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By:-
Harsh Jain
(II year-B.OPTOM.)
TELANGIECTASIA
 It is a condition in which tiny blood vessels cause
thread like red lines or patterns on the skin.
 They are sometimes known as spider veins.
 Diameter 0.52mm to 1.00mm
Cause :
 The exact cause is unknown .
It may be genetic , environmental or combination
of both. It is believed that most causes of
telangiectasia are caused by chronic exposure to
sunlight.
Other possible causes :
Alcoholism: can affect flow of blood in vessels.
Pregnancy: due to large pressure on venules.
Aging : aging blood vessels can begin to weak.
Rosacea: enlarges venules in face, creating flushed
appearance
in nose and cheeks
corticosteroid use: thins and weakens the skin
Scleroderma: hardens and contracts the skin
Symptoms :
1. Pain(related to pressure on venules .)
2. Itching
3. Thread like red marks on skin
TREATMENT
 Laser therapy: Laser target the widened vessels and
seals it.
 Surgery: widen vessels can be removed.
 Sclerotherapy.
 It refers to the rare idiopathic congenital or acquired
retinal vascular malformation.
 It is characterized by the irregular dilation of capillary
bed ,deposition of hard exudates and dilation of
venules and arterioles.
It includes :-
Idiopathic juxtafoveolar retinal telangiectasia.
Coat’s disease.
Leber’s military aneurysm.
Idiopathic juxtafoveolar retinal telangiectasia.
 It is also known as idiopathic macular telangiectasia.
 It rare , congenital or acquired .
 It is a rare condition presenting with mild decrease in
visual acuity due to exudation from the juxtafoveolar
telangitatic retinal capillaries.
 This condition has been divided into three groups 1, 2
and 3 and it is subdivided into A and B depending on
the characteristics of the lesion.
 GROUP ONE : 1 A
Congenital , unilateral, easily visible and macular oedema.
Mild moderate blurring of vision .
About 1.5 disc diameters in area temporal to fovea and
hard exudates . ( 14.112a, b)
FFA shows capillary dilation ( 14.112c ) and late leakage
(14.112 d )
Treatment : laser photocoagulation.
GROUP ONE : 1 B
Affects middle aged men .
FFA shows absence of leakage .( 14.113 c )
TREATMENT : Not appropriate and prognosis is
good.
GROUP TWO : Bilateral parafoveolar
telangiectasia
 Present in the fifth to sixth decades with mild .
 Slowly progressive disturbance of central vision.
 SIGNS:
 Telangiectasia ,stellate plaques of RPE hyperplasia.
 Multipal glistening dots are present.
FFA : shows capillary dilation outside FAZ. ( 14.114A ).
Late leakage ( 14.114b ).
 Most severe form typically present In sixth decade .
 Very rare , acquired .
 Slowly progressive loss of central vision
SIGNS : Aneurysmal dilatation of terminal capillaries and
parafoveal capillaries.
 Optic atrophy may be present.
FFA shows widening of FAZ but absence of leakage .
 Prognosis usually poor.
GROUP THREE : BILATRAL PERIFOVEAL
TELANGIECTASI AND CAPILLARY OCCLUSION :
Coat’s disease
 It is also known as exudative retinopathy of coat’s.
 It is severe from retinal telangiectasia.
Characteristics of coat’s disease:-
 Typically affects one eye of boys in their first decade of
life.( 5 yrs )
 In early stages it is characterized by large areas of intra (
14.120 ) and sub(14.121 a) retinal yellowish exudates and
hemorrhages associated with overlying dilated and retinal
blood vessels and number of small aneurysm near the
posterior pole around the disc.
 It may present with visual loss , strabismus , leukocoria . (
14.118)
 Conditions usually progresses to produce exudative retinal
detachment and retro lantal mass .
• In late stages of complicated cataract ,uveitis and secondary
glaucoma occur which eventually end in pthisis bulbi.
 FFA highlights abnormal vessels , leakage and areas of
capillary dropout.
• Aneurysm formation and hard exudates . ( 14. 121 b )
TREATMENT
Photocoagualation :
 Cryotherapy : with double freeze thaw method .
 It may check progression of the disease if applied in
early stage.
 However, when the retinal detach the treatment
became increasingly difficult and success rate
declines to 33%.
Vitreoretinal surgery : in eyes with deep retinal
detachment .
Leber’s miliiary aneurysm.
 It is essentially localize less severe form of the coat’s
disease presenting in adults with decrease vision.
 It is characterised by local area of fusiform and
saccular aneurysmic (small pouches )dilation of
venules , most common involving the temporal retinal
periphery( 14.115)
 Chronic leakage results in intraretinal hard exuadate
formation which may involve the macula .
FFA during the early phase highlights the vascular
anomalies and show areas of retinal non perfusion.
