Examination, evaluation & Assessment
of Hip
Dr. Abid Ullah PT
Lecturer FIMS
Abbottabad
Email:
dr.abidullahpt@yahoo.com
Anatomy
 Ball and socket joint of synovial joint.
 Connects the pelvic girdle to the lower limb
 Made up of femoral head and acetabulum
 Designed for stability and wide range of movement
 Covered with a thin layer of hyaline cartilage
More Anatomy
 The articular surface of is horse-shoe shaped and is
deficient inferiorly- acetabular notch
 Has a labrum
- is a circular layer of cartilage which surrounds the
outer part of the acetabulum making the socket deeper
and so helping provide more stability
- Acetabular labral tears are a common injury from
major or repeated minor trauma
Iliofemoral Ligament
 This is a strong ligament
which connects the pelvis to
the femur
 at the front of the joint
 It resembles a Y in shape
 Stabilises the hip by limiting
hyperextension
Ligaments
 Pubofemoral ligament
 The pubofemoral ligament attaches the part of the pelvis
known as the pubis (most forward part, either side of the
pubic symphysis) to the femur.
 Ischiofemoral ligament:
 This is a ligament which reinforces the posterior aspect of the
capsule
 attaches the ischium to the two trochanters of the femur.
 Transverse acetabular Ligament:
 Bridges acetabular notch.
 Ligament of head of femur: flat and triangular in shape
 Lies within joint, ensheathed by synovium
Muscles
 Gluteals:
 Gluteus Maximus, Gluteus
Minimus and Gluteus Medius
 Attach to the Ilium and travel
laterally to insert into the greater
trochanter of the femur
 Medius and Minimus abduct and
medially rotate the hip joint, as well
as stabilising the pelvis
 Gluteus maximus extends and
laterally rotates the hip joint
More Muscles
 Quadriceps
 The four Quadricep muscles are
Vastus lateralis, medialis,
intermedius and Rectus femoris
 All attach inferiorly to the tibial
tuberosity
 Rectus femoris originates at the
Anterior Inferior Iliac Spine and acts
to flex the hip
 The 3 other Quad muscles do not
cross the hip joint, and attach
around the greater trochanter and
just below it.
Muscles
 Iliopsoas:
 The is the primary hip flexor
muscle which consists of 2
parts
 Attaches superiorly to the
lower part of the spine and
the inside of the ilium
 Cross the hip joint and insert
to the lesser trochanter of the
femur
Muscles
 Hamstrings:
 The hamstrings are three
muscles which form the
back of the thigh
 Attach superiorly to the
ischial tuberosity
 Cause hip extension
Functional Group of Muscles
Acting on the Hip
Flexors:
 Iliopsoas, sartorius, tensor fascia lata, rectus femorus, pectineus,
adductor longus, brevis, and magnus, gracilis
Extensors:
- hamstrings, addcutor magnus, gluteus maximus
Adductors:
- adductor longus, brevis, and magnus, gracilis, pectineus
Abductors:
- gluteus medius, minimus, tensor fascia lata
External rotators:
- obturator externus, internus, piriformis, quadratus femoris, gluteus
maximus
Internal Rotators:
- gluteus medius, minimus, tensor fascia lata.
Nerves
 Femoral (L2,3,4)
 Obturator (L2, 3, 4)
 Sciatic (L4,5, S1, 2,)
 WHY ARE THESE
IMPORTANT???
- Referred pain to the
knee can hide hip
pathology and vis
versa
Blood Supply
Clinical features of Hip Pathology
 Pain.
 Stiffness.
 Loss of function.
 Crepitations.
 Leg length discrepancy.
Gait
 Antalgic gait: painful, short stance phase
 Ataxic: uncoordinated walk, with a wide base &
 Double-step-gait: in which there is a noticeable
difference in the length or timing of alternate step
 Trendelenburg (abductor lurch) gait: weak abductors
 Waddling gait: bilateral weak abductors, bilateral
DDH
 Steppage gait: foot drop
 Toe-walking
 In-toeing vs out-toeing
 Others: scissoring, etc.
