its an small guide to assess the neuorological status with various pictures , it explains clearly about GCS, MUSCULAR POWER ASSESSMENT , PUPILLARY REACTION & IMPORTANT REFLEXES specially for nurses ....it has brief information about TBI PROTOCAL & RASS SCORE
Neurocritical care triad e Focused neurological examination, brain multimodal...Apollo Hospitals
Intensive care is rightly described as “an art of managing
intense intricacy” and this situation is further complicated in
the care of patients with critical neurological illness owing to limited scope for clinical examination in view of altered
conscious levels.
its an brief information for Intensive nurses about the Neurological assessment ,GCS, braian death assessment & expalins about important brain reflex's pertaining to icu setup, for making this pdf i used out hospitlas protocal, nursing journals.....
Neurocritical care triad e Focused neurological examination, brain multimodal...Apollo Hospitals
Intensive care is rightly described as “an art of managing
intense intricacy” and this situation is further complicated in
the care of patients with critical neurological illness owing to limited scope for clinical examination in view of altered
conscious levels.
its an brief information for Intensive nurses about the Neurological assessment ,GCS, braian death assessment & expalins about important brain reflex's pertaining to icu setup, for making this pdf i used out hospitlas protocal, nursing journals.....
Nurses as the primary care providers would be the immediate health care professional to assess the patient's response and to determine whether he is improving or deteriorating. Signs of brain death can be identified and reported early by a nurse with adequate knowledge.
Nurses as the primary care providers would be the immediate health care professional to assess the patient's response and to determine whether he is improving or deteriorating. Signs of brain death can be identified and reported early by a nurse with adequate knowledge.
Approach to coma
1-Definition
2-Pathophysiology, Causes, and similar condition
3-History and general physical examination
4-Neurological examination
5-Investigation
6-Management
EXTERNAL VENTRICULAR CARE FOR NURSES.pptxMURUGESHHJ
EVD---EVD CARE ESPECAILLY IN ICU SETTINGS MORE ESSENTIAL , THIS PPT EXPLAINS YOU ABOUT EVD IN BRIEF, INDICATIONS, COMPLICATIONS , , EVD CARE PROCEDURE, NURSING DIAGNOSIS & MANGEMENT ASSOCIETED WITH EVD RELATED INFECTIONS ...
HOSPTAL ACQUIRED PNEUMONIAE , PREVENTION AND MANAGEMENT PROTOCALS MURUGESH.pptxMURUGESHHJ
this is an brief explanation for one of most common infection in hospital i.e , HAI, meaning, causes, prevention & management stragies , VAP OR VAE & NVHAP bundles, especially usefull for nurses ...
IMPORTANCE OF ORAL CARE IN ICU MURUGESH HJ.pptxMURUGESHHJ
THIS PPT EXPLAINS ABOUT IMPORTANCE OF ORAL HYGIENE ESPECIALLY FOR UNCONSIOUS PATIENTS, SEVERLY ILL PATIENTS , IT EXPLAINS YOU ABOUT PROCEDURE, IMPORTANT ARTICLES , MOST BENEFICIAL FOR NURSES
HEART SOUNDS ASSESSMENT FOR NURSES MURUGESH.pptxMURUGESHHJ
IT IS AN BRIEF DESCRIPTION ABOUT SIMPLE ASSESMENT OF HEART , ANATOMY & PHYSIOLOGY OF HEART ,HEART SOUNDS, NORMAL & ABNORMAL SOUNDS..ESPECIALLY MOST USEFULL TO NURSES...
Chest auscultation & lung sounds assessment for nursesMURUGESHHJ
its an brief explanation regarding respiratory system & most common sites to assess lung sounds &lobe associated lung infections...visuals explains briefly & clearly about abnormal lung conditions
Artereal blood gas meaning,brief guide for nurses murugeshMURUGESHHJ
ABG-It is an vital &fastest test to assess the patient haemodynamics , this ppt explains you briefly about ABG meaning, components,sampling, allens test & nurses roles....
Mechanical ventilator, common modes, indications,nursing responsibilities MURUGESHHJ
it is an brief summary with diagrammatic presentation for NURSES regarding Mechanical ventilator, uses, complications, types, important terms,common modes, NIV, uses, NURING ROLES & RESPONSIBILITIES for handling INTUBATED patients...
it is an brief description and slides about the CYANOSIS,ISCHEMIA, ISCHEMIC MANAGEMENT MEANING , HEPARIN , HEPARIN USES , IV INFUSION, SIMPLE HEPARIN IV INFSUION CALUCULATION FORMULAE , ANTIDOTE AND NURSING MANAGEMENT,expalins in diagrammatic manner for nurses, ICU nursing educators,primarily its most benificial for INTENSIVE NURSES works in critical area units like ICUs , CT ICUS etc...
