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Opthalmoscopy Uploaded by Parash
- 2.
- 3. Ophthalmoscopy (funduscopy or fundoscopy) is a test that allows a health professional to see inside the fundus of the eye and other structures using an ophthalmoscope (or funduscope).
- 4. It is doneas part of an eye examination and may be done as part of a routine physical examination. It is crucial in determining the health of the retina and the vitreous humor.
- 5.
- 6. • Ophthalmoscope was first invented by Hermann von Helmholtz(1821-1894), a professor of physics from Germany in 1851. • He called it an Augenspiegel (eye mirror)
- 7. • In 1915,Josh Zele and Jon Palumbo invented the world's first hand-held direct illuminating ophthalmoscope • Precursor to the device now used by clinicians around the world • The company started as a result of this invention is Welch Allyn.
- 8. Commonly used brandsin our country • Keeler • Heine •Welch allyn
- 9.
- 10. Direct: This typeof ophthalmoscope is most commonly used during a routine physical examination. Indirect: Indirect ophthalmoscopy provides a wider view of the inside of the eye and allows a better view of the fundus even if the lens is clouded by cataracts. Used by opthalmologist.
- 11. Parts of anophthalmoscope
- 13. How to holdan ophthalmoscope?
- 15.
- 16. Wide angle view: Illuminates the largest area of fundus for the best possible general diagnosis through a large dilated pupil Intermediate angle view: Permits easier access through an undilated pupil and in peripheral examination. Particularly useful in pediatric examination. Macula view: Designed specifically for examination of the macula region of the fundus where a larger beam would create excessive pupillary reaction or patient discomfort. Glaucoma: Projects a graticule onto the retina to assess the optic cup/disc ratio as an aid to glaucoma diagnosis. Slit: Used primarily to determine retinal elevations and depressions, but may also be used to assess anterior chamber depth Fixation Star or Cross: Projects a graticule onto the retina to assess the degree and direction of eccentric fixation, eg, as a result of macula degeneration
- 17. Beam filter Redfree: The red-free filter is used to examine the blood vessels in fine detail. By filtering out the red rays, blood vessels are silhouetted black against a dark green background. Cobalt blue: Used in conjunction with fluorescein dye for the detection and examination of corneal scars and abrasions. Safety: The unique Keeler safety filter cuts out the ultra violet, visible blue and infrared wavelengths said to cause phototoxic retinal damage with prolonged exposure.
- 18.
- 19. Pre-requisite: • Itshould be done in a dark room. • Explain whole of the procedure to the patient. • Pupil is dilated or moderately dilated, but be careful about mydriatic in Glaucoma or Intra ocular implanted lens (IOL). Dilating the pupil with 1% tropicamide or 1% cyclopentolate. This blurs the near vision for 2-3 hrs.
- 20. • Proper positioning:Lying or sitting in chair (better). If lying, move to opposite side when need to examine left eye. • Appropriate direction. • Proper positioning of the examiner. • Both the eye should be seen
- 21.
- 22. • At firstcheck your ophthalmoscope’s battery. • Adjust the ophthalmoscope light to a comfortable brightness.
- 23. • Set the ophthalmoscope lens wheel to zero diopters (D) or correct your visual error by glass or ophthalmoscope lens. • Adjust the focus ring & focus filter.
- 24. • Stand 1hand or half meter apart from the patient in same horizontal plane as patient’s eye. • Ask the patient to look straight ahead at a distant object – patient should continue to look in this direction even if the examiner’s head obscures the target.
- 25. • Patient’s righteye/ your right eye / your right hand /patient’s right side & vice versa.
- 26. A distance ofabout 10-30 cm from the patient try to see through the viewing hole of the ophthalmoscope and focus the light around the patient’s eye. Direction of light should be toward the nose, about 15 degrees from the line of fixation. Instruct the patient to see the distal fixation point with the opposite eye.
