This document provides definitions and information about diet, nutrition, and their importance for oral health. It discusses the major components of a balanced diet including macro-nutrients like carbohydrates, proteins, and fats, as well as micro-nutrients like vitamins and minerals. Specific vitamins and minerals that are important for dental health such as vitamins A, D, and C are explained. The roles of important minerals like calcium, phosphorus and magnesium are also summarized. The document provides recommendations for nutritional assessment and counselling in children.
Dental Plaque
Soft deposits that form the biofilm adhering to the tooth surface or other hard surfaces in the oral cavity, including removable & fixed restorations”
Bowen , 1976
Bacterial aggregations on the teeth or other solid oral structures
Lindhe, 2003
Dental management of children with special health care needsaravindhanarumugam1
hope this will throw a light in understanding special children and dental management of the same particularly for pediatric dentistry PGs .children with genetic diseases and emotionally handicapped ( child abuse and neglect ) are not discussed here as they are separate topics.
dr. aravindhan
In this lecture I explain in step-by-step fashion the basics of Endodontic Diagnosis: Pulp Vitality Tests. a photo guide is attached to the guide to aid in better understanding of the topic
Relationship between the type of food, frequency of intake and various cariogenic and non-cariogenic factors which influence initiation and progression of caries have been studied over the years.
Dental Plaque
Soft deposits that form the biofilm adhering to the tooth surface or other hard surfaces in the oral cavity, including removable & fixed restorations”
Bowen , 1976
Bacterial aggregations on the teeth or other solid oral structures
Lindhe, 2003
Dental management of children with special health care needsaravindhanarumugam1
hope this will throw a light in understanding special children and dental management of the same particularly for pediatric dentistry PGs .children with genetic diseases and emotionally handicapped ( child abuse and neglect ) are not discussed here as they are separate topics.
dr. aravindhan
In this lecture I explain in step-by-step fashion the basics of Endodontic Diagnosis: Pulp Vitality Tests. a photo guide is attached to the guide to aid in better understanding of the topic
Relationship between the type of food, frequency of intake and various cariogenic and non-cariogenic factors which influence initiation and progression of caries have been studied over the years.
Nutrition is very important for a growing child as it not only effects the general health but also the oral health, which are ultimately interrelated. This presentation will help you to understand Nutrition as a Pediatric Dentist.
Introduction
Definitions
Nutrition & Normal occlusion
Calcium
Phosphorous
Vitamins D , A & C
Nutritional deficiencies
Nutrition & Dentofacial growth
Role of Nutritional counseling
Conclusion
Nutritional deficiencies and dentofacial growth /certified fixed orthodontic ...Indian dental academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
Indian dental academy provides dental crown & Bridge,rotary endodontics,fixed orthodontics,
Dental implants courses.for details pls visit www.indiandentalacademy.com ,or call
0091-9248678078
Idk if you are you doing tomorrow morning yet to be treated as I'm sure you are not picking up my room and hospital near Pali road kudi tu hi tu hi hai ki yai kashmiri mirch masala is tinu abong tar ammur hubby is abong tar ammur hubby and I will you be interested please contact the person who are you not replying sooner or later version you doing tomorrow and then you will you go now please send me your address and phone numbers of supreme personality is tinu abong tar chehara I am not yet to be treated as such as I'm unable open to the Prostho department rn you are not picking call in the group about this one also good looking for the students of Bangladesh in my AICTE you doing today also sleeping on it rn Shukla and we have to get up to other room and hospital jodhpur rajasthan me to come to there house and hospital jodhpur rajasthan high court can you please send the link to other room and hospital near Pali Rajasthan State dental council which one is better than the intended recipient please notify us immediately and hospital near Pali Rajasthan State
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
- Video recording of this lecture in English language: https://youtu.be/kqbnxVAZs-0
- Video recording of this lecture in Arabic language: https://youtu.be/SINlygW1Mpc
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Here is the updated list of Top Best Ayurvedic medicine for Gas and Indigestion and those are Gas-O-Go Syp for Dyspepsia | Lavizyme Syrup for Acidity | Yumzyme Hepatoprotective Capsules etc
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
ABDOMINAL TRAUMA in pediatrics part one.drhasanrajab
Abdominal trauma in pediatrics refers to injuries or damage to the abdominal organs in children. It can occur due to various causes such as falls, motor vehicle accidents, sports-related injuries, and physical abuse. Children are more vulnerable to abdominal trauma due to their unique anatomical and physiological characteristics. Signs and symptoms include abdominal pain, tenderness, distension, vomiting, and signs of shock. Diagnosis involves physical examination, imaging studies, and laboratory tests. Management depends on the severity and may involve conservative treatment or surgical intervention. Prevention is crucial in reducing the incidence of abdominal trauma in children.
