• Acute
o Causes – neonates, children, adults
o Management
• Chronic
o Causes – neonates, children, elderly
Appendicitis p 765, p 770 PCCM 105
• Definition
• Incidence
• Causes
• Pathophysiology
• Assessment and common findings
• Management
• Preop care
• Essential health information
Haemorrhoids p 771, p 776 PCCM 124
• Definition
• Causes
• Pathophysiology
• Assessment and common findings p 771 / Clinical features PCCM 124
• Nursing management p 771 / PCCM p 125
• Surgical management
• Post op care
• Essential health information
• External
• Internal
Fissures. Abscesses, fistulas p 772, p 778 PCCM 125
• Definition
• Causes
• Pathophysiology
• Essential patient teaching
Abdominal trauma p 266 (T&E Periods)
• Causes
• Pathophysiology
• Assessment
• Diagnostic
• Common injuries
• Abdominal stabs PCCM p 272
o Clinical features
o Management
• Dangers
• Emergency management
Abdominal compartment syndrome
Jaundice p 785, p 790 PCCM p 115
• Pre hepatic / haemolytic
• Hepatic/ hepatocellular
• Post hepatic / obstructive
Toxic hepatitis p 789, p 794
• Pathophysiology
• Clinical manifestations
• Management
• Essential health information
Poisoning and drug overdose p 58, p 51 PCCM p13
Management (T&E Periods)
• Types of poison
o Paraffin
o Carbon monoxide
o Organophosphate poisoning
Acute pancreatitis p 804, p 809 PCCM p 117
• Definition
• Causes and incidence
• Assessment and common findings p 804, 810 / clinical features PCCM p 117
• Diagnostic tests
o Blood amylase /Urine amylase
• Nursing diagnosis
• Management p 806, 811 /
• PCCM p 117
• Pain relief- nursing care
• Prevention of complications – nursing care
Essential health information
Uterine fibroid (leiomyoma) and new treatment modalitiesMohammed Saadi
This presentation describes Uterine fibroid
Definition
Incidence
Etiology
Risk factors
Clinical manifestation
Red degeneration
Complications of fibroids
Management and the new modalities in treatment
Uterine fibroid (leiomyoma) and new treatment modalitiesMohammed Saadi
This presentation describes Uterine fibroid
Definition
Incidence
Etiology
Risk factors
Clinical manifestation
Red degeneration
Complications of fibroids
Management and the new modalities in treatment
This presentation focuses on common obstetrics emergencies. These include early pregnancy complications such as miscarriages and ectopic pregnancy. As well as abdominal pain. Other include haemorrhage, hypertensive state, and sepsis.
Obstetric fistula is an abnormal opening between the reproductive tract (usually the vagina) and the urinary tract (frequently the bladder) or alimentary tract (usually the rectum) or both. Obstetric fistula typically develops after several days of prolonged or obstructed labour.
For more notes: Join Us on Telegram: https://t.me/OBGYN_Note_Book Or Facebook: https://www.facebook.com/obgyn.books
Slideshare: https://www.slideshare.net/bjlomsecond
Formation of hard, pebble and stone like structure mainly made up of cholesterol in gall bladder is called cholelithiasis.
Know more about cholelithiasis
This presentation focuses on common obstetrics emergencies. These include early pregnancy complications such as miscarriages and ectopic pregnancy. As well as abdominal pain. Other include haemorrhage, hypertensive state, and sepsis.
Obstetric fistula is an abnormal opening between the reproductive tract (usually the vagina) and the urinary tract (frequently the bladder) or alimentary tract (usually the rectum) or both. Obstetric fistula typically develops after several days of prolonged or obstructed labour.
For more notes: Join Us on Telegram: https://t.me/OBGYN_Note_Book Or Facebook: https://www.facebook.com/obgyn.books
Slideshare: https://www.slideshare.net/bjlomsecond
Formation of hard, pebble and stone like structure mainly made up of cholesterol in gall bladder is called cholelithiasis.
