bilateral potentially blinding condition in which obstruction to aqueous outflow is brought about solely by closure of angle by peripheral iris One eye is usually affected before the other
1. Retinitis pigmentosa is a genetically determined, progressive degeneration of retinal photoreceptors that affects both eyes symmetrically. It initially impacts rods, followed by cones.
2. Symptoms include night blindness, loss of peripheral vision, light sensitivity, and eventual loss of central vision. It can be inherited in autosomal recessive, autosomal dominant, or X-linked patterns.
3. Diagnosis involves visual acuity testing, visual fields, color vision, dilated fundus exam showing bone spicule pigmentation, attenuated vessels, and disc pallor. Electroretinography can detect early disease. There is no cure but vitamin supplementation and low-light lifestyle adaptations can
Fungal corneal ulcers are common, caused mainly by Aspergillus in India. Risk factors include ocular trauma, contact lens use, pre-existing eye conditions, and systemic immunosuppression. Diagnosis involves corneal scrapings, cultures, and stains showing fungal hyphae or spores. Treatment consists of topical natamycin or amphotericin B, sometimes with adjunctive debridement, intracameral/intracorneal injections, or therapeutic keratoplasty for severe cases. Systemic antifungals may also be used for extensive infections.
This document discusses corneal ulcers, including their definition, causes, microbiology, pathogenesis, stages, grading, symptoms, clinical examination, investigations, treatment, and complications. Key points include:
- Corneal ulcers are tissue excavations associated with epithelial defects, edema, infiltration and necrosis. They are usually caused by injury or foreign materials that allow microbial infection.
- Common microbes include bacteria (e.g. streptococcus, pseudomonas), fungi (e.g. candida, fusarium), protozoa (e.g. acanthamoeba), and viruses (e.g. herpes).
- Treatment involves local and systemic antibiotics, antifungals, or antiv
This document defines and describes the anatomy and physiology of the lacrimal apparatus, which is responsible for tear formation and drainage in the eye. It discusses how lacrimal syringing is used to test the structural integrity of the lacrimal drainage system by passing fluid through the puncta and observing for any blockages or abnormalities. The document also outlines the equipment needed for lacrimal syringing and provides interpretations of different results that could indicate where in the drainage system a blockage may be located.
Cataract surgery is the most common surgery that we perform on a outpatient basis. Evaluation of the patient is critical and essential for a desirable visual outcome.
This document discusses various types of secondary glaucoma caused by underlying ocular diseases and conditions. It describes the mechanisms of increased intraocular pressure, clinical features, and treatment approaches for different forms of secondary glaucoma including lens-induced glaucoma, inflammatory glaucoma, pigmentary glaucoma, neovascular glaucoma, and steroid-induced glaucoma among others. Management involves treating the underlying condition causing secondary glaucoma as well as lowering intraocular pressure through medical, laser, or surgical means.
This document describes different surgical procedures for lacrimal sac obstructions, including dacryocystorhinostomy (DCR) and dacryocystectomy. It outlines the anatomy, indications, preoperative requirements, steps of conventional DCR including osteotomy of the lacrimal bone and suturing of nasal and sac flaps, and post-operative care. It also discusses endoscopic and endolaser techniques for DCR and the indications for dacryocystectomy. Complications of DCR include wound infections, synechiae formation, and osteotomy stenosis leading to procedure failure.
This document discusses fungal corneal ulcers. It begins by describing the signs and symptoms of fungal ulcers, including pain, redness, defective vision, lid edema, and corneal opacity staining with fluorescein. Diagnosis involves smears, cultures and microscopy to identify causative fungi. Common fungi include Fusarium, Aspergillus, and Candida. Treatment involves topical natamycin or voriconazole drops. Prognosis includes potential complications like scar formation, astigmatism, perforation and fistula formation. Close monitoring is needed due to the difficulty treating fungal infections.
1. Retinitis pigmentosa is a genetically determined, progressive degeneration of retinal photoreceptors that affects both eyes symmetrically. It initially impacts rods, followed by cones.
2. Symptoms include night blindness, loss of peripheral vision, light sensitivity, and eventual loss of central vision. It can be inherited in autosomal recessive, autosomal dominant, or X-linked patterns.
3. Diagnosis involves visual acuity testing, visual fields, color vision, dilated fundus exam showing bone spicule pigmentation, attenuated vessels, and disc pallor. Electroretinography can detect early disease. There is no cure but vitamin supplementation and low-light lifestyle adaptations can
Fungal corneal ulcers are common, caused mainly by Aspergillus in India. Risk factors include ocular trauma, contact lens use, pre-existing eye conditions, and systemic immunosuppression. Diagnosis involves corneal scrapings, cultures, and stains showing fungal hyphae or spores. Treatment consists of topical natamycin or amphotericin B, sometimes with adjunctive debridement, intracameral/intracorneal injections, or therapeutic keratoplasty for severe cases. Systemic antifungals may also be used for extensive infections.
This document discusses corneal ulcers, including their definition, causes, microbiology, pathogenesis, stages, grading, symptoms, clinical examination, investigations, treatment, and complications. Key points include:
- Corneal ulcers are tissue excavations associated with epithelial defects, edema, infiltration and necrosis. They are usually caused by injury or foreign materials that allow microbial infection.
- Common microbes include bacteria (e.g. streptococcus, pseudomonas), fungi (e.g. candida, fusarium), protozoa (e.g. acanthamoeba), and viruses (e.g. herpes).
- Treatment involves local and systemic antibiotics, antifungals, or antiv
This document defines and describes the anatomy and physiology of the lacrimal apparatus, which is responsible for tear formation and drainage in the eye. It discusses how lacrimal syringing is used to test the structural integrity of the lacrimal drainage system by passing fluid through the puncta and observing for any blockages or abnormalities. The document also outlines the equipment needed for lacrimal syringing and provides interpretations of different results that could indicate where in the drainage system a blockage may be located.
Cataract surgery is the most common surgery that we perform on a outpatient basis. Evaluation of the patient is critical and essential for a desirable visual outcome.
