examination of eye, Examination by ophthalmoscope,
assessment of the functions of eye, . PUPILLARY RESPONSE, FUNCTIONAL EXAMINATION, test for Focusing power, confrontation test, Colour sense test and visual acuity testing procedure etc
complete information about the retinal detachment , types, , symptoms , sign, etiology, causes, diagnosis, complications, medical management, nursing management, home care, patient teaching. nursing reserch.
complete information about the retinal detachment , types, , symptoms , sign, etiology, causes, diagnosis, complications, medical management, nursing management, home care, patient teaching. nursing reserch.
Short eye Examination components - that will tell you the main headings of an eye examination in trauma victims.
drawaneeshkatiyar@gmail.com - for further communication.
MALARIA
It is an infectious disease of humans caused by parasitis protozoans belonging to the genus plasmodium.
It is endemic in most parts of India and other tropical countries.
As per WHO, malaria causes one death every minute globally and about 40,000 annual deaths in India.
The disease is transmitted by the bite of an infected female Anopheles mosquito.
Four species of protozoa plasmodium can cause malaria which are P. falciparum, P. vivax, P. ovale and P. malariae.
INTRODUCTION
These are the drugs which are used for the treatment, prophylaxis and prevention of relapses of malaria.
The treatment of malaria is available since 17 century. During those times, the bark of Cinchona tree was used in the crude form.
Later in 1820, quinine was isolated from the bark.
Since 1920, quinine and other drugs are commercially available in the market
OBJECTIVES IN USE OF ANTIMALARIAL DRUGS
The various objectives are:
To prevent clinical attack of malaria.
To treat clinical attack of malaria.
To completely eradicate the parasite from the patient’s body.
To cut down human to mosquito transmission.
THERAPEUTIC CLASSIFICATION
1. CAUSAL PROPHYLACTICS: (Destroy parasite in liver cells and prevent invasion of erythrocytes)
e.g. primaquine, pyrimethamine
2.BLOOD SCHIZONTOCIDES SUPPRESIVES (destroy parasites in the RBC and terminate clinical attacks of malaria): e.g. chloroquine, quinine, mefloquine, halofantrine, pyrimethamine
3. TISSUE SCHIZONTOCIDES used to prevent relapse: act vivax and P. ovale that produce replapses. E.g. primaquine
4. GAMETOCIDAL DRUGS: primaquine, chloroquine, quinine.
1. CHLOROQUINE
It acts as erythrocytic schizontocide against all species of plasmodia.
The parasite disappears from peripheral blood in 1-3 days. It control the clinical attacks of malaria within 1-2 days.
It doesn’t have any gametocidal activity.
It is bitter in taste, so patient should be advised ‘not to chew the tablet’ it is used for the treatment of malaria during pregnancy: no teratogenic effects have been reported.
MECHANISM OF ACTION
Its gets concentrated in the infected RBCs and then is actively taken up by the susceptible plasmodia.
The chloroquine binds to the heme and forms chloroquine heme complex.
Complex inhibits the formation of hemozoin and also damages the Plasmodium memberane
PHARMACOKINETICS
It is well absorbed orally.
50% of the drug is plasma protein bound, gets concentrated in liver, spleen, kidneys, lungs, skin and leukocytes.
The plasma half life is 3-10 days, whereas the terminal half life is 1-2 months. On prolonged use, it gets accumulated selectively in the retina and causes ocular toxicity.
It is partially metabolized in liver and slowly excreted in urine.
INDICATIONS ADVERSE EFFECTS
Clinical drug of choice for malaria.
Extraintestinal amoebiasis.
Rheumatoid arthritis
Infectious mononucleosis.
Mil
These are synthetic antimicrobial having a quinolones structure.
These are active against most of the gram negative bacteria.
In 1960s the first membered of this group, Nalidixic acid, was introduced .
