The document compares and contrasts primary and permanent teeth. Some key differences include:
- Primary teeth are smaller with shorter crowns and thinner enamel and dentin layers.
- Permanent teeth have larger crowns and thicker enamel and dentin.
- The first permanent molar is an important tooth that erupts around 6 years of age and bears significant occlusal forces.
- It plays a key role in arch development and tooth movement, so preserving it is important to prevent problems with spacing, function, and occlusion.
The wasting diseases of teeth, namely attrition, abrasion and dental erosion have taken their toll in the population around the world due to the changing lifestyles, increase in the stress levels and many others factors that were persistent earlier but have suddenly increased drastically. This presentation brings to light the new factors that have attributed to this condition as well as discusses the previous ones.
Differences between primary and permanent dentitionAkshMinhas
A longitudinal radiological study of children (N = 549) who participated in a comprehensive preventive maintenance program showed that caries related events in the approximal surfaces of permanent teeth differed from those in deciduous teeth. Changes in the approximal surfaces of the younger permanent teeth were more pronounced than of the older primary teeth and differed significantly from 1 year to 2.5 years. These findings can be explained by posteruptive maturation of tooth enamel.
The wasting diseases of teeth, namely attrition, abrasion and dental erosion have taken their toll in the population around the world due to the changing lifestyles, increase in the stress levels and many others factors that were persistent earlier but have suddenly increased drastically. This presentation brings to light the new factors that have attributed to this condition as well as discusses the previous ones.
Differences between primary and permanent dentitionAkshMinhas
A longitudinal radiological study of children (N = 549) who participated in a comprehensive preventive maintenance program showed that caries related events in the approximal surfaces of permanent teeth differed from those in deciduous teeth. Changes in the approximal surfaces of the younger permanent teeth were more pronounced than of the older primary teeth and differed significantly from 1 year to 2.5 years. These findings can be explained by posteruptive maturation of tooth enamel.
A detailed look at the differences between the human primary and permanent dentition. Hope you find this informative. for further queries, please contact at dr.mathewthomasm@gmail.com.
Difference between primary and permanent teethprincesoni3954
The presentation features the basic difference between primary and permanent dentition. The differences are tabulated under the headings of crown, roor and pulp.
ORAL HABITS - DEFINITION, CLASSIFICATIONS, CLINICAL FEATURES AND MANAGEMENTKarishma Sirimulla
This seminar consists of description of various oral habit along with definitions, classifications, clinical features and management of oral habits like thumb sucking,tongue thrusting,mouth breathing and other secondary habits
This seminar consists of a brief description about various systemic diseases along with their oral manifestations and treatments along with the special considerations to be followed
This seminar consists of introduction, incidence, etiology, various classifications, history, clinical examination,sequelae of trauma of primary teeth followed by management
Pediatric Endodontics - Indirect and Direct pulp capping,Pulpotomy, Pulpecto...Karishma Sirimulla
this seminar consists of basis differences in root canal pattern between primary and permanet teeth followed by various definitions techniques and medicaments used in indirect pulp capping, direct pulp capping, pulpotomy, pulpectomy, apexogenesis and apexification
this seminar consists of pain,components of pain,pain pathways - ascending and analgesic followed by management of dental pain and local anesthesia,composition,various techniques used and pediatric implications for the administration of the locan anesthetics and the newer agents wich are available in topical,injectable and intra osseous techniques
This seminar contains a brief introduction followed by objectives of bahavior management,various definitions,classification,pedodontic triangle,parenting types,Non-pharmacological methods of behavior management in detail with modifications followed by conclusion.
This seminar consists of an introduction to child psychology followed by psychodynamic theories and its applicatioms followed by description and types of fear and anxietry followed by various behaviour rating scales and classification of behaviour
This seminar consisits of description of various bacterial diseases along with their oral manifestations,diagnosis and treatment.an addition of suitable case reports for better understanding and associated disorders
This seminar consists of various cysts seen in the oral cavity alonh with various classifications and added case repots for better understanding and the various treatment protocols followed for treating various cysts.
This seminar includes features of the normal periodontium seen in children along with various gingival and periodontal diseases seen in children with updated classifications along with clinical features and treatment modalities and a note on clinical assessment of oral cleanliness and periodontal diseases
Oral diseases: a global public health challenge and Ending the neglect of glo...Karishma Sirimulla
This presentation includes various lacunae faced by low and middle income contries due to the dental health policy and also highlights the areas where the reformation has to be made in order to utilize the dental services equally by all group of people
Diet and dental caries - Diet charts and Diet counsellingKarishma Sirimulla
This seminar includes a brief introduction to Diet and Dental caries along with Role of carbohydrates,Proteins and Fats with Dental caries along with diet charts, diet modifications, Diet counselling,Food log and sugar substitutes
Caries activity test - caries prediction,caries susceptibility and clinical i...Karishma Sirimulla
this seminar includes various caries activity tests and key caries risk factors caries susceptibility,cariogram and caries prediction along with its applications
This seminar includes classifcation,etiopathogenesis,Various theories of dental caries,caries patterns in primary and permanent teeth,Caries pattern in adolescets followed by caries risk assessment,CAMBRA,Differences between nursing bottle and rampant cariess,diagnosis which included the advanced digital diagnostic methods like diagnodent,QLF,etc and management with age specific management and flouride therapy age wise .
