PALPITATION
A. Definition.
   Abnormal subjective awareness of the heart
    beat.

   Thumping, pounding, fluttering, jumping, racing,
    skipping)

   But patient may describe palpitation as a feeling
    of breathlessness, excitement, fright etc.

   Palpitation ≠ arrythmias
B. Causes.
   Palpitation may be due to   Rapid heart beat or
                                Slow heart beat or
                                Irregular heart beat.
   Palpitation may be due to
       Primary cardiac disease (Acute or Chronic) or
       Secondary effect on the heart (Systemic disease
       or Drugs)
Common causes are

  (1) Anxiety, Exercise, hyperthyroidism
  (2) Drugs         (Sympathomimetics, Atropine)
  (3) Diet          (Tea, coffee, cola)
  (4) Nicotine(Smoking)
  (5) Abnormal rate
              Sinus tachycardia(100-160)
              Supraventricular tachycardia(160-220)
              Ventricular tachycardia
              Sinus Bradycardia
(6) Abnormal rhythms
            Atrial fibrillation
            Ventricular fibrillation
(7) Extrasystole
            Atrial extrasystole.
            Ventricular extrasystole
(8) Wolff-Parkinson-White (WPW)
(9) Forceful heart beat
            Aortic regurgitation.
Sinus Tachycardia .

 Heart rate = 100 bpm-160bpm

 Causes Fever           Thyrotoxicosis
        Anaemia         Phaeochromocytoma
        Anxiety         Carcinoid tumor
        Exercise        Heart failure
        Pregnancy       Drugs
Sinus bradycardia
Heart rate = < 60 bpm

Causes    Myocardial infarction
          Sick sinus syndrome
          Hypothermia
          Hypothyroidism
          Cholestatic jaundice
          Raised intracranial pressure
          Drugs (Digoxin, Beta blockers.)

Treatment Atropine
Supraventricular Tachycardia (SVT)
Ventricular Tachycardia (VT)

 Heart rate (140-220)
 Causes         Coronary artery disease.
                Valvular heart disease.
                Myocarditis.
                Cardiomyopathy.
                Drugs.
 SVT usually benign.
 VT always malignant and need urgent treatment.
    DC cardiovertion
    Lignocaine, Mexilatine, Flecainide, K, Mg
Paroxysmal atrial tachycardia (PAT).
 Heart rate is usually about 140-220 during attack.

 Clinical feature
     Asymptomatic.
     Symptomatic      Palpitation        Dyspnoea
                      Cheat pain         Fainting
          Sudden onset and sudden disappear.
          Polyuria (due to Atrial Natriuretic peptide)

 ECG      Normal or WPW syndrome during intervals.
          SVT during attack
Treatment
 Carotid sinus massage.

 Increases vagal tone by induced vomiting, Valsalva
  manoeuvre.
 Inj IV Adenosine or IV Verapamil

 Alternative drugs    Beta-blockers.
                       Disopyramide.
                       Digoxin.
                       DC cardioversion.

Prevention
 Above drugs

 Radio frequency ablation.
Atrial Fibrillation.

Causes.
 Coronary artery disease   Alcohol
 Valvular heart disease    Hypertension
 Cardiomyopathy            Pulmonary embolism
 Pericarditis              Congenital heart disease
 Thyrotoxicosis            Pneumonia
 Idiopathic                Bronchial carcinoma
ECG         No P wave.
            Fibrillatory wave.
            Normal but amplitude of QRS complex.

Treatment        Digoxin
                 Amioderone
                 Cardioversion
                 β blocker
                 Verapamil

                 Antiplatelets
Extrasystoles
Premature beats or premature contractions.

  a. Atrial extrasystole.
  b. Ventricular extrasystole.

Clinical features
      Asymptomatic.
      Symptomatic.      Palpitation
                        Irregular beats
                        Missed beat or Strong beat.
                        heart sound have missed or
                              extra beats.
ECG
AE Normal QRS complex with preceding
 abnormal P wave.
VE Broad and bizarre QRS without preceding
    P wave.
    Couplet   2 successive ectopic beat.
    Triplet   3 successive ectopic beat.
    Bigeminy Alternate sinus and ectopic beat.

AE   No treated
VE   Treatment of underlying diseases
VE
   Healthy
    more prominent at rest
    tend to disappear with exercise
    treatment is unnecessary
    low dose β blocker may reduce anxiety and palpitation

   Heart disease During AMI
                  Heart failure
                  Digoxin toxicity
                  Mitral valve prolapse (MVP)
Wolff-Parkinson-White (WPW) syndrome
Pre-excitation syndrome

   Presence of extra conducting tissue (bundle of Kent).
   ECG      1. Short PR interval (< 0.1 sec.)
             2. Delta wave.
             3. Wide QRS complex (> 0.1 sec.)
   Complications Paroxysmal atrial tachycardia(PAT).
                   Atrial fibrillation (Af).
   Trans-venous radiofrequency catheter ablation
   Amiodarone, Disopyramide, Flecainide
   Contrindication – Digoxin, verapamil
C. Diagnosis


   Careful and thorough history is important.

