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MANAGEMENT OF ACUTE
WOUNDS
PRESENTER:
Dr Evans S Masitara
Plastic Surgery
University Of Nairobi
OUTLINE
 Introduction
 Definition
 Classification
 Management
 Complications
INTRODUCTION
 Acute wounds contribute to a big proportion of wounds globally.
 In 2006, 11 million traumatic wounds were managed through the emergency
department in the United States of America .
 In Kenya a study by Botchey et al demonstrated RTAs to be a higher contributor
to acute wounds with the lower limb injury being commonest
 Poorly managed acute wounds contribute to a larger proportion of chronic
wounds especially in developing countries
 Cost Millions of usd per year
DEFINITION
 Acute wound is defined as a recent wound that has yet to progress through the
sequential stages of wound healing
 Can heal in a timely and orderly manner when properly managed.
 It can however transit into chronic wounds when poorly managed
CLASSIFICATION
 Aetiology : Blunt,penetrating,surgical,burn
 Rake and Wakefield : Tidy Vs Untidy
 Relation to Cavity : Penetrating vs Non Penetrating
 Depth : Superficial, Partial thicknes ,Full thickness
 Skin integrity : Open vs closed
 Mechanism : Mechanical, Thermal,Chemical, Radiation,Bite
 Severity ;Simple vs Complex
CLASSIFICATION
TRAUMATIC SURGICAL
LACERATIONS CLEAN
BRUISES CLEAN CONTAMINATED
ABRASIONS CONTAMINATED
CONTUSIONS DIRTY
PUNCTURE WOUNDS
BURNS
TRAUMATIC WOUNDS
LACERATIONS
 Produced by tearing of soft tissues.
 The wounds are often irregular and
jagged.
 Often contaminated with bacteria and
debris
TRAUMATIC WOUNDS
ABRASIONS
 Caused by frictional force to the skin
resulting in an extensive wound that could
be superficial in nature
 Usually do not bleed excessively and can
appear to close up.
 May appear as frictional burns or de-
gloving injuries
TRAUMATIC WOUNDS
PUNCTURE WOUNDS/CUTS /INCISIONS
 Caused by a sharp object piercing through
the skin.
 Commonly associated with injuries to the
deeper structures
 Prone to infection
TRAUMATIC WOUNDS
BRUISES/HEMATOMAS
 Bruises are wounds associated with injuries to
the blood vessels underneath the skin.
 The skin may remain intact trapping the blood
underneath leading to redness and erythema.
 Excessive accumulation of the blood may result
in clots and later on hematoma
TRAUMATIC WOUNDS
COMPLEX TRAUMATIC WOUNDS
 Involve other anatomical structures such as
bone, neuro-vascular structures, thoracic,
abdominal viscera
 Requires MDT
COMPLEX TRAUMATIC WOUNDS
SURGICAL WOUNDS
MANAGEMENT
 Trauma Patient– ATLS
 Primary survey – ABCDE
 FATT
 Secondary Survey – AMPLE History
 Wound assessment
HISTORY
 History of events surrounding the wounds
 Potential wound contamination
 Functional loss or inabilities after the injuries
 Pre-hospital wound care
 Tetanus immunization status
 Comorbidities, current medications
 Allergies and substance abuse,
 Social history
EXAMINATION
 Wound location
 Status of soft tissue
 Length, width and depth of the wound
 Type of tissue in the wound bed such as bone,
tendons or nerves
 Presence of contaminants
 Neuro-vascular status of the extremity
 Functional status of surrounding structures
 DOCUMENT AND PHOTOGRAPH
INVESTIGATIONS
 Blood works –FBC, U N C, X Match,Coagulation
 Radiological – Xray,Trauma series
-FAST
-CT scan
-CT Angiogram
TETANUS
 Active immunization against tetanus has been shown to reduce the incidences of tetanus and
morbidity and mortality
 Wound condition
 Immunization history
 Tetanus prone wounds should be left open and should have thorough Surgical debridement
and removal of all dead and devitalized tissues
Two forms:
 Tetanus-toxoid containing vaccine (e.g. Tdap or Td)
 Tetanus-toxoid containing vaccine (e..g. Tdap or Td) + Tetanus immune globulin
TETANUS PRONE WOUNDS
 Age of wound >6hours
 Avulsion configuration
 - depth >1cm
 Mechanism of Injury, Missiles, crush or burns
 May have signs of infections
 May have devitalized tissues
 May have contaminants such as soil, feacal matter and grass
 May have denervation and ischemic tissues
TETANUS VACCINATION
MEDICATION
 Antibiotics – Prophylaxis when indicated
 Adequate analgesia – NSAIDS
- Opiates
- Local Anaesthesia
- Regional Blocks
- General anesthesia
LOCAL ANAESTHESIA
Drug Maximum dose
 Bupivacaine 0.25% (2.5 mg/ml) 2 mg/kg
 Bupivacaine 0.25% with epinephrine 3 mg/kg
 Lidocaine 1% (10 mg/ml) 4.5 mg/kg
 Lidocaine 1% with epinephrine 7 mg/kg
 Procaine 1% (10 mg/ml) 7 mg/kg
 Procaine 1% with epinephrine 9 mg/kg
SURGICAL DEBRIDEMENT
 Surgical toilet refers to the removal of contaminants in the wound and all necrotic tissues.
