This document discusses the gastrointestinal tract and its components. It describes the structure and function of the oral cavity, esophagus, stomach, small intestine, large intestine, salivary glands, pancreas, liver, and their roles in digestion. Diagrams and histology slides of these organs are also included to illustrate their anatomical features. The document is presented by Prof. Dr. R.R. Deshpande and provides contact information.
Anatomy of urinary bladder. surfaces, border of urinary bladder its relation , ligament support, peritoneal relation in male and females, pouches, blood supply of bladder, nerve supply of bladder, true and false ligament of urinary bladder,
Anatomy of urinary bladder. surfaces, border of urinary bladder its relation , ligament support, peritoneal relation in male and females, pouches, blood supply of bladder, nerve supply of bladder, true and false ligament of urinary bladder,
1st Semester Anatomy - Digestive System - GIT - By thirumurugan.pptxthiru murugan
Digestive System:
Also known as gastrointestinal tract, digestive tract, digestional tract, GI tract, GIT, gut, or alimentary canal.
Consist of alimentary tract and Accessory organs
It involve in Ingestion, Digestion, Absorption and Excretion
It is started from mouth and ended at anal canal, that is assisted and supported by many parts.
Parts of Digestive System: Primary digestive organs & Accessory organs
Primary digestive organs: Mouth, Pharynx, Esophagus, Stomach, Small Intestine, Large Intestine, Rectum, Anal canal
Accessory organs: Teeth, Tongue, Salivary glands, Liver, Gallbladder, Pancreas.
Mouth
First part of GIT, Also known as oral or buccal cavity, It consist of muscles & bones
Tongue or Lingua:Voluntary muscular structure, Occupies floor of mouth
Superior surface consist of numerous papillae contains taste bud for the sense of taste. Attached inferiorly with hyoid bone, Highly mobile muscular part of GIT.
Teeth: Teeth are the hard and whitish substances present in the mouth Which is essential for chewing & speech. They fixed in socket of alveolar ridge in mandible & maxilla (Jaw). Diphyodont is a type of dentition in which two successive sets of teeth are developed during the lifetime. The first set of teeth is temporary or deciduous or milk and the other set is permanent teeth
Salivary glands: These are exocrine glands found in oral cavity that secrete complex fluid known as saliva
Types: Major & Minor salivary glands
Major salivary gland: Parotid, Submandibular & Sublingual
Minor salivary glands: There are 450 minor salivary glands present in oral cavity, lips, cheeks, palate and floor of the mouth
Pharynx: Wide, muscular tube situated behind the nose, mouth & larynx
Cavity of pharynx divided into nasopharynx, oropharynx and laryngopharynx.
Esophagus: The esophagus is a fibromuscular tube, approximately 25cm in length,
It transports food from the pharynx to the stomach.
Stomach: The stomach is a hollow organ in the GIT.
It is the “J” shaped dilated part, situated in the upper part of the abdomen.
Gross Anatomy of stomach: The stomach has four main anatomical divisions; the cardia, fundus, body and pylorus:
Cardia: surrounds the superior opening of the stomach. it consist cardiac sphincter
Fundus: the rounded, often gas filled portion superior to and left of the cardia.
Body: the large central portion inferior to the fundus.
Pylorus: This area connects the stomach to the duodenum. It is divided into the pyloric antrum, pyloric canal and pyloric sphincter.
Pancreas: The pancreas is a soft, finely lobulated, elongated dual (Exo Endocrine) gland.
Liver: It is the largest gland of the body. It involve metabolic activities
The liver is one of the vital organs of the body, responsible for chemical actions that the body needs to survive.
Small intestine: The intestine which is the longest part of the digestive tube is divided into small intestine and large intestine.
Large Intestine or Colon: It is a last part of the GIT
THIS PRESENTATION INCLUDES DETAILED INFORMATION ABOUT ACCESSORY ORGANS OF DIGESTIVE SYSTEM..i,e TEETH, TONGUE, SALIVARY GLANDS, PANCREAS, LIVER AND GALL BLADDER
assessment head, neck and mouth in critical care assessment contain anatomy way of assess, normal finding and abnormal by Dr Rezq Mansour Alqetwi critical care nursing master
1 GNM Anatomy Unit - 7 GIT by thirumurugan.pptxthiru murugan
By:M. Thiru murugan
Unit - 7
Structure and functions of the alimentary tract & is accessory organs.
The process of digestion, absorption & metabolism of food constituents
Digestive system
Also known as gastrointestinal tract, digestive tract, digestional tract, GI tract, GIT, gut, or alimentary canal.
Consist of alimentary tract and Accessory organs
It involve in Ingestion, Digestion, Absorption & Excretion
It is started from mouth and ended at anal canal
Parts of Digestive System
Accessory organs:
Teeth
Tongue
Salivary glands
Liver
Gallbladder
Pancreas
Diagram
Functions of Digestive System
Ingestion: taking of food into the GIT. i.e. eating & drinking.
Propulsion: mixes & moves the contents along the GIT
Digestion: breakdown of food
Absorption: this is the process by which digested food substances pass into the blood for circulation.
Elimination: undigested food substances removed from the GIT as feces by the process of defecation.
Mouth:
First part of GIT
The mouth is an oval-shaped cavity inside the skull.
Parts:
Lips (Labia), vestibule, mouth cavity, gums, teeth, hard & soft palate, tongue and salivary glands. The mouth is also known as the oral cavity or the buccal cavity.
Function of mouth:
Mastication (chewing) of food
Mixing masticated food with saliva
Initiation of swallowing by the tongue
Allowing for the sense of taste
Tongue or Lingua:
The tongue is a muscular organ in the mouth.
Small parts called papillae give the tongue its rough structure.
Many taste buds present on the surfaces of the papillae
Taste buds are sensory organs that are found on your tongue and allow you to experience tastes that are sweet, salty, sour, and bitter
Function of tongue:
Mastication
Deglutition
Speech
Taste
Oral cleaning
Teeth:
The teeth are the hardest white in oral cavity.
There are 20 temporary or deciduous teeth(10 in each Jaw 4 incisors, 2 canines & 4 molars)
The permanent or secondary teeth 32 teeth(16 in each jaw - 4 incisors, 2 canines, 4 premolars & 6 molars )
Function of teeth:
Chewing food
Cutting and grinding food
speech
Structure of tooth:
Crown: part protrude from gums or gingiva
Neck: in between crown & root
Root: bottom part of the tooth; may be single, double, or triple root embedded in bone
Enamel: The hardest, white outer part of the tooth.
Dentin: A layer underlying the enamel.
Pulp: The softer, living inner structure of teeth. Blood vessels and nerves run through the pulp of the teeth.
Cementum: A layer that attach the roots of the teeth to the gums and jawbone.
Periodontal ligament: Tissue that helps hold the teeth tightly in jaw.
