Rapid response systems (RRSs) have become a routine part of the way patients are managed in general wards of acute care hospitals. They have been adopted by national health and safety organisations in North America, Canada, the United Kingdom and Australia and are increasingly being used in other parts of the world.
Studies have almost universally shown significant reductions in outcome indicators such as mortality (up to one third) and cardiac arrest rates (up to 50%). However the validity of these outcomes is questionable as most of these studies are single-centre, before-and-after studies conducted by one or two clinical champions in Rapid Response.
This presentation reveals that the implementation of an Intensivist led Rapid Response Team in an Australian quaternary hospital did not demonstrate such dramatic results. In fact, after one year of service the standardised mortality ratio and the in-hospital cardiac arrest rate remained similar.
The presentation explores some of the operational impacts of a RRS including the replacement of critical thinking with reliance on protocols and the progressive super-specialisation of medical teams. Despite these impacts and relatively static patient outcome data, the service has rapidly become an integral part of the hospital.
Barriers between Intensive Care and ward staff have broken down and quality outcome results have consistently shown ward nurses and doctors feel better prepared, educated and supported in managing clinical deterioration. These surprising results raise the question; should we place more value in quality outcomes?
Justin Bowra - The elephant in the living room
Justin Bowra takes a break from ultrasound to broach the uncool but crucial subject of health care economics.
Health care spending make up a large proportion of the budgets of OECD nations, and it is increasing in relation to GDP. This is an unsustainable situation and something has got to give.
In part 1 of Justin’s talk, he asks the question, where is the money going? The commonly asserted points of the aging population, better medical treatments, litigation and corporatisation of health care contribute. Justin argues, however, that the biggest problem is the system itself. To acknowledge the elephant in the living room is to acknowledge that we as doctors contribute to the problem, but we also have the greatest responsibility to be part of the solution.
In part 2, Justin briefly discusses ways in which the system can be fixed. He touches on taming special interests, shared decision making, surrendering autonomy and to look at the big picture - remembering that what we do for each individual patient has consequences for everyone else.
Physical Therapy in the Emergency Departmentchristaloyd
At the Heart of the Rockies Regional Medical Center in Salida, CO, I got the opportunity to take the lead on doing research and analyzing data to create a presentation describing the benefits of Physical Therapy in an emergency department.
Imogen Mitchell - Morphing the Recalcitrant ClinicianSMACC Conference
Imogen Mitchell’s SMACC Chicago talk 'Morphing the Recalcitrant Clinician’ talks us through the steps to engage the reluctant physician when implementing change.
Imogen initally touches on the stages of physician engagement from aversion, to apathy, to engaged and then outlines the steps to morphing the reluctant physician.
1. Seek out a clinical champion
2. Establish a common purpose/vision
3. Standardise what is standardisable
4. Communication, communication, communication
5. Work out barriers and overcome them
6. Deal with the ‘Whats in it for me?’WIFM
Daily health update for 05/27/2015 for Poway Chiropractor Dr. Rode of Rode Chiropractic in Poway, CA 90264 located on Poway Rd in the Lively Center next to Cully's Restaurant.
Determining Prognosis in Cancer and Non-cancer DiagnosisVITAS Healthcare
This helps physicians, nurses, case managers and social workers understand the trajectories of dying from cancer and non-cancer diagnoses, including heart, lung, kidney and liver disease, stroke, HIV/AIDS, dementia and neurodegenerative diseases. Aided by a better grasp of the decline-related domains involved in poor prognosis, disease progression and disease end stages, attendees will be better positioned to identify patients and residents who are appropriate for hospice care.
Justin Bowra - The elephant in the living room
Justin Bowra takes a break from ultrasound to broach the uncool but crucial subject of health care economics.
Health care spending make up a large proportion of the budgets of OECD nations, and it is increasing in relation to GDP. This is an unsustainable situation and something has got to give.
In part 1 of Justin’s talk, he asks the question, where is the money going? The commonly asserted points of the aging population, better medical treatments, litigation and corporatisation of health care contribute. Justin argues, however, that the biggest problem is the system itself. To acknowledge the elephant in the living room is to acknowledge that we as doctors contribute to the problem, but we also have the greatest responsibility to be part of the solution.
In part 2, Justin briefly discusses ways in which the system can be fixed. He touches on taming special interests, shared decision making, surrendering autonomy and to look at the big picture - remembering that what we do for each individual patient has consequences for everyone else.
Physical Therapy in the Emergency Departmentchristaloyd
At the Heart of the Rockies Regional Medical Center in Salida, CO, I got the opportunity to take the lead on doing research and analyzing data to create a presentation describing the benefits of Physical Therapy in an emergency department.
Imogen Mitchell - Morphing the Recalcitrant ClinicianSMACC Conference
Imogen Mitchell’s SMACC Chicago talk 'Morphing the Recalcitrant Clinician’ talks us through the steps to engage the reluctant physician when implementing change.
Imogen initally touches on the stages of physician engagement from aversion, to apathy, to engaged and then outlines the steps to morphing the reluctant physician.
