This document provides an overview of the physical examination of the cardiovascular system. It describes examining the general appearance and various body systems including height, weight, eyes, nose, mouth, neck, skin, extremities and edema. Specific physical findings are associated with various cardiac conditions. For example, low set ears may indicate Noonan syndrome, thick lips can be seen in Hurler syndrome, and bronze pigmentation could indicate hemochromatosis with potential cardiomyopathy. The document provides a comprehensive guide to the physical exam and relating physical findings to possible underlying heart conditions.
This document provides an overview of the physical examination of the cardiovascular system. It describes examination of posture, gestures, stature, nourishment, hair, head and neck features, skin abnormalities, and extremities. Specific findings related to the head and neck include facial features, eyes, nose, ears, mouth, and neck. Assessment of jugular venous pressure and abnormalities are also covered. The document associates many physical exam findings with potential underlying cardiac conditions.
This document provides an overview of how to conduct a general physical examination, with a focus on examining the cardiovascular system. It describes examining general build and stature, posture, facial appearance, eyes, neck, spine, skin, and extremities. It also discusses common physical findings and their associations with various syndromes and diseases that can manifest cardiovascular issues, such as Marfan syndrome, Turner syndrome, and Cushing's syndrome. Examples of heart murmurs and their causes are also provided. The examination is designed to evaluate any signs, symptoms or physical clues that may indicate underlying cardiovascular conditions.
Clinical assessment of the cardiovascular system. Featured: Main symptoms and physical exam. References from Bates' guide to physical examination and history taking and Medex app.
Cor pulmonale is right ventricular heart failure caused by lung disease that leads to pulmonary hypertension. The most common cause is chronic obstructive pulmonary disease. It can be acute or chronic. Treatment focuses on managing the underlying lung condition, treating pulmonary hypertension with medications, diuretics for volume overload, anticoagulation for those at high risk of clots, and oxygen therapy. Prognosis depends on the specific lung disease, with cor pulmonale due to conditions like COPD generally having a poorer long term outlook.
This document provides details on cardiovascular examination including cardinal symptoms, chest pain characteristics, breathlessness causes, palpitations description, syncope causes, and edema types. It also describes techniques for cardiovascular auscultation including listening locations, sounds, murmur characteristics like timing, intensity location, loudness, quality, pitch, radiation, and changes with maneuvers.
The arterial pulse reflects left ventricular ejection of blood into the aorta. The pulse is determined by factors such as stroke volume, ventricular contractility, and the properties of the arteries. The central pulse contour changes as it is transmitted peripherally due to wave reflections and arterial damping. Abnormal pulses include pulsus parvus, tardus, and anacrotic pulses seen in aortic stenosis. Other abnormal pulses are dicrotic, bisferiens, collapsing, and paradoxus pulses related to various cardiovascular abnormalities. Physical examination of peripheral arteries allows evaluation of the pulse and signs of cardiovascular diseases.
This document contains mnemonics and summaries to aid in remembering various concepts in cardiology. It includes summaries of aortic stenosis characteristics, management of myocardial infarction, causes of pericarditis, heart compensatory mechanisms, distinguishing right and left heart murmurs, causes of ST elevation on ECG, Beck's triad signs, and treatment for myocardial infarction. It also includes mnemonics for jugular venous pressure causes, depressed ST-segment causes, innocent murmur features, murmur locations, cardioselective betablockers, and more.
This document provides an overview of the physical examination of the cardiovascular system. It describes examination of posture, gestures, stature, nourishment, hair, head and neck features, skin abnormalities, and extremities. Specific findings related to the head and neck include facial features, eyes, nose, ears, mouth, and neck. Assessment of jugular venous pressure and abnormalities are also covered. The document associates many physical exam findings with potential underlying cardiac conditions.
This document provides an overview of how to conduct a general physical examination, with a focus on examining the cardiovascular system. It describes examining general build and stature, posture, facial appearance, eyes, neck, spine, skin, and extremities. It also discusses common physical findings and their associations with various syndromes and diseases that can manifest cardiovascular issues, such as Marfan syndrome, Turner syndrome, and Cushing's syndrome. Examples of heart murmurs and their causes are also provided. The examination is designed to evaluate any signs, symptoms or physical clues that may indicate underlying cardiovascular conditions.
Clinical assessment of the cardiovascular system. Featured: Main symptoms and physical exam. References from Bates' guide to physical examination and history taking and Medex app.
Cor pulmonale is right ventricular heart failure caused by lung disease that leads to pulmonary hypertension. The most common cause is chronic obstructive pulmonary disease. It can be acute or chronic. Treatment focuses on managing the underlying lung condition, treating pulmonary hypertension with medications, diuretics for volume overload, anticoagulation for those at high risk of clots, and oxygen therapy. Prognosis depends on the specific lung disease, with cor pulmonale due to conditions like COPD generally having a poorer long term outlook.
This document provides details on cardiovascular examination including cardinal symptoms, chest pain characteristics, breathlessness causes, palpitations description, syncope causes, and edema types. It also describes techniques for cardiovascular auscultation including listening locations, sounds, murmur characteristics like timing, intensity location, loudness, quality, pitch, radiation, and changes with maneuvers.
The arterial pulse reflects left ventricular ejection of blood into the aorta. The pulse is determined by factors such as stroke volume, ventricular contractility, and the properties of the arteries. The central pulse contour changes as it is transmitted peripherally due to wave reflections and arterial damping. Abnormal pulses include pulsus parvus, tardus, and anacrotic pulses seen in aortic stenosis. Other abnormal pulses are dicrotic, bisferiens, collapsing, and paradoxus pulses related to various cardiovascular abnormalities. Physical examination of peripheral arteries allows evaluation of the pulse and signs of cardiovascular diseases.