Flouroquinolones have:
High potency
Expanded antimicrobial spectrum
Better tissue penetration
Good tolerability profile
Very low resistance development
1. NALIDIXIC ACID
It was the first member in quinolones. It was active against gram negative bacteria. It acts by inhibiting bacterial DNA gyrase. It is bacterial in nature.
It is given orally. It attains good concentration in gut, lumen, hence useful in diarrhea.
NALIDIXIC ACID CONTD…
It is most commonly seen in children. And causes neurological toxicity present as headache, drowsiness and vertigo and contraindicated in infants and G6PD deficient patients.
It is given in a dose of 0.5-1g TDS/QID
2. FLUOROQUINOLONES
Fluoroquinolones are quinolones antimicrobials having one or more fluorine substituitions.
The first generation fluoroquinolones have one fluoro substitution and were developed in 1980s.
The second generation fluoroquinolones have additional fluoroquinolones have additional fluoro substitution, which extended the antimicrobial activity and were developed in 1990s.
MECHANISM OF ACTION
The fluoroquinolones inhibits the enzyme DNA gyrase in Gram-negative micro-organism and topoisomerase IV in gram positive micro-organism. This lead to the bactericidal effects of fluoroquinolones.
RESISTANCE
The resistance to fluoroquinolones develops when the bacteria produce a DNA gyrase or topoisomerase IV which have reduced affinity for fluoroquinolones or the bacteria produce efflux pumps across bacterial memberane which shunt out the fluoroquinolones from the bacterial cells.
CLASSIFICATION
FIRST GENERATION FLOUROQUINOLONES :
Norfloxacin
Ofloxacin
Ciprofloxacin
Pefloxacin
SECOND GENERATION FLUOROQUINOLONES:
Levofloxacin
Moxifloxacin
Lomefloxacin
Gemifloxacin
Sparfloxacin
Prulifloxacin
PHARMACOKINETICS
These are given both by oral and intravenous route.
These have good absorption, when given empty stomach and food delays the absorption. These drugs have good tissue penetrability.
These are excreted in urine by glomerular filtration as well as tubular secretion.
INDICATIONS
Bacterial gastroenteritis
Typhoid fever
UTI
Gonorrhoea
Chancroid
Bone, Soft tissue and gynaecological infections
Respiratory infections
Tuberculosis
Gram negative septicemia and meningitis
Conjuctivitis
COMMON ADVERSE EFFECTS
GI SYSTEM: Nausea, vomiting, bad taste and anorexia
CNS: Headache, anxiety, insomnia, restlessness and impairment of concentrations.
Skin: Rash, photosensitivity
Contraindicated in pregnancy.
They should be used in caution in children as a few cases of joint pain and swelling have been reported and a risk of cartilage damage is suspended.
FIRST GENERATION FLUROQUINOLONES
SECOND GENERATION FLUROQUINOLONES
DRUG INTERACTIONS
Antacids decrease the absorption
INTRODUCTION
Sulfonamides were the first effective, synthetic antibacterial agents to be used systemically in man.
They were introduced by Domagk in 1935 and in the next few years several of them were synthesized and widely used. Currently their role in therapeutics is limited because of their toxicity, development of resistance availability of safer drugs.
CLASSIFICATION
Short acting: Sulfisoxazole, Sulfadiazine
Intermediate acting: Sulfamethoxazole
Long-acting: Sulfamethoxypyridazine, sulfadoxine
Poorly absorbed: Sulfasalazine
Topical: Sulfacetamide, mefedine
Silver sulfadiazine.
ANTIBACTERIAL SPECTRUM
It is wide spectrum antibiotic.
It inhibits many gram positive and some gram negative bacteria including streptococci, H. influenza, Norcardia, E. coli, proteus, V. cholerae, some stains of staphylococci, gonococci, memingococci and pneumococci.
They are also effective against Chlamydia, plasmodium falciparum and toxoplasma gondii.
MECHANISM OF ACTION
PABA (Para-aminobenzoic acid)
Folic acid synthesis
DIHYDROFOLIC ACID
Bacteria synthesize their own folic acid from PABA with the help of the enzyme folic acid synthetase. Sulfonamides are structurally similar to PABA and competitively inhibiting the enzymes folic acid synthetase.
