This document discusses different types of retinal detachment. Rhegmatogenous retinal detachment is the most common type and occurs when there is a break or tear in the retina that allows fluid from the vitreous to enter the subretinal space. Tractional retinal detachment occurs when the retina is pulled away from the retinal pigment epithelium due to contraction of fibrous tissue in the vitreous. Exudative retinal detachment occurs when fluid accumulates beneath the retinal pigment epithelium due to inflammation or vascular issues. The document outlines causes, symptoms, examinations, and treatments for each type of retinal detachment.
Enucleation and evisceration. ophthalmology
contains the two methods of eye ball removal, with indications, surgery, complications, treatment, etc
its has animated clips and picture
wonderful slide i have prepaired
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Enucleation and evisceration. ophthalmology
contains the two methods of eye ball removal, with indications, surgery, complications, treatment, etc
its has animated clips and picture
wonderful slide i have prepaired
can be used for clinical as well as educational purpose
Congenital pit is an atypical coloboma usually located on the temporal edge of the disc, associated with irregular defects in the juxtapapillary choroid and pigment epithelium. Macular fibers passing through this area often are affected and corresponding changes in the retinal ganglion cell layer and in the visual field occur.
DR WANI'S TALK ON RETINAL DETACHMENT LECTURE FOR RESIDENTS [DR WANI TALK.pptxvbwani
Dr Wani talks on RD for residents in KLE hospital
This is a detailed talk that deals with all aspects fo RH RD
This talk aims to clear the concepts about RD
It deals the incidence of RH Rd, pathogenesis, cliinical features , diagnosis , treatment options prognosis etc
complete information about the retinal detachment , types, , symptoms , sign, etiology, causes, diagnosis, complications, medical management, nursing management, home care, patient teaching. nursing reserch.
Leukocoria ( or white pupillary reflex) is an abnormal white reflection from the eye.
Leukocoria is a medical sign for a number of several conditions.
- this presentation at annual conference of the Ophthalmic department, faculty of medicine - Al-Azhar University in association with DOS & EOS Cairo, Egypt January 2017
This slide contains information regarding disorders of retina. This can be helpful for proficiency level and bachelor level nursing students. Your feedback is highly appreciated.
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This presentation gives basic information related to handwashing. Handwashing is the most effective method to prevent the majority of the illnesses we encounter on a regular basis.
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Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
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Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
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Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
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Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
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Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
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Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
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New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
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The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
4. Retinal Detachment is the separation of the
neurosensory retina from the pigmented epithelium.
• Even normally potential space between the layers
• Thus, a misnomer------”Retinal Separation”
11. Ret i nal
Det achment
(Commonest variant)
occurs 20
to a full-thickness defect in the sensory retina,
which permits fluid derived from synchytic (liquefied)
vitreous to gain access to the subretinal space.
i.e. associated with retinal breaks (tear or hole) through
which sub-retinal fluid seeps into and separates the
sensory retina from the pigment epithelium
Etiology still not clear
12. Predisposing Factors:
Age: common in 40-60 yrs, however age is no bar
Sex: M>F
Myopia:
Aphakia and pseudophakia:
Retinal degenerations:
Trauma:
Senile posterior vitreous detachment:
13. Pathogenesis:
Senile acute PVD Predisposing Retinal Degenerations Aphakia
Retinal Breaks
Trauma
Degenerated vitreous fluid
seeps through the break and collects as Sub-retinal fluid (SRF)
between the sensory and pigment epithelium
Rhegmatogenous Retinal Detachment
Lincoff’s Rule
14. Prodromal Symptoms:
Floaters (d/t rapid vitreous degeneration)
Photopsia (d/t irritation of retina by vitreous movement)
Symptoms of detached retina:
Localised relative loss in visual field (black curtain)
Sudden appeareance of dark clouds in front of eye
Sudden painless loss of vision
(when detachment is large and central)
C/F:
15. Symptoms of detached retina:
External examination: normal eye
IOP: slightly lower or normal
Marcus Gunn Pupil: in extensive RD
Plane mirror examination: greyish reflex in the quadrant of
detached retina
Ophthalmoscopy: freshly detached retina retinal vessels appears
dark tortuous cords that moves with the
detached retina
Retinal breaks: commonest in upper temporal quadrant
Shaffer sign: vitreous pigment in anterior vitreous
Old retinal detachments
Electroretinography: sub-normal of absent
USG: confirmation of diagnosis
21. Drainage of SRF
• By fine needle insertion through sclera and choroid into sub-
retinal space
• Allows immediate apposition between the sensory and
pigmentary retina
22. Maintainence of
chorioretinal apposition
At least for couple of weeks
Many techniques:
Scleral buckling (external tamponade)
Pneumatic retinopexy
Pars plana viterectomy
Endolaser photocoagulation
Internal tamponade
29. Sol i d Ret i nal
Det achment
(aka Serous RD)Occurs due to retina being mechanically pushed
away by a neoplasm or accumulation of fluid
beneath the retinal pigment epithelium
caused neither by a break nor traction; the SRF is derived
from fluid in the vessels of the NSR or choroid, or both.
Usually associated with inflammatory or vascular lesion
32. C/F:
• NO photopsia, retinal breaks, folds or undulations
• visual field defect may develop suddenly and progress rapidly
• Retina: smooth and convex
may show pigmentary disturbances
• Disturbed pattern of retinal vessels
• Detached retina is very mobile and demonstrate Shifting Fluid
(gravity)
• Transillumination test:
» Transparent or solid
38. • The NSR is pulled away from the RPE by contracting
vitreoretinal membranes in the absence of a retinal break.
• Here retina being mechanically pulled away from its bed by the
contraction of the fibrous tissue in the vitrous (vitreo-retinal
traction band)
Tr act i onal Ret i nal Det achment
39. Etiology:
• Post-traumatic (penetration trauma)
» Contraction of the scar tissue
• Proliferative diabetic retinopathy
• Post hemorrhagic retinitis proliferans
• ROP
• Retinal dysplasia
• Proliferative retinopathy of Eale’s disease
• Toxocariasis
40.
41. C/F:
NO photopsia and floaters
Retinal breaks are usually absent
No shifting of fluid
Detached retina : Concave
Presence of vitreoretinal traction bands
Retinal mobility is severly reduced
The retinal breaks responsible for RD are caused by interplay between dynamic vitreoretinal traction and an underlying weakness in the peripheral retina referred to as predisposing degeneration.