Negative Pressure Wound Therapy also widely known as NPWT, WOUND VAC or TNP(Tropical Negative Pressure) is a widely accepted advanced wound management modality today
This topic is been added in the new edition ( 26th ) of Bailey & Love. This topic covers the types, uses and also the principles of removal of a drain. Every MBBS student should be aware of drains & its uses in surgery.
A presentation on the common hand injuries encountered in the Sub-Saharan region of Africa. At the end of the presentation, common infections of the hand as a complication of hand injuries is elucidated.
Negative Pressure Wound Therapy also widely known as NPWT, WOUND VAC or TNP(Tropical Negative Pressure) is a widely accepted advanced wound management modality today
This topic is been added in the new edition ( 26th ) of Bailey & Love. This topic covers the types, uses and also the principles of removal of a drain. Every MBBS student should be aware of drains & its uses in surgery.
A presentation on the common hand injuries encountered in the Sub-Saharan region of Africa. At the end of the presentation, common infections of the hand as a complication of hand injuries is elucidated.
Triage Meditech is one of the leading Indian medical technology companies acquired a respectable position in Advanced Wound Care arena. We are the leading manufacturers and suppliers of Negative Pressure Wound Therapy (NPWT) products in Indian subcontinent. We have further enhanced our portfolio with Advance Wound Dressings, Colostomy Products, Solutions for Venous Insufficiency, and Surgical Disposables and Consumables. Our R&D team is dedicated to continuous advancement in offerings to create effective products at an affordable cost and helping healthcare professionals and caregivers to offer best practice solutions to their patients. Triage Meditech is an ISO 9001:2008, 13485:2003 certified and DCGI regulated company. We follow WHO Good Manufacturing Practice (GMP) and our products are CE Certified. We have Pan India presence through direct and dealers network and currently we export our products to more than 11 countries.
Our new programmable CCNPWT system delivers controlled negative pressure in the wound site to accelerate healing process. The system delivers continuous, variable and intermittent therapy settings for effective therapy goals. The fully loaded system with safety parameters for leakage, blockage, canister full and system inactive conditions. The robust system has been designed light just about 950gms for mobile patients with a very user friendly operation menu.
CLOSED CYCLIC NEGATIVE PRESSURE WOUND THERAPY (CCNPWT), the flexible PU foam dressing adapts to the contours of deep & irregular surface of the wound bed on application of Negative Pressure Therapy. The Specially designed antimicrobial, hydrophobic, non-linear networked foam dressing removes bacteria colonised wound exudates, enhances dermal perfusion and simultaneously helps promote wound closure by primary or secondary intentions. The CCNPWT acti-foam dressing aggressively promotes uniform healthy granulation tissue formation throughout the wound bed.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
3. INTRODUCTION
Acute wounds contribute to a big proportion of wounds globally.
In 2006, 11 million traumatic wounds were managed through the emergency
department in the United States of America .
In Kenya a study by Botchey et al demonstrated RTAs to be a higher contributor
to acute wounds with the lower limb injury being commonest
Poorly managed acute wounds contribute to a larger proportion of chronic
wounds especially in developing countries
Cost Millions of usd per year
4. DEFINITION
Acute wound is defined as a recent wound that has yet to progress through the
sequential stages of wound healing
Can heal in a timely and orderly manner when properly managed.
It can however transit into chronic wounds when poorly managed
5. CLASSIFICATION
Aetiology : Blunt,penetrating,surgical,burn
Rake and Wakefield : Tidy Vs Untidy
Relation to Cavity : Penetrating vs Non Penetrating
Depth : Superficial, Partial thicknes ,Full thickness
Skin integrity : Open vs closed
Mechanism : Mechanical, Thermal,Chemical, Radiation,Bite
Severity ;Simple vs Complex
7. TRAUMATIC WOUNDS
LACERATIONS
Produced by tearing of soft tissues.
The wounds are often irregular and
jagged.
Often contaminated with bacteria and
debris
8. TRAUMATIC WOUNDS
ABRASIONS
Caused by frictional force to the skin
resulting in an extensive wound that could
be superficial in nature
Usually do not bleed excessively and can
appear to close up.
May appear as frictional burns or de-
gloving injuries
9. TRAUMATIC WOUNDS
PUNCTURE WOUNDS/CUTS /INCISIONS
Caused by a sharp object piercing through
the skin.
Commonly associated with injuries to the
deeper structures
Prone to infection
10. TRAUMATIC WOUNDS
BRUISES/HEMATOMAS
Bruises are wounds associated with injuries to
the blood vessels underneath the skin.
The skin may remain intact trapping the blood
underneath leading to redness and erythema.