Know more about cholelithiasis
Diverticulitis Surgery - Procedure and Recovery Southlake.pdfMeghaSingh194
Diverticulitis happens when small pockets in digestive tract, called as diverticula, get inflamed. Diverticula frequently become inflamed once they become infected. Let's explore more: https://www.southlakegeneralsurgery.com/diverticulitis-surgery-procedure-and-recovery-southlake/
Many people have small pouches in the lining of the colon, or large
intestine, that bulge outward through weak spots. Each pouch is called a
diverticulum. Multiple pouches are called diverticula.
Diverticula are most common in the lower portion of the large intestine,
called the sigmoid colon. When the pouches become inflamed, the
condition is called diverticulitis. Ten to 25 percent of people with
diverticulosis get diverticulitis.
Abortion and other Causes of Early Pregnancy Bleeding.pdfChantal Settley
Describe common causes of bleeding in early pregnancy.
Describe the clinical classifications of abortion, the legal aspects of abortion in Ethiopia, and the safe methods used in health facilities.
Identify the warning signs and the emergency treatment required before referral for early pregnancy bleeding.
Describe the features of woman-friendly comprehensive post-abortion care, including the post-abortion family planning service
List the advantages of regionalised perinatal care.
Describe the functioning of a perinatal-care clinic.
Communicate better with patients and colleagues.
Safely transfer a patient to hospital.
Determine the maternal mortality rate.
Medical problems during pregnancy, labour and the puerperium.pdfChantal Settley
Diagnose and manage cystitis.
Reduce the incidence of acute pyelonephritis in pregnancy.
Diagnose and manage acute pyelonephritis in pregnancy.
Diagnose and manage anaemia during pregnancy.
Identify patients who may possibly have heart valve disease.
Manage a patient with heart valve disease during labour and the puerperium.
Manage a patient with diabetes mellitus.
Explain the wider meaning of family planning.
Give contraceptive counselling.
List the efficiency, contraindications and side effects of the various contraceptive methods.
List the important health benefits of contraception.
Advise a postpartum patient on the most appropriate method of contraception.
Define the puerperium.
List the physical changes which occur during the puerperium.
Manage the normal puerperium.
Assess a patient at the 6-week postnatal visit.
Diagnose and manage the various causes of puerperal pyrexia.
Recognise the puerperal psychiatric disorders.
Diagnose and manage secondary postpartum haemorrhage.
Teach the patient the concept of ‘the mother as a monitor’.
Uterine contractions continue, although less frequently than in the second stage.
The uterus contracts and becomes smaller and, as a result, the placenta separates.
The placenta is squeezed out of the upper uterine segment into the lower uterine segment and vagina. The placenta is then delivered.
The contraction of the uterine muscle compresses the uterine blood vessels and this prevents bleeding. Thereafter, clotting (coagulation) takes place in the uterine blood vessels due to the normal clotting mechanism.
Identify the onset of the second stage of labour.
Decide when the patient should start to bear down.
Communicate effectively with the patient during labour.
Use the maternal effort to the best advantage when the patient bears down.
Make careful observations during the second stage of labour.
Assess the fetal condition during the time the patient bears down.
Accurately evaluate progress in the second stage of labour.
Manage a patient with a prolonged second stage of labour.
Diagnose and manage impacted shoulders.
Monitoring the condition of the fetus during the first stage of labour.pdfChantal Settley
Monitor the condition of the fetus during labour.
Record the findings on the partogram.
Understand the significance of the findings.
Understand the causes and signs of fetal distress.
Interpret the significance of different fetal heart rate patterns and meconium-stained liquor.
Manage any abnormalities which are detected.