Diabetic ketoacidosis meaning,types &management for nurses murugeshMURUGESHHJ
its an brief information about the Diabetic ketoacidosis, causes, signs & symptoms ,hospital management protocals in simple english......provides more information with diagrammatic way ...thank you all
THIS IS AN BRIEF INFORMATION ABOUT AN ONE OF MY FAVOURITE SUBJECT ARDS & & ITS MANAGEMENT ,ROLES OF INTENSIVE NURSES , IT WILL EXPLAINS ABOUT CATEGORIES, PF RATIO, PRONE POSITIONING & NURSING CARE .....FOR THIS I REFFERED OLD SLIDE SHARE PPTS & IN HOSPITAL ROUTINELY PRACTICING POLICIES
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Anti ulcer drugs and their Advance pharmacology ||
Anti-ulcer drugs are medications used to prevent and treat ulcers in the stomach and upper part of the small intestine (duodenal ulcers). These ulcers are often caused by an imbalance between stomach acid and the mucosal lining, which protects the stomach lining.
||Scope: Overview of various classes of anti-ulcer drugs, their mechanisms of action, indications, side effects, and clinical considerations.
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
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3. INTRODUCTION..
Neurological observations collect data on a patient’s neurological status and
can be used for many reasons, including in order to help with diagnosis, as a
baseline observation, following a neurosurgical procedure, and following trauma
(Mooney & Comerford 2003)….
Therefore, it is important that all healthcare professionals are efficient and
accurate in assessing the neurological status of their patients.
It is also important to remember that these changes may occur rapidly over a
short period of time or more gradually, taking place over days or weeks. This is
why accurate neurological assessments and observations are vital in ensuring the
early recognition of neurological deterioration in patients (Koutoukidis et al. 2017;
Mooney & Comerford 2003).
4. BREIF INFORMATION…..
A neurological assessment involves checking the patient
in the main areas in which changes are most likely to occur:
Level of consciousness
Pupillary reaction
Motor function
Sensory function
Vital signs.
5. Glassgow Coma Scale…….
There are many different assessment tools for neurological function, however,
the most widely known and used tool is the Glasgow Coma Scale (GCS).
The patient is assessed and scored in three areas:
1.Eye opening
2.Verbal response
3.Motor response.
The highest possible score is 15, which reflects an individual who is fully alert,
aware and orientated, whereas the lowest possible score is 3 and reflects an unconscious individual.
Although pupil reaction is not included as part of the GCS, but is the vital element to assess the abnormality
6. Components of GCS …..
Behaviour Rating Score
Eye Opening Response
Opens eyes spontaneously Spontaneous 4
Opens eyes in response to speech and sound Sound 3
Opens eyes in response to painful stimuli Pain 2
Does not open eyes None 1
Verbal Response
Oriented to time, person and place Oriented 5
Confused and disoriented Confused 4
Utters incoherent words Words 3
Incomprehensible sounds Sounds 2
Makes no sounds None 1
Motor Response
Obeys two-part requests Obeys commands 6
Localises to painful stimuli Localising 5
Flexion / withdrawal from painful stimuli Normal flexion 4
Abnormal flexion from painful stimuli Abnormal flexion 3
Extension to painful stimuli Extension 2
Makes no movement
7. CONSIOUSNESS ASSESSMENT ;AVPU
scale…
A rapid assessment tool that is utilised in the healthcare field to measure conscious state is the AVPU scale.
A stands for Alert
The patient is aware of the environment and the examiner and is opening their eyes spontaneously. They can
also follow commands and track objects.
V stands for Verbal
The patient’s eyes do not open spontaneously, rather, their eyes only open in response to a verbal stimuli
directed towards them. The patient can respond to this verbal stimuli directly and in a meaningful way.
P stands for Pain
The patient's eyes do not open spontaneously or in response to verbal stimuli. The patient will respond to
painful stimuli directed towards them by moving, moaning or crying out.
U stands for Unresponsive
The client is not responding spontaneously, or to verbal or painful stimuli.
8. PUPILLARY REACTION…..
Assessing Pupillary Reaction
When we are assessing the patient’s pupils, we are gaining information regarding the brain and
determining whether there has been an increase in intracranial pressure.
The pupils are assessed for their size and shape, as well as how they react to the presence of light. They
should be round and equal in size.
The size of the pupils can vary, however, the normal range is 2 to 6 mm in diameter. Upon shining a
bright light into each eye, the pupils should constrict briskly to a smaller size (QAS 2021a).
The reactions to light can be described as brisk, sluggish or non-reactive/fixed.
Both eyes should be checked and compared against each other. Generally, any change that occurs
during an assessment of the pupils indicates a change in the individual’s intracranial pressure and may
signify a neurological emergency.