1. Seek out a clinical champion
2. Establish a common purpose/vision
3. Standardise what is standardisable
4. Communication, communication, communication
5. Work out barriers and overcome them
6. Deal with the ‘Whats in it for me?’WIFM
Daily health update for 05/27/2015 for Poway Chiropractor Dr. Rode of Rode Chiropractic in Poway, CA 90264 located on Poway Rd in the Lively Center next to Cully's Restaurant.
Determining Prognosis in Cancer and Non-cancer DiagnosisVITAS Healthcare
This helps physicians, nurses, case managers and social workers understand the trajectories of dying from cancer and non-cancer diagnoses, including heart, lung, kidney and liver disease, stroke, HIV/AIDS, dementia and neurodegenerative diseases. Aided by a better grasp of the decline-related domains involved in poor prognosis, disease progression and disease end stages, attendees will be better positioned to identify patients and residents who are appropriate for hospice care.
The 10th Annual Utah Health Services Research Conference: Assessment of Actual Pediatric Organ Donation Potential: Neurological and Circulatory Determination of Death. By: Erin E. Bennett, M.D., MPH; Jill Sweney, M.D.; Cecile Aguayo, R.N.; Craig Myrick, R.N.; Armand H. Matheny Antommaria, M.D., Ph.D.; Susan L. Bratton, M.D., MPH.
Patient Centered Research Methods Core, University of Utah, CCTS
Evidence demonstrates that communication is one of the leading contributors to adverse events. Transitions of care epitomize this challenge.
WATCH ON DEMAND: https://goo.gl/M1ovsS
Hazel Talbot gives her insights from working in neonatal and paediatric retrieval.
She delivers her talk with all the passion and dedication that she brings to her work as a neonatal and paediatric transport consultant.
Equipment failure, rapidly deteriorating children and miscommunication are all common challenges that Hazel and her team encounter in their line of work. This is on top of the challenge of caring for neonates and children.
How are children different? They differ in physiology, in disease profiles and even the way they make clinicians feel!
In this talk, Hazel focuses on physiology and disease. The large majority of young deaths in the UK, where Hazel works, are neonates – under 28 days old.
50% of these deaths are due to perinatal diseases. These include congenital malformations, prematurity, sepsis, and congenital heart conditions.
Children are small adults. Small airways, small necks, small lungs. Babies however are not small children. They use the majority of their physiological ability to survive.
When they breath they use most of their diaphragm, compared with an adult who will only use a small proportion. This leaves babies without much reserve. They have a high metabolic rate, and neonates have a right heart dominance with an inability to change their stroke volume.
Hazel urges you to consider this when faced with a sick baby. When thinking about neonatal and paediatric retrieval, Hazel has some key points. Practice, anticipation and knowing your environment and equipment is key to a success outcome.
Hazel drives this message home with a case presentation. This example highlights not only Hazel’s skill and knowledge, but also her ability of communication, leadership, and intuition.
Join Hazel as she gives an insight into her amazing work in neonatal and paediatric retrieval. Come along as she discusses her experiences and tells you how to ward of the Evil Transport Fairy!
For more like this, head to our podcast page. #CodaPodcast
The 10th Annual Utah Health Services Research Conference: Assessment of Actual Pediatric Organ Donation Potential: Neurological and Circulatory Determination of Death. By: Erin E. Bennett, M.D., MPH; Jill Sweney, M.D.; Cecile Aguayo, R.N.; Craig Myrick, R.N.; Armand H. Matheny Antommaria, M.D., Ph.D.; Susan L. Bratton, M.D., MPH.
Patient Centered Research Methods Core, University of Utah, CCTS
Evidence demonstrates that communication is one of the leading contributors to adverse events. Transitions of care epitomize this challenge.
WATCH ON DEMAND: https://goo.gl/M1ovsS
Hazel Talbot gives her insights from working in neonatal and paediatric retrieval.
She delivers her talk with all the passion and dedication that she brings to her work as a neonatal and paediatric transport consultant.
Equipment failure, rapidly deteriorating children and miscommunication are all common challenges that Hazel and her team encounter in their line of work. This is on top of the challenge of caring for neonates and children.
How are children different? They differ in physiology, in disease profiles and even the way they make clinicians feel!
In this talk, Hazel focuses on physiology and disease. The large majority of young deaths in the UK, where Hazel works, are neonates – under 28 days old.
50% of these deaths are due to perinatal diseases. These include congenital malformations, prematurity, sepsis, and congenital heart conditions.
Children are small adults. Small airways, small necks, small lungs. Babies however are not small children. They use the majority of their physiological ability to survive.
When they breath they use most of their diaphragm, compared with an adult who will only use a small proportion. This leaves babies without much reserve. They have a high metabolic rate, and neonates have a right heart dominance with an inability to change their stroke volume.
Hazel urges you to consider this when faced with a sick baby. When thinking about neonatal and paediatric retrieval, Hazel has some key points. Practice, anticipation and knowing your environment and equipment is key to a success outcome.
Hazel drives this message home with a case presentation. This example highlights not only Hazel’s skill and knowledge, but also her ability of communication, leadership, and intuition.
Join Hazel as she gives an insight into her amazing work in neonatal and paediatric retrieval. Come along as she discusses her experiences and tells you how to ward of the Evil Transport Fairy!
For more like this, head to our podcast page. #CodaPodcast
Presentation from the SWEETs 16 conference, Sweden. This presentation works on the applications of simulation for a major change management project in becoming ready for the closure of a paediatric hospital and the impact on an adult emergency department.