They inhibit the enzyme folic acid synthase so folic acid is not synthesized (which is essential bacterial growth).
PHARMACOKINETICS
Sulfonamides are well absorbed, extensively bound to plasma proteins and are well distributed to all tissues.
They are metabolized in the liver and excreted in urine.
They can cross placental barriers.
COMMON USES
SYSTEMIC USES : sulfamethoxazole is used in combination with cotrimoxazolein many bacterial infections.
It is the drug of choice in pneumocystitis in AIDS patient.
Treatment of nocardiosis, toxoplasmosis, ulcerative colitis and rheumatoid arthritis.
TOPICAL USES: ocular sulfacetamide sodium is used in trachoma/inclusion conjuctivitis.
Topical silver sulfadiazine is used for preventing infection on burn surfaces.
Mefinide is active in the presence of pus and against pseudomonas, clostridia which are not inhibited by topical sulfonamides.
USES
Because of the development of resistance and availability of better antimicrobials, which are more effective and less toxic, these are not commonly used now except in a few cases:
UTI
NOCARDIOSIS
TOXOPLASMOSIS
TRACHOMA AND INCLUSION CONJUCTIVITIS
MALARIA
TOPICAL
ULCERATIVE COLLITIS
Contraindication & Precautions: Children younger than 2yrs, Pregnant and breast feeding mother, Renal and hepatic diseases, Hypersensitivity to sulphonamides drug.
Adverse effect: Fever, Rash, Nausea/vomiting, Aplastic Anemia.
DRUG INTERACTIONS
Sulphonamides can increasing the blood thinning effect of warfarin, possibly leading to abnormal bleeding.
Increases blood level of potassium may occur when Sulfamethoxazole trimethoprim is combined with ACE inhibitors.
Su
INTRODUCTION
Erythromycin is the first member of group, and was isolated from a strain of Streptomyces erythreus in 1952.
Rest drugs are semi-synthetic derivatives of erythromycin known as newer macrolides
Some other drugs are dirithromycin, oleandomycin and troleandomycin.
MECHANISM OF ACTION
Macrolide antibiotics are bacteriostatic agents and inhibit the protein synthesis by binding reversibly to 50s ribosomal subunit of sensitive microorganism and interfere with translocation step in the protein synthesis.
Gram positive bacteria's are 100 times more sensitive than gram negative bacteria's by these drugs.
MECHANISM OF ACTION
It is bacteriostatic at low concentration & bactericidal at high concentration
Bactericidal property depends on the concentration, organism concerned and its rate of multiplication
ANTI MICROBIAL SPECTRUM
It is narrow spectrum antibiotic. These antibiotics are more active against gram positive cocci and inactive against most of the aerobic and enteric gram negative bacilli.
In addition, Campylobacter, Legionella, Branhamella catarrhalis, G. vaginalis and Mycoplasma (which are not affected by pencillin are also highly susceptible to erythromycin)
ANTI MICROBIAL SPECTRUM
Moderately sensitive to H. influenza, B. pertussis, C. trachomatis, N. meningitidis and Rickettsiae
Ineffective against Enterobacteriaceae, other gram negative bacilli.
ERYTHROMYCIN
This drug is acid labile, given as enteric coated tablets. Poorly absorbed when given empty stomach and has poor tissue penetration.
DOSE: 250-500mg QID with half life of 1.5 hrs
Indications: a drug of choice in atypical pneumonia, whooping cough and cancroids and as an alternative to penicillin in streptococcal pharyngitis, tonsillitis, mastoiditis.
SIDE EFFECTS: Epigastric distress causing nausea, vomiting and diarrhea. Allergic reactions such as fever and skin eruption.
CLARITHROMYCIN
These drugs are acid stable, good absorption occurs when given empty stomach and has good tissue penetration.
