Prof. Anisuddin Bhatti gave lectures to residents & Junior consultants on PostPolio Residual Paralysis part2 lower limb Reconstructive surgery on 17.04.202. Acknowledged for text and figures as such in reference list.
Deformity: It’s the position of a limb/Joint, from which it cannot be brought back to its normal anatomical position.
Described as abnormalities of :
Length
Angulation
Rotation
Translation
Combination
Deformity: It’s the position of a limb/Joint, from which it cannot be brought back to its normal anatomical position.
Described as abnormalities of :
Length
Angulation
Rotation
Translation
Combination
Osseous anatomy, Types of approaches(Position,landmarks,Incision,Superficial and Deep surgical dissection) , structures at risk, Extensile approaches with diagrams and eponymous .
Hip resurfacing has emerged as a viable alternative to replacement for arthritis in young patients. Selected individuals will benefit by Hip resurfacing arthroplasty offered by the Madras Joint replacement center in India. See if you qualify for this procedure.
Post Polio Residual Palsy: Pathophysiology & Principles of RxAnisuddin Bhatti
Prof. Anisuddin Bhatti, Paeds Orthop Surgeon delivered lecture on Post Polio paralysis and deformities Part 1 on Pathophysio and principles of treatment, through Dr. Ziauddin University Hospital Clifton Karachi webinar on googel.meet, on 3rd April 2021. Acknowledge for material taken from Research papers, slideshare and books as referred in reference list.
ALL (antero-lateral ligament) - extra articular ACL reconstruction - basicsMilind Tanwar
history, need, how to reconstruct, when to reconstruct.
References: *Bonasia, Davide Edoardo et al. "Anterolateral Ligament Of The Knee: Back To The Future In Anterior Cruciate Ligament Reconstruction". Orthopedic Reviews 7.2 (2015)
Biomechanical Results of Lateral Extra-articular
Tenodesis Procedures of the Knee:
A Systematic Review. Erik L. Slette, B.A., Jacob D. Mikula, B.S., Jason M. Schon, B.S., Daniel C. Marchetti, B.A.,
Matthew M. Kheir, B.S., Travis Lee Turnbull, Ph.D., and Robert F. LaPrade, M.D., Ph.D.
Osseous anatomy, Types of approaches(Position,landmarks,Incision,Superficial and Deep surgical dissection) , structures at risk, Extensile approaches with diagrams and eponymous .
Hip resurfacing has emerged as a viable alternative to replacement for arthritis in young patients. Selected individuals will benefit by Hip resurfacing arthroplasty offered by the Madras Joint replacement center in India. See if you qualify for this procedure.
Post Polio Residual Palsy: Pathophysiology & Principles of RxAnisuddin Bhatti
Prof. Anisuddin Bhatti, Paeds Orthop Surgeon delivered lecture on Post Polio paralysis and deformities Part 1 on Pathophysio and principles of treatment, through Dr. Ziauddin University Hospital Clifton Karachi webinar on googel.meet, on 3rd April 2021. Acknowledge for material taken from Research papers, slideshare and books as referred in reference list.
ALL (antero-lateral ligament) - extra articular ACL reconstruction - basicsMilind Tanwar
history, need, how to reconstruct, when to reconstruct.
References: *Bonasia, Davide Edoardo et al. "Anterolateral Ligament Of The Knee: Back To The Future In Anterior Cruciate Ligament Reconstruction". Orthopedic Reviews 7.2 (2015)
Biomechanical Results of Lateral Extra-articular
Tenodesis Procedures of the Knee:
A Systematic Review. Erik L. Slette, B.A., Jacob D. Mikula, B.S., Jason M. Schon, B.S., Daniel C. Marchetti, B.A.,
Matthew M. Kheir, B.S., Travis Lee Turnbull, Ph.D., and Robert F. LaPrade, M.D., Ph.D.
Dr. Anisuddin Bhatti Paediatric Orthopaedic Surgeon DR. Ziauddin University Karachi presented talk on Congenital Vertical Talus at AKU karachi on August 2023 in Orthopaedic Review course. Acknowledged for some text material & photo taken from Published literature.
TKA in valgus knee is challenging procedure seen in up to 10% of cases undergoing TKA. The procedure involves meticulous pre operative planning and intra operative soft tissue release along with modifications in bone cuts for proper implant placement and long term results
Prof. Anisuddin Bhatti Paediatric Orthopaedic surgeon Dr. Ziauddin University hospital Clifton Karachi Pakistan Presented Principles & practice in Clubfoot at AKU Orthopaedic Review Course. October 2023. Acknowledged for Some text material & Photo taken from Global Health publication on Ponseti Clubfoot treatment & internet media.
This is the Presentation on the topic "Pathomechanics of Knee Joint".
The presentation includes images and a clip for proper understanding. The sentences are framed in the way that you can learn it in a easy way.