- 27. • The pupilshould appear pink from 10 cm distance. This is the Red reflex. • Any opacity in the media appear black upon the red reflex. • If total red reflex is lost, it is due to Medial opacity ( cataract, vitreous haemorrhage) or Retinal problem. Pupillary red reflex opacity
- 28. If patient doesn’t cooperate, fix the head by placing your other hand on the patient’s forehead & gently retract the upper eyelid.
- 29. Now come closeto the patient’s head ,bring the ophthalmoscope to within 1-2cm of the eye . Not to touch the eye lash of the patient. Now you can see inside the eye. At first try to see any vessel, then follow it medially to find out the optic disc.
- 30. • Follow theblood vessels as they extend from the optic disc in four directions: superotemporally, inferotemporally, superonasally& inferonasally • Ask the patient to look up to see superior retina, look down to see inferior retina, look temporally to examine temporal retina ,look nasally to examine the nasal retina
- 31. • Finally locatethe centre of the macula by asking the patient to look directly at the light .Macula present two disc temporal from the optic disc
- 32. SOME COMMON MISTAKESthat we can do, must be corrected by the following way: 1.Examine at the same level 2. Never obstruct the opposite eye 3.Never examine the right eye by left eye and left hand & vice versa 4.Never give too much pressure to the head and shoulder
- 33. Common misinterpretations 1.Temporalpallor : Normally paler than nasal, often misinterpreted as abnormal 2.Myopic fundus: Myopic eye is large, so disc appears paler ,may be mistaken for optic atrophy. 3.Hypermetropic fundus: Small eye ,disc appears crowded, mistaken for papilledema
- 34. 4.Drusen: Colloid bodiesthat may occcur on disc, mistaken for papilloedema 5.Pigmentation on the disc edge :Normal-may make disc seem pale 6.Tortuous vessels: normal
- 35.
- 36. • Detection ofany haziness (opacity) in media, • Detection of any optical error. • To look inside of the eye.
- 37. Haziness in media • Corneal opacity, • Lens opacity, • Vitreous opacity. • It can be detected while observing the red reflex by moving the ophthalmoscope; Right/Left or up/down. • If opacity moves opposite to the light:- Corneal opacity. • If opacity moves towards the light :- Vitreous opacity. • If opacity is fixed :- Lens opacity
- 38. Various opacities inmedia Normal red reflex Corneal opacity cataract
- 39. Optical Error •If focus is hazy, adjust the lens of the ophthalmoscope to (-) or (+) and denote myopia or hypermetropia of the patient, but make sure that your eye is error free. • If operator's eye power is normal or if he/she using glasses and Still the focus is hazy, it is due to optical error of the patient.
- 40. • At firstyou will have to turn the focus dial clockwise (plus or black lens), if error is corrected – Patient is Hypermetropic. • If no improvement, then turn the focus dial anticlockwise (minus or red lens), if error is corrected – Patient is Myopic
- 41. What will seein fundus?
- 42.
- 43.
- 44.
- 45. Blood Vessels Vein Artery
- 46. Optic Disc •The optic disc or optic nerve head is the location where ganglion cell axons exit the eye to form the optic nerve • The optic disc represents the beginning of the optic nerve
- 47. • There areno light sensitive rods or cones to respond to a light stimulus at this point. This causes a break in the visual field called "the blind spot" or the "physiological blind spot".
- 48. Things to beseen: 3c • Contour(Margin): – The borders of the optic disc should be clear and well defined • Color: – Typically the optic disc looks like an orange-pink area with a pale centre. The orange-pink appearance represents healthy, well perfused neuro-retinal tissue
- 49. Cup: As mentionedabove the disc has an orange-pink rim with a pale centre. This pale centre is devoid of neuroretinal tissue and is called the cup
- 50. Blood vessels Arteries: They are superficial, tortuous & brighter. Normally arterial walls are invisible, seen as streak, when light is focused bright streak light reflexion is seen.
- 51. • Veins : They are thick, deeper & darker. Normally venous pulsation is visible near the disc. • Total vessels count in disc : 7-10, which include vein & artery. Count only the main vessels not the branches. • Normal vein : artery = 3:2.
- 52.