Dose: 250-500mg BD with half life of 3-6 hrs at low dose and 3-9 hrs at high dose.
Indications: upper and lower RTI, sinusitis, otitis media, atypical pneumonia, skin infections. And H. pylori infection and first line drug in combination regimens in AIDS infection
Side effects: same as erythromycin but better gastric tolerance, reversible hearing loss at high doses.
AZYTHROMYCIN
These drugs are acid stable, good absorption occurs when given empty stomach and has good tissue penetration
Dose: 500mg OD with half life >50 hrs.
Indications: pharyngitis, tonsillitis, sinusitis, otitis media pneumonias, chronic bronchitis. In the prophylaxis and treatment of AIDS infections.
Side effects: nausea vomiting, diarrhea and abdominal pain.
ROXITHROMYCIN
These drugs are acid stable, good absorption occurs when given empty stomach and has good tissue penetration
DOSE: 150mg BD with half life of 12 hrs.
Indications: alternative to erythromycin for respiratory, skin
INTRODUCTION
Aminoglycosides are a class of antibiotics used mainly in the treatment of aerobic gram-negative bacilli infections, although they are also effective against other bacteria including Staphylococci and Mycobacterium tuberculosis.
They are often used in combination with other antibiotics.
Streptomycin – 1944
Actinomycetes – Streptomyces griseus
Bactericidal antibiotics which is interfere with protein synthesis
Used to treat aerobic Gram –ve bacteria
Exhibit ototoxicity and nephrotoxicity
MECHANISM OF ACTION
These drugs inhibit protein synthesis in the bacteria, there permeability is increased and cell contents leak out and death of cell occurs. These drugs leave bactericidal action.
CLINICAL USES
Gram –ve bacillary infection – Septicaemia, pelvic & abdominal sepsis
Bacterial endocarditis – enterococcal, streptococcal or staphylococcal infection of heart valves
Pneumonias, Tuberculosis
Tularemia
Plague, Brucellosis
Topical – Neomycin, Framycetin:- Infections of conjunctiva or external ear and also used it before surgery.
COMMON INDICATIONS OF AMINOGLYCOSIDES
Gram negative bacillary infections particularly septicemia, meningitis, UTI’s renal, pelvic and abdominal sepsis.
Bacterial endocarditis: usually gentamicin is preferred as a part of regimen.
Other infections such as tuberculosis, plague, brucellosis etc.
Topical uses: neomycin, framycetin and sisomicin are used for various topical infections.
NURSING IMPLICATIONS
The renal function should be regularly monitored.
Patients should be regularly enquired about any side effects.
Patients should be warned for not driving or operating the machinery.
Patient should be advised to take plenty of water during the course.
Monitor the sign and symptoms of hearing loss.
7-aminocephalosporanic acid has been modified by addition of different side chains to create a whole family of cephalosporin antibiotics.
these have been conventionally divided into 5 generations
Mechanism of action
All cephalosporins are bactericidal.
As penicillin it also inhibit the synthesis of bacterial cell wall and causing rapid cell lysis.
Inhibition of transpeptidation (Transpeptidase enzymes then cross-link the chains to provide strength to the cell wall and enable the bacterium to resist osmotic lysis)
Imperfect cell wall
Osmotic drive
Activation of autolysin enzymes
Lysis of bacteria
BACTERICIDAL
CLASSIFICATION
Based on
antimicrobial spectrum
Chronological sequence of development
Divided into generations.
First-generation agents
Cephalexin (O)
Cefadroxil (O)
Cefazolin (i.m, i.v)
Cefalothin (withdrawn)
Exhibit good activity against gram-positive bacteria but modest activity against gram negative organisms.
Most gram-positive cocci
Strepto,
Pneumo,
Methicillin sens. Staph. are susceptible to first-generation cephalosporins
Modest activity against E. coli, K. pneumoniae & Proteus mirabilis
Second-generation agents
Cefaclor (O)
Ceforanide
Cefuroxime (i.m , i.v)
Cefoprozil
Exhibit somewhat increased activity against gram negative organisms,
but much less active than third generation agents.
