SlideShare a Scribd company logo
ZERA INTERNATIONAL COLLEGE
OF HEALTH SCIENCES
DEPARTMENT OF CLINICAL MEDICINE
MEDICAL PARASITOLOGY
LECTURE 19: PLATYHELMINTHS
CESTODES (TAPE WORMS)
TAENIA SAGINATA (BEEF TAPWORM) & TAENIA SOILUM (PORK TAPEWORM)
MRS N NG’ANDWE
BSC, BMS
Lecture outline
Quick recap of platyhelminths
Cestodes ( tape worms)
Taenia Solium & Taenia Saginata (pork & beef tapeworm respectively)
-History & distribution
-Habitat
-Morphology
-Life cycle
-Pathogensis & clinical features
-Diagnosis
-Treatment & prevention
History & Distribution
Common name
Taenia saginata Beef tapeworm
Taenia solium Pork tapeworm
T. saginata has been known as an intestinal parasite of man from very ancient times.
The name Taenia is derived from the Greek word meaning ‘tape or band’
T. saginata is worldwide in distribution, but the infection is not found in vegetarians and those
who do not eat beef.
History & distribution
T. solium is also worldwide in distribution except in the countries and communities, which
proscribe pork as taboo
Habitat
The adult worms of both T. saginata and T. solium live in the human small intestine, commonly in
the jejunum
Morphology
Adult Worm of T. saginata
The adult T. saginata worm is opalescent white in color, ribbonlike, dorsoventrally flattended and
segmented measuring 5–10 m in length
Adult Worm of T. solium
The adult worm is usually 23 meters long
Morphology
Eggs
Eggs of both species are indistinguishable.
They are spherical
The eggs of T. saginata are infective only to cattle and not to humans, whereas the eggs of T. solium
are infective to pigs and humans too.
Larva
The larval stage of Taenia is called as cysticercus.
Cysticercus bovis is the larva of T. saginata
Cysticercus cellulosae is the larva of T. solium
Life cycle of Taenia Saginata
T. saginata passes its life cycle in 2 hosts.
Definitive host: Humans are the definitive hosts and harbor the adult worm.
Intermediate host: Cattle (cow or buffalo) are the intermediate host and harbor the larval stage of
the worm.
Infective stage: Cysticercus bovis (larval stage) is the infective stage to man, while eggs are infective to
cattle.
The adult worm lives in the small intestine of man. The gravid segments from the adult worm
breakaway and are expelled singly.
They actively force their way out through the anal sphincter.
The eggs or gravid segments are passed out with feces on the ground.
The eggs deposited in soil remain viable for several weeks.
They are infective to cattle, which ingest the eggs while grazing
Development in cattle
When ingested by cattle (cow or buffalo), the eggshell ruptures releasing larva in the duodenum.
The larva, with their hooklets penetrate the intestinal wall, reach the mesenteric venules or
lymphatics and enter the systemic circulation.
They get filtered out in the striated muscles, particularly in muscles of the tongue, neck, shoulder,
ham, and in the myocardium. In these sites in about 60–70 days they develop in the mature larva,
cysticercus bovis.
The cysticerci can live in flesh of cattle for about 8 months, but can develop further only when
ingested by man, its definitive host
Development in Man
Man acquires infection by ingesting raw or undercooked beef containing cysticerci
The cysticerci are digested out of the meat in the stomach.
In the upper part of the small intestine, they become attached to the mucosa, and by gradual
strobilization develops into the adult worm in about 2–3 months.
The adult worm has a life span of 10 years or more.
Infection is usually with a single worm, but sometimes multiple infection is seen and 25 or more
worms have been reported in patients
Life cycle of Taenia Saginata
Life cycle of Taenia solium
•T. solium causes intestinal taeniasis, its life cycle is similar to that of T. saginata except….
•Definitive host: Man
•Intermediate host: Pig
•Infective stage: Cysticercus cellulosae (larva)
•Humans are infected by consuming inadequately cooked pork containing cysticercus cellulosae (measly
pork).
•Man harboring adult worms may autoinfect oneself either by unhygienic personal habits or by reverse
peristaltic movements of the intestine.
•When Taenia leads to cysticercosis, the life cycle is as follows:
•Definitive host and Intermediate host: Both man
•Infective stage: Eggs of T. solium (not larva)
•Mode of infection: Man acquires infection by ingesting eggs with contaminated food and water.
Life cycle of Taenia solium
•Autoinfection: A man harboring adult worm may autoinfect oneself, either by unhygenic
personal habits or by reverse peristalsis of the intestine.
•The further development of the eggs is similar in man and pigs.
•The larva are released in the duodenum or jejunum and penetrate the intestinal wall.
•They enter the mesenteric venules or lymphatics and are carried in systemic circulation to the
different parts of the body
Life cycle of Taenia solium
•They are filtered out principally in the muscles, where they develop into the larval stage, cysticercus
cellulosae in about 60–70 days.
•In humans, it is a dead end and the larvae die without further development
NOTE
•Intestinal infection with T. solium occurs only in persons eating undercooked pork and usually in
persons of low socioeconomic condition with poor sanitation
•It is uncommon in Jews and Mohammedans, who are not generally pork eaters.
•But cysticercosis may occur in any person residing in endemic areas, even in vegetarians because the
mode of Infection is contamination of food or drink with egg deposited in soil.
•Eggs of T. solium are infective to pigs as well as to man
Life cycle Taenia Solium
Pathogenesis & clinical features
Intestinal Taeniasis
•It can be caused by both T. saginata and T. solium.
•The adult worm, inspite of its large size, causes surprisingly little inconvenience to the patient.
•When the infection is symptomatic, vague abdominal discomformt, indigestion, nausea,
diarrhea, and weight loss may be present.
•Occasional cases of acute intestinal obstruction, acute appendicitis, and pancreatitis have also
been reported.
Pathogenesis & clinical features
Cysticercosis
•It is caused by larval stage (cysticecus cellulosae) of T. solium.
•Cysticercus cellulosae may be solitary or more often multiple.
•Any organ or tissue may be involved, the most common being subcutaneous tissues and
muscles.
•It may also affect the eyes, brain, and less often the heart, liver, lungs, abdominal cavity, and
spinal cord.
•The cysticercus is surrounded by a fibrous capsule except in the eye and ventricles of the brain.
•The larvae evoke a cellular reaction starting with infiltration of neutrophils, eosinophils,
lymphocytes, plasma cells, and at times, giant cells.
•This is followed by fibrosis and death of the larva with eventual calcifcation
Pathogenesis & clinical features
The clinical features depend on the site affected
Subcutaneous nodules are mostly asymptomatic
Muscular cysticerosis may cause acute myositis
Neurocysticerosis (cysticercosis of brain) is the most common and most serious form of cysticercosis.
About 70% of adult onset epilepsy is due to neurocysticercosis.
Other clinical features of neurocysticercosis are increased intracranial tension, hydrocephalus,
psychiatric disturbances, meningoencephalitis, transient paresis, behavioral disorders aphasia, and
visual disturbances.
In ocular cysticercosis, cysts are found in vitreous humor, subretinal space and conjunctiva.
The condition may present as blurred vision or loss of vision, iritis, uveitis, and palpebral conjunctivitis
Diagnosis
•Stool examination (Eggs in stool, antigen detection )
•Sero diagnosis
•Biopsy (cysticercosis)
•Imaging methods ( CT, MRI & Xray) in cysticercosis)
Treatment
Intestinal Taeniasis
•Single dose of praziquantel (10–20 mg/kg) is the drug of choice.
•Niclosamide (2 g), single dose, is another effective drug.
Cysticercosis
•For cysticercosis, excision is the best method, wherever possible.
•Asymptomatic neurocysticercocis requires no treatment.
Treatment
•For symptomatic cerebral cysticercosis, praziquantel in a dose of 50 mg/kg in 3 divided doses for 20–
30 days and albendazole in a dose of 400 mg twice daily for 30 days may be administered.
•Corticosteroids may be given along with praziquantel or albendazole to reduce the inflammatory
reactions caused by the dead cysticerci.
•In addition, antiepileptic drugs should be given until the reaction of the brain has subsided.
Operative intervention is indicated for hydrocephalus
Prevention
Beef and pork to be eaten by man should be subjected to effective inspection for cysticerci in
slaughter house.
Avoidance of eating raw or undercooked beef and pork.
The critical thermal point of cysticercus is 56°C for 5 minutes.
Maintenance of clean personal habits and general sanitary measures