This document contains mnemonics and summaries to aid in remembering various concepts in cardiology. It includes summaries of aortic stenosis characteristics, management of myocardial infarction, causes of pericarditis, heart compensatory mechanisms, distinguishing right and left heart murmurs, causes of ST elevation on ECG, Beck's triad signs, and treatment for myocardial infarction. It also includes mnemonics for jugular venous pressure causes, depressed ST-segment causes, innocent murmur features, murmur locations, cardioselective betablockers, and more.
This document provides an overview of the approach to dyspnea. It defines dyspnea and describes related terms like orthopnea. The mechanisms of orthopnea and paroxysmal nocturnal dyspnea are increased venous return when lying flat overwhelming the heart. Receptors involved in the perception of dyspnea are described. Common causes of acute and chronic dyspnea from cardiovascular, pulmonary, and other systems are listed. The approach involves assessing airway, breathing, vital signs, history, and focused physical exam. Key exam findings that can point to different diagnoses are outlined. Important investigations include spirometry, ABG, imaging, and ECG.
This document provides findings on general physical examination in respiratory diseases. It discusses various findings that may be seen in the level of consciousness, sex, attitude, build, stature, scalp and face, eyes, pallor, icterus, nose/paranasal sinuses, oral cavity, pharynx, cyanosis, neck, and signs related to tobacco smoking. Physical exam can provide clues to respiratory conditions through abnormalities found on inspection of these various body systems and areas.
The document discusses various types of valvular heart disease including stenosis, regurgitation, mitral stenosis, mitral regurgitation, aortic stenosis, aortic regurgitation, tricuspid stenosis, tricuspid regurgitation, pulmonary stenosis and pulmonary regurgitation. For each condition, the causes, consequences, clinical presentation, investigations, and management are described. Common investigations like echocardiography and treatments like valve replacement surgery are also summarized.
presentation regarding investigations and treatment of heart failure in pediatrics, including the management of an emergency , and includes brief description about even drugs used
This document discusses aortic regurgitation (AR), which occurs when the aortic valve does not close properly, allowing blood to flow backward into the left ventricle. It can be caused by damage to the aortic valve leaflets or distortion of the aorta. Common causes include rheumatic heart disease, bicuspid aortic valves, hypertension, and Marfan syndrome. Over time, the left ventricle must work harder to compensate for the backflow of blood, which can lead to enlarged and weakened heart muscles. Symptoms may include palpitations, chest pain, and shortness of breath. Diagnosis involves listening for an early diastolic murmur and confirming the diagnosis with echocardiogram
This document provides information on evaluating and diagnosing chest pain. It begins by defining chest pain and noting that it is a common reason patients present for medical care. It then discusses the causes of chest pain and provides details on distinguishing ischemic from non-ischemic chest pain. Key factors for ischemic cardiac pain are discussed such as onset during exertion and relief with rest. The document provides guidance on evaluating a patient's chest pain by taking a thorough history addressing 10 specific points. Differential diagnoses for chest pain are also reviewed.
This document provides an overview of how to examine the cardiovascular system through history, examination of vital signs, inspection, palpation, and auscultation. It details how to examine the pulse, blood pressure, jugular venous pulse, apex beat, and heart sounds. It describes normal findings as well as abnormalities that may be found and how to characterize different types of murmurs. The examination is supported by additional tests like ECG, chest x-ray, and echocardiogram.
Restrictive cardiomyopathy is characterized by stiff ventricles that do not fill properly, though systolic function is usually preserved initially. It can be caused by infiltrative diseases, fibrosis, or other processes that restrict ventricular filling. On echocardiogram, restrictive cardiomyopathy shows impaired ventricular filling and enlarged atria, while cardiac catheterization reveals elevated diastolic pressures and a distinctive "square root sign" pressure tracing. Treatment focuses on managing symptoms and underlying causes if identifiable, though prognosis is often poor without transplantation.
Chest pain can be cardiac or non-cardiac in origin. Cardiac causes include angina, myocardial infarction, and pericarditis while non-cardiac causes include pulmonary embolism, pneumonia, gallbladder disease, and musculoskeletal disorders. Characteristics of ischemic cardiac chest pain include a central, pressing or squeezing quality that radiates to the jaw/arm and is provoked by exertion. Differential diagnosis of chest pain requires evaluating characteristics like location, quality, duration and associated features to determine cardiac vs. non-cardiac etiology.
This document provides an overview of cardiac failure/congestive heart failure. It begins with an introduction and objectives. It then reviews heart anatomy and physiology, including the structure of the heart, conducting system, heart sounds, and ECG. It defines cardiac failure and discusses epidemiology, causes, pathophysiology, clinical manifestations, classifications, diagnostic process, medical management, and complications. Nursing management is also addressed using the nursing process approach.
The document provides information on performing a precordial examination, including history, examination technique, inspection, palpation, and auscultation. It discusses examining the apical impulse in detail, including location, duration, size, force, and variations such as absence, tapping, hyperdynamic, and heaving. Characteristics and causes of a displaced or diffuse apical impulse are also covered.
This document provides several mnemonics to aid in remembering cardiology concepts. It lists mnemonics for syncope causes (VASOVAGALS and THIS-MADE-ME-DAMN-VAGAL), arrhythmia causes (ACID-ME), atrial fibrillation causes (I-HAVE-A-FIB), congestive heart failure causes (ISCHEMIA-PA-CATHS and EDEMA-TOES), hypotension causes (BP-DECLINED), pericarditis causes (PR-DIP-ST-UP and IT-CAUSED-PERICARDITIS), and restrictive cardiac disease causes (A-STIFFER-CHF).
This document discusses jugular venous pulse (JVP) examination and provides information on three related signs: hepatojugular reflex, Kussmaul's sign, and pulsus paradoxus. It defines each sign and explains their pathophysiology and clinical significance. The hepatojugular reflex test evaluates right heart function by observing changes in JVP during abdominal compression. Kussmaul's sign and pulsus paradoxus involve paradoxical increases or decreases in JVP and blood pressure with inspiration, respectively, and indicate conditions that restrict right ventricular filling. The document also covers techniques for evaluating these physical exam findings.