Salivary glands:
It secreting saliva
There are 3 types
Parotid - each side of the face
Submandibular - below the mandibular
Sublingual – below the tongue
All have ducts to supply saliva in mouth
Functions: chewing and swallowing, Lubricating effect, dissolves food, taste food, Cleaning mouth, fight against pathogenic microorganisms
Pharynx:
Muscular tube situated behind th
Ayurvedic Concept of Waste Products
By Prof. Dr. R. R. Deshpande
• This PPT is very useful for students ,teachers of 1st , 2nd , 3rd ,4th BAMS
• Also useful for Medical Practitioners
• PPT includes Medicinal plants mentioned in CCIM Syllabus
• PPT Contain --- 1) Mutra ( Urine) , 2) Purisha ( Faeces or Stool ) , 3) Sweda ( Sweat ) ,4) Rasa Mal –Kapha , 5) Rakta Mal – Pitta , 6) Mansa Mal – Kha mal , 7) Meda Mal – Sweda , 8) Asthi Mal – Kesha ,Lom ,Nakha, Smashru 9) Majja Mal – Akshi & Vit –Sneha , 10) Shukra Mal –Oja
• Personal & On line classes for BAMS students are available in Marathi or Hindi or English Language
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Ayurvedic Concept of Upadhatu
By Prof. Dr. R. R. Deshpande
• This PPT is very useful for students ,teachers of 1st , 2nd , 3rd ,4th BAMS
• Also useful for Medical Practitioners
• PPT includes Medicinal plants mentioned in CCIM Syllabus
• PPT Contain --- What is Upadhatu ( Secondary Tissues) ?,Difference in Dhatu & Upadhatu ,Stanya ( Breast Milk) ,Raj ( Menstrual Discharge) ,Sira ( Blood Vessels) ,Kandara( Tendons) ,Vasa ( Fat in Meat) ,Twacha ( Skin) ,Sandhi ( Bony Joints) ,Syayu ( Aponerosis, Sphincters ,Tendons)
•
• Personal & On line classes for BAMS students are available in Marathi or Hindi or English Language
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Ayurvedic Concept of Srotas
By Prof. Dr. R. R. Deshpande
• This PPT is very useful for students ,teachers of 1st , 2nd , 3rd ,4th BAMS
• Also useful for Medical Practitioners
• PPT includes Medicinal plants mentioned in CCIM Syllabus
• PPT Contain --- Ayurvedic Concept of Srotas ,Causes for the Pathology of Srotas ,Clinical Features of Pathological Srotas ,Treatment for Pathology in Srotas
• Personal & On line classes for BAMS students are available in Marathi or Hindi or English Language
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Dravyaguna PPT
By Prof. Dr. R. R. Deshpande
• This PPT is very useful for students ,teachers of 2nd BAMS
• Also useful for Medical Practitioners
• PPT includes Medicinal plants mentioned in CCIM Syllabus
• PPT Contain --- Latin Name ,Useful part ,Type of plant –Tree or herb or creeper ,Description of leaves ,Product Picture of Plant
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Stri rog & Prasuti Tantra Question Papers
By Prof. Dr. R. R. Deshpande
This PPT has following Imp Contents – Final Year UG BAMS Question Papers of the Subject –Stri Rog & Prasuti Tantra .This is PDF .Download it & Save in your mobile .You can Zoon in & enlarge the image & read it .Very useful for Ayurved UG & PG students.
Personal & On line classes for BAMS students are available in Marathi or Hindi or English Language
visit – www.ayurvedicfriend.com
Phone – 922 68 10 630
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Dhatu Sarata
By Prof. Dr. R. R. Deshpande
This PPT has following Imp Contents – Concept of Health ,Bioenergy + Body Tissues- Supporters and pillars of our body.+ Healthy & Disease state , To keep fit –Functions of each Dhatu
What is Dhatu Sarata ? ,What is the Importance of Sarata Examination ,Importance of Saravan Dhatu ,Types of Dhatu Sarata ? ,Practical Utility of Dhatu Sarata ,Dashavidha Pariksha What is meaning of word- Sara ? ,Advice for Heena Dhatu Sarata Clinical Features of Each Dhatu Sarata ,Diction
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Virechan – Panchakarma
By Prof. Dr. R. R. Deshpande
• This PPT has following Imp Contents – 1) Introduction of Virechan 2) Causes of Pitta Vitiation 3) Pitta & Rakta –Relation ( Ashraya –Ashrayi) 4) Virechan –Indications 5) Virechan – Contraindications 6) Virechan –Benefits 7) Pre Treatment of Virechan ( Purva Karma) 8) Symptoms of Optimal Snehan or Oiling 9) Virechan – Main Procedure ( Pradhan Karma) 10) Advice after Virechan 11) Post Regimen after Virechan 12) Sansarjankram ( Specific Diet Advice) 13) Symptoms of Proper Virechan ( Samyak) 14) Symptoms of Less Virechan ( Ayog) 15) Symptoms of Excess Virechan ( Ati yog) 16) Pharmacokinetics & Pharmacodynamics of Virechan Dravyas 17) Types of Virechan 18) Drug Formulations 19) Virechan in Clinical Practice Worms Burn wounds Acne Vulgaris ,Urticaria 20) Case study
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Mechanism of Respiration
By Prof. Dr. R. R. Deshpande
• This PPT has following Imp Contents – 1) Mechanism of Respiration 2) 2 Stages of Respiration 3) Muscles of Respiration 4) Bucket Handle movement of ribs 5) Pump Handle movement of Sternum 6) Role of Expiratory Muscles 7) Role of Accessory Muscles 8) Respiration & Ayurved
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Management of Problems in Sub-types of Tri-dosha
By Prof. Dr. R. R. Deshpande
• This PPT has following Imp Contents – Health problems & Management of Sub types of Vata,Pitta & Kapha
• 5 Subt ypes of Vata –Prana,Udan,Vyan,Saman,Apan
• 5 Sub Types of Pitta – Pachak,Ranjak,Sandhak,Bhrajak ,Alovhak
• 5 Sub types of Kapha – Kledak,Bodhak,Avalambak,Schleshak ,Tarpak
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Ayurved Propagation in Germany
By Prof. Dr. R. R. Deshpande
• This PPT has following Imp Contents – 1) Munich ,German Visit in 2012 –By Prof.Dr.Deshpande 2) Details of Travelling 3) Photographs 4) Programme Details 5) Details of Preparation of –Dhanyak Him ; Panchakol Phanta ; Shatavari Kalpa ;Kshir Bala Taila ;Arjun Kshirpak
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Shalya Tantra(Surgery) –Part 1- B
By Prof. Dr. R. R. Deshpande
• This PPT has following Imp Contents –1) Sterilization
• 2) Anaesthesia 3) Positions 4) Incisions 5) Bandages
• 5) IV Fluids 6) Shock
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Shalya Tantra(Surgery) –Part 1- A
By Prof. Dr. R. R. Deshpande
• This PPT has following Imp Contents – 1) Introduction 2) Historical IMP points 3) Abnormalities at birth 4) Instruments Like -- Kidney Tray , Scalpel & Blades Different Forceps ,Retractors ,Catheters ,Ryle’s Tube Flatus Tube ,Sigmoidoscope ,Proctoscope
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Second BAMS Question Papers –Summer 2018
By Prof. Dr. R. R. Deshpande
• This PPT has following Imp Contents – MUHS - Question Papers of Summer 2018 .Subjects – Dravyaguna Paper 1 & 2 ;Rasashastra & Bhaishajya Kalpana –Paper 1 & 2 ; Agadtantra ;Charak Purvardha
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Meda Dhatu
– By Prof.Dr.R.R.Deshpande
Uploaded on 6 July 2018
This PPT is a part of First BAMS .Syllabus of Sharir Kriya .Paper 2 & Part A. Point 1 . Introduction of Dhatu .This PPT contains --- Information of Meda Dhatu
12 Points to study Physiological Aspect of Meda Dhatu –i) Name, Nirukti, Synonyms ii) Meda - Sthana or site iii) Meda -- Swarup, Sanghatan ( Structure & Composition iv) Meda -- Dhatu Prakar ( Types) v) Meda ------ Dhatu --- Tridha Parinaman ( Metabolism) vi) Meda Dhatu Poshan kal ( Time for Formation of Dhatu) vii) Meda Dhatu Guna ( Properties or Attributes) viii) Meda Dhatu Praman ( Quantity) ix) Meda Dhatu Karya ( Functions) x) Meda Dhatu Sarata ( Quality of Dhatu) xi) Mansa -- Upadhatu ( Secondary Tissuers) xii) Meda -- Dhatu Mala ( Waste Products)
2 Points to study Pathological Aspect of each Dhatu – i) Meda Dhatu Vruddhi ( Pathological Excess) ii) Meda Dhatu Kshaya ( Deficiency)
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Mansa Dhatu
– By Prof.Dr.R.R.Deshpande
Uploaded on 3 July 2018
This PPT is a part of First BAMS .Syllabus of Sharir Kriya .Paper 2 & Part A. Point 1 . Introduction of Dhatu .This PPT contains --- Information of Mansa Dhatu
