SlideShare a Scribd company logo
Diseases of the Pulp
Presented by
Dr Rahul Katyayan
For the- Faculty of
Pediatric dentistry.
CONTENTS
⚫ INTRODUCTION
⚫ PULPITIS
⚫ SEQUELAE
⚫ CAUSES
⚫ CLASSIFICATION
⚫ REVERSIBLE PULPITIS
⚫ FOCAL REVERSIBLE PULPITIS
⚫ IRREVERSIBLE PULPITIS
⚫ ACUTE PULPITIS
⚫ CHRONIC PULPITIS
⚫ NECROSIS
⚫ REFERENCES
⮚ formative organ of tooth
⮚ builds primary dentin
during development of tooth
⮚ secondary dentin after
tooth eruption
⮚ reparative dentin in response
to stimulation as long as
odontoblast remain vital
Pulp
⮚ most common cause of
dental pain
⮚ loss of teeth in younger
persons
⮚ usual cause is caries
penetrating the dentin
Pulpitis
Pulpitis
UNTREATED
Death of pulp
Spread of Infection through
apical foramina into periapical tissues
Causes Periapical Periodontitis
⮚ (1)Physical Cause
a)Mechanical
b)Thermal
⮚ (2) Chemical Cause
⮚ (3) Bacterial Cause
Causes of Pulpal
Inflammation
⮚ (a) Mechanical Cause
A. trauma
a) accident (contact sports)
b) iatrogenic damage due to dental procedure
(during cavity or crown preparation)
B. Pathologic wear ( atrrition, abrasion)
C. Cracked tooth syndrome
D. Barometric changes
Causes of Pulpal
Inflammation
⮚ (b) Thermal Cause
✔ uninsulated metallic
restoration
✔ during cavity preparation
✔ polishing
Causes of Pulpal
Inflammation
⮚ (2) Chemical Cause
✔ arise from erosion
✔ or inappropriate use
of acidic dental material
Causes of Pulpal
Inflammation
⮚ (3) Bacterial Cause
✔ can damage pulp
through toxins secreted
by bacteria from caries
✔Microbial colonization in
the pulp by blood borne
microorganisms
(anachoresis).
Causes of Pulpal
Inflammation
⮚ (1) Based on Severity of
Inflammation
⮚ (2) According to Involvement
Classification
⮚ (1) Reversible Pulpitis
⮚ (2) Irreversible Pulpitis
⮚ (3) Pulp Degeneration
⮚ (4) Pulp Necrosis
(1) Based on Severity
of Inflammation
⮚ (1) Reversible Pulpitis
✔ Symptomatic (acute)
✔ Aysptomatic (chronic)
⮚ (2) Irreversible Pulpitis
✔ Acute
• Abnormally responsive to cold
• Abnormally responsive to heat
(1) Based on Severity
of Inflammation
⮚ (2) Irreversible Pulpitis
✔ Chronic
• Asymptomatic with
pulp exposure
• Hyperplastic
• Internal resorption
(1) Based on Severity
of Inflammation
⮚ (3) Pulp Degeneration
✔ Calcific
⮚ (4) Pulp Necrosis
(1) Based on Severity
of Inflammation
⮚ (1) According to extent of inflammation
⮚ (2) According to Severity
⮚ (3) According to presence or
absence of direct
communication
between dental pulp +
oral environment
(2) According to
Involvement
⮚ (1) According to extent of inflammation
✔ Focal or Subtotal or
Partial Pulpitis
✔ Total or Generalized
Pulpitis
(2) According to
Involvement
⮚ (2) According to Severity
✔ Acute
✔ Chronic
(2) According to
Involvement
⮚ (3) According to presence or
absence of direct
communication
between dental pulp +
oral environment
✔ Pulpitis Aperts (open pulpitis)
✔ Pulpitis Clausa (closed pulpitis)
(2) According to
Involvement
⮚ mild to moderate inflammatory
condition of pulp
✔ caused by noxious stimuli
✔ pulp is capable of returning
to un-inflammed state following removal of stimuli
Reversible Pulpitis
⮚ Causes
✔ agent capable of
injuring pulp like:
• trauma
• disturbed occlusal
relationship
• thermal shock
Reversible Pulpitis
⮚ Clinical Features
✔ sharp pain lasting for
a moment
✔ often brought on by cold
than hot food or beverages
and by cold air
Reversible Pulpitis
⮚ Clinical Features
✔ does not continue
when the cause has been
removed
✔ tooth responds to electric
pulp testing at lower
current
Reversible Pulpitis
⮚ Management
✔ prevention
✔ periodic care
✔ early insertion of filling
if a cavity has developed
✔ removal of noxious
stimuli
Reversible Pulpitis
⮚earliest form
⮚ also known as pulp hyperemia
⮚ excessive accumulation of
blood within pulp tissue
⮚ leads to vascular congestion
Focal Reversible
Pulpitis
⮚ Clinical Features
✔ sensitive to thermal
changes
✔ particularly to cold
✔ application of ice or cold
fluids to tooth result in pain
Focal Reversible
Pulpitis
⮚ Clinical Features
✔ disappears upon removal
of thermal irritant or
restoration .
✔ responds to electrical test
stimulant at lower level
of current
Focal Reversible
Pulpitis
⮚ Clinical Features
✔ indicates lower pain
threshold than that of
adjacent normal
teeth
Focal Reversible
Pulpitis
⮚ Clinical Features
✔ teeth show:
• deep carious lesion
• large metallic restoration
• restoration with defective
margins
Focal Reversible
Pulpitis
⮚ Management
✔ removal of irritants
before the pulp is
severely damaged
Focal Reversible
Pulpitis
⮚ persistent inflammatory
condition of pulp
⮚ may be symptomatic or
asymptomatic
⮚ caused by noxious stimulus
Irreversible Pulpitis
⮚ Causes
✔ bacteria involvement of
pulp through caries
✔ chemical
✔ thermal
✔ mechanical injury
Irreversible Pulpitis
⮚ Clinical Features
Early Stage
✔ paroxysm of pain
caused by:
• sudden temperature
changes like cold,
sweet, acid foodstuffs
Irreversible Pulpitis
⮚ Clinical Features
Early Stage
✔ pain often continues
when cause has been
removed
✔ may come and go
spontaneously
Irreversible Pulpitis
⮚ Clinical Features
Early Stage
✔ pain
• sharp
• piercing
• shooting
• generally severe
Irreversible Pulpitis
⮚ Clinical Features
Early Stage
✔ pain
• bending over exacerbates pain which
• lying down is due to change in
• change of position intrapulpal pressure
Irreversible Pulpitis
⮚ Clinical Features
Late Stage
✔ pain
• more severe as if tooth is under
• throbbing constant pressure
Irreversible Pulpitis
⮚ Clinical Features
Late Stage
✔ pain
• patient is often awake
at night due to pain
• increased by heat and
sometimes relieved by cold,
although continued application
of cold may intensify pain