Why Ponseti Technique in Clubfoot management MARCH 2022.pptxAnisuddin Bhatti
Prof Anisuddin Bhatti. Paediatric Orthopaedic surgeon presented talk on Why Ponseti Technique, Concept Evolution in Clubfoot treatment. Presented in AKUH Orthopaedic Review course. March 2022. Acknowledged for some text material & photo taken from published literature
Principles, pitfalls & problems of Paediatrics Fractures AKU 2023.pptxAnisuddin Bhatti
Dr. Anisuddin Bhatti Paediatric Orthopaedic Surgeon DR. Ziauddin University Karachi presented talk on Paediatric fractures principles of treatment at AKU karachi on August 2023 in Orthopaedic Review course. Acknowledged for some text material & photo taken from Published literature.
Principles of Containment in PERTHES AKU August 2023.pptxAnisuddin Bhatti
Prof. Anisuddin Bhatti Paediatric Orthopaedic surgeon Dr. Ziauddin University hospital Clifton Karachi Pakistan Presented how to take clinic examination short case on Congenital Knee dislocation, at AKU Karachi Orthopaedic Review course on August .2023. Acknowledged for some text 7 Photo taken from Published literature.
Prof. Anisuddin Bhatti Paediatric Orthopaedic surgeon Dr. Ziauddin University hospital Clifton Karachi Pakistan Presented how to take clinic examination short case on Clubfoot Deformity, at HMC complex Peshawer on 24.11.2023
Prof. Anisuddin Bhatti Paediatric Orthopaedic surgeon Dr. Ziauddin University hospital Clifton Karachi Pakistan Presented how to take clinic examination short case on Congenital Knee dislocation, at HMC complex Peshawer on 24.11.2023
Prof. Anisuddin Bhatti Orthopaedic & Paediatric Orthopaedic surgeon Dr. Ziauddin University hospital Clifton Karachi Pakistan Presented how to take clinic examination long case on Cerebral Palsy patient, at HMC complex Peshawer on 24.11.2023
Mock Clinical Examination Long case Acetabulum frx.pptxAnisuddin Bhatti
Prof. Anisuddin Bhatti Orthopaedic & trauma surgeon Dr. Ziauddin University hospital Clifton Karachi Pakistan Presented how to take clinic examination long case on Acetabulum fracture, at HMC complex Peshawer on 24.11.2023
Prof. Anisuddin Bhatti Paediatric Orthopaedic Surgeon Dr. Ziauddin University Hospital Clifton Presented Mock Examination technique short case on Clubfoot at HMC Peshawer on 24.11.2023
Mock Examination Short case CKD to long case.pptxAnisuddin Bhatti
Prof. Anisuddin Bhatti Paediatric Orthopaedic Surgeon Dr. Ziauddin University Hospital Clifton Karachi Pakistan presented How to take Short case on congenital Dislocated knee at MOCK Examination at HMC Peshawer on 24.11.2023
Prof. Anisuddin Bhatti Paediatric Orthopaedic Surgeon Dr. Ziauddin University Hospital Clifton Karachi, Pakistan Presented Mock Clinical Examination on CP long case at Hayatabad Medical Complex Peshawer on 24.11.2023.
Bhatti's Functional Scoring System for Developmental Dysplastic HipsAnisuddin Bhatti
Prof. Anisuddin Bhatti, Paediatric Orthopaedic Surgeon, Ziauddin University Hospital, Clifton, Karachi, Pakistan, Innovated a new scoring system for evaluation of Post open reduction DDH (Developmental Dysplastic Hips). The Bhatti's Functional Scoring System elaborates eastern life styles especially daily accustomed sitting habits. BFSS is a comprehensive system that evaluate functional limitations, Range of motion, endurance, limp, and trendelenburg gait if any.
Prof. Anisuddin Bhatti, Paediatric Orthopaedic Surgeon, Dr. Ziauddin University Hospital, Clifton campus Karachi, presented lecture on Congenital Clubfoot and PPV deformity evaluation & treatment. On 31 May 2021 to Resident's of AKUH and others. Acknowledged text & picture source as indicated in reference list.
Prof. Anisuddin Bhatti Paediatric Orthopaedic Surgeon Dr. Ziauddin University Hospital, Clifton, Karachi delivered lecture on DZU Webinar series Lecture 2 on Legg Calve Perthes. Declared few pics and material taken from google.
Prof Anisuddin Bhatti Paediatric orthopaedic surgeon Dr. Ziauddin University Hospital Clifton, Karachi delivered Lectures to trainees and Junior consultants on PERTHES' part-1, [Pathogenesis, Diagnosis, Classification and case discussion] on 20th February 2021, through Dr. Ziauddin Hospital Clifton Webinar series. Part-2 on Perthes' management to be delivered on 6th March 2021. he declares few pictures & material taken from Google.com and mostly his own patients
Prof. Anisuddin Bhatti. Paediatric Orthopaedic surgeon presented this lecture as 4th in series of Dr. Ziauddin University karachi webinar series. Treatment principles & protocols of Open reduction in DDH in age over 8 years. especial reference given to Triple redirection osteotomies.
3a ddh open reduction principles & protocolsAnisuddin Bhatti
Prof. Anisuddin Bhatti, Paeds Orthopaedic surgeon, Dr. Ziauddin University Hospital Clifton Karachi, Pakistan, delivered lecture on Developmental Dysplastic Hips Treatment principles, protocols and procedures on 21.11.2020. he elaborated on principles /protocols of Open reduction. elaborated in detail on Catteral test of stability, Salters osteomy & Pemberton Osteotomy.He also gave example of disaster if principles of open reduction are violated.this lecture series on DDH was mostly for trainees and young Orthop surgeons.