Less active against gram positive cocci & bacilli compared to first gen. drugs.
Use declined
Clinically replaced by 3rd & 4th generation drugs .
Third-generation agents
Cefotaxime
Ceftriaxone
Cefdinir
Cefibuten
Cefpodoxime
Ceftizoxime
Ceftazidime
Cefoperazone (withdrawn)
Highly augmented activity against gram-negative organisms
Less active than first generation agents against gram positive cocci & anaerobes.
All are highly resistant to β-lactamases from gram negative bacteria.
Some inhibit psuedomonas as well; ceftazidime, cefoperazone(withdrawn)
Some members of this group have enhanced ability to cross the blood-brain barrier eg. Ceftriaxone and are effective in treating meningitis caused by pneumococci, meningococci, H. influenzae and susceptible gram negative rods.
Fourth-generation agents
Cefpirome (im/iv)
Cefepime (iv)
Cefozopran (im)
Highly active against G –ve organisms
Similar to third gen drugs for g +ve bacteria
The fourth generation drugs comparable to third generation but more resistant to hydrolysis by β-lactamases.
Effective against bacterial infections resistant to earlier drugs
Fifth-generation agents
Ceftobiprole
Ceftaroline
Active against, g +ve cocci especially MRSA (Methicillin-resistant Staphylococcus aureus) It's tougher to treat than most strains of staphylococcus aureus -- or staph -- because it's resistant to some commonly used antibiotics.
penicillin resistant S. pneumoniae and enterococci
Adverse reactions
Pain after im injection
Thrombophlebitis of injected vein.
Diarrhoea more common with
oral Ceferadine
P/E Cefoperazone (Banned)
DEFINITION CHEMOTHERAPY & ANTIBIOTICS
CHEMOTHERAPY: Chemotherapy is the treatment of infections by substances which destroy or suppress bacteria and other microorganism. The substances / Agents used may natural synthetic or semi – synthetic in nature.
ANTIBIOTICS: An antibiotic is a chemical substance produced by microorganism which prevents the growth of other microorganism or kills the other microorganism. These are natural substances
CHEMOTHERAPY
It is a method of therapy of infectious disease and cancer with chemical agents – chemotherapeutic medicines
ANTIBIOTICS CLASSIFIED AS:
According to the mode of action on Bacteria:
According to the type of Bacteria:
According to the effectiveness against microorganism:
According to the mode of action on Bacteria:
Bacteriostatic: These antibiotics inhibit the growth & multiplication of Bacteria. Eg. Tetracycline, Chloramphenicol, Sulphonamides, Dapsone, Erythromycin, Clindamycin.
Bactericidal: These antibiotics destroy or kill all the Bacteria in the process of multiplication. Eg. Penicillin, Aminoglycosides, Cephalosporin, Fluoroquinolones, Rifampicin, Metronidazole etc.
According to the type of Bacteria:
Gram Positive: Some Antibiotics are effective mainly against Gram Positive Bacteria Eg. Penicillin.
Gram Negative: Some Antibiotics are effective mainly against Gram Negative Bacteria Eg. Streptomycin.
According to the effectiveness against microorganism:
Broad Spectrum: The Antibiotics which acts against wide range of microorganisms. Eg. Tetracycline.
Narrow Spectrum: These Antibiotics are useful against limited microorganisms. Eg. Erythromycin
Toxic Effects: Gastrointestinal irritation, Nausea, Vomiting and diarrhea may occur when given by mouth.
Skin sensitivity may develop with Penicillin or streptomycin causing rashes.
Serious toxic effect may occur due to streptomycin on the vestibular & auditory nerve causing vertigo & deafness
Drug Resistance: Many bacteria soon develops resistance to particular drug after a period of treatment, so that the bacteria will not respond to the same drug for example tubercle bacillus develops resistance to streptomycin quickly.