12 Points to study Physiological Aspect of Mansa Dhatu –i) Name, Nirukti, Synonyms ii) Mansa - Sthana or site iii) Mansa -- Swarup, Sanghatan ( Structure & Composition iv) Mansa -- Dhatu Prakar ( Types) v) Mansa ------ Dhatu --- Tridha Parinaman ( Metabolism) vi) Mansa Dhatu Poshan kal ( Time for Formation of Dhatu) vii) Mansa Dhatu Guna ( Properties or Attributes) viii) Mansa Dhatu Praman ( Quantity) ix) Mansa Dhatu Karya ( Functions) x) Mansa Dhatu Sarata ( Quality of Dhatu) xi) Mansa -- Upadhatu ( Secondary Tissuers) xii) Mansa -- Dhatu Mala ( Waste Products)
2 Points to study Pathological Aspect of each Dhatu – i) Mansa Dhatu Vruddhi ( Pathological Excess) ii) Mansa Dhatu Kshaya ( Deficiency)
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Female reproductive system
By Prof. Dr. R. R. Deshpande
• This PPT has following Imp Contents
• Description of Organs of Female Reproductive System –Vagina,Cervix ,Uterus ,Fallopian Tubes ,Ovary ,
• Menstruation –Slide 54 to 66
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Male reproductive system
By Prof. Dr. R. R. Deshpande
• This PPT has following Imp Contents – Introduction ,Penis ,Scrotum , Testis , Spermatogenesis , Spermatozoon, Epididymis , Vas deferance ,Accessory Glands , Seminal Vesicle , Prostate Gland , Bulbourethral Glans , Spermatozoa , Spermatogenesis , Seminiferous tubules , Control of Spermatogenesis , Fate of Spermatozoa, Metabolism of Spermatozoa , Fertilization of Ovum, Semen
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Pediatrics in GP
By Prof. Dr. R. R. Deshpande
• This PPT has following Imp Contents – Basic Understanding of Paediatrics ,Normal Weight,Mile Stones, About Breast Milk ,Immunization, Marasmus, Kwashiorkor, Rickets, Pica ,Febrile Convulsions, Epilepsy,Chronic Recurrent Cough ,Bed wetting ,Causes of Crying of Baby
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GI Tract –Part 1
By Prof. Dr. R. R. Deshpande
• This PPT has following Imp Contents – 5) Histology of Digestive Tract 6) Salivary glands 7) to 19) –Functions of Liver 21) Five Secretions & Five Movements 25) Functions of Salivary Gland 33) Functions of Gastric secretions 43) Functions of Pancreatic Sectretions 53) Intestinal Juice –Enzymes 54) Functions of Large Intestine 58) Deglutition
• 65) Mastication 73) Movements of Stomach 80) Enteric Nervous system 90) Movements of small Intestine 102) Defecation Reflex 109) Stool
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2024.06.01 Introducing a competency framework for languag learning materials ...Sandy Millin
http://sandymillin.wordpress.com/iateflwebinar2024
Published classroom materials form the basis of syllabuses, drive teacher professional development, and have a potentially huge influence on learners, teachers and education systems. All teachers also create their own materials, whether a few sentences on a blackboard, a highly-structured fully-realised online course, or anything in between. Despite this, the knowledge and skills needed to create effective language learning materials are rarely part of teacher training, and are mostly learnt by trial and error.
Knowledge and skills frameworks, generally called competency frameworks, for ELT teachers, trainers and managers have existed for a few years now. However, until I created one for my MA dissertation, there wasn’t one drawing together what we need to know and do to be able to effectively produce language learning materials.
This webinar will introduce you to my framework, highlighting the key competencies I identified from my research. It will also show how anybody involved in language teaching (any language, not just English!), teacher training, managing schools or developing language learning materials can benefit from using the framework.
The Roman Empire A Historical Colossus.pdfkaushalkr1407
The Roman Empire, a vast and enduring power, stands as one of history's most remarkable civilizations, leaving an indelible imprint on the world. It emerged from the Roman Republic, transitioning into an imperial powerhouse under the leadership of Augustus Caesar in 27 BCE. This transformation marked the beginning of an era defined by unprecedented territorial expansion, architectural marvels, and profound cultural influence.
The empire's roots lie in the city of Rome, founded, according to legend, by Romulus in 753 BCE. Over centuries, Rome evolved from a small settlement to a formidable republic, characterized by a complex political system with elected officials and checks on power. However, internal strife, class conflicts, and military ambitions paved the way for the end of the Republic. Julius Caesar’s dictatorship and subsequent assassination in 44 BCE created a power vacuum, leading to a civil war. Octavian, later Augustus, emerged victorious, heralding the Roman Empire’s birth.
Under Augustus, the empire experienced the Pax Romana, a 200-year period of relative peace and stability. Augustus reformed the military, established efficient administrative systems, and initiated grand construction projects. The empire's borders expanded, encompassing territories from Britain to Egypt and from Spain to the Euphrates. Roman legions, renowned for their discipline and engineering prowess, secured and maintained these vast territories, building roads, fortifications, and cities that facilitated control and integration.
The Roman Empire’s society was hierarchical, with a rigid class system. At the top were the patricians, wealthy elites who held significant political power. Below them were the plebeians, free citizens with limited political influence, and the vast numbers of slaves who formed the backbone of the economy. The family unit was central, governed by the paterfamilias, the male head who held absolute authority.
Culturally, the Romans were eclectic, absorbing and adapting elements from the civilizations they encountered, particularly the Greeks. Roman art, literature, and philosophy reflected this synthesis, creating a rich cultural tapestry. Latin, the Roman language, became the lingua franca of the Western world, influencing numerous modern languages.
Roman architecture and engineering achievements were monumental. They perfected the arch, vault, and dome, constructing enduring structures like the Colosseum, Pantheon, and aqueducts. These engineering marvels not only showcased Roman ingenuity but also served practical purposes, from public entertainment to water supply.
We all have good and bad thoughts from time to time and situation to situation. We are bombarded daily with spiraling thoughts(both negative and positive) creating all-consuming feel , making us difficult to manage with associated suffering. Good thoughts are like our Mob Signal (Positive thought) amidst noise(negative thought) in the atmosphere. Negative thoughts like noise outweigh positive thoughts. These thoughts often create unwanted confusion, trouble, stress and frustration in our mind as well as chaos in our physical world. Negative thoughts are also known as “distorted thinking”.
Operation “Blue Star” is the only event in the history of Independent India where the state went into war with its own people. Even after about 40 years it is not clear if it was culmination of states anger over people of the region, a political game of power or start of dictatorial chapter in the democratic setup.
The people of Punjab felt alienated from main stream due to denial of their just demands during a long democratic struggle since independence. As it happen all over the word, it led to militant struggle with great loss of lives of military, police and civilian personnel. Killing of Indira Gandhi and massacre of innocent Sikhs in Delhi and other India cities was also associated with this movement.
The Art Pastor's Guide to Sabbath | Steve ThomasonSteve Thomason
What is the purpose of the Sabbath Law in the Torah. It is interesting to compare how the context of the law shifts from Exodus to Deuteronomy. Who gets to rest, and why?
How to Split Bills in the Odoo 17 POS ModuleCeline George
Bills have a main role in point of sale procedure. It will help to track sales, handling payments and giving receipts to customers. Bill splitting also has an important role in POS. For example, If some friends come together for dinner and if they want to divide the bill then it is possible by POS bill splitting. This slide will show how to split bills in odoo 17 POS.
