Thrombosis & Haemostasis: Research
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Thrombosis & Haemostasis: Research is an open access, peer reviewed, scholarly journal dedicated to publish articles covering all areas of Thrombosis & Haemostasis. The journal aims to promote research communications and provide a forum for doctors, researchers, physicians and healthcare professionals to find most recent advances in all areas of Thrombosis & Haemostasis. Thrombosis & Haemostasis: Research accepts original research articles, reviews, mini reviews, case reports and rapid communication covering all aspects of Thrombosis & Haemostasis. Thrombosis & Haemostasis: Research strongly supports the scientific up gradation and fortification in related scientific research community by enhancing access to peer reviewed scientific literary works. Austin Publishing Group also brings universally peer reviewed journals under one roof thereby promoting knowledge sharing, mutual promotion of multidisciplinary science.
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Thrombosis
Thrombosis is the formation of a blood clot within a blood vessel. A thrombus is a blood clot that forms in a vessel and remains attached. Thrombi develop at sites of endothelial injury, abnormal blood flow, or hypercoagulability. Arterial thrombi typically arise at sites of atherosclerosis or turbulence, while venous thrombi occur where blood flow is slow. Treatment involves anticoagulants to prevent thrombi from growing or new clots forming.
Infraction - Dr. Abhinav Golla MD. Pathology
1. An infarction is an area of ischemic necrosis in an organ resulting from reduced blood supply, usually due to arterial obstruction.
2. The main causes of infarction are thromboembolism and atherosclerosis, which interrupt arterial blood flow. This leads to localized tissue death from coagulative necrosis.
3. Pathologically, infarcts appear pale and wedge-shaped, with inflammation at the borders and eventual scar formation. Location and outcomes vary by organ, such as potentially lethal myocardial infarction or non-lethal splenic infarction.
Bleeding disorders
This document discusses platelet disorders, coagulation disorders, and vascular disorders that cause purpura. It describes thrombocytopenic purpura which is caused by a platelet deficiency, and thrombasthenic purpura which is a functional platelet disorder. Coagulation disorders discussed include hemophilia A, B, and C which are hereditary deficiencies in coagulation factors VIII, IX, and XI respectively. Tests to detect defects in platelet function and coagulation pathways are also outlined.
Embolism
"Embolism"
this presentation is about embolism where some information about embolism is included. Hope this will help you in your work.
Embolism
This document defines and describes different types of embolism. It states that an embolism is a detached solid, liquid, or gas mass carried by the bloodstream to a distant site. It then lists and provides details on various types of embolisms including venous (pulmonary), arterial, paradoxical, fat, amniotic fluid, air, and septic embolisms. For pulmonary embolisms, it notes they most commonly arise from deep vein thromboses in the legs and can cause further recurrent embolic episodes. It also provides information on symptoms and causes of fat, air, and amniotic fluid embolisms.
Thrombosis
Thrombosis is the formation of a blood clot or thrombus within a blood vessel. It occurs through the interaction of platelets, clotting factors, and altered blood flow. Predisposing factors include injury to blood vessels, stasis or turbulence of blood flow, and hypercoagulability. The clotting process involves platelet adhesion and aggregation at the site of injury, activation of clotting factors, and conversion of fibrinogen to fibrin. Thrombi can cause downstream effects such as ischemia, infarction, embolism, or disseminated intravascular coagulation depending on their location within arteries or veins.
Hemodynamics 1
This document summarizes hemodynamic disorders and edema. It defines edema as abnormal excessive accumulation of fluid in tissues and serous cavities. Edema can be localized or generalized depending on its cause and distribution. The key mechanisms of edema formation are increased hydrostatic pressure, decreased plasma oncotic pressure, lymphatic obstruction, and sodium and water retention. Specific types of edema like pulmonary edema, cerebral edema, hepatic edema, and renal edema are also discussed. The document compares the characteristics of transudate and exudate fluids and provides diagrams to illustrate the pathogenesis of different types of edema.
