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INJURIES OF CHEST
DR.SHAIK EZDA JANI
GENERAL SURGERY PG 1
PNEUMOTHORAX
• INTRODUCTION
• Pneumothorax = presence of air outside the lung,
within the pleural space
• TWO TYPES
1. SPONTANEOUS PNEUMOTHORAX
2. TENSION PNEUMOTHORAX
PNEUMOTHORAX: CAUSES
1.PRIMARY SPONTANEOUS
PNEUMOTHORAX
• usually tall and thin young men
• Due to leak from blebs, vesicles or bullae
• Most common seen at upper lobe of lung
2.SECONDARY SPONTANEOUS
PNEUMOTHORAX
• Occurs when visceral peritoneum leaks
• Due to underlying lung diseases
• Seen in older patients
DIAGNOSIS:
• Chest X-ray:
• CT Chest:
• Occasionally
• Assess cause of spontaneous pneumothorax
• presence of other occult lung disease
TREATMENT:
• depends on size and symptoms
• Smaller pneumothorax may resolve spontaneously
• Progression in the size : intervention with drainage
• Initial spontaneous pneumothorax:
• small bore catheter drainage
• or chest tube and drainage with resolution of the air space and
cessation of air leak
• Persistent air leak (>5 days) or failure of the lung to expand fully
suggests additional intervention
INDICATIONS: SURGICAL
INTERVENTION
● Second ipsilateral pneumothorax
● First contralateral pneumothorax
● Bilateral spontaneous pneumothorax
● Pneumothorax fails to settle despite chest drainage
● Spontaneous haemothorax: professions at risk (e.g. pilots, divers)
● Pregnancy
CHEST DRAIN: INSERTING AND
MANAGING
INSERTION:
1. TRIANGLE OF SAFETY
• Penetration of the skin,
muscle and pleura
• Blunt dissection of the
parietal pleura;
• suture placement
• gauging the distance of
insertion
• digital examination
along the tract into the
pleural space
• withdrawal of
central trochar
and positioning of
drain
• Underwater seal chest drain bottle.
SURGICAL MANAGEMENT
• Can be performed by :
• video-assisted thoracoscopic surgery (VATS) or
• open procedure (Thoracostomy)
• Pleurectomy and pleurodesis
Objectives:
• deal with any leaks from
the lung
• search for and obliterate
any blebs and bullae
• make the visceral pleura
adherent to the parietal
pleura
• Pleural adhesion is achieved in one of three ways:
PLEURECTOMY
PLEURAL ABRASION
CHEMICAL PLEURODESIS
TENSION PNEUMOTHORAX
• Develops due to one way valve air leak
• Mediastinum displace to opposite side
• Compressing the affected lung
• Decreased venous return
• Mc cause penetrating chest trauma
Clinical presentation
• Tachypnea
• Dyspnea
• Distended neck veins
• Tracheal deviation
• Hyper resonance and decreased breath sounds
TREATMENT
• IMMEDIATE DECOMPRESSION
• With large wide bore canula
• Followed by insertion of chest tube in safety triangle
FLAIL CHEST
INTRODUCTION
• Results from blunt trauma associated
with multiple rib fractures
• Defined as three or more ribs fractured
in two or more places.
• blunt force typically also produces an
underlying pulmonary contusion
MECHANISM
DIAGNOSIS
• made clinically in patients who are not ventilated,
• not by radiography
• Confirmation: paradoxical motion of a chest wall segment
• Voluntary splinting of the chest
wall due to:
• Pain
• Mechanically impaired chest wall
movement
• Lung contusion
HYPOXIA
MANAGEMENT:
• Traditionally, mechanical ventilation was
used to ‘internally splint’
• Disadvantage:
• intensive care unit (ICU) resources
• ventilation-dependent morbidity
• Currently:
• oxygen administration
• adequate analgesia (including opiates)
• Physiotherapy
• If chest tube is in place: topical intrapleural local analgesia
• Ventilation (respiratory failure )
• Surgery :
• stabilise the fail segment using internal fxation of the ribs
• patients with
• isolated or severe chest injury
• pulmonary contusion.