Irreversible Pulpitis
⮚ Management
✔ complete removal of pulp
or pulpectomy
✔ if the time is a factor, pulpotomy
should be done as an emergency
procedure
✔ Surgical removal should be considered if the tooth
is not restorable.
Irreversible Pulpitis
Clinical Difference
Reversible Pulpitis Irreversible Pulpitis
⮚ pain generally lasts for few
seconds
⮚ the pain produced by thermal
stimulus disappears as soon as
the stimulus is removed
Pain may last several
minutes or later
⮚ pain lingers even after
the stimulus is removed
⮚ pain may come without
any apparent stimulus
⮚ extensive acute inflammation
of pulp
⮚ frequent sequel of focal
reversible pulpitis
Acute Pulpitis
⮚ Causes
✔ tooth with large carious
lesion
✔ defective restoration
where there has been
recurrent caries
✔ pulp exposure due to
faulty cavity preparation
Acute Pulpitis
⮚ Clinical Features
✔ severe pain is elicited by
thermal changes
✔ pain persists even after
thermal stimulus
disappears or been
removed
Acute Pulpitis
⮚ Clinical Features
✔ may be continuous
✔ intensity may be increased
when patient lies down
✔ application of heat
may cause acute
exacerbation of pain
Acute Pulpitis
⮚ Clinical Features
✔ tooth reacts to electric
pulp vitality tester at a
lower level of current
than adjacent normal
teeth
Acute Pulpitis
⮚ Clinical Features
✔ pressure increases
because of lack of
escape of inflammatory
exudate
✔ rapid spread of inflammation
through pulp with pain
+ necrosis
Acute Pulpitis
⮚ Management
✔ early stages of acute pulpitis pulpotomy
(removal of coronal pulp)
✔ placing material that
favors calcification such
as:
• calcium hydroxide
over entrance of
root canals
Acute Pulpitis
⮚ Management
✔ root canal filling with
inert material like
gutta percha should be
done
Acute Pulpitis
⮚ may develop with or
without episodes of
acute pulpitis
⮚ many pulps under large
carious cavities die painlessly
⮚ 1st indication is then
development of periapical
periodontitis, either with pain
or seen by chance in radiograph
Chronic Pulpitis
⮚ Clinical Features
✔ dull aching type
✔ more often intermittent
than continuous
Chronic Pulpitis
⮚ Management
✔ root canal therapy
✔ followed by crown
restoration
Chronic Pulpitis
⮚ also called as pulp polyp
or pulpitis aperta
⮚ essentially an excessive
exuberant proliferation
of chronically inflammed
dental pulp tissue
Chronic Hyperplastic
Pulpitis
⮚ pulpal inflammation due
to an extensive carious
exposure of a young pulp
⮚ development of granulation
tissue
⮚ covered at times by epithelium
⮚ resulting from long standing
low grade infection
Chronic Hyperplastic
Pulpitis
⮚ Causes
✔ slow progressive
exposure of pulp
✔ bacterial infection
Chronic Hyperplastic
Pulpitis
⮚ Clinical Features
✔ most commonly involved
are deciduous molars +
1st permanent molar
• excellent blood supply
• large root opening
Chronic Hyperplastic
Pulpitis
⮚ Clinical Features
✔ asymptomatic
✔ seen only in teeth of children
+ young adults
Chronic Hyperplastic
Pulpitis
⮚ Clinical Features
✔ polypoid tissue appears
• fleshy
• reddish pulpal mass filling
most of pulp chamber
or cavity
• or even extend beyond
confines of tooth
Chronic Hyperplastic
Pulpitis
⮚ Clinical Features
✔ polypoid tissue appears
• sometimes, if mass is
large enough
• interferes with closure
of mouth
Chronic Hyperplastic
Pulpitis
⮚ Clinical Features
✔ polypoid tissue appears
• may cause discomfort
during mastication
• due to pressure of food
bolus
Chronic Hyperplastic
Pulpitis
⮚ Clinical Features
✔ polypoid tissue appears
• tissue easily bleeds
because of rich network
of blood vessels
• tooth may respond or
not at all to thermal test
Chronic Hyperplastic
Pulpitis
⮚ Management
✔ elimination of polypoid tissue
✔ followed by extirpation of pulp
✔ hyperplastic tissue bleeeding
can be controlled by pressure
✔ pulpectomy or extraction of tooth can also
be done if the tooth is not restorable.
Chronic Hyperplastic
Pulpitis
⮚ death of pulp
⮚ may be partial or total
depending on whether part
or the entire pulp is
involved
Necrosis
⮚ Causes
✔ sequeala of inflammation
✔ can also occur following
trauma
• pulp is destroyed before
an inflammatory reaction
Necrosis
⮚ Types
✔ (1) Coagulation Necrosis
✔ (2) Liquefaction Necrosis
Necrosis
⮚ Types
✔ (1) Coagulation Necrosis
• soluble portion of
tissue is precipitated
or converted into a solid
material
Necrosis
⮚ Types
✔ (1) Coagulation Necrosis
• tissue is converted into
tissue mass consisting
chiefly of coagulated
▪ proteins
▪ fats
▪ water
Necrosis
⮚ Types
✔ (2) Liquefaction Necrosis
• results when proteolytic
enzymes convert the
tissue into softened mass
liquid or amorphous debris
Necrosis
⮚ Clinical Features
✔ no painful symptoms
✔ discoloration of tooth
• 1st indication that the pulp
is dead
Necrosis
⮚ Clinical Features
✔ history of pain lasting from
a few minutes to a few
hours followed by
complete + sudden
cessation of pain
Necrosis
⮚ Management
✔ preparation + obturation of
root canals
Necrosis
References:
❖ Books
⮚ Grossman’s endodontic practice twelfth edition
⮚Textbook of oral pathology shafers
⮚Cawson, R.A: Cawson’s Essentials of Oral Pathology and
Oral Medicine, 8th Edition.(page 60)
⮚ Ghom, Ali & Mhaske, Shubhangi: Textbook of Oral
Pathology. (pages 420-425)
References:
⮚Adrian,J.C.,et al.:J. Am. Dent. Assoc.,83:113,
1971.
⮚Allard, U., et al.:Oral Surg.,48:454, 1979.
⮚Austin, L.T.:J. Am. Dent. Assoc.,17:1930, 1930.
⮚Baume, L.: SSO Schweiz. Monatsschr.
Zahnheilkd.,77:1082, 1965.
References:
⮚Baume, L.: Transactions of the Fourth
International Conference on Endodontics.
Philadelphia: University of Pennsylvania
press, 1968, p. 66 .
⚫Baume, L.J.: Monogr. Oral Sci.,8:1-
220, 1980.
THANK
YOU

