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By- Dr. Armaan SinghBy- Dr. Armaan Singh
Clinical Anatomy of the Back
PROGRESS
TimeTime
Goh et al. Clin Biomech 1999;14:439
SPINE
Consists of
• Cervical Vertebrae
• Thoracic Vertebrae
• Lumbar Vertebrae
• Sacrum
• The strength of the skeletal column is due
to the size and shape of the vertebrae
• Its flexibility is due to the many joints that
are close together
Spine
VERTEBRAL COLUMN
• Lot of stress in variety of sports
• Cervical pathology
• Pain may be referred to upper limb
• Lumber pathology
• Lower limb
YOUNG SPINE
Normal curvature of
infant’s spine
Normal lumbar curve
of toddler’s spine
LOW BACK PAIN IN SPORTS
• 70% of population will suffer from back pain
at some time
• 10% - 15% of sports injuries are spinal
injuries
• 0.6% - 1% have neurological complications
Deyo & Tsui-Wu. Spine 1987;12:264-8
• Majority of sports injuries
to lumbar spine
• Soft tissue and many are
not reported
• Fractures
• Fracture dislocation
• Abrasions, bruising
• Contusions
Tall & De Vault. Clin Sports Med 1993;12:441-8
Low Back Pain in Sports
• Must know the sport
• Must understand the biomechanics and
stresses involved in the sport
• Must examine the spine
in the appropriate position
Low Back Pain in Sports
TYPICAL VERTEBRAE
• Basic parts
• Body and neural arch
• Which consists of
pedicles, lamina and spine
• The transverse processes arise from the
pedicles
• Superior and inferior
articular processes
LUMBAR VERTEBRAE
LUMBAR VERTEBRAE
• Body kidney shaped
• No articular facets for ribs
• Inferior facets face anterolateral
• Superior facets face posteromedial
• Intervertebral notch increase in size
• Accessory processes base
of transverse process
• Mammillary process on posterior aspect of
superior articular process
LUMBAR VERTEBRAE
• Body is convex anteriorly
• Foramina on the posterior aspect are
for the basic vertebral veins, which
drain into the internal vertebral plexus
• The walls of the veins, which are valve
less, have afferent nerve fibers
• Secondaries can spread from pelvis,
prostate, adrenal glands lungs and
breast
• The superior and inferior surfaces of
the body are flat and covered by a thin
layer of hyaline cartilage
• The body of the vertebra consists of
trabecular or cancellous bone
Lumbar Vertebrae
TYPICAL LUMBAR VERTEBRAE
• Superior and inferior articular
processes
• Arise from the junction of the pedicles
and lamina
• Superior face posteromedially
• With rough mammillary processes on
the posterior border
• Inferior face anterolaterally
• Accessory processes at the base of
transverse process
• Prevents rotation
THE LUMBAR FACETS
• Vary from the sagittal disposition at the first
and second, to almost coronal in the lower
• Facet tropism is when the facet on one side
is in the sagittal plane and the other is in the
coronal plane, which adds to rotational
stress
• This change may occur in the lower thoracic
vertebrae
PARS INTERARTICULARIS
• Pars interarticularis
• Portion of lamina between superior
and inferior articular processes
• Site of spondylolysis or
spondylolisthesis
LUMBAR SPINE
• Cancellous bone
• 50% compressive strength
• Facet joints 20% in standing
upright position
LUMBAR VERTEBRAE
LUMBAR VERTEBRAE
LUMBAR SPINE
• Cancellous bone
• 50% of the compressive
strength
• Facet joints, 20% of the strength in
the standing upright position
ANTERIOR LONGITUDINAL LIGAMENT
• Attached mainly to the bodies
• This ligament helps to prevent us from leaning too far
back (hyperextension)
POSTERIOR LONGITUDINAL LIGAMENT
• Attached mainly to the inter
vertebral discs
• This ligament helps to restrict
forward bending (hyperflexion)
LIGAMENTUM FLAVA
• Runs between the laminae
of the neural arches
• Helps to restrict hyperflexion
• It extends to the capsule
of the facet joint
• It is highly elastic and ensures that the
ligament does not buckle in extension
• Gives elasticity to the posterior aspect of
the facet joints
• Helps form the posterior boundary of the
intervertebral foramen
• The ligamentum flava is thicker in the
lumbar region
Ligamentum Flava
SPINAL LIGAMENTS