HAEMOTHORAX
• BLUNT INJURY(m/c)
• torn intercostal vessels
• internal mammary artery
secondary to fractures of
the ribs
• PENETRATING INJURY
• Viscera: both thoracic and
abdominal
• blood leaking through a
hole in the diaphragm
from the positive pressure
abdomen into the
negative pressure thorax
PRESENTATION : HAEMOTHORAX
• haemorrhagic shock
• fat neck veins
• unilateral absence of breath sounds
• dullness to percussion
MANAGEMENT:
• INITIAL TREATMENT:
• correcting the hypovolaemic shock
• insertion of an intercostal drain
• Intubation
• Indication for urgent thoracotomy
• Initial drainage > 1500 mL of blood
• ongoing haemorrhage > 200 mL/h over 3–4 hours
• NOTE: There is no role for clamping a chest tube to tamponade a massive
haemothorax.
• IMPORTANT POINTS:
• if the lung does not reinfLate -> the drain should be placed on low-pressure
(5 cmH2O) suction
• Check for clot occlusion of chest tube
• Second drain is sometimes necessary
• Role of chest radiograph or eFAST
• physiotherapy and active mobilisation
CARDIAC TAMPONADE
PERICARDIAL TAMPONADE
• INTRODUCTION:
• m/c due to penetrating trauma
• All patients with penetrating injury anywhere
near the heart + shock must be considered =
cardiac injury until proven otherwise.
MECHANISM: CARDIAC TAMPONADE
• Accumulation of small amount(50ml) blood
in non distensible pericardial sac
• compression of the heart ;
• obstruction of the venous return,
• decreased filling of the cardiac chambers
during diastole
PRESENTATION
• central venous pressure elevation,
• a decline in arterial pressure
• tachycardia
• mufed heart sound
• deteriorating cyanosis
• agitation
• earliest manifestation is an acute drop in mixed
venous oxygen saturation
DIAGNOSIS
• eFAST
• fuid in the pericardial sac
• Most reliable
• chest radiography
• looking for an enlarged heart shadow
MANAGEMENT:
• NOTE: Pericardiocentesis has no role in the management of cardiac
tamponade secondary to penetrating myocardial injury
• Immediate treatment:
• Operative:
• subxiphoid window or
• by open surgery (sternotomy or left anterolateral thoracotomy)
• with evacuation of the haematoma and repair of the myocardium
THANK YOU

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CHEST TRAUMA JANI 2.pptx

  • 1. INJURIES OF CHEST DR.SHAIK EZDA JANI GENERAL SURGERY PG 1
  • 2. PNEUMOTHORAX • INTRODUCTION • Pneumothorax = presence of air outside the lung, within the pleural space • TWO TYPES 1. SPONTANEOUS PNEUMOTHORAX 2. TENSION PNEUMOTHORAX
  • 4. 1.PRIMARY SPONTANEOUS PNEUMOTHORAX • usually tall and thin young men • Due to leak from blebs, vesicles or bullae • Most common seen at upper lobe of lung
  • 5. 2.SECONDARY SPONTANEOUS PNEUMOTHORAX • Occurs when visceral peritoneum leaks • Due to underlying lung diseases • Seen in older patients
  • 6. DIAGNOSIS: • Chest X-ray: • CT Chest: • Occasionally • Assess cause of spontaneous pneumothorax • presence of other occult lung disease
  • 7. TREATMENT: • depends on size and symptoms • Smaller pneumothorax may resolve spontaneously • Progression in the size : intervention with drainage • Initial spontaneous pneumothorax: • small bore catheter drainage • or chest tube and drainage with resolution of the air space and cessation of air leak • Persistent air leak (>5 days) or failure of the lung to expand fully suggests additional intervention
  • 8. INDICATIONS: SURGICAL INTERVENTION ● Second ipsilateral pneumothorax ● First contralateral pneumothorax ● Bilateral spontaneous pneumothorax ● Pneumothorax fails to settle despite chest drainage ● Spontaneous haemothorax: professions at risk (e.g. pilots, divers) ● Pregnancy
  • 9.
  • 10. CHEST DRAIN: INSERTING AND MANAGING
  • 12. • Penetration of the skin, muscle and pleura • Blunt dissection of the parietal pleura; • suture placement
  • 13. • gauging the distance of insertion • digital examination along the tract into the pleural space • withdrawal of central trochar and positioning of drain
  • 14. • Underwater seal chest drain bottle.
