Infanticide
DEFINITION
• Unlawful destruction of a child
• Under one year of age /12 months of age
• Does not include :
• death of foetus during labour
• When it is destroyed by craniotomy or decapitation
• Feticide- killing of a fetus anytime prior to its birth
• Intrapartum death – death occurring during labor an delivery
• Neonaticide – the act of killing an infant within the first 24 hours of
life
• Neonatal death – Death of a liveborn infant within first 28 days of life
• Perinatal mortality- Stillbirths + early neonatal death (<7 days)
Filicide/ prolicide
• Killing of a child older than 24 hrs by its own parents
• Maternal filicide (commoner than paternal filicide)
• MLI postpartum psychosis
• Charged under a lesser offence in ENGLAND
• (s1 infanticide act of England, 1938)
• Defense is not available to anyone else
Legal Aspects
• India  tried under Section 302 IPC
• Causing death of a living child in the womb Section 299 IPC
Stillbirth/ SADS (sudden antenatal death syndrome)
• WHO defines
• “One which is born after 28th week of pregnancy
• And which did not breathe/show any other signs of life
after being completely born”
• UK (Still birth Act 1992), period is reduced to 24 weeks
Investigation of such a case
• Examination of mother  signs of recent delivery
•  her psychiatric condition
• Examination of the child  stillborn/deadborn
• viable /not
• whether the child was born alive??
• if born alive, how long did he live???
•  what was the cause of death???
Causes
• Anoxia
• Birth trauma (MC: intracranial haemorrage due to excessive moulding )
• Congenital defects
• Erythroblastosis fetalis
• Placental abnormalities
• Prematurity
• Toxaemia of pregnancy
Deadborn child
• One in which the foetus has died in-utero,
• shows one of the following signs after it is completely born
1. Rigor mortis  normal change of death
2. Maceration no air in the liquor
3. Putrefaction air ++
4. Adipocere liquor amnii is retained for longer
5. Mummificationif all liquor amnii
has been absoarbed /drained
Characteristics Still born fetus Dead born fetus
Period of gestation > 28 weeks any
Condition in uterus Was live before labour Was lying dead in uterus
Predominance Illegitimate and immature male
children in primiparae
No such predominance
Cardinal features Signs of prolonged labour Any signs- mummification/
rigor mortis/etc
Cause Anoxia, birth trauma Congenital anamolies,
ABO incompatibility
Maceration
• (Latin macerare, to soften by soaking)
• is degenerative change occurring in a fetus retained in utero after
death.
• It occurs due to the softening effect of soaking on solid tissues (pulpy
fetus).
Salient features:
(1) The earliest sign of maceration is skin slippage (12 hours after IUD)
exposes a red, shiny, moist dermal surface,
particularly noticeable over bony prominences.
(2) 24 hours- fluid-filled bullae
(3) 48 hours
(i)Sweetish, disagreeable odor
(ii) hemolysis  purplish discoloration of internal organs
(iii) Dark, red-stained fluid accumulates in the serous cavities.
(iv) deep dark red appearance (“tobacco juice”)- amniotic fluid
or in the event of passage of meconium, a thick brown
appearance.
(v) laxity of joints - Bones are flexible and readily detached from
soft parts
(vi) Abdominal organs may show green discoloration due to leakage
of bile pigments from the gallbladder
• (4) 3 to 4 days" Umbilical cord is red, soft and smooth
• (5) 5th day"S/c edema up to 5 mm.
• (6) >1 week"
(i) Meconium released
(ii) Protruding autolyzed liver mass - mistaken for omphalocele in USG
(iii) fluid accumulation ~ hydrops fetalis.
(iv) fluid accumulation at the nape of the neck ~cystic hygroma
(v) distortion of skull occurs during vaginal delivery ~ hydrocephalus.
(vi) from purple to brown discoloration.
• (7) Several weeks –
(i) yellow–gray color
(ii) Dehydration results in shrinkage and compaction - formation of
“fetus papyraceous
Lithopedion
• .Very occasionally an extrauterine pregnancy may be retained for
years forming so-called
• (Greek lithos, stone, paidion, small child), a calcified fetus retained in
the abdominal cavity.