• Interspinous ligaments
• Strong supraspinous ligaments
• The inter-transverse ligaments join the
transverse processes and are thin and
membranous in the lumbar region
FIFTH LUMBAR VERTEBRAE
• Larger, superior and inferior articular
facets in the same plane
• Fifth lumbar vertebrae has large
transverse processes
• Arise from the body as well as the
pedicles
ARTHRITIS OF SPINE
• Painful
• Limitation of movement
• Extra projections
• Narrowing of disc spaces
VERTEBRAL JOINTS
• Secondary cartilaginous joints between
the bodies
• Hyaline cartilage covering bodies
• Disc of fibrocartilage in between
• Synovial plane joints between the facets
INTERVERTEBRAL DISCS
• Annulus fibrosis
• Concentric lamina run obliquely
• Type I collagen at periphery, type II near
nucleus
• Weakest portion is the postero-lateral
and posterior
• Periphery has a nerve supply
NUCLEUS PULPOSUS
• Gelatinous, hydrophilic, proteoglycan gel in
collagen matrix
• Lies posterior in the disc
• There are no nerve endings in a mature disc
• Nerve endings are found in the posterior longitudinal ligament and the dura
• Nutrition of the disc is by diffusion via the central 40% of the cartilaginous end
plate
• The discs are thicker in the cervical and lumbar sections of the vertebral
column
• Where there is more movement. The largest disc is between L5 S1
NUCLEUS PULPOSUS
• Hydration of the annulus and nucleus is
proportional to the applied compressional
stress
• In vivo, there is a loss of 1 cm standing
height over the course of the day
• A disc loaded in vitro for four hours by
100% body weight will lose 6% of the
fluid from the nucleus and 13% from the
annulus
• May be due to end plate fracture
• There is more rotational stress in the posterior part of the disc
NUCLEUS PULPOSUS
• The position of the spine determines
where the compressional forces are
greatest
• The posterior longitudinal ligament is
thin and expanded at the level of the
disc
• High compressional loading at
L4,L5,S1 may be due to end plate
fracture and not to rupture of the annulus
• End plate failure is a possible precursor of disc degeneration
AXIAL LOAD AND END-PLATES
END-PLATE MECHANICS
• Functionally, the vertebral end-plate
displays characteristics of a trampoline
• With the sub-end-plate trabecular
bone acting as springs to sustain
and dissipate axial load
• Despite the thinness of the vertebral
end-plate
• The hydraulic nature of marrow and
blood vessels within the vertebral body,
act to dampen axial loads, unless the
local point pressure is too high
END-PLATE MECHANICS
• End-plate lesions can be induced experimentally
before a disc will prolapse through the anulus,
suggesting a protective mechanism over annular
injury and potentially cord or root compression
• Excessive loads may result in perforation of the
end-plate, usually in the region of the nucleus and
often in the path of the developmental notchord
END-PLATE SUSCEPTIBILITY
Schmorl & Junghanns. The human spine in health and disease.
New York: Grune & Stratton, 1965
Notochord
FACET JOINTS
• L1,L2 Facets sagittal plane
• Lower joints in coronal plane
• Synovial plane joints
• Meniscoid structures
• Synovial membrane some contain
fat
• Supplied by medial branch of
dorsal ramus
• Narrowing of disc space, results in
stress on
facet joint
• Highest pressure during
• Combined
• Extension
• Rotation
• Compression
Facet Joints
FACET JOINT SYNDROME
• Extension and rotation
• Pain rising from flexion
• Pain worse standing
• Lateral shift in extension
• Point tenderness over
facet
• Referred leg pain
SEGMENTAL ROTATION
Singer et al. J Musculoskel Res 2001;5: 45-55
• Flexion limited by
disc problems
• Lateral flexion
• Extension limited by
facet joint problems
• Very little rotation
• Extension and rotation
affect facet joints
MOVEMENTS OF LUMBAR SPINE
NERVE SUPPLY
• Nerve supply
• Peripheral annulus
• Facet joint
• Nerve is medial branch dorsal
ramus
BLOOD SUPPLY
• Lumbar arteries
• Internal venous plexuses
• External venous plexuses
• Basivertebral veins
• Valveless
LUMBAR VERTEBRAE
CANCELLOUS BONE
• Cancellous bone
• 50% compressive strength