Super infection: The antibiotics given by mouth kill the normal bacteria inhibiting the alimentary canal and permits the over growth of other insensitive organisms which can cause serious complications. Eg. Fungus cause thrush which may go to the lungs with fatal results.
Hypersensitivity Reaction: Chemotherapeutic agents can cause Hypersensitivity reactions from mild rashes to serve anaphylactic shock. Eg. Penicillin & Sulphonamides.
Vitamin Deficiency: Alteration in vitamin formation and absorption from the bowel take place . So there is deficiency of Vitamin B complex and Vitamin K.
Anemia: In susceptible persons chloramphenicol may produce Aplastic anemia or agranulocytosis. (Action must be taken through proper history about previous drug reaction before administering penicillin sulphonamide and cephalosporin
definition of hydronephrosis,
causes and types of hydronephrosis
pathophysiology of hydronephrosis
clinical manifestation and diagnostic test for hydronephrosis
management
BIOGRAPHY OF FAYE GLENN ABDELLAH, AS AN EDUCATOR AND RESEARCHER, INFLUENCED FAYE ANDELLAH IN THE DEVELOPMENT HER OWN MODEL OF
NURSING, ABDELLAH’S TYPOLOGY OF 21 NURSING PROBLEMS, ASSUMPTION, CONCEPT, STEPS TO IDENTIFY THE CLIENT’S PROBLEM, 11 NURSING SKILLS, USE OF 21 PROBLEMS IN THE NURSING PROCESS AND LIMITATIONS
elimination, bowel elimination, physiology of elimination, process of bowel eliminaton factor impaired bowel, factors improve bowel elimination, alteration in bowel elimination, maintenance of bowel motility, assessment of bowel elimination, characteristics of feces, type of feces, methods for maintain the bowel elimination:- enemas, rectal suppositories and colostomies, types of colostomies, colostomy care
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The French Revolution, which began in 1789, was a period of radical social and political upheaval in France. It marked the decline of absolute monarchies, the rise of secular and democratic republics, and the eventual rise of Napoleon Bonaparte. This revolutionary period is crucial in understanding the transition from feudalism to modernity in Europe.
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2. EXAMINATION OF EYE
An examination of the eye includes an external
examination:-
• Examination by ophthalmoscope, and
• An assessment of the functions of eye.
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4. A. EXTERNAL EXAMINATION
• It consist of inspection of the eyelids,
surrounding tissue and palpebral fissure.
• Palpation of the orbital rim may also be
desirable, depending on the presenting signs
and symptoms
• The conjunctiva and sclera can be inspected
by having the individual look up, and shining a
light while retracting the upper or lower
eyelid.
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5. • The cornea and iris may be similarly
inspected.
• The anterior segment of the eye examined by
visual inspection.
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6. • Note the general appearance of the eyelids,
eyelashes and lacrimal apparatus. Observe
for:-
1. Redness around the eye
2. Discharge or crusting
3. Growths on eyes or eyelids
4. Excessive tearing
• Position and mobility can be observed by
having the patient rotate the yes, looking up,
down and each side.
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7. B. PUPILLARY RESPONSE
• Normal pupils are rounded, centrally placed
and generally equal in size (about 25 percent
of normal individuals have pupils slightly
unequal in size.)
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8. REACTION TO LIGHT:- Seat the patient in an
area with even lighting and instruct him to fix
his gaze on the distant object.
• Cover one eye and shine a flashlight in front
of the exposed eye. The pupil should
(constrict) because of the light. This response
is called a direct reaction.
• The covered pupil should also contract. This
response is called a consensual reaction.
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10. NEAR POINT REACTION:- When the gaze is
changed from the distant object to an object
close at hand, the pupils should contract.
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11. C. FUNCTIONAL EXAMINATION
1. Focusing power is tested by placing a line of
print close to the eye, then slowly moving it
back to the point at which the patient is able
to read it. The nearest point of
accommodation.
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13. 3. Colour sense is tested by using specially
designed colour pates to distinguish reds,
greens and blues.