Radiological Signs of IU Deaths
1. Robert’s sign (1944)
• Earliest sign to appear.
• 12 hours after death.
• Air in aorta, pulmonary vessels or abdomen.
2. Spalding’s sign (1922)
Appears 2 days after death.
Overlapping of fetal skull bones seen on x-ray (or ultrasound) examination.
Due to shrinkage of cerebrum after intrauterine death.
[D/d. May also be positive
(i) during or just prior to labor
(ii) scanty liquor amnii and diminished fetal vitality
(iii) microcephaly
(iv) craniostenosis]
3. Deuel’s halo sign (1947)
3 days after death.
Separation of the subcutaneous fat of the fetal scalp
from the cranial bones.
Seen as a halo on radiography and USG.
First described by Deuel in 1947.
Live birth
• [Explanation 3 of S.299, IPC].
• A child is “live born”, if any part of that child has been brought forth,
even though the child may not have breathed or been completely
born
• The Registration of Births And Deaths Act, 1969
• complete expulsion of fetus from its mother, irrespective of the
duration of pregnancy, and who after such expulsion breathes or
shows any other evidence of life
Signs of Live birth
•Changes in Lungs
•Changes in stomach and intestines
•Changes in middle ear
•Other signs
Before respiration After respiration
Shape of chest 1-2cm below umbilicus Drum shaped
Position of
diaphragm
4th/5th rib 6th/7th rib
Lungs colour Uniformly reddish Mosaic/mottled
Volume Smaller, not covering hear Larger, covers heart
Surface smooth uneven
margins sharp rounded
consistency liverlike Soft, crepitant
Blood within lungs less more
Weight of lungs 35 g (1/70 of body wt) 70g (1/35 of body weight)
Pleura loose stretched
Cut section - lungs Before respiration After respiration
oozing Little frothless blood Abundant frothy blood
oozes on cut section
Bronchi and
bronchioles
empty Contained blood stained
froth
Alveoli Not inflated inflated
MLI
Still born/dead born Live born
Hydrostatic test (Raygat’s test)
Principle
• Upon breathing, both wt and vol of lungs are increased increased.
• Wt due to inflow of blood and
• Vol due to inflow of air.
• vol >>>wt specific gravity of lungs is decreased.
Procedure
• Stage 1 –
• Whole thoracic pluck - both lungs and heart are placed
in a bucket of water.
If the pluck floats If the pluck sinks
"indicates air in lungs“ move to second stage.
** liver - control (floats putrefaction)
Stage 2
• Each brochus is tied, and lungs severed above the ligature.
• Each lung is then placed separately in water.
• If either lung floats, it indicates that the infant may have born alive.
• If either lung sinks, move to third stage.
Stage 3
• Cut each lung in 12-20 pieces
(a) Roll a piece of lung b/w a finger and thumb  crackling crepitant
noise.
(squeeze between thumb and finger bubbles)
(c) observe if they float independently.
If not  next stage
Stage 4
• Each piece is now taken out of water, wrapped in a piece of cloth and
squeezed by putting a weight.
• remove the expiratory reserve volume air, and tidal air.
• Residual air still remains within the alveoli, which can not be taken
out by any means.
Note:--
• floating in earlier stages due to artefacts
• (such as gases introduced by artificial respiration),
• the pieces of lung would not continue to float
till the last stage.
Fallacies
(a) Child respired after birth yet lungs sink
Causes
(1) Absorption of air - Circulation continued after stoppage of
respiration
(2) Atelectasis (non expansion) of lungs.
(3) Alveolar duct membrane - Causing obstruction to entry of air
in alveoli
(4) Diseases.
(b) Child did not respire after birth yet lungs float
• Causes:
(1) Artificial respiration - air may be found in stomach too.
(2) Putrefaction - Putrefactive gases will make the lungs float.
(3) Respiration within the womb [vagitus uterinus]-
if membranes have ruptured, but may die from natural causes
(4) Respiration within the Vagina [vagitus vaginalis] - Similar to above.