Integrating Ayurveda into Parkinson’s Management: A Holistic ApproachAyurveda ForAll
Explore the benefits of combining Ayurveda with conventional Parkinson's treatments. Learn how a holistic approach can manage symptoms, enhance well-being, and balance body energies. Discover the steps to safely integrate Ayurvedic practices into your Parkinson’s care plan, including expert guidance on diet, herbal remedies, and lifestyle modifications.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of the physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar lead (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
6. Describe the flow of current around the heart during the cardiac cycle
7. Discuss the placement and polarity of the leads of electrocardiograph
8. Describe the normal electrocardiograms recorded from the limb leads and explain the physiological basis of the different records that are obtained
9. Define mean electrical vector (axis) of the heart and give the normal range
10. Define the mean QRS vector
11. Describe the axes of leads (hexagonal reference system)
12. Comprehend the vectorial analysis of the normal ECG
13. Determine the mean electrical axis of the ventricular QRS and appreciate the mean axis deviation
14. Explain the concepts of current of injury, J point, and their significance
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. Chapter 3, Cardiology Explained, https://www.ncbi.nlm.nih.gov/books/NBK2214/
7. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
3. CONTENTS
• Definition
• Balanced diet
• Components of nutrition
• Macro-nutrients
• Vitamins
• Minerals and other micro-nutrients
• Assessment of nutritional status
• Diet counselling
• AAPD recommendations
• Nutrition affecting growth and development of infant to
Adolescent
• Nutrition considerations in children with special health Care
needs
• References
3
4. Questions asked previously
• Long essays
1. How is vitamin D formed & activated in the
body. How it regulates body calcium
pool.Add a note on hypo and
hypervitaminosis in growing child.
2. Discuss the role of nutrition in dental and
oral health of children.
3. Discuss the role of nutrition in a changing
society and their dental implications.
4
5. Short essays
1. Vitamin D & Calcium homeostasis
2. Relationship between diet, nutrition & dental caries.
3. Vitamin C
4. Trace elements in dental caries
5.Fat soluble vitamins
6.Dietary recommendations in children with caries risk
7. Balanced diet
8. Stepwise nutritional evaluation of a child
9.Diet diary
10. Diet counselling in rampant caries
5
7. DIET
It is referred to food
and drink regularly
consumed
Nizel 1989
Total oral intake of
substance that provide
nourishment and
energy.
7
8. NUTRITION
Who 1971
Science of food and
its relationship to
health.It is concerned
primarily with the part
played by the nutrients
in body
growth,development
and maintainence
The sum of processes
concerned in the
growth, maintenance
and repair of the living
body as a whole or of
its constituent parts.
8
9. FOOD
Any substance which
when taken into the
body of an organism
may be used either to
supply energy or to
build tissue.
Nizel 1989
Anything that is
eaten,drunk or
absorbed for
maintenance of
life,growth and
repair of the tissues
9
11. BALANCED DIET
• One which supplies all the nutrients in the
right quantity and proportion.
11
12. RDA
12
“levels of intake of
essential nutrients that
on a basis of
scientific knowledge are
adequate to meet the
known
nutrient needs of all
healthy persons.”
