This document discusses protocols for wound debridement. It defines debridement as removing dead, contaminated, or adherent tissue from a wound to facilitate healing. The main types of debridement covered are mechanical, enzymatic, sharp, autolytic, and biologic. Characteristics of necrotic tissue like color, consistency, and adherence are reviewed. Protocols for sharp debridement emphasize preparing the patient, thoroughly removing necrotic tissue from the wound base outward until bleeding edges are seen, and irrigating and dressing the wound. The goal of debridement is to remove barriers to healing and reduce the bacterial burden.
Debridement is an important component of the wound bed preparation (WBP) management Model.
Cause of the wound and patient-centered concerns, debridement is a necessary step in local wound care.
Debridement is the removal of necrotic tissue, exudate, bacteria, and metabolic waste from a wound in order to improve or facilitate the healing process
ABDUL MANAN BIN OTHMAN
BSc (Hons) NPD Northumbria UK, CCWC (Mal)
Assistant Medical Officer
National Wound Care Committee
Wound Care Clinician
Kota Tinggi District Health Office
Debridement is an important component of the wound bed preparation (WBP) management Model.
Cause of the wound and patient-centered concerns, debridement is a necessary step in local wound care.
Debridement is the removal of necrotic tissue, exudate, bacteria, and metabolic waste from a wound in order to improve or facilitate the healing process
ABDUL MANAN BIN OTHMAN
BSc (Hons) NPD Northumbria UK, CCWC (Mal)
Assistant Medical Officer
National Wound Care Committee
Wound Care Clinician
Kota Tinggi District Health Office
different type of lower limb amputation with indication, peri-operative care, surgical steps, post op care complication and different type of prosthesis
different type of lower limb amputation with indication, peri-operative care, surgical steps, post op care complication and different type of prosthesis
There are two main categories of burn surgery: acute and reconstructive. ... It is delivered by a team of trauma surgeons (General Surgeons) that specialize in acute burn care. Complex burns often require consultation with plastic surgeons, who assist with the inpatient and outpatient management of these cases.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
Follow us on: Pinterest
Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
HOT NEW PRODUCT! BIG SALES FAST SHIPPING NOW FROM CHINA!! EU KU DB BK substit...GL Anaacs
Contact us if you are interested:
Email / Skype : kefaya1771@gmail.com
Threema: PXHY5PDH
New BATCH Ku !!! MUCH IN DEMAND FAST SALE EVERY BATCH HAPPY GOOD EFFECT BIG BATCH !
Contact me on Threema or skype to start big business!!
Hot-sale products:
NEW HOT EUTYLONE WHITE CRYSTAL!!
5cl-adba precursor (semi finished )
5cl-adba raw materials
ADBB precursor (semi finished )
ADBB raw materials
APVP powder
5fadb/4f-adb
Jwh018 / Jwh210
Eutylone crystal
Protonitazene (hydrochloride) CAS: 119276-01-6
Flubrotizolam CAS: 57801-95-3
Metonitazene CAS: 14680-51-4
Payment terms: Western Union,MoneyGram,Bitcoin or USDT.
Deliver Time: Usually 7-15days
Shipping method: FedEx, TNT, DHL,UPS etc.Our deliveries are 100% safe, fast, reliable and discreet.
Samples will be sent for your evaluation!If you are interested in, please contact me, let's talk details.
