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Speech problems
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Dysphasia
Overview:
 A deficit in the higher language functions i.e. comprehension and generation.
 Aphasia is a total absence.
 Most commonly due to a left anterior circulation stroke.
 If speech is internally consistent but nonsense, it is confusion not dysphasia.
Receptive (Wernicke's) dysphasia
 Temporal lobe lesion.
 Patient can't follow a command e.g. lift a hand. If the problem is only with a series of commands,
the more they can manage then the better the prognosis.
Expressive (Broca's) dysphasia
 Frontal lobe lesion.
 Can't generate speech.
Nominal aphasia
 Dominant posterior temporo-parietal lesion.
 Difficulty in recalling specific words or names, but rest of speech normal.
 Patient may not be able to name specific objects shown to them.
Conduction dysphasia
 Longitudinal fasciculus lesion.
 Patient can understand and speak, but can't repeat a phrase.
Dysarthria
 Poor articulation.
 Causes: bulbar or pseudobulbar palsy, or (less commonly) facial nerve palsy.
 Examination: test tongue (say la la la) and palate (ka ka ka) for bulbar function,
and lips (ma ma ma) for facial nerve function.
Dysphonia
 Reduced speech volume due to weak respiratory muscles or vocal cords.
 Causes: (pseudo)bulbar palsy, myasthenia gravis, Parkinson's, recurrent laryngeal
nerve invasion.
Bulbar and pseudobulbar palsy
 The 'bulb' refers to the medulla, and bulbar palsy is dysfunction of the
cranial nerves – 9 to 12 – whose nuclei lie within it.
 It presents with dysphonia, dysarthria, and/or dysphagia.
Bulbar palsy
• LMN lesion of the medulla (nuclei) or cranial nerve fibres.
• Causes:
 Brainstem stroke or tumour.
 MND, especially progressive bulbar palsy.
 Guillain Barré.
 Myasthenia gravis.
 Central pontine myelinolysis.
 Iatrogenic: surgery, radiotherapy.
• Distinguishing features:
 Fasciculating tongue which may sit in one side of the mouth.
Pseudobulbar palsy
• UMN lesion of the corticobulbar tract. Commoner than bulbar palsy.
• Causes:
 Stroke e.g. of the bilateral internal capsule.
 MS
 Progressive supranuclear palsy
 MND
 Tumours higher in the brainstem.
 Syphilis
• Distinguishing features:
 Bilateral defects.
 Paralysed tongue with donald duck speech.
 Also non-bulbar symptoms – as corticobulbar tract supplies all motor
cranial nerves – including hyperreflexia (jaw jerk, gag) and facial paralysis.
 Emotional lability.
Thank You
• Information gathered with the assistance of Medicos PDF app. Happy to share this information with
Medical students. Find hundreds of books, slides, notes and articles from
https://bookapp.page.link/slideshare

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Speech problems

  • 1. Speech problems Slide made with the assistance of Medicos PDF app. Find hundreds of books, slides, notes and articles from: https://bookapp.page.link/slideshare
  • 2. Dysphasia Overview:  A deficit in the higher language functions i.e. comprehension and generation.  Aphasia is a total absence.  Most commonly due to a left anterior circulation stroke.  If speech is internally consistent but nonsense, it is confusion not dysphasia. Receptive (Wernicke's) dysphasia  Temporal lobe lesion.  Patient can't follow a command e.g. lift a hand. If the problem is only with a series of commands, the more they can manage then the better the prognosis. Expressive (Broca's) dysphasia  Frontal lobe lesion.  Can't generate speech.
  • 3. Nominal aphasia  Dominant posterior temporo-parietal lesion.  Difficulty in recalling specific words or names, but rest of speech normal.  Patient may not be able to name specific objects shown to them. Conduction dysphasia  Longitudinal fasciculus lesion.  Patient can understand and speak, but can't repeat a phrase.
  • 4. Dysarthria  Poor articulation.  Causes: bulbar or pseudobulbar palsy, or (less commonly) facial nerve palsy.  Examination: test tongue (say la la la) and palate (ka ka ka) for bulbar function, and lips (ma ma ma) for facial nerve function. Dysphonia  Reduced speech volume due to weak respiratory muscles or vocal cords.  Causes: (pseudo)bulbar palsy, myasthenia gravis, Parkinson's, recurrent laryngeal nerve invasion.
  • 5. Bulbar and pseudobulbar palsy  The 'bulb' refers to the medulla, and bulbar palsy is dysfunction of the cranial nerves – 9 to 12 – whose nuclei lie within it.  It presents with dysphonia, dysarthria, and/or dysphagia. Bulbar palsy • LMN lesion of the medulla (nuclei) or cranial nerve fibres. • Causes:  Brainstem stroke or tumour.  MND, especially progressive bulbar palsy.  Guillain Barré.  Myasthenia gravis.  Central pontine myelinolysis.  Iatrogenic: surgery, radiotherapy. • Distinguishing features:  Fasciculating tongue which may sit in one side of the mouth.
  • 6. Pseudobulbar palsy • UMN lesion of the corticobulbar tract. Commoner than bulbar palsy. • Causes:  Stroke e.g. of the bilateral internal capsule.  MS  Progressive supranuclear palsy  MND  Tumours higher in the brainstem.  Syphilis • Distinguishing features:  Bilateral defects.  Paralysed tongue with donald duck speech.  Also non-bulbar symptoms – as corticobulbar tract supplies all motor cranial nerves – including hyperreflexia (jaw jerk, gag) and facial paralysis.  Emotional lability.
  • 7. Thank You • Information gathered with the assistance of Medicos PDF app. Happy to share this information with Medical students. Find hundreds of books, slides, notes and articles from https://bookapp.page.link/slideshare