Speech problems
Dysphasia
Overview:
• A deficit in the higher language functions i.e. comprehension and generation.
• Aphasia is a total absence.
• Most commonly due to a left anterior circulation stroke.
• If speech is internally consistent but nonsense, it is confusion not dysphasia.
Receptive (Wernicke's) dysphasia
• Temporal lobe lesion.
• Patient can't follow a command e.g. lift a hand. If the problem is only with a series of commands, the more they can manage then the better the prognosis.
1. Speech problems
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2. Dysphasia
Overview:
A deficit in the higher language functions i.e. comprehension and generation.
Aphasia is a total absence.
Most commonly due to a left anterior circulation stroke.
If speech is internally consistent but nonsense, it is confusion not dysphasia.
Receptive (Wernicke's) dysphasia
Temporal lobe lesion.
Patient can't follow a command e.g. lift a hand. If the problem is only with a series of commands,
the more they can manage then the better the prognosis.
Expressive (Broca's) dysphasia
Frontal lobe lesion.
Can't generate speech.
3. Nominal aphasia
Dominant posterior temporo-parietal lesion.
Difficulty in recalling specific words or names, but rest of speech normal.
Patient may not be able to name specific objects shown to them.
Conduction dysphasia
Longitudinal fasciculus lesion.
Patient can understand and speak, but can't repeat a phrase.
4. Dysarthria
Poor articulation.
Causes: bulbar or pseudobulbar palsy, or (less commonly) facial nerve palsy.
Examination: test tongue (say la la la) and palate (ka ka ka) for bulbar function,
and lips (ma ma ma) for facial nerve function.
Dysphonia
Reduced speech volume due to weak respiratory muscles or vocal cords.
Causes: (pseudo)bulbar palsy, myasthenia gravis, Parkinson's, recurrent laryngeal
nerve invasion.
5. Bulbar and pseudobulbar palsy
The 'bulb' refers to the medulla, and bulbar palsy is dysfunction of the
cranial nerves – 9 to 12 – whose nuclei lie within it.
It presents with dysphonia, dysarthria, and/or dysphagia.
Bulbar palsy
• LMN lesion of the medulla (nuclei) or cranial nerve fibres.
• Causes:
Brainstem stroke or tumour.
MND, especially progressive bulbar palsy.
Guillain Barré.
Myasthenia gravis.
Central pontine myelinolysis.
Iatrogenic: surgery, radiotherapy.
• Distinguishing features:
Fasciculating tongue which may sit in one side of the mouth.
6. Pseudobulbar palsy
• UMN lesion of the corticobulbar tract. Commoner than bulbar palsy.
• Causes:
Stroke e.g. of the bilateral internal capsule.
MS
Progressive supranuclear palsy
MND
Tumours higher in the brainstem.
Syphilis
• Distinguishing features:
Bilateral defects.
Paralysed tongue with donald duck speech.
Also non-bulbar symptoms – as corticobulbar tract supplies all motor
cranial nerves – including hyperreflexia (jaw jerk, gag) and facial paralysis.
Emotional lability.
7. Thank You
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