 Surgical toilet is the mainstay in the management of acute wounds.
 Acute wounds that have a good surgical toilet done are likely to heal faster than those without.
 Poorly done surgical toilet leads to wound sepsis that results in the wound being arrested in the
inflammatory phase of wound healing.
 Where applicable tourniquet should be utilized to assist in arresting bleeding.
 Patients with extensive wounds may need blood for grouping and cross matching done during
or before surgical toilet.
DEBRIDEMENT
DEBRIDEMENT
Surgical toilet encompasses the following steps
 Wound cleaning and irrigation
 Identification of important anatomical structures
 Removal of devitalize tissues and foreign materials
 Wound closure/ dressing with appropriate dressing materials for delayed closure
CLEANING AND IRRIGATION
 Allows for dilutions and washing away of wound contaminants.
 Pro Proliferative
 Irrigation volumes of 50 to 100 ml per cm of laceration is recommended.
 Adjusted to the wound characteristics and degree of contamination.
 Wound cleaning solution- Isotonic, non hemolytic, nontoxic, colourless ,easy to
sterilize,cheap
 Povidine iodine, chlorohexidine or hydrogen peroxide may be toxic to the cells
 Normal saline,Setrilized water,commercial wound cleansers
IRRIGATION PRESSURE
 No Consensus
 Generally pressure irrigation beneficial
 Very High pressures detrimental >8psi
 Equipment used for irrigation has included
 Bulb syringes, syringes with an attached needle or catheter,
 Intra-venous or irrigation fluid in plastic containers with a pour cap or nozzle, and
pressure canisters
 Newer Calibrated devices in market
 SOAKING NOT ADVISED
IRRIGATION
IDENTIFICATION OF STRUCTURES
 Neurovascular/Tendons
 Identify
 Spare
 Protect
 Tag
 Repair /reconstruction
 Early vs Delayed
DEVITALISED TISSUE
 Remove all contaminants and dead tissue meticulously while protecting viable
tissues.
 Keep reconstruction in mind
 Tissues of uncertain viability may be left till the next session of surgical toilet so as
to give them a chance for survival.
 Dead muscle is identified by color and unresponsiveness to diathermy or cautery.
DEVITALISED TISSUE
WOUND CLOSURE
 General Principles
 Minor non contaminated ,early presentation -1 Closure
 6-10hours –Extremety
 10- 12hours –Scalp and face
 Contaminated wounds- Debride, Dressing ,Relook 48-72hours
WOUND CLOSURE
 Wound closure methods could be by;
Primary wound closure with sutures, staples, glue or tape
 Delayed primary wound closure in wounds that are not clean and require either repeat debridement or
cleaning before closure
 Secondary intention, preserved for small wounds or in patients who cannot undergo surgery. Takes time and
is generally discouraged,associated with scars and contracture formation.
 Skin graft, for extensive wounds with good vascularity bed.
