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HYPERTENSION
BLOOD PRESSURE
The force of
circulating
blood on
walls of the
arteries
DEFINITION
Hypertension also known as high
blood pressure, is a long term
condition in which the blood
in the arteries is persistently
RISK FACTORS
Age: chance of CAD after 50 yrs of age
Alcohol, smoking and DM
Excessive dietary intake of sodium
Gender
Family history
Obesity
Sedentary life style
stress
ETIOLOGY
Primary HTN: it is the elevation in
BP without an identified cause.
Secondary HTN: it is the elevation in
BP with an exact cause. This type is
account for 5-10% of total cases.
The causes of Secondary HTN
includes
• Congenital narrowing of aorta
• Renal disease
• Endocrine disorders like cushing’s
syndrome
• Neurological disorders like brain tumors
and head injury
• Sleep apnea
• Medications like oral contraceptive pills,
NSAID, and coccaine
• Cirrhosis of liver
PATHOPHYSIOLOGY
 The normal blood pressure is maintained by four
mechanisms
 Sympathetic nervous system activities
 Activities of vascular endothelium
 Activities of renal system
 Activities of endocrine system
SYMPATHETIC NERVOUS SYSTEM ACTIVITIES
 When the BP is decreasing the activation of SNS will occur.The
increased SNS activity increases the heart rate and cardiac
contraction.
 The increased the heart rate and cardiac contraction produce
vasoconstriction in the peripheral arterioles and promotes the
release of renin from kidney.
 The net effect of SNS activation is to increase the arterial blood
pressure by increasing cardiac output and systemic vascular
resistance.
BP=CO X SVR
ACTIVITIES OF VASCULAR ENDOTHELIUM
 The vascular endothelium is a single cell layer that lines
the blood vessal.
 It will produce vasoactive substances and growth factors
like nitric acid, endothelin etc.
 These substances are potent vasoconstrictors andcauses
increases blood pressure level.
ACTIVITIES OF RENAL SYSTEM
ACTIVITIES OF ENDOCRINE SYSTEM
 When the angiotensin-II is stimulated in the adrenal cortex, it
will secrete aldosterone.
 The aldosterone will stimulate the kidneys to retain sodium
and water. Thus the BP and cardiac output will getincreased.
CLINICAL FEATURES
 Some times the high blood pressure does not causes any
symptoms, so that it is known as silent killer disease.
 In some patients the symptoms will develop like,
Severe head
ache
Blurred vision
Dizziness
Nausea
Vomiting
Fatigue
epistaxis
Chest pain
Shortness of breath
Irregular heart beat
papilledema
DIAGNOSTIC EVALUATIONS
 History collection and physical examination
 Medical history of diabetes mellitus
 Complete blood count
 Chest x-ray
 ECG
MANAGEMENT
 Mainly the management of hypertension is possible
by two ways, which include
 Life style modification
 Pharmacological therapy
LIFE STYLE MODIFICATION
The life style modification measures mainly includes,
 Weight reduction
 DASH Diet (Dietary approaches to stop hypertension)
 Dietary sodium reduction
 Reduce alcohol
 Exercise
 Stress management
PHARMACOLOGICAL THERAPY
 Diuretics: it helps the kidneys to inhibit the sodium
reabsorption in the distal convoluted tubules, ascending limb
and loop of henle.
Eg: chlorothiazide, furosemide
 Beta blockers: These medications reduces the workload of the
heart and blood vessal and causing the heart to beat slowly and
with less force.
Eg: Atenolol, propanolol
 Alpha blockers: These medications causes
the peripheral vasodilation of blood
vessals.
Eg: Prazosin
 Vasodilators: These medications acting directly on the
muscles in the wall of arteries and preventing the
muscles from tightening and arteries from narrowing.
Eg: Nitroglycerin, Sodium nitro prusside
 ACE Inhibitors: This group of medication
will reduce the conversion of A-I to A-II and
prevents vasoconstriction.
Eg: Captopril, Ramipril
 Calcium channel blockers: These medicines will block the
movement of extra cellular calcium into the cells and causing
vasodilation and decreased heart rate.
Eg: Amlodipine,Verapamil
 Alternative therapies which are helpful to regulate blood
pressure includes acupuncture, relaxation techniquesand
diversional therapies.
NURSING MANAGEMENT
 Proper history collection should be done which includes
family history also.
 Dietary habits should be assessed
 Identify the medical history such as diabetes, CAD, renal
disease etc…
 Instruct the patient to avoid smoking and alcoholism
 Auscultate heart rate and palpate peripheral pulses.
 Identify the use of medications such as contraceptives,
steroids, NSAID etc…
 Monitor vital signs frequently
 Provide diet which is low in sodium and rich with fruits
and vegetables.
