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Sulaimanyia Teaching Hospital
ENT CENTER
ORBITAL COMPLICATIONS OF
ACUTE RHINOSINUSITIS
Presented by:- Dr . Sahar j .Hadi
ENT Center- Sulaimanyia Teaching Hospital- Sulaimanyia- Iraq
CONTENTS
Introduction
Epidemiology
Classification of orbital complications.
Pathogenesis.
Management.
 Medical And Surgical Treatment.
INTRODUCTION
Acute bacterial sinusitis occurs commonly,
usually as a sequel of an upper respiratory
infection. Spread of infection outside the
sinuses results in complicated sinusitis. The
incidence of complications from both acute
and chronic sinusitis has decreased four fold
due to the widespread use of antibiotics for
rhinosinusitis.
COMPLICATION
CHRONIC
DISTANTLOCAL
ACUTE
ANATOMICAL CONSIDRATIONS
Muscular and periorbita
RISK FACTORS
 Seasonal variations.
Gender: Male
 Age increase admission due to complication
(3-6 yeas).
Above 7years intracranial complication.
Adult intracranial complication due to CRS,
while intracranial complications in child due to
ABRS.
 Underdeveloped sinuses, highly vascular
valveless diploic system, immature posterior
table, arachnoid layer.
Pathogenesis
• Through the wall of sinusitisDirect
spread
• Through the subperosteal
venous plexus
Venous
spread
• Perivascular lymphatic spread
to subperiosteal plane
Lymphatic
spread
CLASSIFICATION: according to Chandlers 1970
1-PRESEPTAL CELLULITIS
-complication of ethmoid sinusitis .
-Far most common in pediatric age group.
-Exclude the other causes
-medical comorbidities and immune
deficiencies are risk factors for preseptal
cellulitis.
-Manifests as eyelid swelling, erythema, and
tenderness.
-There are no limitation of extra ocular muscles
movements. and no impairment of visual
acuity.
- Due to impaired venous drainage of the
ethmoidal vessels that are obstructed by
inflammation and pressure.
ORBITAL CELLULITIS
Orbital cellulitis is a postseptal infection .
manifests as diffuse edema of the orbital contents
without a discrete abscess .
There is eyelid edema and erythema, proptosis, and
chemosis with limited or no impairment of
extraocular movements and normal visual acuity
early in the disease process.
Visual changes and ophthalmoplegia indicating
optic neuritis and/or ischemia can occur as the
disease progresses, these are prognostically
worrisome finding.
SUBPERIOSTEAL ABSCESS
 Collection of pus forms at the medial aspect of the
orbit between the periorbita and the lamina
papyracea .
 This is the second most common orbital
complication of sinusitis .
 Sever proptosis, ophthalmoplegia.
 A subperiosteal abscess can displace the orbital
contents and globe downward and laterally with
normal mobility in the early stages.
 An abscess may occasionally rupture the orbital
septum and present in the eyelids.
Orbital abscess
 An orbital abscess occurs when orbital cellulitis
coalesces into a discrete collection of pus
within the orbital tissues.
 This is a serious complication that can be
associated with severe exophthalmos and
chemosis, complete ophthalmoplegia and
visual impairment with a risk for progression to
irreversible blindness.
 On rare occasions there is spontaneous
drainage of purulent material through the
eyelid.
CAVERNOUS SINUS
THROMBOSIS
 can be considered an orbital as well as an
intracranial complication of sinusitis.
 Venous congestion in the orbit results in
bilateral orbital pain, chemosis, proptosis, and
ophthalmoplegia. Cranial nerves III, IV; Vl, V2,
V3, and VI traverse the sinus and can all be
affected.
 Extension of the phlebitis posteriorly into the
cavernous sinus results in progression of
symptoms in the opposite eye.
 It can be associated with sepsis and
meningismus or frank meningitis may be
present.
EVALUTIONS
 HISTORY(Symptoms of URTI + symptoms of ABRS).
 PHYSICAL EXAMINATION.(nasal cavity , orbital EX).
 RADIOLOGY.
Computed tomography (Ct) is considered the gold standard for
orbital complications of ABRS (axial + coronal view).
Aim:
1-To confirm the diagnosis.
2-Define the extent and site of disease.
3-give idea about the treatment modalities.
4-exclude other complications.
INDICATIONS:
1-In preseptal inflammation progress in 24-48hr
despite medical treatment.
2- suspected post septal complications.
3-exclude intracranial abscess.
