This document defines and discusses low birth weight babies, including preterm babies and small for gestational age (SGA) babies. It provides definitions for preterm babies as those born before 37 weeks gestation and low birth weight babies as those weighing less than 2500 grams. SGA babies are defined as those with a birth weight less than the 10th percentile for their gestational age. The document discusses the incidence, etiology, manifestations, management, and complications of low birth weight babies.
This slides contain description about breast feeding, anatomy of breast, types of human milk, good position for latching, holding for the baby, advantages of breast feeding, contraindication of breast feeding, barriers and problems associated with breast feeding with their management
This slides contain description about breast feeding, anatomy of breast, types of human milk, good position for latching, holding for the baby, advantages of breast feeding, contraindication of breast feeding, barriers and problems associated with breast feeding with their management
Every year more than 10 million children die in developing countries due to acute respiratory infections (mostly pneumonia), diarrhea, measles, malaria, or malnutrition - and often to a combination of these illnesses. In 1990s, the WHO, in collaboration with UNICEF and many other agencies, institutions and individuals, responded to this challenge by developing a strategy known as the Integrated Management of Childhood Illness (IMNCI).This strategy adopted in India as Integrated Management of Neonatal and Childhood Illness (IMNCI). IMNCI caters to two groups of children
• 0-2 months, referred to as young infants.
• 2 months to 5 years, referred to as children.
The Apgar score is a test given to newborns soon after birth. This test checks a baby's heart rate, muscle tone, and other signs to see if extra medical care or emergency care is needed. The test is usually given twice: once at 1 minute after birth, and again at 5 minutes after birth
The slides contain description of weaning foods and artifical feeding given to the baby, important points to be considered while preparing feed for the baby
Every year more than 10 million children die in developing countries due to acute respiratory infections (mostly pneumonia), diarrhea, measles, malaria, or malnutrition - and often to a combination of these illnesses. In 1990s, the WHO, in collaboration with UNICEF and many other agencies, institutions and individuals, responded to this challenge by developing a strategy known as the Integrated Management of Childhood Illness (IMNCI).This strategy adopted in India as Integrated Management of Neonatal and Childhood Illness (IMNCI). IMNCI caters to two groups of children
• 0-2 months, referred to as young infants.
• 2 months to 5 years, referred to as children.
The Apgar score is a test given to newborns soon after birth. This test checks a baby's heart rate, muscle tone, and other signs to see if extra medical care or emergency care is needed. The test is usually given twice: once at 1 minute after birth, and again at 5 minutes after birth
The slides contain description of weaning foods and artifical feeding given to the baby, important points to be considered while preparing feed for the baby
Lecture given during Port said fifth neonatology conference, 23-24 October 2014 by Dr.Osama Arafa Abd EL Hameed M. B.,B.CH - M.Sc Pediatrics - Ph. D. Consultant Pediatrician & Neonatologist Head of Pediatrics Department - Port-Fouad Hospital
INTRODUCTION
A newborn, regardless of gestational age or birth weight, who has a greater than average chance of morbidity or mortality because of conditions or circumstances superimposed on the normal course of events associated with birth and the adjustment to extra uterine existence.
FACTORS – TO DEFINE HIGH RISK NEWBORN
DEMOGRAPHIC SOCIAL FACTORS:
Maternal age <16 or >40, unmarried, physical stress, socio-economic status.
PAST MEDICAL HISTORY:
Diabetes Mellitus, genetic disorder, hypertension
PREVIOUS PREGNANCY:
Intrauterine death, neonatal death, IUGR, congenital malformations.
PRESENT PREGNANCY:
Vaginal bleeding, PROM, multiple gestation, pre-eclampsia, abnormal USG findings.
LABOR: AND DELIVERY:
Obstructed labor, fetal distress, forceps delivery, meconium stained liquor.
NEONATE:
Birth weight <2000 or >4000, gestation <37 or >42.
DEFINITIONS
Low birth weight: Live born baby weighing 2500 gram or less at birth. (VLBW: <1500 gm, ELBW: 000 gm).
Preterm: When the infant is born before term i.e. before 38 weeks of gestation.
Premature: When the baby is born before 37 weeks of gestation.
Full term: When the infant is born between 38-42 weeks of gestation.
Post term: When the baby is born after 42 weeks of gestation.