 Monitor the blood cholesterol level frequently
hypertension.pptx adult health nursing ppt

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hypertension.pptx adult health nursing ppt

  • 2. BLOOD PRESSURE The force of circulating blood on walls of the arteries
  • 3. DEFINITION Hypertension also known as high blood pressure, is a long term condition in which the blood in the arteries is persistently
  • 4.
  • 5. RISK FACTORS Age: chance of CAD after 50 yrs of age Alcohol, smoking and DM Excessive dietary intake of sodium Gender Family history Obesity Sedentary life style stress
  • 6. ETIOLOGY Primary HTN: it is the elevation in BP without an identified cause. Secondary HTN: it is the elevation in BP with an exact cause. This type is account for 5-10% of total cases.
  • 7. The causes of Secondary HTN includes • Congenital narrowing of aorta • Renal disease • Endocrine disorders like cushing’s syndrome • Neurological disorders like brain tumors and head injury • Sleep apnea • Medications like oral contraceptive pills, NSAID, and coccaine • Cirrhosis of liver
  • 8.
  • 9.
  • 10.
  • 11. PATHOPHYSIOLOGY  The normal blood pressure is maintained by four mechanisms  Sympathetic nervous system activities  Activities of vascular endothelium  Activities of renal system  Activities of endocrine system
  • 12. SYMPATHETIC NERVOUS SYSTEM ACTIVITIES  When the BP is decreasing the activation of SNS will occur.The increased SNS activity increases the heart rate and cardiac contraction.  The increased the heart rate and cardiac contraction produce vasoconstriction in the peripheral arterioles and promotes the release of renin from kidney.  The net effect of SNS activation is to increase the arterial blood pressure by increasing cardiac output and systemic vascular resistance. BP=CO X SVR
  • 13.
  • 14. ACTIVITIES OF VASCULAR ENDOTHELIUM  The vascular endothelium is a single cell layer that lines the blood vessal.  It will produce vasoactive substances and growth factors like nitric acid, endothelin etc.  These substances are potent vasoconstrictors andcauses increases blood pressure level.
  • 16. ACTIVITIES OF ENDOCRINE SYSTEM  When the angiotensin-II is stimulated in the adrenal cortex, it will secrete aldosterone.  The aldosterone will stimulate the kidneys to retain sodium and water. Thus the BP and cardiac output will getincreased.
  • 17.
  • 18. CLINICAL FEATURES  Some times the high blood pressure does not causes any symptoms, so that it is known as silent killer disease.  In some patients the symptoms will develop like,
  • 20. epistaxis Chest pain Shortness of breath Irregular heart beat papilledema
  • 21. DIAGNOSTIC EVALUATIONS  History collection and physical examination  Medical history of diabetes mellitus  Complete blood count  Chest x-ray  ECG
  • 22. MANAGEMENT  Mainly the management of hypertension is possible by two ways, which include  Life style modification  Pharmacological therapy
  • 23. LIFE STYLE MODIFICATION The life style modification measures mainly includes,  Weight reduction  DASH Diet (Dietary approaches to stop hypertension)  Dietary sodium reduction  Reduce alcohol  Exercise  Stress management
  • 24.
  • 25.
  • 26. PHARMACOLOGICAL THERAPY  Diuretics: it helps the kidneys to inhibit the sodium reabsorption in the distal convoluted tubules, ascending limb and loop of henle. Eg: chlorothiazide, furosemide
  • 27.  Beta blockers: These medications reduces the workload of the heart and blood vessal and causing the heart to beat slowly and with less force. Eg: Atenolol, propanolol
  • 28.  Alpha blockers: These medications causes the peripheral vasodilation of blood vessals. Eg: Prazosin
  • 29.  Vasodilators: These medications acting directly on the muscles in the wall of arteries and preventing the muscles from tightening and arteries from narrowing. Eg: Nitroglycerin, Sodium nitro prusside
  • 30.  ACE Inhibitors: This group of medication will reduce the conversion of A-I to A-II and prevents vasoconstriction. Eg: Captopril, Ramipril
  • 31.  Calcium channel blockers: These medicines will block the movement of extra cellular calcium into the cells and causing vasodilation and decreased heart rate. Eg: Amlodipine,Verapamil
  • 32.  Alternative therapies which are helpful to regulate blood pressure includes acupuncture, relaxation techniquesand diversional therapies.
  • 33. NURSING MANAGEMENT  Proper history collection should be done which includes family history also.  Dietary habits should be assessed  Identify the medical history such as diabetes, CAD, renal disease etc…  Instruct the patient to avoid smoking and alcoholism  Auscultate heart rate and palpate peripheral pulses.
  • 34.  Identify the use of medications such as contraceptives, steroids, NSAID etc…  Monitor vital signs frequently  Provide diet which is low in sodium and rich with fruits and vegetables.  Monitor the blood cholesterol level frequently