1.1 Define and use correctly all of the key terms
1.2 Describe the signs of true labour and distinguish between true and false labour
1.3 Explain to the mother how to recognise the onset of true labour
1.4 Describe the characteristic features and mechanisms of the four stages of labour
1.5 Describe the seven cardinal movements made by the baby as it descends the birth canal in a normal labour
10.2 Preterm labour and preterm rupture of the membranes.pdfChantal Settley
Define preterm labour and preterm rupture of the membranes.
Understand why these conditions are very important.
Understand the role of infection in causing preterm labour and preterm rupture of the membranes.
List which patients are at increased risk of these conditions.
Understand what preventive measures should be taken.
Diagnose preterm labour and preterm rupture of the membranes.
Manage these conditions.
Understand why an antepartum haemorrhage should always be regarded as serious.
Provide the initial management of a patient presenting with an antepartum haemorrhage.
Understand that it is sometimes necessary to deliver the fetus as soon as possible, in order to save the life of the mother or infant.
Diagnose the cause of the bleeding from the history and examination of the patient.
Correctly manage each of the causes of antepartum haemorrhage.
Diagnose the cause of a blood-stained vaginal discharge and administer appropriate treatment.
Define hypertension in pregnancy.
Give a simple classification of the hypertensive disorders of pregnancy.
Diagnose pre-eclampsia and chronic hypertension.
Explain why the hypertensive disorders of pregnancy must always be regarded as serious.
List which patients are at risk of developing pre-eclampsia.
List the complications of pre-eclampsia.
Differentiate pre-eclampsia from pre-eclampsia with severe features.
Give a practical guide to the management of pre-eclampsia.
Provide emergency management for eclampsia.
Manage gestational hypertension and chronic hypertension during pregnancy.
7.2 New Microsoft PowerPoint Presentation (2).pdfChantal Settley
Welcome the woman and ask her to sit near you and facing you.
Smile and make good eye contact with her.
Reassure her that you will always maintain her privacy and confidentiality
Without her permission, do not include a third person in the meeting.
Use simple non-medical language and terminologies throughout that she can understand, and check frequently that she has really understood.
Actively listen to her, using gestures and verbal communication to show her that you are paying attention to what she says.
Encourage her to ask questions, express her needs and concerns, and seek clarification of any information that she does not understand.
6.4 Assessment of fetal growth and condition during pregnancy.pdfChantal Settley
When you have completed this unit you should be able to:
• Assess normal fetal growth.
• List the causes of intra-uterine growth restriction.
• Understand the importance of measuring the symphysis-fundus height.
• Understand the clinical significance of fetal movements.
• Use a fetal-movement chart.
• Manage a patient with decreased fetal movements.
• Understand the value of antenatal fetal heart rate monitoring.
What possible complications to look for:
Antepartum haemorrhage
Pre-eclampsia
proteinuria and a rise in the blood pressure.
Cervical changes
Symphysis-fundus height measurement
below the 10th centile?
above the 90th centile?
To review and act on the results of the screening or special investigations done at the booking visit.
2. To perform the second assessment for risk factors.
If possible, all the results of the screening tests should be obtained at the first visit.
Assess normal fetal growth.
List the causes of intra-uterine growth restriction.
Understand the importance of measuring the symphysis-fundus height.
Understand the clinical significance of fetal movements.
Use a fetal-movement chart.
Manage a patient with decreased fetal movements.
Understand the value of antenatal fetal heart rate monitoring.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
HOT NEW PRODUCT! BIG SALES FAST SHIPPING NOW FROM CHINA!! EU KU DB BK substit...GL Anaacs
Contact us if you are interested:
Email / Skype : kefaya1771@gmail.com
Threema: PXHY5PDH
New BATCH Ku !!! MUCH IN DEMAND FAST SALE EVERY BATCH HAPPY GOOD EFFECT BIG BATCH !
Contact me on Threema or skype to start big business!!
Hot-sale products:
NEW HOT EUTYLONE WHITE CRYSTAL!!
