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TRACHEO
OSEOPHAGIAL
FISTULA
JEBAKUMARI SUTHA.A
LECTURER
GNGA INSTITUTE OF HEALTH SCIENCES
COIMBATORE
TRACHEO ESOPHAGEAL FISTULA
OUTLINES:
 Definition of TEF
 Development
 Epidemiology
 Anatomical variation
 Pathophysiology
 Clinical manifestations
 Diagnostic evaluation
 General management
 Surgical management
 Nursing management
DEFINITION
A tracheoesophageal fistula
(TEF) is a congenital or
acquired communication
between the trachea and
esophagus.
DEVELOPEMENT
 The esophagus and trachea both develop from the
primitive foregut. In a 4- to 6-week-old embryo,
the caudal part of the foregut forms a ventral
diverticulum that evolves into the trachea.
 The longitudinal tracheoesophageal fold fuses to
form a septum that divides the foregut into a
ventral laryngotracheal tube and a dorsal
esophagus. The posterior deviation of the
tracheoesophageal septum causes incomplete
separation of the esophagus from the
laryngotracheal tube and results in a TEF
Epidemiology
 Incidence: 1/4000
 30-50%: congenital anomalies (35%
cardiac; 30% musculoskeletal)
 20-30%: premature, BW<2000g
 VACTERAL and VATER syndrome:
 Vertebral anomalies:
 Anal: imperforate anus
 Cardiac: VSD, PDA, TOF, coarction of aorta, ASD
 Trachea: TEF
 Esophageal: EA
 Renal: renal agenesis,ureteral abnormalities,
hypospadias
 Limb: polydactyly, wrist/knee anomalies
Anatomic Variations
6%
Atresia alone,
 no fistula
 Small stomach,
gasless abdomen
 Usually has a long
gap between the
esophagealends
 GROSS type A
Anatomic Variations
 2%
 Proximal tracheo-
esophageal fistula
 No distal fistula
Small stomach,
 gasless abdomen
 Often has a long
gap between the
Esophageal ends
 GROSS type B
Anatomic Variations
 85%
 Most common
 GROSS type C
Anatomic Variations
 l%
 Proximal and
distal fistulas
("double fistula")
 GROSS type D
Anatomic Variations
 6%
 No atresia of
the esophagus
 Congenital
tracheoesophageal
fistula
"H" or "N" fistula
 GROSS type E
Physiologic effects of distal tracheoesophageal
fistula
Cont ……..
Hyaline membrane disease may necessitate
higher ventilator pressures, which encourage
air to pass through the distal fistula.
A distended abdomen elevates and "splints"
the diaphragm.
Gastric distension may result in gastric rupture
and pneumoperitoneum.
Passage of air through a distal
tracheoesophageal fistula diminishes the
effective tidal volume.
(B)
Aspiration of gastric juices leads to
soiling of the lungs and pneumonia
Gastroesophageal refluxDirection of
gastric fluid proximally through
distal fistula.
Overflow of secretions or
inadvertent feeding may contribute
to aspiration and contamination of
the airway. .
CLINICAL
MANIFESTATIONS
DROOLING OF SALIVA
CHOCKING
RESPIRATORY DISTRESS
INABILITY TO FEED
GASTRIC DISTENSION
ASPIRATION PNEUMONIA
DIAGNOSIS OF ESOPHAGEAL
ATRESIA
 Antenatal Diagnosis (maternal
polyhydramnios, a small stomach, a
distended upper esophageal pouch,
or abnormal swallowing)
 Diagnostic suspicion is increased
when abnormalities known to be
associated with esophageal atresia
are identified.
Fetal MRI
 This 32 week
fetus had
esophageal
atresia and an
absent stomach,
resulting in
marked
polyhydramnios
Clinical Diagnosis
 Prematurity
 Any excessively drooling
(copious, fine, white, frothy
bubbles of mucus in the mouth
and, sometimes, the nose).
Clinical Diagnosis
(A) Diagnosis of
esophageal atresia
is confirmed when a
10-gauge (French)
catheter cannot be
passed beyond 10
cm from the gums.
(B) A smaller-caliber
tube is not used
because it may curl
up in the upper
esophageal
segment, giving a
false impression of
esophageal
continuity.