HYPOTHERMIA
DEFINITION
It is a condition characterized by lowering of body temperature than 36℃.
TYPES OF HYPOTHERMIA
It can be classified according to causes and according to severity.
CLASSIFICATION BASED ON CAUSE:
Primary Hypothermia:
Seen immediately after delivery.
Normal term baby delivered into a warm environment may drop its rectal temperature by 1 – 2℃ shortly after birth and may not achieve a normal stable body temperature until the age of 4 – 8 hours.
In low birth weight baby, the decrease of body temperature may be much greater and more rapid unless special precautions are taken immediately after birth. (Loss at least 0.25℃./min).
Secondary Hypothermia:
This occurs due to factors other than those immediately associated with delivery.
Important contributory factors are: e.g. acute infection especially septicaemia.
CLASSIFICATION BASED ON SEVERITY:
According to severity:
Mild Hypothermia: <36℃.
Moderate Hypothermia: <35.5℃.
Severe Hypothermia: <35℃.
CLINICAL FEATURES
Decrease in body temperature measurement.
Cold skin on trunk and extremities.
Poor feeding in the form of poor suckling
Shallow respiration
Cyanosis
Decrease activity, e.g. weak cry.
FOUR MODALITIES OF HEAT LOSS IN NEONATES
Evaporation: Heat loss that resulted form expenditure of internal thermal energy to convert liquid on an exposed surface to gases, e.g. amniotic fluid, sweat.
Prevention: Carefully dry the neonates after delivery or after bathing.
Radiation: It occurred from body surface to relatively distant objects that are cooler than skin temperature.
Conduction: Heat loss occurred from direct contact between body surface and cooler solid object.
Prevention: Keep the baby out of drafts and close end of heat shield in in
Intra uterine Growth Retardation includes Low birth weight (LBW)
Very low birth weight (VLBW)
Extremely low birth weight (ELBW)
Premature
Small for Gestational Age (SGA)
Large for Gestational Age (AGA)
Intrauterine Growth Retardation (IUGR)
Factors affecting Fetal/Baby size:
Conditions associated with symmetric IUGR
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
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Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
2. DEFINITION:-
1.Preterm baby:-
a)A baby born before 37
completed weeks of gestation
calculating from the first day of
LMP is defined as “preterm
baby.”
- D.C Dutta
b) A fetus delivered between 20
& 37 weeks of gestation is
“preterm baby.”
3. DEFINITION:-
2. Low birth weight (LBW):-
“Defined as one whose birth weight is
less than 2500 grams. Irrespective of
gestational age.”
3. Small for gestational age (SGA):-
“The term is to designate the
newborns with birth weight les
than 10th
percentile or less then
two standard deviation for their
gestational age.”
5. MANIFESTATION:-
1. Anatomical:-
a) The weight is 2500 gms or less & length is
usually less than 44 cm.
b) Head & abdomen are relatively large,
skull bones are soft with wide sutures &
post fontanels.
c) Head circumference is disproportionately
exceeds that of chest.
d) Pinnae of ear are soft & flat.
e) Eyes are kept closed.
f) Skin is red shiny D/T lack of subcutaneous
fat & covered by plentiful lanugo & vernix
caseosa.
6. MANIFESTATION:-
g) Plantar creases are not visible.
h) Testicles are undescended , labia minora are exposed because
labia majora in contact.
i) Nails are not grown right upto the finger tips.
7. FUNCTONALLY:-
a) CNS:- Lethargic & inactivity, poor cough
reflex.
b) CVS:- About 1/3rd
of babies have PDA.
c) RS:- RDS, pulmonary aspiration &
atelactesis are common.
d) GI:- Difficulties in feeding D/T poor
sucking, small capacity of stomach.
Hepatic immaturity leads to
hyperbilirubinaemia which may be
aggravated by delayed feeding,
dehydration & hypoxia.
8. e) Thermoregulation :-
Temperature regulating centre is
immature, heat loss is excessive. Hence
preterm infant develop hypothermia.
f) US:- D/T renal immaturity preterm
infant develop elevation of BUL, BUN,
acidosis & dehydration.
g ) Infection:- low levels of IgG
antibodies preterm babies are prone t
infection.