2 ddh principles & protocols of rx. 0 12 m age Anisuddin Bhatti
Prof. Anisuddin Bhatti, Paeds Orthopaedic Surgeon @ Dr. Ziauddin University Hospital, Clifton Karachi Pakistan, delivered a lecture on Developmental Dysplastic Hip: Principles & protocols of Rx in age less than a year. This was a second lecture in Webinar series of DZU on DDH.third lecture on Principles of DDH surgery shall be delivered after fortnight on Friday November 20th, 2020 @ 09:00-10:00 on DZU webinar through google.
Prof. Anis Bhatti lecture on DDH evaluation & screening ProtocolsAnisuddin Bhatti
Prof. Anisuddin Bhatti, Paediatric Orthopaedic Surgeon, Dr. Ziauddin University Hospital Clifton Karachi, presented webinar on Developmental dysplastic hip, series 1. on <meet.google.com> on 16.10.2020. Presentation mostly for trainees & jr. consultants. He explained in detail, pathoanatomy, screening protocols, ultrasonography & radiological evaluation of DDH cases.
Prof. Anisuddin Bhatti, Paediatric Orthopaedic Surgeon @ Dr. Ziauddin University Hospital, Clifton, Karachi, presents webinar GAIT DISORDERS & ANALYSIS with Sp reference to Trendelenburg gat & Cerebral Palsy Spastic gaits
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
Acute scrotum is a general term referring to an emergency condition affecting the contents or the wall of the scrotum.
There are a number of conditions that present acutely, predominantly with pain and/or swelling
A careful and detailed history and examination, and in some cases, investigations allow differentiation between these diagnoses. A prompt diagnosis is essential as the patient may require urgent surgical intervention
Testicular torsion refers to twisting of the spermatic cord, causing ischaemia of the testicle.
Testicular torsion results from inadequate fixation of the testis to the tunica vaginalis producing ischemia from reduced arterial inflow and venous outflow obstruction.
The prevalence of testicular torsion in adult patients hospitalized with acute scrotal pain is approximately 25 to 50 percent
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Ocular injury ppt Upendra pal optometrist upums saifai etawah
PostPolio Residual Paralysis part2 lower limb
1. POST POLIO DEFORMITY &
RESIDUAL PARALYSIS: Part 2
Development of Regional Deformities &
Reconstructive Surgery
Anisuddin Bhatti
Professor, Dr. Ziauddin Hospital, Clifton
President Rt, Paeds Ortho Society Pakistan & POA
Focal Person, Ponseti International, Pakistan
Dr. Ziauddin University Hospital, Webinar Series
Saturday, 17th April 2021, 09:00-10:00
3. POSTPOLIO DEFORMITIES & RESIDUAL
PARALYSIS
Before Embarking to Reconstructive surgery, it is
necessary to have detailed understanding &
knowledge of:
• Pathologic process
• Pathoanatomy of deformity
• Clinical Assessment
• Principles of Reconstructive Surgery
• Regional Deformities
• Management principles
• Case Discussion
Physiolone.com
4. DECISION MAKING FACTORS
The most important factors that need attention
before embarking to reconstructive surgery
• Age of the patient,
• Functional assessment of limbs & spine
• Socioeconomic background.
• Radiographic examinations.
5. FUNCTIONAL ASSESMENT
1. Muscle Charting / power grades
2. Extent of contractures and
deformities
3. Ambulatory Status & Posture
4. LLD - Shortening of the limb
follow
6. EXTENT OF CONTRACTURES AND DEFORMITIES
• Trendelenburg test
• Thomas test
• Ober / Yount’s test
• Ankle & Foot deformity
evaluation
• Pelvis & spinal curvatures
Slideshare
https://www.slideshare.net/AnisuddinBha
tti1/anis-bhatti-cp-2-clinical-assesment-
2020
Youtube https://youtu.be/IWLnWJ2P-3g
follow
7. AMBULATORY STATUS
• Observation of Gait /
Gait Lab Analysis
• Abductor Lurch
• Extensor Lurch
• Hand to Knee Gait
• The Calcaneus Gait
• Foot Drop Gait
• Short Limb Gait
Slide share
https://www.slideshare.net/AnisuddinBhatti1/4
anisbhatti-gait-disorders
youtube
https://youtu.be/96fZsU5SyYY
follow
8. PROGNOSTIC FACTORS
• i. Severity of initial paralysis
• ii. Diffuseness of its regional distribution
• Iii. Expectations & support
• Iv. Resources availibilty
• In general, the more extensive the paralysis in the
first 10 days of illness the more severe the ultimate
disability.