This document discusses various types of secondary glaucoma caused by underlying ocular diseases and conditions. It describes the mechanisms of increased intraocular pressure, clinical features, and treatment approaches for different forms of secondary glaucoma including lens-induced glaucoma, inflammatory glaucoma, pigmentary glaucoma, neovascular glaucoma, and steroid-induced glaucoma among others. Management involves treating the underlying condition causing secondary glaucoma as well as lowering intraocular pressure through medical, laser, or surgical means.
This document describes different surgical procedures for lacrimal sac obstructions, including dacryocystorhinostomy (DCR) and dacryocystectomy. It outlines the anatomy, indications, preoperative requirements, steps of conventional DCR including osteotomy of the lacrimal bone and suturing of nasal and sac flaps, and post-operative care. It also discusses endoscopic and endolaser techniques for DCR and the indications for dacryocystectomy. Complications of DCR include wound infections, synechiae formation, and osteotomy stenosis leading to procedure failure.
This document discusses fungal corneal ulcers. It begins by describing the signs and symptoms of fungal ulcers, including pain, redness, defective vision, lid edema, and corneal opacity staining with fluorescein. Diagnosis involves smears, cultures and microscopy to identify causative fungi. Common fungi include Fusarium, Aspergillus, and Candida. Treatment involves topical natamycin or voriconazole drops. Prognosis includes potential complications like scar formation, astigmatism, perforation and fistula formation. Close monitoring is needed due to the difficulty treating fungal infections.
Optic atrophy is the degeneration of the optic nerve fibers leading to pallor of the optic disc. It can be classified as primary, secondary, consecutive, glaucomatous, or ischemic based on the underlying cause and appearance of the optic disc. Primary optic atrophy results from diseases proximal to the disc with a chalky white appearance. Secondary optic atrophy occurs after inflammation of the optic disc with a dirty white, blurred disc. Consecutive optic atrophy follows retinal lesions and shows a yellow waxy disc. Treatment focuses on the underlying cause when possible, but once complete atrophy occurs, vision cannot be recovered.
1. Viral corneal ulcers have increased due to antibiotics reducing bacterial flora. Herpes simplex virus is a common cause, initially infecting epithelium and potentially becoming neurotropic. Primary infection involves non-immune individuals while recurrent infections reactivate dormant virus.
2. Herpes simplex keratitis manifestations include punctate epithelial keratitis, dendritic ulcers, and stromal keratitis treated with antivirals like acyclovir along with supportive measures. Herpes zoster ophthalmicus affects the trigeminal nerve causing vesicular skin lesions and ocular complications in 50% of cases like keratitis, treated with antivirals and steroids.
The synoptophore is an ophthalmic instrument used to diagnose and treat imbalances of the eye muscles. It consists of two cylindrical tubes with mirrored bends that allow pictures to be presented simultaneously to both eyes, compensating for any angle of squint. It is used to investigate binocular function in patients with a manifest squint, detect suppression and abnormal retinal correspondence, and measure horizontal, vertical and torsional misalignments. The synoptophore can test for three grades of binocular vision - simultaneous perception, fusion, and stereopsis - and detect whether a patient has normal or abnormal retinal correspondence based on differences between subjective and objective angles of squint.
Primary angle closure glaucoma is caused by blockage of the aqueous humor outflow due to closure or narrowing of the anterior chamber angle. It progresses through stages from primary angle closure suspect to acute angle closure to angle closure glaucoma, where glaucomatous optic nerve damage and visual field defects are present. Risk factors include hyperopia, smaller anterior chamber angle, older age, and Asian ethnicity. Treatment begins with intravenous mannitol or glycerol to lower pressure urgently, followed by topical eye drop medications like beta-blockers or pilocarpine. The definitive treatment is laser iridotomy or trabeculectomy surgery to permanently open the drainage angle.
This document discusses neovascular glaucoma, also known as rubeotic glaucoma. It begins by defining the terminology and describing the clinical features. The main causes of neovascular glaucoma are diabetic retinopathy, central retinal vein occlusion, and carotid artery occlusive disease, all of which result in ocular tissue hypoxia. This hypoxia leads to the release of angiogenic factors like vascular endothelial growth factor that induce new blood vessel growth on the iris and in the anterior chamber angle, causing glaucoma. Later sections discuss theories of neovasculogenesis, angiogenic and vasoinhibitory factors, clinical course, differential diagnosis, medical management, and surgical options.
This document discusses scleritis, an inflammation of the sclera. It defines scleritis and notes it is less common than episcleritis. Scleritis can be classified as anterior or posterior, with anterior scleritis subdivided into non-necrotizing diffuse/nodular or necrotizing forms with or without inflammation. Associated systemic diseases are common in 45% of patients. Signs, symptoms, investigations, and treatment are described depending on the classification of scleritis. Surgical treatment may be needed for extreme scleral thinning or corneal complications.
This document provides information on the anatomy and diseases of the vitreous humor. It discusses that the vitreous humor is a jelly-like structure that fills the back of the eye and provides support. Common diseases include vitreous liquefaction, detachment, hemorrhage, and opacities. Vitreous liquefaction is the most common degenerative change and causes floaters. Posterior vitreous detachment often occurs in older individuals and may lead to retinal tears or breaks. Vitreous opacities can result from inflammatory cells, aggregates, tumors or hemorrhages. Vitreous hemorrhage usually stems from retinal vessels and can cause vision loss.
This document summarizes various ocular emergencies and their management. It categorizes emergencies as immediate (within hours), or non-immediate (within a day). Immediate emergencies include chemical burns, central retinal artery occlusion, and orbital hemorrhage. Non-immediate issues include orbital cellulitis, corneal ulcer, hyphema, and retinal detachment. The document provides details on examining, diagnosing, and treating numerous conditions like acute angle closure glaucoma, corneal abrasion, endophthalmitis, penetrating globe injuries, and more. Management approaches focus on stabilizing the patient, identifying the issue, and initiating appropriate treatment to prevent vision loss and complications.
Central serous retinopathy (CSR) is characterized by a serous retinal detachment in the macula due to leakage of fluid from the choroidal vasculature through the retinal pigment epithelium. It typically affects men in their 40s and 50s and can be associated with stress, corticosteroid use, hypertension, and type A personality. While often self-limiting, CSR can become chronic and cause long-lasting visual impairment without treatment. Management options include observation, stress reduction, oral medications like acetazolamide or ketoconazole to lower cortisol levels, and photodynamic therapy for severe or chronic cases.