Workshop on the benefits of social media, professional learning networks and digital creation and curation. Given at the Laerdal Aus Simulation Users Network Sydney 2016
Deirdre talks ‘bad blood’ – the complex world of critical care haematology.
Critically ill patients frequently have activation of inflammatory and clotting pathways. These are likely adaptive responses in the human.
When they run riot, or the fine balance between pro- and anti-inflammatory states is shifted, there can be significant morbidity and mortality.
Deirdre presents three patients to highlight these issues and what you can do about it. This acronym-busting talk will focus on some acquired haematological disorders in critically ill patients.
Platelets make up a tiny percentage of blood – just 0.01%. However, they have a crucial role to play. A low platelet count can be due to reduced production or increased destruction.
Disseminated Intravascular Coagulation (DIC) is a clinical and laboratory diagnosis that affects about 1% of hospitalised patients. At the most severe end it is associated with bleeding and/or thrombotic complications. Disorders such as thrombotic thrombocytopenia purpura (TTP) and other forms of micro-angiopathic haemolytic anaemia (MAHA) will also be described including the role of ADAMST13.
The knowledge of what is what, is critical, as it will dictate treatment. Heparin-Induced Thrombocytopaenia (HIT) is an uncommon but important condition which is difficult to diagnose in a critically ill patient. It is a heparin dependent pro-thrombotic disorder. There is no good test for HIT.
Have you always wondered about NETs (neutrophil extracellular traps) and their importance? If so this whistle-stop tour of non-malignant hematology in the ICU is for you!
Deirdre drives home the message that low platelets are common in the critically ill and the causes are multifactorial.
Finally, for more like this head to our podcast page. #CodaPodcast
GEMC: Meningitis and Other CNS Infections: Resident TrainingOpen.Michigan
This is a lecture by Dr. Frank Madore from the Ghana Emergency Medicine Collaborative. To download the editable version (in PPT), to access additional learning modules, or to learn more about the project, see http://openmi.ch/em-gemc. Unless otherwise noted, this material is made available under the terms of the Creative Commons Attribution Share Alike-3.0 License: http://creativecommons.org/licenses/by-sa/3.0/.
This Talk is a Summary of:
1. Review the Importance of Quality in CPR
2. Discuss the Safety of “Hands-on” Defibrillation
3. Evaluate Manual vs Mechanical CPR
A presentation given by Luke Wainwright and myself about some of the trials and tribulations and eventual successes with integrating simulation into hospital education programs.
Stuart Lane on prognostication post out of hospital cardiac arrestSMACC Conference
Always controversial, always entertaining, the fearsome but loveable Geordie Stuart Lane gives an excellent summary of a core ICU topic: managing out of hospital cardiac arrests. Nearly at the end of the BCC3 series - and in only a month we're doing it all again, this time in tropical Cairns - come and join us.
Anyone Can Intubate, or Not: Teaching airway skills the antifragile waySMACC Conference
Becoming competent in airway management requires good decision making and and technical skills. Ultimately what matters is how your clinical performance impacts patient outcomes. For this we need to have a clear understanding of what defines success ensuring that its more than just 'getting the tube'. Come to this talk and you'll experience a Canadian take on Guinness, adventure sports, flying a plane and how other factors including failure influence airway management outcomes.
A Physiotherapist Perspective with Michelle Paton
Physiotherapists form a key part of the multi-disciplinary team in the Intensive Care, focusing on both respiratory care and optimisation of function. This talk will discuss the role of physiotherapy across the continuum specifically in the management of an acutely unwell septic patient. I will discuss the focus of a physiotherapy assessment, main treatment aims, some of the barriers for the implementation of physiotherapy in ICU, while identifying strategies to enable appropriate application of physiotherapy techniques.
Communication of prognosis has multiple barriers to achieve shared understanding between patient and clinician. In this slide deck designed for Hospice and Palliative Medicine fellows, I look at some key studies and applied techniques to best address talking about 'How long do I have, doc?'
This slide deck does not cover how to formulate a prognosis.
Care of Sickle Cell Disease Patients: Process Improvement & Change with NursesTosin Ola-Weissmann
Populations with SCD are at risk for disparities primarily because of the lack of knowledge on the part of the healthcare providers regarding the disease; inadequate pain management and prejudice among the staff (Tanabe & Myers, 2007).
On interviewing several nurses in the hospital, many acknowledge that they have never taken care of a patient with SCD and do not know what to assess for. The only nurse with experience of taking care of a SCD patient did not know the complications of the disease and wondered why sickle cell patients “always request pain medication when it’s obvious they are not in pain.”
This presentation is a guide providing essential information to medical professionals on dealing with patients that have sickle cell anemia. In addition, the SCD questionnaire is designed to enhance the assessment of SCD patients by medical professionals in the emergency room and serve as a platform for understanding their vulnerabilities during assessment.
Emphasis of this questionnaire is placed on identifying risk factors for depression, the patient’s socio-economic barriers, lifestyle habits, transportation issues, safe home environment, effective pain management and avenues for possible genetic counseling all of which sickle cell patients have shown vulnerability to (Dorsey & Murdaugh, 2003).