9. OPTIONS: RECONSTRUCTIVE SURGERY
1. Release of contractures : Fasciotomies &
Capsulotomies
II. Re-establishment of power by:
a) Tendon transfer to prevent deformities
b) Muscle transplantation: to replace a
paralyzed muscle
III. Stabilization of a relaxed or flail joint by:
(a) Tenodesis
(b) Construction of Bone block
Tenodesis of Achilles tendon to fibula
Posterior bone block
10. OPTIONS : RECONSTRUCTIVE SURGERY
IV. Correction of Deformities by
a) Osteotomies b) Arthrodesis
V. Limb lengthening (Ilizarov techniques to
release contracture & Limb Lengthening)
VI. Joint replacement surgery
VII. Correction of pelvic obliquity & Spine
deformity & stabilization
12. I. Muscle imbalance:
Contracture of Flexion and
abduction muscles and Paralysis of
Gluteus maximus and Medius
muscles:
•Abductor Lurch / Trendlenburg
sign
•Paralytic dislocation of the hip
Hip Deformities - Causes
Source: J Children Orthop Oct
2015
Benjamin Josef.
Pelvic obliquity due to an
abduction contracture of one hip
13. II. Maintenance of a posture at
ease (Frog leg posture) during acute
and convalescent phase:
• Knees and hips remain flexed and
extremities completely externally
rotated.
• The secondary soft tissue & Iliotibial
band contracture leads to a
permanent deformity of FFC Hip &
Knee.
Cause of Hip deformities
Source Google.com.
14. ILIOTIBIAL BAND CONTRACTURE
Flexion, abduction, external rotation contracture of the
hip & Flexion deformity knee
• The Iliotibial band by virtue of its lateral & anterior position over the
hip joint, causes a flexion and abduction deformity.
• Due to position of comfort leg remain Externally rotated that, the
contracture of external rotators & contribute, further to a fixed
deformity.
• Due to attachements of Iliotibial band from iliac crest down to the
knee joint its its contracture produce double joint deformities: FFD
Hip & Knee.
Source Google.com.
15. ILIOTIBIAL BAND CONTRACTURE
Genu valgum and flexion contracture of the
knee
• With progressive growth, the contracted
iliotibial band (ITB) acts as a taut bowstring
across the knee joint, that gradually abducts
and flexes and cause FFC knee and Genu
valgus.
Genu
valgu
s
16. ILIOTIBIAL BAND CONTRACTURE
External tibial torsion, with or
without knee joint subluxation
• Due to ITB lateral attachment below knee &
its contracture gradually leads to external
tibia & fibula rotation.
• The externl rotation exaggerated by the
strog Biceps femoris
• When the deformity becomes extreme,
tibial condyle subluxates, rotates externaly,
thus head of fibula lies in the popliteal
Ext Tib
Torsion
Knee Subluxation
& FFC
17. ILIOTIBIAL BAND CONTRACTURE
Secondary Ankle and Foot deformities:
(Positional Pes Cavus / Cavo varus)
Results from ill fitted orthrosis that fails to
compensate external tibial torsion caused by
tight ITB.
Mechanism:
The axes of the knee and ankle joints do not occupy the same
horizontal plane in external torsion of the tibia.
When an above-knee orthosis manufactured with these joints
in the same horizontal plane is fitted to a limb with external
tibial torsion, the appliance will force the foot into varus
position so that the ankle is in line with the knee joint
18. ILIOTIBIAL BAND CONTRACTURE
Pelvic obliquity
• When the patient is supine, the iliotibial band contracture
keeps hip in abduction and flexion, the pelvis may remain at a
right angle to the long axis of the spine.
• When the patient stands , the affected extremity is brought
into the weight-bearing position (parallel to the vertical axis of
the trunk), the pelvis assumes an oblique position
On standing upright the iliac crest assumes a low
posture on contracted (Lt.) side and high on the
opposite (Rt) side. On long stading dislocation of
hip occures on other side
19. ILIOTIBIAL BAND CONTRACTURE
Pelvic obliquity:
On standing upright the iliac
crest assumes a low posture
on contracted (Lt.) side and
high on the opposite (Rt)
side. On long stading
dislocation of hip occures on
other side
Source: Campbell Orthop 12th Ed
20. ILIOTIBIAL BAND CONTRACTURE
Scoliosis
• The trunk muscles on the affected side lengthen, and the
muscles on the opposite side contract. An associated lumbar
scoliosis can develop.
• If not corrected, the two contralateral contractures (ITB band on
the affected side and the trunk muscles on the unaffected side)
hold the pelvis in this oblique position until skeletal changes fix
the deformity.
Increased lumbar lordosis
• Bilateral flexion contractures of the hip pull the proximal part of
the pelvis anteriorly; for the trunk to assume an upright position,
a compensatory increase in lumbar lordosis must develop
JaypeeDigital
21. DEFORMITIES CAUSED BY
ILIOTIBIAL BAND CONTRACTURE
• Pes Varus, valgus
• Pelvic obliquity
• Scoliosis
• Lumber lordosis
HIP Deformities:
• FFC
• FFC + Abduction +
External rotation
• Paralytic Dislocation
Knee Deformities:
FFC
FFC + Subluxation
External tibial torsion
23. SURGICAL RECONSTRUCTION
HIP AND KNEE CONTRACTURES
INDICATIONS FOR SURGERY:
• Hip and knee contractures
exceeding 30°
CONTRA INDICATIONS:
• Weakness of one or both arms in
addition to bilateral lower-limb
paralysis, making the use of
crutches difficult or impossible.