Spherical and cylindrical lenses are the two main types of lenses. Spherical lenses have a constant curvature across all meridians, while cylindrical lenses have varying curvatures between meridians. Common spherical lens forms include plano-concave, plano-convex, and bi-convex. Tilting a lens can induce astigmatism, with the cylinder power equal to the sphere power and axis along the tilt meridian. The spherical equivalent represents the average power of a lens and is determined by combining half the cylinder power with the sphere power.
Acute Congestive Glaucoma / Optic Neuritis / Painful Loss Of Vision by Dr. Mu...Zeeshan Hameed
About Author:
Dr. Muhammad Zeeshan Hameed MBBS,FCPS(Resident Eye Surgeon)
GMC/DHQ Teachng Hosptal, Gujranwala Pakistan.
About Presentation:
This presentation covers the complete topic of acute congestive glaucoma, optic neuritis and ddx of sudden painful loss of vision
The document discusses retinal vein occlusion (RVO), specifically central retinal vein occlusion (CRVO), including its demographics, pathogenesis, classification as either ischemic or non-ischemic CRVO, management through examination, investigation and treatment options, and guidelines on systemic evaluation and management. CRVO results from obstruction of venous outflow causing increased pressure and damage to retinal capillaries and is classified based on the location and extent of occlusion, with ischemic CRVO having a poorer visual prognosis without timely treatment.
This document summarizes several disorders of the eyelashes including trichiasis, metaplastic lashes, distichiasis, phthiriasis palpebrarum, madarosis, and poliosis. Trichiasis involves the misdirection of normal lashes toward the eye and can cause punctate epitheliopathy and corneal issues. Metaplastic lashes arise from meibomian gland orifices due to conditions like cicatrizing conjunctivitis. Distichiasis has a second row of lashes from the meibomian glands that may be treated by cryotherapy or surgery. Phthiriasis palpebrarum is an infestation by crab lice.
1) Lacrimal gland tumors are rare, representing only 5-18% of orbital lesions, with an incidence of 1 per million people per year. They commonly affect the elderly.
2) The main types of lacrimal gland tumors are epithelial tumors such as pleomorphic adenoma (50% of epithelial tumors) and adenoid cystic carcinoma (50% of malignant tumors).
3) Pleomorphic adenomas typically present as a painless, slowly progressive mass in the lacrimal gland fossa and are usually treated with complete surgical excision. Adenoid cystic carcinomas have a high morbidity and mortality and often involve perineural spread.
This document discusses the fundamentals of phacodynamics, which refers to the interrelationship between the various functions of a phacoemulsification machine. It provides a history of phacoemulsification and defines the key components and parameters of phaco machines. These include ultrasound energy, fluidics systems for irrigation and aspiration, and parameters like power, vacuum, and aspiration flow rate. The document explains how these components and parameters work together to perform different surgical techniques like sculpting, chopping, and quadrant removal during cataract surgery.
This document discusses primary angle closure glaucoma (PACG), a type of glaucoma where the iris occludes the drainage angle of the eye, obstructing aqueous outflow. PACG is a leading cause of glaucoma worldwide and is particularly common in East Asian populations. It is classified based on the degree of iris occlusion and presence of optic nerve damage and vision loss. Risk factors include older age, female sex, Asian ethnicity, family history, hypermetropia, and shorter axial length. Symptoms include blurred vision, halos around lights, eye pain, and headache. Signs include elevated eye pressure, shallow anterior chamber, iris changes, and optic nerve damage. The document outlines methods for diagnosing
This document summarizes retinal artery occlusion, including classifications, epidemiology, clinical features, risk factors, evaluation, treatment, and prognosis for different types. Central retinal artery occlusion typically causes sudden, painless vision loss and has a poor visual prognosis. Branch retinal artery occlusion often causes partial vision loss and has a better prognosis, with vision recovering to 20/40 or better in most cases. Cilioretinal artery occlusion can occur in isolation or with central retinal vein occlusion, and isolated cases typically have a good visual outcome.
Cystoid macular edema (CME) is characterized by intraretinal swelling and fluid-filled cysts visible on optical coherence tomography (OCT). On fluorescein angiography, it appears as abnormal leaking from retinal blood vessels and pooling of dye in the macula. In severe cases, CME can be associated with inflammation in the vitreous and retina. Common causes include retinal vein occlusion, diabetic retinopathy, uveitis, and complications from cataract surgery or other eye procedures. Treatment focuses on reducing inflammation with corticosteroids or NSAIDs, as inflammation plays a role in many cases of CME.
Acute Rise in IOP (Dr. Rasha, senior resident of ophthalmology)Hind Safwat
There are several potential causes of acute increases in intraocular pressure (IOP), including glaucomatocyclitic crisis (Posner-Schlossman syndrome), inflammatory open-angle glaucoma, retrobulbar hemorrhage or inflammation, traumatic glaucoma, pigmentary glaucoma, neovascular glaucoma, plateau iris syndrome, and malignant glaucoma. IOP increases above 40mmHg can rapidly damage the optic nerve and cause permanent vision loss within hours. Treatment depends on the underlying cause but generally involves topical medications to lower IOP such as beta-blockers, alpha-2 agonists, and carbonic anhydrase inhibitors as well as systemic therapies like oral acetazol
This document provides an overview of primary angle-closure glaucoma (PACG). It defines PACG as a type of glaucoma caused by blockage of the aqueous humor outflow due to closure of the anterior chamber angle. The document classifies PACG and discusses its clinical presentations, including latent, subacute, acute, chronic, and absolute forms. Diagnosis involves gonioscopy and provocative tests. Management includes medications, laser iridotomy, and surgery. The goal is to reopen the drainage angle and lower intraocular pressure to prevent vision loss from optic nerve damage.
