This is a chapter from Grainger and Allison. I have Coolected all images from chapter 20 with caption in this presentation.
In my opinion it will be very benificial to have this in your android. ,
This presentation is almost a complete Pictoral view of Radiograph chest.
This presentation will help radiologist in daily reporting.
This presentation will help physicians, surgeons, anesthetist and almost all medical professionals in diagnosing commonly presenting cardiac diseases.
This will also help all in preparaing TOACS examination.
Right Paratracheal Stripe
Posterior wall of the bronchus intermedius
Left Paratracheal Stripe
Left subclavian artery border
Posterior-superior junction line
This presentation is almost a complete Pictoral view of Radiograph chest.
This presentation will help radiologist in daily reporting.
This presentation will help physicians, surgeons, anesthetist and almost all medical professionals in diagnosing commonly presenting cardiac diseases.
This will also help all in preparaing TOACS examination.
Right Paratracheal Stripe
Posterior wall of the bronchus intermedius
Left Paratracheal Stripe
Left subclavian artery border
Posterior-superior junction line
Role of MDCT MULTISCLICE in coronary artery part 5 (non atherosclerotic coron...AHMED ESAWY
Role of mdc tin coronary artery part 5 (non atherosclerotic coronary abnormalities) dr ahmed esawy
Role of mdc tin coronary artery part 4 (anomalous coronary arteries) dr ahmed esawy
This Presentation is basically image collection from chapter 9 of GRAINGER & ALLISON’S DIAGNOSTIC RADIOLOGY.
This is an effort to present the most authentic images.
A detailed description of ct coronary angiography and calcium scoring with various aspects regarding the preparation, procedure, limitations and a short review regarding post CABG imaging.
This is a chapter from Grainger and Allison. I have Coolected all images from chapter 19 with caption in this presentation.
In my opinion it will be very benificial to have this in your android. ,
Pulmonary Lobar Collapse:Essential Considerations 14 Dr. Muhammad Bin ZulfiqarDr. Muhammad Bin Zulfiqar
This presentation is from 15th chapter of Grainger and Allison--Diagnostic Radiology A TEXTBOOK OF MEDICAL IMAGING.
My aim behind all these presentation is to provide authentic images. As our all radiology revolve around images of diseases. We can put these ppts in our androids for study and references.
Dislocation of joint is very tricky. In this presentation radiological evaluation of Dislocation of various joints will be discussed.
This is one of the best pictoral review of important joint dislocations
Renal Color Doppler Ultrasound.
After studying this presentation one will be able to perform and interpret ultrasound.
This presntation in my opinion is best short analog to text.
In this presentation we will discuss the bone age assessment mainly focusing wrist radiograph.
we shall also highlights some points in adult bone age
Basically it is an introduction. We shall not discuss its judicial importance
Role of medical imaging in developemental dysplasia of Hip Dr muhammad Bin Zu...Dr. Muhammad Bin Zulfiqar
In this presentation we will discuss the role of medical imaging---plain Radiography, Ultrasound,Arthrography, CT and MRI in the evaluation of Developemental dysplasia of hip. Our main focuss will be on Sonographic evaluation.
In this presentation we will discuss the basic of axial trauma from head to pelvis. We will discuss the important key points that aids in the diagnosis of axial trauma
This is a chapter from Grainger and Allison. I have Coolected all images from chapter 21 with caption in this presentation.
In my opinion it will be very benificial to have this in your android.
This presentation is the first series of the MR imaging of Knee.
In this presentation MRI anatomy has been discussed. As we all know good knowledge of medical imaging three dimensional anatomy is key for good reporting.
Hope we all get benifitted.
Suggestions are most welcome
In this presentation we will dscuss the imp imaging features of Posterior fossa tumors in pediatric age group.
Medulloblastoma
Pilocytic Astrocytoma
Ependymoma
Brainstem Glioma
Schwanoma
Meningioma
Epidermoid Cyst
Arachnoid Cyst
In this presentation we will discuss about the
Anatomy of Prostate
Technique of Transrectal US
Carcinoma Prostate and
Different modes of prostatic biopsy.
In this presentation we shall discuss all fractures with specific names .
This is a pictoral review.