• The late phase of FFA shows leakage ( 14.116 c )
 Prognosis is depends upon extent of foveal
involvement ( 14.117 a )
Treatment : photocoagulation ( 14.117 b )
Primary Retinal Telangiectasia
Primary Retinal Telangiectasia
Primary Retinal Telangiectasia

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Primary Retinal Telangiectasia

  • 2. TELANGIECTASIA  It is a condition in which tiny blood vessels cause thread like red lines or patterns on the skin.  They are sometimes known as spider veins.  Diameter 0.52mm to 1.00mm
  • 3. Cause :  The exact cause is unknown . It may be genetic , environmental or combination of both. It is believed that most causes of telangiectasia are caused by chronic exposure to sunlight.
  • 4. Other possible causes : Alcoholism: can affect flow of blood in vessels. Pregnancy: due to large pressure on venules. Aging : aging blood vessels can begin to weak. Rosacea: enlarges venules in face, creating flushed appearance in nose and cheeks corticosteroid use: thins and weakens the skin Scleroderma: hardens and contracts the skin
  • 5. Symptoms : 1. Pain(related to pressure on venules .) 2. Itching 3. Thread like red marks on skin
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  • 7. TREATMENT  Laser therapy: Laser target the widened vessels and seals it.  Surgery: widen vessels can be removed.  Sclerotherapy.
  • 8.  It refers to the rare idiopathic congenital or acquired retinal vascular malformation.  It is characterized by the irregular dilation of capillary bed ,deposition of hard exudates and dilation of venules and arterioles.
  • 9. It includes :- Idiopathic juxtafoveolar retinal telangiectasia. Coat’s disease. Leber’s military aneurysm.
  • 10. Idiopathic juxtafoveolar retinal telangiectasia.  It is also known as idiopathic macular telangiectasia.  It rare , congenital or acquired .  It is a rare condition presenting with mild decrease in visual acuity due to exudation from the juxtafoveolar telangitatic retinal capillaries.  This condition has been divided into three groups 1, 2 and 3 and it is subdivided into A and B depending on the characteristics of the lesion.
  • 11.  GROUP ONE : 1 A Congenital , unilateral, easily visible and macular oedema. Mild moderate blurring of vision . About 1.5 disc diameters in area temporal to fovea and hard exudates . ( 14.112a, b) FFA shows capillary dilation ( 14.112c ) and late leakage (14.112 d ) Treatment : laser photocoagulation.
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  • 13. GROUP ONE : 1 B Affects middle aged men . FFA shows absence of leakage .( 14.113 c ) TREATMENT : Not appropriate and prognosis is good.
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  • 15. GROUP TWO : Bilateral parafoveolar telangiectasia  Present in the fifth to sixth decades with mild .  Slowly progressive disturbance of central vision.  SIGNS:  Telangiectasia ,stellate plaques of RPE hyperplasia.  Multipal glistening dots are present. FFA : shows capillary dilation outside FAZ. ( 14.114A ). Late leakage ( 14.114b ).
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  • 17.  Most severe form typically present In sixth decade .  Very rare , acquired .  Slowly progressive loss of central vision SIGNS : Aneurysmal dilatation of terminal capillaries and parafoveal capillaries.  Optic atrophy may be present. FFA shows widening of FAZ but absence of leakage .  Prognosis usually poor. GROUP THREE : BILATRAL PERIFOVEAL TELANGIECTASI AND CAPILLARY OCCLUSION :
  • 18. Coat’s disease  It is also known as exudative retinopathy of coat’s.  It is severe from retinal telangiectasia.
  • 19. Characteristics of coat’s disease:-  Typically affects one eye of boys in their first decade of life.( 5 yrs )  In early stages it is characterized by large areas of intra ( 14.120 ) and sub(14.121 a) retinal yellowish exudates and hemorrhages associated with overlying dilated and retinal blood vessels and number of small aneurysm near the posterior pole around the disc.  It may present with visual loss , strabismus , leukocoria . ( 14.118)  Conditions usually progresses to produce exudative retinal detachment and retro lantal mass .
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  • 23. • In late stages of complicated cataract ,uveitis and secondary glaucoma occur which eventually end in pthisis bulbi.  FFA highlights abnormal vessels , leakage and areas of capillary dropout. • Aneurysm formation and hard exudates . ( 14. 121 b )
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  • 25. TREATMENT Photocoagualation :  Cryotherapy : with double freeze thaw method .  It may check progression of the disease if applied in early stage.  However, when the retinal detach the treatment became increasingly difficult and success rate declines to 33%. Vitreoretinal surgery : in eyes with deep retinal detachment .
  • 26. Leber’s miliiary aneurysm.  It is essentially localize less severe form of the coat’s disease presenting in adults with decrease vision.  It is characterised by local area of fusiform and saccular aneurysmic (small pouches )dilation of venules , most common involving the temporal retinal periphery( 14.115)  Chronic leakage results in intraretinal hard exuadate formation which may involve the macula .
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  • 28. FFA during the early phase highlights the vascular anomalies and show areas of retinal non perfusion. • The late phase of FFA shows leakage ( 14.116 c )  Prognosis is depends upon extent of foveal involvement ( 14.117 a ) Treatment : photocoagulation ( 14.117 b )