Heart failure is a condition where the heart cannot pump enough blood to meet the body's needs. It has many potential causes, but is often due to problems with the heart muscle itself or valves. Treatment focuses on managing symptoms with diuretics, and slowing progression with ACE inhibitors, beta-blockers, and aldosterone antagonists. Other therapies aim to improve heart function or treat underlying causes. Prognosis depends on severity but ranges from 5-50% annual mortality.
A 32-year-old lady presented with shortness of breath and was found to have severe mitral regurgitation. She underwent mitral valve replacement and is now doing well. A second case report described a 55-year-old lady with a history of mitral stenosis and percutaneous transvenous mitral commissurotomy who now has severe mitral stenosis and regurgitation. She is being treated with mitral valve replacement. The document then discusses mitral regurgitation causes, pathophysiology, presentation, therapy, and acute mitral regurgitation.
This document provides information on various cyanotic heart lesions including Tetralogy of Fallot, Transposition of the Great Arteries, Tricuspid Atresia, Ebstein's Anomaly and Total Anomalous Pulmonary Venous Return. It describes the anatomy, clinical features, diagnosis and management of these conditions. Key cyanotic heart lesions are characterized by mixing of oxygenated and deoxygenated blood resulting in central cyanosis.
The document discusses the anatomy, causes, diagnosis, and management of aortic regurgitation (AR). It provides details on the location of the aortic valve, variants such as bicuspid aortic valve, and common causes of AR including rheumatic heart disease. Physical exam findings, echocardiography parameters, and indications for surgery to replace the aortic valve are summarized. Medical management including vasodilator therapy to reduce afterload is also reviewed.
1. Heart failure affects 1.5-2% of the global population and is characterized by the heart's inability to pump an adequate amount of blood to meet the body's needs.
2. Common causes include coronary heart disease, hypertension, cardiomyopathies, and arrhythmias.
3. Heart failure progresses through stages from no limitation of physical activity to severe limitations where any physical activity causes deterioration of health. Treatment aims to reduce preload and afterload on the heart.
This document provides guidance on performing a general physical examination. It outlines examination of various body systems including build and stature, nutrition, facial appearance, eyes, nose, ears, oral cavity, neck, skin, extremities, and edema. For each system, it describes relevant physical findings, conditions they may indicate, and diagnostic approaches. The focus is on performing a thorough yet efficient exam to identify any abnormalities.
This document provides information on various pediatric medical conditions organized into sections on nutritional deficiencies, rheumatology, genetic syndromes, radiology, ECGs, endocrinology, and miscellaneous topics. Key conditions discussed include Down syndrome, tuberculosis, croup, hypothyroidism, rickets, craniosynostosis, and allergic rhinitis. Diagnostic features, treatments, and complications are outlined for many common pediatric diseases and disorders.
This document provides an overview of the approach to dyspnea. It defines dyspnea and describes related terms like orthopnea. The mechanisms of orthopnea and paroxysmal nocturnal dyspnea are increased venous return when lying flat overwhelming the heart. Receptors involved in the perception of dyspnea are described. Common causes of acute and chronic dyspnea from cardiovascular, pulmonary, and other systems are listed. The approach involves assessing airway, breathing, vital signs, history, and focused physical exam. Key exam findings that can point to different diagnoses are outlined. Important investigations include spirometry, ABG, imaging, and ECG.
This document provides findings on general physical examination in respiratory diseases. It discusses various findings that may be seen in the level of consciousness, sex, attitude, build, stature, scalp and face, eyes, pallor, icterus, nose/paranasal sinuses, oral cavity, pharynx, cyanosis, neck, and signs related to tobacco smoking. Physical exam can provide clues to respiratory conditions through abnormalities found on inspection of these various body systems and areas.
The document discusses various types of valvular heart disease including stenosis, regurgitation, mitral stenosis, mitral regurgitation, aortic stenosis, aortic regurgitation, tricuspid stenosis, tricuspid regurgitation, pulmonary stenosis and pulmonary regurgitation. For each condition, the causes, consequences, clinical presentation, investigations, and management are described. Common investigations like echocardiography and treatments like valve replacement surgery are also summarized.
presentation regarding investigations and treatment of heart failure in pediatrics, including the management of an emergency , and includes brief description about even drugs used
This document discusses aortic regurgitation (AR), which occurs when the aortic valve does not close properly, allowing blood to flow backward into the left ventricle. It can be caused by damage to the aortic valve leaflets or distortion of the aorta. Common causes include rheumatic heart disease, bicuspid aortic valves, hypertension, and Marfan syndrome. Over time, the left ventricle must work harder to compensate for the backflow of blood, which can lead to enlarged and weakened heart muscles. Symptoms may include palpitations, chest pain, and shortness of breath. Diagnosis involves listening for an early diastolic murmur and confirming the diagnosis with echocardiogram
This document provides information on evaluating and diagnosing chest pain. It begins by defining chest pain and noting that it is a common reason patients present for medical care. It then discusses the causes of chest pain and provides details on distinguishing ischemic from non-ischemic chest pain. Key factors for ischemic cardiac pain are discussed such as onset during exertion and relief with rest. The document provides guidance on evaluating a patient's chest pain by taking a thorough history addressing 10 specific points. Differential diagnoses for chest pain are also reviewed.
This document provides an overview of how to examine the cardiovascular system through history, examination of vital signs, inspection, palpation, and auscultation. It details how to examine the pulse, blood pressure, jugular venous pulse, apex beat, and heart sounds. It describes normal findings as well as abnormalities that may be found and how to characterize different types of murmurs. The examination is supported by additional tests like ECG, chest x-ray, and echocardiogram.