More Related Content

Similar to Lecture 19 (Tapeworms).pptx

Cestode
CestodeCestode
Cestode
Aman Ullah
 
Taeniasis
TaeniasisTaeniasis
Taeniasis
Microbiology
 
Taenia solium & Taenia saginata
Taenia solium & Taenia saginataTaenia solium & Taenia saginata
Taenia solium & Taenia saginata
Priyanka Patel
 
Lecture 12 Metazoa_1.pptx
Lecture 12 Metazoa_1.pptxLecture 12 Metazoa_1.pptx
Lecture 12 Metazoa_1.pptx
FLOWERSOFPAKISTAN
 
Arushi Gupta taenia.pptx
Arushi Gupta taenia.pptxArushi Gupta taenia.pptx
Arushi Gupta taenia.pptx
ArushiGupta443767
 
Tapeworms kb labs
Tapeworms   kb labsTapeworms   kb labs
Tapeworms kb labs
dotadota
 
Taenia solium, saginata & neurocysticercosis
Taenia solium, saginata & neurocysticercosisTaenia solium, saginata & neurocysticercosis
Taenia solium, saginata & neurocysticercosis
Menal Wali
 
Taeniasis
TaeniasisTaeniasis
Taeniasis
Vanindar Kumar
 
Taenia saginata
Taenia  saginataTaenia  saginata
Taenia saginata
PriyankaSilwal
 
Cestodes (The Taeenid tapeworms)
Cestodes (The Taeenid tapeworms)Cestodes (The Taeenid tapeworms)
Cestodes (The Taeenid tapeworms)
med zar
 
13_Taenia saginata.pdfchresounvivesity lecturer notes
13_Taenia saginata.pdfchresounvivesity lecturer notes13_Taenia saginata.pdfchresounvivesity lecturer notes
13_Taenia saginata.pdfchresounvivesity lecturer notes
IanLubanga
 
Cestodes PHO.pptx
Cestodes PHO.pptxCestodes PHO.pptx
Cestodes PHO.pptx
Obsa2
 
Cystercercosis and cases0.pptx
Cystercercosis and cases0.pptxCystercercosis and cases0.pptx
Cystercercosis and cases0.pptx
TharushiCiriatthage
 