Optic atrophy is the degeneration of the optic nerve fibers leading to pallor of the optic disc. It can be classified as primary, secondary, consecutive, glaucomatous, or ischemic based on the underlying cause and appearance of the optic disc. Primary optic atrophy results from diseases proximal to the disc with a chalky white appearance. Secondary optic atrophy occurs after inflammation of the optic disc with a dirty white, blurred disc. Consecutive optic atrophy follows retinal lesions and shows a yellow waxy disc. Treatment focuses on the underlying cause when possible, but once complete atrophy occurs, vision cannot be recovered.
1. Viral corneal ulcers have increased due to antibiotics reducing bacterial flora. Herpes simplex virus is a common cause, initially infecting epithelium and potentially becoming neurotropic. Primary infection involves non-immune individuals while recurrent infections reactivate dormant virus.
2. Herpes simplex keratitis manifestations include punctate epithelial keratitis, dendritic ulcers, and stromal keratitis treated with antivirals like acyclovir along with supportive measures. Herpes zoster ophthalmicus affects the trigeminal nerve causing vesicular skin lesions and ocular complications in 50% of cases like keratitis, treated with antivirals and steroids.
The synoptophore is an ophthalmic instrument used to diagnose and treat imbalances of the eye muscles. It consists of two cylindrical tubes with mirrored bends that allow pictures to be presented simultaneously to both eyes, compensating for any angle of squint. It is used to investigate binocular function in patients with a manifest squint, detect suppression and abnormal retinal correspondence, and measure horizontal, vertical and torsional misalignments. The synoptophore can test for three grades of binocular vision - simultaneous perception, fusion, and stereopsis - and detect whether a patient has normal or abnormal retinal correspondence based on differences between subjective and objective angles of squint.
Primary angle closure glaucoma is caused by blockage of the aqueous humor outflow due to closure or narrowing of the anterior chamber angle. It progresses through stages from primary angle closure suspect to acute angle closure to angle closure glaucoma, where glaucomatous optic nerve damage and visual field defects are present. Risk factors include hyperopia, smaller anterior chamber angle, older age, and Asian ethnicity. Treatment begins with intravenous mannitol or glycerol to lower pressure urgently, followed by topical eye drop medications like beta-blockers or pilocarpine. The definitive treatment is laser iridotomy or trabeculectomy surgery to permanently open the drainage angle.
This document discusses neovascular glaucoma, also known as rubeotic glaucoma. It begins by defining the terminology and describing the clinical features. The main causes of neovascular glaucoma are diabetic retinopathy, central retinal vein occlusion, and carotid artery occlusive disease, all of which result in ocular tissue hypoxia. This hypoxia leads to the release of angiogenic factors like vascular endothelial growth factor that induce new blood vessel growth on the iris and in the anterior chamber angle, causing glaucoma. Later sections discuss theories of neovasculogenesis, angiogenic and vasoinhibitory factors, clinical course, differential diagnosis, medical management, and surgical options.
This document discusses scleritis, an inflammation of the sclera. It defines scleritis and notes it is less common than episcleritis. Scleritis can be classified as anterior or posterior, with anterior scleritis subdivided into non-necrotizing diffuse/nodular or necrotizing forms with or without inflammation. Associated systemic diseases are common in 45% of patients. Signs, symptoms, investigations, and treatment are described depending on the classification of scleritis. Surgical treatment may be needed for extreme scleral thinning or corneal complications.
This document provides information on the anatomy and diseases of the vitreous humor. It discusses that the vitreous humor is a jelly-like structure that fills the back of the eye and provides support. Common diseases include vitreous liquefaction, detachment, hemorrhage, and opacities. Vitreous liquefaction is the most common degenerative change and causes floaters. Posterior vitreous detachment often occurs in older individuals and may lead to retinal tears or breaks. Vitreous opacities can result from inflammatory cells, aggregates, tumors or hemorrhages. Vitreous hemorrhage usually stems from retinal vessels and can cause vision loss.
This document summarizes various ocular emergencies and their management. It categorizes emergencies as immediate (within hours), or non-immediate (within a day). Immediate emergencies include chemical burns, central retinal artery occlusion, and orbital hemorrhage. Non-immediate issues include orbital cellulitis, corneal ulcer, hyphema, and retinal detachment. The document provides details on examining, diagnosing, and treating numerous conditions like acute angle closure glaucoma, corneal abrasion, endophthalmitis, penetrating globe injuries, and more. Management approaches focus on stabilizing the patient, identifying the issue, and initiating appropriate treatment to prevent vision loss and complications.
Central serous retinopathy (CSR) is characterized by a serous retinal detachment in the macula due to leakage of fluid from the choroidal vasculature through the retinal pigment epithelium. It typically affects men in their 40s and 50s and can be associated with stress, corticosteroid use, hypertension, and type A personality. While often self-limiting, CSR can become chronic and cause long-lasting visual impairment without treatment. Management options include observation, stress reduction, oral medications like acetazolamide or ketoconazole to lower cortisol levels, and photodynamic therapy for severe or chronic cases.
Spherical and cylindrical lenses are the two main types of lenses. Spherical lenses have a constant curvature across all meridians, while cylindrical lenses have varying curvatures between meridians. Common spherical lens forms include plano-concave, plano-convex, and bi-convex. Tilting a lens can induce astigmatism, with the cylinder power equal to the sphere power and axis along the tilt meridian. The spherical equivalent represents the average power of a lens and is determined by combining half the cylinder power with the sphere power.
Acute Congestive Glaucoma / Optic Neuritis / Painful Loss Of Vision by Dr. Mu...Zeeshan Hameed
About Author:
Dr. Muhammad Zeeshan Hameed MBBS,FCPS(Resident Eye Surgeon)
GMC/DHQ Teachng Hosptal, Gujranwala Pakistan.
About Presentation:
This presentation covers the complete topic of acute congestive glaucoma, optic neuritis and ddx of sudden painful loss of vision
The document discusses retinal vein occlusion (RVO), specifically central retinal vein occlusion (CRVO), including its demographics, pathogenesis, classification as either ischemic or non-ischemic CRVO, management through examination, investigation and treatment options, and guidelines on systemic evaluation and management. CRVO results from obstruction of venous outflow causing increased pressure and damage to retinal capillaries and is classified based on the location and extent of occlusion, with ischemic CRVO having a poorer visual prognosis without timely treatment.
