Mr. Mohammed Ghouse, a 64-year-old male with hypertension, presented with chest pain and was admitted to the hospital. Tests showed extensive heart ischemia. He underwent coronary angiography which revealed triple vessel disease and atherosclerosis. His condition was stabilized with medications and an IABP device, but he later developed ventricular tachycardia and died despite resuscitative efforts. The cause of death was determined to be an acute myocardial infarction due to reduced blood flow from the blocked coronary arteries.
This highly energetic lecture presents the pathophysiology of S-T elevation myocardial infarction in an easy to understand style to help you best identify, triage and treat patients presenting with acute coronary syndromes. Using the latest research behind the AHA Guidelines changes, AHA National Faculty Rom Duckworth will help you better coordinate with you partners along the continuum of cardiac care. Emphasis is placed on risk factors, recognizing truly sick patients and coordinating care with hospital personnel.
Learning Objectives: Students will learn:
-The pathophysiology of S-T elevation myocardial infarction.
-The difference between STEMI, NSTEMI and unstable angina.
-Differing treatment methods and priorities for different cardiac syndromes.
-The function and importance of 12 lead ECG and prehospital diagnostic testing.
-The roles and responsibilities of EMS providers as the key element in “door-to-balloon” and “door-to-needle” time for STEMI patients.
www.romduck.com
www.RescueDigest.com
This highly energetic lecture presents the pathophysiology of S-T elevation myocardial infarction in an easy to understand style to help you best identify, triage and treat patients presenting with acute coronary syndromes. Using the latest research behind the AHA Guidelines changes, AHA National Faculty Rom Duckworth will help you better coordinate with you partners along the continuum of cardiac care. Emphasis is placed on risk factors, recognizing truly sick patients and coordinating care with hospital personnel.
Learning Objectives: Students will learn:
-The pathophysiology of S-T elevation myocardial infarction.
-The difference between STEMI, NSTEMI and unstable angina.
-Differing treatment methods and priorities for different cardiac syndromes.
-The function and importance of 12 lead ECG and prehospital diagnostic testing.
-The roles and responsibilities of EMS providers as the key element in “door-to-balloon” and “door-to-needle” time for STEMI patients.
www.romduck.com
www.RescueDigest.com
Acute coronary syndrome result from a sudden blockage in a coronary artery. this blockage causes unstable angina or heart attack (MI), depending on the location and amount of blockage.
people who experience an ACS usually have chest pressure or ache, shortness of breath and fatigue.
People who think they are experiencing ACS should call for emergency help.
Doctors use ECG and blood test (troponin level) to determine whether a person is experiencing an ACS.
Treatment varies depending on the type of syndrome but usually include attempts to increase blood flow to affected area.
This presentation describes the emergency department management of sinus tachycardia, supraventricular tachycardia, atrial flutter, atrial fibrillation, ventricular tachycardia and ventricular ectopic
A presentatation on Acute coronary syndrome made while in Emergency Department. If you are making a presentation on ACS, you may want to add more on TIMI score as it is important. Some problems with display of pictures/diagrams due to ?conversion problems. Based on AHA Guidelines 2010 and from Harrison's 18th Ed.. Made using OpenOffice.
this is a slide on myocardial infraction to figure you out what exactly it is !
though i have not mentioned the diet based causes ............etc.
so enjoy
Acute coronary syndrome result from a sudden blockage in a coronary artery. this blockage causes unstable angina or heart attack (MI), depending on the location and amount of blockage.
people who experience an ACS usually have chest pressure or ache, shortness of breath and fatigue.
People who think they are experiencing ACS should call for emergency help.
Doctors use ECG and blood test (troponin level) to determine whether a person is experiencing an ACS.
Treatment varies depending on the type of syndrome but usually include attempts to increase blood flow to affected area.
This presentation describes the emergency department management of sinus tachycardia, supraventricular tachycardia, atrial flutter, atrial fibrillation, ventricular tachycardia and ventricular ectopic
A presentatation on Acute coronary syndrome made while in Emergency Department. If you are making a presentation on ACS, you may want to add more on TIMI score as it is important. Some problems with display of pictures/diagrams due to ?conversion problems. Based on AHA Guidelines 2010 and from Harrison's 18th Ed.. Made using OpenOffice.
this is a slide on myocardial infraction to figure you out what exactly it is !
though i have not mentioned the diet based causes ............etc.
so enjoy
Myocardial Infarction - Case Presentation and an OverviewAbubakkar Raheel
Case Presented by Final Year MBBS sudents of Frontier Medical College at the 1st Clinico-Pathological Conference for the year 2015.The Presentation is divided into two parts. First part is about a case of an Acute ST Segment elevated Myocardial Infarction with. Its management at the Hospital and the findings. Second part is about the pathophysiology, Cinical signs and symptoms and an effective gold standard treatment of MI.
