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DR HARDEV SINGH
P.G IN ORTHOPAEDICS
BHMRC
NEW DELHI
What is a compartment?
Closed area of muscles
group, nerves & blood
vessels surrounded by
fascia
Pressure: 5-15 mmhg
Definition:
An increased pressure within enclosed
osteofascial space that reduces capillary
perfusion below level necessary for
tissue viability;
the underlying mechanism is:
- increased volume within space
- decreased space for contents
- combination of both
What is a compartment
syndrome?
 intra comp. pressure
(35-40 mmhg)
 capillaries collapse
 Blood flow to muscles
and nerves
 Bl.Vs collapse
Pathophysiology:
Increased compartment pressure
leads to increased venous pressure
which decreases A-V gradient
resulting in muscle and nerve
ischemia.
Pathophysiology:
Normal tissue pressure
0-4 mm Hg
8-10 with exertion
Absolute pressure theory
30 mm Hg - Mubarak
45 mm Hg - Matsen
Consequences –vicious cycle
Why is it dangerous?
Nerves:
neuropraxia: will
regenerate
Ischemia: cell death
Muscles: contracture
(Volkmann's ischemic
contracture)
Gangrene
Compartment Syndrome- CAUSES
Causes
 Fractures
 Contusions
 Surgery
 Post Ischemic swelling after arterial occlusion
 Major vascular trauma
 Crush injuries
 Burns
 Prolonged limb compression
Causes
Fracture of a long bone
(Supracondylar humerus,
forearm, hand,tibia and
foot)
CAUSES
Drilling &
reaming
Dissection
Tourniquet
CAUSES
Tight cast
swelling
Bluish
discolorationnumbness
CAUSES
Severe bruised muscle
(even if there is no fracture)
Don’t take contusion lightly
COMPARTMENT SYNDROME
Signs and Symptoms
Increased Pressure and Tightness
Progressive pain out of proportion to initial
injury
Markedly swollen area
Progressive neurologic deficit
Seven P’s
Pain
Pressure
Pain with passive stretch
Parethesia
Paresis/ Paralysis
Pulselessness
Pallor
SYMPTOMS
Severe pain
inappropriate to the
injury(not relieved even
with morphia)
SYMPTOMS
Burning of the affected limb
Tight muscle(rigid)tightness feeling
Numbness: bad sign
SIGNS & DIAGNOSIS
Passive stretching of fingers or toes (muscle
stretch)will lead to severe pain (diagnostic sign)
Never wait for signs of ischemia (5 Ps):irreversible
damage
STRECH TEST
It is possible to strech
the affected muscles by
passively moving the
joints in direction
opposite to that of the
damaged muscles,s
action (( e.g. ::: passive
extension of fingers
produces pain in flexor
compartment of forearm
Technique
STRYKER TECHNIQUE
MERCURY
MANOMETER
Wick hand held
instrument
Whiteside maneuver Wick hand held instrument
syringe
3 way stopcock
mmhg
mano.
electrode
Direct
reading
Stryker Stic System
Easy to use
Can check multiple compartments
Different areas in one compartment
Complications related to CS
Late Sequelae
 Volckmann’s contracture
 Weak dorsiflexors
 Claw toes
 Sensory loss
 Chronic pain
 Amputation
Compartment Syndrome
Tissue Survival
• Muscle
– 3-4 hours - reversible changes
– 6 hours - variable damage
– 8 hours - irreversible changes
• Nerve
– 2 hours - looses nerve conduction
– 4 hours - neuropraxia
– 8 hours - irreversible changes
COMPARTMENT SYNDROME
Management
Non surgical management:
 Remove any tight bandage, soaked dressing
 Cast should be removed completely
 Elevation
•management
Surgical management:
(FASCIOTOMY)
Open skin and fascia
down to a compartment
It is a surgical procedure
where the fascia is cut to
relieve tension or
pressure commonly to
treat the resulting loss of
circulation to the tissue
Fasciotomy Principles
Make early diagnosis
Long extensile incisions
Release all fascial compartments
Preserve neurovascular structures
Debride necrotic tissues
Coverage within 7-10 days
Compartment Syndrome
Indications for Fasciotomy
Unequivocal clinical findings
Pressure within 15-20 mm hg of DBP
Rising tissue pressure
Significant tissue injury or high risk pt
> 6 hours of total limb ischemia
Injury at high risk of compartment syndrome
Single Incision
• Perifibular Fasciotomy
– Matsen et al (1980)
– Single incision just
posterior to fibula
– Common peroneal nerve
Double Incision
Mubarak et al JBJS 1977
Forearm Fasciotomy
Volar-Henry
approach
Include a carpal
tunnel release
Release lacertus
fibrosus and fascia
Protect median
nerve, brachial artery
and tendons after
release
Compartment syndrome foot
9 compartments
-medial superficial
lateral calcaneal
interossei(4)
adductor.
Suspicion with
-lisfranc fracture
-calcaneal fracture
Compartment syndrome foot
 Dorsal incision to
release the interossei and
adductor
Median incision to
release the medial
superficial lateral and
calcaneal compartment
Flexor digitorum
longus
Gastroc-soleus
Superficial peroneal
Intermuscular septu
Fasciotomy of Hand
10 separate osteofascial
compartments
dorsal interossei (4)
palmar interossei (3)
thenar and hypothenar
(2)
adductor pollicis (1)
Close skin by 2ry
sutures after oedema
subsides
It may need skin graft
Wound Management
Wound is not closed at initial surgery
Second look debridement with consideration for
coverage after 48-72 hrs
Limb should not be at risk for further swelling
Pt should be adequately stabilized
Usually requires skin graft
DPC possible if residual swelling is minimal
Flap coverage needed if nerves, vessels, or bone
exposed
Goal is to obtain definitive coverage within 7-10
days
Wound Management
After the fasciotomy, a bulky compression dressing and
a splint are applied.
“VAC” (Vacuum Assisted Closure) can be used
 Foot should be placed in neutral to prevent equinus
contracture.
Incision for the fasciotomy usually can be closed after
three to five days
Complications Related to
Fasciotomies
Altered sensation within the margins of the wound (77%)
Dry, scaly skin (40%)
Pruritus (33%)
Discolored wounds (30%)
Swollen limbs (25%)
Tethered scars (26%)
Recurrent ulceration (13%)
Muscle herniation (13%)
Pain related to the wound (10%)
Tethered tendons (7%)
Fitzgerald, McQueen Br J Plast Surg 2000Fitzgerald, McQueen Br J Plast Surg 2000
Compartment syndrome is a serious syndrome, Which needs
to be diagnosed early.
Palpable pulse doesn’t exclude compartment syndrome
If diagnosis and fasciotomy were done within 24 hrs, the
prognosis is good.
If delayed, complications will develop.
The earlier you diagnose, the safer you are

