Osteomyelitis
 It is the infection of bone marrow.
- It is seen in infants and children
than adults because of presence of spongy bone in anterior
wall of maxilla. Infection may start in dental sac and then
spread to the maxilla, but less often it is primary infection
of maxillary sinus.
Clinical Features: Erythema, Swelling of cheek, oedema of
lower lid, purulent nasal discharge and fever.
Subperiosteal abscess followed by fistulae may form in
infraorbital region, alveolus or palate or in zygoma.
Treatment- large doses of antibiotics, drainage of any abscess
and removal of sequestra.
2. - More often seen in
adults.
Results from acute infection of frontal sinus either directly
or through the venous spread. It can also follow trauma or
surgery of frontal sinus in the presence of acute infection.
Pus may form externally under the periosteum as soft
doughy swelling, or internally as extradural abscess.
Treatment- large doses of antibiotics, drainage of abscess
and trephening of frontal sinus through its floor.
Orbital Complications
 The orbit is the structure most commonly involved in
complicated sinusitis.
 Orbital extension is usually the result of ethmoid sinusitis.
 Children are more prone to orbital complications,
probably secondary to high incidence of URI and
sinusitis.
Subperiosteal abscess
 Pus collects outside the bone under the periosteum.
 A subperiosteal abscess from ethmoids forms on the
medial wall of orbit and displaces the eyeball forward,
downward and laterally.
 From the frontal sinus, abscess is situated just above
and behind the medial canthus and displaces eyeball
downwards and laterally
 From the maxillary sinus, abscess forms in the floor of
the orbit and displaces the eyeball upwards and
forwards.
Orbital Cellulitis
 When pus breaks through the periosteum and finds its
way into the orbit, it spreads between the orbital fat,
extraocular muscles, vessels and nerves.
 Clinical features will include oedema of lids,
exophthalmos, chemosis of conjunctiva and restricted
movements of the eye ball. Vision is affected causing
partial or total loss which is sometimes permanent.
Patient may run high fever.
 Orbital cellulitis is potentially dangerous because of the
risk of meningitis and cavernous sinus thrombosis.
Orbital Abscess
 Intraorbital abscess usually forms along lamina
papyracea or the floor of frontal sinus.
 Clinical picture is similar to that of orbital cellulitis.
 Diagnosis can be easily made by CT scan or ultrasound
of the orbit.
 Treatment is i.v. antibiotics and drainage of the abscess
and that of the sinus (ethmoidectomy or trephination of
frontal sinus).
Superior orbital Fissure syndrome
 Infection of sphenoid sinus can rarely affect structures
of superior orbital fissure. Symptoms consist of deep
orbital pain, frontal headache and progressive paralysis
of CN VI, III and IV, in that order.
Orbital apex Syndrome
It is superior orbital fissure syndrome with additional
involvement of the optic nerve and maxillary division of
the trigeminal (V2)

Orbital Complications of Sinusitis

  • 1.
    Osteomyelitis  It isthe infection of bone marrow. - It is seen in infants and children than adults because of presence of spongy bone in anterior wall of maxilla. Infection may start in dental sac and then spread to the maxilla, but less often it is primary infection of maxillary sinus. Clinical Features: Erythema, Swelling of cheek, oedema of lower lid, purulent nasal discharge and fever. Subperiosteal abscess followed by fistulae may form in infraorbital region, alveolus or palate or in zygoma. Treatment- large doses of antibiotics, drainage of any abscess and removal of sequestra.
  • 2.
    2. - Moreoften seen in adults. Results from acute infection of frontal sinus either directly or through the venous spread. It can also follow trauma or surgery of frontal sinus in the presence of acute infection. Pus may form externally under the periosteum as soft doughy swelling, or internally as extradural abscess. Treatment- large doses of antibiotics, drainage of abscess and trephening of frontal sinus through its floor.
  • 3.
    Orbital Complications  Theorbit is the structure most commonly involved in complicated sinusitis.  Orbital extension is usually the result of ethmoid sinusitis.  Children are more prone to orbital complications, probably secondary to high incidence of URI and sinusitis.
  • 6.
    Subperiosteal abscess  Puscollects outside the bone under the periosteum.  A subperiosteal abscess from ethmoids forms on the medial wall of orbit and displaces the eyeball forward, downward and laterally.  From the frontal sinus, abscess is situated just above and behind the medial canthus and displaces eyeball downwards and laterally  From the maxillary sinus, abscess forms in the floor of the orbit and displaces the eyeball upwards and forwards.
  • 7.
    Orbital Cellulitis  Whenpus breaks through the periosteum and finds its way into the orbit, it spreads between the orbital fat, extraocular muscles, vessels and nerves.  Clinical features will include oedema of lids, exophthalmos, chemosis of conjunctiva and restricted movements of the eye ball. Vision is affected causing partial or total loss which is sometimes permanent. Patient may run high fever.  Orbital cellulitis is potentially dangerous because of the risk of meningitis and cavernous sinus thrombosis.
  • 8.
    Orbital Abscess  Intraorbitalabscess usually forms along lamina papyracea or the floor of frontal sinus.  Clinical picture is similar to that of orbital cellulitis.  Diagnosis can be easily made by CT scan or ultrasound of the orbit.  Treatment is i.v. antibiotics and drainage of the abscess and that of the sinus (ethmoidectomy or trephination of frontal sinus).
  • 9.
    Superior orbital Fissuresyndrome  Infection of sphenoid sinus can rarely affect structures of superior orbital fissure. Symptoms consist of deep orbital pain, frontal headache and progressive paralysis of CN VI, III and IV, in that order. Orbital apex Syndrome It is superior orbital fissure syndrome with additional involvement of the optic nerve and maxillary division of the trigeminal (V2)