• Facet joints 20% in standing
upright position
Normal bone Osteoporotic bone
ANATOMICAL ABNORMALITIES
• Spina Bifida Occulta
• Facet Tropism
• Kyphosis
• Scoliosis
ANATOMICAL ABNORMALITIES
Kyphosis Scoliosis
ANATOMICAL ABNORMALITIES
• Hemi-vertebra
• Spina Bifida Occulta
• Facet Tropism
• Scoliosis
• Kyphosis
ANATOMICAL ABNORMALITIES
• Unilateral lumbarisation
• Unilateral sacralisation
THE SPINE IN SPORTS
• Spine injury epidemiology
• Contact vs. non-contact sports
• Spine injury mechanisms
• Overuse – overload – overlooked
• Vertebral end-plate injury
• Disc injury
• Future issues
EPIDEMIOLOGY
Cooke & Lutz. Phys Med Rehab Clinics N Am 2000;11:837
Cooke & Lutz. Phys Med Rehab Clinics N Am 2000;11:837-65
EPIDEMIOLOGY
• Back pain in the community is 60% - 80%
• Recurrence of back pain is
70% - 90%
• Progression to chronic back pain is 5% -
10%
LOW BACK PAIN IN SPORTS
• Majority of sports injuries are to the
lumbar spine
• Many soft tissue injuries are not
reported
• Fractures
• Fracture dislocation
• Abrasions, bruising
• Contusions
Tall & De Vault. Clin Sports Med 1993;12:441-8
CHRONIC LOW BACK PAIN
• Local structures
• Muscles
• Ligaments
• Poor lifting techniques
• Joints
• Bones
BACK PAIN
Local structures
• Muscles, ligaments
• Joints
Referred pain
• Abdominal organs
• Pelvic organs
Must out rule
• Infection
• Tumours
ACUTE LOW BACK PAIN
• Non-specific low back pain
• Usually settles quickly
• History
• Examination
• Pain relief
• Stay as active as possible within limit
of pain
ACUTE LOW BACK PAIN
• Nerve root pain
• Leg pain worse than back pain
• Numbness and pins and needles
• Neurological signs
• Refer to specialist
• If it does not resolve in
first 4 weeks
INVESTIGATE LOW BACK PAIN
• Under 20 or over 55 years
• Non-mechanical pain
• Past history cancer
• Thoracic pain
• Steroids or HIV
• Unwell, weight loss
• Widespread neurology
• Structural deformity
• Gait disturbance or sphincter disturbance
CHRONIC LOW BACK PAIN
Pain referred
• Abdominal organs
• Pelvic organs
Must out rule
• Infection
• Tumours
PAIN REFERRED
YOUNG ATHLETE
• Junior rugby team 15 years of age
• M. Scheuermann
• 5 Spina bifida occulta
• The scrum half had degenerative
facet joint changes
SACROILIAC JOINT – SCIATIC NERVE
SPINAL STENOSIS
• Congenital or acquired
• Abnormally short pedicles or lamina
• Formation of osteophytes
• Osteo-arthritis of facet joints
• Pain aggravated by walking
• Relieved by rest
SPINAL STENOSIS
PREDISPOSING FACTORS
• Intrinsic factors
• Anatomical abnormalities
• Biomechanical
• Extrinsic factors
• Sport
• Surfaces
• Equipment
• Training
PREDISPOSING FACTORS BACK PAIN
• Poor posture
• Overweight
• Unfit
PREDISPOSING FACTORS
• Poor core stability
• Weak abdominal muscles
• Weak gluteal muscles
• Muscle imbalance
PREDISPOSING FACTORS
• Poor core stability
• Weak abdominal muscles
• Weak gluteal muscles
• Muscle imbalance
• Pronated or cavus feet
PREDISPOSING FACTORS
• Badly designed furniture
• No back support
• Poor posture at work
ACUTE LOW BACK PAIN
ANNULAR TEARS
• Loaded compression with rotatory
component
• As little as 3 degrees of
high torque rotation
• Facets protect disc
• As annulus fails, facets
joints may be injured
ANNULAR BULGE
DISC LESION
YOUNG ATHLETE
• Junior rugby team 15 years of age
• M. Scheuermann
• 5 Spina bifida occulta
• The scrum half had degenerative
facet joint changes
SCHEUERMANN’S DISEASE
Greene et al. J Pediatr Orthop 1985;5:1
SPONDYLOLISTHESIS
PARS INTERARTICULARIS
• Pars interarticularis, portion of lamina
between superior and inferior articular
processes
• Site of spondylolysis or spondylolisthesis
SPONDYLOLISTHESIS
SPONDYLOLYSIS AND SPONDYLOLISTHESIS
PARS INTERARTICULARIS; FACET JOINT
SPONDYLOLISTHESIS
RAPID FLEXION AND EXTENSION
• Gymnastics, flips
• Vaulting
• Ballet, arabesque
• Lifting during dance
• Diving
• Butterfly swimming
• Decathlon
• Pole vaulting
ANKYLOSING SPONDYLITIS, INFECTION
465 ATHLETES LOW BACK PAIN
(M318;F147)
male (39) female(14)
Spina Bifida Occulta (SBO)
6.6%(21) 4.1%(6)
Lumbarisation
3.5%(11) 1.4%(2)
Sacralisation
2.2% (7) 6.1% (9)
Spondylolisthesis (13)