More Related Content

What's hot

Periodontal flap
Periodontal flapPeriodontal flap
Periodontal flap
akshay shete
 
Patterns of bone destruction in periodontics
Patterns of bone destruction in periodontics Patterns of bone destruction in periodontics
Patterns of bone destruction in periodontics
Maneesh Ahammed Syed
 
Pulpal & periradicular diseases & their diagnosis
Pulpal & periradicular diseases & their diagnosisPulpal & periradicular diseases & their diagnosis
Pulpal & periradicular diseases & their diagnosis
Aditi Singh
 
Periodontal surgery
Periodontal surgeryPeriodontal surgery
Periodontal surgery
Enas Elgendy
 
HEMISECTION
HEMISECTIONHEMISECTION
HEMISECTION
Dr AJINS CB
 
External root resorption (ERR)
External root resorption (ERR)External root resorption (ERR)
External root resorption (ERR)
Dentist Khawla
 
Apexification and apexogenesis
Apexification and apexogenesisApexification and apexogenesis
Apexification and apexogenesis
Dr Ravneet Kour
 
Periodontal pocket
Periodontal pocketPeriodontal pocket
Periodontal pocket
Navneet Randhawa
 
Direct and Indirect pulp capping
Direct and Indirect pulp cappingDirect and Indirect pulp capping
Direct and Indirect pulp capping
Anju Thomas
 
Cracked tooth syndrome
Cracked tooth syndromeCracked tooth syndrome
Cracked tooth syndrome
fattahaa
 
Periodontal abscess
Periodontal abscessPeriodontal abscess
Periodontal abscess
neha_mathur2132
 
Periapical periodonitis
Periapical periodonitisPeriapical periodonitis
Periapical periodonitis
Hamzeh AlBattikhi
 
Revascularisation
RevascularisationRevascularisation
Revascularisation
Parul Malhotra
 
Odontoma (Doctor Faris Alabeedi MSc, MMedSc, PgDip, BDS.)
Odontoma (Doctor Faris Alabeedi MSc, MMedSc, PgDip, BDS.)Odontoma (Doctor Faris Alabeedi MSc, MMedSc, PgDip, BDS.)
Odontoma (Doctor Faris Alabeedi MSc, MMedSc, PgDip, BDS.)Doctor Faris Alabeedi
 
Gow gates & vazirani akinosi technique of nerve
Gow  gates & vazirani akinosi technique of nerveGow  gates & vazirani akinosi technique of nerve
Gow gates & vazirani akinosi technique of nerve
POOJAKUMARI277
 
Diagnodent
DiagnodentDiagnodent
DiagnodentMccart
 
Dentinal hypersensitivity
Dentinal hypersensitivityDentinal hypersensitivity
Dentinal hypersensitivity
MrinaliniDr
 
Vestibuloplasty
VestibuloplastyVestibuloplasty
Vestibuloplasty
vrushupatel
 

What's hot (20)

Periodontal flap
Periodontal flapPeriodontal flap
Periodontal flap
 
Patterns of bone destruction in periodontics
Patterns of bone destruction in periodontics Patterns of bone destruction in periodontics
Patterns of bone destruction in periodontics
 
Pulpal & periradicular diseases & their diagnosis
Pulpal & periradicular diseases & their diagnosisPulpal & periradicular diseases & their diagnosis
Pulpal & periradicular diseases & their diagnosis
 
Periodontal surgery
Periodontal surgeryPeriodontal surgery
Periodontal surgery
 
HEMISECTION
HEMISECTIONHEMISECTION
HEMISECTION
 
External root resorption (ERR)
External root resorption (ERR)External root resorption (ERR)
External root resorption (ERR)
 
Apexification and apexogenesis
Apexification and apexogenesisApexification and apexogenesis
Apexification and apexogenesis
 
Periodontal pocket
Periodontal pocketPeriodontal pocket
Periodontal pocket
 
Direct and Indirect pulp capping
Direct and Indirect pulp cappingDirect and Indirect pulp capping
Direct and Indirect pulp capping
 
Cracked tooth syndrome
Cracked tooth syndromeCracked tooth syndrome
Cracked tooth syndrome
 
Periodontal abscess
Periodontal abscessPeriodontal abscess
Periodontal abscess
 
Periapical periodonitis
Periapical periodonitisPeriapical periodonitis
Periapical periodonitis
 
Revascularisation
RevascularisationRevascularisation
Revascularisation
 
Dental implants _perio_
Dental implants _perio_Dental implants _perio_
Dental implants _perio_
 
Odontoma (Doctor Faris Alabeedi MSc, MMedSc, PgDip, BDS.)
Odontoma (Doctor Faris Alabeedi MSc, MMedSc, PgDip, BDS.)Odontoma (Doctor Faris Alabeedi MSc, MMedSc, PgDip, BDS.)
Odontoma (Doctor Faris Alabeedi MSc, MMedSc, PgDip, BDS.)
 