Hip Joint Orthopedic Tests
 Congenital Hip Dysplasia
 Congenital hip dysplasia is a condition in which the
femoral head is displaced out of the acetabular cavity.
 Often bilateral. Girls affected more often than boys.
 The acetabular cavity is shallow or more vertical than
normal.
Congenital Hip Dysplasia
 Clinical Signs and Symptoms
 Decreased hip flexibility
 Limited hip abduction
 Painless limp
 Hip pain
 Shortened extremity
Allis Test
 Procedure: Infant supine, flex the knees, Feet
should approximate one another on the table.
 Positive Test: A difference in the height of the knees
is a positive test.
 Short knee on the affected side – posterior displacement
of the femoral head or a short tibia.
 Long knee on the affected side – anterior displacement
of the femoral head or increase in tibia length.
Allis Test
Ortalani’s Click Test
 Procedure: Infant supine. Grasp both thighs with
thumbs on the lesser trochanters. Flex and abduct the
thighs b/l.
 Positive Test: Palpable or audible click is a positive
sign. The click signifies displacement of the femoral
head in or out of the acetabular cavity.
Ortalani’s Click Test
Hip Fractures
 Hip fractures occur most frequently in the elderly
population.
 Most common types are intertrochanteric and
intracapsular.
 Intertrochanteric and femoral head fractures typically
do NOT disrupt the blood supply.
 Intracapsular fractures disrupt the blood supply to the
femoral head and can lead to avascular necrosis
Hip Fractures
 Clinical Signs and Symptoms
 Hip pain
 Shortened extremity
 Externally rotated extremity
 Referred pain to medial thigh
Anvil Test
 Procedure: Patient supine. Tap the inferior
calcaneous with your fist.
 Positive Test: Local pain in the hip joint may
indicate a femoral head fracture or joint pathology.
 Pain in the thigh or leg secondary to trauma may
indicate a femoral, tibial, or fibula fracture.
 Pain local to the calcaneous may indicate a calcaneal
fracture.
Anvil Test
Hip Contracture
 A hip joint contracture is a condition of soft tissue
stiffness that restricts joint motion.
 This can be caused by immobility due to spasticity,
paralysis, ossification, bone trauma, or joint trauma.
 A frequently moved joint is unlikely to develop a
contracture deformity.
 The joint capsule, ligaments, or muscle tendon units
can be involved.
Hip Contracture
 Clinical Signs and Symptoms
 Stiff hip joint
 Limited hip range of motion
 Inability to position joint in the neutral position
 Hip joint pain on range of motion
Thomas Test Procedure: A test to determine if a patient has tightness of hip flexors
i.e rectus femoris or iliopsoas muscles.
 The patient is positioned supine on the table. The subject is then asked
to bring both knees to their chest and to hold the untested limb with
their hands. The examiner then passively lowers the test limb to the
table. If the limb remains up off the table, a hip flexor contracture is
suspected. To differentiate muscles, the examiner then passively
extends the knee placing the rectus femoris on slack. If the test limb
lowers to the table the rectus femoris is shortened. If the test limb
remains off the table with the knee flexed the iliopsoas is shortened.
 Positive Test:
 No tightness – suspect restriction at the hip joint structure or joint
capsule.
If tightness is palpated on the side of the involuntary flexed knee –
hip flexure contraction is suspected.
Thomas Test
Ely’s Test
 Structure : iliopsoas & rectusfemorus
 Procedure: Patient prone. Grasp ankle and
passively flex the knee to the buttock.
 Positive Test: If the patient has a tight rectus
femoris or hip flexion contracture, the hip on the same
side will flex, raising the buttock off the table.
Ely’s Test
General Hip Joint Lesions
 Common problems associated with the hip joint
include the following:
 Osteoarthritis, sprains, fractures, dislocations, bursitis,
tendinitis, synovitis, and avascular necrosis of the
femoral head.
 The following tests determine whether a general lesion
of the hip is present. Further diagnostic imaging can
determine the exact pathology.
General Hip Joint Lesions
 Clinical Signs and Symptoms
 Hip pain
 Shortened extremity
 Externally rotated extremity
 Referred pain to medial thigh
 Trochanteric Bursitis
• Pain over the lateral hip and possibly
• down the lateral thigh to the knee
• Discomfort after standing
• hip elevated and adducted and the pelvis dropped on
• the opposite side.