We specializes in exporting high quality Research chemical, medical intermediate, Pharmaceutical chemicals and so on. Products are exported to USA, Canada, France, Korea, Japan,Russia, Southeast Asia and other countries.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
4. Necrotic Tissue
• Necrotic tissue impairs wound healing as it is a physical
barrier to:
• Granulation tissue formation
• Wound contraction
• Re-epithelialization
• Necrotic tissue may also harbor bacteria, which could
lead to wound infection, thus impairing wound
healing
• The more necrotic tissue there is in a wound, the:
• More severe the damage is
• Longer it will take the close the wound
5. Necrotic Tissue
• As tissues die they change in:
• Color
• Consistency
• Adherence
6. Necrotic Tissue:Color
• As the depth/severity of the wound increases, the color
of the necrotic tissue changes:
• White/gray
• Tan/yellow
• Brown/black
7. Necrotic Tissue: Consistency
• As the tissues dry out, the consistency of the necrotic
tissue changes:
• Mucinous
• Soft, stringy
• Soft, soggy
• Hard
HardSoft, soggy
Soft, stringy
Mucinious
8. Necrotic Tissue : Adherence
• Adhesiveness of the debris to the wound bed and the
ease with which the two are separated
• Necrotic tissue tends to be more adherent:
• The deeper or more severe the damage is
• The less moist the wound is
11. Type of Necrosis :ByWound
Etiology
• Arterial/ischemic wounds:
• Dry gangrene
• Thick, dry, desiccated black/gray appearance
• Firmly adherent
• May be surrounded by an erythematous halo
• Neurotropic wounds:
• Do not present with necrotic tissue in wound typically
• Have hyperkeratosis surrounding wound
• Venous leg ulcers:
• Eschar or slough
• Usually yellow fibrous material
• Pressure Sores:
• Relates to the depth of the injury
13. What is Debridement?
• The process of removing dead, contaminated, or
adherent tissue and/or foreign material from a wound
• Five primary methods:
• Mechanical Debridement
• Enzymatic Debridement
• Sharp Debridement
• Autolytic Debridement
• Biologic Debridement
14. Mechanical Debridement:
• “The use of some outside force to remove dead tissue”,
i.e.:
• Wet to dry gauze dressings
• Wound irrigation
• Whirlpool
• Wet to dry gauze continues to be the most commonly
used debridement technique despite it’s multiple
disadvantages
15. Mechanical Debridement
Continued
• Advantages:
• Familiar to health care providers
• Wound irrigation can reduce bacterial burden
• Whirlpool may soften necrotic debris
• Disadvantages (wet-to-dry gauze):
• Non-selective
• Rarely applied correctly
• Painful
• More costly (labor and supplies)
• May cause maceration
• Releases airborne organisms and causes cross-
contamination
16. Enzymatic Debridement:
“Applying a concentrated, commercially prepared
(proteolytic) enzyme to the surface of the necrotic tissue,
in the expectation that it will aggressively degrade
necrosis by digesting devitalized tissue”
Cannot be used on dry wounds …
17. Enzymatic Debridement
Continued
Advantages:
Selective
Effective in combination with other debridement techniques
Disadvantages:
Enzymatic use is prolonged more than necessary, increasing
costs
Can be slow – 3-30 days to achieve a completely clean wound
bed (it is faster than autolysis however)
Requires a specific pH range (may cause local irritation due to pH
changes)
May be inactivated by contact with heavy metals (zinc or silver)
Risk of maceration and infection
Requires frequent dressing changes (1-3 times per day)
18. Sharp Debridement
• Performed either one time (surgical) or sequentially
(conservative)
• Surgical sharp debridement:
• Use of scalpel, scissors, or other sharp instruments
• Removal of viable and non-viable tissue
• Most rapid and effective
• May convert chronic wound into an acute wound
• Requires analgesics and availability of cautery equipment
• Indicated for removal of thick, adherent and/or large amounts of non-
viable tissue and when advancing cellulitis or signs of sepsis are present
• Requires a certain level of expertise, education and skill
• Risk of bleeding
19. Sharp Debridement :
Continued
• Conservative sharp wound debridement (CSWD):
• Use of scalpel, scissors, or other sharp instruments
• Rapid and effective
• Used in combination with enzymatic, mechanical,
and/or autolytic debridement to speed the removal of
non-viable necrotic debris/tissue
• Can be performed in any health-care setting by non-
physician clinicians (if they have the knowledge, skill,
judgment and authority to do so)..