This document summarizes several disorders of the eyelashes including trichiasis, metaplastic lashes, distichiasis, phthiriasis palpebrarum, madarosis, and poliosis. Trichiasis involves the misdirection of normal lashes toward the eye and can cause punctate epitheliopathy and corneal issues. Metaplastic lashes arise from meibomian gland orifices due to conditions like cicatrizing conjunctivitis. Distichiasis has a second row of lashes from the meibomian glands that may be treated by cryotherapy or surgery. Phthiriasis palpebrarum is an infestation by crab lice.
1) Lacrimal gland tumors are rare, representing only 5-18% of orbital lesions, with an incidence of 1 per million people per year. They commonly affect the elderly.
2) The main types of lacrimal gland tumors are epithelial tumors such as pleomorphic adenoma (50% of epithelial tumors) and adenoid cystic carcinoma (50% of malignant tumors).
3) Pleomorphic adenomas typically present as a painless, slowly progressive mass in the lacrimal gland fossa and are usually treated with complete surgical excision. Adenoid cystic carcinomas have a high morbidity and mortality and often involve perineural spread.
This document discusses the fundamentals of phacodynamics, which refers to the interrelationship between the various functions of a phacoemulsification machine. It provides a history of phacoemulsification and defines the key components and parameters of phaco machines. These include ultrasound energy, fluidics systems for irrigation and aspiration, and parameters like power, vacuum, and aspiration flow rate. The document explains how these components and parameters work together to perform different surgical techniques like sculpting, chopping, and quadrant removal during cataract surgery.
This document discusses primary angle closure glaucoma (PACG), a type of glaucoma where the iris occludes the drainage angle of the eye, obstructing aqueous outflow. PACG is a leading cause of glaucoma worldwide and is particularly common in East Asian populations. It is classified based on the degree of iris occlusion and presence of optic nerve damage and vision loss. Risk factors include older age, female sex, Asian ethnicity, family history, hypermetropia, and shorter axial length. Symptoms include blurred vision, halos around lights, eye pain, and headache. Signs include elevated eye pressure, shallow anterior chamber, iris changes, and optic nerve damage. The document outlines methods for diagnosing
This document summarizes retinal artery occlusion, including classifications, epidemiology, clinical features, risk factors, evaluation, treatment, and prognosis for different types. Central retinal artery occlusion typically causes sudden, painless vision loss and has a poor visual prognosis. Branch retinal artery occlusion often causes partial vision loss and has a better prognosis, with vision recovering to 20/40 or better in most cases. Cilioretinal artery occlusion can occur in isolation or with central retinal vein occlusion, and isolated cases typically have a good visual outcome.
Cystoid macular edema (CME) is characterized by intraretinal swelling and fluid-filled cysts visible on optical coherence tomography (OCT). On fluorescein angiography, it appears as abnormal leaking from retinal blood vessels and pooling of dye in the macula. In severe cases, CME can be associated with inflammation in the vitreous and retina. Common causes include retinal vein occlusion, diabetic retinopathy, uveitis, and complications from cataract surgery or other eye procedures. Treatment focuses on reducing inflammation with corticosteroids or NSAIDs, as inflammation plays a role in many cases of CME.
Acute Rise in IOP (Dr. Rasha, senior resident of ophthalmology)Hind Safwat
There are several potential causes of acute increases in intraocular pressure (IOP), including glaucomatocyclitic crisis (Posner-Schlossman syndrome), inflammatory open-angle glaucoma, retrobulbar hemorrhage or inflammation, traumatic glaucoma, pigmentary glaucoma, neovascular glaucoma, plateau iris syndrome, and malignant glaucoma. IOP increases above 40mmHg can rapidly damage the optic nerve and cause permanent vision loss within hours. Treatment depends on the underlying cause but generally involves topical medications to lower IOP such as beta-blockers, alpha-2 agonists, and carbonic anhydrase inhibitors as well as systemic therapies like oral acetazol
This document provides an overview of primary angle-closure glaucoma (PACG). It defines PACG as a type of glaucoma caused by blockage of the aqueous humor outflow due to closure of the anterior chamber angle. The document classifies PACG and discusses its clinical presentations, including latent, subacute, acute, chronic, and absolute forms. Diagnosis involves gonioscopy and provocative tests. Management includes medications, laser iridotomy, and surgery. The goal is to reopen the drainage angle and lower intraocular pressure to prevent vision loss from optic nerve damage.
Acute angle closure crisis is an ophthalmic emergency caused by sudden rise in intraocular pressure due to blockage of aqueous outflow. It presents with severe eye pain, blurred vision, nausea and elevated eye pressure. Immediate treatment involves intravenous acetazolamide and topical eye drops to lower pressure. If pressure is not controlled, further interventions like laser iridotomy or surgery may be needed to relieve the block and prevent permanent vision loss from optic nerve damage. Long term management involves treatment of the underlying primary angle closure glaucoma with pressure lowering eye drops or surgery and monitoring for glaucoma progression.
Acute angle closure glaucoma (AACG) is characterized by a sudden increase in intraocular pressure (IOP) due to blockage of the eye's drainage angle by the iris. Emergency treatment involves rapidly lowering IOP through medications like acetazolamide and topical beta-blockers, as well as steroids to reduce inflammation. If medical therapy is ineffective, further interventions like osmotic agents, corneal indentation, or laser peripheral iridotomy within 24-48 hours may be needed to fully resolve the condition and prevent future attacks.
Primary angle closure glaucoma (PACG) involves blockage of the drainage angle between the iris and cornea, increasing intraocular pressure. It has acute, subacute, and chronic stages depending on symptoms and pressure changes. Treatment involves lowering pressure through medications, laser iridotomy to open the angle, or surgery if needed. A red eye from PACG presents with severe vision loss, pain, mid-dilated pupil, and high pressure, distinguishing it from conjunctivitis, uveitis, or other causes.
Glaucoma is a condition where fluid pressure inside the eye rises above healthy levels, which can damage the optic nerve and cause vision loss or blindness if untreated. The two main types are primary open-angle glaucoma, the most common, and closed-angle glaucoma, which is a medical emergency. Treatment aims to lower intraocular pressure through eyedrops, laser treatment, or surgery depending on the type of glaucoma. Regular eye exams are important for early detection and treatment.