Unit 8 - Information and Communication Technology (Paper I).pdfThiyagu K
This slides describes the basic concepts of ICT, basics of Email, Emerging Technology and Digital Initiatives in Education. This presentations aligns with the UGC Paper I syllabus.
This is a presentation by Dada Robert in a Your Skill Boost masterclass organised by the Excellence Foundation for South Sudan (EFSS) on Saturday, the 25th and Sunday, the 26th of May 2024.
He discussed the concept of quality improvement, emphasizing its applicability to various aspects of life, including personal, project, and program improvements. He defined quality as doing the right thing at the right time in the right way to achieve the best possible results and discussed the concept of the "gap" between what we know and what we do, and how this gap represents the areas we need to improve. He explained the scientific approach to quality improvement, which involves systematic performance analysis, testing and learning, and implementing change ideas. He also highlighted the importance of client focus and a team approach to quality improvement.
How to Create Map Views in the Odoo 17 ERPCeline George
The map views are useful for providing a geographical representation of data. They allow users to visualize and analyze the data in a more intuitive manner.
Ethnobotany and Ethnopharmacology:
Ethnobotany in herbal drug evaluation,
Impact of Ethnobotany in traditional medicine,
New development in herbals,
Bio-prospecting tools for drug discovery,
Role of Ethnopharmacology in drug evaluation,
Reverse Pharmacology.
1. Gastro Intestinal Tract
• Presented By –
• Prof.Dr.R.R.Deshpande
(M.D in Ayurvdic
Medicine & M.D. in
Ayurvedic Physiology)
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Sharir Kriya -- Paper 1 –Part B –Point 5
• Presented By –
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Medicine & M.D. in Ayurvedic Physiology)
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3. Sharir Kriya Paper 1-Part B –Set 2
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• Prof & HOD
• CARC ,Pune 44
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Sharir Kriya Hand Book –
1st to last year BAMS
• Best for Fast Revision
• Paper 1,Paper 2
• Practicals
• Instruments
• Histology
• IMP Schlok
• All basics of
Dodha,Dhatu & Mala
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Sharikriya Paper Practical Book
• As per Very New
Syllabus formed By
CCIM IN 2012
• Ayurvedic Practicals
like Prakruti,sara,Agni
• Modern
Haematological
Practicals
• CNS & CVS
Examination
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Clinical Examination
• Systemic Examination
of 8 systems
• Ayurvedic Srotas
Examination
• Clinical significance of
Lab Tests &
Radiology,USG,2D
Echo
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Sharir Kriya Paper 1
• Book in English
• Total CCIM Syllabus
covered
• Chaukhamba Sanskrit
Pratisthan Publication
• Popular Nationwide &
In Germany also
• Dosha & Prakruti
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Sharir Kriya Paper 2
• Book in English
• Total CCIM Syllabus
covered
• Chaukhamba Sanskrit
Pratisthan Publication
• Popular Nationwide &
In Germany also
• Dhatu,Mala
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Prof.Dr.Deshpande’s
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7/10/2016 Prof.Dr.R.R.Deshpande 10
11. 7/10/2016 Prof.Dr.R.R.Deshpande 11
Digestion
• The food, after ingestion, passes along the
various parts of the digestive tract ---
• Where it is converted into simple
absorbable constituents.
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Structure of Digestive Tract
• 1) Oral cavity is lined by stratified epithelium.
• 2) From oesophagus onwards, the digestive
tract on transverse section show 4 layers -
• Serous layer (outermost)
• Muscular layer consists of longitudinal & circular
muscle fibers
• Submucous layer
• Mucous layer.
17. 7/10/2016 Prof.Dr.R.R.Deshpande 17
Structure & appearance of teeth
• A tooth consists of a crown, a neck & a
root.
• The crown is the portion projecting above
the gum.
• The slight constriction where it is
surrounded by the gum margin is the neck
& the part buried in the alveolus of the jaw
is the root
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Structure & appearance of teeth
• The main mass of the tooth consists of a
substance called dentine, a very hard
material which resembles bone.
• The exposed portion of the tooth is
covered with a thin layer of enamel,
which is even stronger than dentine.
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Structure & appearance of teeth
• The interior of a tooth is hollow & is called
the pulp cavity.
• This contains soft connective tissue, small
blood vessels & nerves, which enter the
root of the tooth through a fine canal.
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Structure & appearance of teeth
• The teeth are firmly fixed in the alveoli of
the jaw ---
• By a special calcified cement substance &
a strong layer of connective tissue called
the periodontal membrane
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Structure & appearance of teeth
• The incisor teeth have a crown consisting of a
sharp cutting edge, & a single pointed root.
•
• The canines have a crown, which terminates in
a point & a single root.
22. Structure & appearance of teeth
• The premolars have a single root & a
crown consisting of 2 elevations or cusps
(which explains why they are sometimes
called bicuspids)
• The molars have a square - shaped
crown with 4 cusps. The upper molars
possess 3 roots, the lower 2 roots.
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Structure & appearance of teeth
• The incisors are the sharp cuttings, or
biting, teeth situated in front.
• To the lateral side of the incisors are the
canine teeth (eye teeth), used for
grasping.
• Behind the canines are the premolars &
the molars, or grinding teeth.
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Stomach
• Stomach has a J - shaped structure.
• It communicates above with oesophagus
through cardiac orifice & below with duodenum
through pyloric orifice.
• It’s main parts are fundus, body & pylorus.
•
32. Stomach
• The body is bound by lesser & greater
curvatures.
• The lesser curvature extends from cardiac
end to incisuria angularis.
•
• The greater curvature extends from fundus
to pylorus
7/10/2016 Prof.Dr.R.R.Deshpande 32
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Small Intestine
• Extend from gastric pyloric orifice onwards up to
ileo colic junction which is guarded with ileocolic
sphincter.
• Small intestines are about 6. 5 meters in length,
first 20 - 25 cm portion is formed by the
duodenum, about 2/5 th by jejunum & 3/5th by
ileum.
34. Small Intestine
• The duodenum & jejunum are mainly
concerned with digestive processes,--
• While the main function of ileum is
absorption (Hence it is richly supplied
with villi)
7/10/2016 Prof.Dr.R.R.Deshpande 34
37. Histology of Small Intestine
• Slide - Serous, muscularis (Longitudinal,
circular),submucosa, mucosa, villi, crypts
of Liberkuhn
• Mucosal folds = villi. Villi - Lined with
ciliated columnar epithelium.
• At base of mucosa - Intestinal glands
(Crypts of Liberkuhn).
• In mucosa - Goblet cells,
Enteroendocrine glands.
7/10/2016 37Prof.Dr.R.R.Deshpande
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Large Intestine
• It is about 1. 5 meters in length & is called as
large intestines because, it has large diameter
than small intestines
•
• It extends from ileo - colic junction to anus.
40. Large Intestine
• Its different parts are ----
• caecum (and appendix),
• ascending colon, hepatic flexure, transverse
colon, splenic flexure, descending colon, iliac
colon,
• sigmoid colon & anal sphincter (involuntary in
function)
• & at the lower end at anal canal is external anal
sphincter (Voluntary in function)
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Important glands
in connection with digestion
• 1) Salivary glands
• Salivary glands secrete saliva. 3 pairs of salivary glands
are
• submaxillary (submandibular) glands open through
Wharton duct at side of frenum of tongue at the floor of
the mouth
• parotid gland - open through stenson’s duct to
opposite the 2nd upper molar tooth,
• sublingual glands open through ducts of Rivinus (10 -
12 ducts) at side of frenum of tongue, at the floor of the
mouth.
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2) Pancreas
• It is an elongated racemose gland; situated at
posterior wall of the abdomen.
