Stroke is a major cause of death and disability in India. Rehabilitation after a stroke aims to help patients regain independence and function through a team-based approach. Rehabilitation occurs in phases, beginning in the acute post-stroke period and continuing through outpatient care. It focuses on improving impairments, preventing complications, and enhancing quality of life through restoration and compensation strategies that capitalize on the brain's neuroplasticity. Common post-stroke impairments include motor deficits, cognitive issues, swallowing difficulties, and pain. An individualized rehabilitation plan incorporating exercise, therapy, and lifestyle changes can help patients achieve their goals.
“The ability of neurons to change their function, chemical profile or structure is referred to as neuroplasticity.”
Neuroplasticity includes :
- Habituation
- Learning & memory
- Cellular recovery after injury
Stroke rehabilitation and its aspects to work with patients with hemiplegia and other effects of stroke, other than that you will see some pictures of the used interventions and adaptive equipment used with stroke patients
It is a technique developed by Janet H Carr and Roberta B Shepherd which provides physiotherapists and occupational therapists with an approach to stroke rehabilitation that is clear, relevant, and effective, building on the research-based model created by the authors
“The ability of neurons to change their function, chemical profile or structure is referred to as neuroplasticity.”
Neuroplasticity includes :
- Habituation
- Learning & memory
- Cellular recovery after injury
Stroke rehabilitation and its aspects to work with patients with hemiplegia and other effects of stroke, other than that you will see some pictures of the used interventions and adaptive equipment used with stroke patients
It is a technique developed by Janet H Carr and Roberta B Shepherd which provides physiotherapists and occupational therapists with an approach to stroke rehabilitation that is clear, relevant, and effective, building on the research-based model created by the authors
Stroke (cerebrovascular accident [CVA]) is the sudden loss of neurological function caused by an interruption of the blood flow to the brain.
Impairments may resolve spontaneously as brain swelling subsides (reversible ischemic neurological deficit), generally within 3 weeks.
Residual neurological impairments are those that persist
longer than 3 weeks and may lead to lasting disability
Physiotherapy management of spasticity using diffrent modalities as well as manual techniques is described along with possible dosage ijn clinical use is also menstined.
The Bobath concept is a problem-solving approach used in the evaluation and treatment of individuals with movement and postural control disturbances due to a lesion of the central nervous system.
It is named after Berta Bobath, a physiotherapist, and her husband Karel Bobath, a psychiatrist/neuropsychiatrist, who proposed the approach for treating patients affected with Central Nervous System anomalies.
Procedure: in a “trial & error” fashion in 1948.
Concept of compensatory training.
Neglects the potential of hemiplegic side.
It is an interactive problem-solving approach that focuses on continuing reassessment with attention to individual goals, developing working hypotheses, treatment plans, and relevant objective measures to evaluate interventions.
Therapist should have:
Good posture & movement analysis skills.
PRINCIPLES
NDT THERAPY WORKS
ALWAYS TREAT THE PATIENT AS A WHOLE
WORK SIMULTANEOUSLY ON PATIENTS STRENGTHS & WEAKNESSES
INDIVIDUALIZED FOR EVERY PATIENT BASED ON ICF MODEL
GAIN THE INFORMATION FROM PAST, PRESENT & FUTURE
TEAMWORK IS CRITICAL FOR REHAB PURPOSES
UNDERSTANDING THE CONCEPT OF TYPICAL DEVELOPMENT (MOTOR CONTROL)
TRANSFERENCE OF TRAINING IN DAILY LIFE
HANDS ON INTERVENTION TO ENHANCE MOTOR LEARNING & FUNCTIONS
This presentation contains detailed knowledge about Down's Syndrome its types, clinical presentation, diagnosis, medical and physio therapeutic management of the condition.
Down syndrome is a condition in which a person has an extra chromosome. Chromosomes are small “packages” of genes in the body. They determine how a baby’s body forms and functions as it grows during pregnancy and after birth. Typically, a baby is born with 46 chromosomes. Babies with Down syndrome have an extra copy of one of these chromosomes, chromosome 21. A medical term for having an extra copy of a chromosome is ‘trisomy.’ Down syndrome is also referred to as Trisomy 21. This extra copy changes how the baby’s body and brain develop, which can cause both mental and physical challenges for the baby.