This seminar includes various cephelograms and various hard tissue analyses by diffreent authors followed by differences in various ethnicities and pediatric implications
This seminar includes various isolation methods which are direct and indirect with eloboration about rubber dam usage and application along with the advantages along with soft tissue isolation methods
This seminar includes hemostasis,mechanism of blood clotting and associated blood dyscrasias commonly seen in children and their treatments with a note on antifibrinolytics
THIS SEMINAR INCLUDES DEFINATION,TYPES OF INFLAMMATIONS AND MEDIATORS OF INFLAMMATION FOLLOWED BY REGENERATION,REPAIR AND WOUND HEALING BY PRIMARY AND SECONDARY INTENTIONS OF SOFT AND HARD TISSUES.HEALING OF EXTRACTION SOCKETS AND WEEKLY CHANGES IN HEALING OF EXTRACTION SOCKET.LOCAL AND SYSTEMIC FACTORS OF INFLAMMATION ABD COMPLICATIONS OF WOUND HEALING
Oral microbiology:normal oram microflora and Dental plaqueKarishma Sirimulla
this seminar includes microbiolofical aspects of bacteria and their living systems and oral microflora along with detailed description about plaque which includes composition,classification,formation of plaque and the plaque hypothesis followed by pathogenesis of plaque
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
2. The human dentition is termed heterodont
• In comparison, a homodont dentition is one in which all of the teeth
are the same in form and type. This sort of dentition is found in some
of the lower vertebrates.
• Sets of teeth: Diphyodont (Human):
• 2 sets of teeth – 1. Decidous
2. Permanent.
INTRODUCTION
3. Deciduous dentition - Eruption : about six months to two years of age
No. of teeth presents : 20
Other non-scientific name : "milk" teeth/" baby" teeth/ "temporary"
teeth.
• 2. Permanent dentition – Eruption: from 6-21 years of age.
No. of teeth presents: 32
4. Eruption sequence follows a pattern – Incisors-First Molars-
Canines-Second molars.
This pattern is generally followed by both arches, with the
mandibular arch preceding the maxillary arch.
The loss of deciduous teeth tends to mirror the eruption
sequence.
Caries susceptibility is reverse of this order
5. Three periods of dentition, since the deciduous and permanent dentitions
overlap in time.
These periods are summarized in the following manner:
1. Primary dentition period
2. Mixed dentition period
3. Permanent dentition Period
6. Morphological & Anatomical Difference b/w
Primary & Permanent Tooth
Primary Teeth
Crown:
- Shorter.
- Narrow Occlusal
table.
- Constricted in
cervical portion.
Permanent Teeth
Crown:
- Bigger
- Broad Occlusal table.
- Cervical constriction is
not well marked.
7. 1) Bluish white in color ,refractive
index similar to that of milk(
RI=1).
2) Cuspids are slender and to be
more conical.
3) Cervical ridges are more
pronounced especially on buccal
aspect of first primary molar
4) Occlusal plane is relatively flat.
5) Molars are bulbous and are
sharply constricted cervically
6) 1 st molar is smaller in
dimension than the 2nd molar
1) Grayish white to yellowish white
in color.
2) Cuspids are less conical.
3) The cervical ridges are flatter
4) Occlusal plane has relatively
curved contour.
5) They have less constriction at the
neck.
6) 1 st molar is larger in dimension
than the 2nd molar.
8. • Thinner enamel and dentin
layers.
• Enamel rods in the cervical
area directed Occlusally.
• Broad and flat contacts.
• Color is usually lighter.
• Prominent mesio-buccal
cervical bulge seen in
primary molars.
• Incisors have no
developmental grooves or
mammelons.
9. Pulp chamber is larger in relation to crown
size.(smaller)
Pulpal outline follows DEJ more closely.
( less closely)
Pulp horns are closer to the outer surface.
Mesial pulp horn extends to a closer
approximation of surface than the distal
pulp horn.(comparatively away)
High degree of cellularity and vascularity
in tissue.
High potential for repair.
PULP
10. Comparatively less tooth structure.
Root canals are more ribbon like. the
radicular pulp follows a thin , tortuous
and branching path (well defined with
less branching. )
Floor of pulp chamber is porous.
Accessory canals in primary pulp
chamber floor leads directly into
interradicular furcation.
11. PRIMARY TEETH PERMANENT TEETH
• Roots are larger and more slender in
comparison to crown size.