American food &
nutrition board
Assuming 2400 kcal as 1 unit of energy, RDA is
expressed as a proportion of this.(ICMR, Hyderabad)
Holliday and Segar formula
age of the child
Weech formula
For bedside approximation of expected weight and
height
Infant weight = (age in months)+9/2
18. COMPONENTS OF DIET
18
MAJOR NUTRIENTS
Carbohydrates: 65 to 80 per
cent
Proteins :7 to 15 per cent
Fats/lipids:10 to 30 per cent
TRACE NUTRIENTS
MICRO NUTRIENTS
RDA < 1mg/day
Vitamins
Minerals
19. Proteins
24 amino acids of which 9 are essential amino acids
and the remaining are non essential amino acids.
Functions
Body building
Tissue repair & maintenance
Synthesis of antibodies,plasma proteins,Hb,
Hormones,Enzymes
Deficiency:PEM
19
21. Kwashiorkor
21
Prof Cicely Williams
‘red boy’ “deposed
child”.
Classic signs:
Stunted growth
Hepatomegaly
Anaemia
Oedema
Grading
Grade I- pedal
oedema
Grade II- I +facial
oedema
Grade III – II +
paraspinal and chest
oedema
Grade IV- III +
ascites
22. MARASMUS
.
• Affected children exhibit extreme wasting.
• Old man appearance to jaws and skin and bones.
• Wasting of brown fat occurs first.
• Marasmic Kwashiorkar: when marasmic children
develop oedema.
22
Greek word
Marasmos
=Wasting
Grading
Grade I: wasting starting in
axilla and groin.
Grade II: I + wasting in thigh
and buttock region
Grade III: II+ chest and
abdomen
Grade IV: buccal pad of fat
23. Proteins & Oral health
Atrophy of the gingiva
Degeneration of cementum and supporting periodontal
tissues
23
Malocclusion In A Pem Child Hypoplasia Of Pem Child
Tongue: Bright
red,loss of
papilla,edema
Dry mouth
Fissured lip
Loss of
circumoral
pigmentation
Reduced caries
activity(Lack of
substrate)
Delayed
eruption
24. VITAMINS
• Vitamins and Minerals form the protective
foods and are also called functional foods.
• Vitamins do not yield energy but enable the
body to use other nutrients
24
Fat soluble
Water
soluble
25. Vitamin A
25
Importance
Plays an
important
role in Walds
visual cycle
Deficiency
-Night blindness
Xeropthalmia
-Bitots spots
Keratomalacia
Oral Manifestations
1. Keratinising Metaplasia of
epithelium(increased keratin
formation)
2. Occlusion of salivary gland
ducts with keratin.
3. Enamel hypoplasia
4. Atypical dentin formation
5. Epithelial invasion of
connective tissues
6. Delayed eruption of teeth.
RDA:
1500 IU (500 μg)
26. Vitamin D (Anti Rachitic Vitamin)
26
1,25 –DHCC is active form.
Acts like a hormone.
Calcitriol increases the serum
calcium & phosphorus level by
increasing intestinal absorbtion
& reducing renal excretion
27. Vitamin D (Anti Rachitic Vitamin)
27
Deficiency
Rickets in children
Osteomalacia in adults
Pigeon chest is a feature
Renal rickets (CRF)
Plasma
calcitriol
Alkaline
phosphatase
Oral manifestations
1. Delayed eruption of
primary & permanent
teeth.
2. Developmental
anomalies of enamel
and dentin
3. Wide predentin zone
4. Increased
interglobular dentin
5. Elongated pulp horns
28. Vitamin E(Shady lady of nutrition)
• Antioxidant
• Protects liver from toxic
compounds
• Maintains germinal health of
gonads
• Protects RBC from hemolysis
• Prevents heart disease by
preventing oxidation of LDL
• Decreased male fertility
• Enacephalomalcia
• Nutritional muscular
dystrophy
Deficiency • Loss of pigmentation
• Atrophic degenerative
changes in enamel.