Hydrostatic test is not Necessary
One is sure fetus was born dead –
(i) Born before age of viability [28 wks]
(ii) Macerated or mummified
(iii)Monster [eg anencephalic]
(iv)Bruising on lungs - indicating efforts
at artificial respiration.
One is sure fetus was born alive
(i) Stomach - contains milk
(i) Umbilical cord - has separated and
a scar has formed.
Changes in Stomach and intestines
• (1) Breslau’s second life test;
Stomach and intestines are removed after tying double ligatures at
each end,
put in water.
float if respiration had taken place; otherwise they sink
Principle - Air is swallowed into the stomach and intestines during
respiration, making them buoyant
(3) False +ve –
(i) Resuscitation attempts
(ii) Bacterial gas formation [putrefaction]
(4) Survival period – can be calculated:
(i) Immediate after birth – gas in stomach (upto 15 mins)
(ii) 1-2 h – Gas reaches small intestines
(iii) 5-6 h – colon
(iv) 12 h - Rectum.
• 5) Drawbacks and Fallacies –
• (i) Useless in putrefaction
• (ii) Air may be swallowed by the child in attempting to free the air
passages of fluid obstructions in cases of stillbirth.
Changes in the middle ear
• (1) Wreden’s test –
• Principle - During embryonic life, the middle ear contains gelatinous
tissue.
• During efforts of breathing,
• the sphincter at the pharyngeal end of Eustachian tube relaxes and
some air enters the middle ear.
• (i) Regular procedure –
• Skull cap is removed and base of skull submerged in water.
• Petrous part of temporal bone [which forms roof of the middle ear] is
opened.
• If a bubble of air escapes from middle ear, the child was born alive.
• (ii) Simpler method –
• Dip ears in water. Puncture tympanic membrane"Bubble of air
escapes" Live birth.
Other signs
a. Blood
• (1) Nucleated RBC"disappear in 24 h
• (2) Fetal Hb [synthesized mainly in liver]“
(i) Before birth"80-90%
(ii) 3 rd month"7-8%
(iii) 6 m"disappears completely.
b. Meconium
Completely excreted from the large intestine in 24-48 h after normal birth
May be completely excreted before birth in (a) breech presentation (b)
severe anoxia.
Caput succadeneum vs cephalheamatoma
Caput succadeneum Cephalhaematoma
Cause Edema b/w skin and galea Rupture of periosteal capillary
Contents Serosanguinous fluid blood
Suture
lines
Extends across suture
lines
Never extends across them
Size larger Smaller
Site bilateral unilateral
Colour No discoloration Discolouration+
Caput succadeneum Cephalhaematoma
Impulse on crying _ -
Appearance Immediately after birth 24-48 hours
Complications None Jaundice
Calcification and
ossification
Disappearance After birth it starts
disappearing
Increases for 24-48 hours
then recedes
MLI Sign of live birth Regression~period of
survival
• D. Changes in skin
• E. Changes in Umbilical cord
• F. Placenta
• G. Circulation
Blood vessel Time of closure
Umbilical arteries 10hrs to 3 days
Left umbilical vein 3-5 days
Ductus venosus 3-5 days
Ductus arteriousus 7-10 days
Foramen Ovale 2-3 months
CAUSES OF DEATH
Precipitate labour
is the expulsion of fetus within less than 3 h of
commencement of contractions
Salient features
• (1) All three stages of labor are merged in one
• (2) Delivery occurs suddenly and rapidly even without the knowledge
of mother
• (3) Delivery may occur even unconsciously during like intoxication,
anaesthasia
Risk factors are
• (i) Multipara [extremely rare in primi]
• (ii) Placental abruption
• (iii) Roomy pelvis
• (iv) Small premature baby.
Maternal complications
i. cervical and grade 3 perineal tear
ii. post-partum hemorrhage
iii. retained placenta
Child may die from
(i) Suffocation
(ii) Drowning
(iii) Head injury –
(iv) Hemorrhage - from torn end of the cord
PM findings
(i) Placenta – is attached to newborn
(ii) Umbilical cord – lacerated, torn
(iii) Hair and injured scalp – show grass, gravel, mud, sand.