Parasitology helmith 2011
Parasitology helmith 2011Parasitology helmith 2011
Parasitology helmith 2011
Mercury Lin
 
Parasites in histopathology
Parasites in histopathologyParasites in histopathology
Parasites in histopathology
Dr. Varughese George
 
Cysticercosis
CysticercosisCysticercosis
Cysticercosis
OPTOM FASLU MUHAMMED
 
Class Trematoda. Tapeworms as parasites of human being
Class Trematoda. Tapeworms as parasites of human beingClass Trematoda. Tapeworms as parasites of human being
Class Trematoda. Tapeworms as parasites of human being
Eneutron
 
L2 ASCARIASIS.ppt
L2 ASCARIASIS.pptL2 ASCARIASIS.ppt
L2 ASCARIASIS.ppt
NaaelHAli1
 

Similar to Lecture 19 (Tapeworms).pptx (20)

Cestode
CestodeCestode
Cestode
 
Taeniasis
TaeniasisTaeniasis
Taeniasis
 
Taenia solium & Taenia saginata
Taenia solium & Taenia saginataTaenia solium & Taenia saginata
Taenia solium & Taenia saginata
 
Lecture 12 Metazoa_1.pptx
Lecture 12 Metazoa_1.pptxLecture 12 Metazoa_1.pptx
Lecture 12 Metazoa_1.pptx
 
Taenia Solium and Taenia Saginata
Taenia Solium and Taenia SaginataTaenia Solium and Taenia Saginata
Taenia Solium and Taenia Saginata
 
Arushi Gupta taenia.pptx
Arushi Gupta taenia.pptxArushi Gupta taenia.pptx
Arushi Gupta taenia.pptx
 
Tapeworms kb labs
Tapeworms   kb labsTapeworms   kb labs
Tapeworms kb labs
 
Taenia solium, saginata & neurocysticercosis
Taenia solium, saginata & neurocysticercosisTaenia solium, saginata & neurocysticercosis
Taenia solium, saginata & neurocysticercosis
 
Taeniasis
TaeniasisTaeniasis
Taeniasis
 
Taenia saginata
Taenia  saginataTaenia  saginata
Taenia saginata
 
Cestodes (The Taeenid tapeworms)
Cestodes (The Taeenid tapeworms)Cestodes (The Taeenid tapeworms)
Cestodes (The Taeenid tapeworms)
 
13_Taenia saginata.pdfchresounvivesity lecturer notes
13_Taenia saginata.pdfchresounvivesity lecturer notes13_Taenia saginata.pdfchresounvivesity lecturer notes
13_Taenia saginata.pdfchresounvivesity lecturer notes
 
Cestodes PHO.pptx
Cestodes PHO.pptxCestodes PHO.pptx
Cestodes PHO.pptx
 
Cystercercosis and cases0.pptx
Cystercercosis and cases0.pptxCystercercosis and cases0.pptx
Cystercercosis and cases0.pptx
 
Parasitology helmith 2011
Parasitology helmith 2011Parasitology helmith 2011
Parasitology helmith 2011
 
Parasites in histopathology
Parasites in histopathologyParasites in histopathology
Parasites in histopathology
 
Cysticercosis
CysticercosisCysticercosis
Cysticercosis
 
Class Trematoda. Tapeworms as parasites of human being
Class Trematoda. Tapeworms as parasites of human beingClass Trematoda. Tapeworms as parasites of human being
Class Trematoda. Tapeworms as parasites of human being
 
Nematodoses10
Nematodoses10Nematodoses10
Nematodoses10
 
L2 ASCARIASIS.ppt
L2 ASCARIASIS.pptL2 ASCARIASIS.ppt
L2 ASCARIASIS.ppt
 

More from NellyPhiri5

Lecture 3 -Screening tests.ppt presentation
Lecture 3 -Screening tests.ppt presentationLecture 3 -Screening tests.ppt presentation
Lecture 3 -Screening tests.ppt presentation
NellyPhiri5
 
Lecture 16 APH.pptx. power point lecture
Lecture 16 APH.pptx. power point lectureLecture 16 APH.pptx. power point lecture
Lecture 16 APH.pptx. power point lecture
NellyPhiri5
 
BURNS.pptx lecture presentation surgery.
BURNS.pptx lecture presentation surgery.BURNS.pptx lecture presentation surgery.
BURNS.pptx lecture presentation surgery.
NellyPhiri5
 
SUTURING AND SUTURE MATERIALS.lecture.pptx
SUTURING AND SUTURE MATERIALS.lecture.pptxSUTURING AND SUTURE MATERIALS.lecture.pptx
SUTURING AND SUTURE MATERIALS.lecture.pptx
NellyPhiri5
 
TYPES OF INCISIONS. presentation lecture
TYPES OF INCISIONS. presentation lectureTYPES OF INCISIONS. presentation lecture
TYPES OF INCISIONS. presentation lecture
NellyPhiri5
 
Spirochetes ppt microbiology and immunology
Spirochetes ppt  microbiology and immunologySpirochetes ppt  microbiology and immunology
Spirochetes ppt microbiology and immunology
NellyPhiri5
 
Contraception_Lecture_i mweene.pptx
Contraception_Lecture_i mweene.pptxContraception_Lecture_i mweene.pptx
Contraception_Lecture_i mweene.pptx
NellyPhiri5
 
