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TOPIC :- SHOCK
MR . ROMAN BAJRANG
BASIC BS.C NURSING 2ND YEAR
Reliance institute ofnursing
INTRODUCTION
 Shock is a failure of cardio vascular system to
deliver enough oxygen & nutrients to meet
cellular metabolic needs .Shock may develop
rapidly or slowly all the system of the body
included in this in shock condition the body will
struggle for survive then the haemostatic
mechanism ( coagulation or blood clotting )will
occur it provide blood circulation. It is a remedy
to stop hemorrhage if shock is due to
hemorrhage
DEFINITION
Shock is defined as a complex life
threatening condition characterized by
inadequate blood flow to the tissue & cells of
the body
INCIDENCE
 HYPOVOLUMIC
Affects all ages
Affects males & females equally
 CARDIOGENIC SHOCK
Typically affects patient with are of infraction
involving 40% or more left ventricular muscle
mass
 SEPTICSHOCK
Possible in any person with impaired immunity
TYPES
 Hypovolemic (inadequate circulatory
contractility)
 Cardiogenic (decrease myocardial
contractility)
 Obstructive or (inadequate circulatory blood
flow caused by a physical impairment or
obstructive)
 Distribute [decrease vascular resistance ]
* Anaphylacticshock
*Septic shock
*Neurogenic shock
HYPOVOLUMIC SHOCK
DEFINITION
 It is a most common shock characterized by the
decreased intra vascular volume
ETIOLOGY
 EXTERNAL FLUID LOSS
Surgery
vomiting
diarrhea
excessive urination
 INTERNAL FLUIDSHIFTING
Hemorrhage
burns
ascitis
edema
PATHOPHYSIOLOGY
Decreased blood volume
Decreased venous return
Decreased stroke volume
Decreased cardiac output
Decreased B.P
Decreased tissue perfusion
CLINICAL MANIFESTATION
 RAPID PULSE
 TACHYPNEA
 COOL & CLAMMYSKIN
 CYANOSIS
MEDICAL MANAGEMENT
 Restore intravascular
volume
 Re -distribute fluid volume
 Correct underlined causes
NURSING MANAGEMENT
 Safe administration of the prescribed fluid
 Right documentation
 Administering blood transfusion
 Monitor the hemodynamic pressure, vital
sign, arterial blood gas
 Monitor cardiac & respiratory status closely
 Administer oxygen
CARDIOGENICSHOCK
DEFINITION
 It occurs due to the impaired heart ability to
contract & ejection of blood
 ETIOLOGY
 CORONARY CAUSE
Myocardial infraction
 NON CORONARYCAUSE
Cardiomyopathy
vulvar damage
cardiac temponade
PATHOPHYSIOLOGY
Decreased cardiac contractility
Decreased stroke volume
Decreased cardiac output
Pulmonary decreased systemic decreased
Congestion tissue perfusion coronary
artery
perfusion
CLINICAL MANIFESTATION
 Angina pain
 Decreased systolic B.P
 Mental confusion
 Fatigue
 Pulmonary congestion
 Decreased urine out put
 Hemodynamic instability
MEDICAL MANAGEMENT
 Correction of underlined causes
 Initiation of first line treatment
 Oxygenation
 Pain control
 Hemodynamic monitoring
PHARMACHOLOGICAL THERAPY
 DOBUTAMINE
 NITROGLYCERINE
 DOPAMINE
 OTHERVASOACTIVEMEDICATION
 ANTIARRYTHEMICMEDICATION
NURSING MANAGEMENT
 Monitoring hemodynamic status
 Administering medication & Intravenous
therapy
 Enhancing safety & comfort
COMPLICATION
 NEUROLOGIC IMPAIRMENT
 ACUTE RESPIRATORY DISSTRESS
SYNDROME
 RENAL FAILURE
CIRCULATORYSHOCK
Shock is a life-threatening
medical condition of low blood
perfusion to tissues resulting
in cellular injury and
inadequate tissue function
SEPTIC SHOCK
 DEFINITION: Severe bacterial infectionor
septicemia induces septic shock
 ETIOLOGY
Infection
gram negative bacteria causinginfection
staphylococcal infection
U.T.I
indwelling lines & catheter (inserted into
bladder )
respiratory infection
DISEASE: D.M ,AIDS
PATHOPHYSIOLOGY
Vasodilation
Mal distribution of blood volume
Decreased venous return
Decreased stroke volume (the volume of
blood pumped from the left ventricle per beat ( 70 ml ) 70kg
Decreased cardiac out put
Decreased tissueperfusion
CLINICAL MANIFESTATION
 FIRST STAGE
 Patient is febrile
 Bounding pulse ( a strong throbbing increase lub dub )felt
over one of the arteries in the body . It is due to a forceful
heartbeat .