12/26/17 8MR.SACHIN GADADE
9. MANAGEMENT :-
CARE OF PRETERM BABY AFTER
BIRTH :-
Immediate management after
birth:-
1.Cord to be clamped quickly to
prevent hypervolemia & later on
development of
hyperbilirubinaemia.
2.Air passage should be cleared.
3.Adequate O2 therapy (O2
concentration 35% )
10. 4. Vit. K 1mg IM to prevent
hemorrhages.
5. Body should be handled
carefully. Bathing is not
appropriate for a
premature baby.
11. PRINCIPLES OF MANAGEMENT :-
1. To maintain body temperature -
2. Respiratory support -
3. Prevention of infection –
4. To maintain nutrition –
5. Adequate nursing care -
13. PATHOLOGY:-
High conc. Of O2 for prolonged period
Induces vasoconstriction specially temporal
portion of retina
Anoxia damage to endothelium
Regeneration of new vessels in area occurs
after the O2 therapy is withdrawn
13
14. Extension of revascularization beyond the retina into
vitreous
Dilatation of vessels – rupture
Fibrosis
Adhesions – detach retina
Blindness
15. Definition:-
Babies with a birth weight of less
than 10th
percentile for their gestational
age. They are also termed as small for
gestational age (SGA)
12/26/17 15MR.SACHIN
16. Maternal :-
short stature mother
primi or grand multipara
teenage pregnancy
low Pre-Pregnant weight
maternal illness- anemia, heart disease, malaria
complications of pregnancy – PIH
smoking, alcoholism or drug abuse by mother
poor weight gain during pregnancy
previous similar baby
Causes
12/26/17 16MR.SACHIN
17. Placental factors:-
disorders of placental implantation
Abruptio placenta
single umbilical artery
structural & functional abnormalities of placenta
umbilical cord.
Fetal Factors:-
first born babies are generally smaller
twin or multiple pregnancy
intrauterine infections
genetic or chromosomal aberrations
12/26/17 17MR.SACHIN
18. Environment factors :-
poor socioeconomic status
nutritional habits
cultural practices
12/26/17 18MR.SACHIN
19. Types :-
the babies with SFD are found as three different
types.
1. Malnourished small for dates infants:
These babies appear long, thin and alert
They look marasmic poor subcutaneous fat & poor
muscle mass.
They have excess skin folds on the buttocks & thigh.
The difference in head circumference and chest
circumference is more then 3 cms.
The internal organs such as liver thymus & lungs are
shrunken but pulmonary alveoli are mature as per the
gestation.
12/26/17 19MR.SACHIN
20. 12/26/17 SHASHIKANT 20
In these infants the growth arrest
probably occurred in the later part
of pregnancy due to reduction in
the cell size but not the cell number
Prognosis of physical growth is
better
21. 2. Hypo plastic SFD :-
In case of intrauterine infection, genetic
defects & chromosomal aberration growth
retardation occurs in the early part of the
pregnancy, the no of body cell is reduced these
babies are small including the head size, prognosis
is poor permanent mental& physical growth
retardation.
12/26/17 21MR.SACHIN
22. 12/26/17 SHASHIKANT 22
3. Mixed group:-
There is reduction in the cell no. & the cell size
because of the adverse factors operate during both
the early & mid pregnancy
23. Common problems of SFD Babies
Birth asphyxia SFD infant suffer from prenatal asphyxia due to
maternal factors & placental insufficiency. Some of them pass me
conium in utero due to distress & are liable to develop me conium
aspirant syndrome.
Fetal hypoxia & introspection death due to place dysfunction
Inappropriate thermoregulation
12/26/17 23SHASHIKANT
24. 12/26/17 SHASHIKANT 24
Pulmonary hemorrhage polycythemia due to
unknown cause
Increased risk of infections
Poor growth potential
Metabolic changes these infants develop
hypoglycemia due to poor reserves of glycogen & fat.
Hypokalemia is frequent because of transient
hypoparathyroidism.
25. Management :-
Whenever a SFD fetus is suspected careful
observation of mother is made to determine LBW with
the help of USG. Majority of fetal deaths occur after
36th wk gestation so correct diagnosis is Essential.
Mother is advised for adequate bed rest in left lateral
position.
To correct malnutrition by balanced diet: 300 extra
calories per day to be taken
Avoid smoking & alcohol
Fetal growth & assessment of well being of for his to be
done, by NST.
12/26/17 25MR.SACHIN