Lecture 3 (T. cruzi) .pptx
Lecture 3 (T. cruzi) .pptxLecture 3 (T. cruzi) .pptx
Lecture 3 (T. cruzi) .pptx
NellyPhiri5
 
approachtopleuraleffusion-140205151929-phpapp01.pdf
approachtopleuraleffusion-140205151929-phpapp01.pdfapproachtopleuraleffusion-140205151929-phpapp01.pdf
approachtopleuraleffusion-140205151929-phpapp01.pdf
NellyPhiri5
 
12.2NUCLEOTIDES & NUCLEIC ACIDS-1.pptx
12.2NUCLEOTIDES & NUCLEIC ACIDS-1.pptx12.2NUCLEOTIDES & NUCLEIC ACIDS-1.pptx
12.2NUCLEOTIDES & NUCLEIC ACIDS-1.pptx
NellyPhiri5
 
shockbyara1-141208091205-conversion-gate02 (1).pdf
shockbyara1-141208091205-conversion-gate02 (1).pdfshockbyara1-141208091205-conversion-gate02 (1).pdf
shockbyara1-141208091205-conversion-gate02 (1).pdf
NellyPhiri5
 
Lecture 12 (G+ & G- cocci ;STAPH& NEISSERIA).pptx
Lecture 12 (G+ & G- cocci ;STAPH& NEISSERIA).pptxLecture 12 (G+ & G- cocci ;STAPH& NEISSERIA).pptx
Lecture 12 (G+ & G- cocci ;STAPH& NEISSERIA).pptx
NellyPhiri5
 
hematology.pptx
hematology.pptxhematology.pptx
hematology.pptx
NellyPhiri5
 
Lecture 2 - NATURAL HISTORY OF A DISEASE.pptx
Lecture 2 - NATURAL HISTORY OF A DISEASE.pptxLecture 2 - NATURAL HISTORY OF A DISEASE.pptx
Lecture 2 - NATURAL HISTORY OF A DISEASE.pptx
NellyPhiri5
 
Legg-Calve-Perthes Disease.pptx
Legg-Calve-Perthes Disease.pptxLegg-Calve-Perthes Disease.pptx
Legg-Calve-Perthes Disease.pptx
NellyPhiri5
 
FOURNIER’S GANGRENE.pptx
FOURNIER’S GANGRENE.pptxFOURNIER’S GANGRENE.pptx
FOURNIER’S GANGRENE.pptx
NellyPhiri5
 
epistaxis.pptx
epistaxis.pptxepistaxis.pptx
epistaxis.pptx
NellyPhiri5
 
Lecture 18B (Viruses).pptx
Lecture 18B (Viruses).pptxLecture 18B (Viruses).pptx
Lecture 18B (Viruses).pptx
NellyPhiri5
 
SURGICAL_WOUNDS,CLASSIFICATIONS,WOUND_HEALING_AND_COMPLICATIONS.ppt
SURGICAL_WOUNDS,CLASSIFICATIONS,WOUND_HEALING_AND_COMPLICATIONS.pptSURGICAL_WOUNDS,CLASSIFICATIONS,WOUND_HEALING_AND_COMPLICATIONS.ppt
SURGICAL_WOUNDS,CLASSIFICATIONS,WOUND_HEALING_AND_COMPLICATIONS.ppt
NellyPhiri5
 
OPEN FRACTURES.pptx
OPEN FRACTURES.pptxOPEN FRACTURES.pptx
OPEN FRACTURES.pptx
NellyPhiri5
 

More from NellyPhiri5 (20)

Lecture 3 -Screening tests.ppt presentation
Lecture 3 -Screening tests.ppt presentationLecture 3 -Screening tests.ppt presentation
Lecture 3 -Screening tests.ppt presentation
 
Lecture 16 APH.pptx. power point lecture
Lecture 16 APH.pptx. power point lectureLecture 16 APH.pptx. power point lecture
Lecture 16 APH.pptx. power point lecture
 
BURNS.pptx lecture presentation surgery.
BURNS.pptx lecture presentation surgery.BURNS.pptx lecture presentation surgery.
BURNS.pptx lecture presentation surgery.
 
SUTURING AND SUTURE MATERIALS.lecture.pptx
SUTURING AND SUTURE MATERIALS.lecture.pptxSUTURING AND SUTURE MATERIALS.lecture.pptx
SUTURING AND SUTURE MATERIALS.lecture.pptx
 
TYPES OF INCISIONS. presentation lecture
TYPES OF INCISIONS. presentation lectureTYPES OF INCISIONS. presentation lecture
TYPES OF INCISIONS. presentation lecture
 
Spirochetes ppt microbiology and immunology
Spirochetes ppt  microbiology and immunologySpirochetes ppt  microbiology and immunology
Spirochetes ppt microbiology and immunology
 
Contraception_Lecture_i mweene.pptx
Contraception_Lecture_i mweene.pptxContraception_Lecture_i mweene.pptx
Contraception_Lecture_i mweene.pptx
 
Lecture 3 (T. cruzi) .pptx
Lecture 3 (T. cruzi) .pptxLecture 3 (T. cruzi) .pptx
Lecture 3 (T. cruzi) .pptx
 
approachtopleuraleffusion-140205151929-phpapp01.pdf
approachtopleuraleffusion-140205151929-phpapp01.pdfapproachtopleuraleffusion-140205151929-phpapp01.pdf
approachtopleuraleffusion-140205151929-phpapp01.pdf
 
