etiology, local names, definition, transmission, source of infection, epidemiology, pathogenesis, clinical signs, diagnosis, differential diagnosis, treatment prevention and control
etiology, local names, definition, transmission, source of infection, epidemiology, pathogenesis, clinical signs, diagnosis, differential diagnosis, treatment prevention and control
Lecturer notes for metabolic diseases in Cattle.which is benificial for student of BVSc& AH/DVM and MVsc student. It is My first presentation need your feedback for more presentation like this.
Babesiosis is the diseased state caused by the protozoal (single celled) parasites of the genus Babesia. Infection in a dog may occur by tick transmission, direct transmission via blood transfer from dog bites, blood transfusions, or transplacental transmission.
Lecturer notes for metabolic diseases in Cattle.which is benificial for student of BVSc& AH/DVM and MVsc student. It is My first presentation need your feedback for more presentation like this.
Babesiosis is the diseased state caused by the protozoal (single celled) parasites of the genus Babesia. Infection in a dog may occur by tick transmission, direct transmission via blood transfer from dog bites, blood transfusions, or transplacental transmission.
It a bone marrow failure syndrome. Patients with severe Anaemia and neutropenia exhibiting a yellow hypercellular marrow on postmortem examination.
Features are. Pancytopenia, reticulocytopenia, bone marrow hypercellularity, heart murmur, pale-skin, gum, nail bus, epistaxis, mouth or throat infection, Anaemia etc. No gender differences based on the aetiology classified into two types Acquired and hereditary.
Ophthalmic eye care presentation, medical residency training, health care and malaria, Vision and malaria, malaria blindness, complications of malaria, ocular malaria
Anemia is the blood disorder, characterized by the reduction in:
1. Red blood cell (RBC) count
2. Hemoglobin content
3. Packed cell volume (PVC).
This occurs because of
Decreased production of RBC
Increased destruction of RBC
Excess loss of blood from the body
Hemolytic anemia occurs when the bone marrow is unable to increase production to make up for the premature destruction of red blood cells and the abnormal breakdown of red blood cells either in the blood vessels (intravascular hemolysis) or elsewhere in the body (extravascular). It has numerous possible causes, ranging from relatively harmless to life-threatening. The general classification of hemolytic anemia is either inherited or acquired. Treatment depends on the cause and nature of the breakdown.Symptoms of hemolytic anemia are similar to other forms of anemia (fatigue and shortness of breath), but in addition the breakdown of red cells leads to jaundice and increases the risk of particular long-term complications such as gallstones and pulmonary hypertension.
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Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Basavarajeeyam is an important text for ayurvedic physician belonging to andhra pradehs. It is a popular compendium in various parts of our country as well as in andhra pradesh. The content of the text was presented in sanskrit and telugu language (Bilingual). One of the most famous book in ayurvedic pharmaceutics and therapeutics. This book contains 25 chapters called as prakaranas. Many rasaoushadis were explained, pioneer of dhatu druti, nadi pareeksha, mutra pareeksha etc. Belongs to the period of 15-16 century. New diseases like upadamsha, phiranga rogas are explained.
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- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
2. Definition:-
Anemia is defined as the reduction in the quality and
or number of erythrocytes below normal for that
particular age, species, breed and geographic location.
3. Clinical signs
Depends on the degree of anemia, the duration and the
underlying cause.
In acute blood loss, the animal usually presents with
tachycardia, pale mucous membranes, bounding or weak
peripheral pulses and hypotension. If hemolysis is present,
the animal may be icteric. Animals with chronic anemia
have had time to accommodate, and their clinical
presentation is usually more indolent with vague signs of
lethargy, weakness, and anorexia.
Splenomegaly, abdominal distention, and/or heart
murmur may be present, depending on the underlying
cause of anemia.
5. ‘Anaemia’ can be conveniently grouped
as :-
Production low, but destruction normal-
i)Dyshemopoietic anemias
Production normal but destruction excessive -
i)Hemolytic anemias
ii) Hemorrhagic anemias
6. Dyshemopoietic anaemias
Anaemies in which there is defect in the formation of
erythrocytes
The defect may lie in the formation of stroma protein
or in the formation of Hb etc.
Porphyrinopathies : Porphyrins are necessary for the
normal synthesis of heme. If certain enzymes are
lacking, then heme is not synthesized.
7. A. Diminished stroma protein formation
Blood picture- macrocytic and normochromic or hypochromic.
Bone marrow is megaloblastic showing numerous megaloblasts
and giant metamyelocytes.
Dietetic deficiency of Extrinsic factor: Cobalt-cyanocobalamine
Dietetic deficiency of Folic acid
Deficiency of the intrinsic factor ( an enzyme secreted by gastric
mucosa)
Failure to store the Erythrocyte Maturation Factor
Failure to use the Erythrocyte Maturation Factor
Hypopituitarism
8. B. Diminished hemoglobin formation:
Blood picture- normocytic or microcytic and hypochromic.
Dietetic deficiency of iron: can occur due to
i) Deficient intake
ii) Detective absorption
Dietetic deficieney of copper
Dietetic defciency of ascorbic acid
Dietetic deficiency of pyrydoxine or nicotinic acid or
riboflavin
Deficiency of Thyroxine
9. C. Toxic inhibition
Blood picture- normochromic and microcytic
Chemical Poisons: Nitrogen mustard, Folic acidd
antagonists, Streptomycin, chloromycetin Metals like
Bismuth, Arsenic and Gold, Benzol, hair dyes, insecticides.