This presentation will be very helpful for radiologist to have in their androids to help them in rapid reporting
In this presentation all images of Chapter 18 from Grainger and Allison have been discussed.
Our aim is to discuss authentic material .
This is only for educational purposes.
In this chapter air space infilteration have been discussed. Ground glass haze and consolidation are discussed in detail.
This presentation is a selection of images from 17th chapter of grainger and allison.
Our aim is to provide standard and proved cases of the disease process.
This all is for educational purpose
Objectives of this presentation are
Introduction to ct
Cross sectional anatomy
Common important pathologies
This presentation is aimed to educate beginers to help in ct interpretetion.
16 High Resolution Computed Tomography of Interstitial and Occupational Lung ...Dr. Muhammad Bin Zulfiqar
This presentation is collection of images from chapter 16 of Grainger and Allison.
Inthis we will discuss the ILD.
This is only for educational purposes.
This Presentation is a collection of chapter 5 images from Grainger and Allison.
Our aim is to study authentic data.
This is only for educational purposes
In this presentation we will discuss role of high resolution in characterizing normal variant and pathologies of spinal pathologies.
This is a pictoral review.
This presentation provides sufficient material for anyone who wants is interested in interventional radiology. Here we will discuss the available facilities, mechanisms and equipments.
In my opinion this presentation will prove a footstep in interventional radiology
Hepatocellular carcinoma—role of interventional radiologist Dr. Muhammad Bin ...Dr. Muhammad Bin Zulfiqar
In these presentation we will discuss the merits, demrits and outcomes of various interventional radiology modalities for the treatment of hepatocellular carcinoma
This presentation is from 13th chapter of Grainger and Allison--Diagnostic Radiology A TEXTBOOK OF MEDICAL IMAGING.
My aim behind all these presentation is to provide authentic images. As our all radiology revolve around images of diseases. We can put these ppts in our androids for study and references.
In this presentation we will focus on aetiological factors that cause infirtility. Our focus is on US depiction of these aetiological factors to help physician in the management of infirtility.
We have nothing to do with direct radiological intervention in the management of infirtility in this presentation.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
HOT NEW PRODUCT! BIG SALES FAST SHIPPING NOW FROM CHINA!! EU KU DB BK substit...GL Anaacs
Contact us if you are interested:
Email / Skype : kefaya1771@gmail.com
Threema: PXHY5PDH
New BATCH Ku !!! MUCH IN DEMAND FAST SALE EVERY BATCH HAPPY GOOD EFFECT BIG BATCH !
Contact me on Threema or skype to start big business!!
Hot-sale products:
NEW HOT EUTYLONE WHITE CRYSTAL!!
5cl-adba precursor (semi finished )
5cl-adba raw materials
ADBB precursor (semi finished )
ADBB raw materials
APVP powder
5fadb/4f-adb
Jwh018 / Jwh210
Eutylone crystal
Protonitazene (hydrochloride) CAS: 119276-01-6
Flubrotizolam CAS: 57801-95-3
Metonitazene CAS: 14680-51-4
Payment terms: Western Union,MoneyGram,Bitcoin or USDT.
Deliver Time: Usually 7-15days
Shipping method: FedEx, TNT, DHL,UPS etc.Our deliveries are 100% safe, fast, reliable and discreet.
Samples will be sent for your evaluation!If you are interested in, please contact me, let's talk details.
We specializes in exporting high quality Research chemical, medical intermediate, Pharmaceutical chemicals and so on. Products are exported to USA, Canada, France, Korea, Japan,Russia, Southeast Asia and other countries.
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...
20 congenital heart disease Dr. Muhammmad Bin Zulfiqar
1. DR. MUHAMMAD BIN ZULFIQAR
PGR IV FCPS SIMS/SHL
radiombz@gmail.com
20 Congenital Heart Disease:
General Principles and Imaging Grainger and Allison
2. • FIGURE 20-1 ■ Perioperative CXR. (A) A 3-year-old patient
following total cavopulmonary connection surgery, postoperative
CXR demonstrating tube positions in intensive care. Note two chest
and one mediastinal drains, endotracheal tube and veno-venous
collateral occluder device (right upper zone). (B) Third
postoperative day following extubation and removal of mediastinal
drain. Note change in cardiomediastinal contour caused by large
pericardial clot, requiring evacuation.