Glaucoma is an eye disorder characterized by changes in the optic nerve and loss of vision. It occurs when the intraocular pressure is too high for the optic nerve to function normally. There are several types including primary open-angle glaucoma which is the most common. Risk factors include age over 40, family history, and high eye pressure. Symptoms may include vision loss and visual field defects. Treatment aims to lower eye pressure through eye drops, laser treatment, or surgery depending on the type and severity of glaucoma. Homeopathic medicines can help improve circulation, drainage, and blood supply to slow progression and control symptoms.
INTRODUCTION
ETIOLOGY
RISK FACTORS
PATHOPHYSIOLOGY
CLASSIFICATION
CLINICAL FEATURES
DIAGNOSTIC MEASURES
MANAGEMENT
Medical
Surgical
Nursing
CONCLUSION
BIBLIOGRAPHY
POST TEST
This document provides information about glaucoma, including its etiology, pathophysiology, clinical manifestations, types, and treatment options. It begins with an introduction to glaucoma as a group of disorders characterized by optic nerve damage and vision loss associated with increased intraocular pressure. The document then discusses the etiology and pathophysiology of glaucoma, describing how increased pressure damages the optic nerve. It outlines the clinical signs and symptoms of various types of glaucoma and concludes by explaining non-pharmacological and pharmacological treatment approaches that aim to lower intraocular pressure and prevent further vision loss.
Glaucoma is a group of eye disorders characterized by optic nerve damage and vision loss caused by increased pressure in the eye. The document discusses the definition, epidemiology, causes, clinical features, diagnosis, and management of primary open angle glaucoma and primary angle-closure glaucoma. Key points include that glaucoma has various clinical manifestations, causes include blockage of aqueous outflow, signs involve optic nerve changes and visual field loss, and treatment aims to lower intraocular pressure through medication, laser treatment, or surgery.
Glaucoma and dgharia of bgf vfcvf vf .pptxmekulecture
Under normal conditions, only approximately 1 mL of the 125 mL of glomerular filtrate that is formed each minute is excreted in the urine.
The other 124 mL is reabsorbed in the tubules.
This means that the average output of urine is approximately 60 mL/hour… approximately 1.5L urine per day.
1. Glaucoma is a group of eye disorders characterized by increased intraocular pressure (IOP) that can damage the optic nerve and result in vision loss.
2. It is caused by an imbalance between the production and drainage of aqueous humor in the eye, which increases IOP.
3. The main types are open-angle glaucoma and angle-closure glaucoma. Treatment involves medications, laser treatments, surgery, and maintaining proper IOP levels to prevent further vision loss.
4. Nursing care focuses on teaching patients about proper medication administration, eye drop techniques, the importance of follow up exams, managing pain and anxiety, and promoting independent living through low vision adaptations.
1) Primary angle-closure glaucoma occurs when the iris blocks the drainage angle of the eye, preventing aqueous humor from draining properly and increasing intraocular pressure. It is usually caused by pupillary block from apposition of the iris and lens.
2) Acute primary angle closure presents with sudden severe eye pain, blurred vision, headache, and nausea. Chronic primary angle closure develops slowly over time with peripheral anterior synechiae formation.
3) Risk factors include hyperopia, smaller eye size, and increasing age when the lens thickens. Treatment involves medical management to lower pressure urgently followed by laser iridotomy or surgery to permanently open the drainage angle.
Glaucoma is a group of eye diseases characterized by increased pressure in the eye due to excess fluid. If left untreated, it can cause damage to the optic nerve and result in vision loss and blindness. The two most common types are open-angle glaucoma, which develops slowly over time, and angle-closure glaucoma, which develops acutely and requires urgent treatment to prevent vision loss. Treatment involves medications or surgery to lower eye pressure and prevent further damage to the optic nerve. Nursing care focuses on administering eye drop medications correctly, providing a safe environment, teaching patients self-care techniques, and ensuring follow-up medical care.
This presentation describes all clinical aspects of primary angle closure glaucoma in a concentrated and simplified manner....you can watch the illustrated presentation at the following link:
1. Glaucoma is a group of eye disorders characterized by increased intraocular pressure and optic nerve damage that can lead to vision loss.
2. Risk factors include age, family history, diabetes, eye injuries or abnormalities.
3. Treatment involves lifelong use of eye drops or surgery to lower pressure and prevent further vision loss.
Glaucoma types, Pathogenesis, Diagnosis and TreatmentPranatiChavan
Glaucomas are ocular disorders characterized by changes in the optic nerve head (optic disk) and by loss of visual sensitivity and field.
There are two major types of glaucoma: open-angle glaucoma, which accounts for most cases and closed-angle glaucoma.
This document provides an overview of glaucoma, including its pathophysiology, types, diagnosis, and treatment. Glaucoma is an optic neuropathy characterized by optic nerve damage and visual field loss. It is traditionally associated with elevated intraocular pressure but can occur with normal pressure as well. The two main types are open-angle glaucoma, the most common, and closed-angle glaucoma, which has a sudden onset. Treatment involves medications, laser therapy, or surgery to lower intraocular pressure and prevent further nerve damage.
GLAUCOMA of human eye for certificate nursesokumuatanas1
This document provides information about glaucoma, including:
1. Glaucoma is a group of eye conditions marked by increased pressure in the eye that can damage the optic nerve and lead to vision loss.
2. The two main types are open-angle glaucoma, the most common type caused by blocked drainage canals, and angle-closure glaucoma caused by a narrowed drainage angle.
3. Risk factors include age over 40, family history, and other medical conditions. Treatment involves medications, laser treatments, or surgeries to lower pressure and improve drainage in the eye.
The choroid is the vascular layer of the eye located between the retina and sclera. It develops from mesenchyme surrounding the eye. The choroid consists of three layers - an outer layer of large vessels, a middle layer of medium vessels, and an inner layer of densely packed capillaries. It receives its blood supply from the short and long posterior ciliary arteries and is innervated by both the sympathetic and parasympathetic nervous systems to regulate blood flow. The choroid plays an important role in nourishing the outer retina and maintaining a smooth interface for retinal attachment.
thesis statement is a sentence that sums up the central point of your paper or essay. It usually comes near the end of your introduction.