Chronic interstitial Nephritis
Oesophagastomiasis
chronic infections like Tuberculosis, Brucellosis and
Rheumatic fever
Ionising radiation
10. D. Aplastic anemia:
Blood picture- normochromic and normocytic
This occurs due to aplasia of bone marrow where there is
utter inactivity
Classification-
i) Primary or idiopathic : rather rare.
ii) Secondary
A) exhaaustion- chronic haemorrhages due to GI parasites,
neoplasm, vit C & K deficiency
B) Toxic – radiation, chemical poisoning
C) metabolic- in sows suffering from protein malnutrition
during pregnancy
11. E. Myelophthisic anemia:
There is replacement of bone marrow by other tissues.
Since in this disease immature forms of granulocytes are found
in the peripheral blood, it is also known as leuco-erythroblastic
anemia. This condition is found in-
1. Secondary metastasis of other tumors- lymphatic leukemia in
dog and cat.
2. Osteodystrophies- where the myeloid tissue is replaced by
connective tissue
3. Primary tumors of the reticulo-endothelial system- Nieman-
Pick Disease , Hodgkin’s Disease etc.
12. HEMOLYTIC ANEMIAS
In this condition intravascular destruction of erythrocytes
occurs.
Anemia is normochromic and macrocytic becoming
hypochromic and microcytic as the iron Stores are used up
Causes-
1) Abnormal auto-antibodies the presence of which may be
A. Primary or idiopathic. or
B. Secondary due to
i) Malignant disease-lymphatic neoplasms; ovarian tumors,
Gastro-intestinal carcinoma.
ii) Collagen diseases- disseminated lupus erythematosis.
iii) Viral disease - infectious mononucleosis
13. 2. Abnormal iso-antibodies: Due to the presence of
hemolysins in the plasma which is produced by
A) incompatible blood transfusion
B) injection of blood products
C) pregnancy- blood group antigens of the foetus pass to the
dam which does not possess these antigens.
3. Toxic
A) Chemicals:
i) Copper poisoning
ii) Onion poisoning
iii) Poisoning by castor seeds
iv) Phenothiazine poisoning
v) Naphthalene poisoning
vi) lead poisoning
vii) Hypersensitivity to certain drugs like sulphanilamide,
quinine etc
viii) snake venom
14. B) Post-parturient hemoglobinuria
C) infection
a) Protozoa- Anaplasmosis, Babesiosis, Ehrlichia canis.
b) Bacteria- Leptospira, Clostridia, Streptococci and
Staphylococci.
c)Viruses -Equine infectious anemia, feline infectious
anemia.
D. Hypersplenism
E. Cold hemoglobinuria in calves
15. HEMORRHAGIC ANEMIAS
In this condition extravascular destruction of
erythrocytes occurs.
The types are determined by
l. the amount of blood lost.
2. the rate at which the blood is lost
3. the diet controlling the balance between blood loss
and production
16. A. Acute haemorrhagic anaemia
Injury
sweet clover poisoning
Warfarin poisoning
bracken fern poisoning.
B. Chronic hemorrhagic anemia
blood sucking worms; Hemonchus, Fasciola ,
Bunostomum. in cattle and sheep, Strongyles in horses,
Ancylostomes in dogs.
Ectoparasites - ticks, lice and fleas
Protozoa- Coccidiosis in dogs
Hemorrhagic diseases- Chronic bovine hematuria
17. C. Purpura and hemorrahagic diseases
Purpura is accumulation of blood, under the skin due to
spontaneous rupture of the capillaries.
1. Vascular disorders ;
i. Purpuric infections : This is found in various diseases
characterised by petechial hemorrhages . eg. Hemorrhagic
septicemia, Anthrax etc. The Cause is injury to the capillaries
and venules by the toxins. In viremic diseases endothelium is
directly damaged due to the multiplication of the virus in the
endothelial cells eg. Infectious canine hepatitis and hog
cholera
ii. Allergic purpura or purpura hemorrhagica : -This is a
symptom of post-infectious toxemia as in Strangles, fistulous
withers, poll evil and emphysema of guttural pouches.
18. iii. Congenital purpura: Purpura may develop in the foetus.
The mechanism is suggested to be similar to the one found
in erythroblastosis foetalis. lso-agglminins formed
against platelets in the mother pass into the foetus via
placenta and produce thrombocytopenia.
iv. Senile purpura: in human beings
v. Vitamin C deficiency : Hemorrhages occur due to
increased capillary permeability and capillary
fragility since cement substance of capillary wall is not
synthesized. However this condition may not be met with
in animals since vitamin C is synthesized in their gut.
19. 2. Impaired Clotting mechanism:
a) Thrombocytopenia
b) haemophilia
c)prothrombin deficiency due to liver diseases,
Deficiency of Vit K, Impaired absorption of Vitamin K,
Poisoning by dieoumarin and Warfarin
d) Presence of Circulating anticoagulants
3) Mouldy corn poisoning in cattle and swine
4) Epistaxis in horse