• Derangement of
ameloblasts.
Oral
manifestations
28
29. Vitamin K(Coagulation vitamin)
• Brings about post translational
modification of 2,7,9,10 protein c
protein s factors.
• Deficiency is uncommon may result in
prolonged CT
• Prothrombin levels below 35% results
in bleeding on brushing.
• Below 20% results in spontaneous
bleeding.
29
30. Vitamin B1(THIAMINE)
Plays important role in metabolism of
carbohydrates, alcohol and branched
chain amino acids.
30
Deficiency is seen in populations consuming polished rice.
Dry beri beri (peripheral neuritis)
Wet beri beri( Cardiac manifestations)
Cerebral or Wernickes encephalopathy with Kosakoff’s psychosis.
Oral manifestations
Burning tongue
Senstive OMM
Loss of taste
31. Vitamin B2 (Riboflavin)
• Has a vital role in cellular oxidation.
• It is a cofactor in number of enzymes involved with energy
metabolism.
• Deficiency manifests as angular stomatitis,
cheilosis, atrophic papillae on tongue
• In severe cases, tongue becomes glazed and
smooth due to complete atrophy of papillae.
• Lips: red and shiny because of epithelial
desquamation.
31
32. Niacin
• Nicotinic acid is essential for metabolism of
carbohydrate, proteins, and fat.
• It is also essential for normal functioning of skin,
intestinal and nervous system.
• Part of NADP co-enzymes.
• Deficiency state is termed PELLAGRA which
leads to dermatitis, diarrhoea and dementia.
• Casal’s necklace and glove and stocking type
dermatitis occurs in the exposed parts..
• Glossitis & Stomatitis
32
33. Pyridoxine
33
•Plays an important role in the metabolism of
amino acids, fats, and carbohydrates
It keeps up the level of GABA, an
inhibitory neurotransmitter.
• Deficiency:
Peripheral Neuritis
Rashes
Convulsions
•Oral manifestations:
Cheilosis
Glossitis
Angular stomatitis
34. Folic acid
34
Folic acid plays an important role in the synthesis of
nucleic acids and development of Red blood cells in the
bone marrow.
Deficiency leads to megaloblastic anemia,
diarrhoea, and knuckle and periungual pigmentation.
Oral manifestations: Chelitis,glossitis
Preconceptional administration can prevent neural tube
defects in the baby.(RDA : 400mcg in pregnancy)
35. Vitamin B12(Cyanocobalamine)
35
Takes part in synthesis of fatty acids in myelin.
Essential in nucleic acid synthesis.
Deficiency leads to pernicious anemia,peripheral
neuritis,degeneration of spinal cord .
Oral Manifestations:
Sore painful tongue, glossitis and glossodynia
Beefy red tongue
Small shallow ulcers with atrophy of papillae with a
loss of normal muscle tone, called as Hunter’s
glossitis.
36. Vitamin C (Ascorbic Acid)
Functions
Converts proline to
hydroxyproline, which is a
constituent of collagen.
Involved in collagen synthesis
and teeth formation.
Increases iron absorption.
Acts as antioxidant due to its
reducing property.
Deficiency
Scurvy
Spongy ,sore gums,loose
teeth,anemia,swollen
joints,haemorrhage.
“Cork screw hair pattern”
“Woody legs”
Oral manifestations
Pathognomic signs swollen and
spongy papillae , particularly
interdental papillae producing
scurvy buds.
In severe cases haemorrhages
to periodontal membranes
followed by loose teeth.
36
38. Calcium
A major element of the body.
• Provides rigidity and strength to
bones and teeth.
• Calcium plays an important role
in blood coagulation, muscle
contraction, myocardial action,
and neuro muscular irritability
and is responsible for integrity
of various membranes.
38
Vitamin D
ParathhormoneCalcitonin
Deficiency
Osteomalacia, rickets, fracture
susceptible bones.
Impaired enamel apatite crystals
formation.
Low blood calcium causes TETANY.
