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 Tetralogy Of Fallot is a congenital heart defect which
is classicaly understood to involve four anatomical
abnormalitiesof the heart (although only three of
them are always present) it is the most common
cyanotic heart defect,and its also known as blue baby
syndrome
 it was described in 1888 by the french physician
etienne-louis arthur fallot, after whom it is named.
INVOLVES FOUR HEART DEFECTS
 PULMONARY STENOSIS
 AN OVERRIDING AORTA
 A LARGE VENTRICULAR SEPTAL DEFECT(VSD)
 RIGHT VENTRICULAR HYPERTROPHY
 GERMAN MEASLES(Rubella)& Some other
VIRAL ILLNESS.
 POOR NUTRITION
 ALCOHOL USE
 AGE(being older than 40)
 DIABETES
 HERIDITY
 CHILDREN WITH GENETIC DISORDERS (Down
syndrome, DiGeorge Syndrome)
 BLUE COLOUR TO THE SKIN(CYANOSIS)
 CLUBBING OF FINGERS
 DIFFICULTY FEEDING(POOR FEEDING HABIT)
 DYSPNEA ON EXERTION
 POLYCYTHEMIA
 HEART MURMUR
A TET spell occurs when the
oxygen level in the blood suddenly drops.
This causes the baby to become very blue.
He or she also may have trouble
breathing, become very tired and limp,not
respond to a parents voice or
touch,become very fussy,or pass out.
 It occurs while baby awakening from sleep
after crying or during
 During or after defecation
 or during or immediately following feeding
SQUATTING
Obstruction of Femoral Arteries
Increased Peripheral vascular resistance
Increased LV After load
Increased LV Pressure
Decreased Right ventricular over Left
Ventricular pressure gradient
.
Decreased shunting of blood from right ventricle to left
ventricle (R to L shunt) through the VSD
Improved flow to the pulmonary artery
Better alveolar perfusion
Better oxygenation
Decreased symptoms of TOF spell
 HAVE A HARD TIME BREATHING
 BECOME VERY TIRED AND LIMP
 NOT RESPONDING TO A PARENTS VOICE OR
TOUCH
 BECOME VERY FUSSY
 LOSS OF CONSCIOUSNESS
 PHYSICAL EXAMINATION
 ECHOCARDIOGRAPHY
 EKG(Electro cardiogram)
 CHEST X RAY
 PULSE OXIMETRY
 CARDIAC CATHETERIZATION
 BLALOCK-TAUSSING SHUNT:-
Anastamosis between subclavian
artery and pulmonary artery.
 POTT’S PROCEDURE:-
Side to side anastamosis of left
pulmonary artery to descending aorta.
 WATERSON-COOLEY PROCEDURE:-
Anastamosis of right pulmonary
artery to descending aorta.
 Pulmonary artery atresia
 Major associated anomalies
 Multiple previous surgeries
 Absent pulmonary valve syndrome
 Young or Old age
 Small pulmonary arteries
 Multiple VSDs
 Low birth weight
 Coexisting cardiac anomalies
 Closure of the ventricular septal defect in a manner
that ensures left ventricular aortic continuity by
using the VSD patches.
 Infundibular muscle resection
 Pulmonic valve is opened by using Hegars dilator.
 NURSING DIAGNOSIS:DECREASED CARDIAC OUTPUT
 ↓ BP
 Tachycardia
 Jugular venous distention(jvd)
 Sudden stoppage of drainage from chest tubes
 ↓ in HB and hematocrit
 Cool,clammy skin
 ↓ urine output
NURSING INTERVENSIONS RATIONALE
LOW CARDIAC OUTPUT Inflammatory response associated with
CPB, Myocardial ischemia From aortic
clamping or circulatory arrest.
Assess Peripheral and central pulses Good peripheral pulses and adequate
capillary refill are signs of good CO
Assess for mental status changes Early signs of cerebral hypoxia are
restlessness with confusion and loss of
consciousness later stages
Assess respiratory rate, rhythm,and
breath sounds
Rapid shallow respirations and
presence of wheezes are characteristic
of decreased output.