• Pancreas secretes pancreatic juice & also it
has alpha & beta cells of islets of Langerhans.
• The beta cells secrete insulin & alpha cells
glucagon.
• Pancreatic juice contains digestive enzymes
(trypsin, chymotrypsin)
51. Histology of Pancreas
• Slide - Pancreatic acini, Islets of
Langerhans - Alpha and beta cells.
• Pancreas - Both Exocrine and Endocrine,
Divided into Lobes,
• Pancreatic Acini → secrets Pancreatic
Juice, Cells of Islets of
• Langerhans → Beta cells - Insulin, Alpha
cells → Glucagon.
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Liver
• It is an important & essential metabolic
organ of the body.
• The liver cells secrete bile.
• Although bile is not a digestive juice, in its
strict sense, it is helpful, rather essential,
for digestion & absorption of fats, hence it
is considered along with digestive juices.
56. Histology of Liver
• Slide --- Central vein, Hepatocytes, Branch of
Hepatic Artery and portal vein + Bile duct;
Kupffer cells, sinusoid, Bile canaliculi.
• Liver - Largest gland - covered with
connective tissue sheath (Glisson’s sheath)
• Bile - Formed inside the Hepatocytes.
• Sinusoids - Contain R.E cells (Kupffer cells)
• Functions of Liver -- Formation of Bile,
phagocytosis by Kupffer cells, Vitamin-Ironfat
• storage.
7/10/2016 56Prof.Dr.R.R.Deshpande
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Gall bladder
• It stores & concentrates the bile
received from liver via hepatic duct.
• Through cystic duct it joins hepatic duct
to form common bile duct, which in turn
joins pancreatic duct to form ampulla of
Vater guarded by “Sphincter of Oddi”,
which relaxes after fatty meal to allow
drainage of bile in the duodenum
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Functions of Liver -1
• 1) In Connection With Blood & Circulation
• RBC formation, in foetal life.
• RBC destruction in adult life.
• Store house of blood & regulates blood volume.
• Manufactures prothrombin & fibrinogen &
thus essential for clotting. Mast cells form
heparin & prevent intravascular clotting.
61. 7/10/2016 Prof.Dr.R.R.Deshpande 61
Functions of Liver -2
• Related to activity of its R.E. system in immune
mechanism.
• It transfers blood from portal to systemic
circulation.
• Manufactures all plasma proteins.
• Stores iron, haematinic factor known as Vit.
B12 & copper & thus helps in the formation of
red cells & haemoglobin.
• Hepatic & portal circulation control
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Functions of Liver - 3
• 2) Manufactures Bile
• Bile is secreted continuously from the liver cells & stored
in the gall bladder. Bile contains water, total solid, mucin
& pigments, neutral fat, fatty acids, phospholipids,
cholesterol & inorganic ions.
• Bile acids in conjugation with glycerin & taurine form the
compounds - bile salts - glycochloric acid & taurocholic
acid respectively. Bile salts perform important
function of emulsification of fats
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Functions of Liver - 4
• 3) Relation With Carbohydrate Metabolism
• Converts non glucose monosaccharides into
glucose.
• Converts lactic acid, pyruvic acid & glycerol into
glucose & also glycogen.
• Stores carbohydrates in the form of
glycogen.
• Takes part in blood sugar regulation.
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Functions of Liver - 5
• Manufactures fats from carbohydrates
etc.
• Alcohol metabolism - The liver is the
main seat of alcohol metabolism. The
direct effect of alcohol may be alcoholic
fatty liver, which is the cause of increased
hepatic fatty acid oxidation.
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Functions of Liver - 6
• 4) Relation With Fat Metabolism
• It stores fat.
• It helps in the oxidation of fat, releasing
energy in the form of A.T.P.
• Site of synthesis of cholesterol from
acetate.
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Functions of Liver - 7
• Synthesizes fats from carbohydrates &
proteins.
• It is the seat of ketone body formation.
• Unused free fatty acid (FFA) released from
fat depot is converted to triglycerides &
other lipids to meet energy requirement
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Functions of Liver - 8
• In a carbohydrate deficiency, the fat
metabolism in the liver is increased & fat is
partially converted to glucose or glycogen.
•
• Fat soluble vitamins, eg. - A, D, E & K
are stored here
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Functions of Liver - 9
• 5) Relation With Protein Metabolism
• Synthesis of some amino acids takes place
here.
• Plasma proteins are manufactured here
except immune globulin.
• Main seat of urea & uric acid formation.
• It is the seat of specific dynamic action of
protein.
• It is the seat of nitrogen metabolism
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Functions of Liver - 10
• 6) Hormone Metabolism
• Reduces the circulating adrenal cortical &
sex hormones by degradation &
conjugation.
• Inactivation of insulin, glucageon, anti -
diuretic hormones (ADH) & anterior
pituitary tropic hormones etc. occur here.
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Functions of Liver - 11
• 7) Relation With Vitamins
• Manufactures prothrombin with the help of vit. K.
• It forms vit. A from carotene & stores vit. A & D.
• Chronic liver disease is always associated with
folic acid deficiency.
• The liver is the principal storage organ for
vit. B12.
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Functions of Liver - 12
• 8) Excretory Functions
• Certain heavy metals are temporarily fixed
by the liver cells, which are then excreted
in the bile.
• Various toxins, bacteria & drugs are
excreted through bile.
• Cholesterol & bile pigments are excreted
in the bile.
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Functions of Liver - 13
• 9) Detoxicating & Protective Functions
• The liver is the site of detoxication of
different toxic substances either produced
in the body or taken along with food.
• 10) Takes Part In Heart Regulation
• The liver produces a large amount of heat
& takes part in heart regulation.
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Functions of Liver - 14
• 11) It is also responsible for the liberation
of a depressor principle.
• 12) It is the store house of Fe, fat
soluble vitamins, glycogen, labile
protein, fat etc.
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Digestive system
• Functional term that differentiate between
dead & living body i.e. metabolism.
• 1) Accumulation or storage of energy is
called as anabolism
• 2) Loss of energy - catabolism
• Due to catabolism, our body is able to
perform physical & mental activities.
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Digestive system
• Let us try to understand, how energy enters in
living body, how it get stores, or loosed by body
(Energy turn over) ?
• The human body gets the energy from food.
Actually sunrays, which are electromagnetic
radiation, are the true source of energy in world.
• Human living cells do not have the capacity
for absorbing energy of sun rays & form the
food by themselves.
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Digestive system
• Plants capacity of absorbing energy of sun rays is mainly
due to green pigments called as chlorophyll.
• Chlorophyll molecules take CO2 & water from air & soil
respectively & they are tied together to form a compound
carbohydrate.
• This binding function is done with the help of energy
from sunrays.
• So radiation energy is converted into material form
of food, which a human being can use as a source of
energy for performing physical & mental activities.
78. 7/10/2016 Prof.Dr.R.R.Deshpande 78
In Plant -- Polysaccharides
• Glucose is called as monosaccharides
(Glucose).
• In plant, chain of glucose molecules is
formed (to store more energy) which are
called as polysaccharides
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Proximal Principles of Food
• 1) Our food is mainly made up of
polysaccharides or complex carbohydrate
• 2) Binding carbohydrates with nitrogen
molecule, higher energy molecules of
proteins are formed.
• 3) Fats are very high energy molecules
which are formed by combining
carbohydrates with fatty acid (COOH)
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Complex to Simpler molecules
• For performing body activities, the food that is
complex molecules should be broken up for
absorption purpose. In digestive system, due to
action of different enzymes & chemicals (Bile,
HCL) complex molecules break up & simpler
molecules are formed
• Carbohydrates → Glucose
• Protein → Amino acid
• Fats → Fatty acid & glycerol
81. 7/10/2016 Prof.Dr.R.R.Deshpande 81
Functions of CVS & RS
• This simpler molecule should reach to
different cells of body. This transport is
done through blood. Proper blood
circulation is maintained by
cardiovascular system.