 Flaps are recommended for wounds with poor vascularity base or with exposed bone and neuro-vascular
structures are best covered by flaps
WOUND CLOSURE
SKIN GRAFTS
SOLEUS FLAP
PARAUMBILICAL FLAP
FREE FLAP
Complications
 Chronicity
 Severe Infection
 Wound dehiscence
 Limb Loss
 Osteomyelitis
 Malignant Transformation
REFFERENCES
THANK YOU

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Management of acute wounds

  • 1. MANAGEMENT OF ACUTE WOUNDS PRESENTER: Dr Evans S Masitara Plastic Surgery University Of Nairobi
  • 2. OUTLINE  Introduction  Definition  Classification  Management  Complications
  • 3. INTRODUCTION  Acute wounds contribute to a big proportion of wounds globally.  In 2006, 11 million traumatic wounds were managed through the emergency department in the United States of America .  In Kenya a study by Botchey et al demonstrated RTAs to be a higher contributor to acute wounds with the lower limb injury being commonest  Poorly managed acute wounds contribute to a larger proportion of chronic wounds especially in developing countries  Cost Millions of usd per year
  • 4. DEFINITION  Acute wound is defined as a recent wound that has yet to progress through the sequential stages of wound healing  Can heal in a timely and orderly manner when properly managed.  It can however transit into chronic wounds when poorly managed
  • 5. CLASSIFICATION  Aetiology : Blunt,penetrating,surgical,burn  Rake and Wakefield : Tidy Vs Untidy  Relation to Cavity : Penetrating vs Non Penetrating  Depth : Superficial, Partial thicknes ,Full thickness  Skin integrity : Open vs closed  Mechanism : Mechanical, Thermal,Chemical, Radiation,Bite  Severity ;Simple vs Complex
  • 6. CLASSIFICATION TRAUMATIC SURGICAL LACERATIONS CLEAN BRUISES CLEAN CONTAMINATED ABRASIONS CONTAMINATED CONTUSIONS DIRTY PUNCTURE WOUNDS BURNS
  • 7. TRAUMATIC WOUNDS LACERATIONS  Produced by tearing of soft tissues.  The wounds are often irregular and jagged.  Often contaminated with bacteria and debris
  • 8. TRAUMATIC WOUNDS ABRASIONS  Caused by frictional force to the skin resulting in an extensive wound that could be superficial in nature  Usually do not bleed excessively and can appear to close up.  May appear as frictional burns or de- gloving injuries
  • 9. TRAUMATIC WOUNDS PUNCTURE WOUNDS/CUTS /INCISIONS  Caused by a sharp object piercing through the skin.  Commonly associated with injuries to the deeper structures  Prone to infection
  • 10. TRAUMATIC WOUNDS BRUISES/HEMATOMAS  Bruises are wounds associated with injuries to the blood vessels underneath the skin.  The skin may remain intact trapping the blood underneath leading to redness and erythema.  Excessive accumulation of the blood may result in clots and later on hematoma
  • 11. TRAUMATIC WOUNDS COMPLEX TRAUMATIC WOUNDS  Involve other anatomical structures such as bone, neuro-vascular structures, thoracic, abdominal viscera  Requires MDT
  • 14. MANAGEMENT  Trauma Patient– ATLS  Primary survey – ABCDE  FATT  Secondary Survey – AMPLE History  Wound assessment
  • 15. HISTORY  History of events surrounding the wounds  Potential wound contamination  Functional loss or inabilities after the injuries  Pre-hospital wound care  Tetanus immunization status  Comorbidities, current medications  Allergies and substance abuse,  Social history
  • 16. EXAMINATION  Wound location  Status of soft tissue  Length, width and depth of the wound  Type of tissue in the wound bed such as bone, tendons or nerves  Presence of contaminants  Neuro-vascular status of the extremity  Functional status of surrounding structures  DOCUMENT AND PHOTOGRAPH
  • 17. INVESTIGATIONS  Blood works –FBC, U N C, X Match,Coagulation  Radiological – Xray,Trauma series -FAST -CT scan -CT Angiogram
  • 18. TETANUS  Active immunization against tetanus has been shown to reduce the incidences of tetanus and morbidity and mortality  Wound condition  Immunization history  Tetanus prone wounds should be left open and should have thorough Surgical debridement and removal of all dead and devitalized tissues Two forms:  Tetanus-toxoid containing vaccine (e.g. Tdap or Td)  Tetanus-toxoid containing vaccine (e..g. Tdap or Td) + Tetanus immune globulin
  • 19. TETANUS PRONE WOUNDS  Age of wound >6hours  Avulsion configuration  - depth >1cm  Mechanism of Injury, Missiles, crush or burns  May have signs of infections  May have devitalized tissues  May have contaminants such as soil, feacal matter and grass  May have denervation and ischemic tissues
  • 21.