 COMMON RISK FACTORS:
 Intubation during surgery
 extubation
 Mechanical ventilation
 Perioperative CPB time
 Ease of intubation
NURSING INTERVENTIONS RATIONALE
Assess respiratory rate,rhythm,and
depth every hour
Rapid, shallow respirations may occur
from hypoxia or from acidosis
Assess for any increasing in work of
breathing
After extubation patients may
experience acute respiratory distress
syndrome.
Assess pulse oximetry and ABGs O2 saturation should be kept at 90%
or greater
Change position every 2 hours This facilitates movement and
drainage of secretions
NURSING INTERVENTIONS RATIONALE
Observe and documents serial
laboratory data: na,K+,CL,MG,CA+
Levels
Hemodilution from ECC and resultant
fluid shifts changes in fluid composition
Monitor ECG for changes Widening QRS,ST
changes,arrhythmias,and
atrioventricular blocks are seen with
electrolyte imbalance
Monitor for hyperglycemia Tight glycemic control significantly
reduces the incidence of morbidity and
mortality
 COMMON RELATED FACTORS:
 Intensive care unit environment
 Unfamiliarity with postoperative care
 Threat of pain related to major surgery
 Threat of death
NURSING INTERVENTIONS RATIONALE
Recognize patients level of fear Controlling fear helps reduce
physiology reaction that can aggravate
condition and ↑ o2 consumption
Approach the child and parent in a
confident and composed manner
This approach increases the patient’s
feeling security
Avoid unnecessary conversations
between team members in front of
patients
This reduces patients misconceptions
and fear or anxiety
Tof

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Tof

  • 1.
  • 2.
  • 3.  Tetralogy Of Fallot is a congenital heart defect which is classicaly understood to involve four anatomical abnormalitiesof the heart (although only three of them are always present) it is the most common cyanotic heart defect,and its also known as blue baby syndrome  it was described in 1888 by the french physician etienne-louis arthur fallot, after whom it is named.
  • 4. INVOLVES FOUR HEART DEFECTS  PULMONARY STENOSIS  AN OVERRIDING AORTA  A LARGE VENTRICULAR SEPTAL DEFECT(VSD)  RIGHT VENTRICULAR HYPERTROPHY
  • 5.
  • 6.
  • 7.  GERMAN MEASLES(Rubella)& Some other VIRAL ILLNESS.  POOR NUTRITION  ALCOHOL USE  AGE(being older than 40)  DIABETES  HERIDITY  CHILDREN WITH GENETIC DISORDERS (Down syndrome, DiGeorge Syndrome)
  • 8.  BLUE COLOUR TO THE SKIN(CYANOSIS)  CLUBBING OF FINGERS  DIFFICULTY FEEDING(POOR FEEDING HABIT)  DYSPNEA ON EXERTION  POLYCYTHEMIA  HEART MURMUR
  • 9.
  • 10.
  • 11. A TET spell occurs when the oxygen level in the blood suddenly drops. This causes the baby to become very blue. He or she also may have trouble breathing, become very tired and limp,not respond to a parents voice or touch,become very fussy,or pass out.
  • 12.  It occurs while baby awakening from sleep after crying or during  During or after defecation  or during or immediately following feeding
  • 13.
  • 14.
  • 15. SQUATTING Obstruction of Femoral Arteries Increased Peripheral vascular resistance Increased LV After load Increased LV Pressure Decreased Right ventricular over Left Ventricular pressure gradient .
  • 16. Decreased shunting of blood from right ventricle to left ventricle (R to L shunt) through the VSD Improved flow to the pulmonary artery Better alveolar perfusion Better oxygenation Decreased symptoms of TOF spell
  • 17.  HAVE A HARD TIME BREATHING  BECOME VERY TIRED AND LIMP  NOT RESPONDING TO A PARENTS VOICE OR TOUCH  BECOME VERY FUSSY  LOSS OF CONSCIOUSNESS
  • 18.  PHYSICAL EXAMINATION  ECHOCARDIOGRAPHY  EKG(Electro cardiogram)  CHEST X RAY  PULSE OXIMETRY  CARDIAC CATHETERIZATION
  • 19.
  • 20.
  • 21.  BLALOCK-TAUSSING SHUNT:- Anastamosis between subclavian artery and pulmonary artery.  POTT’S PROCEDURE:- Side to side anastamosis of left pulmonary artery to descending aorta.  WATERSON-COOLEY PROCEDURE:- Anastamosis of right pulmonary artery to descending aorta.
