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P R E S E N T E D B Y ;
L T ; A Y E M Y A T H A N D A R
( 2 1 / 1 0 0 ) M H
ACUTE CORONARY SYNDROME
Acute Coronary Syndrome is
 Due to decreased blood
flow in the coronary
arteries so that part of
the heart muscle is
unable to function
properly or dies
 Clinical symptoms that
equate to myocardial
ischaemia
ACS is subdivided in three types depending on ECG
changes and blood test result,
1. ST elevation myocardial
infarction (STEMI)
2. Non-ST elevation myocardial
infarction (NSTEMI)
3. Unstable angina
Causes of ACS
. Result of plaque disruption in the coronary
arteries
. Common risk factors are
* Smoking
* Hypertension
* Diabetes
* Hyperlipidemia
* Family obesity, poor nutritional practices
Signs and Symptoms of ACS are:
 Midsternal or substernal compression or crushing chest
discomfort
 May be described as pressure, tightness or heaviness in the
chest
 Unexplained indigestion or epigastric pain
 Radiate to neck, jaw, shoulder, back and one or both arms
Investigations
 Cardiac Troponins T (Normal - less than 0.04 ng/ml)
 Cardiac Troponins I (Normal - between 0 and 0.04
ng/mL) , High-Sensitivity cardiac troponin (hs-cTn)
(Normal - < 14ng/L)
 CKMB (Normal 5 to 25 IU/L)
 Cath Profiles For Procedure
1. CP,ESR
2. U&C, Electrolyte, Lipid Profile, LFT, TFT
3. Serology
4. PT, INR
Non Invasive - Echo
Regional Wall Motion Septal Hypokinesia
Non Invasive – Exercise ECG (Treadmill)
Non Invasive – CT Ca++ Score
ST-Elevation Myocardial Infarction
Result from complete and prolonged occlusion of an
epicardial coronary blood vessel
ST-Elevation Myocardial Infarction
Non-STEMI
 Result from severe coronary artety narrowing,
transient occulsion
 Elevation of cardiac biomarkers in the absence of ST
elevation
Unstable Angina
 Heart doesn’t get enough blood flow and oxygen
 Occur at rest
 Become more frequent, severe and prolonged
 Not response to rest or nitroglycerine
Approach-to-diagnosis and risk stratification of
ACS
Approach-to-diagnosis and Management
Treatments
(1) Medications
(1) Antiplatelet
(2) Anti Ischaemic
(3) Anti Coagulant
Treatments
(2) Thrombolysis
IV infusion
Streptokinase 15 lks +
NS 100 ml within 1 hr
(100ml/hr)
Treatments
(3) Percutaneous coronary intervention ( PCI )
Treatments
(4) Coronary bypass surgery
Nursing Management
Assessment
 Changes in vital signs ( BP, PR,SaO2,HR)
 Shortness of breath or dyspnoea
 Tachycardia
 Dizzness, changes in level of consciousness
 Pallor
 Nausea and vomiting
 Anxiety
 Irritability
 Decrease peripheral pulse
Diagnosis
 Acute pain related to decrease blood and oxygen
supply to myocardium.
 Activity intolerance r/t cardiac dysfunction and
imbalance in oxygen supply.
 Anxiety r/t pain and perceived thread of death.
 Knowledge deficit r/t anxiety seondary to not
knowing the disease process.
Planning
 may be relieved from currently suffered chest pain.
 may be coped with the anxiety.
 may be understand the causes and treatment plans
of the disease.
Implementation
 Assess respirations, BP and heart rate with each
episodes of chest pain.
 Provide comfort measures such as back rub.
 Maintain bed rest during pain with position of
comfort.
 Prepare for the administration of prescribed
medications and monitor response to drug therapy.
 Caution patient to avoid activities that incerase
cardiac output.
 Observe skin colour, temperature and capillary refill
time.
 Monitor intake and output and calculate 24 hr fluid
balance.
 Administer oxygen as indicated.
 Decrease the patient’s anxiety by explaining the
disease, cauative factors DOs and DON’Ts, and
available treatments.
Implementation – Con;
Evaluation
The patient had
1. relieved from pain .
2. relieved from anxiety.