DECISION MAKING FACTORS:
• Age
• Severity of Contracture
oYoung child with recent
contracture : release Tight
tensor fasciae latae and Iliotibial
band,
oTeenage & Adolescent: Divide
additional ligamentous and
tendinous structures
24. RELEASE OF FFD HIP & KNEE:
Iliotibial band contracture
SOUTTAR’S SLIDE / CAMPBEL’S RELEASE
• Release of TFL and Gluteus maximus from from their origins
(anterolateral part of the hip) to correct hip FFD.
OBER-YOUNT PROCEDURE
Sartorius,TFL, Rectus femorus,Gluteus medius and minimus, Iliopsoas
tendon.
YOUNT’S RELEASE
Resection of the thickened anterolateral fascia latae, to correct knee
contracture. CAUTION:
Avoid damaging the femoral, popliteal arteries & common peroneal nerve.
Divide biceps under direct vision to avoid risk of damaging the adjacent lateral
popliteal nerve.
25. FFD HIP & KNEE:
ILIOTIBIAL BAND CONTRACTURE
Before & After SOUTTAR’S SLIDE , YOUNT’S
RELEASE. Source: M. Sai Krishna
Origin of Sartorius, TFL & G. Med released
+/- Resection of redundant iliac crest.
Source: Campbell Op Orthop
Campbell’s Slide for FFC Hip
Source: Tachdjian
26. PARALYSIS of
GLUTEUS MAXIMUS & MEDIUS
• Unstable hip and an unsightly and fatiguing
limp
• Gluteus Medius alone Palsy: Trendelenburg
Gait
• Gluteus maximus alone paralysis: Backward
lurch
Treatment
• Sharrards’ Posterior transfer of iliopsoas for
paralysis of the gluteus medius and maximus
muscles Trendelenburg Gait
Backward lurch
27. MUSCLE TRNSFER TO RESTORE DYNAMIC BALANCE @ HIP
PREVENT PELVIC OBLIQUITY & SUBLUXATIO
Age of Onset of
Paralysis
Principles
< 2 years • Early stage: Adductor tenotomy +/- OR
• Late Stage: IlioPsoas transfer @ the age 4-5 yrs.
• If Coxa Valga > 1500 , it is best to correct deformity
to FNS angle 1100 before IlioPsos transfer.
> 2yeras • IlioPsoas transfer may be postponed & Stability of
hip monitored periodically +/- without Adductor
tenotomy.
• When Coxa Valga exceed 1600 and femoral head
starts to subluxate laterally
• In patient <6yrs, PFVDR to reduce FNS angle to 1050.
• In older >6yrs pt. PFVDRO to reduce FNS angle to
0
28. PARALYSIS OF THE GLUTEUS MAXIMUS & MEDIUS
Treatment
Sharrards’ Posterior transfer of the iliopsoas
• Iliopsoas with lesser troch detached, Psoas &
Iliacus mobilzed, origin of ilacus freed fron
origin, hole made in Ilium.
• IlioPsoas is tendon passed from medial to
lateral through hole. IlioPsoas tendon with
lessor troch secured to greater troch with
screw.
Weisinger modification: IlioPsoas muscle +
Tendon redirected lateraly through Sciatic
Notch inserted into great troc.
30. COBRA PLATE COMPRESSION
DISPLACEMENT OSTEOTOMY AD
DHS
HIP ARTHRODESIS
Position of hip fusion:
• Neutral abduction,
• External rotation of 0-300,
• Flexion 20-250
• Avoid abduction and
internal rotation
• This position is design to
minimize excessive lumbar
spine motion and opposite
knee motion which
32. QUADRICEPS FEMORIS PARALYSIS
QUADRICEPS GAIT & GENU RECURVATUM
• With a Weak Quads, knee is stablized with Hand Knee gait
• Mild genu recurvatum: adequate strong triceps surae &
hamstring muscles. knee is stabilized by locking in
hyperextension.
• During stance phase, quadriceps weakness is compensated
by tilting trunk and center of gravity of the body forward.
Rx with bracing is sufficient to walk satisfactorily.
• The only functional disabilities are difficulty climbing steps
and running.
• Decompensation further leads to gross recurvatum
deformity with bony changes
Source: Benjamin Josef. J.
Children Orthop. Oct 2015.
Source: Bhatti ZHC March 2021.
33. QUADRICEPS FEMORIS PARALYSIS
Gross Genu Recurvatum
MUSCLE TRANSFER
Muscle transfer to restore knee extension
power:
Common:
• Biceps femoris
• Semitendinosus
Other:
• Sartorius
• Tensor fasciae latae, and
• Adductor longus
Transfer of biceps femoris and semitendinosus tendon
34. QUADRICEPS FEMORIS PARALYSIS
GENU RECURVATUM
with STRUCTURAL CHANGES
• With Wt bearing & Gravity, the proximal tibial shaft
bows posteriorly
• Partial subluxation of the tibia may gradually occur.
• There is frequently calcaneus deformity of foot.
Treatment
• Closing wedge osteotomy for genu recurvatum.
• Triple tenodesis for genu recurvatum
• Other Surgical methods of correcting genu recurvatum.
A . Irwin's technique. B. Modified dome osteotomy. C.