- 53. White/yellow lesions: Cottonwool spots (soft exudates): White fluffy spots with indistinct margin caused by retinal ischemia due to accumulation of axonal proteins in the nerve fiber layer. Causes: Severe HTN, DM, retinal vein occlusion ,SLE,AIDS. Cotton wool
- 54. Hard exudates: Bright yellowish sharp-edged lesions consist of lipid deposition that result from leakage of plasma from abnormal retinal capillaries. Causes: DM, HTN. Chorioretinal atrophy: Well defined punched out lesion. Cause: Previous retinal inflammation, injury Hard exudate Hard exudate
- 55. Black lesion: Retinalpigment hypertrophy: Black lesion like bony spicules in periphery. Causes: Retinitis pigmentosa due to any cause, previous injury/laser. Retinitis pigmentosa
- 56. Laser burns: black edged round lesion. Usually in regular pattern. Moles: flat, usually round. Normal findings. Melanoma: raised irregular malignant tumour. Laser burns Malignant melanoma
- 57. Red lesion Dothaemorrhage: Thin vertical haemorrhage that may be difficult to differentiate from microaneurysms seen adjacent to blood vessels. Cause: DM. Blot haemorrhage: Larger full thickness haemorrhages in the deeper layer of retina .Rounded, localized. Causes: DM Dot haemorrhage Blot haemorrhage
- 58. Flame haemorrhage: Superficialbleed, shaped by nerve fibres into a fan with point towards the disc. Cause: HTN, retinal vein oclusion.
- 59. Deep large haemorrhage: Retinal/pre-retinal. Causes: Bleeding diathesis.
- 60. Subhyaloid haemorrhage: Irregularsuperficial with flat top. Causes: Subarachnoid haemorrhage.
- 61. Pathology in OpticDisc Common abnormality in optic disc: • Optic disc swelling (Papilloedema/ Papillitis) • Optic atrophy. • Glaucomatous cupping. • Abnormal vessels.
- 62. Optic disc swelling Optic nerve head swelling can be inflammatory or non-inflammatory . If non-inflammatory: Papilloedema If Inflammatory: Papillitis.
- 63. Papilledema • Causedby raised intracranial pressure. • Loss of venous pulsation (normally absent in 15% people.) • Disc is abnormally red. • Margins are blurred, upper nasal quadrant first, then lower nasal, then temporal margin.
- 64. • Physiological cupbecomes obliterated. • Retinal veins are slightly distended. • If papilloedema develops rapidly, there will be marked engorgement of the retinal veins with haemorrhages & exudates on & arround the disc. • If develops slowly, may be little or no vascular change.
- 66. PAPILLITIS Ophthalmoscopy •Ophthalmoscopy may show no abnormalities on retrobulbar optic neuritis. • Dilatation of retinal arteries and veins on optic nerve disc . • Possible petty splinter hemorrhages on the optic nerve disc .
- 67. • Retinal edema around the optic disc. • Optic nerve disc has blurred margins • Reddish (hyperemic) optic nerve disc due to dilatation of blood vessels . • Possible white exudates on the optic nerve disc .
- 68. PAPILLITIS PAPILLOEDEMA Usuallyunilateral Usually bilateral Marked dimness of vision. May be slight dimness of vision. Not loss. Loss of afferent pupillary reflex Not loss. Visual field defect is usually central, particularly for red & green. Peripheral constriction or enlargement of blind spot. Eye ball is painful & tender. Not painful/tender.
- 69. Optic Atrophy Featuresof optic atrophy • Disc is small. • Pale. • Loss of function. Added may be • Reduced number of vessels (< 7). • Margin may be sharp / blurred.
- 70. Types 1. primary 2. secondary
- 71. Primary optic atrophy • Due to disease of the optic nerve. • Disc is flat, pale/white. • Clear-cut, sharp margins. • Decreased / loss of vision Secondary optic atrophy • Due to long standing papilloedema. • Disc is greyish-white. • Indistinct margins. • Decreased / loss of vision.
- 72. Papilloedema Optic atrophy In both picture disc margins are blurred/indistinct & vessels count decreased, but in secondary optic atrophy disc colour is pale & in papilloedema disc colour is abnormally red.