12.2NUCLEOTIDES & NUCLEIC ACIDS-1.pptx
12.2NUCLEOTIDES & NUCLEIC ACIDS-1.pptx12.2NUCLEOTIDES & NUCLEIC ACIDS-1.pptx
12.2NUCLEOTIDES & NUCLEIC ACIDS-1.pptx
 
shockbyara1-141208091205-conversion-gate02 (1).pdf
shockbyara1-141208091205-conversion-gate02 (1).pdfshockbyara1-141208091205-conversion-gate02 (1).pdf
shockbyara1-141208091205-conversion-gate02 (1).pdf
 
Lecture 12 (G+ & G- cocci ;STAPH& NEISSERIA).pptx
Lecture 12 (G+ & G- cocci ;STAPH& NEISSERIA).pptxLecture 12 (G+ & G- cocci ;STAPH& NEISSERIA).pptx
Lecture 12 (G+ & G- cocci ;STAPH& NEISSERIA).pptx
 
hematology.pptx
hematology.pptxhematology.pptx
hematology.pptx
 
Lecture 2 - NATURAL HISTORY OF A DISEASE.pptx
Lecture 2 - NATURAL HISTORY OF A DISEASE.pptxLecture 2 - NATURAL HISTORY OF A DISEASE.pptx
Lecture 2 - NATURAL HISTORY OF A DISEASE.pptx
 
Legg-Calve-Perthes Disease.pptx
Legg-Calve-Perthes Disease.pptxLegg-Calve-Perthes Disease.pptx
Legg-Calve-Perthes Disease.pptx
 
FOURNIER’S GANGRENE.pptx
FOURNIER’S GANGRENE.pptxFOURNIER’S GANGRENE.pptx
FOURNIER’S GANGRENE.pptx
 
epistaxis.pptx
epistaxis.pptxepistaxis.pptx
epistaxis.pptx
 
Lecture 18B (Viruses).pptx
Lecture 18B (Viruses).pptxLecture 18B (Viruses).pptx
Lecture 18B (Viruses).pptx
 
SURGICAL_WOUNDS,CLASSIFICATIONS,WOUND_HEALING_AND_COMPLICATIONS.ppt
SURGICAL_WOUNDS,CLASSIFICATIONS,WOUND_HEALING_AND_COMPLICATIONS.pptSURGICAL_WOUNDS,CLASSIFICATIONS,WOUND_HEALING_AND_COMPLICATIONS.ppt
SURGICAL_WOUNDS,CLASSIFICATIONS,WOUND_HEALING_AND_COMPLICATIONS.ppt
 
OPEN FRACTURES.pptx
OPEN FRACTURES.pptxOPEN FRACTURES.pptx
OPEN FRACTURES.pptx
 

Recently uploaded

TESDA TM1 REVIEWER FOR NATIONAL ASSESSMENT WRITTEN AND ORAL QUESTIONS WITH A...
TESDA TM1 REVIEWER  FOR NATIONAL ASSESSMENT WRITTEN AND ORAL QUESTIONS WITH A...TESDA TM1 REVIEWER  FOR NATIONAL ASSESSMENT WRITTEN AND ORAL QUESTIONS WITH A...
TESDA TM1 REVIEWER FOR NATIONAL ASSESSMENT WRITTEN AND ORAL QUESTIONS WITH A...
EugeneSaldivar
 
Fish and Chips - have they had their chips
Fish and Chips - have they had their chipsFish and Chips - have they had their chips
Fish and Chips - have they had their chips
GeoBlogs
 
Sectors of the Indian Economy - Class 10 Study Notes pdf
Sectors of the Indian Economy - Class 10 Study Notes pdfSectors of the Indian Economy - Class 10 Study Notes pdf
Sectors of the Indian Economy - Class 10 Study Notes pdf
Vivekanand Anglo Vedic Academy
 
Overview on Edible Vaccine: Pros & Cons with Mechanism
Overview on Edible Vaccine: Pros & Cons with MechanismOverview on Edible Vaccine: Pros & Cons with Mechanism
Overview on Edible Vaccine: Pros & Cons with Mechanism
DeeptiGupta154
 
Sha'Carri Richardson Presentation 202345
Sha'Carri Richardson Presentation 202345Sha'Carri Richardson Presentation 202345
Sha'Carri Richardson Presentation 202345
beazzy04
 
Operation Blue Star - Saka Neela Tara
Operation Blue Star   -  Saka Neela TaraOperation Blue Star   -  Saka Neela Tara
Operation Blue Star - Saka Neela Tara
Balvir Singh
 
The French Revolution Class 9 Study Material pdf free download
The French Revolution Class 9 Study Material pdf free downloadThe French Revolution Class 9 Study Material pdf free download
The French Revolution Class 9 Study Material pdf free download
Vivekanand Anglo Vedic Academy
 
Chapter 3 - Islamic Banking Products and Services.pptx
Chapter 3 - Islamic Banking Products and Services.pptxChapter 3 - Islamic Banking Products and Services.pptx
Chapter 3 - Islamic Banking Products and Services.pptx
Mohd Adib Abd Muin, Senior Lecturer at Universiti Utara Malaysia
 
Additional Benefits for Employee Website.pdf
Additional Benefits for Employee Website.pdfAdditional Benefits for Employee Website.pdf
Additional Benefits for Employee Website.pdf
joachimlavalley1
 