Restrictive cardiomyopathy is characterized by stiff ventricles that do not fill properly, though systolic function is usually preserved initially. It can be caused by infiltrative diseases, fibrosis, or other processes that restrict ventricular filling. On echocardiogram, restrictive cardiomyopathy shows impaired ventricular filling and enlarged atria, while cardiac catheterization reveals elevated diastolic pressures and a distinctive "square root sign" pressure tracing. Treatment focuses on managing symptoms and underlying causes if identifiable, though prognosis is often poor without transplantation.
Chest pain can be cardiac or non-cardiac in origin. Cardiac causes include angina, myocardial infarction, and pericarditis while non-cardiac causes include pulmonary embolism, pneumonia, gallbladder disease, and musculoskeletal disorders. Characteristics of ischemic cardiac chest pain include a central, pressing or squeezing quality that radiates to the jaw/arm and is provoked by exertion. Differential diagnosis of chest pain requires evaluating characteristics like location, quality, duration and associated features to determine cardiac vs. non-cardiac etiology.
This document provides an overview of cardiac failure/congestive heart failure. It begins with an introduction and objectives. It then reviews heart anatomy and physiology, including the structure of the heart, conducting system, heart sounds, and ECG. It defines cardiac failure and discusses epidemiology, causes, pathophysiology, clinical manifestations, classifications, diagnostic process, medical management, and complications. Nursing management is also addressed using the nursing process approach.
The document provides information on performing a precordial examination, including history, examination technique, inspection, palpation, and auscultation. It discusses examining the apical impulse in detail, including location, duration, size, force, and variations such as absence, tapping, hyperdynamic, and heaving. Characteristics and causes of a displaced or diffuse apical impulse are also covered.
This document provides several mnemonics to aid in remembering cardiology concepts. It lists mnemonics for syncope causes (VASOVAGALS and THIS-MADE-ME-DAMN-VAGAL), arrhythmia causes (ACID-ME), atrial fibrillation causes (I-HAVE-A-FIB), congestive heart failure causes (ISCHEMIA-PA-CATHS and EDEMA-TOES), hypotension causes (BP-DECLINED), pericarditis causes (PR-DIP-ST-UP and IT-CAUSED-PERICARDITIS), and restrictive cardiac disease causes (A-STIFFER-CHF).
This document discusses jugular venous pulse (JVP) examination and provides information on three related signs: hepatojugular reflex, Kussmaul's sign, and pulsus paradoxus. It defines each sign and explains their pathophysiology and clinical significance. The hepatojugular reflex test evaluates right heart function by observing changes in JVP during abdominal compression. Kussmaul's sign and pulsus paradoxus involve paradoxical increases or decreases in JVP and blood pressure with inspiration, respectively, and indicate conditions that restrict right ventricular filling. The document also covers techniques for evaluating these physical exam findings.
Heart failure is a condition where the heart cannot pump enough blood to meet the body's needs. It has many potential causes, but is often due to problems with the heart muscle itself or valves. Treatment focuses on managing symptoms with diuretics, and slowing progression with ACE inhibitors, beta-blockers, and aldosterone antagonists. Other therapies aim to improve heart function or treat underlying causes. Prognosis depends on severity but ranges from 5-50% annual mortality.
A 32-year-old lady presented with shortness of breath and was found to have severe mitral regurgitation. She underwent mitral valve replacement and is now doing well. A second case report described a 55-year-old lady with a history of mitral stenosis and percutaneous transvenous mitral commissurotomy who now has severe mitral stenosis and regurgitation. She is being treated with mitral valve replacement. The document then discusses mitral regurgitation causes, pathophysiology, presentation, therapy, and acute mitral regurgitation.
This document provides information on various cyanotic heart lesions including Tetralogy of Fallot, Transposition of the Great Arteries, Tricuspid Atresia, Ebstein's Anomaly and Total Anomalous Pulmonary Venous Return. It describes the anatomy, clinical features, diagnosis and management of these conditions. Key cyanotic heart lesions are characterized by mixing of oxygenated and deoxygenated blood resulting in central cyanosis.
The document discusses the anatomy, causes, diagnosis, and management of aortic regurgitation (AR). It provides details on the location of the aortic valve, variants such as bicuspid aortic valve, and common causes of AR including rheumatic heart disease. Physical exam findings, echocardiography parameters, and indications for surgery to replace the aortic valve are summarized. Medical management including vasodilator therapy to reduce afterload is also reviewed.
1. Heart failure affects 1.5-2% of the global population and is characterized by the heart's inability to pump an adequate amount of blood to meet the body's needs.
2. Common causes include coronary heart disease, hypertension, cardiomyopathies, and arrhythmias.
3. Heart failure progresses through stages from no limitation of physical activity to severe limitations where any physical activity causes deterioration of health. Treatment aims to reduce preload and afterload on the heart.
This document provides guidance on performing a general physical examination. It outlines examination of various body systems including build and stature, nutrition, facial appearance, eyes, nose, ears, oral cavity, neck, skin, extremities, and edema. For each system, it describes relevant physical findings, conditions they may indicate, and diagnostic approaches. The focus is on performing a thorough yet efficient exam to identify any abnormalities.
This document provides information on various pediatric medical conditions organized into sections on nutritional deficiencies, rheumatology, genetic syndromes, radiology, ECGs, endocrinology, and miscellaneous topics. Key conditions discussed include Down syndrome, tuberculosis, croup, hypothyroidism, rickets, craniosynostosis, and allergic rhinitis. Diagnostic features, treatments, and complications are outlined for many common pediatric diseases and disorders.
This document provides guidance on performing a physical examination, beginning with introducing yourself to the patient and ensuring their comfort. It describes examining each body system using inspection, palpation, percussion and auscultation. Key signs to examine include vital signs, facial features, skin signs like jaundice and cyanosis, the oral cavity, hair, neck, nails, and edema. Specific diagnostic facial features and skin signs associated with various conditions are highlighted. The levels of pitting edema are defined.