30% had SBO; 21 of 56 had other pathology
MECHANISM OF INJURIES
• Compression or weight loading
• Torque or rotation
• Tensile stresses produced by excessive
motion of spine
• Hyperextension and flexion
Watkins & Dillin, 1985
COMPRESSION OR WEIGHT LOADING
• Sports requiring
• Massive strength
• High body weight
• Weight lifter
• Hooker and No 8
• Wrestling
• Line back American football
Watkins & Dillin, 1985
WEIGHT LIFTING
• 40 % weight lifters have
low back pain
• Greatest stress is when weight is lifted
above the head
• Dangerous time is shift from spinal flexion to
extension
Aggrawal et al. Br J Sports Med 1979;13:58-61
AXIAL COMPRESSIVE LOADING
• Head on collisions
• Motor sports
• Boating accidents
• Wrestling
• Horseback riding
• Bicycling
• Bobsleigh
AXIAL COMPRESSIVE LOADING
AXIAL COMPRESSIVE LOADING
AXIAL COMPRESSIVE LOADING
COMPRESSION STRESS
ROTATIONAL STRESS
ROTATIONAL STRESS
SPONDYLOLISTHESIS
RAPID FLEXION AND EXTENSION
• Gymnastics, flips
• Vaulting
• Ballet, arabesque
• Lifting during dance
• Diving
• Butterfly swimming
• Decathlon
• Pole vaulting
AUSTRALIAN FOOTBALL LEAGUE
Seward & Orchard. 2000 AFL Injury Report, Australian Sports Commission
GOLF
• Highest incidence of back injuries in
professional sports
• Torsional stress is lessened by spreading the
stress over the entire spine
• Rigid abdominal control
• Parallel shoulders and pelvis
Watkins and Dillin, 1985
SUSTAINED POSTURES -
HYPEREXTENSION
SUSTAINED POSTURES -
HYPEREXTENSION
SUSTAINED POSTURES -
HYPEREXTENSION
SUSTAINED POSTURES - FLEXION
SCOLIOSIS DUE TO UNILATERAL SPORTS
• Racquet sports
• Fencing
• Sweep rowing
• Javelin
• Freestyle unilateral breathing
SCOLIOSIS DUE TO UNILATERAL SPORTS
RUNNING
• Poor posture
• Poor abdominal
• Pronated feet
• Muscle imbalance
• Leg length discrepancy
• Osteoporosis
CRICKET
• Bowlers
• Rotational forces
• Extension followed by rotation
and flexion

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Clinical anatomy of_the_back by- dr. armaan singh

  • 1. By- Dr. Armaan SinghBy- Dr. Armaan Singh Clinical Anatomy of the Back
  • 2. PROGRESS TimeTime Goh et al. Clin Biomech 1999;14:439
  • 3. SPINE Consists of • Cervical Vertebrae • Thoracic Vertebrae • Lumbar Vertebrae • Sacrum
  • 4. • The strength of the skeletal column is due to the size and shape of the vertebrae • Its flexibility is due to the many joints that are close together Spine
  • 5. VERTEBRAL COLUMN • Lot of stress in variety of sports • Cervical pathology • Pain may be referred to upper limb • Lumber pathology • Lower limb
  • 6. YOUNG SPINE Normal curvature of infant’s spine Normal lumbar curve of toddler’s spine
  • 7. LOW BACK PAIN IN SPORTS • 70% of population will suffer from back pain at some time • 10% - 15% of sports injuries are spinal injuries • 0.6% - 1% have neurological complications Deyo & Tsui-Wu. Spine 1987;12:264-8
  • 8. • Majority of sports injuries to lumbar spine • Soft tissue and many are not reported • Fractures • Fracture dislocation • Abrasions, bruising • Contusions Tall & De Vault. Clin Sports Med 1993;12:441-8 Low Back Pain in Sports
  • 9. • Must know the sport • Must understand the biomechanics and stresses involved in the sport • Must examine the spine in the appropriate position Low Back Pain in Sports
  • 10. TYPICAL VERTEBRAE • Basic parts • Body and neural arch • Which consists of pedicles, lamina and spine • The transverse processes arise from the pedicles • Superior and inferior articular processes
  • 12. LUMBAR VERTEBRAE • Body kidney shaped • No articular facets for ribs • Inferior facets face anterolateral • Superior facets face posteromedial • Intervertebral notch increase in size • Accessory processes base of transverse process • Mammillary process on posterior aspect of superior articular process
  • 13. LUMBAR VERTEBRAE • Body is convex anteriorly • Foramina on the posterior aspect are for the basic vertebral veins, which drain into the internal vertebral plexus • The walls of the veins, which are valve less, have afferent nerve fibers • Secondaries can spread from pelvis, prostate, adrenal glands lungs and breast