• climbing stairs aggravate the condition.
 Psoas Bursitis
• Pain in the groin or anterior thigh
• Aggravated during excessive hip flexion
 Ischiogluteal Bursitis
• Pain is around the ischial tuberosities,
• increase when sitting
Patrick Test (Faber)
 Procedure: Patient supine. Flex leg and place foot flat
on table. Grasp femur and press it into the acetabular
cavity. Cross leg to opposite knee. Stabilize ASIS opposite
and press down on knee of side tested.
 Positive Test:
 Pain in the hip – inflammatory process in the hip joint
 Pain secondary to trauma – may indicate fracture
 Pain may indicate avascular necrosis of femoral head
 Faber – Flexion, abduction, & external rotation
 If pain is elicited on the ipsilateral side anteriorly, it is
suggestive of a hip joint disorder on the same side. If pain is
elicited on the contralateral side posteriorly around the
sacroiliac join
Patrick Test (Faber)
Trendelenburg Test
 Procedure: Patient standing. Grasp waist.
Thumbs on PSIS . Instruct patient to flex one leg at a
time.
 Positive Test:
 If the patient cannot stand on one leg because of pain
 If the opposite pelvis falls or fails to rise
 This tests the integrity of the hip joint opposite the side
of hip flexion
Trendelenburg Test
Laguerre’s Test
 Procedure: Patient supine. Flex the hip and knee
to 90 degrees. Rotate the thigh outward and the knee
medially. Press down on the knee with one hand and
pull up on the ankle with the other.
 Positive Test: This test externally forces the head
of the femur into the acetabular cavity.
 May indicate an inflammatory process in the joint such
as osteoarthritis.
 Pain secondary to trauma – suspect fracture of the
acetabular cavity or rim.
Laguerre’s Test
Avascular necrosis (AVN) of the hip
(osteonecrosis)
Osteonecrosis
 also called avascular necrosis or aseptic necrosis.
 is a painful condition that occurs when the blood
supply to the bone is disrupted
 Because bone cells die without a blood supply,
osteonecrosis can ultimately lead to destruction of the
hip joint and arthritis.
 The pain localized to the groin area, but it may in the
ipsilateral buttock, knee, or greater trochanteric
region.
 Painful symptoms exacerbated with weight bearing
but are relieved by rest.
 Causes
 Atraumatic
 Excessive corticosteroid
 Alcoholism
 Other medical conditions — sickle cell disease, SLE, arterial
embolism, thombosis, and vasculitis
 traumatic
 Femoral neck fractures
 Hip dislocation, subluxation, displaced femoral neck fracture
can damage the fragile retinacular vessels, which supply the
femoral head and result in femoral head necrosis.
 Symptoms
 Osteonecrosis develops in stages. Hip pain is typically the
first symptom. This may lead to a dull ache or throbbing
pain in the groin or buttock area. As the disease progresses,
it will become more difficult to stand and put weight on the
affected hip, and moving the hip joint will be painful.
 How long it takes for the disease to progress through these
stages varies from several months to over a year. It is
important to diagnose this disease early, because some
studies show that early treatment is associated with better
outcomes.
 Passive range of motion of the hip is limited and
painful, especially forced internal rotation.
 A distinct limitation of passive abduction is usually
noted.
 A straight-leg raise against resistance provokes pain in
most symptomatic cases.
 Passive internal and external rotation of the extended
leg ("log roll test") may elicit pain that is consistent
with an active capsular synovitis.
Postoperative Management
 Immobilization
 After THA there is no need for immobilization of the
operated hip
 postoperative rehabilitation emphasizes early
movement
 the operated limb may need to remain in a position of
slight abduction and neutral rotation
 An abduction pillow or wedge typically is sufficient to
maintain the position.
 Weight-Bearing Considerations
 After cemented THA, typically patients are permitted
to bear as much weight as tolerated almost
immediately after surgery
 Cementless or hybrid THA, it is often necessary to
limit weight bearing on the operated limb for the first
few weeks or up to 3 months
 Exercise and Functional Training

Hip asseement

  • 1.