This document provides guidance on performing a general physical examination in 4 steps: inspection, palpation, percussion, and auscultation. Key areas of examination include general appearance, hands, pulse, blood pressure, face, neck, and jugular veins. Abnormal findings may indicate various underlying conditions. A thorough physical exam following these steps can provide important clinical clues.
External markers of congenital heart diseaseKurian Joseph
This document lists various congenital syndromes and their associated external physical features and cardiovascular system abnormalities. Some examples included are Down's syndrome which can cause short stature, brachydactyly, and defects like atrial septal defects; Ellis-Van Creveld syndrome with short limbs, polydactyly, and defects like atrial septal defects; and Turner's syndrome with short stature, webbed neck, and defects like coarctation of the aorta and bicuspid aortic valves. Many syndromes are associated with multiple external physical anomalies and cardiovascular system defects.
Common Orofacial Syndromes in dentistry.pptxPseudoPocket
This document discusses several genetic syndromes including their causes, clinical manifestations, and dental features. Ascher syndrome is characterized by a double lip, eyelid drooping, and non-toxic thyroid enlargement. Beckwith-Wiedemann syndrome involves overgrowth and can cause a large tongue, abdominal wall defects, and tumors. Cowden syndrome is linked to PTEN gene mutations and causes skin growths on the face and hands.
This document provides guidance on performing a physical examination, beginning with introducing yourself to the patient and ensuring their comfort. It describes examining each body system using inspection, palpation, percussion and auscultation. Vital signs including pulse, blood pressure, temperature and respiratory rate should be assessed initially. Important diagnostic facial features, jaundice, cyanosis, pallor and other skin signs are highlighted. The oral cavity, hair, neck, nails, edema and clubbing are also examined. Common causes and clinical findings for different conditions are outlined for each system and sign.
Approach to a Child with Congenital Heart DiseseCSN Vittal
This document discusses congenital heart diseases (CHD), specifically ventricular septal defects (VSD). It begins by outlining the approach to diagnosing and classifying CHD. It then describes the different types of VSDs based on their anatomical location, including perimembranous, muscular, inlet, and supracristal defects. The clinical manifestations, physical exam findings, ECG patterns, chest x-ray appearances, and echocardiogram features of VSDs are discussed in detail.
Sarcoidosis was diagnosed in a 71-year-old man presenting with dyspnea, cough, and joint pain over the past month. Imaging showed randomly distributed lung nodules and interlobular septal thickening. Biopsy revealed noncaseating granulomas consistent with sarcoidosis. Sarcoidosis is typically diagnosed in younger adults but can present atypically in elderly patients with nonspecific symptoms. Imaging may also demonstrate atypical findings, making diagnosis more challenging for older patients. Treatment can still provide benefit for elderly patients diagnosed with sarcoidosis.
This document discusses cyanotic congenital heart disease (CCHD), which is defined as a cardiovascular birth defect that results in systemic arterial desaturation due to a right-to-left shunt. CCHDs can be classified based on pulmonary blood flow as having reduced, increased, or near normal flow. Common types of CCHDs with reduced pulmonary blood flow include tetralogy of Fallot and pulmonary atresia. CCHDs with increased pulmonary blood flow can present with features of congestive heart failure. The clinical approach to CCHDs involves delineating the anatomical and physiological abnormalities through assessment of anatomy, pulmonary circulation, systemic circulation, and ventricular function.
This document provides guidance on important physical examination findings related to respiratory diseases. It discusses examining the level of consciousness, physical build, voice, signs of breathing difficulty, clubbing, cyanosis, pallor, and use of accessory muscles. Specific examination points for the head, eyes, mouth, neck, chest, lungs, heart, abdomen and extremities are covered. Key signs related to conditions like obstructive sleep apnea, pulmonary embolism, lung cancer, and tuberculosis are highlighted. A general physical exam can provide clues to respiratory pathology.
This document provides an overview of the clinical examination procedures used in endocrinology. It describes the examination of general appearance including facial features, build, skin and nails. It then examines specific areas in more depth including hair, eyes, ears, mouth and various glands and organs. It outlines common endocrine-related abnormalities that may be observed in each area and their potential causes. Key syndromes and disorders are highlighted along with diagrams to illustrate certain clinical findings.
This document provides guidance on performing a head to toe examination in children. It describes examining the head, hair, scalp, fontanelles, face, eyes, mouth, tongue, ears, nose, neck, limbs, chest, spine, abdomen, and genitalia. For each body part, common abnormalities are listed such as flat or prominent occiput, microcephaly, cephalhematoma, strabismus, low set ears, cleft palate, delayed dentition, goiter, lymphadenopathy, and neck stiffness. The document provides details on assessing each body part and common clinical findings to note during a pediatric examination.
History taking & examination of cardiovascular systemgrkmedico
This document provides information on the history, complaints, symptoms, physical exam findings, and etiologies of congenital heart disease. Key points include:
- Congenital heart disease often presents in childhood or adolescence with complaints of increased precordial activity, failure to thrive, cyanosis, breathlessness, cough, and recurrent respiratory infections.
- Physical exam may reveal signs like clubbing, cyanosis, edema, irregular pulses, displaced apex beat, murmurs, and cardiomegaly.
- Etiologies include arrhythmogenic right ventricular dysplasia, cyanotic congenital heart disease, acyanotic congenital heart disease, and various genetic syndromes that are associated with an increased risk of congen
This document provides guidance on performing a physical examination of the central nervous system. It outlines examination of various body systems and signs, including attitude, level of consciousness, head, facies, eyes, ears, mouth, neck, skin, extremities, and spine. It describes abnormalities to examine for, such as craniosynostosis deformities of the head, facial palsies, ptosis, neurocutaneous lesions, clubbing, and peripheral nerve disorders. The examination aims to detect neurological deficits or systemic diseases that may involve the CNS.