5cl-adba precursor (semi finished )
5cl-adba raw materials
ADBB precursor (semi finished )
ADBB raw materials
APVP powder
5fadb/4f-adb
Jwh018 / Jwh210
Eutylone crystal
Protonitazene (hydrochloride) CAS: 119276-01-6
Flubrotizolam CAS: 57801-95-3
Metonitazene CAS: 14680-51-4
Payment terms: Western Union,MoneyGram,Bitcoin or USDT.
Deliver Time: Usually 7-15days
Shipping method: FedEx, TNT, DHL,UPS etc.Our deliveries are 100% safe, fast, reliable and discreet.
Samples will be sent for your evaluation!If you are interested in, please contact me, let's talk details.
We specializes in exporting high quality Research chemical, medical intermediate, Pharmaceutical chemicals and so on. Products are exported to USA, Canada, France, Korea, Japan,Russia, Southeast Asia and other countries.
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
Follow us on: Pinterest
Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
2. Acute Constipation
The onset of acute constipation is sudden and it lasts
for several days.
It’s usually caused by medication, blockage,
dehydration, prolonged activity, or missing a bowel
movement.
With women who are pregnant, it can develop when
the womb is pressed against the intestine.
Swallowing indigestible objects and lead poisoning
are also a cause sometimes, as well as general
anaesthesia, which may affect the bowel muscles a
couple days after surgery.
2018/10/02Compiled by C Settley
2
3. Acute Constipation
The medications below may slow down the faeces passage
through the intestine and provoke acute constipation:
Epilepsy anticonvulsants
Diuretics
Antidepressants
Iron supplements
Calcium-channel blockers, and other heart medications
Morphine, codeine, and other pain medications
Some antacids
Some cold and cough medications that contain dextromethorphan
2018/10/02Compiled by C Settley
3
4. Chronic constipation
Chronic constipation can be defined as the instance of two
or more of the specified below inside an eight week period:
Three bowel movements each week.
One incident of faecal incontinence each week.
Big stools in the rectum or apparent upon examination of
the abdomen.
Withholding behaviour.
Defecation that is painful.
2018/10/02Compiled by C Settley
4
5. Chronic constipation
Normal constipation lasts for a brief period and its symptoms resolve in a
fairly short time.
With chronic constipation, however, the symptoms last for a longer
duration, usually around three or more months and in some cases, years.
It is a long-term condition that can impact a person’s social and work life.
It is generally not relieved with OTC laxatives or lifestyle changes and could
require medical attention.
Although the occasional onset of constipation is common, in some cases,
people will experience chronic constipation, which interferes with their
ability to do daily tasks.
2018/10/02Compiled by C Settley
5
6. Chronic constipation
Chronic constipation treatment will depend on the underlying cause.
The treatment typically starts with lifestyle and diet changes which are
meant for increasing the speed of the stool moving through the intestines.
When simple measures like this fail, laxative tablets are often the next step.
For acute constipation, stimulant laxatives like Dulcolax® tablets are often
used.
Other treatments for acute constipation may also include suppositories &
enemas.
2018/10/02Compiled by C Settley
6
7. Appendicitis
A condition in which the appendix becomes inflamed and filled with pus,
causing pain.
11-30 years more prone to developing
Males are more common affected
2018/10/02Compiled by C Settley
7
8. The appendix
Normally, the appendix sits in the lower right abdomen.
The function of the appendix is unknown.
One theory is that the appendix acts as a storehouse for good
bacteria, “rebooting” the digestive system after diarrheal illnesses.
2018/10/02Compiled by C Settley
8
9. Appendicitis: Causes
A blockage in the lining of the appendix that results in infection is the likely
cause of appendicitis.
The bacteria multiply rapidly, causing the appendix to become inflamed,
swollen and filled with pus.
If not treated promptly, the appendix can rupture.