  • 22.  Pulmonary artery atresia  Major associated anomalies  Multiple previous surgeries  Absent pulmonary valve syndrome  Young or Old age  Small pulmonary arteries  Multiple VSDs  Low birth weight  Coexisting cardiac anomalies
  • 23.
  • 24.  Closure of the ventricular septal defect in a manner that ensures left ventricular aortic continuity by using the VSD patches.  Infundibular muscle resection  Pulmonic valve is opened by using Hegars dilator.
  • 25.
  • 26.  NURSING DIAGNOSIS:DECREASED CARDIAC OUTPUT  ↓ BP  Tachycardia  Jugular venous distention(jvd)  Sudden stoppage of drainage from chest tubes  ↓ in HB and hematocrit  Cool,clammy skin  ↓ urine output
  • 27. NURSING INTERVENSIONS RATIONALE LOW CARDIAC OUTPUT Inflammatory response associated with CPB, Myocardial ischemia From aortic clamping or circulatory arrest. Assess Peripheral and central pulses Good peripheral pulses and adequate capillary refill are signs of good CO Assess for mental status changes Early signs of cerebral hypoxia are restlessness with confusion and loss of consciousness later stages Assess respiratory rate, rhythm,and breath sounds Rapid shallow respirations and presence of wheezes are characteristic of decreased output.
  • 28.  COMMON RISK FACTORS:  Intubation during surgery  extubation  Mechanical ventilation  Perioperative CPB time  Ease of intubation
  • 29. NURSING INTERVENTIONS RATIONALE Assess respiratory rate,rhythm,and depth every hour Rapid, shallow respirations may occur from hypoxia or from acidosis Assess for any increasing in work of breathing After extubation patients may experience acute respiratory distress syndrome. Assess pulse oximetry and ABGs O2 saturation should be kept at 90% or greater Change position every 2 hours This facilitates movement and drainage of secretions
  • 30. NURSING INTERVENTIONS RATIONALE Observe and documents serial laboratory data: na,K+,CL,MG,CA+ Levels Hemodilution from ECC and resultant fluid shifts changes in fluid composition Monitor ECG for changes Widening QRS,ST changes,arrhythmias,and atrioventricular blocks are seen with electrolyte imbalance Monitor for hyperglycemia Tight glycemic control significantly reduces the incidence of morbidity and mortality
  • 31.  COMMON RELATED FACTORS:  Intensive care unit environment  Unfamiliarity with postoperative care  Threat of pain related to major surgery  Threat of death
  • 32. NURSING INTERVENTIONS RATIONALE Recognize patients level of fear Controlling fear helps reduce physiology reaction that can aggravate condition and ↑ o2 consumption Approach the child and parent in a confident and composed manner This approach increases the patient’s feeling security Avoid unnecessary conversations between team members in front of patients This reduces patients misconceptions and fear or anxiety

Editor's Notes

  1. Nursing Care Plan for Tetralogy of Fallot Tetralogy of FallotDefinitionTetralogy of Fallot is a congenital heart disease with cyanosis, a combination of the four main symptoms are:obstruction of the flow out of the right ventricle (pulmonary stenosis), ventricular septal defect, the position of the right of the aorta and right ventricular hypertrophy together form a tetralogy of Fallot. Clinical manifestationscyanosis dyspnoea dyspnoea attacks paroksimal (blue anoxia attacks) delay in growth and development normal rate of blood vessels systolic murmur Assessment - Nursing Care Plan for Tetralogy of FallotData that is commonly found in patients with tetralogy of Fallot are:thorough cyanosis of mucous membranes or lips, tongue, conjunctiva. Cyanosis also occur at the time of crying, eating, tight, soak in water, can be peripheral or central. dyspnoea usually accompanies the activity of eating, crying or tension / stress. weakness, commonly in the legs. growth and development not in accordance with age. digital clubbing headache epistaxis Nursing Diagnosis for Tetralogy of FallotRisk for Decreased cardiac output related to structural abnormalities of the heart. Activity Intolerance related to imbalance in the fulfillment of oxygen to the body's needs. Impaired growth and development related to inadequate oxygenation, tissue nutrisis needs, social isolation. Risk for infection related to the general conditions is inadequate.