(iv) Head –
(a) Caput succedaneum and moulding of head - absent
(b) fracture of skull.
(c) IC hemorrhages
(v) Air passages and lungs –depending on where the child fell or
drowned.
Medico-legal Importance
• (i) Infanticide - Plea
• (ii) False allegation - Alternatively in a true precipitate labor case,
mother may be accused of infanticide.
Head injury in precipitate labour and Blunt trauma
Precipitate labour Blunt trauma
Bruises on the vertex Bruises- anywhere
Lacerations on the scalp- Present Absent
Fractures- parietal bones- fissured
fractures meeting sagittal suture
at right angles
Extensive comminuted and
depressed fractures
Brain- not injured Brain- contusions, haemorrhages
Criminal causes
Act of commission Act of Omission
Asphyxia
Burns
Trauma
Poisons
Abandoned child is called FONDLING
Person in-charge of the infant [mother,
father, guardian etc] can be charged u/s
302, IPC if they fail to take care of child
and allow him to die.
Failure to
(i) Clear the air passages – these may be
obstructed by amniotic fluid or mucus
(ii) Tie the cord – after it is cut. May
cause death by hemorrhage
Abandoning of children
Child < 12years
Section 317 IPC
7 years imprisonment
Concealment of birth by secret disposal of dead body
before, after or during its birth,
2 y imprisonment or fine or both
[Section.318, IPC].
• CHILD ABUSE
Child abuse is the
• physical,
• sexual or
• emotional mistreatment or neglect of a child.
Battered baby syndrome
• Caffey’s syndrome,
• Caffey-Kempe syndrome,
• Maltreatment syndrome,
• Non Accidental Injury of Childhood (NAIC),
• parent-infant traumatic stress syndrome (PITS),
• Tardieu’s syndrome
• clinical condition in which young children
• usually under 3 years of age
• are beaten repeatedly over the most trivial provocation
• Salient features:
• (1) Lack of provocation
• (2) Precipitating factors – Often child’s irritating actions eg crying,
refusal to be quiet, persistent soiling of napkins etc
• (3) Deprivation of nutrition, care and affection is present
• (4) Delay - attention sought
• (5) Cerebral palsy - 10-15% cases of cerebral palsy may be the result
of the battered baby syndrome.
• (6) History is incompatible with injuries
Injuries
• Injuries are due to direct manual violence [commonest].
(1) Tear of the frenulum
(2) Soft tissue
Pinch marks – butterfly marks
Slap marks – show as clear bruises [or as lines of petechial hgs]
resembling fingers
(3)Scalp injuries –
very characteristic. Caused by vigorous pulling on scalp hair
• Subgaleal hematoma
• Traumatic alopecia – bald patches on scalp
• (4) Eyes
(i) Black eye
(ii) Hemorrhages [Retinal, Subconjunctival, Subhyaloid, Vitreous]
(iii) Lens displacement
(iv) Retinal separation.
• (5) Skeletal injuries
(i) Skull fractures, especially in occipito-parietal area
(ii) Transverse and spiral fractures of long bones,
(twisting of arms and legs etc )
(6) Traction lesions - jerking of shaking a child’s limbs
Injuries of the periosteum of long bones are seen without fractures
(i) Periosteal hematomas
(ii) Periosteal shearing
(iii) Epiphyseal separation – seen radiologically as small fragments
separated from the main bone
(iv) Avulsion of metaphysis or chipping of the edges of metaphysis. -
corner fractures and bucket handle fractures.
• (7) Anteroposterior compression of chest
Fractures of ribs in midaxillary line
• (8) Side-to-side compression of chest –
along posterior angles of ribs.
After 1-2 weeks, callus forms.
X-Rays these are seen as a “string of beads” [syn, rosary bead]
paravertebral gutter [Nobbing fracture]
Visceral injuries
(1) Brain and meninges
(2) Lungs – Post-traumatic pulmonary pseudocysts [PTPPCs]
(3) Liver and spleen - Bursting injuries
(4) Hollow viscera – Ruptures
Tearing of mesentery
Thank you

infanticide.pptx

  • 1.
  • 2.