  • 14. • The superior and inferior surfaces of the body are flat and covered by a thin layer of hyaline cartilage • The body of the vertebra consists of trabecular or cancellous bone Lumbar Vertebrae
  • 15. TYPICAL LUMBAR VERTEBRAE • Superior and inferior articular processes • Arise from the junction of the pedicles and lamina • Superior face posteromedially • With rough mammillary processes on the posterior border • Inferior face anterolaterally • Accessory processes at the base of transverse process • Prevents rotation
  • 16. THE LUMBAR FACETS • Vary from the sagittal disposition at the first and second, to almost coronal in the lower • Facet tropism is when the facet on one side is in the sagittal plane and the other is in the coronal plane, which adds to rotational stress • This change may occur in the lower thoracic vertebrae
  • 17. PARS INTERARTICULARIS • Pars interarticularis • Portion of lamina between superior and inferior articular processes • Site of spondylolysis or spondylolisthesis
  • 18. LUMBAR SPINE • Cancellous bone • 50% compressive strength • Facet joints 20% in standing upright position
  • 21. LUMBAR SPINE • Cancellous bone • 50% of the compressive strength • Facet joints, 20% of the strength in the standing upright position
  • 22. ANTERIOR LONGITUDINAL LIGAMENT • Attached mainly to the bodies • This ligament helps to prevent us from leaning too far back (hyperextension)
  • 23. POSTERIOR LONGITUDINAL LIGAMENT • Attached mainly to the inter vertebral discs • This ligament helps to restrict forward bending (hyperflexion)
  • 24. LIGAMENTUM FLAVA • Runs between the laminae of the neural arches • Helps to restrict hyperflexion • It extends to the capsule of the facet joint • It is highly elastic and ensures that the ligament does not buckle in extension
  • 25. • Gives elasticity to the posterior aspect of the facet joints • Helps form the posterior boundary of the intervertebral foramen • The ligamentum flava is thicker in the lumbar region Ligamentum Flava
  • 26. SPINAL LIGAMENTS • Interspinous ligaments • Strong supraspinous ligaments • The inter-transverse ligaments join the transverse processes and are thin and membranous in the lumbar region
  • 27. FIFTH LUMBAR VERTEBRAE • Larger, superior and inferior articular facets in the same plane • Fifth lumbar vertebrae has large transverse processes • Arise from the body as well as the pedicles
  • 28. ARTHRITIS OF SPINE • Painful • Limitation of movement • Extra projections • Narrowing of disc spaces
  • 29. VERTEBRAL JOINTS • Secondary cartilaginous joints between the bodies • Hyaline cartilage covering bodies • Disc of fibrocartilage in between • Synovial plane joints between the facets
  • 30. INTERVERTEBRAL DISCS • Annulus fibrosis • Concentric lamina run obliquely • Type I collagen at periphery, type II near nucleus • Weakest portion is the postero-lateral and posterior • Periphery has a nerve supply
  • 31. NUCLEUS PULPOSUS • Gelatinous, hydrophilic, proteoglycan gel in collagen matrix • Lies posterior in the disc • There are no nerve endings in a mature disc • Nerve endings are found in the posterior longitudinal ligament and the dura • Nutrition of the disc is by diffusion via the central 40% of the cartilaginous end plate • The discs are thicker in the cervical and lumbar sections of the vertebral column • Where there is more movement. The largest disc is between L5 S1
  • 32. NUCLEUS PULPOSUS • Hydration of the annulus and nucleus is proportional to the applied compressional stress • In vivo, there is a loss of 1 cm standing height over the course of the day • A disc loaded in vitro for four hours by 100% body weight will lose 6% of the fluid from the nucleus and 13% from the annulus • May be due to end plate fracture • There is more rotational stress in the posterior part of the disc
  • 33. NUCLEUS PULPOSUS • The position of the spine determines where the compressional forces are greatest • The posterior longitudinal ligament is thin and expanded at the level of the disc • High compressional loading at L4,L5,S1 may be due to end plate fracture and not to rupture of the annulus • End plate failure is a possible precursor of disc degeneration