This document presents the case of a 14-year-old female patient who presents with a 1-day fever and a 1-month history of a rash on her cheeks and nose with accompanying burning sensation when exposed to sunlight. Her other symptoms include mouth and nose ulcers for 1 week. Her history and examination are documented. A provisional diagnosis of systemic lupus erythematosus is made based on her presenting signs and symptoms. Relevant details on SLE including epidemiology, pathogenesis, diagnostic criteria, clinical manifestations and management are then summarized.
This document provides information on various diseases that affect equines in India. It begins with background on the equine population in India and then lists and describes several important viral diseases (Hendra, equine influenza, equine herpes virus, equine infectious anemia, African horse sickness, equine viral arteritis, West Nile fever, equine encephalitis) and bacterial diseases (glanders, strangles, tetanus, Rhodococcus equi, leptospirosis, botryomycosis). For each disease, it discusses the causative agent, transmission, pathogenesis, clinical signs, lesions, and current status or outbreaks in India. Considerable detail is provided for Hendra virus, equ
This document provides guidance on examining the head and face during a general physical examination. It outlines key aspects to evaluate for the head, eyes, conjunctiva, sclera and lens, mouth and tongue, nose and lips, and facial features. Conditions that certain clinical findings may indicate are also listed. The examination is designed to identify abnormalities, diseases, or signs of underlying illnesses.
Evaluation of cyanotic child & management of cyanoticDhanesh Bhardwaj
- The document discusses the evaluation and management of cyanotic children and cyanotic spells.
- Cyanosis is clinically determined through history, examination, and oxygen saturation probes. It can be central, peripheral, or mixed and is caused by cardiac, pulmonary, hematologic or infectious issues.
- The diagnosis involves assessing arterial oxygen saturation, hemoglobin concentration, and hyperoxia testing.
- Cyanotic spells are medical emergencies triggered by stress that cause increased respiration, deeper cyanosis, limpness and potentially convulsions or death. Treatment involves supportive measures and addressing the underlying cause. Prevention includes medications, iron supplements, and surgical correction of structural issues.
Similar to General Physical Examination in Cardiovascular Diseases (20)
Acid base disorders - acidosis alkalosis metabolic respiratoryChetan Ganteppanavar
This document discusses acid-base disorders, including:
1. It defines acid-base terminology like acid, base, pH, acidosis, alkalosis, and anion gap.
2. It describes the four main types of simple acid-base disorders: metabolic acidosis, metabolic alkalosis, respiratory acidosis, and respiratory alkalosis.
3. It provides examples of causes, clinical features, and treatment approaches for different acid-base disorders like diabetic ketoacidosis, lactic acidosis, and respiratory alkalosis.
Hepatitis C - Etiology Pathogenesis Clinical Features Diagnosis ManagementChetan Ganteppanavar
Hepatitis C is a viral infection that affects an estimated 2-3% of the world's population. It is transmitted parenterally or through exposure to infected blood or bodily fluids. Approximately 70-130 million individuals are chronically infected. While some acute cases resolve, others progress to chronic infection which can lead to cirrhosis or liver cancer over time. Diagnosis involves testing for HCV antibodies and RNA. Genotypes 1-6 exist globally. Management involves antiviral therapy with pegylated interferon and ribavirin or newer direct acting antiviral agents targeting viral proteins. Treatment aims to achieve a sustained virologic response and prevent progression of liver disease.
This document provides information on disorders of the parathyroid gland. It discusses the anatomy and histology of the parathyroid glands. It describes how parathyroid hormone is synthesized and its role in calcium regulation. It covers different types of hyperparathyroidism including primary, secondary, and tertiary. Primary hyperparathyroidism can be caused by adenomas, hyperplasia, or carcinoma. It discusses clinical manifestations, diagnosis, and differential diagnosis of primary hyperparathyroidism. It also covers hypercalcemia associated with other conditions like malignancy, immobilization, and familial hypocalciuric hypercalcemia.
Mutistep carcinogenesis refers to the process by which normal cells transform into cancerous cells through the accumulation of multiple genetic mutations over time. These mutations can be caused by environmental or inherited factors and affect genes that regulate cell growth (oncogenes) or cell cycle arrest (tumor suppressor genes). The accumulation of mutations in genes that control processes like apoptosis, cell proliferation, and DNA repair enable cells to proliferate uncontrollably and form malignant tumors.
The document provides the name, title, and place of work of an individual. Dr Chandrashekhar K is an Assistant Professor at KIMS in Hubballi, India. The document identifies an individual and their professional role and affiliation in a brief manner.
Motor neuron disease - Etiology, Pathogenesis, Clinical Features, Classificat...Chetan Ganteppanavar
This document discusses motor neuron diseases, including amyotrophic lateral sclerosis (ALS). It provides details on the classification, symptoms, signs, diagnosis, prognosis, and management of ALS and related conditions. Key points include that ALS is characterized by the degeneration of both upper and lower motor neurons, leading to muscle weakness, atrophy, and fasciculations. Diagnosis involves finding signs of both upper and lower motor neuron involvement. Prognosis is typically worse if onset is bulbar or simultaneous in multiple limbs. Treatment focuses on managing symptoms while no treatments have been proven to slow disease progression.
1. Peripheral neuropathies can involve sensory nerves, motor nerves, or both and may affect a single nerve, multiple contiguous nerves, or multiple non-contiguous nerves.
2. Common causes of peripheral neuropathy include diabetes, paraproteinemia, alcohol misuse, renal failure, and HIV infection.
3. Mononeuropathy involves a single nerve and implies a local compressive, traumatic, or vascular process. Mononeuropathy multiplex damages multiple non-contiguous nerves simultaneously through ischemia or microangiopathy.
4. Polyneuropathy presents as symmetrical distal sensory and motor deficits in a stocking-glove pattern and can be caused by diabetes, alcoholism, and vitamin B12 deficiency.