20. Autolytic Debridement
The process of using the body’s own mechanisms (enzymes) to
remove nonviable tissue”
The collection of fluid at the wound site, “promotes rehydration
of the dead tissue and allows enzymes within the wound to
digest necrotic tissue”
May be accomplished by the use of any moisture-retentive
dressings, i.e. hydrocolloids, hydrogels, hypertonic
dressings/gels, and/or transparent films
21. Autolytic Debridement
Continued
Advantages:
Painless in the majority of people with wounds
Effective, versatile, and easy to perform
Selective
Low cost
Can be used in conjunction with other debridement
techniques
Disadvantages:
Slow
Caregiver education required for compliance
22. Biologic Debridement
A.k.a. larval/maggot debridement therapy (use of
medical grade green bottle fly larvae/maggots)
Controlled “application of disinfected maggots to the
wound
to remove the nonviable tissue”
Maggots are left in the wound for 2-3 days . They
secrete “proteolytic enzymes that break down
necrotic tissue and then ingest the liquefied tissue”
The secretions also have antimicrobial properties,
promote growth of human fibroblasts and improve
granulation tissue formation
23. Biologic Debridement
Continued:
Advantages:
Reduces bacterial burden
Growth-stimulating effects
Selective
Disadvantages:
Limited number of studies
‘Yuck factor’
Availability of sterile medical grade maggots
Lack of policies and procedures
24. Why Debride?
• To remove the physical barrier to
epidermal resurfacing, contraction, or
granulation
• To reduce bacteria burden by removing necrotic tissue
• To convert a chronic wound to an acute wound
by stimulating the healing cascade
• To facilitate earlier coverage of the
wound with active dressings or
biologicals
25. How Do WeDebride?
• After a thorough holistic assessment of the person and their wound,
and determination that debridement is indicated, you must first
choose the most appropriate type(s) of debridement. This is
dependent on the:
• Knowledge, skill and authority of the health care practitioner
• Availability of required resources
• Overall condition of the person with the wound, and their ‘heal
ability’
• Characteristics of the wound and wound tissue
• Presence of wound related pain
• Required speed and tissue selectivity of debridement
• Costs associated with available debridement techniques
• Presence of wound infection
• Physical environment
26. Red/Yellow/Black System
Red
Wound bed is clean and wound tissue is red/pink
Goal: maintain moist wound healing environment
Yellow
*
Wound bed has slough/fibrin present and tissue may be a combo of red/pink +
ivory/canary yellow/green (depending if infection is present)
Not all yellow is bad – granulation grows through yellow fibrin. Healthy tendon may appear white/yellow
Goal: maintain moist wound healing environment whilst managing excessive exudates and removing slough
via sharp, mechanical, enzymatic, and/or
autolytic debridement
Black
*
Wound bed has non-viable tissue present. Tissue combo may be dark brown/
grey/ black +/- red/pink +/- ivory/canary yellow/green.
Goal (healable wound and eschar is not stable and on heel): remove non-viable tissue via sharp, mechanical,
enzymatic and/or autolytic debridement
27. Zones of necrotic wound
There are three zones of necrotic wound
1. Zone of Coagulation
Containing maximum damaged area
2. Zone of Stasis
Containing thrombosed vessels and lymphatics
1. Zone of Hyperemia
Containing engorged vessels
28. Protocols of Sharp
debridement..
Most commonly used type of debridement in our setup..
Have thorough understanding of tissues
Preparing patient for debridement
Adequate resuscitation
Start broad spectrum antibiotics as soon as possible
Inform the patient about the procedure and its significance in wound healing
Procedure
Wear cap ,mask , gown and gloves
Apply disinfectant such as povidate iodine on and around the area
Drape the area properly
Start debriding from the base of the wound
29. The reason for starting from base of the wound is that
becox the blood from upper margins fall into base and
give false impression of red bleeding tissue
Be generous while debriding tissues and consider every
debridement as last debridement
Debride tissues until red bleeding margins are seen
The goal here is to excise the area having thrombosed vessels
and lymphatics
Irrigate the area with normal saline and bactericidal agent
such as hydrogen peroxide
Dry the area with clean sponge and do dressing