Acute pupillary dilation in patients who have suffered a head injury is thought to be caused by
compression of the third cranial nerve from brain oedema and herniation, or alternatively, from a
decrease of blood flow to the brain stem, resulting in brain stem ischaemia (Koutoukidis et al. 2017;
Majdan 2015…….
9. MOTOR RESPONSE –LIMBS STRENGTH
Limb strength can be described as either:
Normal power
Mild weakness
Severe weakness
Spastic flexion
Extension
No response.
Generally, this assessment focuses on the arms and legs and will look for any
improvement or deterioration in function. However, it must be noted that lower limb
function may impact spinal function in some patients and this can disrupt the
assessment findings …..
10. INTRA CRANIAL PRESSURE ….
Elevated intracranial pressure (ICP) is seen in ;
head trauma, [1] hydrocephalus, intracranial hemorrhage, sub-arachnoid hemorrhage from ruptured
brain aneurysm, intracranial tumors, [3] hepatic encephalopathy, [4] and cerebral edema. Intractable
elevated ICP can lead to death or devastating neurological damage
either by reducing cerebral perfusion pressure (CPP) [6] and causing cerebral ischemia or by compressing and
causing herniation of the brainstem or other vital structures. Prompt recognition is crucial in order to intervene
appropriately
CUSHINGS TRIAD…
refers to a set of signs that are indicative of increased
intracranial pressure (ICP), or increased pressure in the brain.
Cushing's triad consists of
bradycardia (also known as a low heart rate), HR < 50B/MIN
irregular respirations, some times TACHYPNOEA OR BRADYPNOEA
a widened pulse pressure BP > 150/80MMHG
12. PUPILLARY REFLEX……
PUPILLARY REFLEX
Eyes allow for visualization of the world by receiving and processing light stimuli.
The pupillary light reflex constricts the pupil in response to light, and pupillary
constriction is achieved through the innervation of the iris sphincter muscle……..
Pupillary light reflex is used to assess the brain stem function. Abnormal
pupillary light reflex can be found in optic nerve injury, oculomotor nerve
damage, brain stem lesions, such as tumors, and medications like barbiturates.
13. CORNEAL REFLEX….
CORNEAL REFLEX
A reflex closing of the eyelids when the cornea is touched or a puff of air is
blown on to it, mediated by the fifth cranial trigeminal nerve (sensory) and the
seventh cranial facial nerve (motor), often diminished or absent in people who
wear contact lenses..
The corneal reflex, also known as the blink reflex or eyelid reflex, is an
involuntary blinking of the eyelids elicited by stimulation of the cornea, though
could result from any peripheral stimulus.
14. GAG REFLEX…..
GAG REFLEX….
The gag reflex, also called the pharyngeal reflex, is a contraction of the throat
that happens when something touches the roof of your mouth, the back of your
tongue or throat, or the area around your tonsils. This reflexive action helps to
prevent choking and keeps us from swallowing potentially harmful substances
15. COUGH REFLEX…..
COUGH REFLEX
Coughing is an important defensive reflex that enhances clearance of secretions
and particulates from the airways and protects from aspiration of foreign
materials occurring as a consequence of aspiration or inhalation of particulate
matter, pathogens, accumulated secretions, postnasal drip, inflammation, and
mediators ……
16. OCULO CEPHALIC REFLEX ( DALLS EYE)
….
OCULO CEPHALIC REFLEX (DALLS EYE )…..
The doll's eyes reflex, or oculocephalic reflex, is produced by moving the patient's head left to right or up and down. When the reflex is
present, the eyes of the patient remain stationary while the head is moved, thus moving in relation to the head.
OVERVIEW
Oculocephalic and oculovestibular reflexes are primarily used to determine whether a patient’s brainstem is intact (e.g. coma or
brain death assessment)
ensure the C-spine is cleared.
the patient’s eyes are held open.
the head is briskly turned from side to side with the head held briefly at the end of each turn.
a positive response occurs when the eyes rotate to the opposite side to the direction of head rotation, thus indicating that the brainstem
(CN3,6,8) is intact.
a similar result is seen when the head is flexed and extended — a positive result is downward deviation of the eyes during extension, and
upward deviation during flexion (the eyelids, if closed, may also open as part of the ‘doll’s head phenomenon’). These vertical responses
indicates that the brainstem (CN3,4,8) is intact.
The eyes should gradually return to the mid-position in a smooth, conjugate movement if the brainstem is intact.
Patients with metabolic coma (e.g. hepatic failure) may have exaggerated, brisk oculocephalic reflexes.