2018/10/02Compiled by C Settley
9
10. Faecolith: It is a hardening of faeces into lumps of varying size and may occur anywhere in the
intestinal tract but is typically found in the colon. It is also called appendicolith when it occurs in the
appendix and is sometimes concomitant with appendicitis.
Lymphoid hyperplasia is an increase in the number of normal cells (called lymphocytes) that are
contained in lymph nodes.
2018/10/02Compiled by C Settley
10
11. Appendicitis: Pathophysiology
Obstruction of the appendix results in an inflammatory reaction
Leads to oedema and distention
Pressure is applied on the intramural blood vessels
This leads to vascular engorgement (swelling of tissue)
That results in gangrene of the appendix
In addition, the mucosal wall ulcerates, becomes infected and
ruptures
This spreads infection to the peritoneum, hence the associated
peritonitis or abscess formation
2018/10/02Compiled by C Settley
11
13. Assessment
Diagnostic tests
FBC: elevated white cell count above 11 000mm3. neutrophil
count above 75%.
X-rays: Dilated loops of bowel indicating paralytic ileus, air or
fluid levels in case of obstruction and free air consistent with
perforation.
2018/10/02Compiled by C Settley
13
14. Appendicitis: Medical Management
Immediate surgical removal of the appendix through a laparotomy
Drainage of the abscess may be indicated if the complication has
developed
IV therapy should commence surgery to correct fluid and
electrolyte imbalance and to administer AB’s for infection
If appendix has not perforated at the time of the surgery, the
patient’s recovery is usually smooth and the patient is discharged
within 5-7 days or earlier
2018/10/02Compiled by C Settley
14
15. Appendicitis: Nursing Management
Relieve anxiety
Relieve pain
Prevent systematic infection
Correct fluid and electrolyte imbalance
Nutrition
Prepare patient for surgery
2018/10/02Compiled by C Settley
15
17. Haemorrhoids
Swollen and inflamed veins in the rectum and anus
that cause discomfort and bleeding.
2018/10/02Compiled by C Settley
17
18. Haemorrhoids: Symptoms
Painless bleeding during bowel movements —small
amounts of bright red blood on toilet tissue or in the toilet
Itching or irritation in anal region
Pain or discomfort
Swelling around anus
A lump near anus, which may be sensitive or painful (may
be a thrombosed hemorrhoid)
Hemorrhoid symptoms usually depend on the location.
2018/10/02Compiled by C Settley
18
19. Haemorrhoids
Internal hemorrhoids.
These lie inside the rectum. Occasionally, straining can push an internal
hemorrhoid through the anal opening. This is known as a protruding or
prolapsed hemorrhoid and can cause pain and irritation.
External hemorrhoids.
These are under the skin around anus. When irritated, external hemorrhoids can
itch or bleed.
Thrombosed hemorrhoids.
Sometimes blood may pool in an external hemorrhoid and form a clot
(thrombus) that can result in severe pain, swelling, inflammation and a hard
lump near the anus.
2018/10/02Compiled by C Settley
19
20. A venous plexus is a congregation of multiple veins.
2018/10/02Compiled by C Settley
20
21. Haemorrhoids: Causes
Straining during bowel movements
Sitting for long periods of time on the toilet
Chronic diarrhea or constipation
Obesity
Pregnancy
Anal intercourse
Low-fiber diet
Rectal surgery
Loss of muscle tone
Anal intercourse
Heavy lifting
2018/10/02Compiled by C Settley
21
22. Haemorrhoids: Pathophysiology
The covering epithelium is damaged by the hard bowel
movement, and the underlying veins bleed.
With spasm of the sphincter complex elevating pressure, the
internal hemorrhoidal veins can spurt.
Internal hemorrhoids can deposit mucus onto the perianal tissue
with prolapse.
2018/10/02Compiled by C Settley
22
25. Haemorrhoids: Complications
Anemia. Rarely, chronic blood loss from hemorrhoids may cause
anemia, in which you don't have enough healthy red blood cells
to carry oxygen to your cells.