  • 34. AXIAL LOAD AND END-PLATES
  • 35. END-PLATE MECHANICS • Functionally, the vertebral end-plate displays characteristics of a trampoline • With the sub-end-plate trabecular bone acting as springs to sustain and dissipate axial load • Despite the thinness of the vertebral end-plate • The hydraulic nature of marrow and blood vessels within the vertebral body, act to dampen axial loads, unless the local point pressure is too high
  • 36. END-PLATE MECHANICS • End-plate lesions can be induced experimentally before a disc will prolapse through the anulus, suggesting a protective mechanism over annular injury and potentially cord or root compression • Excessive loads may result in perforation of the end-plate, usually in the region of the nucleus and often in the path of the developmental notchord
  • 37. END-PLATE SUSCEPTIBILITY Schmorl & Junghanns. The human spine in health and disease. New York: Grune & Stratton, 1965 Notochord
  • 38. FACET JOINTS • L1,L2 Facets sagittal plane • Lower joints in coronal plane • Synovial plane joints • Meniscoid structures • Synovial membrane some contain fat • Supplied by medial branch of dorsal ramus
  • 39. • Narrowing of disc space, results in stress on facet joint • Highest pressure during • Combined • Extension • Rotation • Compression Facet Joints
  • 40. FACET JOINT SYNDROME • Extension and rotation • Pain rising from flexion • Pain worse standing • Lateral shift in extension • Point tenderness over facet • Referred leg pain
  • 41. SEGMENTAL ROTATION Singer et al. J Musculoskel Res 2001;5: 45-55
  • 42. • Flexion limited by disc problems • Lateral flexion • Extension limited by facet joint problems • Very little rotation • Extension and rotation affect facet joints MOVEMENTS OF LUMBAR SPINE
  • 43. NERVE SUPPLY • Nerve supply • Peripheral annulus • Facet joint • Nerve is medial branch dorsal ramus
  • 44. BLOOD SUPPLY • Lumbar arteries • Internal venous plexuses • External venous plexuses • Basivertebral veins • Valveless
  • 46. CANCELLOUS BONE • Cancellous bone • 50% compressive strength • Facet joints 20% in standing upright position Normal bone Osteoporotic bone
  • 47. ANATOMICAL ABNORMALITIES • Spina Bifida Occulta • Facet Tropism • Kyphosis • Scoliosis
  • 49. ANATOMICAL ABNORMALITIES • Hemi-vertebra • Spina Bifida Occulta • Facet Tropism • Scoliosis • Kyphosis
  • 50. ANATOMICAL ABNORMALITIES • Unilateral lumbarisation • Unilateral sacralisation
  • 51. THE SPINE IN SPORTS • Spine injury epidemiology • Contact vs. non-contact sports • Spine injury mechanisms • Overuse – overload – overlooked • Vertebral end-plate injury • Disc injury • Future issues
  • 52. EPIDEMIOLOGY Cooke & Lutz. Phys Med Rehab Clinics N Am 2000;11:837
  • 53. Cooke & Lutz. Phys Med Rehab Clinics N Am 2000;11:837-65 EPIDEMIOLOGY • Back pain in the community is 60% - 80% • Recurrence of back pain is 70% - 90% • Progression to chronic back pain is 5% - 10%
  • 54. LOW BACK PAIN IN SPORTS • Majority of sports injuries are to the lumbar spine • Many soft tissue injuries are not reported • Fractures • Fracture dislocation • Abrasions, bruising • Contusions Tall & De Vault. Clin Sports Med 1993;12:441-8
  • 55. CHRONIC LOW BACK PAIN • Local structures • Muscles • Ligaments • Poor lifting techniques • Joints • Bones
  • 56. BACK PAIN Local structures • Muscles, ligaments • Joints Referred pain • Abdominal organs • Pelvic organs Must out rule • Infection • Tumours
  • 57. ACUTE LOW BACK PAIN • Non-specific low back pain • Usually settles quickly • History • Examination • Pain relief • Stay as active as possible within limit of pain
  • 58. ACUTE LOW BACK PAIN • Nerve root pain • Leg pain worse than back pain • Numbness and pins and needles • Neurological signs • Refer to specialist • If it does not resolve in first 4 weeks
  • 59. INVESTIGATE LOW BACK PAIN • Under 20 or over 55 years • Non-mechanical pain • Past history cancer • Thoracic pain • Steroids or HIV • Unwell, weight loss • Widespread neurology • Structural deformity • Gait disturbance or sphincter disturbance