Gow gates & vazirani akinosi technique of nerve
Gow  gates & vazirani akinosi technique of nerveGow  gates & vazirani akinosi technique of nerve
Gow gates & vazirani akinosi technique of nerve
 
Diagnodent
DiagnodentDiagnodent
Diagnodent
 
Gingival curettage
Gingival curettageGingival curettage
Gingival curettage
 
Dentinal hypersensitivity
Dentinal hypersensitivityDentinal hypersensitivity
Dentinal hypersensitivity
 
Vestibuloplasty
VestibuloplastyVestibuloplasty
Vestibuloplasty
 

Similar to Diseases of pulp

Pulp diseases
Pulp diseasesPulp diseases
Pulp diseases
Panda Park
 
pulp lecture-1.pdf_108808.pdf_239382.pdf
pulp lecture-1.pdf_108808.pdf_239382.pdfpulp lecture-1.pdf_108808.pdf_239382.pdf
pulp lecture-1.pdf_108808.pdf_239382.pdf
MohamedFarag457087
 
Endodonticemergencies 170917100430
Endodonticemergencies 170917100430Endodonticemergencies 170917100430
Endodonticemergencies 170917100430
sivaram12321
 
Endodontic emergencies
Endodontic emergenciesEndodontic emergencies
Endodontic emergencies
Nivedha Tina
 
Pulpitis
PulpitisPulpitis
Pulpitis
Amanjot Singh
 
PULP AND PERIAPICAL LESIONS OF THE TOOTH ppt
PULP AND PERIAPICAL LESIONS OF THE TOOTH pptPULP AND PERIAPICAL LESIONS OF THE TOOTH ppt
PULP AND PERIAPICAL LESIONS OF THE TOOTH ppt
K BHATTACHARJEE
 
comprehensive case history recording in pediatric dentistry
comprehensive case history recording in pediatric dentistrycomprehensive case history recording in pediatric dentistry
comprehensive case history recording in pediatric dentistry
drsavithaks
 
Diseases of pulp for ugs
Diseases of pulp for ugsDiseases of pulp for ugs
Diseases of pulp for ugs
ashujarb
 
Diseases of pulp
Diseases of pulpDiseases of pulp
Diseases of pulp
Shubham Parmar
 
Pulpitis
PulpitisPulpitis
Pulpitis
IAU Dent
 
Diseases of pulp
Diseases of pulpDiseases of pulp
Diseases of pulp
Dr. Santhu Sadasivan
 
Diseases of pulp and periapical tissues
Diseases of pulp and periapical tissues Diseases of pulp and periapical tissues
Diseases of pulp and periapical tissues
madhusudhan reddy
 
sequle of pulpitis.pptx
sequle of pulpitis.pptxsequle of pulpitis.pptx
sequle of pulpitis.pptx
Royal Dental College Library
 
Pulpal diseases
Pulpal diseasesPulpal diseases
Pulpal diseases
Addis Dlove
 
Endodontic emergencies (only pretreatment)
Endodontic emergencies (only pretreatment)Endodontic emergencies (only pretreatment)
Endodontic emergencies (only pretreatment)
Dr. Riya Oswal
 
Pulp & periapical disease
Pulp & periapical diseasePulp & periapical disease
Pulp & periapical disease
Al-Azhar University, Cairo, Egypt
 
Endodontic emergencies
Endodontic emergenciesEndodontic emergencies
Endodontic emergencies
Dr. Arpit Viradiya
 
Periodontal abscess : A Review
Periodontal  abscess : A ReviewPeriodontal  abscess : A Review
Periodontal abscess : A Review
Navneet Randhawa
 
DISEASES OF DENTAL PULP AND PERI RADICULAR TISSUES.ppt
DISEASES OF DENTAL PULP AND PERI RADICULAR TISSUES.pptDISEASES OF DENTAL PULP AND PERI RADICULAR TISSUES.ppt
DISEASES OF DENTAL PULP AND PERI RADICULAR TISSUES.ppt
consendosbpdch
 

Similar to Diseases of pulp (20)

Pulipitis
PulipitisPulipitis
Pulipitis
 
Pulp diseases
Pulp diseasesPulp diseases
Pulp diseases
 
pulp lecture-1.pdf_108808.pdf_239382.pdf
pulp lecture-1.pdf_108808.pdf_239382.pdfpulp lecture-1.pdf_108808.pdf_239382.pdf
pulp lecture-1.pdf_108808.pdf_239382.pdf
 
Endodonticemergencies 170917100430
Endodonticemergencies 170917100430Endodonticemergencies 170917100430
Endodonticemergencies 170917100430
 
Endodontic emergencies
Endodontic emergenciesEndodontic emergencies
Endodontic emergencies
 
Pulpitis
PulpitisPulpitis
Pulpitis
 
PULP AND PERIAPICAL LESIONS OF THE TOOTH ppt
PULP AND PERIAPICAL LESIONS OF THE TOOTH pptPULP AND PERIAPICAL LESIONS OF THE TOOTH ppt
PULP AND PERIAPICAL LESIONS OF THE TOOTH ppt
 
comprehensive case history recording in pediatric dentistry
comprehensive case history recording in pediatric dentistrycomprehensive case history recording in pediatric dentistry
comprehensive case history recording in pediatric dentistry
 
Diseases of pulp for ugs
Diseases of pulp for ugsDiseases of pulp for ugs
Diseases of pulp for ugs
 
Diseases of pulp
Diseases of pulpDiseases of pulp
Diseases of pulp
 
Pulpitis
PulpitisPulpitis
Pulpitis
 
Diseases of pulp
Diseases of pulpDiseases of pulp
Diseases of pulp
 
Diseases of pulp and periapical tissues
Diseases of pulp and periapical tissues Diseases of pulp and periapical tissues
Diseases of pulp and periapical tissues
 
sequle of pulpitis.pptx
sequle of pulpitis.pptxsequle of pulpitis.pptx
sequle of pulpitis.pptx
 
Pulpal diseases
Pulpal diseasesPulpal diseases
Pulpal diseases
 
Endodontic emergencies (only pretreatment)
Endodontic emergencies (only pretreatment)Endodontic emergencies (only pretreatment)
Endodontic emergencies (only pretreatment)
 
Pulp & periapical disease
Pulp & periapical diseasePulp & periapical disease
Pulp & periapical disease
 
Endodontic emergencies
Endodontic emergenciesEndodontic emergencies
Endodontic emergencies
 
Periodontal abscess : A Review
Periodontal  abscess : A ReviewPeriodontal  abscess : A Review
Periodontal abscess : A Review
 