3. • FIGURE 20-2 ■ Physiological assessment using CXR.
(A) Pulmonary plethora in a patient with a VSD. Note
the increased number and size of discrete vessels
without haziness. (B) Pulmonary oedema in a supine
patient with cor triatriatum (membranous obstruction
to LA outflow) resulting in increased pulmonary venous
pressure. Note cardiomegaly, perihilar alveolar
haziness/consolidation and peribronchial cuffing
4. • FIGURE 20-3 ■ Pulmonary oligaemia. (A) Supine AP CXR, in an 8-
week-old patient with tetralogy of Fallot with severe pulmonary
stenosis and cyanosis. Note black lungs with sparse, small-calibre
vessels. (B) Supine AP CXR, in the same patient following
construction of a right modified Blalock–Taussig (BT) shunt on the
next day. Note the increased size of the left cardiac contour due to
increased LV filling, increased pulmonary vascular markings, now
plethoric, suggestive of high pulmonary blood flow arising from the
shunt. Indeed, the patient had compromised systemic perfusion
due to redistribution of cardiac output to the lungs, necessitatin
clipping the shunt to reduce its calibre
5. • FIGURE 20-4 ■ Atrial septal defects. (A) Schematic drawing of ASD positions. (B)
b-SSFP CMR image. Four-chamber view showing a large secundum ASD with
posterior extension. The absence of a posterior rim (arrow) precludes insertion of
an ASD closure device. Note the dilated right atrium (RA), and right ventricle (RV),
and flattened interventricular septum. (C) b-SSFP CMR image. Axial view showing a
large superior sinus venosus defect, with PAPVD of the right upper and right
middle pulmonary veins, straddling the deficient atrial septum (arrow). (D) Plot of
instantaneous flow (measured by velocity-encoded phase-contrast MRI) as a
function of time showing a left-to-right shunt through an ASD; note increased
pulmonary blood flow
6. • FIGURE 20-4 ■ Atrial septal defects. (A) Schematic drawing of ASD
positions. (B) b-SSFP CMR image. Four-chamber view showing a large
secundum ASD with posterior extension. The absence of a posterior rim
(arrow) precludes insertion of an ASD closure device. Note the dilated
right atrium (RA), and right ventricle (RV), and flattened interventricular
septum. (C) b-SSFP CMR image. Axial view showing a large superior sinus
venosus defect, with PAPVD of the right upper and right middle pulmonary
veins, straddling the deficient atrial septum (arrow). (D) Plot of
instantaneous flow (measured by velocity-encoded phase-contrast MRI) as
a function of time showing a left-to-right shunt through an ASD; note
increased pulmonary blood flow
7. • FIGURE 20-5 ■ Atrioventricular septal defects. (A) Schematic
drawing of orthogonal views of a common atrioventricular valve:
shortaxis view from below (left), long-axis (top right), 4-chamber
(bottom right). (B) Valve view showing a complete AVSD in a patient
with right atrial isomerism and double outlet RV. Valve leaflets: SB =
superior bridging leaflet, RAS = right anterosuperior leaflet, RI =
right inferior (mural) leaflet, IB = inferior bridging leaflet, LM = left
mural leaflet. (C) b-SSFP CMR image showing 4-chamber view of a
balanced complete AVSD. There are large atrial and ventricular
components. Note the VSD (arrow) and moderate left AV valve
regurgitation (arrowhead).
8. • FIGURE 20-6 ■ Ventricular septal defects. (A)
Schematic drawing of VSD positions viewed
from the right ventricular aspect. (B) b-SSFP
CMR image of a VSD (arrow) with overriding
aorta in a patient with tetralogy of Fallot. (C)
Coronal oblique view following correction
with VSD patch (arrowhead).