Important structures associated with neural control of locomotion- CPGs, Peripheral receptors and afferents, Basal ganglia, Cerebellum, Brainstem, Cerebellar Cortex.
Physiotherapy in MND
Dr. Quazi Ibtesaam Huma (MPT)
Dr. Suvarna Ganvir (Phd, Prof & HOD)
Dept. of Neurophysiotherapy
DVVPF’s College of Physiotherapy
Content
Introduction
Types of MND
Clinical Features of MND
Diagnostic Procedure
Management: 1) Pharmaceutical
2) Physiotherapy
Motor Neuron Disease
Motor Neuron Disease are a group of neurodegenerative disorders that affects the nerves in the spine and brain to progressively lose its function.
Motor neuron diseases (MND) include a heterogeneous spectrum of inherited and sporadic (no family history) clinical disorders of the upper motor neurons (UMNs), lower motor neurons (LMNs), or a combination of both.
Types of MND
Amyotrophic Lateral Sclerosis
Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disease, characterized by progressive degeneration of motor neurons in the spinal cord, brain stem, and motor cortex, leading to progressive muscle atrophy and weakness.
Clinical Features
UPPER MOTOR NEURON
Loss of Dexterity
Muscle Weakness
Spasticity
Hyperreflexia
Pathological reflexes
LOWER MOTOR NEURON
Muscle Weakness
Muscle Atrophy
Hypotonicity
Hyporeflexia
Fasciculation
Muscle Cramp
Impairment related to LMN
Other clinical features
Diagnostic Criteria
Diagnostic Procedure
EMG-
It include signs of active denervation, such as fibrillation potentials and positive sharp waves;
Signs of chronic denervation, such as large motor unit potentials (increased duration, increased proportion of polyphasic potentials, increased amplitude)
Unstable motor unit potential
Nerve Conduction Velocity Studies,
Muscle And Nerve Biopsies,
Neuroimaging Studies - MRI
Management- Multidisciplinary Approach
Physical Therapy Examination
Cognition
Pain
Psychosocial Function
Joint integrity, ROM and Muscle strength.
Motor Function: Gross motor and Fine motor
Muscle tone and reflexes
Cranial nerve integrity
Sensations
Gait
Respiratory Function
Physiotherapy goals in MND treatment.
Pain reduction
Prevention for contractures
Maintenance of joint mobility
Regular review of posture
Positioning to relieve discomfort
House Modification and ergonomic advice.
Management of Sialorrhea and Pseudobulbar Affect
Management for Dysphagia
PEG procedure.
A PEG may be recommended as the disease progresses.
A PEG is a type of gastrostomy tube inserted via endoscopic surgery that creates a permanent opening into the stomach for the introduction of food.
Studies have found that PEG insertion may prolong survival. Patients with PEG were found to live 1 to 4 months longer than those individuals who refused it.
Management of Dysphagia
A palatal lift prosthesis may be prescribed for individuals with good articulation but who have a breathy voice quality or decreased loudness because of excessive air loss through the nose.
The device, a dental appliance designed to attach to the existing teeth and to elevate the soft palate, is custom-made by a prosthodontist.
Stroke (cerebrovascular accident [CVA]) is the sudden loss of neurological function caused by an interruption of the blood flow to the brain.
Impairments may resolve spontaneously as brain swelling subsides (reversible ischemic neurological deficit), generally within 3 weeks.
Residual neurological impairments are those that persist
longer than 3 weeks and may lead to lasting disability
Physiotherapy management of spasticity using diffrent modalities as well as manual techniques is described along with possible dosage ijn clinical use is also menstined.
The Bobath concept is a problem-solving approach used in the evaluation and treatment of individuals with movement and postural control disturbances due to a lesion of the central nervous system.
It is named after Berta Bobath, a physiotherapist, and her husband Karel Bobath, a psychiatrist/neuropsychiatrist, who proposed the approach for treating patients affected with Central Nervous System anomalies.