Open-up wedge osteotmy
A. Closing wedge tiabial osteotom
B. Prox. Tibail bow / sloped plateua
C. 5 months after CWO
Source: Campbel operative orthop
36. FFC KNEE
Cause: Contracture of the ITB & Quads Palsy with normal /
partialy paralysed hamstrings.
Treatment:
• <15 - 200 FFC Knee– Posterior hamstring lengthening and
capsulotomy.
• 20-700 FFC Knee – Supracondylar extension osteotomy of the
femur
• >700 FFC Knee – Division of ITB and hamstring tendons,
combined with posterior capsulotomy.
Post OP Care: Skeletal traction Two pins in the distal tibia &
proximal tibia to avoid posterior subluxation of the tibia.
• Long-term use of a long-leg brace for bone remodeling.
Supracondylar extension osteotomy
of the femur.
Source: Benjamin Josef. J. Children Orthop. Oct 2015.
Sourse: Campbell Op Orthop
37. FLAIL KNEE
• Knee is unstable in all directions.
• Insufficient muscle power for tendon transfer to
overcome this instability.
Treatment
• Locking knee long leg knee brace.
• Knee arthrodesis
39. PEABODY’S CLASSIFCATION
INVERTOR EXTENSOR INSUFFICIENCY
Limited extensor invertor insufficiency
• Tibialis Anterior muscle paralysis produces slowly
progressive deformity
1. Equinus 2. Cavus 3. Varying degree of plano
valgus
Treatment:
• Redistributed of muscle power by transferring the
EHL tendon to base of 1st metatarsal + plantar
fasciotomy.
40. PEABODY’S CLASSIFCATION
INVERTOR EXTENSOR INSUFFICIENCY
TYPE A: Gross extensor & invertor insufficiency
• Paralysis of Extensors of toes and Tibialis Anterior,
with relatively normal Tibialis Posterior muscle.
• Produces - Equinus – later Equino Valgus
Treatment:
• Transfer of Peroneus Longus to dorsum of 1st cunieform bone.
• Talo-navicular arthrodesis is combined if deformity is fixed.
TYPE B: Paralysis of both Tibialis Anterior & Tibialis Posterior and Toe
extensors
• Rx: Transfer of both Peroneals to dorsum of foot. Source: Benjamin Josef.
J. Children Orthop. Oct 2015.
41. PEABODY’S CLASSIFCATION
EVERTORS INSUFFICIENCY
Evertor insufficiency
Paralysis of Peroneal muscles producing:
• Varus foot
• Deformity produce Slight to moderate
impairment
Treatment:
• Mild Deformity: Transfer of EHL to base of 5th
MT.
• Severe Deformity: Tibialis anterior to cuboid, EHL
Source: M. Sai Krishna.
42. TALIPES EQUINUS
Mechanism:
• Planter flexors are stronger than
dorsiflexors and tight Tendo
Achilles.
• If associated lateral imbalance is
there Equinuovarus or
Equinovalgus may result.
43. Rx. TALIPES EQUINUS
• 1. No intervention : mild equinus
• 2. Conservative management: exercises, serial casting,
orthosis and molded shoe wear.
• 3 .SURGICAL MANAGEMENT:
a) soft tissue procedures to release contracture
b) bony procedures
• Cambells Posterior bone block operation
• Lambrinudi procedure
• Pantalar arthrodesis
44. CAMPBELLS POSTERIOR BONE BLOCK
Usually combined with Triple arthrodesis
To correct lateral instability as well
• Mechanical block created, posterior to talus , superior to
calcaneum, to impinge on posterior lip of distal tibia, to
prevent planter flexion.
• Dorsiflexion preserved
• COMPLICATIONS: Recurrence, OA, Talus flatening & Ankylosis of ankle
A. Bruce Gil
45. LAMBRINUDI PROCEDURE
TaloNavicular & CalneoCuboid Fussion.
• Wedge of bone resected from distal &
planter surface of Talus.
• Remaining Talus Wedged in trough
made in Navicular
• CalcaneoCuboid cartilage removed.
• Forefoot realinged in corrected position
COMPLICATIONS:
• Residual deformity, Degenerative Arthritis
• Pseudoarthrosis, Talus Flatening
46. TALIPES EQUINUS VARUS_TEV
Mechanism:
• Weakness of Peroneals & Tibialis anterior &
Normal triceps surae: (Lateral Imbalance).
• Equinus produced increases mechanical
advantage of TP which in turn encourages the
fixation of hind foot inversion and forefoot
adduction and supination.
• Cavus and clawing develop when toe
extensors help to dorsiflex the ankle.
Source: M. Sai Krishna.
47. Rx. TALIPES EQUINUS VARUS_TEV
Young
children
4-8 yrs
Early stage Coservative Rx. Operative Rx.
Double bar brace with ankle
stop •
Stretching of plantar fascia
and posterior ankle structure
with wedging casting •
• TA lengthening &
Posterior capsulotomy
• Anterior transfer of tibialis
posterior or Split transfer
of tibialis anterior to
insertion of p.brevis (if
tibialis posterior is weak)
• Anterior transfer of medial
half of tendo-calcaneous(
Caldwell)
48. Rx. TALIPES EQUINUS VARUS _ TEV
Children
>8yrs:
Procedure
• Steindlers fasciotomy
• Triple arthrodesis
• Anterior transfer of tibialis
posterior
• Modified jones procedure
• When TP is weak TA is
transferred laterally to midline.
