Auditory brainstem response (ABR)
Approximately 1 of every 1000 children is born deaf. Many more are born with less severe degrees of hearing impairment, while others may acquire hearing loss during early childhood.
combination of technological advances in ABR and otoacoustic emissions (OAE) testing methods are used for evaluation of hearing in newborns.
Congenital malformation of external ear and it’s managementYousuf Choudhury
Congenital malformations of external ear are the most common malformations presented to otolaryngologists and facial plastic surgeons. Hence in the pursuit of in-depth knowledge, a seminar on the topic was presented by me in the month of May 2017 at ENT-HNS dept, Silchar Medical College.
Auditory brainstem response (ABR)
Approximately 1 of every 1000 children is born deaf. Many more are born with less severe degrees of hearing impairment, while others may acquire hearing loss during early childhood.
combination of technological advances in ABR and otoacoustic emissions (OAE) testing methods are used for evaluation of hearing in newborns.
Congenital malformation of external ear and it’s managementYousuf Choudhury
Congenital malformations of external ear are the most common malformations presented to otolaryngologists and facial plastic surgeons. Hence in the pursuit of in-depth knowledge, a seminar on the topic was presented by me in the month of May 2017 at ENT-HNS dept, Silchar Medical College.
The nasal septum is the cartilage and bone in your nose. The septum divides the nasal cavity (inside your nose) into a right and left side. When the septum is off-center or leans to one side of the nasal cavity, it has “deviated.” Healthcare providers call this a deviated nasal septum.
Nasal polyps are soft, painless, noncancerous growths on the lining of your nasal passages or sinuses. They hang down like teardrops or grapes. They result from chronic inflammation and are associated with asthma, recurring infection, allergies, drug sensitivity or certain immune disorders.
This is the recent development in the surgical management of adenoid hypertrophy. traditional adenoidectomy is contraindicated as it needs proper positioning of the patient. Comparative study between the conventional versus endoscopic technique showed less blood loss and better post operative airway improvement as there is direct visualization and clearance of the airway without injuring the eustachian tube orifice
Myringotomy (from Latin myringa "eardrum") is a surgical procedure in which a tiny incision is created in the eardrum to relieve pressure caused by excessive buildup of fluid, or to drain pus from the middle ear. A tympanostomy tube is inserted into the eardrum to keep the middle ear aerated for a prolonged time and to prevent reaccumulation of fluid. Without the insertion of a tube, the incision usually heals spontaneously in two to three weeks. Depending on the type, the tube is either naturally extruded in 6 to 12 months or removed during a minor procedure.
Myringoplasty is the closure of the perforation of pars tensa of the tympanic membrane. When myringoplasty is combined with ossicular reconstruction, it is called tympanoplasty. The operation is performed with the patient supine and face turned to one side.
inflammation of the ear, usually distinguished as otitis externa (of the passage of the outer ear), otitis media (of the middle ear), and otitis interna (of the inner ear; labyrinthitis).
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
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Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
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2 Case Reports of Gastric Ultrasound
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
3. HEARING LOSS
Hearing Loss is Defined as Impairment of hearing and
severity vary from mild to moderate or Profound.
Hearing loss is Characterized By:
• Type of loss(conductive, sensory, neural)
• Locations of the problem (External ear, Middle ear,
Cochlea, auditory, nerve, central )
• Mode of Onset
• Rate of progression
• Degree on loss
• The Conditions that causes it (etiology)
• Bilateral and Unilateral
5. CONDUCTIVE HEARING LOSS
1. Negative Rinne test i.e. BC > AC.
2. Weber lateralised to poorer ear.
3. Normal absolute bone conduction.
4. Low frequencies affected more.
5. Audiometry shows bone conduction better then air
conduction with air-bone gap. Greater the air-bone
gap, more is the conductive loss
6. Loss is not more than 60bD.
7. Speech discrimination is good.
6. MANAGEMENT
1. Removal of canal obstructions.
2. Removal of fluid.
3. Removal of mass from middle ear.
4. Stapedectomy.
5. Tympanoplasty.
6. Hearing Aid.
7. TYMPANOPLASTY
It is an Operation to Eradicate disease in the middle
ear and to reconstruct hearing mechanism.
Types of tympanoplasty:
Type I : Defect perforation of tympanic membrane repaired with Graft.
Type II : Defect perforation of tympanic membrane with erosion of
malleus.
Type III : Malleus and incus are absent. Graft is placed directly on the
stapes head.
Type IV : Only the footplate of stapes is present. It is exposed to the
external ear, and Grafth is placed between the oval and
round
windows.
Type V : Fenestration Operation.
9. SENSORINEURAL HEARING LOSS
Positive Rinne test i.e. AC > BC.
Weber lateralised to better ear.
Bone conduction reduced on Schwabach and
absolute
bone conduction tests.
More often involving high frequencies.
No gap between air and bone conduction curve on
audiometry.
Loss may exceed 60 dB.
Speech discrimination is poor.
There is difficulty in hearing in the presence of
noise.
10. AETIOLOGY OF SNHL
a) Infection of labyrinth-viral
b) Trauma of labyritnth
c) Noise induced Hearing loss
d) Ototoxic drugs
e) Presbycusis
f) Meniere’s disease
g) Acoustic neuroma
h) Sudden hearing loss
i) Familial progressive SNHL
j) Systemic disorders
15. OTOTOXICITY
The Drugs and Chemicals that are Ototoxic.
Aminoglycoside Anitibiotics
Cisplatan
Salicylates
Quinine
Diueritics like fursemide, bumetide
Macrolide antibiotics
Glycopeptide Antibiotics Vencomycine
16. CLINICAL FEATURES OF OTOTOXICITY
1. Tinnitus
2. Hearing loss
3. Balance disturbances (Disequilibrium)
Investigations
1. Monitoring of the Drug concentration in the body.
2. PTA
3. Otoacoustic emission
4. BERA
17. MANAGEMENT OF OTOTOXICITY
1. Early recognition and discontinuation of the
drug.
2. Hearing AID.
3. Tinnitus should be treated with mild
hypnotics or by Tinnitus maskers.
4. Disequilibrium. Reassurance Physiotherapy
Including vestibular Exercises, avoidance of
walking in darkness, Unnecessary Head
movements.
20. TREATMENT OF PRESSBYACUSIS
Prophylaxis
Avoidance of noise
Avoidance of High fat diet
Avoidance of cold excessive smoking and stress
Psychological Support
Hearing AID
Drugs (B1,B6,B12 & Iron) may be tried in long
term to prevent deterioration of hearing
21. SUDDEN SNHL CAUSES
Only 10 to 15 percent of the people diagnosed with
SSHL have an identifiable cause. The most common
causes are:
Infectious diseases
Trauma, such as a head injury
Autoimmune diseases such as Cogan’s syndrome
Ototoxic drugs (drugs that harm the sensory cells in
the inner ear)
Blood circulation problems
A tumor on the nerve that connects the ear to the
brain
Neurologic diseases and disorders, such as multiple
sclerosis
22. INVESTIGATION OF SUDDEN SNHL
Careful History & Examination
Auditory functions test
Radiological examinations of temporal bone to rule
out acoustic neuroma
Serial viral antibody studies for VDRL, FTA
Blood glucose level for diabetes
ESR and circulating immune complexes to rule out
autoimmune pathology.
23. TREATMENT
Bed rest
Sedation to relieve anxiety and associated
giddiness
Steroid therapy
Inhalation of carbogen
Vasodilator drugs
Low molecular weight dextran
Hyperbaric oxygen therapy
Antiviral agents have been tried in patients with
suspected viral etiology.