• These molecules are oxidized with oxygen
to liberate energy. O2 for oxidation is
provided by respiratory system.
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Role of systems
• Waste product formed during oxidation
are carried back to kidneys, lungs, skin
through excretory system.
• The total regulation & co - ordination of
all these metabolic activities is done by
nervous system & endocrine system
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How to study ?
• To study the physiological aspect, we
have to consider ---
• Composition,
• Function &
• Regulation.
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Salivary secretions
• 1) Tubo acinar glands
• Acini - lobule - columnar epithelium
manifacting of enzymes
• Duct - cuboidal epithelium - secretes
bicarbonates - HCO3-
• 2) Blood group determination from saliva
is important in medicolegal cases.
95. 7/10/2016 Prof.Dr.R.R.Deshpande 95
Salivary secretions
• 3) Decomposition of carbohydrates forms
acid.
• This acid medium will dissolves the
calcium & caries of teeth will develop.
• To avoid this, function of saliva (killing
of microorganism) is important
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Salivary secretions
• In the mouth partial digestion of
carbohydrates take place as follows –
Starch is converted → Dextrin &
maltose
• Polysaccharide → Diasaccharide
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Salivary secretions
• This digestion is completed by pancreatic
amylase.
• The importance of proper chewing food lies in
above concept.
• If we do not give the proper time for action of
salivary amylase there will be burden on
Pancreas.
• Naturally functional capacity of Pancreas will be
reduced at early stage of life & patient will land
into Diabetes mellitus.
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Salivary secretions
• 5) Parasympathetic nerves are
“secretomotor” in action in G.I. tract i.e.
they will increase secretion & also
motility.
• These nerves stimulate acini.
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3 Phases
• a) Cephalic phase - saliva increases after thought of
food or sight of food.
• b) Oral phase - It is most important. It is due to contact
of food with Taste buds. Signals pass to brain from these
Taste buds (Gustatory pathway)
• c) Gastric phase - Presence of food in stomach
increases salivary secretions. After eating spicy food,
saliva increases, as a protective mechanism
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Salivary secretions
• 6) a) Mumps virus has specific affinity to
salivary cells.
• b) Hyperthyroidism - serum calium level
increases.
• Hence stone formation takes place in may
places in body.
• (Gall stones, salivary stones, &
pancreatic stones)
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Pepsinogen to Pepsin
• 1) Salivary glands are 3 in number but
gastric glands are many.
• 2) Pepsinogen (Gen = genesis) is
inactivated form. Due to HCl only, it is
converted into active form i.e. pepsin
• 3) Ferric (Fe3+) cannot be absorbed. Due
to effect of HCl it is converted into ferrous
form (Fe++) which can be absorbed.
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Vitamin C with Iron
• 4) Ferrous iron can only be absorbed &
this form is needed for hemoglobin
formation.
• Iron preparation is always given with
vitamin C (ascorbic acid) vitamin C keeps
iron in ferrous form.
• 5) Proteins that we eat are polypeptide
(amino acid linkage) Due to pepsin break
down of this linkage takes place
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Mucin prevents ulcer
• 6) Mucin is alkaline & sticky. It forms the
buffer coat on gastric mucosa.
• In gastric mucosa rapid mitosis takes
place.
• Above 2 facts prevent ulceration in
stomach (Peptic ulcer)
• 99% of ulcers occur in duodenum because
there is no protective mechanism.
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Mid Night Pain
• 7) Presence of food & distention of
stomach causes afferent sensation. Hence
vagotomy is the surgical treatment in
peptic ulcer (afferent & efferent both are
cut)
• 8) In duodenum ulcers mid night pain is
common (i.e. after 2 ½ to 3 hr. after eating
pain starts. That means when food comes
in duodenum from stomach pain starts.)
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Gastric Emptying Time
• 9) HCl maintains appetite hence hyper acidity
patients eat frequently. In hyper acidity patient
gastric emptying time is also shorter.
• 10) Gastric moments are affected by type of
food
• More acidic food - fast emptying time
• More carbohydrates or protein - faster emptying
time
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Fats delay Emptying Time
• More fats - delayed emptying time
• Reason - Due to fatty food CCK hormone
is stimulated. This hormone inhibits
gastrin, hence motility is reduced
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Investigations
• 11) For testing of stomach functioning
following 3 tests can be done
• Gastric analysis (Out dated)
• Barium meal X - ray
• Gastroscopy - by Gastroenterologist
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Treatment for hyperacidity
• 1) Milk neutralizes HCl → thus milk diet is important.
• Anticholinergic drugs
• 2) which block vaso vagal action.
• 3) Histamine blocking drugs (H2 Receptor Blockers)
• Tab omez (Omeprazole) - 1BD
• Tab Histac - 1 BD
• When there is pyloric stenosis (complication of ulcer)
Surgical treatment is ‘gastrojejunostomy
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Pancreatic Enzymes
• 1) Enterokinase enzyme is present in
Duodenum (intestinal juice)
• Which converts inactive enzyme (Trypsinogen)
into active form (Trypsin)
• 2) In pancreas, enzymes are not in active form -
Due to trypsin inhibitor.
• Enzymes get activated when they come in
duodenum
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Acute Pancreatitis
• 3) When Trypsin inhibitor is absent Auto
digestion of Pancrease takes place.
• Exocrine & endocrine functions lost - which is
irreversible.
• This condition is called as Acute Pancreatitis.
• This case is of acute abdomen.
• Patient get severe stabbing pain in the
epigastric region, referred to back.
• Alcohol consumption is predisposing factor
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4) Bile secretion
• Bile is formed in Liver &
• Stored in gall - bladder &
• Through the bile duct it enters into
duodenum
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Bile -- Contents
• i) Bile salts (Sodium taurocolate,
Sodium glycolate)
• ii) Bile pigments (Bilirubin & Biliverdin)
• iii) Cholesterol (ester)
• iv) HCO- 3 (Na & K)
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Bile - Function
• Emulsification of fats (Big fat molecules
are broken down into smaller particles.)
Hence surface area increases, so action of
Lipase becomes easier)
• Note - In urine examination test for Bile
salt (Sulphur test) is based on this
information.
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Control of bile secretion &
expulsion of bile from Gall bladder
• Due to CCK hormone, Gall bladder contraction
increases.
• Note - Bile does not contain any enzyme
• 1) But still it is very necessary for fat digestion (for action
of Lipase enzyme) by emulsification of fat
• 2) Pathologically - Gall stones can cause Obstructive
jaundice
• Gall stones are common in fat, fertile, female of forty
complaining of flatulence. (Remember as ‘5 F’)
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Large intestinal functions 1
• 1) Absorption of H2O, electrolytes (Cl, HCO-3)
• 2) Colonic bacteria - They are useful for production of
Vit. B12 & Vit. K. These lactobacillus are the example of
symbiosis.
• Due to antibiotics these intestinal bacterial flora gets
disturbed so antibiotics should be supplemented with
vitamin tablets.
• Buttermilk contains these lacto bacillus so this is best
diet in patients of Colitis.
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Large intestinal functions 2
• 3) Movements of large intestine push
undigested food ahead into the rectum.
• After filled with faecal matter signal goes
through spinal cord to the brain
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Large intestinal functions
• Dorsal column sensory tract → Parital lobe → Frontal
lobe → Motar centre of evacuation.
• It will decide according to place & condition, whether to
evacuate or not.
• Then signal from pyramidal tract come to spinal cord.
•
• From spinal cord parasympathetic fibers contract
rectum.
• Through CNS motar signals to external sphincters are
given, to open it.