Rob Reid: Redesigning primary care: the Group Health journeyThe King's Fund
Rob Reid, Senior Investigator at Group Health Research Institute, explains the journey taken by Group Health in support of integrated primary care. A case study in how primary care can be delivered effectively and efficiently to a population, Rob laid out the challenges facing general practice in the States, and how Group Health worked to improve the situation for both patients and the workforce.
CORTICAL SPREADING DEPOLARISATION IN NEUROLOGICAL DISEASE – AN INTRODUCTION
By Toby Jeffcote
Cortical spreading depolarization (CSD) is a spreading loss of ion homeostasis, altered vascular response, change in synaptic architecture, and subsequent depression in electrical activity following an inciting neurological injury.
It was first described by Leão in 1944, a disturbance in neuronal electrophysiology has since been demonstrated in a number of animal studies, and recently a few human studies that examine the occurrence of this depolarizing phenomenon in the setting of a variety of pathological states, including migraines, cerebrovascular accidents, epilepsy, intracranial hemorrhages, and traumatic brain injuries. The onset of CSD has been demonstrated experimentally following a disruption in the neuronal environment leading to glutamate-induced toxicity. This initial event leads to pathological changes in the activity of ion channels that maintain membrane potential. Recovery mechanisms such as sodium-potassium pumps that aim to restore homeostasis fail, leading to osmolar shifts of fluid, swelling of the neuron, and ultimately a measurable depression in cortical activity that spreads in the order of millimeters per minute. Equally important is the resulting change in vascular response. In healthy tissue, increased electrical activity is coupled with release of vasodilatory factors such as nitric oxide and arachidonic acid metabolites that increase local blood flow to meet increased energy expenditure. In damaged tissue, not only is the restorative vascular response lacking but a vasoconstrictive response is promoted and the ischemia that follows adds to the severity of the initial injury. Tissue threatened by this ischemic response is then at elevated risk for CSD propagation and falls into a vicious cycle of electrical and hemodynamic disturbance. Efforts have been made to halt this spreading cortical depression using N-methyl-D-aspartate receptor antagonists and other ion channel blockers to minimize the damaging effects of CSD that can persist long after the triggering insult.
Celia Bradford takes us through the latest on the management of subdural haemorrhage (SDH). She covers acute SDH, chronic SDH and middle meningeal artery embolisation, a novel treatment for chronic SDH management in certain circumstances.
Andy Neill - More neuroanatomy pearls for neurocritical careSMACC Conference
Andy Neill shares some more neuroanatomy wisdom that's highly practical for anyone working with neuro emergencies. This time he covers brain herniation syndromes, hydrocephalus, extradural vs subdural haematomas, cervical spinal imaging, vertebral artery dissection and "things you read on CT reports but don't know what they mean"!
Andrew Udy talks about Brain Tissue Oxygen Monitoring:
It’s Not What You’ve Got It’s What You Do With It
The BONANZA Trial
Andrew Udy talks about the ongoing BONANZA Trial which is assessing whether an algorithm that incorporates both ICP and brain tissue oxygen (PbTO2) can improve outcomes after traumatic brain injury (TBI). Like with all monitoring, how the PbTO2 is interpreted and managed is critical and the devil is in the detail!
More on BONANZA here
More on BOOST3 here
R. Loch Macdonald, M.D., Ph.D.
Community Neurosciences Institute
Fresno, California, USA
Angiographic vasospasm and more accurately, delayed cerebral ischemia, continue to contribute to morbidity and mortality in patients with aneurysmal subarachnoid hemorrhage (SAH). It is known that angiographic vasospasm is common after SAH, occurring in two-thirds of patients. Cerebral infarctions that developed days after the SAH have been attributed to angiographic vasospasm, occuring in about a third of patients, although this has always been controversial. Angiographic vasospasm theoretically can only damage the brain by restricting blood flow but there is no easy, accurate, widely available method to measure cerebral blood flow and this is not the measurement we need. Blood flow depends on metabolic demand so what we need to know to determine if angiographic vasospasm is causing ischemia is oxygen extraction fraction in the brain tissue supplied the the spastic artery. Without this measurement, the attribution of ischemia to vasospasm is subjective. Since angiographic vasospasm is essentially the only detectable delayed phenomenon after SAH, we focus on it and apply tremendous resources to preventing or reversing the vasospasm. Undoubtedly angiographic vasospasm can cause cerebral infarctions, but it has to be severe and flow limiting. But SAH is a complex disease. There are many other causes for cerebral infarctions after SAH, the most common being due to the aneurysm repair procedure. And a given degree of vasospasm may cause infarction in a volume-depleted patient with poor collateral blood supply but not in a patient without these things. There also are hypodense brain lesions after SAH that are due to intracerebral hemorrhages. There can be hypodensities in the brain directly under usually thick SAH where the brain dies. This observation in particular supports a role for cortical spreading depolarizations/ischemia as a cause of infarction after SAH. Other macromolecular processes that are hypothesized to cause brain damage after SAH include microthromboembolism, changes in the microcirculation, delayed brain cell apoptosis and capillary transit time heterogeneity. Determining the importance of these things is hindered by the lack of an easy way to detect them in patients. It is also known that poor grade patients, who presumably have more early brain injury and ischemia than good grade patients, are more prone to delayed cerebral ischemia, suggesting increased sensitivity to secondary insults of the already injured brain. We also assume delayed neurological deterioration when attributed to vasospasm or delayed cerebral ischemia, is purely due to ischemia. While knowledge about what happens pathophysiologically after SAH is increasing, management of delayed cerebral ischemia still focuses on detecting angiographic vasospasm and then augmenting the blood pressure to improve cerebral blood flow or dilating the spastic arteries with balloons or drugs.