Pathology of hemodynamic disorders Part 1 nov 2017 Sufia Husain
This document provides an overview of hemodynamic disorders including edema, hyperemia, congestion, hemorrhage, thrombosis, and infarction. It begins by defining edema and its various types. It then discusses hyperemia and congestion, distinguishing between the active process of hyperemia and the passive process of congestion. The document outlines the causes, pathogenesis and morphology of thrombosis. It also discusses endothelial injury, abnormal blood flow, and hypercoagulable states as they relate to the development of thrombosis. Specific conditions that can lead to endothelial dysfunction and abnormal blood flow are highlighted.
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Thrombosis
Thrombosis is the formation of a blood clot within a blood vessel. A thrombus is a blood clot that forms in a vessel and remains attached. Thrombi develop at sites of endothelial injury, abnormal blood flow, or hypercoagulability. Arterial thrombi typically arise at sites of atherosclerosis or turbulence, while venous thrombi occur where blood flow is slow. Treatment involves anticoagulants to prevent thrombi from growing or new clots forming.
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1. An infarction is an area of ischemic necrosis in an organ resulting from reduced blood supply, usually due to arterial obstruction.
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3. Pathologically, infarcts appear pale and wedge-shaped, with inflammation at the borders and eventual scar formation. Location and outcomes vary by organ, such as potentially lethal myocardial infarction or non-lethal splenic infarction.
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Embolism
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Thrombosis
Thrombosis is the formation of a blood clot or thrombus within a blood vessel. It occurs through the interaction of platelets, clotting factors, and altered blood flow. Predisposing factors include injury to blood vessels, stasis or turbulence of blood flow, and hypercoagulability. The clotting process involves platelet adhesion and aggregation at the site of injury, activation of clotting factors, and conversion of fibrinogen to fibrin. Thrombi can cause downstream effects such as ischemia, infarction, embolism, or disseminated intravascular coagulation depending on their location within arteries or veins.
Hemodynamics 1
This document summarizes hemodynamic disorders and edema. It defines edema as abnormal excessive accumulation of fluid in tissues and serous cavities. Edema can be localized or generalized depending on its cause and distribution. The key mechanisms of edema formation are increased hydrostatic pressure, decreased plasma oncotic pressure, lymphatic obstruction, and sodium and water retention. Specific types of edema like pulmonary edema, cerebral edema, hepatic edema, and renal edema are also discussed. The document compares the characteristics of transudate and exudate fluids and provides diagrams to illustrate the pathogenesis of different types of edema.
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Ischemia and Infarction
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1. Thrombosis is the formation of a blood clot (thrombus) within the circulatory system of a living organism. It is caused by endothelial injury, changes in blood flow, and hypercoagulability according to Virchow's triad.
2. Thrombi form in arteries and veins, and can cause obstruction, ischemia, infarction, or embolization if parts break off. Common sites are the legs, lungs, heart, and brain.
3. Embolism occurs when a thrombus or other mass travels through the bloodstream and blocks a vessel in another part of the body, potentially causing infarction or sepsis. Pulmonary embolism from deep leg vein
4.Microcirculatiomicrocirculation presentation
This document discusses various types of microcirculatory disturbances including arterial hyperemia, venous hyperemia, ischemia, thrombosis, embolism, and disturbances of lymph circulation. It provides details on the mechanisms, causes, signs, and outcomes of each type of disturbance. Key components of microcirculation like the coagulation cascade and formation of thrombi are also explained.
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Ischemia is defined as a condition of inadequate blood supply to an area of tissue.
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- Non-coding RNAs including microRNAs and long non-coding RNAs that regulate gene expression.
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thrombosisembolismandinfarction-180117180555.pptx
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Disorders of blood vessels.
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- Atherosclerosis, the thickening of arterial walls due to fat and white blood cell deposition.
- Aneurysms, abnormal artery dilations that can be fusiform, saccular, or dissecting.
- Embolisms, blood clots moving in circulation that become lodged in small vessels.