Strangulated hemorrhoid. If the blood supply to an internal
hemorrhoid is cut off, the hemorrhoid may be "strangulated,"
another cause of extreme pain.
2018/10/02Compiled by C Settley
25
26. A: Strangulated internal hemorrhoid;
B: Acutely thrombosed external hemorrhoid.
2018/10/02Compiled by C Settley
26
27. Haemorrhoids: Nursing management
Preventing constipation by eating a high fibre diet
Avoiding prolonged periods of standing/sitting
Warm sitz baths
Inserting soothing anal suppositories
Warm compresses
Anal hygiene
exercise
2018/10/02Compiled by C Settley
27
28. Haemorrhoids: Surgical management
A hemorrhoidectomy.
Indications:
Thrombosis
Prolonged bleeding
Complicated prolapses
Intolerable itching
Discomfort
Intense pain
Post op care:
Support
Pillow
Pain meds
Stool softener
2018/10/02Compiled by C Settley
28
29. Fissures, abscesses & fistulas:
Common problems developed from trauma or infection in the
anorectal area
Fissure: A small tear in the lining of the anus.
Anal fissure may occur when passing hard or large stools.
An anal fissure can cause pain and bleeding during bowel movements.
This condition usually heals on its own in four to six weeks. Common
treatments include dietary fibre and stool softeners, as well as creams to
the affected area.
2018/10/02Compiled by C Settley
29
30. Fissures, abscesses & fistulas:
Common problems developed from trauma or infection in the
anorectal area
Anal abscess: An abscess causes tenderness, swelling, and pain. These
symptoms clear when the abscess is drained. The patient may also
complain of fever, chills, and general weakness or fatigue.
2018/10/02Compiled by C Settley
30
31. Fissures, abscesses & fistulas:
Common problems developed from trauma or infection in the
anorectal area
Anal fistula: An infected tunnel between the skin and the anus.
An anal fistula is an infected tunnel between the skin and the anus, the
muscular opening at the end of the digestive tract. Most anal fistulas are
the result of an infection in an anal gland that spreads to the skin.
Symptoms include pain, swelling and discharge of blood or pus from the
anus.
Surgery is usually required to treat anal fistula.
2018/10/02Compiled by C Settley
31
33. Fissures, abscesses & fistulas:
Summary
Bleeding, pain, or drainage from the anus can occur
with several illnesses, so a physician should always be
consulted.
Often the diagnosis is anal fissure, abscess, or fistula.
These are problems that are usually easy to diagnose
and correct.
A variety of treatments, including surgery, are
available to correct these conditions.
2018/10/02Compiled by C Settley
33
34. Abdominal trauma
Abdominal trauma is an injury to the abdomen.
Signs and symptoms include abdominal pain,
tenderness, rigidity, and bruising of the external
abdomen.
Complications may include blood loss and infection.
Diagnosis may involve ultrasonography, computed
tomography, and peritoneal lavage, and treatment
may involve surgery.
2018/10/02Compiled by C Settley
34
35. Abdominal trauma: Common injuries
Lacerated liver
Injuries to kidney and intestines
Ruptured spleen, liver, stomach, diaphragm & bladder
Dangers:
Peritonitis
Inflammation of the membrane lining the abdominal wall and covering the
abdominal organs.
Shock
Cullen’s sign
superficial oedema and bruising in the subcutaneous fatty tissue around the umbilicus
Abdominal compartment syndrome
A condition caused by abnormally increased pressure within the abdomen.
Gunshots, poly-trauma, stab wounds, haemorrhage
2018/10/02Compiled by C Settley
35
36. Abdominal trauma:
Emergency management
Laparotomy
Clear airway
High Fowlers
Oxygen
Control bleeding
Replace fluid loss
Blood transfusion
Nasogastric tube to decompress the abdomen and stomach
Catheter
Vital signs
2018/10/02Compiled by C Settley
36
37. Jaundice
Jaundice is caused by a build-up of bilirubin, a waste material, in the blood.