  • 60. CHRONIC LOW BACK PAIN Pain referred • Abdominal organs • Pelvic organs Must out rule • Infection • Tumours
  • 62. YOUNG ATHLETE • Junior rugby team 15 years of age • M. Scheuermann • 5 Spina bifida occulta • The scrum half had degenerative facet joint changes
  • 63. SACROILIAC JOINT – SCIATIC NERVE
  • 64. SPINAL STENOSIS • Congenital or acquired • Abnormally short pedicles or lamina • Formation of osteophytes • Osteo-arthritis of facet joints • Pain aggravated by walking • Relieved by rest
  • 66. PREDISPOSING FACTORS • Intrinsic factors • Anatomical abnormalities • Biomechanical • Extrinsic factors • Sport • Surfaces • Equipment • Training
  • 67. PREDISPOSING FACTORS BACK PAIN • Poor posture • Overweight • Unfit
  • 68. PREDISPOSING FACTORS • Poor core stability • Weak abdominal muscles • Weak gluteal muscles • Muscle imbalance
  • 69. PREDISPOSING FACTORS • Poor core stability • Weak abdominal muscles • Weak gluteal muscles • Muscle imbalance • Pronated or cavus feet
  • 70. PREDISPOSING FACTORS • Badly designed furniture • No back support • Poor posture at work
  • 72. ANNULAR TEARS • Loaded compression with rotatory component • As little as 3 degrees of high torque rotation • Facets protect disc • As annulus fails, facets joints may be injured
  • 75. YOUNG ATHLETE • Junior rugby team 15 years of age • M. Scheuermann • 5 Spina bifida occulta • The scrum half had degenerative facet joint changes
  • 76. SCHEUERMANN’S DISEASE Greene et al. J Pediatr Orthop 1985;5:1
  • 78. PARS INTERARTICULARIS • Pars interarticularis, portion of lamina between superior and inferior articular processes • Site of spondylolysis or spondylolisthesis
  • 82. SPONDYLOLISTHESIS RAPID FLEXION AND EXTENSION • Gymnastics, flips • Vaulting • Ballet, arabesque • Lifting during dance • Diving • Butterfly swimming • Decathlon • Pole vaulting
  • 84. 465 ATHLETES LOW BACK PAIN (M318;F147) male (39) female(14) Spina Bifida Occulta (SBO) 6.6%(21) 4.1%(6) Lumbarisation 3.5%(11) 1.4%(2) Sacralisation 2.2% (7) 6.1% (9) Spondylolisthesis (13) 30% had SBO; 21 of 56 had other pathology
  • 85. MECHANISM OF INJURIES • Compression or weight loading • Torque or rotation • Tensile stresses produced by excessive motion of spine • Hyperextension and flexion Watkins & Dillin, 1985
  • 86. COMPRESSION OR WEIGHT LOADING • Sports requiring • Massive strength • High body weight • Weight lifter • Hooker and No 8 • Wrestling • Line back American football Watkins & Dillin, 1985
  • 87. WEIGHT LIFTING • 40 % weight lifters have low back pain • Greatest stress is when weight is lifted above the head • Dangerous time is shift from spinal flexion to extension Aggrawal et al. Br J Sports Med 1979;13:58-61
  • 88. AXIAL COMPRESSIVE LOADING • Head on collisions • Motor sports • Boating accidents • Wrestling • Horseback riding • Bicycling • Bobsleigh
  • 95. SPONDYLOLISTHESIS RAPID FLEXION AND EXTENSION • Gymnastics, flips • Vaulting • Ballet, arabesque • Lifting during dance • Diving • Butterfly swimming • Decathlon • Pole vaulting
  • 96. AUSTRALIAN FOOTBALL LEAGUE Seward & Orchard. 2000 AFL Injury Report, Australian Sports Commission
  • 97. GOLF • Highest incidence of back injuries in professional sports • Torsional stress is lessened by spreading the stress over the entire spine • Rigid abdominal control • Parallel shoulders and pelvis Watkins and Dillin, 1985
  • 102. SCOLIOSIS DUE TO UNILATERAL SPORTS • Racquet sports • Fencing • Sweep rowing • Javelin • Freestyle unilateral breathing
  • 103. SCOLIOSIS DUE TO UNILATERAL SPORTS
  • 104. RUNNING • Poor posture • Poor abdominal • Pronated feet • Muscle imbalance • Leg length discrepancy • Osteoporosis
  • 105. CRICKET • Bowlers • Rotational forces • Extension followed by rotation and flexion

Editor's Notes