By Catherine Bell and Andrew Udy
Catherine Bell takes us through how to troubleshoot problems commonly encountered when looking after patients who have an external ventricular drain (EVD) in situ. Issues with using brain tissue oxygen monitors are also discussed. A highly practical session aimed at bedside clinicians.
There is no such thing as mild, moderate and severe TBI - by Andrew UdySMACC Conference
Part 2 of a debate over the classification of TBI. Andrew Udy then argues that this classification is fundamentally flawed. He discusses the issues with the Glasgow Coma Scale, and therefore the follow-on issues in TBI classification, including all the confounders to the GCS, the issues with timing of the score as well as GCS not taking baseline function or specifics subtypes of TBI into account. He makes teh argument that biomarkers may better categorise the diffuse entity we call TBI.
TBI Debate - Mild, moderate and severe categories workSMACC Conference
Andrew Chow, Intensivist with a neurosurgical background, argues that the current categorisation system for traumatic brain injury (TBI) works, and makes sense! He tackles us through the history of this system, and why it’s important to differentiate different types of TBI. The arguments in favour of this categorisation include the consistency and benefits of a universal language, the implications for triage and management, and the fact that this system has been endorsed by all major organisations
Dr Nick Little is an experienced Neurosurgeon who's looked after patients with traumatic brain injury for his whole career. Here he discusses the difficulties of prognostication following traumatic brain injury (TBI). He talks about the statistics of outcomes following mild, moderate and severe TBI and then goes on to tackle the harder topic of how we try to work out what an individual would want if they knew the spectrum of outcomes that they may face. The issues with the clinical examination findings we use to prognosticate are covered, as well as which imaging findings he finds most helpful. He also mentions the difficulties with current prognostic calculators.
Historically, when it came to brain injury, ketamine had a bad rap. Much of that dogma was dispelled in the last decade, and ketamine is now frequently used as an induction agent in acute brain injury, especially traumatic brain injury, due to it’s favorable effects on haemodynamics.
However a new application of ketamine is now being explored - whether ketamine may be able to reduce secondary brain injury.
Managing Complications of Chronic SCI by Bonne LeeSMACC Conference
20 million people around the world are living with a spinal cord injury (SCI). The medical issues they develop over the years differ to any other patient cohort.
These complications include autonomic dysreflexia, management of pressure areas, specific infections, nuanced peri-operative care and highly specific issues such as baclofen pump management and syringomyelia
Do look at the NeuroResus section on this and listen to Spinal Rehab Specialist Bonne Lee talk about this side of SCI care.
Keywords
SCI, spinal, spinal cord injury, autonomic dysreflexia, pressure areas, infection, peri-operative care, baclofen pump, syringomyelia, chronic SCI, spinal trauma, spinal rehab, incomplete SCI
Tania is a neurologist and epileptologist with expertise in continuous EEG (cEEG) and status epilepticus (SE). This talk covers what a seizure is, what status is, including focal and generalised status epilepticus.
So why do we do cEEGs for patients with suspected SE?
To confirm the diagnosis
To see if patient just post ictal or still seizing
To establish that the clinical and electric seizures have stopped
To see if burst suppression is achieved
To exclude other differential diagnoses
She makes a good argument for why cEEG is such an important tool in managing SE.
In the questions after the talk, the issue of availability of cEEG in the Australian setting was discussed. Limited montage EEGs are discussed including their pros and cons.
Stuart Browne is a Neuro Rehab specialist from Sydney. These slides accompany a talk he gave at the Brian Symposium in 2023. He discusses what "severe disability" really means.
Severe disability is more common than many realise - about 6% of the Australian population.
Stuart discusses how health is more than simply physical recovery and how it is a multidimensional construct. He covers how permanent disability doesn't necessarily equate to a poor quality of life. He also discusses the long timespan of recovery, which is often much longer than appreciated.
He specifically discusses "Locked-in Syndrome" and how the survivors have surprisingly positive self-reported health-related quality of life and well-being.
Stuart also covers how severely disabled people face various forms of discrimination.
Shree Basu is a Paediatirc Intensivist in Sydney. These slides from the Brain Symposium 2023 accompany the talk she gave. She discusses how Paediatric stroke presents, what neuroimaging is required and what interventions are available, including thrombolysis and the role of endovascular thrombectomy.
Hypertensing Spinal Cord Injury - gold standard or wacky?SMACC Conference
After spinal cord injury (SCI), there aren’t many interventions we have available that actually make a difference.
Augmenting blood pressure to increase spinal cord perfusion pressure is an attractive concept that may improve neurological outcomes following SCI. We know that hypotension can make SCI worse. Clinical studies looking at blood pressure augmentation are mostly old, retrospective and flawed in various ways.
Aiming for a MAP of > 85 for 5-7 days is recommended by guidelines but why this pressure and duration are good questions.