DISEASES OF DENTAL PULP AND PERI RADICULAR TISSUES.ppt
DISEASES OF DENTAL PULP AND PERI RADICULAR TISSUES.pptDISEASES OF DENTAL PULP AND PERI RADICULAR TISSUES.ppt
DISEASES OF DENTAL PULP AND PERI RADICULAR TISSUES.ppt
 

Recently uploaded

NVBDCP.pptx Nation vector borne disease control program
NVBDCP.pptx Nation vector borne disease control programNVBDCP.pptx Nation vector borne disease control program
NVBDCP.pptx Nation vector borne disease control program
Sapna Thakur
 
planning for change nursing Management ppt
planning for change nursing Management pptplanning for change nursing Management ppt
planning for change nursing Management ppt
Thangamjayarani
 
KDIGO 2024 guidelines for diabetologists
KDIGO 2024 guidelines for diabetologistsKDIGO 2024 guidelines for diabetologists
KDIGO 2024 guidelines for diabetologists
د.محمود نجيب
 
How to Give Better Lectures: Some Tips for Doctors
How to Give Better Lectures: Some Tips for DoctorsHow to Give Better Lectures: Some Tips for Doctors
How to Give Better Lectures: Some Tips for Doctors
LanceCatedral
 
Ocular injury ppt Upendra pal optometrist upums saifai etawah
Ocular injury  ppt  Upendra pal  optometrist upums saifai etawahOcular injury  ppt  Upendra pal  optometrist upums saifai etawah
Ocular injury ppt Upendra pal optometrist upums saifai etawah
pal078100
 
Alcohol_Dr. Jeenal Mistry MD Pharmacology.pdf
Alcohol_Dr. Jeenal Mistry MD Pharmacology.pdfAlcohol_Dr. Jeenal Mistry MD Pharmacology.pdf
Alcohol_Dr. Jeenal Mistry MD Pharmacology.pdf
Dr Jeenal Mistry
 
Physiology of Chemical Sensation of smell.pdf
Physiology of Chemical Sensation of smell.pdfPhysiology of Chemical Sensation of smell.pdf
Physiology of Chemical Sensation of smell.pdf
MedicoseAcademics
 
Superficial & Deep Fascia of the NECK.pptx
Superficial & Deep Fascia of the NECK.pptxSuperficial & Deep Fascia of the NECK.pptx
Superficial & Deep Fascia of the NECK.pptx
Dr. Rabia Inam Gandapore
 
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journey
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness JourneyTom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journey
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journey
greendigital
 
Triangles of Neck and Clinical Correlation by Dr. RIG.pptx
Triangles of Neck and Clinical Correlation by Dr. RIG.pptxTriangles of Neck and Clinical Correlation by Dr. RIG.pptx
Triangles of Neck and Clinical Correlation by Dr. RIG.pptx
Dr. Rabia Inam Gandapore
 
How STIs Influence the Development of Pelvic Inflammatory Disease.pptx
How STIs Influence the Development of Pelvic Inflammatory Disease.pptxHow STIs Influence the Development of Pelvic Inflammatory Disease.pptx
How STIs Influence the Development of Pelvic Inflammatory Disease.pptx
FFragrant
 
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...
kevinkariuki227
 
Dehradun #ℂall #gIRLS Oyo Hotel 9719300533 #ℂall #gIRL in Dehradun
Dehradun #ℂall #gIRLS Oyo Hotel 9719300533 #ℂall #gIRL in DehradunDehradun #ℂall #gIRLS Oyo Hotel 9719300533 #ℂall #gIRL in Dehradun
Dehradun #ℂall #gIRLS Oyo Hotel 9719300533 #ℂall #gIRL in Dehradun
chandankumarsmartiso
 
Knee anatomy and clinical tests 2024.pdf
Knee anatomy and clinical tests 2024.pdfKnee anatomy and clinical tests 2024.pdf
Knee anatomy and clinical tests 2024.pdf
vimalpl1234
 
Pharynx and Clinical Correlations BY Dr.Rabia Inam Gandapore.pptx
Pharynx and Clinical Correlations BY Dr.Rabia Inam Gandapore.pptxPharynx and Clinical Correlations BY Dr.Rabia Inam Gandapore.pptx
Pharynx and Clinical Correlations BY Dr.Rabia Inam Gandapore.pptx
Dr. Rabia Inam Gandapore
 
Pictures of Superficial & Deep Fascia.ppt.pdf
Pictures of Superficial & Deep Fascia.ppt.pdfPictures of Superficial & Deep Fascia.ppt.pdf
Pictures of Superficial & Deep Fascia.ppt.pdf
Dr. Rabia Inam Gandapore
 
HOT NEW PRODUCT! BIG SALES FAST SHIPPING NOW FROM CHINA!! EU KU DB BK substit...
HOT NEW PRODUCT! BIG SALES FAST SHIPPING NOW FROM CHINA!! EU KU DB BK substit...HOT NEW PRODUCT! BIG SALES FAST SHIPPING NOW FROM CHINA!! EU KU DB BK substit...
HOT NEW PRODUCT! BIG SALES FAST SHIPPING NOW FROM CHINA!! EU KU DB BK substit...
GL Anaacs
 
Maxilla, Mandible & Hyoid Bone & Clinical Correlations by Dr. RIG.pptx
Maxilla, Mandible & Hyoid Bone & Clinical Correlations by Dr. RIG.pptxMaxilla, Mandible & Hyoid Bone & Clinical Correlations by Dr. RIG.pptx
Maxilla, Mandible & Hyoid Bone & Clinical Correlations by Dr. RIG.pptx
Dr. Rabia Inam Gandapore
 
New Drug Discovery and Development .....
New Drug Discovery and Development .....New Drug Discovery and Development .....
New Drug Discovery and Development .....
NEHA GUPTA
 
Physiology of Special Chemical Sensation of Taste
Physiology of Special Chemical Sensation of TastePhysiology of Special Chemical Sensation of Taste
Physiology of Special Chemical Sensation of Taste
MedicoseAcademics
 

Recently uploaded (20)