    DEFINITION • Unlawful destructionof a child • Under one year of age /12 months of age • Does not include : • death of foetus during labour • When it is destroyed by craniotomy or decapitation
  • 3.
    • Feticide- killingof a fetus anytime prior to its birth • Intrapartum death – death occurring during labor an delivery • Neonaticide – the act of killing an infant within the first 24 hours of life • Neonatal death – Death of a liveborn infant within first 28 days of life • Perinatal mortality- Stillbirths + early neonatal death (<7 days)
  • 4.
    Filicide/ prolicide • Killingof a child older than 24 hrs by its own parents • Maternal filicide (commoner than paternal filicide) • MLI postpartum psychosis • Charged under a lesser offence in ENGLAND • (s1 infanticide act of England, 1938) • Defense is not available to anyone else
  • 5.
    Legal Aspects • India tried under Section 302 IPC • Causing death of a living child in the womb Section 299 IPC
  • 6.
    Stillbirth/ SADS (suddenantenatal death syndrome) • WHO defines • “One which is born after 28th week of pregnancy • And which did not breathe/show any other signs of life after being completely born” • UK (Still birth Act 1992), period is reduced to 24 weeks
  • 8.
    Investigation of sucha case • Examination of mother  signs of recent delivery •  her psychiatric condition • Examination of the child  stillborn/deadborn • viable /not • whether the child was born alive?? • if born alive, how long did he live??? •  what was the cause of death???
  • 9.
    Causes • Anoxia • Birthtrauma (MC: intracranial haemorrage due to excessive moulding ) • Congenital defects • Erythroblastosis fetalis • Placental abnormalities • Prematurity • Toxaemia of pregnancy
  • 10.
    Deadborn child • Onein which the foetus has died in-utero, • shows one of the following signs after it is completely born 1. Rigor mortis  normal change of death 2. Maceration no air in the liquor 3. Putrefaction air ++ 4. Adipocere liquor amnii is retained for longer 5. Mummificationif all liquor amnii has been absoarbed /drained
  • 11.
    Characteristics Still bornfetus Dead born fetus Period of gestation > 28 weeks any Condition in uterus Was live before labour Was lying dead in uterus Predominance Illegitimate and immature male children in primiparae No such predominance Cardinal features Signs of prolonged labour Any signs- mummification/ rigor mortis/etc Cause Anoxia, birth trauma Congenital anamolies, ABO incompatibility
  • 12.
    Maceration • (Latin macerare,to soften by soaking) • is degenerative change occurring in a fetus retained in utero after death. • It occurs due to the softening effect of soaking on solid tissues (pulpy fetus).
  • 13.
    Salient features: (1) Theearliest sign of maceration is skin slippage (12 hours after IUD) exposes a red, shiny, moist dermal surface, particularly noticeable over bony prominences. (2) 24 hours- fluid-filled bullae
  • 14.
    (3) 48 hours (i)Sweetish,disagreeable odor (ii) hemolysis  purplish discoloration of internal organs (iii) Dark, red-stained fluid accumulates in the serous cavities. (iv) deep dark red appearance (“tobacco juice”)- amniotic fluid or in the event of passage of meconium, a thick brown appearance. (v) laxity of joints - Bones are flexible and readily detached from soft parts (vi) Abdominal organs may show green discoloration due to leakage of bile pigments from the gallbladder
  • 15.
    • (4) 3to 4 days" Umbilical cord is red, soft and smooth • (5) 5th day"S/c edema up to 5 mm. • (6) >1 week" (i) Meconium released (ii) Protruding autolyzed liver mass - mistaken for omphalocele in USG (iii) fluid accumulation ~ hydrops fetalis. (iv) fluid accumulation at the nape of the neck ~cystic hygroma (v) distortion of skull occurs during vaginal delivery ~ hydrocephalus. (vi) from purple to brown discoloration. • (7) Several weeks – (i) yellow–gray color (ii) Dehydration results in shrinkage and compaction - formation of “fetus papyraceous
  • 16.
    Lithopedion • .Very occasionallyan extrauterine pregnancy may be retained for years forming so-called • (Greek lithos, stone, paidion, small child), a calcified fetus retained in the abdominal cavity.