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14. 4. Visual acuity testing is done with Snellen
chart or one of its modifications. Each eye is
tested separately, both with and without
glasses, if worn.
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16. • The test is performed at a distance of 6 meters (20
feet).
• Vision is expressed by a fraction, the numerator
denoting the distance at which the test was
performed (normally, 6 metres or 20 feet), and the
denominator denoting the smallest line of letters
which could be read at that distance.
• If a patient is seated 6 meters from the chart and the
smallest lie of letters he is able to read is the one
that should be read at a distance of 30 meters, then
his vision is expressed at 6/30 (20/100).
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17. • If the largest letters on the chart cannot be
read at a distance of 6 meters, the patient is
moved toward the chart until he can read the
largest letters.
• Vision is then expressed as a fraction, with the
numerator denoting the distance at which the
largest line could be read and the
denominator denoting the number of largest
line.
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18. • If the patient cannot read the largest line at a
distance of one meter, the examiner tests the
patient’s ability to see hand motion in front of his
face.
• If the patient cannot see the examiner’s hand at a
distance of one or two meters, he is tested for light
perception.
• A light is flashed from different directions and the
patient is asked from which direction the light
appears and when it goes on and it goes off.
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19. • If the patient can do this, the examination is
recorded as ‘light perception present’.
• If no light perception is present , a person is
technically blind.
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20. ASSESSING SYMPTOMS
In addition to the examinations mentioned
previously, the patient should be assessed:-
• Discomfort or pain in or around the eye
• Photophobia (abnormal sensitivity to light)
• Nystagmus (involuntary and rapid movement of
eyeball.)
• Strabismus (deviation of eye from the normal
physiological axis: ‘crossed vision’)
• Blurred vision
• ‘Spot 'or ‘light 'in the visual field
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21. EYE TESTS AND EXAMS
OPHTHALMOSCOPY:-
• It is an examination of the back part of the eyeball
(fundus), which includes the retina, optic disc,
choroid and blood vessels. Ophthalmoscope
examination takes about 5 and 10 minutes. There
are different types of ophthalmoscopy.
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22. THERE ARE TWO TYPES:-
DIRECT OPHTHALMOSCOPY
• Patient will be seated in a
darkened room. The health
care provider performs this
common exam by shining a
beam of light through the
pupil using an instrument
called an ophthalmoscope.
• It allows the examiner to
view the back of eyeball.
INDIRECT OPHTHALMOSCOPY
• Patient will lie or sit in a semi
reclined position. The health
care provider holds patient
eye open while shining a very
bright light into the eye using
an instrument worn on the
head.
• Some pressure may be
applied to the eyeball using a
small, blunt tool. Patient will
be asked to look in various
direction.10/19/2018
23. 2. APPLANATION METHOD
• This eye test helps
doctors to diagnose
glaucoma by measuring
the amount of pressure
needed to flattern a
portion of the cornea.
• This is done by taking a
thin strip of paper
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24. 3. CORNEAL TOPOGRAPHY
• Corneal topography is a computer assisted diagnostic tool
that creates a three-dimensional map of the surface
curvature of the cornea.
• The cornea (the front window of the eye) is responsible for
about 70 percent of the eye’s focusing power. An eye
with normal vision has an evenly rounded cornea, but if the
cornea is too flat, too steep, or unevenly curved, less than
perfect vision results.
• The greatest advantage of corneal topography is its ability to
detect irregular conditions invisible to most conventional
testing.
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25. • This is an eye test used to evaluate the blood
circulation in the retina. It is useful in helping
diagnose diabetic retinopathy and retinal
detachment.
• During this eye test, a special dye called fluorescein,
is injected into a vein in the arm and it quickly travels
to blood vessels inside the eye.
• When it reach to the eye a specialized camera is
used to the photograph the fluorescein as it
circulates through the blood vessels in the back of
the eye.