    Examination, evaluation &Assessment of Hip Dr. Abid Ullah PT Lecturer FIMS Abbottabad Email: dr.abidullahpt@yahoo.com
  • 2.
    Anatomy  Ball andsocket joint of synovial joint.  Connects the pelvic girdle to the lower limb  Made up of femoral head and acetabulum  Designed for stability and wide range of movement  Covered with a thin layer of hyaline cartilage
  • 4.
    More Anatomy  Thearticular surface of is horse-shoe shaped and is deficient inferiorly- acetabular notch  Has a labrum - is a circular layer of cartilage which surrounds the outer part of the acetabulum making the socket deeper and so helping provide more stability - Acetabular labral tears are a common injury from major or repeated minor trauma
  • 6.
    Iliofemoral Ligament  Thisis a strong ligament which connects the pelvis to the femur  at the front of the joint  It resembles a Y in shape  Stabilises the hip by limiting hyperextension
  • 7.
    Ligaments  Pubofemoral ligament The pubofemoral ligament attaches the part of the pelvis known as the pubis (most forward part, either side of the pubic symphysis) to the femur.  Ischiofemoral ligament:  This is a ligament which reinforces the posterior aspect of the capsule  attaches the ischium to the two trochanters of the femur.  Transverse acetabular Ligament:  Bridges acetabular notch.  Ligament of head of femur: flat and triangular in shape  Lies within joint, ensheathed by synovium
  • 8.
    Muscles  Gluteals:  GluteusMaximus, Gluteus Minimus and Gluteus Medius  Attach to the Ilium and travel laterally to insert into the greater trochanter of the femur  Medius and Minimus abduct and medially rotate the hip joint, as well as stabilising the pelvis  Gluteus maximus extends and laterally rotates the hip joint
  • 9.
    More Muscles  Quadriceps The four Quadricep muscles are Vastus lateralis, medialis, intermedius and Rectus femoris  All attach inferiorly to the tibial tuberosity  Rectus femoris originates at the Anterior Inferior Iliac Spine and acts to flex the hip  The 3 other Quad muscles do not cross the hip joint, and attach around the greater trochanter and just below it.
  • 10.
    Muscles  Iliopsoas:  Theis the primary hip flexor muscle which consists of 2 parts  Attaches superiorly to the lower part of the spine and the inside of the ilium  Cross the hip joint and insert to the lesser trochanter of the femur
  • 11.
    Muscles  Hamstrings:  Thehamstrings are three muscles which form the back of the thigh  Attach superiorly to the ischial tuberosity  Cause hip extension
  • 12.
    Functional Group ofMuscles Acting on the Hip Flexors:  Iliopsoas, sartorius, tensor fascia lata, rectus femorus, pectineus, adductor longus, brevis, and magnus, gracilis Extensors: - hamstrings, addcutor magnus, gluteus maximus Adductors: - adductor longus, brevis, and magnus, gracilis, pectineus Abductors: - gluteus medius, minimus, tensor fascia lata External rotators: - obturator externus, internus, piriformis, quadratus femoris, gluteus maximus Internal Rotators: - gluteus medius, minimus, tensor fascia lata.
  • 13.
    Nerves  Femoral (L2,3,4) Obturator (L2, 3, 4)  Sciatic (L4,5, S1, 2,)  WHY ARE THESE IMPORTANT??? - Referred pain to the knee can hide hip pathology and vis versa
  • 14.
  • 15.
    Clinical features ofHip Pathology  Pain.  Stiffness.  Loss of function.  Crepitations.  Leg length discrepancy.
  • 16.
    Gait  Antalgic gait:painful, short stance phase  Ataxic: uncoordinated walk, with a wide base &  Double-step-gait: in which there is a noticeable difference in the length or timing of alternate step  Trendelenburg (abductor lurch) gait: weak abductors  Waddling gait: bilateral weak abductors, bilateral DDH  Steppage gait: foot drop  Toe-walking  In-toeing vs out-toeing  Others: scissoring, etc.