  • 17.
    Radiological Signs ofIU Deaths 1. Robert’s sign (1944) • Earliest sign to appear. • 12 hours after death. • Air in aorta, pulmonary vessels or abdomen.
  • 18.
    2. Spalding’s sign(1922) Appears 2 days after death. Overlapping of fetal skull bones seen on x-ray (or ultrasound) examination. Due to shrinkage of cerebrum after intrauterine death. [D/d. May also be positive (i) during or just prior to labor (ii) scanty liquor amnii and diminished fetal vitality (iii) microcephaly (iv) craniostenosis]
  • 20.
    3. Deuel’s halosign (1947) 3 days after death. Separation of the subcutaneous fat of the fetal scalp from the cranial bones. Seen as a halo on radiography and USG. First described by Deuel in 1947.
  • 21.
    Live birth • [Explanation3 of S.299, IPC]. • A child is “live born”, if any part of that child has been brought forth, even though the child may not have breathed or been completely born • The Registration of Births And Deaths Act, 1969 • complete expulsion of fetus from its mother, irrespective of the duration of pregnancy, and who after such expulsion breathes or shows any other evidence of life
  • 22.
    Signs of Livebirth •Changes in Lungs •Changes in stomach and intestines •Changes in middle ear •Other signs
  • 23.
    Before respiration Afterrespiration Shape of chest 1-2cm below umbilicus Drum shaped Position of diaphragm 4th/5th rib 6th/7th rib Lungs colour Uniformly reddish Mosaic/mottled Volume Smaller, not covering hear Larger, covers heart Surface smooth uneven margins sharp rounded consistency liverlike Soft, crepitant Blood within lungs less more Weight of lungs 35 g (1/70 of body wt) 70g (1/35 of body weight) Pleura loose stretched
  • 24.
    Cut section -lungs Before respiration After respiration oozing Little frothless blood Abundant frothy blood oozes on cut section Bronchi and bronchioles empty Contained blood stained froth Alveoli Not inflated inflated MLI Still born/dead born Live born
  • 25.
    Hydrostatic test (Raygat’stest) Principle • Upon breathing, both wt and vol of lungs are increased increased. • Wt due to inflow of blood and • Vol due to inflow of air. • vol >>>wt specific gravity of lungs is decreased.
  • 26.
    Procedure • Stage 1– • Whole thoracic pluck - both lungs and heart are placed in a bucket of water. If the pluck floats If the pluck sinks "indicates air in lungs“ move to second stage. ** liver - control (floats putrefaction)
  • 27.
    Stage 2 • Eachbrochus is tied, and lungs severed above the ligature. • Each lung is then placed separately in water. • If either lung floats, it indicates that the infant may have born alive. • If either lung sinks, move to third stage.
  • 28.
    Stage 3 • Cuteach lung in 12-20 pieces (a) Roll a piece of lung b/w a finger and thumb  crackling crepitant noise. (squeeze between thumb and finger bubbles) (c) observe if they float independently. If not  next stage
  • 29.
    Stage 4 • Eachpiece is now taken out of water, wrapped in a piece of cloth and squeezed by putting a weight. • remove the expiratory reserve volume air, and tidal air. • Residual air still remains within the alveoli, which can not be taken out by any means.
  • 30.
    Note:-- • floating inearlier stages due to artefacts • (such as gases introduced by artificial respiration), • the pieces of lung would not continue to float till the last stage.
  • 31.
    Fallacies (a) Child respiredafter birth yet lungs sink Causes (1) Absorption of air - Circulation continued after stoppage of respiration (2) Atelectasis (non expansion) of lungs. (3) Alveolar duct membrane - Causing obstruction to entry of air in alveoli (4) Diseases.
  • 32.
    (b) Child didnot respire after birth yet lungs float • Causes: (1) Artificial respiration - air may be found in stomach too. (2) Putrefaction - Putrefactive gases will make the lungs float. (3) Respiration within the womb [vagitus uterinus]- if membranes have ruptured, but may die from natural causes (4) Respiration within the Vagina [vagitus vaginalis] - Similar to above.
  • 33.