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Compartment syndrome

Editor's Notes

  1. Seven P’s: Pain- pain in excess of the presenting injury Pressure- affected compartment may be tense to palpation Pain with passive stretch- pain increases with passive ROM Paresthesia- Numbness over the cutaneus distribution of the nerve that runs through the affected compartment Paresis/Paralysis- Ischemia or necrosis of the nerves or muscles develops within the affected compartment Pulses- could be absent but frequently palpable Pallor- skin discoloration may be visible due to impaired venous drainage Co-Morbidities include: Mild: ~ mm weakness ~fatigue ~ Myositis ossificans Severe ~ Severe life threatening vascular compromise ~ decreased arteriovenous gradient **To Remember** ~ Do not elevate limb above hear because it decreases the arteriovenous gradient therefore decreasing blood flow ~ Instead limb should be elevated at heart level to maintain arteriovenous gradient and assist with venous drainage ~ Remove any compression casts or dressings
  2. Studies have shown that nerve tissue is the most sensitive to ischemic changes. Nerve conduction is lost in 1-2 hours of total ischemia and survive up to 4 hrs with only neuropraxia changes, while axonotmesis and irreversible changes occur after 8 hrs. Muscle may survive up to 4 hours with reversible changes, variable damage occurs by 6 hrs, and irreversible changes after 8 hrs under conditions of warm ischemia.
  3. These are indications for surgical decompression. A missed CS > 24-48 hours should not be opened. (see Rockwood and Green 5th edition and Campbell’s 10 th edition) The damage cannot be reversed and there is a significant infection rate when the dead tissue is exposed to the hospital environment. The surgeon must deal with the residual contractures of the ischemic muscle and not risk the chance of infection. Some have suggested that the scarred ant tib muscle can serve as a check rein and limit foot drop sequelae.
  4. Volar approach use Henry Approach to release superficial and deep flexors flexors followed by the pronators and supinator. The dorsal approach is centered over the proximal forearm and can be used to release the Mobile Wad (BR, ECRL,ECRB). It is important to remember when treating and electrical injury with a compartment syndrome, the most damage is deep along the bone (i.e. the Pro and Sup muscles) and may require debridement at the initial surgery…Do not miss this!
  5. Volar approach use Henry Approach to release superficial and deep flexors flexors followed by the pronators and supinator. The dorsal approach is centered over the proximal forearm and can be used to release the Mobile Wad (BR, ECRL,ECRB). It is important to remember when treating and electrical injury with a compartment syndrome, the most damage is deep along the bone (i.e. the Pro and Sup muscles) and may require debridement at the initial surgery…Do not miss this!
  6. Volar approach use Henry Approach to release superficial and deep flexors flexors followed by the pronators and supinator. The dorsal approach is centered over the proximal forearm and can be used to release the Mobile Wad (BR, ECRL,ECRB). It is important to remember when treating and electrical injury with a compartment syndrome, the most damage is deep along the bone (i.e. the Pro and Sup muscles) and may require debridement at the initial surgery…Do not miss this!