Your thesis will look a bit different depending on the type of essay you’re writing. But the thesis statement should always clearly state the main idea you want to get across. Everything else in your essay should relate back to this idea.
Example: Thesis statement
Despite Oscar Wilde’s Aestheticist claims that art needs no justification or purpose, his work advocates Irish nationalism, women’s suffrage, and socialism.
You can write your thesis statement by following four simple step
The retina (from "net") is the innermost, light-sensitive layer of tissue of the eye of most vertebrates and some molluscs. The optics of the eye create a focused two-dimensional image of the visual world on the retina, which then processes that image within the retina and sends nerve impulses along the optic nerve to the visual cortex to create visual perception
The retina is the tissue layer located in the back of your eye. This layer transforms light into nerve signals that are then sent to the brain for interpretation.
When your blood pressure is too high, the retina’s blood vessel walls may thicken. This may cause your blood vessels to become narrow, which then restricts blood from reaching the retina. In some cases, the retina becomes swollen.
Over time, high blood pressure can cause damage to th
orneal ulcer, also called keratitis, is an inflammatory or, more seriously, infective condition of the cornea involving disruption of its epithelial layer with involvement of the corneal stroma. It is a common condition in humans particularly in the tropics and in farming. In developing countries, children afflicted by vitamin A deficiency are at high risk for corneal ulcer and may become blind i
Lung volumes and lung capacities refer to the volume of air in the lungs at different phases of the respiratory cycle.
The average total lung capacity of an adult human male is about 6 litres of air.[1]
Tidal breathing is normal, resting breathing; the tidal volume is the volume of air that is inhaled or exhaled in only a single such breath.
The average human respiratory rate is 30–60 breaths per minute at birth,[2] decreasing to 12–20 breaths per minute in adults.[3
Dry eye disease is a common condition that occurs when your tears aren't able to provide adequate lubrication for your eyes. Tears can be inadequate and unstable for many reasons. For example, dry eyes may occur if you don't produce enough tears or if you produce poor-quality tears. This tear instability leads to inflammation and damage of the eye's surface.
Dry eyes feel uncomfortable. If you have dry eyes, your eyes may sting or burn. You may experience dry eyes in certain situations, such as on an airplane, in an air-conditioned room, while riding a bike or after looking at a computer screen for a few hours
SUPERIOR OBLIQUE PALSY MANAGEMENT [Autosaved].pptxudayasree30
These tissues are known as trochlea. The superior oblique muscle allows the eye to be turned downward and inward. When the fourth cranial nerve is injured or diseased, it can cause paralysis of the superior oblique muscle. This is known as superior oblique palsy, trochlear nerve palsy, or fourth nerve palsy.
The International Classification of Diseases 11 (2018) classifies vision impairment into two groups, distance and near presenting vision impairment.
Distance vision impairment:
Mild – visual acuity worse than 6/12 to 6/18
Moderate – visual acuity worse than 6/18 to 6/60
Severe – visual acuity worse than 6/60 to 3/60
Blindness – visual acuity worse than 3/60
Nearsightedness (myopia) is a common vision condition in which near objects appear clear, but objects farther away look blurry. It occurs when the shape of the eye — or the shape of certain parts of the eye — causes light rays to bend (refract) inaccurately. Light rays that should be focused on nerve tissues at the back of the eye (retina) are focused in front of the retina.
Nearsightedness usually develops during childhood and adolescence, and it usually becomes more stable between the ages of 20 and 40. Myopia tends to run in families.
A basic eye exam can confirm nearsightedness. You can compensate for the blurry vision with eyeglasses, contact lenses or refractive surgery.
5-hydroxytryptamine or 5-HT or Serotonin is a neurotransmitter that serves a range of roles in the human body. It is sometimes referred to as the happy chemical since it promotes overall well-being and happiness.
It is mostly found in the brain, intestines, and blood platelets.
5-HT is utilised to transport messages between nerve cells, is known to be involved in smooth muscle contraction, and adds to overall well-being and pleasure, among other benefits. 5-HT regulates the body's sleep-wake cycles and internal clock by acting as a precursor to melatonin.
It is hypothesised to regulate hunger, emotions, motor, cognitive, and autonomic processes.
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Travel vaccination in Manchester offers comprehensive immunization services for individuals planning international trips. Expert healthcare providers administer vaccines tailored to your destination, ensuring you stay protected against various diseases. Conveniently located clinics and flexible appointment options make it easy to get the necessary shots before your journey. Stay healthy and travel with confidence by getting vaccinated in Manchester. Visit us: www.nxhealthcare.co.uk
DECLARATION OF HELSINKI - History and principlesanaghabharat01
This SlideShare presentation provides a comprehensive overview of the Declaration of Helsinki, a foundational document outlining ethical guidelines for conducting medical research involving human subjects.
These lecture slides, by Dr Sidra Arshad, offer a simplified look into the mechanisms involved in the regulation of respiration:
Learning objectives:
1. Describe the organisation of respiratory center
2. Describe the nervous control of inspiration and respiratory rhythm
3. Describe the functions of the dorsal and respiratory groups of neurons
4. Describe the influences of the Pneumotaxic and Apneustic centers
5. Explain the role of Hering-Breur inflation reflex in regulation of inspiration
6. Explain the role of central chemoreceptors in regulation of respiration
7. Explain the role of peripheral chemoreceptors in regulation of respiration
8. Explain the regulation of respiration during exercise
9. Integrate the respiratory regulatory mechanisms
10. Describe the Cheyne-Stokes breathing
Study Resources:
1. Chapter 42, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 36, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 13, Human Physiology by Lauralee Sherwood, 9th edition
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2. An attack of acute primary angle closure glaucoma occurs
due to a sudden total angle closure leading to severe rise in
IOP.
May occur due to pupillary block.
This is sight threatening emergency.
3. CLINICAL FEATURES
SYMPTOMS
Pain: Typically acute attack is characterised by sudden onset
of very severe pain in the eye which radiates along the
branches of 5th nerve.