    Hydrostatic test isnot Necessary One is sure fetus was born dead – (i) Born before age of viability [28 wks] (ii) Macerated or mummified (iii)Monster [eg anencephalic] (iv)Bruising on lungs - indicating efforts at artificial respiration. One is sure fetus was born alive (i) Stomach - contains milk (i) Umbilical cord - has separated and a scar has formed.
  • 34.
    Changes in Stomachand intestines • (1) Breslau’s second life test; Stomach and intestines are removed after tying double ligatures at each end, put in water. float if respiration had taken place; otherwise they sink Principle - Air is swallowed into the stomach and intestines during respiration, making them buoyant
  • 35.
    (3) False +ve– (i) Resuscitation attempts (ii) Bacterial gas formation [putrefaction] (4) Survival period – can be calculated: (i) Immediate after birth – gas in stomach (upto 15 mins) (ii) 1-2 h – Gas reaches small intestines (iii) 5-6 h – colon (iv) 12 h - Rectum.
  • 36.
    • 5) Drawbacksand Fallacies – • (i) Useless in putrefaction • (ii) Air may be swallowed by the child in attempting to free the air passages of fluid obstructions in cases of stillbirth.
  • 37.
    Changes in themiddle ear • (1) Wreden’s test – • Principle - During embryonic life, the middle ear contains gelatinous tissue. • During efforts of breathing, • the sphincter at the pharyngeal end of Eustachian tube relaxes and some air enters the middle ear.
  • 38.
    • (i) Regularprocedure – • Skull cap is removed and base of skull submerged in water. • Petrous part of temporal bone [which forms roof of the middle ear] is opened. • If a bubble of air escapes from middle ear, the child was born alive. • (ii) Simpler method – • Dip ears in water. Puncture tympanic membrane"Bubble of air escapes" Live birth.
  • 39.
    Other signs a. Blood •(1) Nucleated RBC"disappear in 24 h • (2) Fetal Hb [synthesized mainly in liver]“ (i) Before birth"80-90% (ii) 3 rd month"7-8% (iii) 6 m"disappears completely. b. Meconium Completely excreted from the large intestine in 24-48 h after normal birth May be completely excreted before birth in (a) breech presentation (b) severe anoxia.
  • 40.
    Caput succadeneum vscephalheamatoma Caput succadeneum Cephalhaematoma Cause Edema b/w skin and galea Rupture of periosteal capillary Contents Serosanguinous fluid blood Suture lines Extends across suture lines Never extends across them Size larger Smaller Site bilateral unilateral Colour No discoloration Discolouration+
  • 41.
    Caput succadeneum Cephalhaematoma Impulseon crying _ - Appearance Immediately after birth 24-48 hours Complications None Jaundice Calcification and ossification Disappearance After birth it starts disappearing Increases for 24-48 hours then recedes MLI Sign of live birth Regression~period of survival
  • 45.
    • D. Changesin skin • E. Changes in Umbilical cord • F. Placenta • G. Circulation Blood vessel Time of closure Umbilical arteries 10hrs to 3 days Left umbilical vein 3-5 days Ductus venosus 3-5 days Ductus arteriousus 7-10 days Foramen Ovale 2-3 months
  • 46.
    CAUSES OF DEATH Precipitatelabour is the expulsion of fetus within less than 3 h of commencement of contractions Salient features • (1) All three stages of labor are merged in one • (2) Delivery occurs suddenly and rapidly even without the knowledge of mother • (3) Delivery may occur even unconsciously during like intoxication, anaesthasia
  • 47.
    Risk factors are •(i) Multipara [extremely rare in primi] • (ii) Placental abruption • (iii) Roomy pelvis • (iv) Small premature baby.
  • 48.
    Maternal complications i. cervicaland grade 3 perineal tear ii. post-partum hemorrhage iii. retained placenta Child may die from (i) Suffocation (ii) Drowning (iii) Head injury – (iv) Hemorrhage - from torn end of the cord
  • 49.