NVBDCP.pptx Nation vector borne disease control program
NVBDCP.pptx Nation vector borne disease control programNVBDCP.pptx Nation vector borne disease control program
NVBDCP.pptx Nation vector borne disease control program
 
planning for change nursing Management ppt
planning for change nursing Management pptplanning for change nursing Management ppt
planning for change nursing Management ppt
 
KDIGO 2024 guidelines for diabetologists
KDIGO 2024 guidelines for diabetologistsKDIGO 2024 guidelines for diabetologists
KDIGO 2024 guidelines for diabetologists
 
How to Give Better Lectures: Some Tips for Doctors
How to Give Better Lectures: Some Tips for DoctorsHow to Give Better Lectures: Some Tips for Doctors
How to Give Better Lectures: Some Tips for Doctors
 
Ocular injury ppt Upendra pal optometrist upums saifai etawah
Ocular injury  ppt  Upendra pal  optometrist upums saifai etawahOcular injury  ppt  Upendra pal  optometrist upums saifai etawah
Ocular injury ppt Upendra pal optometrist upums saifai etawah
 
Alcohol_Dr. Jeenal Mistry MD Pharmacology.pdf
Alcohol_Dr. Jeenal Mistry MD Pharmacology.pdfAlcohol_Dr. Jeenal Mistry MD Pharmacology.pdf
Alcohol_Dr. Jeenal Mistry MD Pharmacology.pdf
 
Physiology of Chemical Sensation of smell.pdf
Physiology of Chemical Sensation of smell.pdfPhysiology of Chemical Sensation of smell.pdf
Physiology of Chemical Sensation of smell.pdf
 
Superficial & Deep Fascia of the NECK.pptx
Superficial & Deep Fascia of the NECK.pptxSuperficial & Deep Fascia of the NECK.pptx
Superficial & Deep Fascia of the NECK.pptx
 
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journey
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness JourneyTom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journey
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journey
 
Triangles of Neck and Clinical Correlation by Dr. RIG.pptx
Triangles of Neck and Clinical Correlation by Dr. RIG.pptxTriangles of Neck and Clinical Correlation by Dr. RIG.pptx
Triangles of Neck and Clinical Correlation by Dr. RIG.pptx
 
How STIs Influence the Development of Pelvic Inflammatory Disease.pptx
How STIs Influence the Development of Pelvic Inflammatory Disease.pptxHow STIs Influence the Development of Pelvic Inflammatory Disease.pptx
How STIs Influence the Development of Pelvic Inflammatory Disease.pptx
 
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...
 
Dehradun #ℂall #gIRLS Oyo Hotel 9719300533 #ℂall #gIRL in Dehradun
Dehradun #ℂall #gIRLS Oyo Hotel 9719300533 #ℂall #gIRL in DehradunDehradun #ℂall #gIRLS Oyo Hotel 9719300533 #ℂall #gIRL in Dehradun
Dehradun #ℂall #gIRLS Oyo Hotel 9719300533 #ℂall #gIRL in Dehradun
 
Knee anatomy and clinical tests 2024.pdf
Knee anatomy and clinical tests 2024.pdfKnee anatomy and clinical tests 2024.pdf
Knee anatomy and clinical tests 2024.pdf
 
Pharynx and Clinical Correlations BY Dr.Rabia Inam Gandapore.pptx
Pharynx and Clinical Correlations BY Dr.Rabia Inam Gandapore.pptxPharynx and Clinical Correlations BY Dr.Rabia Inam Gandapore.pptx
Pharynx and Clinical Correlations BY Dr.Rabia Inam Gandapore.pptx
 
Pictures of Superficial & Deep Fascia.ppt.pdf
Pictures of Superficial & Deep Fascia.ppt.pdfPictures of Superficial & Deep Fascia.ppt.pdf
Pictures of Superficial & Deep Fascia.ppt.pdf
 
HOT NEW PRODUCT! BIG SALES FAST SHIPPING NOW FROM CHINA!! EU KU DB BK substit...
HOT NEW PRODUCT! BIG SALES FAST SHIPPING NOW FROM CHINA!! EU KU DB BK substit...HOT NEW PRODUCT! BIG SALES FAST SHIPPING NOW FROM CHINA!! EU KU DB BK substit...
HOT NEW PRODUCT! BIG SALES FAST SHIPPING NOW FROM CHINA!! EU KU DB BK substit...
 
Maxilla, Mandible & Hyoid Bone & Clinical Correlations by Dr. RIG.pptx
Maxilla, Mandible & Hyoid Bone & Clinical Correlations by Dr. RIG.pptxMaxilla, Mandible & Hyoid Bone & Clinical Correlations by Dr. RIG.pptx
Maxilla, Mandible & Hyoid Bone & Clinical Correlations by Dr. RIG.pptx
 
New Drug Discovery and Development .....
New Drug Discovery and Development .....New Drug Discovery and Development .....
New Drug Discovery and Development .....
 
Physiology of Special Chemical Sensation of Taste
Physiology of Special Chemical Sensation of TastePhysiology of Special Chemical Sensation of Taste
Physiology of Special Chemical Sensation of Taste
 