9. • FIGURE 20-7 ■ Severe coarctation of the aorta. (A) PA CXR showing characteristic
bilateral rib-notching (arrow), secondary to the development of collateral
circulation. (B) Black-blood, spin-echo, oblique sagittal image through the aorta
showing a tight discrete coarctation (arrow). (C) Volume-rendered 3D
reconstruction of MR angiography showing a tight coarctation (arrowhead), and
multiple enlarged collateral vessels. (D) Echocardiographic continuous-wave
Doppler profile of the coarctation region, demonstrating increased velocity across
the stenosis, 4.18 m/s (blue cross), corresponding to a pressure gradient of 70
mmHg from the simplified Bernoulli equation. There is also markedly increased
diastolic velocity, characteristic in coarctation, termed ‘diastolic tail’ (red star
10. • FIGURE 20-7 ■ Severe coarctation of the aorta. (A) PA CXR showing characteristic
bilateral rib-notching (arrow), secondary to the development of collateral
circulation. (B) Black-blood, spin-echo, oblique sagittal image through the aorta
showing a tight discrete coarctation (arrow). (C) Volume-rendered 3D
reconstruction of MR angiography showing a tight coarctation (arrowhead), and
multiple enlarged collateral vessels. (D) Echocardiographic continuous-wave
Doppler profile of the coarctation region, demonstrating increased velocity across
the stenosis, 4.18 m/s (blue cross), corresponding to a pressure gradient of 70
mmHg from the simplified Bernoulli equation. There is also markedly increased
diastolic velocity, characteristic in coarctation, termed ‘diastolic tail’ (red star
11. • FIGURE 20-8 ■ Coronary artery anomalies. Schematic diagram of
the coronary arteries viewed in the axial oblique plane on CMR.
RA = right atrium, LA = left atrium, LV = left ventricle, RVOT = right
ventricular outflow tract, LAD = left anterior descending artery, RCA
= right coronary artery, LCX = left circumflex artery. (A) Anomalous
LCX from RCA. (B) Anomalous RCA from left main stem (LMS), with
interarterial course between pulmonary artery and aorta. (C)
Anomalous RCA from LMS passing posteriorly between the aorta
and atria. (D) Anomalous left coronary artery arising from RCA with
interarterial course between the pulmonary trunk and aorta. (E)
Anomalous left coronary artery arising from RCA passing anterior to
pulmonary trunk. (F) Anomalous left coronary artery arising from
RCA passing posteriorly between aorta and atria
12. • FIGURE 20-8 ■ Coronary artery anomalies. Schematic diagram of
the coronary arteries viewed in the axial oblique plane on CMR.
RA = right atrium, LA = left atrium, LV = left ventricle, RVOT = right
ventricular outflow tract, LAD = left anterior descending artery, RCA
= right coronary artery, LCX = left circumflex artery. (A) Anomalous
LCX from RCA. (B) Anomalous RCA from left main stem (LMS), with
interarterial course between pulmonary artery and aorta. (C)
Anomalous RCA from LMS passing posteriorly between the aorta
and atria. (D) Anomalous left coronary artery arising from RCA with
interarterial course between the pulmonary trunk and aorta. (E)
Anomalous left coronary artery arising from RCA passing anterior to
pulmonary trunk. (F) Anomalous left coronary artery arising from
RCA passing posteriorly between aorta and atria
13. • FIGURE 20-9 ■ Tetralogy of Fallot. (A, B) Right ventricular outflow
tract, morphological specimen and corresponding black-blood
spin-echo image in coronal view. The deviated outlet septum
(asterisk), aortic root (arrowhead) and hypertrophied septoparietal
trabeculations (arrow) are shown. (C) b-SSFP images of unrepaired
tetralogy of Fallot: inflow/outflow view of the left ventricle (LV)
shows a VSD with overriding aorta (Ao)—note the severe
hypertrophy of the right ventricle (RV). (D) Black-blood, spin-echo
image of right modified Blalock–Taussig shunt; 3.5-mm gortex tube
from innominate artery to right pulmonary artery (arrow).
14. • FIGURE 20-9 ■ Tetralogy of Fallot. (A, B) Right ventricular outflow
tract, morphological specimen and corresponding black-blood
spin-echo image in coronal view. The deviated outlet septum
(asterisk), aortic root (arrowhead) and hypertrophied septoparietal
trabeculations (arrow) are shown. (C) b-SSFP images of unrepaired
tetralogy of Fallot: inflow/outflow view of the left ventricle (LV)
shows a VSD with overriding aorta (Ao)—note the severe
hypertrophy of the right ventricle (RV). (D) Black-blood, spin-echo
image of right modified Blalock–Taussig shunt; 3.5-mm gortex tube
from innominate artery to right pulmonary artery (arrow).