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Movements of GI tract
• Deglutition
• 1) Process of swallowing of food.
• 2) Phases ---
• i) Oral phase
• ii) Pharyngeal phase
• iii) Oesophageal phase.
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i) Oral Phase
• Masticated food is mixed with saliva.
• Bolus is pushed back to pharynx, by
movement of tongue.
• This phase is voluntarily controlled by
muscle of tongue. (Supplied by
Hypoglossal nerve)
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ii) Pharyngeal phase - Involuntary
• Events are
• a) Elevation of trachea.
• b) Epiglottis falls on trachea, to close it.
• c) Nasopharynx is closed by contraction of muscle of soft
palate.
• d) Pharyngo - oesophageal sphincter opens.
• e) Vocal cords are approximated.
• f) In all these events, respiration stops temporarily
(Deglutition Apnoea)
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Deglutition - Control
• Reflex mechanism.
• Presence of food in pharynx, stimulate touch
receptors. Afferent impulses travel via 9th, 10th
& 5th cranial nerves to the brain stem.
• Deglutition center is situated in medulla.
Efferent impulses come through 9th, 10th & 11th
cranial nerves & bring about synchronized
events of this phase
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iii) Oesophageal Phase
• Involuntary phase 2 components
• a) Primary peristalsis - due to activity of
pacemaker at pharyngo - oesophageal center.
•
• b) Secondary peristalsis - presence of food
causes distention of oesophagus.
• Afferent impulses travel via Vagus nerve. From
Vagus center efferent impulses come via Vagus
nerve & increase peristalsis.
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Deglutition
• When food reaches cardiac sphincter, it
opens by receptive relaxation.
• Food enters into stomach & sphincter
closes.
• This prevents regurgitation.
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Deglutition - Pathology
• Dysphagia - Difficulty in deglution
• Main cause - Cancer of oesophagus. Also occurs in
‘Achalasia cardia - when cardiac sphincter is not relaxing
properly.
• Regurgitation of food from nose or into Trachea
• (Causing coughing reflex)
• This can happen if nerves are damaged near pharynx -
Pharyngeal phase cannot occur in synchronized manner
(eg. Diphtheria)
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2) Mastication
• Definition - Process of grinding of food
under teeth.
• Teeth functions - Incisors - To cut the
food.
• Molars & premolars - Mastication
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Function of Mastication
• Big food particles are converted to small
particles.
• Surface area increases & digestive
enzymes can act better.
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Muscles of mastication
• 1) Temporalis - Originates from temporal
bone & is inserted on mandible. Fan
shaped muscle.
• 2) Masseter - Originates from maxillary
part of bone & is inserted on mandible.
• Functions - These muscles when contract
upward, movement of jaw takes place
which helps in grinding
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Muscles of mastication
• 3) Medial & lateral Pterygoid - Located
on inner side of mouth.
• Due to these muscles side to side
movement of jaw occurs which helps in
grinding.
• All these muscles are supplied by -
Mandibular branch of ‘Trigeminal nerve’
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Mastication Reflex
• This is stretch reflex.
• Due to presence of food bolus in the
mouth jaw drops down.
• Stretch receptors in the muscles of
mastication are stimulated.
• Afferent impulses go through Trigeminal
nerve - to its motor nucleus in pons.
145. Mastication Reflex
• Efferent impulses again via mandibular
nerve give signals to the muscle to
contract. Jaw is elevated.
• Process occurs again & again
automatically.
• This process can also controlled
voluntarily, by motor cortex. We can stop
or start the act of mastication
voluntarily.
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146. Mastication – Pathology 1
• Injury or pain or stiffness of TM joint -
movement of mastication suffers.
• In Tetanus there is a spasm of muscles of
mastication - leading to ‘locked jaw’ &
mastication reflex suffers.
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147. Mastication- Pathology 2
• If molars, premolars are in inadequate
number - i.e. in the old age → mastication
suffers.
• If tongue is injured, mastication suffers
because for mastication, movements of
tongue are helpful to push the food under
teeth.
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148. 3) Movements of stomach
• When food enters into stomach it
undergoes receptive relaxation.
• Then food is stored.
• Mixing waves & propulsive waves
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149. 3) Movements of stomach
• Mixing waves are initiated at rate of every
20 sec. & are due BER (Basic Electrical
Rhythm). This helps to mixed the food
with gastric juice.
• Then waves start moving down from
fundus to pylorus. They become powerful
which increases pressure of antral
contents.
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150. Emptying of stomach
• Antral peristalsis (Pyloric pump) &
relaxation of pyloric sphincter, releases
gastric contents to duodenum.
• In stomach food is stored for 2 to 2&1/2
hrs.
• It is called as “Gastric emptying time”.
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151. Emptying of stomach
• It can be studied by ‘Barium Meal
examination (BA swallow)
• Waves of BER, become 5 to 6 times
powerful which push fluid food (Chyme)
towards pylorus.
• This is called ‘Pyloric pump activity’.
Emptying of stomach is proportionate to
the volume of food
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152. Factors, which modify, gastric emptying
• Stomach factors
• Food in the stomach try to hasten gastric
emptying.
• Stimulation of vagus increases pyloric
pump activity & also causes relaxation of
sphincter.
• Hormone gastrin also has the same effect
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153. Factors, which modify, gastric emptying
• Duodenal factors
• With feed back inhibition, distension of
duodenum stimulates local nerve
plexuses, which inhibit gastric emptying.
• Duodenal hormones like GIP & CCK - PZ
cause inhibition of gastrin & inhibit
gastric emptying
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154. Factors, which modify,
gastric emptying
• Fatty food -- Delays gastric emptying.
• Chilly food, Protein food has got quick
emptying effect.
• In pyloric stenosis (which is a complication
of chronic duodenal ulcer) Gastric
emptying is delayed.
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155. Enteric nervous system
• Nerve supply to Gastrointestinal Tract
• There are 2 types of Nerve supply
• 1) Intrinsic Nerve supply
• 2) Extrinsic Nerve supply
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156. 1) Intrinsic Nerve supply
• Enteric Nerve system is present within the wall
of Digestive tract from oesophagus to anus.
• Nerve fibers form 2 networks as
• i) Auerbach Plexus & ii) Meissner Plexus.
These plexuses contain stretch & chemo
receptors .
• Enteric Nervous system is controlled by Extrinsic
nerves.
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157. 1) Auerbach Plexus
• Called as Myenteric Nerve plexus.
• It is present in between inner circular muscle layer &
outer longitudinal muscle layer.
• Main function is to regulate the movements of GI
Tract.
• Some nerve fibers accelerate the movements by
secreting excitatory neurotransmitter like Ach, Serotonin,
Substance P.
• Other fibers inhibit the GI motility by secreting the
inhibitory neurotransmitter like vasoactive Intestinal
Polypeptide (VIP), Neurotensin, enkephalin
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158. 2) Meissner Nerve Plexus
• Called as submucous Nerve Plexus
• .Present in between the muscular &
submucosal layer of GI Tract.
• Function is to regulate secretory
functions of Digestive Tract.
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159. 2) Extrinsic Nerve supply
• Both Sympathetic & Parasympathetic
divisions innervate the GI Tract .
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160. 1) Sympathetic Nerve Fibers
• Preganglionic fibers arise from lateral horns of
spinal cord between 5th thoracic & 2nd lumber
segments .
• They terminate in the celiac & mesenteric
ganglia .
• Sympathetic nerve fibers inhibit the movements
& decrease the secretions of GI Tract.
• This happens due to neurotransmitter
Noradrenaline .There is also constriction of
sphincters
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161. 2) Parasympathetic Nerve fibers
• They pass through some of cranial & sacral
nerves .
• Nerve fibers to mouth & salivary glands pass
through facial & glossopharyngeal nerves.