Students, digital devices and success - Andreas Schleicher - 27 May 2024..pptx
Students, digital devices and success - Andreas Schleicher - 27 May 2024..pptxStudents, digital devices and success - Andreas Schleicher - 27 May 2024..pptx
Students, digital devices and success - Andreas Schleicher - 27 May 2024..pptx
EduSkills OECD
 
Synthetic Fiber Construction in lab .pptx
Synthetic Fiber Construction in lab .pptxSynthetic Fiber Construction in lab .pptx
Synthetic Fiber Construction in lab .pptx
Pavel ( NSTU)
 
aaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaa
aaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaa
aaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaa
siemaillard
 
Unit 8 - Information and Communication Technology (Paper I).pdf
Unit 8 - Information and Communication Technology (Paper I).pdfUnit 8 - Information and Communication Technology (Paper I).pdf
Unit 8 - Information and Communication Technology (Paper I).pdf
Thiyagu K
 
Cambridge International AS A Level Biology Coursebook - EBook (MaryFosbery J...
Cambridge International AS  A Level Biology Coursebook - EBook (MaryFosbery J...Cambridge International AS  A Level Biology Coursebook - EBook (MaryFosbery J...
Cambridge International AS A Level Biology Coursebook - EBook (MaryFosbery J...
AzmatAli747758
 
CLASS 11 CBSE B.St Project AIDS TO TRADE - INSURANCE
CLASS 11 CBSE B.St Project AIDS TO TRADE - INSURANCECLASS 11 CBSE B.St Project AIDS TO TRADE - INSURANCE
CLASS 11 CBSE B.St Project AIDS TO TRADE - INSURANCE
BhavyaRajput3
 
PART A. Introduction to Costumer Service
PART A. Introduction to Costumer ServicePART A. Introduction to Costumer Service
PART A. Introduction to Costumer Service
PedroFerreira53928
 
Language Across the Curriculm LAC B.Ed.
Language Across the  Curriculm LAC B.Ed.Language Across the  Curriculm LAC B.Ed.
Language Across the Curriculm LAC B.Ed.
Atul Kumar Singh
 
Polish students' mobility in the Czech Republic
Polish students' mobility in the Czech RepublicPolish students' mobility in the Czech Republic
Polish students' mobility in the Czech Republic
Anna Sz.
 
The Roman Empire A Historical Colossus.pdf
The Roman Empire A Historical Colossus.pdfThe Roman Empire A Historical Colossus.pdf
The Roman Empire A Historical Colossus.pdf
kaushalkr1407
 
How to Break the cycle of negative Thoughts
How to Break the cycle of negative ThoughtsHow to Break the cycle of negative Thoughts
How to Break the cycle of negative Thoughts
Col Mukteshwar Prasad
 

Recently uploaded (20)

TESDA TM1 REVIEWER FOR NATIONAL ASSESSMENT WRITTEN AND ORAL QUESTIONS WITH A...
TESDA TM1 REVIEWER  FOR NATIONAL ASSESSMENT WRITTEN AND ORAL QUESTIONS WITH A...TESDA TM1 REVIEWER  FOR NATIONAL ASSESSMENT WRITTEN AND ORAL QUESTIONS WITH A...
TESDA TM1 REVIEWER FOR NATIONAL ASSESSMENT WRITTEN AND ORAL QUESTIONS WITH A...
 
Fish and Chips - have they had their chips
Fish and Chips - have they had their chipsFish and Chips - have they had their chips
Fish and Chips - have they had their chips
 
Sectors of the Indian Economy - Class 10 Study Notes pdf
Sectors of the Indian Economy - Class 10 Study Notes pdfSectors of the Indian Economy - Class 10 Study Notes pdf
Sectors of the Indian Economy - Class 10 Study Notes pdf
 
Overview on Edible Vaccine: Pros & Cons with Mechanism
Overview on Edible Vaccine: Pros & Cons with MechanismOverview on Edible Vaccine: Pros & Cons with Mechanism
Overview on Edible Vaccine: Pros & Cons with Mechanism
 
Sha'Carri Richardson Presentation 202345
Sha'Carri Richardson Presentation 202345Sha'Carri Richardson Presentation 202345
Sha'Carri Richardson Presentation 202345
 
Operation Blue Star - Saka Neela Tara
Operation Blue Star   -  Saka Neela TaraOperation Blue Star   -  Saka Neela Tara
Operation Blue Star - Saka Neela Tara
 
The French Revolution Class 9 Study Material pdf free download
The French Revolution Class 9 Study Material pdf free downloadThe French Revolution Class 9 Study Material pdf free download
The French Revolution Class 9 Study Material pdf free download
 
Chapter 3 - Islamic Banking Products and Services.pptx
Chapter 3 - Islamic Banking Products and Services.pptxChapter 3 - Islamic Banking Products and Services.pptx
Chapter 3 - Islamic Banking Products and Services.pptx
 
Additional Benefits for Employee Website.pdf
Additional Benefits for Employee Website.pdfAdditional Benefits for Employee Website.pdf
Additional Benefits for Employee Website.pdf
 
Students, digital devices and success - Andreas Schleicher - 27 May 2024..pptx
Students, digital devices and success - Andreas Schleicher - 27 May 2024..pptxStudents, digital devices and success - Andreas Schleicher - 27 May 2024..pptx
Students, digital devices and success - Andreas Schleicher - 27 May 2024..pptx
 