 Flushed skin
 Tachycardia
 Decreased urine out put
 Decreased bowel sound
 SECOND STAGE
 Low B.P
 Skin cool & pale
 Temperature normal or below normal
MEDICAL MANAGEMENT
 Identify the cause & eliminate the cause
CULTURE
o Blood specimen
o Urine specimen
o Drainage of wound
o Remove old I.V line & put new I.V line
o Antibiotic coated I.Vline should be established
o Abcess are drained
o Fluid replacement
NEUROGENICSHOCK
DEFINITION
 It cause vasodilation due to loss of
sympathetic tone ( damage to the
central nervous system
 ETIOLOGY
 Spinal cord injuries
 Nervous tissue damage
 Depressant action of nervous system
 Lack of glucose
CLINICAL MANIFESTATION
Dry ,warm skin rather than cool Moist
skin
Bradycardia rather than the
tachycardia
ANAPHYLACTI
C SHOCK
DEFINITION
It occurs in severe allergic reaction
ETIOLOGY
 Penicillin sensitivity
 Transfusion reaction
 Vasodilation
 Capillary permeability( the capacity of a blood
vessel wall to allow for the flow of small
molecules ( drug, nutrient ,water,iron) or even
whole cells ( lymphocytes on their way to the
site of inflammation)
CLINICAL MANIFESTATION
 Mild
peripheral tingling, sensation of warm ,
fullness of mouth & throat , nasal
congestion
 Moderate
flushing (red in mouth ), warmth, anxiety
& itching
 Severe
bronchospasm, dyspnea, cough,
cyanosis
MEDICAL MANAGEMENT
 Identify the cause & solve it
 Decrease vasodilatation
 Administer medication
epinephrine
diphenhydramine
nebulization
albuterol
NURSING MANAGEMENT
Assess the patient for any previous allergy
to medicine, fruits etc
When we administer any new medicine we
have to observe the side effect of it
We must have the knowledge about the
sign symptoms of shock
Observe the response to the treatment
ASSESMENT&DIAGNOSTIC PROCEDURE
NON INVASIVEPROCEDURE
-spirometry
-pulse oximeter
-arterial blood gas analysis
C.V.P MEASUREMENT
E.C.G
CHESTX-RAY
HEAMODYNAMIC MONITORING
THANK

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SHOCK 12

  • 1. TOPIC :- SHOCK MR . ROMAN BAJRANG BASIC BS.C NURSING 2ND YEAR Reliance institute ofnursing
  • 2.
  • 3. INTRODUCTION  Shock is a failure of cardio vascular system to deliver enough oxygen & nutrients to meet cellular metabolic needs .Shock may develop rapidly or slowly all the system of the body included in this in shock condition the body will struggle for survive then the haemostatic mechanism ( coagulation or blood clotting )will occur it provide blood circulation. It is a remedy to stop hemorrhage if shock is due to hemorrhage
  • 4. DEFINITION Shock is defined as a complex life threatening condition characterized by inadequate blood flow to the tissue & cells of the body
  • 5. INCIDENCE  HYPOVOLUMIC Affects all ages Affects males & females equally  CARDIOGENIC SHOCK Typically affects patient with are of infraction involving 40% or more left ventricular muscle mass  SEPTICSHOCK Possible in any person with impaired immunity
  • 6. TYPES  Hypovolemic (inadequate circulatory contractility)  Cardiogenic (decrease myocardial contractility)  Obstructive or (inadequate circulatory blood flow caused by a physical impairment or obstructive)  Distribute [decrease vascular resistance ] * Anaphylacticshock *Septic shock *Neurogenic shock
  • 7.
  • 9. DEFINITION  It is a most common shock characterized by the decreased intra vascular volume ETIOLOGY  EXTERNAL FLUID LOSS Surgery vomiting diarrhea excessive urination  INTERNAL FLUIDSHIFTING Hemorrhage burns ascitis edema
  • 10. PATHOPHYSIOLOGY Decreased blood volume Decreased venous return Decreased stroke volume Decreased cardiac output Decreased B.P Decreased tissue perfusion
  • 11. CLINICAL MANIFESTATION  RAPID PULSE  TACHYPNEA  COOL & CLAMMYSKIN  CYANOSIS
  • 12. MEDICAL MANAGEMENT  Restore intravascular volume  Re -distribute fluid volume  Correct underlined causes
  • 13. NURSING MANAGEMENT  Safe administration of the prescribed fluid  Right documentation  Administering blood transfusion  Monitor the hemodynamic pressure, vital sign, arterial blood gas  Monitor cardiac & respiratory status closely  Administer oxygen
  • 14.