Procedure: in a “trial & error” fashion in 1948.
Concept of compensatory training.
Neglects the potential of hemiplegic side.
It is an interactive problem-solving approach that focuses on continuing reassessment with attention to individual goals, developing working hypotheses, treatment plans, and relevant objective measures to evaluate interventions.
Therapist should have:
Good posture & movement analysis skills.
PRINCIPLES
NDT THERAPY WORKS
ALWAYS TREAT THE PATIENT AS A WHOLE
WORK SIMULTANEOUSLY ON PATIENTS STRENGTHS & WEAKNESSES
INDIVIDUALIZED FOR EVERY PATIENT BASED ON ICF MODEL
GAIN THE INFORMATION FROM PAST, PRESENT & FUTURE
TEAMWORK IS CRITICAL FOR REHAB PURPOSES
UNDERSTANDING THE CONCEPT OF TYPICAL DEVELOPMENT (MOTOR CONTROL)
TRANSFERENCE OF TRAINING IN DAILY LIFE
HANDS ON INTERVENTION TO ENHANCE MOTOR LEARNING & FUNCTIONS
This presentation contains detailed knowledge about Down's Syndrome its types, clinical presentation, diagnosis, medical and physio therapeutic management of the condition.
Down syndrome is a condition in which a person has an extra chromosome. Chromosomes are small “packages” of genes in the body. They determine how a baby’s body forms and functions as it grows during pregnancy and after birth. Typically, a baby is born with 46 chromosomes. Babies with Down syndrome have an extra copy of one of these chromosomes, chromosome 21. A medical term for having an extra copy of a chromosome is ‘trisomy.’ Down syndrome is also referred to as Trisomy 21. This extra copy changes how the baby’s body and brain develop, which can cause both mental and physical challenges for the baby.
Important structures associated with neural control of locomotion- CPGs, Peripheral receptors and afferents, Basal ganglia, Cerebellum, Brainstem, Cerebellar Cortex.
Physiotherapy in MND
Dr. Quazi Ibtesaam Huma (MPT)
Dr. Suvarna Ganvir (Phd, Prof & HOD)
Dept. of Neurophysiotherapy
DVVPF’s College of Physiotherapy
Content
Introduction
Types of MND
Clinical Features of MND
Diagnostic Procedure
Management: 1) Pharmaceutical
2) Physiotherapy
Motor Neuron Disease
Motor Neuron Disease are a group of neurodegenerative disorders that affects the nerves in the spine and brain to progressively lose its function.
Motor neuron diseases (MND) include a heterogeneous spectrum of inherited and sporadic (no family history) clinical disorders of the upper motor neurons (UMNs), lower motor neurons (LMNs), or a combination of both.
Types of MND
Amyotrophic Lateral Sclerosis
Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disease, characterized by progressive degeneration of motor neurons in the spinal cord, brain stem, and motor cortex, leading to progressive muscle atrophy and weakness.
Clinical Features
UPPER MOTOR NEURON
Loss of Dexterity
Muscle Weakness
Spasticity
Hyperreflexia
Pathological reflexes
LOWER MOTOR NEURON
Muscle Weakness
Muscle Atrophy
Hypotonicity
Hyporeflexia
Fasciculation
Muscle Cramp
Impairment related to LMN
Other clinical features
Diagnostic Criteria
Diagnostic Procedure
EMG-
It include signs of active denervation, such as fibrillation potentials and positive sharp waves;
Signs of chronic denervation, such as large motor unit potentials (increased duration, increased proportion of polyphasic potentials, increased amplitude)
Unstable motor unit potential
Nerve Conduction Velocity Studies,
Muscle And Nerve Biopsies,
Neuroimaging Studies - MRI
Management- Multidisciplinary Approach
Physical Therapy Examination
Cognition
Pain
Psychosocial Function
Joint integrity, ROM and Muscle strength.
Motor Function: Gross motor and Fine motor
Muscle tone and reflexes
Cranial nerve integrity
Sensations
Gait
Respiratory Function
Physiotherapy goals in MND treatment.