Diseases of pulp

  • 1. Diseases of the Pulp Presented by Dr Rahul Katyayan For the- Faculty of Pediatric dentistry.
  • 2. CONTENTS ⚫ INTRODUCTION ⚫ PULPITIS ⚫ SEQUELAE ⚫ CAUSES ⚫ CLASSIFICATION ⚫ REVERSIBLE PULPITIS ⚫ FOCAL REVERSIBLE PULPITIS ⚫ IRREVERSIBLE PULPITIS ⚫ ACUTE PULPITIS ⚫ CHRONIC PULPITIS ⚫ NECROSIS ⚫ REFERENCES
  • 3. ⮚ formative organ of tooth ⮚ builds primary dentin during development of tooth ⮚ secondary dentin after tooth eruption ⮚ reparative dentin in response to stimulation as long as odontoblast remain vital Pulp
  • 4. ⮚ most common cause of dental pain ⮚ loss of teeth in younger persons ⮚ usual cause is caries penetrating the dentin Pulpitis
  • 5. Pulpitis UNTREATED Death of pulp Spread of Infection through apical foramina into periapical tissues Causes Periapical Periodontitis
  • 6. ⮚ (1)Physical Cause a)Mechanical b)Thermal ⮚ (2) Chemical Cause ⮚ (3) Bacterial Cause Causes of Pulpal Inflammation
  • 7. ⮚ (a) Mechanical Cause A. trauma a) accident (contact sports) b) iatrogenic damage due to dental procedure (during cavity or crown preparation) B. Pathologic wear ( atrrition, abrasion) C. Cracked tooth syndrome D. Barometric changes Causes of Pulpal Inflammation
  • 8. ⮚ (b) Thermal Cause ✔ uninsulated metallic restoration ✔ during cavity preparation ✔ polishing Causes of Pulpal Inflammation
  • 9. ⮚ (2) Chemical Cause ✔ arise from erosion ✔ or inappropriate use of acidic dental material Causes of Pulpal Inflammation
  • 10. ⮚ (3) Bacterial Cause ✔ can damage pulp through toxins secreted by bacteria from caries ✔Microbial colonization in the pulp by blood borne microorganisms (anachoresis). Causes of Pulpal Inflammation
  • 11. ⮚ (1) Based on Severity of Inflammation ⮚ (2) According to Involvement Classification
  • 12. ⮚ (1) Reversible Pulpitis ⮚ (2) Irreversible Pulpitis ⮚ (3) Pulp Degeneration ⮚ (4) Pulp Necrosis (1) Based on Severity of Inflammation
  • 13. ⮚ (1) Reversible Pulpitis ✔ Symptomatic (acute) ✔ Aysptomatic (chronic) ⮚ (2) Irreversible Pulpitis ✔ Acute • Abnormally responsive to cold • Abnormally responsive to heat (1) Based on Severity of Inflammation
  • 14. ⮚ (2) Irreversible Pulpitis ✔ Chronic • Asymptomatic with pulp exposure • Hyperplastic • Internal resorption (1) Based on Severity of Inflammation
  • 15. ⮚ (3) Pulp Degeneration ✔ Calcific ⮚ (4) Pulp Necrosis (1) Based on Severity of Inflammation
  • 16. ⮚ (1) According to extent of inflammation ⮚ (2) According to Severity ⮚ (3) According to presence or absence of direct communication between dental pulp + oral environment (2) According to Involvement
  • 17. ⮚ (1) According to extent of inflammation ✔ Focal or Subtotal or Partial Pulpitis ✔ Total or Generalized Pulpitis (2) According to Involvement
  • 18. ⮚ (2) According to Severity ✔ Acute ✔ Chronic (2) According to Involvement
  • 19. ⮚ (3) According to presence or absence of direct communication between dental pulp + oral environment ✔ Pulpitis Aperts (open pulpitis) ✔ Pulpitis Clausa (closed pulpitis) (2) According to Involvement
  • 20. ⮚ mild to moderate inflammatory condition of pulp ✔ caused by noxious stimuli ✔ pulp is capable of returning to un-inflammed state following removal of stimuli Reversible Pulpitis
  • 21. ⮚ Causes ✔ agent capable of injuring pulp like: • trauma • disturbed occlusal relationship • thermal shock Reversible Pulpitis
  • 22. ⮚ Clinical Features ✔ sharp pain lasting for a moment ✔ often brought on by cold than hot food or beverages and by cold air Reversible Pulpitis
  • 23. ⮚ Clinical Features ✔ does not continue when the cause has been removed ✔ tooth responds to electric pulp testing at lower current Reversible Pulpitis
  • 24. ⮚ Management ✔ prevention ✔ periodic care ✔ early insertion of filling if a cavity has developed ✔ removal of noxious stimuli Reversible Pulpitis
  • 25. ⮚earliest form ⮚ also known as pulp hyperemia ⮚ excessive accumulation of blood within pulp tissue ⮚ leads to vascular congestion Focal Reversible Pulpitis
  • 26. ⮚ Clinical Features ✔ sensitive to thermal changes ✔ particularly to cold ✔ application of ice or cold fluids to tooth result in pain Focal Reversible Pulpitis
  • 27. ⮚ Clinical Features ✔ disappears upon removal of thermal irritant or restoration . ✔ responds to electrical test stimulant at lower level of current Focal Reversible Pulpitis
  • 28. ⮚ Clinical Features ✔ indicates lower pain threshold than that of adjacent normal teeth Focal Reversible Pulpitis
  • 29. ⮚ Clinical Features ✔ teeth show: • deep carious lesion • large metallic restoration • restoration with defective margins Focal Reversible Pulpitis
  • 30. ⮚ Management ✔ removal of irritants before the pulp is severely damaged Focal Reversible Pulpitis
  • 31. ⮚ persistent inflammatory condition of pulp ⮚ may be symptomatic or asymptomatic ⮚ caused by noxious stimulus Irreversible Pulpitis
  • 32. ⮚ Causes ✔ bacteria involvement of pulp through caries ✔ chemical ✔ thermal ✔ mechanical injury Irreversible Pulpitis
  • 33. ⮚ Clinical Features Early Stage ✔ paroxysm of pain caused by: • sudden temperature changes like cold, sweet, acid foodstuffs Irreversible Pulpitis
  • 34. ⮚ Clinical Features Early Stage ✔ pain often continues when cause has been removed ✔ may come and go spontaneously Irreversible Pulpitis
  • 35. ⮚ Clinical Features Early Stage ✔ pain • sharp • piercing • shooting • generally severe Irreversible Pulpitis
  • 36. ⮚ Clinical Features Early Stage ✔ pain • bending over exacerbates pain which • lying down is due to change in • change of position intrapulpal pressure Irreversible Pulpitis
  • 37. ⮚ Clinical Features Late Stage ✔ pain • more severe as if tooth is under • throbbing constant pressure Irreversible Pulpitis
  • 38. ⮚ Clinical Features Late Stage ✔ pain • patient is often awake at night due to pain • increased by heat and sometimes relieved by cold, although continued application of cold may intensify pain Irreversible Pulpitis