  • 15. SURGICAL MANAGEMENT • Can be performed by : • video-assisted thoracoscopic surgery (VATS) or • open procedure (Thoracostomy) • Pleurectomy and pleurodesis
  • 16. Objectives: • deal with any leaks from the lung • search for and obliterate any blebs and bullae • make the visceral pleura adherent to the parietal pleura
  • 17. • Pleural adhesion is achieved in one of three ways: PLEURECTOMY PLEURAL ABRASION CHEMICAL PLEURODESIS
  • 18. TENSION PNEUMOTHORAX • Develops due to one way valve air leak • Mediastinum displace to opposite side • Compressing the affected lung • Decreased venous return • Mc cause penetrating chest trauma
  • 19. Clinical presentation • Tachypnea • Dyspnea • Distended neck veins • Tracheal deviation • Hyper resonance and decreased breath sounds
  • 20. TREATMENT • IMMEDIATE DECOMPRESSION • With large wide bore canula • Followed by insertion of chest tube in safety triangle
  • 22. INTRODUCTION • Results from blunt trauma associated with multiple rib fractures • Defined as three or more ribs fractured in two or more places. • blunt force typically also produces an underlying pulmonary contusion
  • 24. DIAGNOSIS • made clinically in patients who are not ventilated, • not by radiography • Confirmation: paradoxical motion of a chest wall segment
  • 25. • Voluntary splinting of the chest wall due to: • Pain • Mechanically impaired chest wall movement • Lung contusion HYPOXIA
  • 26. MANAGEMENT: • Traditionally, mechanical ventilation was used to ‘internally splint’ • Disadvantage: • intensive care unit (ICU) resources • ventilation-dependent morbidity
  • 27. • Currently: • oxygen administration • adequate analgesia (including opiates) • Physiotherapy • If chest tube is in place: topical intrapleural local analgesia • Ventilation (respiratory failure ) • Surgery : • stabilise the fail segment using internal fxation of the ribs • patients with • isolated or severe chest injury • pulmonary contusion.
  • 28. HAEMOTHORAX • BLUNT INJURY(m/c) • torn intercostal vessels • internal mammary artery secondary to fractures of the ribs • PENETRATING INJURY • Viscera: both thoracic and abdominal • blood leaking through a hole in the diaphragm from the positive pressure abdomen into the negative pressure thorax
  • 29. PRESENTATION : HAEMOTHORAX • haemorrhagic shock • fat neck veins • unilateral absence of breath sounds • dullness to percussion
  • 30. MANAGEMENT: • INITIAL TREATMENT: • correcting the hypovolaemic shock • insertion of an intercostal drain • Intubation • Indication for urgent thoracotomy • Initial drainage > 1500 mL of blood • ongoing haemorrhage > 200 mL/h over 3–4 hours
  • 31. • NOTE: There is no role for clamping a chest tube to tamponade a massive haemothorax. • IMPORTANT POINTS: • if the lung does not reinfLate -> the drain should be placed on low-pressure (5 cmH2O) suction • Check for clot occlusion of chest tube • Second drain is sometimes necessary • Role of chest radiograph or eFAST • physiotherapy and active mobilisation
  • 33. PERICARDIAL TAMPONADE • INTRODUCTION: • m/c due to penetrating trauma • All patients with penetrating injury anywhere near the heart + shock must be considered = cardiac injury until proven otherwise.
  • 34. MECHANISM: CARDIAC TAMPONADE • Accumulation of small amount(50ml) blood in non distensible pericardial sac • compression of the heart ; • obstruction of the venous return, • decreased filling of the cardiac chambers during diastole
  • 35. PRESENTATION • central venous pressure elevation, • a decline in arterial pressure • tachycardia • mufed heart sound • deteriorating cyanosis • agitation • earliest manifestation is an acute drop in mixed venous oxygen saturation
  • 36. DIAGNOSIS • eFAST • fuid in the pericardial sac • Most reliable • chest radiography • looking for an enlarged heart shadow
  • 37. MANAGEMENT: • NOTE: Pericardiocentesis has no role in the management of cardiac tamponade secondary to penetrating myocardial injury • Immediate treatment: • Operative: • subxiphoid window or • by open surgery (sternotomy or left anterolateral thoracotomy) • with evacuation of the haematoma and repair of the myocardium
  • 38.