The autonomic nervous system (ANS) controls involuntary body functions through the sympathetic and parasympathetic divisions. The sympathetic division is activated during stress and prepares the body for fight or flight through increased heart rate and respiration. The parasympathetic division acts to calm the body and restore homeostasis. Dysregulation of the ANS is implicated in several psychiatric disorders like depression, anxiety, and schizophrenia through altered heart rate variability and sympathetic/parasympathetic balance. Studying ANS function provides insights into the physiological effects of mental illness and potential treatment strategies like vagus nerve stimulation.
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This document discusses enteral nutrition, including indications, contraindications, types, administration sites and tubes, feeding protocols, monitoring, and complications. The main points are:
- Enteral nutrition is indicated for inadequate oral intake of 5-7 days or inability to take oral feedings due to illnesses.
- Types of enteral nutrition include oral supplements, polymeric feeds, and disease-specific formulas.
- Administration sites are gastric or post-pyloric feeds via nasogastric, nasojejunal, or surgical tubes like PEG or jejunostomy.
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2. PDC is a multienzyme complex located in the mitochondrial matrix consisting of three enzymes and 60 subunits.
3. The citric acid cycle (TCA cycle) is the final common pathway for the oxidation of acetyl-CoA derived from carbohydrates, fatty acids, and amino acids. It operates under aerobic conditions.
Amputation is the surgical removal of all or part of a limb. There are several types and levels of amputation depending on the condition and location. The main types are non-weight bearing and weight bearing amputations. Some common levels include ray amputations of the toes, below knee amputations, and above knee amputations. It is important to properly mark the incision, ensure adequate blood supply to flaps, round bone cuts, and provide post-operative exercises.
Jaundice, or icterus, is caused by increased levels of bilirubin in the blood which deposits in tissues. It can be a sign of liver disease or hemolytic disorders. Bilirubin is produced from the breakdown of heme in red blood cells and transported to the liver where it is conjugated and excreted in bile or reabsorbed and excreted in urine. Defects in bilirubin production, uptake, conjugation or excretion by the liver can lead to jaundice. The document discusses the causes, signs, and differential diagnosis of jaundice.
Antibodies, also known as immunoglobulins, are Y-shaped proteins produced by plasma cells in response to antigens. They have antigen binding sites that recognize specific antigens with high specificity. There are five main classes of immunoglobulins - IgG, IgM, IgA, IgD, and IgE - which differ in structure, concentration in serum, location in the body, and functions such as enhancing phagocytosis, neutralizing pathogens, and initiating immune responses.
The HMP pathway, also known as the pentose phosphate pathway or phosphogluconate oxidative pathway, is an alternate pathway to glycolysis where glucose-6-phosphate is oxidized to produce NADPH and pentoses like ribose-5-phosphate. It occurs in the cytosol of liver, adipose tissue, adrenal cortex, and red blood cells. The pathway has two phases - an oxidative phase that generates NADPH and a non-oxidative phase that produces pentoses. Glucose-6-phosphate dehydrogenase, which catalyzes the first reaction of the oxidative phase, is regulated by NADPH levels. The uronic acid pathway is another oxidative pathway for glucose that produces glucuronic
Hemoglobinopathies are inherited abnormalities of hemoglobin synthesis characterized by structurally abnormal hemoglobin variants. Sickle cell anemia is the prototype hemoglobinopathy caused by a single point mutation that results in production of abnormal hemoglobin S. This leads to polymerization of hemoglobin S molecules under conditions of low oxygen, causing distortion of red blood cells into a sickle shape and various complications. Other hemoglobinopathies include Hemoglobin C, E, and D disease, which typically have milder phenotypes.
Heme is an important prosthetic group found in hemoglobin, myoglobin, and cytochromes. It is synthesized through a pathway involving 8 enzymes, with deficiencies leading to various porphyrias. The acute hepatic porphyrias involve deficiencies in enzymes from the middle of the pathway, resulting in accumulation of aminolevulinic acid and porphobilinogen that can cause severe abdominal pain, neuropathy, and psychiatric symptoms. Diagnosis involves urine and stool tests showing elevated levels of pathway intermediates. Treatment focuses on managing acute attacks and avoiding precipitating factors.
This document discusses diabetes mellitus and insulin. It defines diabetes as a metabolic disorder characterized by chronic hyperglycemia resulting from defects in insulin secretion or action. It notes diabetes is associated with risks of late complications affecting the eyes, kidneys, nerves, and cardiovascular system. The document outlines diagnostic criteria for diabetes and classifications including type 1, type 2, gestational, and secondary forms. It describes complications of diabetes such as retinopathy, kidney failure, heart disease, and neuropathy. The document discusses insulin, its structure, biosynthesis, and mechanisms of action. It provides details on six classes of oral medications used to treat diabetes, including how sulfonylureas, biguanides, thiazolidinediones, alpha-
8 Surprising Reasons To Meditate 40 Minutes A Day That Can Change Your Life.pptxHolistified Wellness
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TEST BANK For An Introduction to Brain and Behavior, 7th Edition by Bryan Kolb, Ian Q. Whishaw, Verified Chapters 1 - 16, Complete Newest Versio
TEST BANK For An Introduction to Brain and Behavior, 7th Edition by Bryan Kolb, Ian Q. Whishaw, Verified Chapters 1 - 16, Complete Newest Version
TEST BANK For An Introduction to Brain and Behavior, 7th Edition by Bryan Kolb, Ian Q. Whishaw, Verified Chapters 1 - 16, Complete Newest Version
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Muktapishti is a traditional Ayurvedic preparation made from Shoditha Mukta (Purified Pearl), is believed to help regulate thyroid function and reduce symptoms of hyperthyroidism due to its cooling and balancing properties. Clinical evidence on its efficacy remains limited, necessitating further research to validate its therapeutic benefits.