Pain reduction
Prevention for contractures
Maintenance of joint mobility
Regular review of posture
Positioning to relieve discomfort
House Modification and ergonomic advice.
Management of Sialorrhea and Pseudobulbar Affect
Management for Dysphagia
PEG procedure.
A PEG may be recommended as the disease progresses.
A PEG is a type of gastrostomy tube inserted via endoscopic surgery that creates a permanent opening into the stomach for the introduction of food.
Studies have found that PEG insertion may prolong survival. Patients with PEG were found to live 1 to 4 months longer than those individuals who refused it.
Management of Dysphagia
A palatal lift prosthesis may be prescribed for individuals with good articulation but who have a breathy voice quality or decreased loudness because of excessive air loss through the nose.
The device, a dental appliance designed to attach to the existing teeth and to elevate the soft palate, is custom-made by a prosthodontist.
Dr Nikhil Nadkarni
MD ,DM (PGI ,Chandigarh)
Advanced fellowship Gastroenterology (Mayo Clinic ,USA)
Associate Director
Department of Gastroenterology & Hepatology
Max Super specialty Hospital
Mohali,Punjab
Dr Kishore Kumar Ubrangala, MD
Professor, Dept. of Medicine,
Yenepoya Medical College,
Yenepoya (Deemed to be) University, Mangalore, India.
sankish@gmail.com
Heamocon 2020 . Lecture by Dr Prashant at Yenepoya Medical college Mangalor...YMC Medicine
DR Prashant presented on 8th March at Yenepoya Medical college
Click to connect with the author
https://www.linkedin.com/in/prashanth-balanthimogru-a47ab438
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
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Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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Stroke rehabilitation approach
1. PRESENTER :- Dr. Abhinav Kinagi
MODERATOR :- Dr. Kishore Kumar Ubrangala
27/05/2020
2. Stroke burden in INDIA
Stroke Rehabilitation – Definition, Team
Phase of Rehabilitation, their focus & setting
Concept of Neuroplasticity
Approaches
Common Post-stroke impairments & their
management
Life style changes after stroke
Employment & Return to work
Driving after stroke
Take Home Message
3. 4th leading cause of death (ICMR 2016)
5th leading cause of Disability Adjusted Life Years
(DALY) in India in 2016
Responsible for 3.5% of DALY in India
Crude Prevalence Rate :- 116-163/1,00,000
persons (2016)
More than half will have significant deficits in
mobility, cognition, and their ability to perform
activities of daily living (ADLs)
4.
5. After a stroke, you may have to change or re-
learn how you live day to day.
Getting quality rehab from a strong team of
therapists leads to better recovery
The goal of rehab is to become as independent
as possible.
6. Particular emphasis will be on:
the recovery of function (performance)
prevention of secondary complications
reduction of caregiver burden, and
improvement of overall quality of life.
Physical medicine and rehabilitation or
Physiatry
7. Rehabilitation is a team effort. This team
communicates about and coordinates the
care to help achieve your goals
Physician and Neurologist
Physiatrist
Physical therapist (PT)
10. Rehab programs focus on assessing and
improving:
ADLs
Mobility
Communication skills
Cognitive skills
Social skills
Psychological functioning
11. Once thought that there was no regenerative
ability in the brain or spinal cord – limiting
potential for functional recovery.
NEUROPLASTICITY
– resulting form active exercise
12. Complementary strategies of approaches to
stroke recovery:
Compensatory
Restorative (Remedial, Therapeutic)
13. Finding a new way to perform and complete a
task
Using of a cane in the setting of hemiparesis
resulting in safer walking.
Installing a ramp in lieu of stairs.
Learning to use the left arm to complete
tasks in the setting of right hemiparesis.
14. Attempt to develop new or repair damaged
neural networks through active exercise
Ex: Constraint-induced movement therapy
Both approaches used simultaneously:
The rehabilitation team chooses the
appropriate therapies to enhance
performance and facilitate recovery.