15. • FIGURE 20-10 ■ Transposition of the great arteries. (A) b-SSFP CMR image
showing an oblique sagittal outlet view of the aorta arising from the right
ventricle (RV) and pulmonary artery arising posteriorly from the left ventricle (LV).
(B) Schematic drawing of the arterial switch repair of TGA, showing the Le Compte
manoeuvre with the translocation of the aorta and pulmonary artery. Note sites of
coronary artery ‘button’ removal and subsequent reimplantation into the neo-
aortic root. (C) b-SSFP CMR image showing the pulmonary arteries straddling the
aorta following the arterial switch procedure with Le Compte manoeuvre. (D)
Volume-rendered 3D reconstruction of a contrast-enhanced MRA showing bilateral
proximal branch pulmonary artery narrowing.
16. • FIGURE 20-10 ■ Transposition of the great arteries. (A) b-SSFP CMR
image showing an oblique sagittal outlet view of the aorta arising from
the right ventricle (RV) and pulmonary artery arising posteriorly from the
left ventricle (LV). (B) Schematic drawing of the arterial switch repair of
TGA, showing the Le Compte manoeuvre with the translocation of the
aorta and pulmonary artery. Note sites of coronary artery ‘button’ removal
and subsequent reimplantation into the neo-aortic root. (C) b-SSFP CMR
image showing the pulmonary arteries straddling the aorta following the
arterial switch procedure with Le Compte manoeuvre. (D) Volume-
rendered 3D reconstruction of a contrast-enhanced MRA showing bilateral
proximal branch pulmonary artery narrowing.
17. • FIGURE 20-11 ■ Congenitally corrected
transposition of the great arteries. (A) b-SSFP
CMR image of CCTGA showing the discordant
atrioventricular connection, with anterior LV.
Note the apical offset of the left-sided tricuspid
valve. (B) Schematic drawing of CCTGA and
frequent associated lesions.
18. • FIGURE 20-11 ■ Congenitally corrected transposition of the great
arteries. (A) b-SSFP CMR image of CCTGA showing the discordant
atrioventricular connection, with anterior LV. Note the apical offset
of the left-sided tricuspid valve. (B) Schematic drawing of CCTGA
and frequent associated lesions.
19. • FIGURE 20-12 ■ Total anomalous pulmonary venous drainage. (A)
PA CXR in a patient with unobstructed supracardiac TAPVD. Note
dilated ascending vein (arrow) returning all pulmonary blood to the
brachiocephalic vein. The arrowhead shows the dilated SVC. (B)
Volume-rendered 3D reconstruction of MR angiography showing
total anomalous infracardiac drainage of the pulmonary veins. Note
the narrowing of the veins as they pass through the diaphragm
(arrow) before draining into the portal vein (arrowhead).
20. • FIGURE 20-13 ■ Single ventricle. (A) b-SSFP CMR image showing
hypoplastic left heart syndrome, with severe hypertrophy of the systemic
RV. Note the large interatrial communication (arrowed), allowing mixing of
systemic and pulmonary venous return. (B) Volume-rendered 3D
reconstruction of an MR angiogram showing the Glenn, bidirectional
cavopulmonary anastomosis (arrow) and (C) a lateral tunnel total
cavopulmonary anastomosis (arrow) to the right pulmonary artery
(arrowhead). (D) b-SSFP CMR image showing severe ascites (arrow), and
right pleural effusion (arrowhead) in a patient with a failing TCPC
circulation and protein-losing enteropathy.
21. • FIGURE 20-13 ■ Single ventricle. (A) b-SSFP CMR image showing
hypoplastic left heart syndrome, with severe hypertrophy of the systemic
RV. Note the large interatrial communication (arrowed), allowing mixing of
systemic and pulmonary venous return. (B) Volume-rendered 3D
reconstruction of an MR angiogram showing the Glenn, bidirectional
cavopulmonary anastomosis (arrow) and (C) a lateral tunnel total
cavopulmonary anastomosis (arrow) to the right pulmonary artery
(arrowhead). (D) b-SSFP CMR image showing severe ascites (arrow), and
right pleural effusion (arrowhead) in a patient with a failing TCPC
circulation and protein-losing enteropathy.