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Hemodynamic disorders.pdf
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THROMBOSIS & SHOCK
The document discusses thrombosis and shock. It defines thrombosis as the formation of a blood clot in a blood vessel. It then describes different types of thrombosis including deep vein thrombosis, portal vein thrombosis, and cerebral venous sinus thrombosis. The causes and mechanisms of thrombosis are also explained. The document also defines shock and describes the different types including cardiogenic, hypovolemic, septic, neurogenic, and anaphylactic shock. It outlines the signs, symptoms and clinical stages from non-progressive to progressive to irreversible shock.
FPATH
Deep vein thrombosis occurs when a blood clot forms in the deep veins of the leg, usually caused by prolonged immobility. When sitting for long periods, such as on a plane, the calf muscles do not contract and blood circulation decreases, allowing a clot to form. Part of the clot can break off and travel through the bloodstream as an embolism to the lungs, blocking vessels and causing a pulmonary embolism. Symptoms of deep vein thrombosis include leg pain and swelling, while a pulmonary embolism causes shortness of breath, chest pain, and other issues. Both conditions are serious and require medical treatment.
4.Circulation.disorders.pdf
This document provides a summary of various pathologies related to blood circulation. It discusses edema, hyperemia, ischemia, hemorrhage, thrombosis, and embolism. It defines these terms and describes their causes, classifications, and clinical significance. For example, it explains that edema is the accumulation of tissue fluid, and discusses different types like traumatic or inflammatory edema. It also covers topics like venous plethora, arterial hyperemia, ischemia, mechanisms of bleeding, classifications of hematomas, blood stasis, classifications of thrombi, and outcomes of thrombosis.
Thrombosis
Thrombosis is the formation of a blood clot within blood vessels. A thrombus is a blood clot that forms at the site of endothelial injury or abnormal blood flow and can cause vascular obstruction. Virchow's triad describes the three main factors that contribute to thrombus formation: endothelial injury, abnormal blood flow, and hypercoagulability. Thrombi form in different locations depending on the underlying cause, and can propagate and embolize, becoming potentially life-threatening.
Hemodynamic disorder
Hemodynamic disorders affect the heart and blood vessels and include conditions like edema, hyperemia, congestion, hemorrhage and thrombosis. Thrombosis involves blood clot formation within intact vessels and can develop due to endothelial injury, alterations in normal blood flow like stasis and turbulence, and hypercoagulability of the blood, as described by Virchow's triad. Endothelial injury, turbulence and stasis promote activation and dysfunction of endothelial cells, while hypercoagulability refers to genetic or acquired alterations that increase coagulation and the risk of clots.
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- 1. THROMBOSIS
- 2. WHAT IS THROMBOSIS? Thrombosis is a kind of blood clot inside a blood vessel, obstructing the flow of blood through the circulatory system. Thrombosis may occur in arteries or in veins.
- 4. TYPES OF THROMBOSIS Venous thrombosis Arterial thrombosis There are two types of thrombosis:
- 5. Venous thrombosis: A venous thrombosis is a blood clot that occurs within the vein. It leads to congestion of the affected part of the body.
- 6. Arterial thrombosis: A arterial thrombosis is a blood clot that occurs within the artery It affects the blood supply and leads to damage of the tissue supplied by the artery
- 7. VARIOUS DISEASES OF VENOUS THROMBOSIS Deep vein thrombosis Paget-schroetter disease Budd chiari syndrome Portal vein thrombosis Renal vein thrombosis Cerebral venous sinus thrombosis Jugular vein thrombosis Cavernous sinus thrombosis
- 8. VARIOUS DISEASES OF VENOUS THROMBOSIS Stroke Myocardial infarction Limb ischemia Hepatic artery thrombosis
- 9. RISK FACTORS OF THROMBOSIS Stroke Heart failure Obesity Trauma Dehydration Dysrhythmias Acute respiratory failure Vasoconstriction Post partum period Cancers
- 11. CAUSES OF THROMBOSIS The composition of blood The quality of vessel wall Nature of blood flow
- 12. SYMPTOMS OF THROMBOSIS Cramping pain in the affected area Tenderness Swelling Skin over the affected area becomes reddish or bluish color Warm skin in the area of clot
- 13. VARIOUS TREATMENTS FOR THROMBOSIS Anticoagulation Thrombolysis Surgery Endovascular treatment Antiplatelet agents