The chest radiograph
 A plain radiograph
will confirm the tube
has not reached the
stomach
The Gasless Abdomen
 Absence of gas in the
abdomen suggests
that the patient has
either atresia without
a fistula or atresia
with a proximal
fistula only
Contrast studies
 should be performed by
an experienced pediatric
radiologist, or after
transfer to the tertiary
institution, and with the
use of a small amount
(0.5 to 1 mL) of water-
soluble contrast. Care
must be taken to avoid
aspiration.
General management
 Primary repair of TEF is urgent
 Associated anomalies: CV, GU,
musculoskeletal
 Protect the lungs from aspiration pneumonia
 Avoidance of feeding
 Positioning of the infant slightly head up to
minimize regurgitation of gastric contents
through fistula
 Intermittent suction
 Antibiotic therapy and physiotherapy in babies
with contaminated lung
SURGICAL MANAGEMENT
 one stage repair: optimal surgical
management
 fistula is ligated, proximal and distal ends of the esophagus are
anastomosed
 right posterolateral extrapleural thoracotomy
 left thoracotomy if the patient has right aortic arch
 gastrostomy:
 high risk infants unable to withstand a thoracotomy
 decompress the stomach and prevent regurgitation via fistula into the
lungs
 local anesthesia:
 general anesthesia: protect the lungs from aspiration during surgical
manipulation
SURGICAL MANAGMENT
 Definitive repair
 24-72 hours later, when infant can withstand both
surgery and anesthesia
 Method: right thoracotomy using a posterolateral
extrapleural approach
 Fistula is ligated, esophageal segments are
anastomosed
 If distal esophagus is too short
Fistula is ligated
exteriorization of the upper pouch through an
esophagostomy is performed
wait till weight of 9kg or 1 y/o to perform
anastomosis
 Respiratory difficulty
after feedings in a 3-day-
old boy. Barium
esophagogram clearly
shows an H-shaped
fistula between the
trachea and the middle
segment of the
esophagus (arrowhead).
Barium is filling the
bronchi of the right
lower lobe (arrows).
Tracheoesophageal fistula
without atresia (type E).
Tracheoesophageal fistula
without atresia (type E).
 Esophagogram shows
a fistula (arrow)
arising from the
anterior portion of
the esophagus (e) and
passing cephalad to
the posterior portion
of the trachea (t).
NURSING MANAGEMENT
 SMALL AND FREQUENT FEEDS
 IV FLUIDS MAINTANENCE
 MAINTAIN UPRIGHT POSITION
 PREPARATION OF PARENTS FOR SURGERY
 FIXATION OF NG TUBE FOR
DECOMPRESSION
tracheo oesophagal fistula   gihs

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tracheo oesophagal fistula gihs

  • 2.
  • 3. TRACHEO ESOPHAGEAL FISTULA OUTLINES:  Definition of TEF  Development  Epidemiology  Anatomical variation  Pathophysiology  Clinical manifestations  Diagnostic evaluation  General management  Surgical management  Nursing management
  • 4. DEFINITION A tracheoesophageal fistula (TEF) is a congenital or acquired communication between the trachea and esophagus.
  • 5. DEVELOPEMENT  The esophagus and trachea both develop from the primitive foregut. In a 4- to 6-week-old embryo, the caudal part of the foregut forms a ventral diverticulum that evolves into the trachea.  The longitudinal tracheoesophageal fold fuses to form a septum that divides the foregut into a ventral laryngotracheal tube and a dorsal esophagus. The posterior deviation of the tracheoesophageal septum causes incomplete separation of the esophagus from the laryngotracheal tube and results in a TEF
  • 6.
  • 7. Epidemiology  Incidence: 1/4000  30-50%: congenital anomalies (35% cardiac; 30% musculoskeletal)  20-30%: premature, BW<2000g  VACTERAL and VATER syndrome:  Vertebral anomalies:  Anal: imperforate anus  Cardiac: VSD, PDA, TOF, coarction of aorta, ASD  Trachea: TEF  Esophageal: EA  Renal: renal agenesis,ureteral abnormalities, hypospadias  Limb: polydactyly, wrist/knee anomalies
  • 8.
  • 9.