TREATMENT: (all need admission , antibiotic)
>3 years oral
Borad
spectrum
antibiotic
+conservative
treatment
IV antibiotic
+- sinus
drainage
IV antibiotic
+sinus
drainage
+abscess
drainage
IV antibiotic
+- sinus
drainage
+- abscess
drainage
INDICATIONS OF SURGICAL
INTERVENTIONS
INDICATIONS OF MEDICAL TREATMENT
IN MEDIALSUBPERIOSTEAL ABSCESS
Abscess width<4mm
Surgical Treatment
Orbital Decompression
Indications Orbital
Cellulitis/Abscess
Retro Orbital
Hemorrhage
Graves Orbitopathy
Complications
Diplopia
Impair vision
Post.op/epistaxis
Post/op sinusitis
vedio
THANK YOU

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Orbital complications of acute Rhinosinusitis

  • 1. Sulaimanyia Teaching Hospital ENT CENTER ORBITAL COMPLICATIONS OF ACUTE RHINOSINUSITIS Presented by:- Dr . Sahar j .Hadi ENT Center- Sulaimanyia Teaching Hospital- Sulaimanyia- Iraq
  • 2. CONTENTS Introduction Epidemiology Classification of orbital complications. Pathogenesis. Management.  Medical And Surgical Treatment.
  • 3. INTRODUCTION Acute bacterial sinusitis occurs commonly, usually as a sequel of an upper respiratory infection. Spread of infection outside the sinuses results in complicated sinusitis. The incidence of complications from both acute and chronic sinusitis has decreased four fold due to the widespread use of antibiotics for rhinosinusitis.
  • 6.
  • 8. RISK FACTORS  Seasonal variations. Gender: Male  Age increase admission due to complication (3-6 yeas). Above 7years intracranial complication. Adult intracranial complication due to CRS, while intracranial complications in child due to ABRS.  Underdeveloped sinuses, highly vascular valveless diploic system, immature posterior table, arachnoid layer.
  • 9. Pathogenesis • Through the wall of sinusitisDirect spread • Through the subperosteal venous plexus Venous spread • Perivascular lymphatic spread to subperiosteal plane Lymphatic spread
  • 11.
  • 12. 1-PRESEPTAL CELLULITIS -complication of ethmoid sinusitis . -Far most common in pediatric age group. -Exclude the other causes -medical comorbidities and immune deficiencies are risk factors for preseptal cellulitis. -Manifests as eyelid swelling, erythema, and tenderness. -There are no limitation of extra ocular muscles movements. and no impairment of visual acuity. - Due to impaired venous drainage of the ethmoidal vessels that are obstructed by inflammation and pressure.
  • 13. ORBITAL CELLULITIS Orbital cellulitis is a postseptal infection . manifests as diffuse edema of the orbital contents without a discrete abscess . There is eyelid edema and erythema, proptosis, and chemosis with limited or no impairment of extraocular movements and normal visual acuity early in the disease process. Visual changes and ophthalmoplegia indicating optic neuritis and/or ischemia can occur as the disease progresses, these are prognostically worrisome finding.
  • 14. SUBPERIOSTEAL ABSCESS  Collection of pus forms at the medial aspect of the orbit between the periorbita and the lamina papyracea .  This is the second most common orbital complication of sinusitis .  Sever proptosis, ophthalmoplegia.  A subperiosteal abscess can displace the orbital contents and globe downward and laterally with normal mobility in the early stages.  An abscess may occasionally rupture the orbital septum and present in the eyelids.
  • 15. Orbital abscess  An orbital abscess occurs when orbital cellulitis coalesces into a discrete collection of pus within the orbital tissues.  This is a serious complication that can be associated with severe exophthalmos and chemosis, complete ophthalmoplegia and visual impairment with a risk for progression to irreversible blindness.  On rare occasions there is spontaneous drainage of purulent material through the eyelid.
  • 16. CAVERNOUS SINUS THROMBOSIS  can be considered an orbital as well as an intracranial complication of sinusitis.  Venous congestion in the orbit results in bilateral orbital pain, chemosis, proptosis, and ophthalmoplegia. Cranial nerves III, IV; Vl, V2, V3, and VI traverse the sinus and can all be affected.  Extension of the phlebitis posteriorly into the cavernous sinus results in progression of symptoms in the opposite eye.  It can be associated with sepsis and meningismus or frank meningitis may be present.
  • 17. EVALUTIONS  HISTORY(Symptoms of URTI + symptoms of ABRS).  PHYSICAL EXAMINATION.(nasal cavity , orbital EX).  RADIOLOGY. Computed tomography (Ct) is considered the gold standard for orbital complications of ABRS (axial + coronal view). Aim: 1-To confirm the diagnosis. 2-Define the extent and site of disease. 3-give idea about the treatment modalities. 4-exclude other complications.
  • 18. INDICATIONS: 1-In preseptal inflammation progress in 24-48hr despite medical treatment. 2- suspected post septal complications. 3-exclude intracranial abscess.
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  • 21. TREATMENT: (all need admission , antibiotic) >3 years oral Borad spectrum antibiotic +conservative treatment IV antibiotic +- sinus drainage IV antibiotic +sinus drainage +abscess drainage IV antibiotic +- sinus drainage +- abscess drainage
  • 23. INDICATIONS OF MEDICAL TREATMENT IN MEDIALSUBPERIOSTEAL ABSCESS Abscess width<4mm
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