Synthetic Fiber Construction in lab .pptx
Synthetic Fiber Construction in lab .pptxSynthetic Fiber Construction in lab .pptx
Synthetic Fiber Construction in lab .pptx
 
aaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaa
aaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaa
aaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaa
 
Unit 8 - Information and Communication Technology (Paper I).pdf
Unit 8 - Information and Communication Technology (Paper I).pdfUnit 8 - Information and Communication Technology (Paper I).pdf
Unit 8 - Information and Communication Technology (Paper I).pdf
 
Cambridge International AS A Level Biology Coursebook - EBook (MaryFosbery J...
Cambridge International AS  A Level Biology Coursebook - EBook (MaryFosbery J...Cambridge International AS  A Level Biology Coursebook - EBook (MaryFosbery J...
Cambridge International AS A Level Biology Coursebook - EBook (MaryFosbery J...
 
CLASS 11 CBSE B.St Project AIDS TO TRADE - INSURANCE
CLASS 11 CBSE B.St Project AIDS TO TRADE - INSURANCECLASS 11 CBSE B.St Project AIDS TO TRADE - INSURANCE
CLASS 11 CBSE B.St Project AIDS TO TRADE - INSURANCE
 
PART A. Introduction to Costumer Service
PART A. Introduction to Costumer ServicePART A. Introduction to Costumer Service
PART A. Introduction to Costumer Service
 
Language Across the Curriculm LAC B.Ed.
Language Across the  Curriculm LAC B.Ed.Language Across the  Curriculm LAC B.Ed.
Language Across the Curriculm LAC B.Ed.
 
Polish students' mobility in the Czech Republic
Polish students' mobility in the Czech RepublicPolish students' mobility in the Czech Republic
Polish students' mobility in the Czech Republic
 
The Roman Empire A Historical Colossus.pdf
The Roman Empire A Historical Colossus.pdfThe Roman Empire A Historical Colossus.pdf
The Roman Empire A Historical Colossus.pdf
 
How to Break the cycle of negative Thoughts
How to Break the cycle of negative ThoughtsHow to Break the cycle of negative Thoughts
How to Break the cycle of negative Thoughts
 