39. Phosphorus
Second most abundant mineral.
Formation of bone and tooth.
Absorption and transport of nutrients.
Regulates acid – base balance.
energy released due to metabolism of carbohydrates, fats
and proteins is accomplished by phosphates (ADP).
Phosphates play an important role in cell protein
synthesis. It is a part of DNA and RNA.
39
40. Magnesium
Magnesium is essential for cellular respiration,
functioning chiefly as an activator for numerous
important coenzymes such as carboxylase and Co
enzyme A.
Plays an important role in synthesis of carbohydrates,
fats and proteins.
Helps in regulation of acid base balance of the
Magnesium is present in enamel and dentin but
more in dentin.
40
Magnesium
deficiency causes
chronic
malabsorption
syndrome, acute
diarrhea, renal
failure, weakness,
tremors,convulsions,
hyper excitability.
41. Iron
Iron is necessary for formation of hemoglobin, brain development and
function.
Regulates body temperature and muscle activity.
Increases the production of T CELLS.
It helps in the production of antibodies.
Iron binds oxygen to blood cells, and helps in oxygen transport and cell
respiration.
41
42. Iron Deficiency
42
Stages
1. Decreased storage of iron
without any detectable
abnormalities.
2. Intermediate deficiency of iron
stores getting exhausted
but no evidence of anemia.
3. Overt iron deficiency with
decreased hemoglobin
concentration.
less than 12
g/dl for a child.
Iron-deficiency anaemia –
hypochromic
microcytic anemia
characterized by low
serum iron, increased serum
iron-binding
capacity, decreased serum
ferritin, and
decreased marrow iron
stores.
43. Clinical features of Anemia
• Weakness, fatigue, pallor, tingling of extremities, brittle nails.
• Spoon shaped nails (koilonychias), altered hair growth.
ORAL MANIFESTATIONS
•Inflammation of the tongue, atrophy of tongue.
•Smooth shiny red appearance of tongue.
•Dysphagia, grayish mucous membrane.
•Angular stomatitis.
•Combination of above all features is termed as
• PLUMMER VINSON SYNDROME.
43
44. Iodine
• Iodine is an integral part of the thyroid
hormones THYROXIN and tri IODO THYRONINE
whose function is to maintain the control of
energy metabolism of the body.
44
45. Deficiency of Iodine
• Hypothyroidism
• CRETINISM and MYXEDEMA
are pathological conditions
• Skin is dry and coarse
• Metabolism is slow.
• Macroglossia
• Stunted growth, delayed tooth
eruption
• Retarded mental activity
45
46. Hyperthyroidism
• The excessive activity of the
thyroid gland that is
brought on by a deficiency
of iodine characterized by
increased pulse
rate,temperature and blood
pressure with nervousness ,
irritability,sweating, weight
loss, dyspnea, and
tiredness. Patients may also
develop EXOPTHALMOUS.
46
47. TRACE ELEMENTS AND DENTAL CARIES
• Aside from fluorine certain trace elements in diet possess
increased resistance or susceptibility to caries.
• Molybdenum(Hungary)
• Vanadium(Tank &
Storvick)
• Strontium(works with F)
CARIOSTATIC
• Selenium(Oregon)
• Magnesium
• Cadmium
CARIOGENIC
51. ANTHROPOMETRICS
• STATURE:CDC guidelines define a height-for
age value less than 5th percentile as short
stature.
• CHEST CIRCUMFERENCE:Measured at the
nipple midway between inspiration and
expiration.
51
Rayner and Rudolf
low weight
for-age is a
marker of
Failure to
thrive
Head circumference
35cm at birth
50 cm by 3 yrs
55. Food Frequency Questionnaire
• List of around 100
items +servings,
• Daily ,monthly
• Errors in noting
serving size
• Long,tedious
55
56. Diet history
• Interview method
56
• Establish overall eating
pattern
• 24 hour recall
• Household measures of
servings
First
part
• Cross check
• Food preferences
established
Second
part
57. Food diary
• Amount , frequency,
type of food
consumed
• Period of collection
1-7 days
• Reliable,but difficult
to maintain
57
59. • Diet counseling (Katz 1981)- it is the correction of
diet imbalance that could affect the patients general
health and sometimes is also reflected in his oral
health.
