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DUODENAL ATRESIA
Dr AKSHAY
JR
Department of General surgery
Mch calicut
INCIDENCE
• 1 in 6000-10000 live births
Embryology
• The endoderm-gut tube-3-4weeks
• 6th week gut epithelium
proliferates rapidly,resulting in
obliteration of the intestinal
lumen—recanalizes in next few
weeks
• Errors in recanalization –primary
cause
• This differs from other atresias-
intrauterine vascular accidents
during the later phases of gestation
Associated anomalies
• Down syndrome 28%
• Annular pancreas 23.1%
• Congenital heart disease 22.6%
• Malrotation 19.7%
• EA/TEF 8.5
• Genitourinary 8.0
• Anorectal 4.4
• Other bowel atresia 3.5
Gray and Skandalakis Classification
DA
Type1(92%) Type2(1%) Type3(7%)
Type 1
• Continuity is maintained,may be
normal on exterior,but obstructed by
a intact membrane or diaphragm
• Wind Sock deformity
• May be totally intact obstructing the
lumen completely
• Traction on the membrane with a
catheter may cause dimpling at the
origin of diaphragm-helps find out
where the membrane starts
Wind Sock deformity
• A variant of type1 DA
• Membrane is thin and elongated
• Base of the membrane in D2
• Balloons out distally intp D3 /D4
• Thus externally the obstruction
appears considerably more distal
than it is actually
Type 2
• Continuity not maintained,but
connected by a fibrous cord
• Mesentery intact
Type 3
• No connection between the
two blind ends of the
duodenum
• V-shaped mesenteric defect
Duodenal web
• Perforate/imperforate
wen/septa
• Diameter of the opening
directly determines the degree
of obstruction and is therefore
inversely related to symptoms
Site of obstruction
• Commonest D1 and D2 -85%
• Complete-81%
• Incomplete-19%
• Majority(75-95%) is seen distal to ampulla of vater-bilious
emesis,minimal distension
• May asscociated with extrinsic compression –annular pancreas,pre
dudodenal portal vein
Clinical features
Antenatally
• Polyhydramnious -50%
• Double bubble
Postnatal
• Bilious emesis from birth
• May be blood stained
• Epigastric distension,dehydration,emaciation
• Downs facies
Delayed presentation
• Recurrent vomiting
• Fb/food bolus impaction
• Failure to thrive
Diagnosis
• A/N-USG-double bubble sign-due to
fluid
• P/N-Xray-Double bubble sign –due
to air
If inconclusive
Inject 30-60ml air –repeat plain xray
Or a Contrast study
Preoperative preperation
• Ng tube decompression
• Replace GI loses
• PICC line
• TPN-(gastroperasis due to chronic obstruction)
Surgery-technique
• KIMURA’S diamond shaped DUODENO DUODENOSTOMY-Trans
anastomotic fedding tube
• Webs are excised without injuring ampulla of vater
• Other techniques
• Tapering duodenoplasty+anastomosis
• Duodenojejunostomy
Per operative
• Rule out distal atresia-using 6fr foleys catheter
• Rule out malrotation
• Donot miss out wind sock deformity
Post operative complications
• Prolonged gastroparesis-2-3 weeks to subside-use of prokinetic –
erythromycin in suboptimal dose (10mg/kg/dose)
• Anastomotic leak
• Anastamotic stricture-endoscopic dilataion
Outcome
• 45-95% survival
• Outcome depends on associated major anamolies
Thank you

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DUODENAL ATRESIA.pptx

  • 1. DUODENAL ATRESIA Dr AKSHAY JR Department of General surgery Mch calicut
  • 2. INCIDENCE • 1 in 6000-10000 live births
  • 3. Embryology • The endoderm-gut tube-3-4weeks • 6th week gut epithelium proliferates rapidly,resulting in obliteration of the intestinal lumen—recanalizes in next few weeks • Errors in recanalization –primary cause • This differs from other atresias- intrauterine vascular accidents during the later phases of gestation
  • 4. Associated anomalies • Down syndrome 28% • Annular pancreas 23.1% • Congenital heart disease 22.6% • Malrotation 19.7% • EA/TEF 8.5 • Genitourinary 8.0 • Anorectal 4.4 • Other bowel atresia 3.5
  • 5. Gray and Skandalakis Classification DA Type1(92%) Type2(1%) Type3(7%)
  • 6. Type 1 • Continuity is maintained,may be normal on exterior,but obstructed by a intact membrane or diaphragm • Wind Sock deformity • May be totally intact obstructing the lumen completely • Traction on the membrane with a catheter may cause dimpling at the origin of diaphragm-helps find out where the membrane starts
  • 7. Wind Sock deformity • A variant of type1 DA • Membrane is thin and elongated • Base of the membrane in D2 • Balloons out distally intp D3 /D4 • Thus externally the obstruction appears considerably more distal than it is actually
  • 8. Type 2 • Continuity not maintained,but connected by a fibrous cord • Mesentery intact
  • 9. Type 3 • No connection between the two blind ends of the duodenum • V-shaped mesenteric defect
  • 10. Duodenal web • Perforate/imperforate wen/septa • Diameter of the opening directly determines the degree of obstruction and is therefore inversely related to symptoms
  • 11. Site of obstruction • Commonest D1 and D2 -85% • Complete-81% • Incomplete-19% • Majority(75-95%) is seen distal to ampulla of vater-bilious emesis,minimal distension • May asscociated with extrinsic compression –annular pancreas,pre dudodenal portal vein
  • 12. Clinical features Antenatally • Polyhydramnious -50% • Double bubble Postnatal • Bilious emesis from birth • May be blood stained • Epigastric distension,dehydration,emaciation • Downs facies
  • 13. Delayed presentation • Recurrent vomiting • Fb/food bolus impaction • Failure to thrive
  • 14. Diagnosis • A/N-USG-double bubble sign-due to fluid • P/N-Xray-Double bubble sign –due to air If inconclusive Inject 30-60ml air –repeat plain xray Or a Contrast study
  • 15. Preoperative preperation • Ng tube decompression • Replace GI loses • PICC line • TPN-(gastroperasis due to chronic obstruction)
  • 16. Surgery-technique • KIMURA’S diamond shaped DUODENO DUODENOSTOMY-Trans anastomotic fedding tube
  • 17. • Webs are excised without injuring ampulla of vater • Other techniques • Tapering duodenoplasty+anastomosis • Duodenojejunostomy
  • 18. Per operative • Rule out distal atresia-using 6fr foleys catheter • Rule out malrotation • Donot miss out wind sock deformity
  • 19. Post operative complications • Prolonged gastroparesis-2-3 weeks to subside-use of prokinetic – erythromycin in suboptimal dose (10mg/kg/dose) • Anastomotic leak • Anastamotic stricture-endoscopic dilataion
  • 20. Outcome • 45-95% survival • Outcome depends on associated major anamolies