Rasamanikya is a excellent preparation in the field of Rasashastra, it is used in various Kushtha Roga, Shwasa, Vicharchika, Bhagandara, Vatarakta, and Phiranga Roga. In this article Preparation& Comparative analytical profile for both Formulationon i.e Rasamanikya prepared by Kushmanda swarasa & Churnodhaka Shodita Haratala. The study aims to provide insights into the comparative efficacy and analytical aspects of these formulations for enhanced therapeutic outcomes.
Local Advanced Lung Cancer: Artificial Intelligence, Synergetics, Complex Sys...Oleg Kshivets
Overall life span (LS) was 1671.7±1721.6 days and cumulative 5YS reached 62.4%, 10 years – 50.4%, 20 years – 44.6%. 94 LCP lived more than 5 years without cancer (LS=2958.6±1723.6 days), 22 – more than 10 years (LS=5571±1841.8 days). 67 LCP died because of LC (LS=471.9±344 days). AT significantly improved 5YS (68% vs. 53.7%) (P=0.028 by log-rank test). Cox modeling displayed that 5YS of LCP significantly depended on: N0-N12, T3-4, blood cell circuit, cell ratio factors (ratio between cancer cells-CC and blood cells subpopulations), LC cell dynamics, recalcification time, heparin tolerance, prothrombin index, protein, AT, procedure type (P=0.000-0.031). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and N0-12 (rank=1), thrombocytes/CC (rank=2), segmented neutrophils/CC (3), eosinophils/CC (4), erythrocytes/CC (5), healthy cells/CC (6), lymphocytes/CC (7), stick neutrophils/CC (8), leucocytes/CC (9), monocytes/CC (10). Correct prediction of 5YS was 100% by neural networks computing (error=0.000; area under ROC curve=1.0).
35. REVISED GHENTS CRITERIA:
1. Skeletal system
2. Ocular system
3. Cardiovascular system
4. Lungs
5. Skin
6. Dura
36. • SKELETAL SYSTEM:
MAJOR SIGNS: FEW TAPPS
1. Flat foot
2. Elbow Extension reduced (<170°)
3. Wrist and thumb sign
4. Tall stature
5. Protrusio acetabulae
6. Pectus carinatum
7. Pectus excavatum requiring surgery
8. Scoliosis >20°
37. • MINOR SIGNS:
1. Pectus excavatum of moderate intensity
2. Hypermobility of joints
3. High arched palate
4. Abnormal facial appearance
ORGAN INVOLVEMENT CRITERIA:
2 major signs or 1 major plus 2 minor signs
51. • 1. Face – Facial dysmorphism, facies, local features
• 2. Eyes
• 3. Nose
• 4. Ears
• 5.Oral cavity
• 6. Neck
B. HEAD AND NECK
52. • 1. FACIAL DYSMPORPHISM:
i. Hypertelorism
ii. Epicanthic folds
iii. Broad flat nose
iv. Low set ears
v. Thick lips
vi. Abnormal teeth
vii. Short neck
viii.Low hair line
82. SCLERA
Scleral abnormality CV condition
Icterus CHF
large pulmonary infarct
Hemolysis due to prosthetic
valves
Infective endocarditias
Drugs- ATT
Blue sclera OI
Ehler-Danlos syndrome
Marfans syndrome
Familial
98. NOSE:
Nose findings Conditions
Broad flat nose Cornelia de Lange syndrome
Down syndrome
William syndrome
Hurlers syndrome
Broad nose Acromegaly
Thin beaked nose Robinstein- Taybi syndrome
PDA VSD
99. EAR ABNORMALITIES
Ear findings Conditions
Low set ears Noonan syndrome
Turners syndrome
Klippel-Feil syndrome
Down syndrome
Cornelia de Lange syndrome
Rubintein – Taybi syndrome
Defomed ears Polychondritis
Ear lobe crease CAD
Deafness Turners syndrome
Klippel fleil syndrome
Rubella
Multiple lentigines syndrome
Jervell and Lange-Nielson
syndrome
113. TONGUE:
Tongue findings Condition
Colour-
Blue
Scarlet red
Magenta
Black
Pale tongue
Cyanosis
Niacin
Riboflavin deficiency
Actinomycosis
IDA Shock
Macroglossia Acromegaly
Myxedema
Downs syndrome
Hurler syndrome
Cretin
Glossoptosis Pierre Robin syndrome
114.
115. PALATE
Palate findings Conditions
High arched palate Marfans syndrome
Pierre Robin syndrome
Cleft/perforated palate Tertiary syphilis
Velocardiofacial syndrome
Tuberculosis
Pierre Robin syndrome
137. Xanthomas Hyperlipoproteinemia type
Palmar xanthomas Type III
Tendinous xanthomas Type II
Eruptive xanthomas Types I and V
Tuberous xanthomas Hyperlipidemia, myxedema
Liver disorders
146. 1. ABNORMALITIES OF DIGITS
Digits description Disease
a. Arachnodactyly Marfan syndrome
b. Polydactyly Ellis-van Creveld and
Laurence-Moon-Biedl
syndromes
c. Syndactyly Ellis-van Creveld and
Laurence-Moon-Biedl
syndromes
d. Clindactyly Down, Ellis-van Creveld and
Hurler’s syndromes
e. Brachydactyly Down and Turner syndromes,
hyperparathyroidism
f. Sclerodactyly Scleroderma
150. 2. ABNORMALITIEES OF THUMB
Abnormal thumbs Conditions CV abnormality
Fingerized thumbs Holt-Oram
syndrome
ASD, VSD,
Conduction defects
Single thumb like
digit
Cornelia de Lange
syndrome
PS VSD COA
Broad thumbs Rubinstein-Taybi
syndrome
ASD VSD PDA
171. REFERENCES:
1. Harrisons principle of Internal Medicine 20th edition
2. Diagnosis of Cardiovascular diseases Jonathan
Abrahams
3. Clinical examination in cardiology Vijay Raghav Rao