• This will enable the doctor to diagnose any
circulation problems, swelling, leaking or abnormal
blood vessels.10/19/2018
27. 4. PUPILLARY DILATION TEST
• During this eye test, the eye doctor places
special drops (Tropicamide) in the eye that
cause the pupil to dilate. By dilating the
pupils, doctor can examine retina for any signs
of disease.
10/19/2018
29. • This is measures the ability to see objects at
special distances. Often doctors will ask the
patient to look at a chart usually 20 feet away
and try to read it while looking through a
special; instrument know n as a phoropter.
• The eye test is useful in helping to diagnose
presbyopia (long-sightedness ), hyperopia
(nearby objects are blurry), myopia (lose
objects appear clearly, but far ones don't.)
and astigmatism (eye does not focus light
evenly).
10/19/2018
30. 6. FLUORESCEIN ANGOGRAPHY
• Fluorescein angiography, fluorescent
angiography, or fundus fluorescein
angiography is a technique for examining the
circulation of the retina and choroid using a
fluorescent dye and a specialized camera.
10/19/2018
31. 7. COLOR FUNDUS PHOTOGRAPHY
• Fundus camera to record color images of the
condition of the interior surface of the eye, in order
to document the presence of disorders and monitor
their change over time.
• A fundus camera or retinal camera is a specialized
low power microscope with an attached camera
designed to photograph the interior surface of the
eye, including the retina, retinal vasculature, optic
disc, macula, and posterior pole (i.e. the fundus).
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32. • The retina is imaged to document conditions
such as diabetic retinopathy, age related
macular degeneration, macular edema and
retinal detachment.
10/19/2018
33. 8. SLIT- LAMP EXAM
• Once patient in the examination
chair, the doctor will place an
instrument in front of patient on
which to rest chin and forehead.
• This helps steady head for the
exam. Doctor may put drops in
eyes to make any abnormalities
on the surface of cornea more
visible.
• The drops contain a yellow dye
called fluorescein, which will
wash away the tears. Additional
drops may also be put in eyes to
allow pupils to dilate, or get
bigger.10/19/2018
34. • The doctor will use a low-powered microscope, along
with a slit lamp, which is a high-intensity light. They
will look closely at eyes. The slit lamp has different
filters to get different views of the eyes. Some
doctor’s offices may have devices that capture digital
images to track changes in the eyes over time.
• During the test, the doctor will examine all areas of
your eye, including the:- eyelids, conjunctiva, iris,
lens, sclera, cornea, retina and optic nerve.
10/19/2018
35. 9. TONOMETERY
• Tonometry is the
procedure eye care
professionals perform
to determine the
intraocular pressure,
the fluid pressure inside
the eye. It is an
important test in the
evaluation of patients
at risk from glaucoma.
• (normal pressure range
is 12 to 22 mm Hg)10/19/2018
36. • Before the tonometry test, eye doctor will put
numbing eye drops in eyes so that patient
don’t feel anything touching them.
• Once eye is numb, doctor may touch a tiny,
thin strip of paper that contains orange dye to
the surface of eye to stain it. This helps
increase the accuracy of the test.
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37. • Doctor will then put a machine called a "slit-
lamp" in front of patient. Patient will rest chin
and forehead on the supports provided.
• The lamp will then be moved toward eyes
until just the tip of the tonometer probe
touches cornea. By flattening cornea just a bit,
the instrument can detect the pressure in eye.
Doctor will adjust the tension until they get a
proper reading.
10/19/2018
38. • Because eyes are numb, patient will feel no
pain during the procedure. Tonometry is
extremely safe. However, there’s a very
minimal risk that cornea could be scratched
when the tonometer touches eye.
• Even if this happens, however, it will normally
heal itself within a few days.
10/19/2018
39. 10. ULTRASOUND BIOMICROSCOPY
• Ultrasound bio microscopy
is a type of ultrasound eye
exam that makes a more
detailed image than regular
ultrasound. High-energy
sound waves are bounced
off the inside of the eye and
the echo patterns are
shown on the screen of an
ultrasound machine
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