• Furcation is more towards cervical area
so that root trunk is smaller .
• Roots are narrower mesio-distally.
• At the cervical region, the roots of the
primary molars flare outward and
continue to flare as they approach the
apices to accommodate permanent tooth
buds.
• Undergo physiologic resorption during
shedding of primary teeth.
• Roots are shorter and bulbous
in comparison to crown.
• Placement of furcation is apical
, thus the root trunk is larger.
• Roots are broader
mesiodistally.
• Marked flaring of roots is
absent.
• Physiologic resorption is
absent.
ROOTS
12. ENAMEL
PRIMARY TEETH PERMANENT TEETH
• Bands of retzius are less common. This
maybe partly responsible for the
bluish white color.
• Neonatal lines are present in all teeth.
• Enamel is thinner and has a more
consistent depth of about 1mm
thickness throughout the entire crown
• Enamel rods at the cervical slopes
occlusally from the DEJ.
• Bands of retzius are more
common.
• Neonatal lines are only
present in 1st molars
• The enamel is thicker
and has a thickness of
about 2-3mm.
• Enamel rods are oriented
gingivally.
13.
14. DENTIN
Dentinal tubules are less
regular.
Dentin thickness is half that of
permanent teeth. Thickness is
limited in some places.
Less dense and easy to cut.
Interglobular dentin is absent.
Dentinal tubules are
more regular.
Dentin is thicker.
Dentin is denser and
difficult to cut.
Interglobular dentin is
present.
15. PERIODONTIUM
• Cementum is very thin and of the
primary type. Secondary cementum is
characteristically absent.
• Alveolar atrophy is rare.
• Gingivitis is generally absent in a
healthy child. Similarly recession is in
frequent.
• Secondary cementum is
present.
• Alveolar atrophy occurs.
• Gingivitis is common in
adults.
16. HISTOLOGICAL DIFFERENCES
• Roots have enlarged apical
foramens. Thus , the abundant
blood supply demonstrates a more
typical inflammatory response.
• Incidence of reparative dentin
formation beneath carious lesion is
more extensive and irregular.
• Foramens are restricted. Thus
reduced blood supply favors a
calcific response and healing
by calcific scarring.
• Reparative dentin formation
is less.
17. OTHER KEY POINTS
• Primary Teeth - More prone to
acid attack, thus rapidly
demineralised to dental caries.
• Dentin is less mineralised .
• Lamina dura is relatively thick.
• Premolars –Absent
• Mesiodistal diameter of crown is
more then cervico incisal length.
• Permanent Teeth- Less prone to
caries attack.
• Dentin is more mineralised.
• Lamina dura is relatively thin.
• Premolars-Present
• Cervico incisal length is more
then the mesiodistal dimension.
18.
19. The Importance Of The First Permanent
Molar
The first permanent molar is a very important tooth from both
functional and developmental point of view.It is otherwise known
as "six year molar"
This tooth erupts between 5 and 1/2 to 6 and 1/2 years of age.It is
the first non-succedaneous tooth to erupt in to the oral cavity.As it
has a large occlusal table,it bears the maximum load of occlusal
forces.
20. It is considered a key to occlusion.As this tooth erupts posterior to
the deciduous second molar(milk tooth),it is also instrumental in
establishing the arch perimeter.
The first permanent molar also has maximum root surface and so is
the most important anchorage unit used in tooth movement
21. Due to the presence of deep pits and fissures, food lodgement is
common in this tooth resulting in rapid decay.
Extraction or removal of this tooth leads to problems in space
management, tooth movement, mastication and occlusion.
So every attempt should be made to save this tooth in case it
gets decayed.
22. YOUNG PERMANENT TEETH
1)Vital pulp therapy for teeth diagnosed with a normal pulp or reversible pulpitis
Protective liner/Indirect pulp treatment
Apexogenesis (root formation). Formation of the apex in vital, young,
permanent teeth can be accomplished by im-plementing the appropriate vital pulp
therapy (i.e., indirect pulp treatment, direct pulp capping, partial pulpotomy for
carious exposures and traumatic exposures).
Direct pulp cap : When a small exposure of the pulp is encountered during cavity
preparation and after hemorrhage control is obtained, the exposed pulp is capped
with a material such as calcium hydroxide88-92 or MTA92 prior to placing a
restoration that seals the tooth from microleakage
23. Partial pulpotomy for carious exposures.
Partial pulpotomy for traumatic exposures
(Cvek pulpotomy).
The partial pulpotomy for traumatic exposures is a
procedure in which the inflamed pulp tissue beneath an
exposure is re-moved to a depth of one to three
millimeters or more to reach the deeper healthy tissue.
Pulpal bleeding is controlled using bacteriocidal irrigants
such as sodium hypochlorite or chlorhexidine, and the site
then is covered with calcium hydroxide or MTA