  • 17.
    Hip Joint OrthopedicTests  Congenital Hip Dysplasia  Congenital hip dysplasia is a condition in which the femoral head is displaced out of the acetabular cavity.  Often bilateral. Girls affected more often than boys.  The acetabular cavity is shallow or more vertical than normal.
  • 18.
    Congenital Hip Dysplasia Clinical Signs and Symptoms  Decreased hip flexibility  Limited hip abduction  Painless limp  Hip pain  Shortened extremity
  • 19.
    Allis Test  Procedure:Infant supine, flex the knees, Feet should approximate one another on the table.  Positive Test: A difference in the height of the knees is a positive test.  Short knee on the affected side – posterior displacement of the femoral head or a short tibia.  Long knee on the affected side – anterior displacement of the femoral head or increase in tibia length.
  • 20.
  • 21.
    Ortalani’s Click Test Procedure: Infant supine. Grasp both thighs with thumbs on the lesser trochanters. Flex and abduct the thighs b/l.  Positive Test: Palpable or audible click is a positive sign. The click signifies displacement of the femoral head in or out of the acetabular cavity.
  • 22.
  • 23.
    Hip Fractures  Hipfractures occur most frequently in the elderly population.  Most common types are intertrochanteric and intracapsular.  Intertrochanteric and femoral head fractures typically do NOT disrupt the blood supply.  Intracapsular fractures disrupt the blood supply to the femoral head and can lead to avascular necrosis
  • 24.
    Hip Fractures  ClinicalSigns and Symptoms  Hip pain  Shortened extremity  Externally rotated extremity  Referred pain to medial thigh
  • 25.
    Anvil Test  Procedure:Patient supine. Tap the inferior calcaneous with your fist.  Positive Test: Local pain in the hip joint may indicate a femoral head fracture or joint pathology.  Pain in the thigh or leg secondary to trauma may indicate a femoral, tibial, or fibula fracture.  Pain local to the calcaneous may indicate a calcaneal fracture.
  • 26.
  • 27.
    Hip Contracture  Ahip joint contracture is a condition of soft tissue stiffness that restricts joint motion.  This can be caused by immobility due to spasticity, paralysis, ossification, bone trauma, or joint trauma.  A frequently moved joint is unlikely to develop a contracture deformity.  The joint capsule, ligaments, or muscle tendon units can be involved.
  • 28.
    Hip Contracture  ClinicalSigns and Symptoms  Stiff hip joint  Limited hip range of motion  Inability to position joint in the neutral position  Hip joint pain on range of motion
  • 29.
    Thomas Test Procedure:A test to determine if a patient has tightness of hip flexors i.e rectus femoris or iliopsoas muscles.  The patient is positioned supine on the table. The subject is then asked to bring both knees to their chest and to hold the untested limb with their hands. The examiner then passively lowers the test limb to the table. If the limb remains up off the table, a hip flexor contracture is suspected. To differentiate muscles, the examiner then passively extends the knee placing the rectus femoris on slack. If the test limb lowers to the table the rectus femoris is shortened. If the test limb remains off the table with the knee flexed the iliopsoas is shortened.  Positive Test:  No tightness – suspect restriction at the hip joint structure or joint capsule. If tightness is palpated on the side of the involuntary flexed knee – hip flexure contraction is suspected.
  • 30.
  • 31.
    Ely’s Test  Structure: iliopsoas & rectusfemorus  Procedure: Patient prone. Grasp ankle and passively flex the knee to the buttock.  Positive Test: If the patient has a tight rectus femoris or hip flexion contracture, the hip on the same side will flex, raising the buttock off the table.
  • 32.
  • 33.
    General Hip JointLesions  Common problems associated with the hip joint include the following:  Osteoarthritis, sprains, fractures, dislocations, bursitis, tendinitis, synovitis, and avascular necrosis of the femoral head.  The following tests determine whether a general lesion of the hip is present. Further diagnostic imaging can determine the exact pathology.
  • 34.
    General Hip JointLesions  Clinical Signs and Symptoms  Hip pain  Shortened extremity  Externally rotated extremity  Referred pain to medial thigh
  • 35.