Nausea, vomiting and prostrations are frequently
associated with pain.
Severe unilateral headache
4. Rapidly progressive impairment of vision, redness,
photophobia and lacrimation develop in all cases.
Past history: About 5 percent patients give history of typical
previous intermittent attacks of subacute angle-closure
glaucoma.
5. SIGNS
Lids may be oedematous.
Conjunctiva is chemosed, and congested, (both conjunctival
and ciliary vessels are congested).
Cornea becomes oedematous and insensitive.
Anterior chamber is very shallow.
Aqueous flare or cells may be seen in anterior chamber.
8. Angle of anterior chamber is completely closed as seen on
gonioscopy (shaffer grade 0).
Iris may be discoloured.
Pupil is semi-dilated, vertically oval and fixed. It is non-
reactive to both light and accommodation
9.
10. Angle structures from anterior to
posterior
Schwalbe’s line
Trabecular meshwork
Scleral spur
Ciliary body band
Root of iris
11.
12. IOP is markedly elevated, usually between 40 and 70 mm of
Hg.
Optic disc is oedematous and hyperaemic.
Fellow eye shows shallow anterior chamber and a narrow
angle (latent angle closure glaucoma).
19. MANAGEMENT
It is a serious ocular emergency and needs to be managed
aggressively.
Immediate medical therapy to lower IOP.
Definitive treatment.
Prophylaxis of fellow eye.
Long term glaucoma surveillance and IOP management in
both eyes.
20. immediate medical therapy to lower IOP:
Systemic hyperosmotic agents if IOP is more than 40 mmHg
intravenous mannitol 20%(1-2gm/kg body weight)
Oral hyperosmotics eg: 50% glycerol (1gm/kg body weight)
in lemon juice may be given.
C/I in diabetes mellitus
21. Systemic carbonic anhydrase inhibitors:
Eg: acetazolamide 500 mg IV stat followed by 250mg tablet 3
times a day.
Topical antiglaucoma drugs:
Beta-blockers eg: 0.5% timolol or 0.5% betaxolol.
Alpha adrenergic agonists eg: brimonidine 0.1-0.2%
Prostaglandin analogue eg: latanoprost 0.005%
22. Pilocarpine eyedrops should be started after the IOP is bit
lowered by hyperosomtic agents.
At higher pressure iris sphincter is ischaemic and
unresponsive to pilocarpine.
Initially 2 percent pilocarpine should be administered every
30 minutes for 1-2 hours and then 6 hourly
23. Central corneal indentation with a squint hook or
indentation goniolens to force aqueous into the angle.
Epithelial oedema can be cleared first with topical 50%
glycerol to improve visualization and to avoid abrasion
24. Analgesics and antiemetics may be required.
Topical steroids like 1% prednisolone acetate or
dexamethasone eye drops administered 3 – 4 times a day
reduces inflammation.
25. Definitive therapy
Laser peripheral iriotomy:
goniscopy should be performed as soon as cornea becomes
clear.
Laser PI should be performed if PAS are seen in <270 angle.
LPI re-establishes communication between posterior and
anterior chamber so it bypasses pupillary block and
immediately relieves the crowding of the angle.
26. Filtration surgery: It should be performed in cases where
IOP is not controlled with the best medical therapy
following an attack of acute congestive glaucoma and also
when peripheral anterior synechiae are formed in more than
270 degrees of the angle of the anterior chamber.
Mechanism: Filtration surgery provides an alternative to the
angle for drainage of aqueous from anterior chamber into
subconjunctival space.
27. Clear lens extraction by phacoemulsification with
intraocular lens implantation by has recent been
recommended by some workers.
28. Prophylactic treatment in the normal fellow eye
Prophylactic laser iridotomy (preferably) or surgical
peripheral iridectomy should be performed on the fellow
asymptomatic eye.
It should be done as early as possible as chances of acute
attack are 50% in such eyes.
29. Long term glaucoma surveillance and IOP management in
both eyes.
It is must to ultimately prevent glaucomatous blindness.
Eyes treated with PI may develop PACG at any time, so it
should be treated as when required.
Filtration surgery may fail anytime during course and hence
need to be repeated with antimetabolites.
30. Sequelae of acute PAC
Postsurgical acute PAC
Spontaneous angle reopening
Ciliary body shut down
Vogt’s triad
31. Postsurgical acute PAC:
This refers to the clinical status of the eye after laser
peripheral iridotomy (PI) for an attack of acute PAC. It may
occur in two clinical settings :
i. With normalized IOP after successful laser PI, the eye
usually quitens after some time with or without marks of an
acute attack.
ii. With raised IOP after unsuccessful laser PI, there occurs a
state of chronic congestive glaucoma. It needs to be treated
by trabeculectomy operation
32. Spontaneous angle opening:
It may very rarely occur in some cases and the attack of
acute PACG may subside itself without treatment.
Treatment of such cases is laser peripheral iridotomy.
33. Ciliary body shut down:
It refers to temporary cessation of aqueous humour
secretion due to ischaemic damage to the ciliary epithelium
after an attack of acute PACG.
Clinical features in this stage are similar to acute congestive
glaucoma except that the IOP is low and pain is markedly
reduced
34. Treatment includes:
Topical steroid drops to reduce inflammation.
Laser iridotomy should be performed when the cornea
becomes clear and IOP should be monitored.
Trabeculectomy is required when IOP rises constantly.
35. Vogt’s triad
It may be seen in patients with any type of postcongesive
glaucoma and in treated cases of acute congestive glaucoma.
It is characterized by:
Glaucomflecken (anterior subcapsular lenticular opacity),
Patches of iris atrophy, and
Slightly dilated non-reacting pupil (due to sphincter
atrophy).
36.
37. A patient presents with sudden onset of severe unilateral
eye pain, headache associated with blurred vision, rainbow
colored haloes around bright light, nausea and vomiting.
Examination revealed a fixed midpoint pupil and a hazy,
cloudy cornea with marked conjunctival congestion. What
could be the diagnosis? Discuss the pathophysiology and
treatment of the above condition.
38. Differential diagnosis of acute red eye. Describe clinical
features and management of acute congestive glaucoma.