A very informative case on ACS for budding Clinical pharmacists, it covers Classification of ACS with types of MI's as well pharmaceutical care plan for the management of ACS
Caring patient on Mechanical Ventilator Shanta Peter
Mechanical ventilators are used now in general wards , not only in ICU -to save patient's life. We need to care patient and ventilator while working with it ..
Endovascular and surgical treatment of pulmonary embolism 26.11.17Ivo Petrov
Interventional treatment (thrombus fragmentation and supraselective fibrinolysis) of high and intermediate risk patients with pulmonary embolism.
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ACS covering STEMI and NSTEMI. MI is the most common cause of death in the developed countries and it is important to have a basic information and reach doctor as soon as possible to avoid complications and sudden death.
Percutaneous Transvenous Mitral Commissurotomy in 71 Years Old Woman with Mit...M A Hasnat
AAS Majumder, MA Hasnat, AKMM Islam, M Ullah, MZ Rahman, S Zaman, MM Rahman, MH Alamgir.
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(Cardiovasc. j. 2013; 6(1): 68-70)
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An Interesting Case of Stuck Mitral Valve by Clot, Post Thrombolysis Develope...Premier Publishers
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Case presentation on myocardial infraction
1.
2. Mr. Mohammed Ghouse ,a 64 YO Male was
admitted to FMMH on 16th Nov2015
He deceased due to Acute Myocardial
Infraction –Cardiogenic Shock on
18thNov2015
3. The patient was with k/c/o Hypertension
4hrs of sudden onset of retrosternal rt sided
throbbing type of chest pain
4. Hematology
Tests 16Nov 17Nov
Hemoglobin(11-16.5)
14.8 11.4
PCV (35-50%) 45.6 -
TC(3500-10000) 12100 31300
Mcv/mch/mchc(80-100/27-31/32-36) 88.5/28.8/32.5 89.0/29.3/32.9
Platelets(1-4lac cells/cum) 336000 259000
PTT control test 29.9/56.5 -
P.Smear - 3.90/34.7
6. Echocardiogram reports
Left ventricle
*hypokinesia- inability of movemnt ,rigidity
*LVH-Left Ventricular Hyprtrophy ,thickening of LV myocadium
Regional Wall Motion Hypokinesia of artero lateral
wall
Size Normal
Thickness Concentric LVH
Ejection fracton Reduced
LA Normal
RA Normal
RV Normal
7. Heart Valves
Final diagnosis :
Ischemic heart disease
Concentric LVH
Sclerotic Aortic valve
No clot /PAH/PE
Mild LV dysfunction
Mitral valves Normal
Tricuspid valve Normal
Aortic valve Sclerotic
Pulmonary valve Normal
8. Coronary angiogram
Coronary profile
Left main Distal segment had 100% occlusion
LAD Faintly seen through retropad
collaterals
Diagnals Not seen
LCx Not seen
PCA Dominant vessel,3.5 mm big
85%calcifcation on proximal part
,type B lesion ,mid segment has
insignified lesions ,distal segment
has calcified 90% type B lesions
PDA and PLV Large branching vessels without
disease
12. Patient K/c/o hypertension presented with c/o chest
pain of one day duration.ECG showed extensive
Anterolateral wall ischemia .patient was admitted to
ICCU .CAG was done on 16Nov2015 which showed
Atherosclerotic mains with triple vessel disease
,patient had hypertension for which he was put on
isotropic support
In view of persistent hypertension IABP was inserted
on consent of patient party .there was symptomatic
improvement on the patient’s condition. But on
18nov2015 at around 7am he developed VT and had
cardio respiratory arrest and in spite of all
resuscitative measures ,he patient could not be
revived
13. Definition
Myocardial infarction or Acute Myocardial
infarction occurs when blood flow stops to a
part of the heart causing damage to the
myocardium
14. Etiology
Myocardial Infarction results from the lack of oxygen
supply to the working myocardium. The regional
infarcts are due to lack of blood flow that occurs
due to thrombus or atheroma
Autopsies of the inpatients dying of acute MI reveal an
acute thrombus overlying atherosclerotic plaque in
more than95%of cases .The remaining cases are
cause by severe coronary diseases
He risk factors include high blood pressure, obesity,
smoking habits, excess alcohol intake ,cholesterol,
diabetes .
17. Assessment
Dueto the above factors te patient was diagnosed
to be suffering from Acute
myocardial infarction
Plan
Firdt the patient should be stabilised by giving
oxygen therapy folwed by emergency
medications and then initiate the
pharmacological therapy