• Preganglionic parasympathetic nerve fibers to
oesophagus, stomach, small intestine & upper
part of large intestine pass through Vagus
nerve.
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162. 2) Parasympathetic Nerve fibers
• Preganglionic fibers to lower part of large
intestine arise from 2nd ,3rd ,4th sacral
segments & pass through Pelvic nerve.
• Parasympathetic nerve fibers accelerate
the movements & increase the secretions
of GI Tract, with Neurotransmitter Ach.
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163. 4) Movements of small intestine
• Peristalsis - Definition
• Wave of relaxation, followed by wave of
contraction.
• This is the basic property of all tubular
structures.
• In intestine it is more prominent due to
Myenteric plexus
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164. Types of movement
• 1) Mixing movement –
• Only 1 segments contracts & relaxes
(Segmental peristalsis).
• Wave is not spreading to the next
segment.
• Food is mixed with digestive juices
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165. Types of movement
• 2) Propulsive waves –
• Spread from one segment to another &
food is pushed ahead & ahead.
• Speed = 5 to 6 hrs are required to reach
the food from duodenum to Caecum
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166. Types of movement
• 3) Rush peristalsis –
• When irritation of mucosa, high
segments contracts & large quantity of
food is pushed ahead.
• Structures lined by smooth muscle
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167. Types of movement
• 4) Anti peristalsis –
• Normally wave of peristalsis - From oral to
rectal direction.
• But when there is excessive irritation
(Gastro - enteritis, food poisoning,
obstruction) - Waves move in opposite
direction, leading to vomiting
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168. Movements of Villi
• Due to movement of submucosa of
intestine thin coat of muscle contract due
to presence of food & distention of small
intestine.
• Villi movement help in absorption
process
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169. Role of ilio caecal valve
• It allows unidirectional movement of
food.
• Contents of cecum cannot regurgitate
back.
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170. Causes of Peristalsis
• 1) Role of Nerves
• i) Role of Myenteric plexus (Intrinsic supply)
• In muscularis layer of intestine this plexus is
present. Presence of food & distension of
intestine stimulate this plexus & they send signal
to smooth muscle -increasing movement.
• This is called as local reflex/ Myenteric reflex.
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171. Causes of Peristalsis
• ii) Role of sympathetic & parasympathetic
nerves (Extrinsic supply)
• Parasympathetic nerves - increases the
peristalsis.
• Sympathetic nerves - decreases
peristalsis.
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172. Causes of Peristalsis
• iii) Role of Hormones
• CCKPZ, Gastrin, Serotonin etc. hormones
directly act on smooth muscle & increase
peristalsis
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173. Functions of Peristalsis
• Mixing of food with digestive juices.
• Push the food on absorptive surfaces, to
facilitate absorption
• Undigested food is pushed to large
intestine.
• To increase the blood supply of
intestine.
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174. Pathology
• In cholera, food poisoning - peristalsis is
stimulated excessively - which causes
loose motion & dehydration.
•
• Postoperatively or due to
anticholinergic drug (Tab Baralgan, Tab
Spasmindon) - Movements are reduced.
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175. Defecation Reflex
• Mass peristaltic movement, push faecal
matter from sigmoid colon into the rectum.
•
• Distension of rectal wall stimulates -
Stretch receptors.
• This receptors send - sensory nerve
impulses to the - sacral spinal cord
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176. Defecation Reflex
• Motor impulses, from the spinal cord
travel along parasympathetic nerves -
back to the descending colon, sigmoid
colon, rectum & anus.
• Contraction of longitudinal rectal muscle,
shortens the rectum, which increases the
inside pressure.
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177. Defecation Reflex
• This pressure, along with voluntary
contraction of diaphragm & abdominal
muscle – --
• Open the internal sphincter & faeces are
expelled through the anus.
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178. Defecation Reflex
• If external sphincter voluntarily relaxed -
defecation occurs.
• But, if it is voluntarily constricted,
defecation can be postponed.
• In infants, the defecation reflex causes -
automatic emptying of the rectum,
because voluntary control of the external
anus sphincter has not yet developed
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181. Faeces
• After the last stage of digestion, after
absorption of water solid or semisolid
waste part is formed which is called as
faeces.
• Quantity - Roughly about 150 gm of solid
stool is passed in 24 hours.
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182. Composition
• If vegetable course cereals & cellulose are excluded
from the diet, the faeces show a fairly constant
components as follows -
• 1) Water - 65 %
• 2) Solid - 35 %
• i) Ash - 15 % (Mainly, Ca, P4, Fe)
• ii) Ether soluble substances (Fat) - 15 %
• iii) Nitrogen - 5 %
• iv) Other - Desquamated epithelial cells, bacteria,
mucous, undigested & unabsorbed food.
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183. Stool
• Reaction - Generally, neutral or acid.
• Colour - Due to the presence of
Stercobilin, derived from the bile pigments.
• Odour - Mainly due to aromatic
substances like indole, skatole & also
gases like H2S.
• Under normal condition about 500 cc of
gas is passed out per day.
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184. Importance of Cellulose
• Cellulose serves the important purpose of
increasing the bulk of stool &
• Thus stimulating the movement of large
intestine.
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185. Faeces formation 1
• Last stage of digestion in the large intestine
• The last stage of digestion occurs through
bacterial action & no enzymes are secreted by
the colon.
• Mucus is secreted by the glands of the large
intestine, but no enzymes are secreted.
• Chyme is prepared for elimination by the action
of bacteria.
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186. Faeces formation 2
• These bacteria ferment any remaining
carbohydrates & release hydrogen, CO2 &
methane gas. These gases contribute to
flatus (Gas) in the colon.
• They also convert remaining proteins to
amino acids & breakdown the amino acids
into simpler substances that is indol,
skatole, hydrogen sulphide & fatty acids
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187. Faeces formation 3
• Some of the Indol & Skatole is carried of
in the faeces & contributes to their odour
• The rest are absorbed & transported to the
liver where they are converted into less
toxic compounds & excreted in the urine.
• Bacteria also decompose bilirubin into
simpler pigments (Stercobilinogen) which
gives faeces brown colour.
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188. Faeces formation 4
• Several vitamins needed for normal
metabolism including some B complex
vitamins & Vit. K are synthesized by
bacterial action & absorbed.
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189. Absorption & faeces formation
• By the time the chyme has remained into
large intestine for 3 - 10 Hr. It becomes
solid or semisolid as a result of absorption
of water & is known as faeces.
• Chemically faeces consist of water,
inorganic salts, sloughed of epithelial cells
from the mucosa of the GI tract, bacteria,
products of bacterial decomposition &
undigested part of food.
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190. Physiology of daefecation
• 1) Mass peristaltic movements push
faecal matter from sigmoid colon into the
rectum.
• 2) The resulting distension of the rectal
wall stimulates pressure - sensitive
receptor initiating. Reflex of defecation
which results in emptying of the rectum.
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191. Daefecation
• The external sphincter is voluntarily
controlled.
• If it is voluntarily relaxed defecation occurs, if it is
voluntarily constricted defecation can be
postponed.
• Voluntarily contraction of diaphragm &
abdominal muscle aid defecation by increasing
the pressure inside the abdomen.
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192. Daefecation Reflex
• In infants the defecation reflex causes
automatic emptying of the rectum, without
the voluntary control of the external anal
sphincter
• In certain instances of spinal cord injury,
the reflex is abolished & defecation
requires supportive measures such as
laxative
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193. Pathology
• 1) Diarrhoea - means frequent defecation of liquid
faeces, caused by increased motility of the intestine.
Since chyme passes too quickly through the small
intestine & faeces pass too quickly through the large
intestine. There is not enough time for absorption.
Vomiting & diarrhoea can result in dehydration &
electrolyte imbalance.
• 2) Constipation - means infrequent or difficult
defecation
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