- 73. Optic cup andCup Disc ratio(CDR) • The optic cup is the white, cup-like area in the center of the optic disc. • The ratio of the size of the optic cup to the optic disc (or cup-to-disc ratio) is the cup disc ratio. • Normally the cup should take up less than 50% of the disc,i.e. CDR is <.5 • The CDR is measured to diagnose Glaucoma
- 74. Glaucoma CDR= .4 CDR= .77
- 75. Progression of glucomatous Optic nerve Damage
- 76. Hypertensive retinopathy Grade1 : Arteriolar thickening, tortuosity, increased reflectiveness (‘Silver wiring’). Grade 2: Grade 1 plus constriction of veins at arterial crossings (‘Arteriovenous nipping/nicking’). Grade 3: Grade 2 plus evidence of retinal ischaemia (‘Flame shaped or blot hemorrhage and cotton wool exudate’). Grade 4: Grade 3 plus papilloedema.
- 77.
- 78.
- 79. Grade 1 Silverwiring: – It’s the appearance of blood vessels in which the arterial wall becomes so completely opaque that the blood column is not seen and the central light reflex occupies all of the width of the arteriole. – The light is completely reflected, yielding a white ‘line,’ likened to a silver wire,
- 80. Grade 2 NormalAV nipping
- 81. Grade 2 •AV nicking: A vascular abnormality in the retina of the eye, visible on ophthalmologic examination, in which a vein is compressed by an arteriovenous crossing • The vein appears "nicked" as a result of constriction or spasm
- 82. Grade 3 Cottonwool exudate Blot Haemorrhage Flame shaped
- 83.
- 84.
- 85. Diabetec Retinopathy Classificationof Diabetic Retinopathy –Non-proliferative ‘background’ retinopathy without maculopathy, –Maculopathy, – Pre-proliferative retinopathy, – Proliferative retinopathy
- 86. Non-proliferative ‘background retinopathywithout maculopathy Blot hemorrhage Dot hemorrhage Hard Exudate
- 87. Maculopathy Hard exudate Dot and blot Haemorrhage Macular oedema Macular oedema, exudates, dot & blot hemorrhage
- 88. Pre proliferative retinopathy Features of pre-proliferative retinopathy: –Venous loops & beading, dot-blot haemorrhage, large retinal hemorrhage, cotton wool exudates, macular oedema with reduced visual acuity, perimacular exudates, retinal hemorrhages of any size. But no proliferative changes.
- 89.
- 90. Proliferative diabetec retinopathy Abnormal blood vessels
- 91.
- 92. Fundoscopy findings in different conditions
- 93. Retinitis pigmentosa 1.Retinal pigmentation 2.Thin Blood vessels 3.Pale optic disc
- 94. Central retinal veinocclusion 1.Dilated and tortuous retinal veins 2.Diffuse intraretinal haemorrhage in all 4 quadrants 3.Cotton wool spots 4.Swollen optic disk 5. Retinal oedema
- 95. Central retinal arteryocclusion 1. Retina appears pale due to Retinal edema 2. Optic disc swelling 3. Macula with cherry-red spot on white-yellow background
- 96. Acute Leukaemia Acuteleukemia: Intra-retinal white-centered hge. (Roth spot) Cotton-wool spots
- 97. Aplastic anaemia Discedema Retinal hge
- 98. Vitreous haemorrhage preretinal-unclotted blood with boatshaped configuration, moving towards gravity
- 99. Sub arachnoid Haemorrhage Disc swelling Retinal hge Subhyaloid hge Vitreous hge
- 100.
- 101. Retinal detachment Mobile Convex Corrugated retina
- 102. Retinoblastoma Leukocoria Direct visualization of tumor (Multi globulat- -ed white mass with overlying retinal detachment)
- 103. Roth’s spot Bacterialendocarditis DM , Leukemia Pernicious anemia HTN,AIDS
- 104. Cytoid body Systemiclupus erythematosus.
- 105. IOL (intra ocularlens) Anterior chamber IOL Posterior chamber IOL
- 106.