59
60. Five ‘W’ and one ‘H’ of diet consultation
• WHO may be benefited?
• WHAT are the objectives of diet and nutrition counseling?
• WHY is counseling beneficial?
• WHEN is counseling conducted?
• WHERE should the counseling occur?
• HOW to counsel?
60
61. DENTAL HEALTH DIET SCORE
• Screening device
• Food score (adequate intake of foods from each of the food
groups) + Nutrient score(consuming foods from especially
recommended groups of ten nutrients) - Sweet score (frequent
ingestion of foods that are overtly sweet)
• 60-100- acceptable- dietary counselling not given unless
requested
• 56 or less indicated and recommended
61
62. Instruction for calculating a dental
health diet score
Record
(what,when,
how much)
Circled
Uncircled
Check
8 columns of
food
Check & add
nutrient
score(56)
62
Sweet
score
Put it all
together
63. Food group RDA NO.OF
SERVINGS
POINTS
MILK 3 X 8 HIGHEST
POSSIBLE
SCORE 24
MEAT 2 X 12 24
FRUITS AND
VEGETABLES
1 X 6 6
OTHERS 2 X 6 12
BREAD AND
CEREALS
4 X 6 24
FOOD GROUP SCORE 96 IS THE HIGHEST
63
FOOD GROUP EVALUATION CHART
64. Protein and Niacin
7
Vitamin A 7 Iron 7 Folic acid
Cheese
Dried beans
Dried peas
Eggs
Fish
Meat
Milk
Nuts
poultry
Apricot margarine
Broccoli milk
Butter peaches
Cantaloupe squashes
Carrots spinach
Eggs sweet
potato
Greens
Liver
Beef
Broccoli
Eggs
Green
leafy
vegetables
Liver
Oyster
Sardines
shrimp
Asparagus
Broccoli
Cereals
Kidney
Liver
Spinach yeasts
64
NUTRIENT EVALUATION CHART
66. Step 4-Scoring the sweet
• Classify each sweet into liquid, solid and sticky or slowly dissolving
• For each time the sweet was eaten either at the end of a meal or
between meals(atleast 20 mins apart) place a check in the frequency
column
1. Add the checks
2. (Highest score-35)
66
FORM FREQUENCY POINTS
liquid 5
Solid and sticky 10
Slowly dissolving 15
67. Step 5
• Now put it all together
• Food group score
• 72-96-excellent
• 64-72-adequate
• 56-64-barely adequate nutrition counselling
• 56 or less-not adequate
67
68. • Sweet score
• 5 or less- excellent
• 10 -good
• 15 or more-watch out zone
• If your sweet score is in “watch out” zone your dentist will
talk about improvements
68
69. 69
Interviewing
• Separate room
• Indicates respect for
privacy
Physical Setting
• Eye contact
• Verbal,Non verbal
• Teaching,motivation
Effective
communication
70. Steps of decision making
Awareness
Interest
Involvement
Action
Habit
70
71. COUNSELLING APPROACHES
• Role of patient is passive
• Decision made by counselor
for patientDIRECTIVE
• Counselor’s role is merely to
aid the patient
• More recommendedNON-DIRECTIVE
71
72. NIZEL’S RULES
• 4 rules are
72
Maintain overall
nutrition
Vary slightly from the
normal diet pattern
Should meet body’s
requirements
Should take into
consideration
patient’s likes and
dislikes
73. Limit no. of eating to three meals per
day
Increase intake of protective food
Decrease carbohydrate
Wean from taste of sweet
Raw fruit and raw vegetable
Fluoridated water
73
For prevention of dental caries general principles
are as follows
74. Breast-feeding of infants
with care to wiping or
brushing as the first
primary tooth begins to
erupt
Educating association
between frequent
consumption of
carbohydrates and caries.