16. As soon as the patient is medically stable and
capable
Multidisciplinary rehabilitation team
17. Early mobilization is defined as ‘trained
rehabilitation therapists and nursing staff
assisting the patient with task-specific
activities’.
Early and regular mobilization in short
sessions within 24 hours of diagnosis of
stroke predicted better outcomes at 3
months.
18. Coma at onset of stroke
Urinary incontinence
Poor cognitive function
Severe hemiplegia
Perceptual and spatial disorders
Depression
19. An independent level of function before
stroke is a positive factor, and full
assessment of premorbid function is
recommended.
20. Use to reduce neural inflammation, modify
synaptic function, and restore cortical
network balance.
Dopamine and catecholamine agonists &
anti-cholinergic antagonist
Selective serotonin reuptake inhibitor (SSRI).
(FLAME) trial
21. To facilitate recovery, minimize
complications, and maximize function
following stroke.
The most common settings are: an inpatient
rehabilitation facility, skilled nursing facility,
long-term acute care hospital, home-based
services, and outpatient services.
Home health care agency (HHCAs)
22.
23. Neurogenic bladder and bowel dysfunction
are defined as any alteration in the
controlled, predictable, and socially
acceptable elimination of bodily waste after
neurologic injury.
24.
25.
26. 2nd leading cause of impaired cognition and
dementia
30% of stroke survivors develop dementia
within 1 year of the onset of their stroke
Even minor strokes may affect cognition and
executive functions,(TIA) transient ischemic
attacks also may be associated with impaired
cognition
27. The areas most affected: memory, language &
communication, orientation, attention, and
executive function
Impact the patient’s ability to perform ADLs
Younger stroke survivors - will often affect
their chance of returning to work (RTW)
28. Evaluation by physiatry and neuropsychology,
and is followed by treatment by speech
language pathologist (SLP) and occupational
therapy (OT)
Major goal: Improving the impairments (such
as retraining memory function), and/or
establish new patterns of cognitive activity
A secondary goal: provide compensation
strategies to cope with the disabling impact of
the impairment.
29.
30.
31. Aphasia is a disorder of language resulting from
damage to the language-dominant hemisphere of
the brain (usually the left side)
Non-fluent aphasia
Fluent aphasia
Treatment by the SLP
32. Include Apraxia of speech and Dysarthria.
Apraxia - (an impairment in “motor
planning”)
Dysarthria - 20% to 30% - Articulation deficit
Treatment by SLP
33. Visuo-spatial neglect
43% of patients - right hemispheric damage
17% - left hemispheric damage
Encouragingly, neglect following stroke
usually improves within a few weeks after
onset.
34. Persons with stroke who have
poor balance,
low balance confidence,
fear of falls and/or are at risk for falls
should be provided with a balance-training
program
35. Eye exercises for treatment of convergence
insufficiency are recommended
36. Affects 25% to 65% of patients
Results from abnormal functioning of the
muscles of the mouth, pharynx and upper
esophageal sphincter
37. Food and/or drink exiting the patient’s
nose
Oxygen desaturation
Persistent cough
Change in voice quality during meals
38. Pocketing
Earliest - Sensation of food being stuck in
the throat
Silent Aspiration
40. History taking
Video fluoroscopic swallow study, also known as the
modified barium swallow (MBS) study
SLP
Fiberoptic endoscopic evaluation of swallowing test
Treatment strategies: strengthening exercises,
compensatory strategies during swallowing, and
changes in food type, amount, and consistency.
Supervision during meals
41. Stroke survivors may experience pain resulting
from
central &
peripheral mechanisms
as well as - psychological factors
43. Chronic neuropathic disorder typically experienced
within the first month after stroke but ranging from 1
week to 10 years
Prevalence - 7% to 35%
Pain - intermittent or constant, with sensory
abnormalities, and is described as burning, aching, icy,
pricking, and lacerating
Pharmacologic agents : TCA, SSRIs, Membrane
stabilizing agents such as gabapentin and pregabalin,
AED, Corticosteroids, and Opioids.
Nonpharmacologic approach : Prescription of exercise
therapy to improve strength, flexibility, and function.