Old red blood cells travel to the liver, where they're broken down.
Bilirubin is the yellow pigment formed by the breakdown of these old cells.
Jaundice occurs when your liver doesn't metabolize bilirubin the way it's
supposed to.
An inflamed liver or obstructed bile duct can lead to jaundice, as well as
other underlying conditions.
Symptoms include a yellow colour to the skin and whites of the eyes, dark
urine, and itchiness.
2018/10/02Compiled by C Settley
37
38. Types of Jaundice
Pre hepatic/Haemolytic
Haemolytic jaundice occurs as a result of haemolysis,
or an accelerated breakdown of red blood cells,
leading to an increase in production of bilirubin.
Hepatic/Hepatocellular
Hepatocellular jaundice occurs as a result of liver
disease or injury.
Post hepatic/Obstructive
Obstructive jaundice occurs as a result of an
obstruction in the bile duct.
2018/10/02Compiled by C Settley
38
39. Toxic hepatitis
Toxic hepatitis is an inflammation of the liver in reaction to certain
substances exposed to.
Toxic hepatitis can be caused by alcohol, chemicals, drugs or nutritional
supplements.
In some cases, toxic hepatitis develops within hours or days of exposure to a
toxin.
2018/10/02Compiled by C Settley
39
40. Acute pancreatitis
The pancreas is an organ in the upper abdomen that produces digestive fluids
and the hormone insulin.
Acute pancreatitis means inflammation of the pancreas that develops quickly.
The main symptom is abdominal pain.
It usually settles in a few days but sometimes it becomes severe and very serious.
The most common causes of acute pancreatitis are gallstones and drinking a lot
of alcohol.
2018/10/02Compiled by C Settley
40
41. Acute pancreatitis: Causes
Gallstones
Heavy alcohol use
Drugs such as angiotensin-converting
enzyme (ACE) inhibitors, azathioprine,
furosemide, 6-mercaptopurine,
pentamidine, sulfa drugs, and valproate
Estrogen use in women with high levels
of lipids in the blood
High levels of calcium in the blood
(which may be caused by
hyperparathyroidism)
Viruses such as mumps
High levels of triglycerides in the blood
(hypertriglyceridemia)
Damage to the pancreas caused by
surgery or endoscopy (such as
endoscopic retrograde
cholangiopancreatography [ERCP])
Damage to the pancreas caused by
blunt or penetrating injuries
Cancer of the pancreas, or other
blockages of the pancreatic duct
Hereditary pancreatitis, including a small
percentage of people with cystic fibrosis
or cystic fibrosis genes
Cigarette smoking
Kidney transplantation
Pregnancy (rare)
2018/10/02Compiled by C Settley
41
42. Acute pancreas: Symptoms
Abdominal pain
Coughing, vigorous movement, and deep breathing may worsen the pain. Sitting
upright and leaning forward may provide some relief.
Feeling of wanting to vomit.
Some people, especially those who develop acute pancreatitis because of
heavy alcohol use, may never develop any symptoms other than moderate to
severe pain.
Some people feel terrible. They look sick and are sweaty and have a fast pulse
(100 to 140 beats a minute) and shallow, rapid breathing.
At first, body temperature may be normal, but it may increase in a few hours to
between 100° F and 101° F (37.7° C and 38.3° C).
Blood pressure is usually low and tends to fall when the person stands, causing
light-headedness.
Occasionally, the whites of the eyes (sclera) become yellowish.
2018/10/02Compiled by C Settley
42
43. Poisoning and drug overdose:
Management
Identify the poison
Place in recovery position
Establish or maintain open airway
Assess for signs of breathing
Check pulse and skin colour
Call for help
Transport to hospital
2018/10/02Compiled by C Settley
43