  • 16. DEFINITION  It occurs due to the impaired heart ability to contract & ejection of blood  ETIOLOGY  CORONARY CAUSE Myocardial infraction  NON CORONARYCAUSE Cardiomyopathy vulvar damage cardiac temponade
  • 17. PATHOPHYSIOLOGY Decreased cardiac contractility Decreased stroke volume Decreased cardiac output Pulmonary decreased systemic decreased Congestion tissue perfusion coronary artery perfusion
  • 18. CLINICAL MANIFESTATION  Angina pain  Decreased systolic B.P  Mental confusion  Fatigue  Pulmonary congestion  Decreased urine out put  Hemodynamic instability
  • 19. MEDICAL MANAGEMENT  Correction of underlined causes  Initiation of first line treatment  Oxygenation  Pain control  Hemodynamic monitoring
  • 20. PHARMACHOLOGICAL THERAPY  DOBUTAMINE  NITROGLYCERINE  DOPAMINE  OTHERVASOACTIVEMEDICATION  ANTIARRYTHEMICMEDICATION
  • 21. NURSING MANAGEMENT  Monitoring hemodynamic status  Administering medication & Intravenous therapy  Enhancing safety & comfort
  • 22. COMPLICATION  NEUROLOGIC IMPAIRMENT  ACUTE RESPIRATORY DISSTRESS SYNDROME  RENAL FAILURE
  • 23.
  • 25. Shock is a life-threatening medical condition of low blood perfusion to tissues resulting in cellular injury and inadequate tissue function
  • 26.
  • 27.
  • 28.
  • 29.
  • 30. SEPTIC SHOCK  DEFINITION: Severe bacterial infectionor septicemia induces septic shock  ETIOLOGY Infection gram negative bacteria causinginfection staphylococcal infection U.T.I indwelling lines & catheter (inserted into bladder ) respiratory infection DISEASE: D.M ,AIDS
  • 31. PATHOPHYSIOLOGY Vasodilation Mal distribution of blood volume Decreased venous return Decreased stroke volume (the volume of blood pumped from the left ventricle per beat ( 70 ml ) 70kg Decreased cardiac out put Decreased tissueperfusion
  • 32. CLINICAL MANIFESTATION  FIRST STAGE  Patient is febrile  Bounding pulse ( a strong throbbing increase lub dub )felt over one of the arteries in the body . It is due to a forceful heartbeat .  Flushed skin  Tachycardia  Decreased urine out put  Decreased bowel sound  SECOND STAGE  Low B.P  Skin cool & pale  Temperature normal or below normal
  • 33. MEDICAL MANAGEMENT  Identify the cause & eliminate the cause CULTURE o Blood specimen o Urine specimen o Drainage of wound o Remove old I.V line & put new I.V line o Antibiotic coated I.Vline should be established o Abcess are drained o Fluid replacement
  • 35. DEFINITION  It cause vasodilation due to loss of sympathetic tone ( damage to the central nervous system  ETIOLOGY  Spinal cord injuries  Nervous tissue damage  Depressant action of nervous system  Lack of glucose
  • 36. CLINICAL MANIFESTATION Dry ,warm skin rather than cool Moist skin Bradycardia rather than the tachycardia
  • 38. DEFINITION It occurs in severe allergic reaction ETIOLOGY  Penicillin sensitivity  Transfusion reaction  Vasodilation  Capillary permeability( the capacity of a blood vessel wall to allow for the flow of small molecules ( drug, nutrient ,water,iron) or even whole cells ( lymphocytes on their way to the site of inflammation)
  • 39. CLINICAL MANIFESTATION  Mild peripheral tingling, sensation of warm , fullness of mouth & throat , nasal congestion  Moderate flushing (red in mouth ), warmth, anxiety & itching  Severe bronchospasm, dyspnea, cough, cyanosis
  • 40. MEDICAL MANAGEMENT  Identify the cause & solve it  Decrease vasodilatation  Administer medication epinephrine diphenhydramine nebulization albuterol
  • 41. NURSING MANAGEMENT Assess the patient for any previous allergy to medicine, fruits etc When we administer any new medicine we have to observe the side effect of it We must have the knowledge about the sign symptoms of shock Observe the response to the treatment
  • 42. ASSESMENT&DIAGNOSTIC PROCEDURE NON INVASIVEPROCEDURE -spirometry -pulse oximeter -arterial blood gas analysis C.V.P MEASUREMENT E.C.G CHESTX-RAY HEAMODYNAMIC MONITORING
  • 43. THANK