49. TRIPLE ARTHRODESIS
A. Oblique incision over
sinus tarsi to expose
Subtalar, TaloNavicular &
Calcanio cuboid Jts.
B. Cartilage & Cortical
bone removed,
appropriate wedges are
removes as nesseccary
C. Wedges necessary for
valgus deformity.
D. Wedges necessary for
varus defformity
51. TALIPES EQUINO VALGUS
Cause:
• Tibialis anterior and Tibialis posterior are weak and Peroneal longus and brevis are
strong and the triceps sure is strong and contracted. Triceps surae pulls the foot into
equinus and the Peroneals into valgus.
Skeletally immature – Treatment:
• Early Stage: Double bar brace with ankle stop • Shoe
with an arch support and medial heel wedge •
Repeated stretching and wedging cast
• TA lengthening
• Anterior transfer of peroneals
• Grice and Green arthrodesis Subtalar arthrodesis
and Anterior transfer of Peroneals Source: M. Sai Krishna. M.Pardhasaradhi
52. TALIPES EQUINO VALGUS
Skeletally mature :- Treatment:
•TA lengthening
•Triple arthrodesis followed by
anterior transfer of peroneals
•Modified Jones
Source: Benjamin Joseh
54. TALIPES CAVO-VARUS
Cause:
• Imbalance of extrinsic muscles or by unopposed
short toe flexors and other intrinsic muscle
Rx.
• Plantar fasciotomy , Release of intrinsic muscles
and resecting motor branch of medial and lateral
plantar nerves before tendon surgery
• Peroneus longus is transferred to the base of the
second MT
• EHL is transferred to the neck of neck of 1st MT
Source: M. Sai Krishna. M.Pardhasaradhi
55. TALIPES CALCANEUS
Cause: Triceps Surae Weaknes with unopposed action of dorsiflexors
Rx.
• Plantar fasciotomy: Intrinsic muscle release before tendon transfer
• Transfer of TP and PL and FHL tendons to calcaneous.
• Green and Grice
• Posterior transfer of Tibialis Anterior ( Peabody )
• When EHL and EDL strength is good, both Tibials and Peroneials
can be transferred posteriorly and EHL, EDL transferred proximally
to act as dorsiflexors of ankle.
• If adequate muscles are not available:Tenodesis of
Tendoachiles to fibula is done ( Westin )
Source: M. Sai Krishna. i
57. FLAIL FOOT
Cause:
• All muscles paralysed distal to the knee. Equinus
deformity results because passive plantar flexion and
Cavoequinus deformity because – intrinsic muscle may
retain some function.
Treatment:
• Radical plantar release
• Tenodesis
• In older pt mid foot wedge resection
may be required
• ANKLE ARTHRODESIS
58. CLAW TOES
Deformity: Hyperextension of MTP and flexion
of IP
Cause: long toe extensors substitutes
dorsiflexion of ankle
Treatment: For lateral 4 toes :
• Procedure I : division of extensor tendon by z-plasty
incision,dorsal capsulotomy of MTP joint.
• Procedure 2: Girdlestone- Taylor tendon transfer
Dorsolateral incision. Divide the long flexor tendon
and suture them to lateral side of proximal phalanx
to extensor expansion.
Girdlestone- Taylor tendon transfer
59. CLAW TOES
Girdlestone- Taylor tendon transfer Dorsolateral
incision. Divide the long flexor tendon and suture
them to lateral side of proximal phalanx to extensor
expansion.
60. CLAW TOES: BIG TOE
Rx. Great Toe: Dickson and
Diveley procedure.
• EHL tendon is divided
proximal to IP joint &
transferred to Flexor Hallucis
Longus tendon to taut flexor
tendons.
• Distal part of extensor tendon
sutured to soft tissues on
dorsum of proximal phalanx to
assist maintain opposition of
raw surfaces of IP joint.
• Arthrodesis of interphalangeal
joint.
Source: Slideshare.net
61. CLAW TOES: BIG TOE
Modified Jone’s procedure:
• Division of EHL proximal to IP
joint.
• Proximal slip fixed to neck of
1st metatarsal.
• Distal slip fixed to soft tissues.
• Arthrodesis of IP joint by K
wire fixation
Source: Slideshare.net
62. CAVUS AND CLAW FOOT
Primary deformity :
• Forefoot Equinus resulting in clawing of toes.
• Mild Clawing disappear if mild cavus of short duration is
corrected.
Sever Deformity:
• In severe cavus large callosities or even ulcerations may
develop beneath the metatarsal heads.
• Clawing may lead to dorsal dislocation of MTP joint.
• In severe cases all plantar stuctures may contract.
63. Rx. CAVUS AND CLAW FOOT
Rx.
Mild Deformity:
• Conservative : metatarsal bar on the shoe, metatarsal pads.
• Surgical measures:
oDivision PL tendon and imbricate to PB, considering that the
deformity is due to imbalance of Tibialis Anterior and PL.
oArthrodesis of all IP joints, considering that clawing is caused by
disturbance of function of intrinsic muscles of foot. mild cavus
with clawing
64. Rx. CAVUS AND CLAW FOOT
Rx.