  • 39. ⮚ Management ✔ complete removal of pulp or pulpectomy ✔ if the time is a factor, pulpotomy should be done as an emergency procedure ✔ Surgical removal should be considered if the tooth is not restorable. Irreversible Pulpitis
  • 40. Clinical Difference Reversible Pulpitis Irreversible Pulpitis ⮚ pain generally lasts for few seconds ⮚ the pain produced by thermal stimulus disappears as soon as the stimulus is removed Pain may last several minutes or later ⮚ pain lingers even after the stimulus is removed ⮚ pain may come without any apparent stimulus
  • 41. ⮚ extensive acute inflammation of pulp ⮚ frequent sequel of focal reversible pulpitis Acute Pulpitis
  • 42. ⮚ Causes ✔ tooth with large carious lesion ✔ defective restoration where there has been recurrent caries ✔ pulp exposure due to faulty cavity preparation Acute Pulpitis
  • 43. ⮚ Clinical Features ✔ severe pain is elicited by thermal changes ✔ pain persists even after thermal stimulus disappears or been removed Acute Pulpitis
  • 44. ⮚ Clinical Features ✔ may be continuous ✔ intensity may be increased when patient lies down ✔ application of heat may cause acute exacerbation of pain Acute Pulpitis
  • 45. ⮚ Clinical Features ✔ tooth reacts to electric pulp vitality tester at a lower level of current than adjacent normal teeth Acute Pulpitis
  • 46. ⮚ Clinical Features ✔ pressure increases because of lack of escape of inflammatory exudate ✔ rapid spread of inflammation through pulp with pain + necrosis Acute Pulpitis
  • 47. ⮚ Management ✔ early stages of acute pulpitis pulpotomy (removal of coronal pulp) ✔ placing material that favors calcification such as: • calcium hydroxide over entrance of root canals Acute Pulpitis
  • 48. ⮚ Management ✔ root canal filling with inert material like gutta percha should be done Acute Pulpitis
  • 49. ⮚ may develop with or without episodes of acute pulpitis ⮚ many pulps under large carious cavities die painlessly ⮚ 1st indication is then development of periapical periodontitis, either with pain or seen by chance in radiograph Chronic Pulpitis
  • 50. ⮚ Clinical Features ✔ dull aching type ✔ more often intermittent than continuous Chronic Pulpitis
  • 51. ⮚ Management ✔ root canal therapy ✔ followed by crown restoration Chronic Pulpitis
  • 52. ⮚ also called as pulp polyp or pulpitis aperta ⮚ essentially an excessive exuberant proliferation of chronically inflammed dental pulp tissue Chronic Hyperplastic Pulpitis
  • 53. ⮚ pulpal inflammation due to an extensive carious exposure of a young pulp ⮚ development of granulation tissue ⮚ covered at times by epithelium ⮚ resulting from long standing low grade infection Chronic Hyperplastic Pulpitis
  • 54. ⮚ Causes ✔ slow progressive exposure of pulp ✔ bacterial infection Chronic Hyperplastic Pulpitis
  • 55. ⮚ Clinical Features ✔ most commonly involved are deciduous molars + 1st permanent molar • excellent blood supply • large root opening Chronic Hyperplastic Pulpitis
  • 56. ⮚ Clinical Features ✔ asymptomatic ✔ seen only in teeth of children + young adults Chronic Hyperplastic Pulpitis
  • 57. ⮚ Clinical Features ✔ polypoid tissue appears • fleshy • reddish pulpal mass filling most of pulp chamber or cavity • or even extend beyond confines of tooth Chronic Hyperplastic Pulpitis
  • 58. ⮚ Clinical Features ✔ polypoid tissue appears • sometimes, if mass is large enough • interferes with closure of mouth Chronic Hyperplastic Pulpitis
  • 59. ⮚ Clinical Features ✔ polypoid tissue appears • may cause discomfort during mastication • due to pressure of food bolus Chronic Hyperplastic Pulpitis
  • 60. ⮚ Clinical Features ✔ polypoid tissue appears • tissue easily bleeds because of rich network of blood vessels • tooth may respond or not at all to thermal test Chronic Hyperplastic Pulpitis
  • 61. ⮚ Management ✔ elimination of polypoid tissue ✔ followed by extirpation of pulp ✔ hyperplastic tissue bleeeding can be controlled by pressure ✔ pulpectomy or extraction of tooth can also be done if the tooth is not restorable. Chronic Hyperplastic Pulpitis
  • 62. ⮚ death of pulp ⮚ may be partial or total depending on whether part or the entire pulp is involved Necrosis
  • 63. ⮚ Causes ✔ sequeala of inflammation ✔ can also occur following trauma • pulp is destroyed before an inflammatory reaction Necrosis
  • 64. ⮚ Types ✔ (1) Coagulation Necrosis ✔ (2) Liquefaction Necrosis Necrosis
  • 65. ⮚ Types ✔ (1) Coagulation Necrosis • soluble portion of tissue is precipitated or converted into a solid material Necrosis
  • 66. ⮚ Types ✔ (1) Coagulation Necrosis • tissue is converted into tissue mass consisting chiefly of coagulated ▪ proteins ▪ fats ▪ water Necrosis
  • 67. ⮚ Types ✔ (2) Liquefaction Necrosis • results when proteolytic enzymes convert the tissue into softened mass liquid or amorphous debris Necrosis
  • 68. ⮚ Clinical Features ✔ no painful symptoms ✔ discoloration of tooth • 1st indication that the pulp is dead Necrosis
  • 69. ⮚ Clinical Features ✔ history of pain lasting from a few minutes to a few hours followed by complete + sudden cessation of pain Necrosis
  • 70. ⮚ Management ✔ preparation + obturation of root canals Necrosis
  • 71. References: ❖ Books ⮚ Grossman’s endodontic practice twelfth edition ⮚Textbook of oral pathology shafers ⮚Cawson, R.A: Cawson’s Essentials of Oral Pathology and Oral Medicine, 8th Edition.(page 60) ⮚ Ghom, Ali & Mhaske, Shubhangi: Textbook of Oral Pathology. (pages 420-425)
  • 72. References: ⮚Adrian,J.C.,et al.:J. Am. Dent. Assoc.,83:113, 1971. ⮚Allard, U., et al.:Oral Surg.,48:454, 1979. ⮚Austin, L.T.:J. Am. Dent. Assoc.,17:1930, 1930. ⮚Baume, L.: SSO Schweiz. Monatsschr. Zahnheilkd.,77:1082, 1965.
  • 73. References: ⮚Baume, L.: Transactions of the Fourth International Conference on Endodontics. Philadelphia: University of Pennsylvania press, 1968, p. 66 . ⚫Baume, L.J.: Monogr. Oral Sci.,8:1- 220, 1980.