    PM findings (i) Placenta– is attached to newborn (ii) Umbilical cord – lacerated, torn (iii) Hair and injured scalp – show grass, gravel, mud, sand. (iv) Head – (a) Caput succedaneum and moulding of head - absent (b) fracture of skull. (c) IC hemorrhages (v) Air passages and lungs –depending on where the child fell or drowned.
  • 50.
    Medico-legal Importance • (i)Infanticide - Plea • (ii) False allegation - Alternatively in a true precipitate labor case, mother may be accused of infanticide.
  • 51.
    Head injury inprecipitate labour and Blunt trauma Precipitate labour Blunt trauma Bruises on the vertex Bruises- anywhere Lacerations on the scalp- Present Absent Fractures- parietal bones- fissured fractures meeting sagittal suture at right angles Extensive comminuted and depressed fractures Brain- not injured Brain- contusions, haemorrhages
  • 52.
    Criminal causes Act ofcommission Act of Omission Asphyxia Burns Trauma Poisons Abandoned child is called FONDLING Person in-charge of the infant [mother, father, guardian etc] can be charged u/s 302, IPC if they fail to take care of child and allow him to die. Failure to (i) Clear the air passages – these may be obstructed by amniotic fluid or mucus (ii) Tie the cord – after it is cut. May cause death by hemorrhage
  • 53.
    Abandoning of children Child< 12years Section 317 IPC 7 years imprisonment
  • 54.
    Concealment of birthby secret disposal of dead body before, after or during its birth, 2 y imprisonment or fine or both [Section.318, IPC].
  • 55.
    • CHILD ABUSE Childabuse is the • physical, • sexual or • emotional mistreatment or neglect of a child.
  • 56.
    Battered baby syndrome •Caffey’s syndrome, • Caffey-Kempe syndrome, • Maltreatment syndrome, • Non Accidental Injury of Childhood (NAIC), • parent-infant traumatic stress syndrome (PITS), • Tardieu’s syndrome
  • 57.
    • clinical conditionin which young children • usually under 3 years of age • are beaten repeatedly over the most trivial provocation
  • 58.
    • Salient features: •(1) Lack of provocation • (2) Precipitating factors – Often child’s irritating actions eg crying, refusal to be quiet, persistent soiling of napkins etc • (3) Deprivation of nutrition, care and affection is present • (4) Delay - attention sought • (5) Cerebral palsy - 10-15% cases of cerebral palsy may be the result of the battered baby syndrome. • (6) History is incompatible with injuries
  • 59.
    Injuries • Injuries aredue to direct manual violence [commonest]. (1) Tear of the frenulum (2) Soft tissue Pinch marks – butterfly marks Slap marks – show as clear bruises [or as lines of petechial hgs] resembling fingers (3)Scalp injuries – very characteristic. Caused by vigorous pulling on scalp hair • Subgaleal hematoma • Traumatic alopecia – bald patches on scalp
  • 63.
    • (4) Eyes (i)Black eye (ii) Hemorrhages [Retinal, Subconjunctival, Subhyaloid, Vitreous] (iii) Lens displacement (iv) Retinal separation. • (5) Skeletal injuries (i) Skull fractures, especially in occipito-parietal area (ii) Transverse and spiral fractures of long bones, (twisting of arms and legs etc )
  • 64.
    (6) Traction lesions- jerking of shaking a child’s limbs Injuries of the periosteum of long bones are seen without fractures (i) Periosteal hematomas (ii) Periosteal shearing (iii) Epiphyseal separation – seen radiologically as small fragments separated from the main bone (iv) Avulsion of metaphysis or chipping of the edges of metaphysis. - corner fractures and bucket handle fractures.
  • 65.
    • (7) Anteroposteriorcompression of chest Fractures of ribs in midaxillary line • (8) Side-to-side compression of chest – along posterior angles of ribs. After 1-2 weeks, callus forms. X-Rays these are seen as a “string of beads” [syn, rosary bead] paravertebral gutter [Nobbing fracture]
  • 67.
    Visceral injuries (1) Brainand meninges (2) Lungs – Post-traumatic pulmonary pseudocysts [PTPPCs] (3) Liver and spleen - Bursting injuries (4) Hollow viscera – Ruptures Tearing of mesentery
  • 69.