3. understood about the disease and Dos and
DON’Ts

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ACUTE CORONARY SYNDROME.pptx

  • 1. P R E S E N T E D B Y ; L T ; A Y E M Y A T H A N D A R ( 2 1 / 1 0 0 ) M H ACUTE CORONARY SYNDROME
  • 2. Acute Coronary Syndrome is  Due to decreased blood flow in the coronary arteries so that part of the heart muscle is unable to function properly or dies  Clinical symptoms that equate to myocardial ischaemia
  • 3. ACS is subdivided in three types depending on ECG changes and blood test result, 1. ST elevation myocardial infarction (STEMI) 2. Non-ST elevation myocardial infarction (NSTEMI) 3. Unstable angina
  • 4.
  • 5. Causes of ACS . Result of plaque disruption in the coronary arteries . Common risk factors are * Smoking * Hypertension * Diabetes * Hyperlipidemia * Family obesity, poor nutritional practices
  • 6. Signs and Symptoms of ACS are:  Midsternal or substernal compression or crushing chest discomfort  May be described as pressure, tightness or heaviness in the chest  Unexplained indigestion or epigastric pain  Radiate to neck, jaw, shoulder, back and one or both arms
  • 7. Investigations  Cardiac Troponins T (Normal - less than 0.04 ng/ml)  Cardiac Troponins I (Normal - between 0 and 0.04 ng/mL) , High-Sensitivity cardiac troponin (hs-cTn) (Normal - < 14ng/L)  CKMB (Normal 5 to 25 IU/L)  Cath Profiles For Procedure 1. CP,ESR 2. U&C, Electrolyte, Lipid Profile, LFT, TFT 3. Serology 4. PT, INR
  • 8. Non Invasive - Echo Regional Wall Motion Septal Hypokinesia
  • 9. Non Invasive – Exercise ECG (Treadmill)
  • 10. Non Invasive – CT Ca++ Score
  • 11.
  • 12.
  • 13. ST-Elevation Myocardial Infarction Result from complete and prolonged occlusion of an epicardial coronary blood vessel
  • 15. Non-STEMI  Result from severe coronary artety narrowing, transient occulsion  Elevation of cardiac biomarkers in the absence of ST elevation
  • 16. Unstable Angina  Heart doesn’t get enough blood flow and oxygen  Occur at rest  Become more frequent, severe and prolonged  Not response to rest or nitroglycerine
  • 17. Approach-to-diagnosis and risk stratification of ACS
  • 19. Treatments (1) Medications (1) Antiplatelet (2) Anti Ischaemic (3) Anti Coagulant
  • 20. Treatments (2) Thrombolysis IV infusion Streptokinase 15 lks + NS 100 ml within 1 hr (100ml/hr)
  • 21. Treatments (3) Percutaneous coronary intervention ( PCI )
  • 24. Assessment  Changes in vital signs ( BP, PR,SaO2,HR)  Shortness of breath or dyspnoea  Tachycardia  Dizzness, changes in level of consciousness  Pallor  Nausea and vomiting  Anxiety  Irritability  Decrease peripheral pulse
  • 25. Diagnosis  Acute pain related to decrease blood and oxygen supply to myocardium.  Activity intolerance r/t cardiac dysfunction and imbalance in oxygen supply.  Anxiety r/t pain and perceived thread of death.  Knowledge deficit r/t anxiety seondary to not knowing the disease process.
  • 26. Planning  may be relieved from currently suffered chest pain.  may be coped with the anxiety.  may be understand the causes and treatment plans of the disease.
  • 27. Implementation  Assess respirations, BP and heart rate with each episodes of chest pain.  Provide comfort measures such as back rub.  Maintain bed rest during pain with position of comfort.  Prepare for the administration of prescribed medications and monitor response to drug therapy.
  • 28.  Caution patient to avoid activities that incerase cardiac output.  Observe skin colour, temperature and capillary refill time.  Monitor intake and output and calculate 24 hr fluid balance.  Administer oxygen as indicated.  Decrease the patient’s anxiety by explaining the disease, cauative factors DOs and DON’Ts, and available treatments. Implementation – Con;
  • 29. Evaluation The patient had 1. relieved from pain . 2. relieved from anxiety. 3. understood about the disease and Dos and DON’Ts