Hypertensive therapy is relatively safe and easy to implement but not without risk.
Tessa discusses the pros and cons, how this is managed practically and what the future may hold in this area.
Mark Weedon takes us through the increasingly utilised concept of an optimal cerebral perfusion pressure (CPPopt) for each unique patient. He discusses the background to CPPopt, including intrcranial pressure (ICP), the Monroe Kelly hypothesis, neurovascular coupling, and cerebral autoregulation in health and following brain injury. He shows how intracranial pressure is affected by intracranial compliance and how this affects ICP waveforms. Cerebral perfusion pressure in relation to the Brain Trauma Foundation guidelines is covered including management of elevated ICP (EICP). The currently recommended tiered approach to managing cerebral perfusion pressure and EICP is mentioned citing recent guidelines. He uses a clinical case of a TBI to illustrate how the CPPopt can be ascertained and used to guide therapy, including the easy to perform “MAP Challenge”. Mark also describes the Pressure Reactivity Index (PRx) and how it can be used as a target for therapy. Finally, he covers the exciting results of the preliminary COGiTATE pilot study.
Social Worker Victoria Whitfield and Bereavement councilor Louise Sayers discuss the power of words when health professionals are communicating topics around of death and serious injury with relatives and patients in critical care. They use role plays to bring theories to life.
Sepsis and Antimicrobial Stewardship - Two Sides of the Same CoinSMACC Conference
Appropriate use of antimicrobials is primarily a patient safety issue, and is the key aim of an effective antimicrobial stewardship program. We discuss the challenges in the management of a patient with sepsis, and how decision-making is usually done in the absence of effective diagnostics. Time dependent protocols and the knowledge that undertreatment of a patient with sepsis will lead to poor outcomes will lead to prescribing that may be driven by fear. Antimicrobial resistance is associated with over-use of antimicrobials but is usually not the immediate concern. Antimicrobial stewardship programs should work closely with sepsis teams to ensure that sepsis pathways are implemented across the whole hospital, and that key principles of judicious use are embedded within the clinical pathway.
Being able to prognosticate in the aftermath of a traumatic brain injury (TBI) is important as it assists with counselling patients and families. Moreover, it helps rationally allocate healthcare resources.
However, due to the heterogenous nature of TBI and variable pre brain injury patient factors and post brain injury course, this has proven to be a difficult task.
Large cohort studies have enabled improved accuracy in the prediction of 6 month mortality and unfavourable outcome.
Furthermore, many of the factors that contribute to long-term outcome have also emerged. However, it is not yet possible to use them in prediction algorithms or mathematical models.
There is emerging evidence that pre injury psychosocial and demographic factors may be of more relevance than injury severity. Moreover, that 'outcome' becomes increasingly subjective and complex as the post injury duration increases.
We end with three brief vignettes which highlight the fraught nature of long term outcome prediction.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
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RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
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Resus Room Service - Sarah Webb
1. Room Service Resuscitation
Sarah Webb
@sarahbebewebb
Nurse Practitioner, Intensive Care Unit
Royal North Shore Hospital,
Sydney, Australia
MN (Nurse Practitioner), MN (ICU), Gdip (Midwifery), Gcert (ICU), Bach Nurs
2.
3.
4.
5. CERS CNC
David Wastell
RRT Clinical Lead
Prof Carole Foot
Resuscitation CNC
Sarah Webb
RRT Director
Dr Liz Hickson
11th February 2013
12. Your Logo
Staff deskilling
• 95% strongly disagreed that the RRT caused deskilling
• 91% disagree RRT calls are required because nursing staff have
mismanaged their patients
Patient Safety
• 87% agreed RRT help teaches how to manage sick patients
• 96% agreed the system allowed them to seek help when worried
Effective Teamwork
• 96% agreed RRT encourage effective teamwork
• 89% disagreed that they would be reluctant to call the RRT for fear of
being criticised
13. Intranet Paging
EMR RR Form
CPA Form
Key
Performance
Indicators
Track and Trigger audits
Surveys
Resus Trolley Transition
REACH
Medical ALS
11 Met with Merits
State Innovation Award
Team of the Year Award
Quality
14 Chapter Movie
Weekly RRT Teaching
Weekly mock events
93% education saturation
Education
18. References
1 Jones DA, DeVita MA, Bellomo R. Rapid response teams. N Engl J Med
2011; 365: 139-146. 2 Brennan TA, Leape LL, Laird NM, et al. Incidence
of adverse events and negligence in hospitalized patients — results from
the Harvard Medical Practice Study I. N Engl J Med 1991; 324: 370-376.
3 Wilson RM, Runciman WB, Gibberd RW, et al. The Quality in Australian
Health Care Study. Med J Aust 1995; 163: 458-471. 4 Kohn LT, Corrigan
JM, Donaldson MS, editors. To err is human: building a safer health
system. Washington, DC: National Academy Press, 2000. 5 Hillman K.