  1. The late natural history the thoracic kyphosis is for progressive deformation: This deformation occurs within the vertebral bodies in women with a greater contribution from disc degeneration in males.[Goh et al 1999] Those sports that predispose the individual to hyper-kyphosis during the adolescent years and beyond, should be studied to determine the association between the structural deformation and back pain. [[Those of us destined to keyboard activity will identify with the evolutionary trend depicted above]]
  2. As can be seen on the left, the vertebral end-plate is a tenuous cartilaginous membrane which from direct measurement is approximately 0.5mm thick, connected to trabecular bone within the cortical shell. Lesions of the end-plate arising from sporting activities are reported to be frequent. Typical aetiology involves dynamic compressive axial loads, common in landing sports: eg: gymnastics Discal material is extruded through the end-plate into the vertebral body. At the time of injury, the lesion may be painful due to the inflammatory response to the lesion. It has been postulated that such injury predisposes the disc to early degenerative change [Roberts et al, 1997 European Spine Journal 6: 387] The late stage of healing involves sclerosis of bone around the site of injury, demonstrated on the right [arrow] form a CT at T11-12
  3. The notochordal streak, as depicted by Schmorl & Junghanns from their classic text, showing a foetal specimen [left] and the progressive apoptosis of these cells during maturation and differentiation of the disc and vertebral body. Typically, Schmorl’s nodes occur close to this site, suggesting both a functional and genetic predisposition to compressive load failure of the end-plates in some individuals.
  4. In a recent CT study, 42 patients suspected of disc and facet joint pain were rotated within the CT scanner prior to scanning. They were fixed into this position for both right and left side scans which were compared with their neutral [conventional] scan images. Very slight separation of the facet joints occurred at most levels and particularly where the anatomical alignment was mostly coronal [usually the lowest segments]. Joints with a more sagittal alignment, typically the upper lumbar spine, appeared to offer greater resistance to torsion.
  5. Spine injuries can occur through overuse, overload, trauma or a combination of these events. Injuries may occur to any component of the mobile segment: disc, vertebral bodies or facet joints, however disc and end-plate lesions are the most commonly affected.
  6. The reported statistics for back pain according to Cooke & Lutz [2000] are: Lifetime prevalence of back pain in the community = 60-80% Back pain recurrence = 70-90% Progression to chronic back pain = 5-10% Back pain a common feature in most sports, particularly competitive contact sports. Back pain is higher in young athletes compared with age matched controls and, Back pain may occur in response to various conditions of load, fatigue and trauma
  7. The reported statistics for back pain according to Cooke & Lutz [2000] are: Lifetime prevalence of back pain in the community = 60-80% Back pain recurrence = 70-90% Progression to chronic back pain = 5-10% Back pain a common feature in most sports, particularly competitive contact sports. Back pain is higher in young athletes compared with age matched controls and, Back pain may occur in response to various conditions of load, fatigue and trauma
  8. The incidence of end-plate lesions in sport participants varies, however, these may result from pre-existing anatomical abnormalities. In the case of Scheuermann’s disease, there can be multiple end-plate lesions over many segments. According to Sorenson, the characteristics of this disease involve four or more segments with lesions of the end-plates, and corresponding vertebral wedging. Accentuated kyphosis and a painful thoraolumbar spine are the main clinical features.
  9. AFL is a dynamic game that involves various types of play and physical encounters. Injuries are common, predominantly to the knee [ACL], hamstring and groin regions. In a major survey of AFL injuries undertaken by Drs Seward & Orchard, identified a recurrence rate of 32% for injuries to the neck, back, and ribs.