17. OCULOVESTIBULAR REFLEX(CALORIC
TEST
OCULOVESTIBULAR REFLEX ( COLD CALORIC TEST)
Oculovestibular reflex (caloric stimulation):
the head is elevated to 30 degrees above horizontal so that the lateral semicircular canal is vertical, and so that stimulation with generate a
maximal response.
check that the tympanum is intact and that the external ear canal is clear — C-spine clearance is not necessary.
introduce iced water into the external ear canal through a small catheter until one of the following occurs:
nystagmus (in the intact brainstem the slow phase is towards the irrigated ear)
ocular deviation
200mL of iced water has been instilled.
allow 5 minutes between testing ears to allow re-equilibration of the oculovestibular system.
as consciousness is lost, the fast component (towards the non-irrigated ear) is lost and the slow component deviates the eye in the direction of
the irrigated ear.
Vertical oculovestibular eye responses can be assessed by irrigating both ears simultaneously.
If the brainstem is intact, cold water causes the eyes to deviate downwards and warm water causes the eyes to deviate upwards.
The positive brainstem responses described above are those seen in a comatose patient with an intact brainstem.
19. TBI PROTOCAL --
TOTAL BRAIN INJURY ---
SUDDEN OR PROGREESIVE MASS ERRUPTION OR CIRCULATION BLOCKAGE OR
SUDDEN BRAIN ANOXEMIA OR SKULL PIERCING TRAUMA MAY LEEDS TO
REVERSIBLE OR IIREVERSIBLE INJURY TO THE BRAIN…
MAIN TYPES OF BRAIN INJURY IS
01.CONCUSSION –IS A SUUDDEN OR SHAKING MOVEMNT , MINOR AND MOST
COMMAN IT WILL HEAL FASTEN…..
02.CONTUSSION - IS A BRUISE OF THE BRAIN TISSUE, JUST LIKE ONE MIGHT HAVE A
BRUISE ON THEIR SKIN. AND LIKE ANY OTHER BRUISE, THEY ARE CAUSED BY THE BREAKING
AND LEAKING OF SMALL BLOOD VESSELS …SEVERE CONTUSIONS MAY CAUSE A
LOSS OF CONSCIOUSNESS, CONFUSION, TIREDNESS, EMOTIONAL
DISTRESS, OR AGITATION. MORE SEVERE CONTUSIONS MAY CAUSE THE
BRAIN TO SWELL, COULD PREVENT PROPER OXYGENATION, AND OTHER
SERIOUS CONSEQUENCES.
20. TBI protocol ….contd…..
03. PENENRATING INJURY- MOST COMMONLY EXTERNAL INJURY TO THE
SKULL…OBJECT THAT PIERCING & INJURING THE BRAIN …..IN CASES LIKE
ACCIDENTAL SLIP OR FALL INJURY …RTA HEAD INJURY….ASSALT OR
GUNSHOT….. MOST FATAL & REQUIRES IMMEDIATE INTERVENTIONS ..
04.ANOXEMIC BRAIN INJURY – FOR 4-5 MINUTES BRAIN CAN SURVIVE
WITHOUT OXYGEN …STILL NO OXYGEN ..ANOXEMIA --- COMA OR BRAIN
DEATH …
CAUSES –SUDDEN BLOCAKGE OF CIRCULATION EXAMPLE- BY STROKE
EITHER ISCHEMIC OR HEMORRHAGIC SHOCK…….MASS OR TUMOUR
DEVELOPMENT ………
21. TBI PROTOCAL ….important points
Its an important gudelines to follow in order to avoid complications like POST
TRAUMATIC BRAIN INJURY …
It includes few points to follow ..
****HEAD POSITION -30-45 DEGREE ( REVERSE TRENDLUNBURG position)
****TEMPERATURE SHOULD BE MAINTAIN 34-60 DEGREE
***KEEP BP MAP 80-90MMHG
***MAINTAIN PCO2 RANGE 35-40 & PO2 95-105..
22. ***SODIUM LEVEL 140-150
HAEMOGLOBIN >10%GM
****RBS 5.1-8MMOL
***GOOD DIET( ELECTROLYTES BALANCED )
***MAINTAIN RASS SCORE -4 TO -5
***ANTI STRESSORS OR ANTI CONVULSANTS OR OSMOTIC DIURETICS AS PER
ADVICE
***FLEET ENEMA ( IN ORDER TO KEEP NORMAL INTRA ABDOMIINAL
PRESSURE)
TBI PROTOCAL ….important points
23. RASS SCORE…..
RICHMOND AGTATION AND SEDATION SCALE ….
IT IS AN SEDATION ASSEMENT TOOL , MAINLY USING IN CRITICAL AREAS LIKE
ICUS,Ots…..
TO MONITOR THE LEVEL OF CONSCIOUSNESS OR TO UNDERSTAND PATIENT COPING
WITH VENTILATOR OR TO DESCRIBE THE ALERTNESS OR AGITATION ………