  • 10. Anatomic Variations 6% Atresia alone,  no fistula  Small stomach, gasless abdomen  Usually has a long gap between the esophagealends  GROSS type A
  • 11. Anatomic Variations  2%  Proximal tracheo- esophageal fistula  No distal fistula Small stomach,  gasless abdomen  Often has a long gap between the Esophageal ends  GROSS type B
  • 12. Anatomic Variations  85%  Most common  GROSS type C
  • 13. Anatomic Variations  l%  Proximal and distal fistulas ("double fistula")  GROSS type D
  • 14. Anatomic Variations  6%  No atresia of the esophagus  Congenital tracheoesophageal fistula "H" or "N" fistula  GROSS type E
  • 15. Physiologic effects of distal tracheoesophageal fistula
  • 16. Cont …….. Hyaline membrane disease may necessitate higher ventilator pressures, which encourage air to pass through the distal fistula. A distended abdomen elevates and "splints" the diaphragm. Gastric distension may result in gastric rupture and pneumoperitoneum. Passage of air through a distal tracheoesophageal fistula diminishes the effective tidal volume.
  • 17. (B) Aspiration of gastric juices leads to soiling of the lungs and pneumonia Gastroesophageal refluxDirection of gastric fluid proximally through distal fistula. Overflow of secretions or inadvertent feeding may contribute to aspiration and contamination of the airway. .
  • 25. DIAGNOSIS OF ESOPHAGEAL ATRESIA  Antenatal Diagnosis (maternal polyhydramnios, a small stomach, a distended upper esophageal pouch, or abnormal swallowing)  Diagnostic suspicion is increased when abnormalities known to be associated with esophageal atresia are identified.
  • 26.
  • 27. Fetal MRI  This 32 week fetus had esophageal atresia and an absent stomach, resulting in marked polyhydramnios
  • 28. Clinical Diagnosis  Prematurity  Any excessively drooling (copious, fine, white, frothy bubbles of mucus in the mouth and, sometimes, the nose).
  • 29. Clinical Diagnosis (A) Diagnosis of esophageal atresia is confirmed when a 10-gauge (French) catheter cannot be passed beyond 10 cm from the gums. (B) A smaller-caliber tube is not used because it may curl up in the upper esophageal segment, giving a false impression of esophageal continuity.
  • 30. The chest radiograph  A plain radiograph will confirm the tube has not reached the stomach
  • 31. The Gasless Abdomen  Absence of gas in the abdomen suggests that the patient has either atresia without a fistula or atresia with a proximal fistula only
  • 32. Contrast studies  should be performed by an experienced pediatric radiologist, or after transfer to the tertiary institution, and with the use of a small amount (0.5 to 1 mL) of water- soluble contrast. Care must be taken to avoid aspiration.
  • 33. General management  Primary repair of TEF is urgent  Associated anomalies: CV, GU, musculoskeletal  Protect the lungs from aspiration pneumonia  Avoidance of feeding  Positioning of the infant slightly head up to minimize regurgitation of gastric contents through fistula  Intermittent suction  Antibiotic therapy and physiotherapy in babies with contaminated lung
  • 34. SURGICAL MANAGEMENT  one stage repair: optimal surgical management  fistula is ligated, proximal and distal ends of the esophagus are anastomosed  right posterolateral extrapleural thoracotomy  left thoracotomy if the patient has right aortic arch  gastrostomy:  high risk infants unable to withstand a thoracotomy  decompress the stomach and prevent regurgitation via fistula into the lungs  local anesthesia:  general anesthesia: protect the lungs from aspiration during surgical manipulation
  • 35. SURGICAL MANAGMENT  Definitive repair  24-72 hours later, when infant can withstand both surgery and anesthesia  Method: right thoracotomy using a posterolateral extrapleural approach  Fistula is ligated, esophageal segments are anastomosed  If distal esophagus is too short Fistula is ligated exteriorization of the upper pouch through an esophagostomy is performed wait till weight of 9kg or 1 y/o to perform anastomosis
  • 36.  Respiratory difficulty after feedings in a 3-day- old boy. Barium esophagogram clearly shows an H-shaped fistula between the trachea and the middle segment of the esophagus (arrowhead). Barium is filling the bronchi of the right lower lobe (arrows). Tracheoesophageal fistula without atresia (type E).
  • 37. Tracheoesophageal fistula without atresia (type E).  Esophagogram shows a fistula (arrow) arising from the anterior portion of the esophagus (e) and passing cephalad to the posterior portion of the trachea (t).
  • 38. NURSING MANAGEMENT  SMALL AND FREQUENT FEEDS  IV FLUIDS MAINTANENCE  MAINTAIN UPRIGHT POSITION  PREPARATION OF PARENTS FOR SURGERY  FIXATION OF NG TUBE FOR DECOMPRESSION