Lecture 19 (Tapeworms).pptx

  • 1. ZERA INTERNATIONAL COLLEGE OF HEALTH SCIENCES DEPARTMENT OF CLINICAL MEDICINE MEDICAL PARASITOLOGY LECTURE 19: PLATYHELMINTHS CESTODES (TAPE WORMS) TAENIA SAGINATA (BEEF TAPWORM) & TAENIA SOILUM (PORK TAPEWORM) MRS N NG’ANDWE BSC, BMS
  • 2. Lecture outline Quick recap of platyhelminths Cestodes ( tape worms) Taenia Solium & Taenia Saginata (pork & beef tapeworm respectively) -History & distribution -Habitat -Morphology -Life cycle -Pathogensis & clinical features -Diagnosis -Treatment & prevention
  • 3. History & Distribution Common name Taenia saginata Beef tapeworm Taenia solium Pork tapeworm T. saginata has been known as an intestinal parasite of man from very ancient times. The name Taenia is derived from the Greek word meaning ‘tape or band’ T. saginata is worldwide in distribution, but the infection is not found in vegetarians and those who do not eat beef.
  • 4. History & distribution T. solium is also worldwide in distribution except in the countries and communities, which proscribe pork as taboo
  • 5. Habitat The adult worms of both T. saginata and T. solium live in the human small intestine, commonly in the jejunum
  • 6. Morphology Adult Worm of T. saginata The adult T. saginata worm is opalescent white in color, ribbonlike, dorsoventrally flattended and segmented measuring 5–10 m in length Adult Worm of T. solium The adult worm is usually 23 meters long
  • 7. Morphology Eggs Eggs of both species are indistinguishable. They are spherical The eggs of T. saginata are infective only to cattle and not to humans, whereas the eggs of T. solium are infective to pigs and humans too. Larva The larval stage of Taenia is called as cysticercus. Cysticercus bovis is the larva of T. saginata Cysticercus cellulosae is the larva of T. solium
  • 8.
  • 9. Life cycle of Taenia Saginata T. saginata passes its life cycle in 2 hosts. Definitive host: Humans are the definitive hosts and harbor the adult worm. Intermediate host: Cattle (cow or buffalo) are the intermediate host and harbor the larval stage of the worm. Infective stage: Cysticercus bovis (larval stage) is the infective stage to man, while eggs are infective to cattle. The adult worm lives in the small intestine of man. The gravid segments from the adult worm breakaway and are expelled singly. They actively force their way out through the anal sphincter. The eggs or gravid segments are passed out with feces on the ground. The eggs deposited in soil remain viable for several weeks. They are infective to cattle, which ingest the eggs while grazing
  • 10. Development in cattle When ingested by cattle (cow or buffalo), the eggshell ruptures releasing larva in the duodenum. The larva, with their hooklets penetrate the intestinal wall, reach the mesenteric venules or lymphatics and enter the systemic circulation. They get filtered out in the striated muscles, particularly in muscles of the tongue, neck, shoulder, ham, and in the myocardium. In these sites in about 60–70 days they develop in the mature larva, cysticercus bovis. The cysticerci can live in flesh of cattle for about 8 months, but can develop further only when ingested by man, its definitive host
  • 11. Development in Man Man acquires infection by ingesting raw or undercooked beef containing cysticerci The cysticerci are digested out of the meat in the stomach. In the upper part of the small intestine, they become attached to the mucosa, and by gradual strobilization develops into the adult worm in about 2–3 months. The adult worm has a life span of 10 years or more. Infection is usually with a single worm, but sometimes multiple infection is seen and 25 or more worms have been reported in patients
  • 12. Life cycle of Taenia Saginata
  • 13. Life cycle of Taenia solium •T. solium causes intestinal taeniasis, its life cycle is similar to that of T. saginata except…. •Definitive host: Man •Intermediate host: Pig •Infective stage: Cysticercus cellulosae (larva) •Humans are infected by consuming inadequately cooked pork containing cysticercus cellulosae (measly pork). •Man harboring adult worms may autoinfect oneself either by unhygienic personal habits or by reverse peristaltic movements of the intestine. •When Taenia leads to cysticercosis, the life cycle is as follows: •Definitive host and Intermediate host: Both man •Infective stage: Eggs of T. solium (not larva) •Mode of infection: Man acquires infection by ingesting eggs with contaminated food and water.
  • 14. Life cycle of Taenia solium •Autoinfection: A man harboring adult worm may autoinfect oneself, either by unhygenic personal habits or by reverse peristalsis of the intestine. •The further development of the eggs is similar in man and pigs. •The larva are released in the duodenum or jejunum and penetrate the intestinal wall. •They enter the mesenteric venules or lymphatics and are carried in systemic circulation to the different parts of the body
  • 15. Life cycle of Taenia solium •They are filtered out principally in the muscles, where they develop into the larval stage, cysticercus cellulosae in about 60–70 days. •In humans, it is a dead end and the larvae die without further development NOTE •Intestinal infection with T. solium occurs only in persons eating undercooked pork and usually in persons of low socioeconomic condition with poor sanitation •It is uncommon in Jews and Mohammedans, who are not generally pork eaters. •But cysticercosis may occur in any person residing in endemic areas, even in vegetarians because the mode of Infection is contamination of food or drink with egg deposited in soil. •Eggs of T. solium are infective to pigs as well as to man
  • 17. Pathogenesis & clinical features Intestinal Taeniasis •It can be caused by both T. saginata and T. solium. •The adult worm, inspite of its large size, causes surprisingly little inconvenience to the patient. •When the infection is symptomatic, vague abdominal discomformt, indigestion, nausea, diarrhea, and weight loss may be present. •Occasional cases of acute intestinal obstruction, acute appendicitis, and pancreatitis have also been reported.
  • 18. Pathogenesis & clinical features Cysticercosis •It is caused by larval stage (cysticecus cellulosae) of T. solium. •Cysticercus cellulosae may be solitary or more often multiple. •Any organ or tissue may be involved, the most common being subcutaneous tissues and muscles. •It may also affect the eyes, brain, and less often the heart, liver, lungs, abdominal cavity, and spinal cord. •The cysticercus is surrounded by a fibrous capsule except in the eye and ventricles of the brain. •The larvae evoke a cellular reaction starting with infiltration of neutrophils, eosinophils, lymphocytes, plasma cells, and at times, giant cells. •This is followed by fibrosis and death of the larva with eventual calcifcation
  • 19. Pathogenesis & clinical features The clinical features depend on the site affected Subcutaneous nodules are mostly asymptomatic Muscular cysticerosis may cause acute myositis Neurocysticerosis (cysticercosis of brain) is the most common and most serious form of cysticercosis. About 70% of adult onset epilepsy is due to neurocysticercosis. Other clinical features of neurocysticercosis are increased intracranial tension, hydrocephalus, psychiatric disturbances, meningoencephalitis, transient paresis, behavioral disorders aphasia, and visual disturbances. In ocular cysticercosis, cysts are found in vitreous humor, subretinal space and conjunctiva. The condition may present as blurred vision or loss of vision, iritis, uveitis, and palpebral conjunctivitis
  • 20. Diagnosis •Stool examination (Eggs in stool, antigen detection ) •Sero diagnosis •Biopsy (cysticercosis) •Imaging methods ( CT, MRI & Xray) in cysticercosis)
  • 21. Treatment Intestinal Taeniasis •Single dose of praziquantel (10–20 mg/kg) is the drug of choice. •Niclosamide (2 g), single dose, is another effective drug. Cysticercosis •For cysticercosis, excision is the best method, wherever possible. •Asymptomatic neurocysticercocis requires no treatment.
  • 22. Treatment •For symptomatic cerebral cysticercosis, praziquantel in a dose of 50 mg/kg in 3 divided doses for 20– 30 days and albendazole in a dose of 400 mg twice daily for 30 days may be administered. •Corticosteroids may be given along with praziquantel or albendazole to reduce the inflammatory reactions caused by the dead cysticerci. •In addition, antiepileptic drugs should be given until the reaction of the brain has subsided. Operative intervention is indicated for hydrocephalus
  • 23. Prevention Beef and pork to be eaten by man should be subjected to effective inspection for cysticerci in slaughter house. Avoidance of eating raw or undercooked beef and pork. The critical thermal point of cysticercus is 56°C for 5 minutes. Maintenance of clean personal habits and general sanitary measures