     Trochanteric Bursitis •Pain over the lateral hip and possibly • down the lateral thigh to the knee • Discomfort after standing • hip elevated and adducted and the pelvis dropped on • the opposite side. • climbing stairs aggravate the condition.  Psoas Bursitis • Pain in the groin or anterior thigh • Aggravated during excessive hip flexion  Ischiogluteal Bursitis • Pain is around the ischial tuberosities, • increase when sitting
  • 36.
    Patrick Test (Faber) Procedure: Patient supine. Flex leg and place foot flat on table. Grasp femur and press it into the acetabular cavity. Cross leg to opposite knee. Stabilize ASIS opposite and press down on knee of side tested.  Positive Test:  Pain in the hip – inflammatory process in the hip joint  Pain secondary to trauma – may indicate fracture  Pain may indicate avascular necrosis of femoral head  Faber – Flexion, abduction, & external rotation  If pain is elicited on the ipsilateral side anteriorly, it is suggestive of a hip joint disorder on the same side. If pain is elicited on the contralateral side posteriorly around the sacroiliac join
  • 37.
  • 38.
    Trendelenburg Test  Procedure:Patient standing. Grasp waist. Thumbs on PSIS . Instruct patient to flex one leg at a time.  Positive Test:  If the patient cannot stand on one leg because of pain  If the opposite pelvis falls or fails to rise  This tests the integrity of the hip joint opposite the side of hip flexion
  • 39.
  • 40.
    Laguerre’s Test  Procedure:Patient supine. Flex the hip and knee to 90 degrees. Rotate the thigh outward and the knee medially. Press down on the knee with one hand and pull up on the ankle with the other.  Positive Test: This test externally forces the head of the femur into the acetabular cavity.  May indicate an inflammatory process in the joint such as osteoarthritis.  Pain secondary to trauma – suspect fracture of the acetabular cavity or rim.
  • 41.
  • 43.
    Avascular necrosis (AVN)of the hip (osteonecrosis)
  • 44.
    Osteonecrosis  also calledavascular necrosis or aseptic necrosis.  is a painful condition that occurs when the blood supply to the bone is disrupted  Because bone cells die without a blood supply, osteonecrosis can ultimately lead to destruction of the hip joint and arthritis.  The pain localized to the groin area, but it may in the ipsilateral buttock, knee, or greater trochanteric region.  Painful symptoms exacerbated with weight bearing but are relieved by rest.
  • 45.
     Causes  Atraumatic Excessive corticosteroid  Alcoholism  Other medical conditions — sickle cell disease, SLE, arterial embolism, thombosis, and vasculitis  traumatic  Femoral neck fractures  Hip dislocation, subluxation, displaced femoral neck fracture can damage the fragile retinacular vessels, which supply the femoral head and result in femoral head necrosis.
  • 46.
     Symptoms  Osteonecrosisdevelops in stages. Hip pain is typically the first symptom. This may lead to a dull ache or throbbing pain in the groin or buttock area. As the disease progresses, it will become more difficult to stand and put weight on the affected hip, and moving the hip joint will be painful.  How long it takes for the disease to progress through these stages varies from several months to over a year. It is important to diagnose this disease early, because some studies show that early treatment is associated with better outcomes.
  • 47.
     Passive rangeof motion of the hip is limited and painful, especially forced internal rotation.  A distinct limitation of passive abduction is usually noted.  A straight-leg raise against resistance provokes pain in most symptomatic cases.  Passive internal and external rotation of the extended leg ("log roll test") may elicit pain that is consistent with an active capsular synovitis.
  • 48.
    Postoperative Management  Immobilization After THA there is no need for immobilization of the operated hip  postoperative rehabilitation emphasizes early movement  the operated limb may need to remain in a position of slight abduction and neutral rotation  An abduction pillow or wedge typically is sufficient to maintain the position.
  • 49.
     Weight-Bearing Considerations After cemented THA, typically patients are permitted to bear as much weight as tolerated almost immediately after surgery  Cementless or hybrid THA, it is often necessary to limit weight bearing on the operated limb for the first few weeks or up to 3 months  Exercise and Functional Training