Educating about health
risks associated with excess
consumption of simple
carbohydrates, fat, saturated
fat, and sodium.
The AAPD encourages
74
Pediatric dentists and other
health care providers who treat
children to provide dietary and
nutrition counseling
75. Food and beverage
manufacturers to make
nutritional content on
food labels more
prominent and
“consumer-friendly”.
Furthermore the AAPD encourages
75
School health
education programs and
food services to
promote nutrition
programs
Pediatric dentists and other
health care providers to
recommend or prescribe
sugar-free medications
whenever possible.
76. Nutritional considerations from
infancy to adolescence
• 0-1 year
76
Milk substitutes:
Regular unmodified cow’s milk is not
suitable:
Insufficient source of vitamin C and iron.
It may cause gastrointestinal bleeding.
Its solute load is too heavy for the infant’s
renal system to handle .
Low-fat milk: should not be used,
Insufficient energy provision.
Lack of essential fatty acids
On guard against dehydration
77. Infant -Toddler
2 years
• Reduction in apetite
• Dietary needs for
proteins and minerals
remain high.
• During first two
years of life: 40-50%
of energy should
come from fat.
• Consume 3 regular
meals per day
77
Older than 2 years:
Roughly 30% should come from fat, with no
more than 10% from either saturated
fats or polyunsaturated fats.
Carbohydrates: 55-60% of calorie
requirements with no more than 10% from
simple
sugars.
78. Preschooler
3-6 years
78
• Physical growth occurs in spurts.
• Fewer calories are required, but relatively
high protein and mineral needs remain.
• Child should be helped to lose ‘baby fat’ by
increasing physical activity rather than by
severely restricting calories.
• Wholesome, nutritious, low sugar snacks
can promote adequate intake of essential
nutrients without adding calories or
promoting dental caries.
79. School going child(6-12 year)
• Decline in food
requirements per unit
body weight( Because
of reduction in growth
rate.)
• Children should be
encouraged to have
breakfast.
• Thus, emphasis on high
nutrient density : High
ratio of nutrients to
calories
79
80. Adolescence
12-18 years
• Peer pressure & sociocultural influence.
• Weight control
• Development of eating disorders.
• Cigarette smoking to lose weight
• Nutritional requirements are influenced primarily by onset of
puberty and the final growth spurt of childhood.
• Increased needs for energy, protein, minerals and vitamins.
• “Female athlete triad” : American College of Sports Medicine
(ACMS) in 1992.
Seen among adolescent female athletes
• Disordered eating behaviours
• Amenorrhoea
• Osteoporosis
80
81. Special children considerations
81
Estimated 40%
Factors to be considered
Decreased apetite
Parental overindulgence
,overprotection
Poor oral hygiene & prevention
Long term use of cariogenic
medications
Xerostomia
82. Recommendations(AAPD)
Dental home by
age 1
Oral hygiene
management(2-2-2
rule)
Antimicrobial
products
Remineralising
agents
Management of
pain & discomfort,
xerostomia
82
84. REFERENCES
1. Nutrition in preventive dentistry – Nizel and Papas.277-308
2. Finn SB. Clinical Pedodontics. 4th edn. WB Saunders company,
Philadelphia;2004.
3. Damle SG. Textbook of pediatric dentistry. 4th edn.Arya Medi
Publications, New Delhi.2014.
4. Marwah N. Textbook of pediatric dentistry.3rd edn.
5. Tandon S. Textbook of Pedodontics. 2nd edn. Paras medical
publishers.2009
6. Diet Counselling – A Primordial Level of Prevention of Dental Caries. Dr.
Girish V Chour, Dr. Rashmi G Chour. IOSR .2014;13:64-70.
8. American Association of Pedodontics and American Academy of Pediatric
dentistry . Policy on dietary recommendations for infants children and
adolescents. Reference Manual; 37:no. 6.15-16.
84