Excessive accumulation of the blood may result
in clots and later on hematoma
11. TRAUMATIC WOUNDS
COMPLEX TRAUMATIC WOUNDS
Involve other anatomical structures such as
bone, neuro-vascular structures, thoracic,
abdominal viscera
Requires MDT
15. HISTORY
History of events surrounding the wounds
Potential wound contamination
Functional loss or inabilities after the injuries
Pre-hospital wound care
Tetanus immunization status
Comorbidities, current medications
Allergies and substance abuse,
Social history
16. EXAMINATION
Wound location
Status of soft tissue
Length, width and depth of the wound
Type of tissue in the wound bed such as bone,
tendons or nerves
Presence of contaminants
Neuro-vascular status of the extremity
Functional status of surrounding structures
DOCUMENT AND PHOTOGRAPH
17. INVESTIGATIONS
Blood works –FBC, U N C, X Match,Coagulation
Radiological – Xray,Trauma series
-FAST
-CT scan
-CT Angiogram
18. TETANUS
Active immunization against tetanus has been shown to reduce the incidences of tetanus and
morbidity and mortality
Wound condition
Immunization history
Tetanus prone wounds should be left open and should have thorough Surgical debridement
and removal of all dead and devitalized tissues
Two forms:
Tetanus-toxoid containing vaccine (e.g. Tdap or Td)
Tetanus-toxoid containing vaccine (e..g. Tdap or Td) + Tetanus immune globulin
19. TETANUS PRONE WOUNDS
Age of wound >6hours
Avulsion configuration
- depth >1cm
Mechanism of Injury, Missiles, crush or burns
May have signs of infections
May have devitalized tissues
May have contaminants such as soil, feacal matter and grass
May have denervation and ischemic tissues
22. MEDICATION
Antibiotics – Prophylaxis when indicated
Adequate analgesia – NSAIDS
- Opiates
- Local Anaesthesia
- Regional Blocks
- General anesthesia
23. LOCAL ANAESTHESIA
Drug Maximum dose
Bupivacaine 0.25% (2.5 mg/ml) 2 mg/kg
Bupivacaine 0.25% with epinephrine 3 mg/kg
Lidocaine 1% (10 mg/ml) 4.5 mg/kg
Lidocaine 1% with epinephrine 7 mg/kg
Procaine 1% (10 mg/ml) 7 mg/kg
Procaine 1% with epinephrine 9 mg/kg
24. SURGICAL DEBRIDEMENT
Surgical toilet refers to the removal of contaminants in the wound and all necrotic tissues.
Surgical toilet is the mainstay in the management of acute wounds.
Acute wounds that have a good surgical toilet done are likely to heal faster than those without.
Poorly done surgical toilet leads to wound sepsis that results in the wound being arrested in the
inflammatory phase of wound healing.
Where applicable tourniquet should be utilized to assist in arresting bleeding.
Patients with extensive wounds may need blood for grouping and cross matching done during
or before surgical toilet.
27. DEBRIDEMENT
Surgical toilet encompasses the following steps
Wound cleaning and irrigation
Identification of important anatomical structures
Removal of devitalize tissues and foreign materials
Wound closure/ dressing with appropriate dressing materials for delayed closure
28. CLEANING AND IRRIGATION
Allows for dilutions and washing away of wound contaminants.
Pro Proliferative
Irrigation volumes of 50 to 100 ml per cm of laceration is recommended.
Adjusted to the wound characteristics and degree of contamination.
Wound cleaning solution- Isotonic, non hemolytic, nontoxic, colourless ,easy to
sterilize,cheap
Povidine iodine, chlorohexidine or hydrogen peroxide may be toxic to the cells
Normal saline,Setrilized water,commercial wound cleansers
29. IRRIGATION PRESSURE
No Consensus
Generally pressure irrigation beneficial
Very High pressures detrimental >8psi
Equipment used for irrigation has included
Bulb syringes, syringes with an attached needle or catheter,
Intra-venous or irrigation fluid in plastic containers with a pour cap or nozzle, and
pressure canisters
Newer Calibrated devices in market
SOAKING NOT ADVISED
31. IDENTIFICATION OF STRUCTURES
Neurovascular/Tendons
Identify
Spare
Protect
Tag
Repair /reconstruction
Early vs Delayed
32. DEVITALISED TISSUE
Remove all contaminants and dead tissue meticulously while protecting viable
tissues.
Keep reconstruction in mind
Tissues of uncertain viability may be left till the next session of surgical toilet so as
to give them a chance for survival.
Dead muscle is identified by color and unresponsiveness to diathermy or cautery.
34. WOUND CLOSURE
General Principles
Minor non contaminated ,early presentation -1 Closure
6-10hours –Extremety
10- 12hours –Scalp and face
Contaminated wounds- Debride, Dressing ,Relook 48-72hours
35. WOUND CLOSURE
Wound closure methods could be by;
Primary wound closure with sutures, staples, glue or tape
Delayed primary wound closure in wounds that are not clean and require either repeat debridement or
cleaning before closure
Secondary intention, preserved for small wounds or in patients who cannot undergo surgery. Takes time and
is generally discouraged,associated with scars and contracture formation.
Skin graft, for extensive wounds with good vascularity bed.
Flaps are recommended for wounds with poor vascularity base or with exposed bone and neuro-vascular
structures are best covered by flaps