The changing role of acute care hospitals. Med J Aust 1999; 170: 325-
328. 6 Adhikari NKJ, Fowler RA, Bhagwanjee S, Rubenfeld GD. Critical
care and the global burden of critical illness in adults. Lancet 2010; 375:
1339-1346. 7 Donni-Lenhoff FG, Hedrick HL. Growth of specialization in
graduate medical education. JAMA 2000; 284: 1284-1289. 8 Schein RM,
Hazday N, Pena M, et al. Clinical antecedents to in-hospital
cardiopulmonary arrest. Chest 1990; 98: 1388-1392. 9 Hillman KM,
Bristow PJ, Chey T, et al. Antecedents to hospital deaths. Intern Med J
2001; 31: 343-348. 10 Hillman KM, Bristow PJ, Chey T, et al. Duration of
life-threatening antecedents prior to intensive care admission. Intensive
Care Med 2002; 28: 1629-1634. 11 Donaldson LJ, Panesar SS, Darzi A.
Patient-safety-related hospital deaths in England: thematic analysis of
incidents reported to a national database, 2010–2012. PLoS Med 2014;
11: e1001667. 12 Hillman K, Chen J, Cretikos M, et al; MERIT Study
Investigators. Introduction of the medical emergency team (MET)
359.el-369.el. 15 Chen J, Ou L, Hollis SJ. How accurate are the diff
erent predictive models in identifying deteriorating patients? The
ViEWS may not be as clear as we fi rst thought. Crit Care Med 2014;
42: 986-987. 16 Gerdik C, Vallish RO, Miles K, et al. Successful
implementation of a family and patient activated rapid response
team in an adult level 1 trauma center. Resuscitation 2010; 81:
1676-1681. 17 Loekito E, Bailey J, Bellomo R, et al. Common
laboratory tests predict imminent death in ward patients.
Resuscitation 2013; 84: 280-285. 18 Esmonde L, McDonnell, Ball C,
et al. Investigating the eff ectiveness of critical care outreach
services: a systematic review. Intensive Care Med 2006; 32: 1713-
1721. 19 Clinical Excellence Commission. Between the Flags project:
the way forward. Sydney: CEC, 2008.
http://www.cec.health.nsw.gov.
au/__documents/programs/between-the-fl ags/2013/btf-the-
wayforward-2008.pdf (accessed Jul 2014). 20 Chan PS, Jain R,
Nallmothu BK, et al. Rapid response teams. A systematic review and
meta-analysis. Arch Intern Med 2010; 170: 18-26. 21 Hillman KM.
Run, don’t walk. Crit Care Med 2012; 40: 2712-2713. 22 Hillman K,
Alexandrou E, Flabouris M, et al. Clinical outcome indicators in
acute hospital medicine. Clin Intensive Care 2000; 11: 89-94. 23
Chen J, Bellomo R, Flabouris A, et al; MERIT Study Investigators for
the Simpson Centre and the ANZICS Clinical Trials Group. The
relationship between early emergency team calls and serious
adverse events. Crit Care Med 2009; 37: 148-153. 24 Buist M,
Bernard S, Nguyen TV, et al. Association between clinically
abnormal observations and subsequent in-hospital mortality: a
prospective study. Resuscitation 2004; 62: 137-141. 25 Bellomo R,
Goldsmith D, Uchino S, et al. A prospective before-and-after trial of
Editor's Notes
Hello everyone. This presentation is about Room Service resuscitation but before I start I’d like to wet your appetite. Take a look at this…
So I’d like to do is shine some light on one of these systems…the rapid response system.
But before you all fall asleep don’t worry I’m not going tell you all about yet another single centre, before and after study that showed reduced cardiac arrests and hospital mortality…. Because we weren’t able to show this…
In fact, what I think Rapid Response is all about is comfort and cost. What sort of a hotel do you prefer to stay in? Would you rather stay in a 5 star height of luxury hotel that costs about $400 a night or would you rather stay in a more simple establishment for $100 a night.
Both have the essentials; a bed, toilet facilities and adequate food. But the five star hotel is much more expensive and much more enjoyable to stay in, even staff love working there because it’s such a nice enviornment, and not only is the food restaurant quality there’s even a service that delivers it to your own room.
2 years ago I was part of a team that implemented the first rapid response service in our organisation.
This picture was taken on the very first day that our rapid response service began. All of us only had began in these positions just a few months before.
There are three main reasons are hospital decided to implement our roles and the RRT…
We had 16 serious clinical incidents in one year…that were all related to clinical deterioration.
Our hospital was being accredited under new national standards which we were going to fail because we didn’t have a rapid response system.
And finally our hospital was also being rebuilt so in terms of change management this was an ideal time to overhaul the resuscitation processes.
This was a really interesting time for us because we go to see first hand the impact of a rapid response system.
What we really noticed was a before and after effect, a shift from a reliance on critical thinking to a reliance on protocols.
Before we had the RRT our ward nursing and medical colleagues had to be independent in assessing and trouble shooting clinical deterioration appropriately, critical care expertise was only activated when ward staff had exhausted their own clinical skills. These days it often seems like critical thinking has been replaced by protocols such as mandatory escalation, where ward nurses and doctors activate a critical care team based on a track and trigger observation chart.
The reason track and trigger systems have been implemented in national health and safety standards around world is because there is a well established link between abnormal physiological parameters and adverse patient events such as unexpected death, cardiac arrest or admission to ICU. The theory is that the ward nurses track the observations, and when they fall out of normal parameters they are triggered to call the critical care team. A second set of eyes who review the patient and treat the cause of deterioration before it cascades into a sentinel event.