44. Shoulder pain on the weak limb in a person
with hemiplegic stroke.
It is present in 25% of stroke survivors.
46. Pain can develop as early as 2 weeks after
stroke but is more commonly seen at 2 to 3
months.
Treatment: Effective pain control with similar
agents used to treat CPSP, subacromial
steroid injection, physical modalities, and a
program of rehabilitation.
47. Resting hand/wrist splints along with regular
stretching and spasticity management
Resting ankle splints used at night and during
assisted standing may be considered for
prevention of ankle contracture in a
hemiplegic limb.
48. Prophylactic-dose subcutaneous heparin
(unfractionated heparin or low-molecular-
weight heparin) should be used for the duration
of the
acute and rehabilitation hospital stay or
until the stroke survivor regains mobility.
52. Feelings of sadness
Hopelessness or helplessness
Irritability
Changes in eating, sleeping and thinking
53. Pseudobulbar affect, also called “emotional
lability,”
“reflex crying” or “labile mood,”
can cause :-
Rapid mood changes — a person may “spill
over into tears” for no obvious reason and
then quickly stop crying or start laughing.
Crying or laughing that doesn’t match a
person’s mood.
Crying or laughing at unusual times or that
lasts longer than seems appropriate.
54.
55. Feeling of exhaustion and a lack of energy and
effort
Frequency :- 29% to 77%
May inhibit participation and progress in
rehabilitation
The symptom often develops after physical or
mental activity and usually improves with rest
57. Frequency :- 30%
Commonly found in the shoulder, flexor muscles
of the upper arm (elbow, wrist, and fingers), and
extensor muscles of the lower limb (knee and
ankle)
Complications :- pain, contracture, worsened
mobility, poor quality of life, and increased
caregiver burden
Early detection and management of spasticity
may help mitigate these complications.
58. PT or OT
Stretching modalities
Oral antispasm agents (Baclofen, Tizanidine,
and Dantrolene)
Physiatric management
True joint contracture needs a surgical
consultation.
59. Frequency :- 57% to 75%
Common complaints - decline in libido,
decrease in coital frequency, reduction in
vaginal lubrication and orgasm in women, and
poor or failed erection and ejaculation in men.
Pharmacologic management
Counseling by the multidisciplinary team
60.
61. Don’t smoke and avoid second-hand smoke.
Improve your eating habits
(Eat foods low in saturated fat, trans fat, sodium
and added sugars)
Be physically active
Take your medicine as directed
Get your blood pressure checked regularly
Reach and maintain a healthy weight
Decrease your stress level
Seek emotional support when it’s needed
Have regular medical checkup
62.
63. Stroke in the young
To help determine if the stroke survivor is
able to safely RTW, a referral may be made to
OT or PT for a functional capacity evaluation.
64.
65. Limitations :- motor, visual, and cognitive
impairments
State regulations
Specialized centers where PT or OT assist
with screening and driver evaluation
Neuropsychology evaluation
66.
67.
68.
69.
70.
71.
72.
73.
74.
75.
76.
77.
78.
79.
80.
81.
82.
83.
84. There is life – and hope – after Stroke.
With time, new routines will become second
nature.
Rehabilitation can build patient’s strength,
capability and confidence.
It can help the patient to continue his/her
daily activities despite the effects of Stroke
85. Team approach
Evidence based practice
Neuroplasticity and motor learning principle
Early mobilisation
Aerobic training
Comprehensive attention to all post-stroke
impairments
86. Leroy R Lindsay, Diane A T, Michel W O. Updated Approach to Stroke
Rehabilitation. Med Clin N Am 2020;
https://www.stroke.org/ (American Stroke Association)
Guidelines for Prevention & Management of Stroke, National Programme
for Prevention and Control of Cancer, Diabetes, Cardiovascular Diseases
& Stroke (NPCDCS) - Directorate General of Health Services Ministry of
Health and Family Welfare – GOI 2019
https://www.heart.org/ (American Heart Association )
Harrisons Principles of Internal Medicine - 20th edition