  • 22. MEDICATION  Antibiotics – Prophylaxis when indicated  Adequate analgesia – NSAIDS - Opiates - Local Anaesthesia - Regional Blocks - General anesthesia
  • 23. LOCAL ANAESTHESIA Drug Maximum dose  Bupivacaine 0.25% (2.5 mg/ml) 2 mg/kg  Bupivacaine 0.25% with epinephrine 3 mg/kg  Lidocaine 1% (10 mg/ml) 4.5 mg/kg  Lidocaine 1% with epinephrine 7 mg/kg  Procaine 1% (10 mg/ml) 7 mg/kg  Procaine 1% with epinephrine 9 mg/kg
  • 24. SURGICAL DEBRIDEMENT  Surgical toilet refers to the removal of contaminants in the wound and all necrotic tissues.  Surgical toilet is the mainstay in the management of acute wounds.  Acute wounds that have a good surgical toilet done are likely to heal faster than those without.  Poorly done surgical toilet leads to wound sepsis that results in the wound being arrested in the inflammatory phase of wound healing.  Where applicable tourniquet should be utilized to assist in arresting bleeding.  Patients with extensive wounds may need blood for grouping and cross matching done during or before surgical toilet.
  • 25.
  • 27. DEBRIDEMENT Surgical toilet encompasses the following steps  Wound cleaning and irrigation  Identification of important anatomical structures  Removal of devitalize tissues and foreign materials  Wound closure/ dressing with appropriate dressing materials for delayed closure
  • 28. CLEANING AND IRRIGATION  Allows for dilutions and washing away of wound contaminants.  Pro Proliferative  Irrigation volumes of 50 to 100 ml per cm of laceration is recommended.  Adjusted to the wound characteristics and degree of contamination.  Wound cleaning solution- Isotonic, non hemolytic, nontoxic, colourless ,easy to sterilize,cheap  Povidine iodine, chlorohexidine or hydrogen peroxide may be toxic to the cells  Normal saline,Setrilized water,commercial wound cleansers
  • 29. IRRIGATION PRESSURE  No Consensus  Generally pressure irrigation beneficial  Very High pressures detrimental >8psi  Equipment used for irrigation has included  Bulb syringes, syringes with an attached needle or catheter,  Intra-venous or irrigation fluid in plastic containers with a pour cap or nozzle, and pressure canisters  Newer Calibrated devices in market  SOAKING NOT ADVISED
  • 31. IDENTIFICATION OF STRUCTURES  Neurovascular/Tendons  Identify  Spare  Protect  Tag  Repair /reconstruction  Early vs Delayed
  • 32. DEVITALISED TISSUE  Remove all contaminants and dead tissue meticulously while protecting viable tissues.  Keep reconstruction in mind  Tissues of uncertain viability may be left till the next session of surgical toilet so as to give them a chance for survival.  Dead muscle is identified by color and unresponsiveness to diathermy or cautery.
  • 34. WOUND CLOSURE  General Principles  Minor non contaminated ,early presentation -1 Closure  6-10hours –Extremety  10- 12hours –Scalp and face  Contaminated wounds- Debride, Dressing ,Relook 48-72hours
  • 35. WOUND CLOSURE  Wound closure methods could be by; Primary wound closure with sutures, staples, glue or tape  Delayed primary wound closure in wounds that are not clean and require either repeat debridement or cleaning before closure  Secondary intention, preserved for small wounds or in patients who cannot undergo surgery. Takes time and is generally discouraged,associated with scars and contracture formation.  Skin graft, for extensive wounds with good vascularity bed.  Flaps are recommended for wounds with poor vascularity base or with exposed bone and neuro-vascular structures are best covered by flaps
  • 41. Complications  Chronicity  Severe Infection  Wound dehiscence  Limb Loss  Osteomyelitis  Malignant Transformation

Editor's Notes

  1. Morelle lavel lesions