But the sensitivity of these systems is a serious concern for us. This chart represents the percentages of RRT activation in our first year. We had 3740 calls and 37% of our calls were for low blood pressure.
The criteria for this is a systolic blood pressure of less than 90mmHg. Now of course, this might be seriously concerning in a small cohort of patients but the majority of these calls are treated with simple medicine not critical care.
In fact of all those 3740 calls only 9% resulted in transfer to a higher level of care. Facts like this make us think is this really the work of critical care teams?
Who are the right people to provide this service? A secondary effect that we noticed was that the RRT compounded the progressive specialisation of medical and surgical teams. What I mean by this is that specialist doctors are becoming more focused on their particular specialty. And the problem with this is, that the more focused specialties become, the bigger the clinical gap between them becomes.
We are the best to equipped to treat these patients and prevent adverse events but shouldn’t home teams be more holistic than a surgical site or single organ dysfunction? And if we’re having to review all the patients in the hospital who have deranged observations then who is ultimately responsible for them and where does that responsibility end?
On the other hand when I have my hip replacement I’d rather have a surgeon who does nothing but hips all day everyday… and when I become hypotensive a week later I’d rather be reviewed by people who know the most about systemic inflammatory response.
The third effect that we really noticed was the impact that the RRT had on the clinicians.
From our perspective after the first month this is how I think most of us felt once we had seen our 40th call for a SBP 88. This isn’t what critical care is about. Where are the cardiac arrests and emergency intubations.
For the RRT nurses it was still kind of fun… most of us had only ever worked in ICU so we were quite excited to check out the rest of the hospital. But for our doctors who’d already spent years working on the wards there really was sense of sense of deflation. It was incredibly frustrating for them to follow these very rigid protocols when simple medicine would have sufficed and we wondered if all we were doing was deskilling the ward staff.
Now the ward staff were barely represented in the movie at the beginning of this talk and prior to the RRT we had a very different view of our ward colleagues as I’m sure they did of us…
but what we hadn’t appreciated was that as the wards were getting busier and busier, the patients were aging and becoming more complex and the home teams were becoming more focused on their specialty… the ones left holding the pieces were the ward staff.
RR Service might have replaced critical thinking with protocols but those protocols actually empower these guys to call for help they need it, it also creates unique teaching opportunities with critical care team which is espcecially valuable for the junior ward doctors.
In fact when we ran a survey for are ward colleagues in the first year of the RRT and the results were really surpising.
In terms of deskilling they didn’t see the RRT made them deskill at all.
In terms of patients of patient safety they felt the system actually improved their ability to manage sick patients and they now felt they had someone they could call if they were worried.
In terms of team work almost all of them felt that the rrt encouraged effective teamwork…
Now this data isn’t the stuff that usually influences hospital administrators to continue funding services but I think this demonstrate an important cultural shift, a breaking down of the barriers between ICU and the wards and a new alliance between ICU and the ward stuff which makes our working environment and especially the wards environment much more enjoyable.
Implementing this system was an enormous effort and it certainly wasn’t just our team, there were numerous key stakeholders in the executive committee as well as nursing and medical clinicians both in the ICU and throughout the hospital who contributed to this project.
The education alone was huge. We made a 14 part movie that could be played to the mass audiences, we ran weekly teaching RRT teaching sessions, weekly mock events on the wards and at the end of the year we showed 93% education saturation throughout the hospital.
We rigorously followed our KPIs… we worked with the electronic medical record team to implement an intranet paging system, we built electronic forms for rapid response and cardiac arrest calls to make sure that our data was as clean and robust as possible.
We ran numerous Quality Initiatives; Observation audits, staff satisfaction surveys, we standardised and simplified the resuscitation equipment in the entire hospital, we dramatically increased the advanced life support accreditaion of our junior medical staff from 2% to 55% in the first year, we smashed hospital accrediation, we won a state innovation award and we even implemented a process were patients and relatives could call the rrt directly.
There was nothing held back... Nothing left in the tank.
When it comes to evaluating Rapid Response Teams, the holy grails are improved cardiac arrests, Icu admissions and hospital mortality. and you can see our results don’t actually look that promising.
But having lived and breathed this data, counting arrest forms, analysing excel spreadsheets, pivot tables and data dictionaries, I actually take these KPIs with a grain of salt because there’s a surprising amount of variability in the way this information can caulcuated and the factors that influence them are vast. I can bore anyone who ineterested in this over lunch but for the purporse of this talk… Rapid Response Teams may not dramatically change our patient outcomes the way we had hoped.
So if our patient outcomes haven’t really changed what have we achieved then? Have we just upgraded from our 2 star hotel? Have we just implemented a form of room service?
In summary, RRTs shift a reliance on critical thinking to a reliance on protocols, they compound the progressive specialisation of our home medical teams, they may not involve the most exciting resuscitation medicine, they may not dramatically improve our patient outcomes but they do supbstantially improve working environment for our ward colleagues and break down barriers between ICU and the wards.. Which I think can only be beneficial for the patients.
So I’d like to leave you with one last image I mean message.
The next time your booking your hotel remember to book the five star one with the room service, your outcomes are probably going to be same but the journey will be much more enjoyable.