ECT is mainly used to treat severe depression when antidepressant drugs and therapy have failed or the person is at risk of suicide. Modern ECT involves placing electrodes on the head and passing a small electric current through the brain to induce a seizure, similar to an epileptic fit. A course of ECT usually involves 3 to 15 treatments, and has risks similar to minor surgery but can effectively treat life-threatening depression for 60-70% of patients. However, ECT remains controversial due to concerns about side effects like memory impairment and because of its past portrayal and use.
Goldman grand rounds, electroconvulsive therapy (ect)...mysterious rebootLisa E Goldman, MD, MSW
How does ECT work? Is it dangerous? Is it scary? Does it work? What mental health conditions can be effectively treated by ECT? Is it true that ECT works better than prescription antidepressants? What are common side effects of ECT and how are this managed? Has the practice of ECT changed over time? What patient factors are the best predictors of positive and negative outcomes of ECT treatment? Come with me on a journey as I present my grand rounds lecture for the department of psychiatry as I complete my 4th year of psychiatry residency at UTHSC Memphis. These are exciting times. Recovery is definitely an option.
The presentation part of a two-day workshop on ECT held at al-Hussain University Hospital of al-Azhar Univeristy. It was accompanied by one-to-one training on the Thymatron system IV machine in the ECT suite of al-Hussain hospital.
Goldman grand rounds, electroconvulsive therapy (ect)...mysterious rebootLisa E Goldman, MD, MSW
How does ECT work? Is it dangerous? Is it scary? Does it work? What mental health conditions can be effectively treated by ECT? Is it true that ECT works better than prescription antidepressants? What are common side effects of ECT and how are this managed? Has the practice of ECT changed over time? What patient factors are the best predictors of positive and negative outcomes of ECT treatment? Come with me on a journey as I present my grand rounds lecture for the department of psychiatry as I complete my 4th year of psychiatry residency at UTHSC Memphis. These are exciting times. Recovery is definitely an option.
The presentation part of a two-day workshop on ECT held at al-Hussain University Hospital of al-Azhar Univeristy. It was accompanied by one-to-one training on the Thymatron system IV machine in the ECT suite of al-Hussain hospital.
electro convulsive therapy is imparting or introducing the electrical seizures in to the brain.
the seizures will be introducing bilaterally that is frontal temporal region and dominant side of the brain.
ect is the only therapy which was practiced in the ancient times .after that medicines have come. now it has wide variety of tequniques which is very common and came in to practice .
this ect will be done under anesthesia and with out anesthesia.
unilateral and bilateral ECT IS PRESENT .most commonly unilateral ect being practiced. The sitting of the ect may be up to 25 .every 3 sittings will be present once in a month .
NURSES responsibility is to educate the patient regarding treatment and indications and it's side effects .family also have to include while this education process.
ect means introducing the artificial grand mal seizures in to the brain.
ancient times even before ect psychiatric patients treated by the electrical eels. That passes a 1000 volts .
there are pre ect procedure
intra ect procedure
post ect procedure. Nurses must take part in these all 3 roles.
electro convulsive therapy is imparting or introducing the electrical seizures in to the brain.
the seizures will be introducing bilaterally that is frontal temporal region and dominant side of the brain.
ect is the only therapy which was practiced in the ancient times .after that medicines have come. now it has wide variety of tequniques which is very common and came in to practice .
this ect will be done under anesthesia and with out anesthesia.
unilateral and bilateral ECT IS PRESENT .most commonly unilateral ect being practiced. The sitting of the ect may be up to 25 .every 3 sittings will be present once in a month .
NURSES responsibility is to educate the patient regarding treatment and indications and it's side effects .family also have to include while this education process.
ect means introducing the artificial grand mal seizures in to the brain.
ancient times even before ect psychiatric patients treated by the electrical eels. That passes a 1000 volts .
there are pre ect procedure
intra ect procedure
post ect procedure. Nurses must take part in these all 3 roles.
Treatment for depression therapy options for depressionchriswans335
Over the years, advances have been made in treatments for Depression Therapy. The early treatments consisted of medication(s). The medications in the beginning left patients seriously lethargic and barely able to function. There is a variety of treatments available. Together with your physician you can determine which treatment protocol is appropriate for you.
Medications have come a long way over the years and you can lead a normal life and a successful one. There are different classes of medications, each working differently. All of them work on the chemicals in the brain Serotonin and Norepinephrine.
ECT - Electroconvulsive Therapy also known as shock therapy. Simply put electrodes are placed on the patients and shocks are delivered from which a seizure will occur. The after effects include confusion, memory loss, and prolonged treatment may lead to movement disorders.
This is also called talk therapy or counseling. Through various techniques, you will be led to review your life. There may be past issues, trauma, or chronic illness present. All of these can trigger Depression.
There are many treatments, some of which are older and rarely used. Others are old and still used. Still, other is new and may be experimental. The important thing to know here is that there are options that you can review with your doctor to see the path of treatment that is most appropriate for your needs.
AJMRR has a wide network of very good academicians and researchers. Its review process is very strict and the papers not fulfilling the criteria are outright rejected. Referees are constantly working hard to maintain the standards of Journal.
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
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Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists
Ect2
1. Electroconvulsive therapy (ECT) ECT is mainly used to treat people with severe depression. Some people do not get better when they are treated with anti-depressant drugs and psychological therapy.
2. ECT is only used when a person is at risk of suicide or is so depressed that they stop eating, drinking, or taking part in routine everyday activities. It is prescribed as a course of treatment by a psychiatrist if the psychiatrist has tried all other possible treatments and these have not worked.
3. Modern ECT Electrode’s are placed on the side of a patient’s head just above the temples. The patient is given anesthetic injections and a muscle relaxant to stop muscle contractions that can lead to broken bones. A small electric current is passed through the brain.
5. The patient is given a course of ECT usually between 3 and 15 treatments. ECT has the same risks as any minor surgical treatment.
6. ECT is very controversial. One reason is that ECT used to be dangerous and led to intense seizures that broke bones. Old ECT
7. Another reason is how ECT has been portrayed in the media One Flew Over The Cuckoo's Nest
8. Modern ECT is very different but there can still be serious side effects. Modern ECT
9. Limitations Possible physical side effects of ECT include impaired memory, cardiovascular changes and headaches (Datto, 2000). Psychological effects may be evident as well. The Department of Health report (1999) found that among those receiving ECT within the last two years, 30% reported that it had resulted in permanent fear and anxiety. Source: Psychology AS; The Complete Companion
10. ECT can save lives ECT can be an effective treatment for life threatening depression. Comer (2002) found that 60-70% of ECT patients improve after treatment. Sherwin Nuland, a surgeon, who suffered from severe depression discusses in this talk how ECT saved his life with no damaging side effects. TED talk on ECT Strengths
11. Ethics People with a serious mental illness who are at risk of self harm or are thought to be a risk to other people can be sectioned under the Mental Health Act.
12. This means they can be taken to a place of safety, usually a secure psychiatric unit, and given treatment against their will.
13. They may not consent to the treatment they are given and may be held against their will.