Moderate Deformity:
• Young children :
o Steindler’s fasciotomy
• Older children :
o Dwyers calcaneal
osteotomy.
o Japas V osteotomy
Steindler’s fasciotomy • stripping of fat and muscles from both
superficial and deep surfaces. • Transverse division of fascia close to
calcaneal attachment. • Release of long plantar ligament extending
from calcaneus to cuboid.
65. CAVUS AND CLAW FOOT
SURGICAL PROCEDURES
Moderate / Sever Deformity
Cole’s Anterior wedge
osteotomy:
Indication:
Cavus without varus or calcaneus or
gross muscle imbalance.
Advantage : preserves mid tarsal and
sub-talar joints
Disadvantage: shortens the dorm of
foot.
Osteotomy of the navicular
and cuboid and defect is
closed by elevating the
forefoot.
66. CAVUS AND CLAW FOOT
SURGICAL PROCEDURES
Japas V osteotomy:
• Apex of “V” is proximal at highest point of
cavus
• Lateral limb extends to cuboid
• Medial limb through intermediate
cuneiform to medial border of foot.
• No bone is excised
• Proximal border of distal fragment is
pressed plantarwards, while metatarsal
heads are elevated correcting the
deformity.
67. CAVUS AND CLAW FOOT
SURGICAL PROCEDURES
Hibb’s operation:
• EDL tendons is divided
and proximal end is
inserted to 3rd cuneiform.
• EHL tendon is divided
and fixed to neck of 1st
metatarsal.
• Interphalangeal joint
arthrodesis.
68. DORSAL BUNION
• Shaft of 1st MT is dorsiflexed and great toe is plantar flexed
resulting in prominent head of 1st metatarsal. If severe may
result in subluxation of MTP joint.
Pathogenesis:
• Imbalance between Tib. A and PL: normally TA raises the 1st
cuneiform and 1st MT and PL opposes this action. Unopposed
action of Tib. A causes this deformity.
• Weakness of Anterior and lateral compartment muscles.
unopposed action of posterior compartment muscles causes
excessive plantar flexion of great toe.
Rx.
Transfer of PL & Midline transfer of TA to 3rd Cuniform
Before the transfer of PL, the effect of its loss on 1st MT must be considered. Every transfer of
PL should be accompanied with midline transfer of TA to 3rd cuneform.
Source: Slideshare.net
69. DORSAL BUNION
Rx: LAPIDUS TECHNIQUE
• Wedge of bone is removed from metatarso-
cuneform and naviculo-cuneform joint. •
• If Tib. A is overactive, transfer it to 2nd or 3rd
cuneiform. •
• FHL is detached and brought dorsally and
attached to 1st metatarsal, converting it into a
plantar flexor of metatarsal rather than great
toe.
• Subcutaneous plantar tenotomy
• capsulotomy of 1st MTP joint.
Source: Slideshare.net
70. DORSAL BUNIONUE
Hammond Techniq
• Any deforming tendon
except the FHL is divided
and transferred to dorsum
of foot to correct MT
displacement.
• Fusion of joint.
73. ZOOM.COM
Anisuddin Bhatti Is Inviting You To A Scheduled Zoom Meeting
On 18th April 2021@ 12:00 -13:00
Topic: Post Polio Residual Paralysis & Deformities Par t Iii. Upper Limb
Time: This Is A Recurring Meeting Meet Anytime
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74. CASE DISCUSSION
19 Years Old Female Known Case Of Poliomyelitis
H/O: Contractures and deformity of right leg for the last 17 years.
On examination:
Right knee FFD 90 degree
Hip FFD 90 degree
Abduction contracture 45 degree
Right leg Genu Valgum
LLD of ~11 cm
Distal neurovascular intact
Walks with Gluteal crutch
Xray revealed: Hypoplastic limb, contractures.
No dislocation.
Demand:
Release of contracture and brace able straight leg
Walk with the straight leg
75. DECISION MAKING CHART
Patient # xx yrs. FM, PPRP_xx yr. Walks xxxxxxx
AREA DEFORMITY DEFICIT SUBSTITUTE REMARKS
HIP
KNEE
ANKLE & FOOT
LLD
DIAGNOSIS
DEMAND
DECISION
76. Patient: 19 yrs. FM, PPRP_17 yr. Walks with gluteal Cructh, projected knee forward
Area Deformity Deficit Substitute Remarks
Hip FFC_Flx 900
Abduction 450
Glutie 3+, Abd 3+
Flexors 4
Iliopsoa, Rectus
abdominus
Not required, nearly
balanced power
Knee 900
Genu valgum
Quad 3+ Semitendinosis Not required, nearly
balanced power
Ankle & Foot None Controled All avaible Not required,
balanced power. No
deformity
LLD 11 cm, walks with gluteal crutch with Ugly posture of 90-90 HK contracture
Diagnosis Postural contracture Hip & Knee FFC 90-90, LLD 11cm, Balanced ankle power & no
deformity
Demand Elimination of contractures, Potural Stability & lengthening.
Decision Release of contratures: Hip_Campbells release + Knee_Z-Lengthening + Posterior
Capsulotomy. Followed by Shoe elevation
Stage 2: Limb Lengthening