   Definitive diagnosis may be obtained by doing
    ECG during attacks or ambulatory ECG
    monitoring.
The evaluation of patient with palpitation.
   Continuous or intermittent?
   Regular or irregular heartbeat?
   Approximate heart rate?
   Discrete attacks or not? If yes, is the onset
    abrupt? Or how do attacks terminate?
   Any associated symptoms? Eg. Chest pain,
    lightheadedness, polyuria.
   Any precipitating factors? Eg. Exercise, alcohol.
   Evidence of structural heart disease? Eg.
    Coronary heart disease, valvular heart disease.
Regular heart beat



      NO
                                       Yes



Ectopics
Atrial fibrillation             Discrete attacks



                       Yes                         NO



                       SVT                   Sinus tachycardia
                                             High stroke volume
                                                     Anaemia
                                                     Anxiety
                                                     AR
Palpitation

Palpitation

  • 1.
  • 2.
    A. Definition.  Abnormal subjective awareness of the heart beat.  Thumping, pounding, fluttering, jumping, racing, skipping)  But patient may describe palpitation as a feeling of breathlessness, excitement, fright etc.  Palpitation ≠ arrythmias
  • 3.
    B. Causes.  Palpitation may be due to Rapid heart beat or Slow heart beat or Irregular heart beat.  Palpitation may be due to Primary cardiac disease (Acute or Chronic) or Secondary effect on the heart (Systemic disease or Drugs)
  • 4.
    Common causes are (1) Anxiety, Exercise, hyperthyroidism (2) Drugs (Sympathomimetics, Atropine) (3) Diet (Tea, coffee, cola) (4) Nicotine(Smoking) (5) Abnormal rate Sinus tachycardia(100-160) Supraventricular tachycardia(160-220) Ventricular tachycardia Sinus Bradycardia
  • 5.
    (6) Abnormal rhythms Atrial fibrillation Ventricular fibrillation (7) Extrasystole Atrial extrasystole. Ventricular extrasystole (8) Wolff-Parkinson-White (WPW) (9) Forceful heart beat Aortic regurgitation.
  • 6.
    Sinus Tachycardia . Heart rate = 100 bpm-160bpm Causes Fever Thyrotoxicosis Anaemia Phaeochromocytoma Anxiety Carcinoid tumor Exercise Heart failure Pregnancy Drugs
  • 7.
    Sinus bradycardia Heart rate= < 60 bpm Causes Myocardial infarction Sick sinus syndrome Hypothermia Hypothyroidism Cholestatic jaundice Raised intracranial pressure Drugs (Digoxin, Beta blockers.) Treatment Atropine
  • 8.
    Supraventricular Tachycardia (SVT) VentricularTachycardia (VT) Heart rate (140-220) Causes Coronary artery disease. Valvular heart disease. Myocarditis. Cardiomyopathy. Drugs. SVT usually benign. VT always malignant and need urgent treatment. DC cardiovertion Lignocaine, Mexilatine, Flecainide, K, Mg
  • 9.
    Paroxysmal atrial tachycardia(PAT). Heart rate is usually about 140-220 during attack. Clinical feature Asymptomatic. Symptomatic Palpitation Dyspnoea Cheat pain Fainting Sudden onset and sudden disappear. Polyuria (due to Atrial Natriuretic peptide) ECG Normal or WPW syndrome during intervals. SVT during attack
  • 10.
    Treatment  Carotid sinusmassage.  Increases vagal tone by induced vomiting, Valsalva manoeuvre.  Inj IV Adenosine or IV Verapamil  Alternative drugs Beta-blockers. Disopyramide. Digoxin. DC cardioversion. Prevention  Above drugs  Radio frequency ablation.
  • 11.
    Atrial Fibrillation. Causes. Coronaryartery disease Alcohol Valvular heart disease Hypertension Cardiomyopathy Pulmonary embolism Pericarditis Congenital heart disease Thyrotoxicosis Pneumonia Idiopathic Bronchial carcinoma
  • 12.
    ECG No P wave. Fibrillatory wave. Normal but amplitude of QRS complex. Treatment Digoxin Amioderone Cardioversion β blocker Verapamil Antiplatelets
  • 13.
    Extrasystoles Premature beats orpremature contractions. a. Atrial extrasystole. b. Ventricular extrasystole. Clinical features Asymptomatic. Symptomatic. Palpitation Irregular beats Missed beat or Strong beat. heart sound have missed or extra beats.
  • 14.
    ECG AE Normal QRScomplex with preceding abnormal P wave. VE Broad and bizarre QRS without preceding P wave. Couplet 2 successive ectopic beat. Triplet 3 successive ectopic beat. Bigeminy Alternate sinus and ectopic beat. AE No treated VE Treatment of underlying diseases
  • 15.
    VE  Healthy more prominent at rest tend to disappear with exercise treatment is unnecessary low dose β blocker may reduce anxiety and palpitation  Heart disease During AMI Heart failure Digoxin toxicity Mitral valve prolapse (MVP)
  • 16.
    Wolff-Parkinson-White (WPW) syndrome Pre-excitationsyndrome  Presence of extra conducting tissue (bundle of Kent).  ECG 1. Short PR interval (< 0.1 sec.) 2. Delta wave. 3. Wide QRS complex (> 0.1 sec.)  Complications Paroxysmal atrial tachycardia(PAT). Atrial fibrillation (Af).  Trans-venous radiofrequency catheter ablation  Amiodarone, Disopyramide, Flecainide  Contrindication – Digoxin, verapamil
  • 17.
    C. Diagnosis  Careful and thorough history is important.  Definitive diagnosis may be obtained by doing ECG during attacks or ambulatory ECG monitoring.
  • 18.
    The evaluation ofpatient with palpitation.  Continuous or intermittent?  Regular or irregular heartbeat?  Approximate heart rate?  Discrete attacks or not? If yes, is the onset abrupt? Or how do attacks terminate?  Any associated symptoms? Eg. Chest pain, lightheadedness, polyuria.  Any precipitating factors? Eg. Exercise, alcohol.  Evidence of structural heart disease? Eg. Coronary heart disease, valvular heart disease.
  • 19.
    Regular heart beat NO Yes Ectopics Atrial fibrillation Discrete attacks Yes NO SVT Sinus tachycardia High stroke volume Anaemia Anxiety AR