible
  • 10.  The onset of acute osteomyelitis is accompanied by fever and chills, rapid pulse and respiration, and sometimes nausea and vomiting. Dehydration and acidosis may accompany the toxemia, and albuminuria is a frequent finding.
  • 11. The white cell count may show an appreciable leukocytosis. In addition, there is a decided "shift to left. Toxemia is indicated by the presence of immature cells.
  • 12. Acute osteomyelitis causing considerable bone destruction and resulting in a radiolucent appearance of the ramus
  • 13. RADIOGRAPHIC FINDINGS  Enlargement of the marrow spaces.  Later the cortex becomes involved,  Forms osteolytic channels  Surround dead pieces of bone, or sequestrum.  Larger radiolucent areas denote active destruction of bone.
  • 14. Treatment  Complete bed rest,  a high-protein and high caloric diet,  Adequate multivitamins. Dehydration - administration of intravenous solutions.
  • 15.  Blood transfusions when RBC count is low.  Analgesics for pain. Antibiotic therapy. Penicillin - immediate drug of choice.
  • 16. Chronic Suppurative Osteomyelitis Chronic osteomyelitis, results from infection by subvirulent organisms. The failure to drain the pus results in accumulation of pus and consequent elevation of periosteum from the bone. The subperiosteal blood vessels are stretched, breaks resulting in ischemia.
  • 17.  Due to ischemia, cortical bone becomes devitalized Such a devitalized piece of bone appears sclerosed and becomes a foreign body known as sequestrum.  The mandibular premolar regions are mostly involved. This is due to the thrombosis of the inferior dental vessels exerting its pressure on the inferior dental canal.
  • 18. Chronic osteomyelitis – large sequestrum
  • 19. Clinical Features  Similar to those of acute osteomyelitis.  The pain is less severe  The temperature is still elevated  Leukocytosis is only slightly greater than normal.
  • 20. Teeth may not be loose or sore, so that mastication is at least possible. Acute exacerbations of the chronic stage may occur periodically.  The suppuration may perforate the bone to form a fistulous tract  This form should be treated on the same principles as its acute counterpart.
  • 21. Radiographic findings The sequestrum often appears radiopaque,  separated by a zone of radiolucency, - “moth eaten appearance”  A layer of subperiosteal new bone formation – “involucrum”  Appears as a linear laminated opacity, parallel to the cortical surface.
  • 22. Chronic osteomyelitis –– MMaannddiibbllee –– IIIIII mmoollaarr rreeggiioonn
  • 23. Chronic osteomyelitis – Mandible (Ramus).
  • 25. TREATMENT  Emperical antibiotic therapy  Sequestrectomy and curettage  Saucerization – cleaning up the bone cavity rendering the cavity broad.
  • 26.  Partial or total removal of cortex – known as decortiication  Cavity is packed with iodoform or whitehead’s varnish
  • 27. NON SUPPURATIVE OSTEOMYELITIS CHRONIC FOCAL SCLEROSING OSTEOMYELITIS (Condensing Osteitis)  Chronic focal scelrosing osteomyelitis is an unusual reaction of bone to infection,  Occurrs in instances of extremely high tissue resistance  In cases of a low – grade infection.
  • 28. Clinical Features  Exclusively in young persons  before the age of 20 years.  Tooth most commonly is the mandibular first molar.  Mild pain associated with an infected pulp.
  • 29. RADIOGRAPHIC FINDINGS  Well – circumscribed radiopaque mass of sclerotic bone  surrounds and extends below the apex of one or both roots  The entire root outline is nearly always visible
  • 31.  An important feature in distinguishing it from the benign cementoblastoma Radiopacity stands out in distinct contrast to the trabeculation of the normal bone.  This is basically a reaction of bone to a mild bacterial infection.
  • 32. TREATMENT The tooth is treated endodontically or extracted.  The sclerotic bone constituting the osteomyelitis is not attached to the tooth. Remains after the tooth is removed.
  • 33. Chronic Diffuse sclerosing Osteomyelitis Chronic diffuse sclerosing osteomyelitis is a condition analogous to the focal form of the disease.  Represents a proliferative reaction of the bone.  Entry for the infection is through diffuse periodontal disease.
  • 34. CLINICAL FEATURES  May occur at any age  Most common in older persons. Especially in edentulous mandibular jaws or edentulous areas Presents no clinical indications of its presence
  • 35. Exposure of the necrotic bone – Intraoral view of exposed bone
  • 36. An acute exacerbation of the dormant chronic infection  Spontaneous formation of a fistula opening onto the mucosal surface to establish drainage  vague pain  bad taste in the mouth.
  • 37. RADIOGRAPHIC FEATURES  Diffuse sclerosis of bone.  Radiopaque lesion may be extensive - bilateral  The border between the sclerosis and the normal bone is often indistinct.
  • 38. TREATMENT  Lesion is usually too extensive to be removed surgically,  Yet it frequently undergoes acute exacerbations Antibiotic administration.
  • 39. If a tooth is present in one of these sclerotic areas and must be extracted the probability must be recognized.
  • 40. Garre's Osteomyelitis Of The Mandible chronic osteomyelitis with proliferative periostitis, Periostitis Ossificans  Nonsuppurative process in which there is  Peripheral sub periosteal bone deposition  Caused by mild irritation and infection.
  • 41.  Affects children and young adults  Generally involves the mandible.  The infectious process localizes in the periosteum
  • 42. A patient, aged 10 years, with Garre’s osteomyelitis of the mandible
  • 43.  Spreads only slightly into the interior of the bone.  Bony thickening is visible in the radiograph.  Garre’s osteomyelitis should be distinguished from infantile cortical hyperostosis, or Caffey's disease.
  • 44.  Caffey believed this disease to be of infectious origin  Because of the accompanying high fever  Elevated sedimentation rate. The disease is selflimiting and eventually regresses.
  • 45. Radiograph shows extensive proliferation of subperiosteal bone
  • 46. TREATMENT  Unusually removal of the infected tooth  Curettage of the socket are curative. Surgery should be done only if there is obvious facial asymmetry after at least a 6- month waiting period.
  • 47. Osteomyelitis associated with systemic diseases  In addition to nonspecific forms of osteomyelitis, there are several specific types that accompany certain systemic diseases.  They include tuberculosis, actinomycosis and syphilis.
  • 48. TREATMENT  Generally, treatment involves management of the systemic pathosis, as well as local forms of therapy.  Osteomyelitis of the mandible also has been reported as a complication of sickle cell anemia.