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HYPERTENSION
Ratheesh R L
Lecturer
Murlidhar college of nursing
Rajkot
DEFINITION
 Hypertension (HTN or HT), also known as high blood
pressure (HBP), is a long term medical condition in
which the blood pressure in the arteries is persistently
elevated.
 The SBP will be more than or equal of 140 mmHg and
DBP will be more than or equal of 90 mmHg
TYPES
 Pre hypertension: SBP: 120-139 mmHg
DBP: 80-89 mmHg
 Hypertension stage I: SBP: 140-159 mmHg
DBP: 90-99 mmHg
 Hypertension stage II: SBP: More or equal to 160 mmHg
DBP: More or equal to 100 mmHg
 Pregnancy induced HTN: because of increased production of
hormones and enzymes during pregnancy.
ETIOLOGY
 Primary HTN: it is the elevation in BP without an
identified cause.
 Secondary HTN: it is the elevation in BP with an
exact cause. This type is account for 5-10% of total
cases.
 The causes of Secondary HTN includes
 Congenital narrowing of aorta
 Renal disease
 Endocrine disorders like cushing’s syndrome
 Neurological disorders like brain tumors and
head injury
 Sleep apnea
 Medications like oral contraceptive pills,
NSAID, and coccaine
 Cirrhosis of liver
RISK FACTORS
 Age: chance of CAD after 50 yrs of age
 Alcohol, smoking and DM
 Excessive dietary intake of sodium
 Gender
 Family history
 Obesity
 Sedentary life style
 stress
PATHOPHYSIOLOGY
 The normal blood pressure is maintained by four
mechanisms
 Sympathetic nervous system activities
 Activities of vascular endothelium
 Activities of renal system
 Activities of endocrine system
SYMPATHETIC NERVOUS SYSTEM ACTIVITIES
 When the BP is decreasing the activation of SNS will occur. The
increased SNS activity increases the heart rate and cardiac
contraction.
 The increased the heart rate and cardiac contraction produce
vasoconstriction in the peripheral arterioles and promotes the
release of renin from kidney.
 The net effect of SNS activation is to increase the arterial blood
pressure by increasing cardiac output and systemic vascular
resistance.
BP=CO X SVR
ACTIVITIES OF VASCULAR ENDOTHELIUM
 The vascular endothelium is a single cell layer that lines
the blood vessal.
 It will produce vasoactive substances and growth factors
like nitric acid, endothelin etc..
 These substances are potent vasoconstrictors and causes
increases blood pressure level.
ACTIVITIES OF RENAL SYSTEM
ACTIVITIES OF ENDOCRINE SYSTEM
 When the angiotensin-II is stimulated in the adrenal cortex, it
will secrete aldosterone.
 The aldosterone will stimulate the kidneys to retain sodium
and water. Thus the BP and cardiac output will get increased.
CLINICAL FEATURES
 Some times the high blood pressure does not causes any
symptoms, so that it is known as silent killer disease.
 In some patients the symptoms will develop like,
 Severe head ache
 Blurred vision
 Dizziness
 Nausea
 Vomiting
 Fatigue
 Confusion epistaxis
 Chest pain
 Shortness of breath
 Irregular heart beat
 papilledema
DIAGNOSTIC EVALUATIONS
 History collection and physical examination
 Medical history of diabetes mellitus
 Complete blood count
 Chest x-ray
 ECG
MANAGEMENT
 Mainly the management of hypertension is possible
by two ways, which include
 Life style modification
 Pharmacological therapy
LIFE STYLE MODIFICATION
The life style modification measures mainly includes,
 Weight reduction
 DASH Diet (Dietary approaches to stop hypertension)
 Dietary sodium reduction
 Reduce alcohol
 Exercise
 Stress management
PHARMACOLOGICAL THERAPY
Various groups of drugs are used for the
treatment of hypertension, collectively these drugs are called as
anti-hypertensive drugs, which includes,
 Diuretics: it helps the kidneys to inhibit the sodium
reabsorption in the distal convoluted tubules, ascending limb
and loop of henle.
Eg: chlorothiazide, furosemide
 Beta blockers: These medications reduces the workload of the
heart and blood vessal and causing the heart to beat slowly and
with less force.
Eg: Atenolol, propanolol
 Alpha blockers: These medications causes the peripheral
vasodilation of blood vessals.
Eg: Prazosin
 Vasodilators: These medications acting directly on the
muscles in the wall of arteries and preventing the
muscles from tightening and arteries from narrowing.
Eg: Nitroglycerin, Sodium nitro prusside
 ACE Inhibitors: This group of medication will reduce
the conversion of A-I to A-II and prevents
vasoconstriction.
Eg: Captopril, Ramipril
 Calcium channel blockers: These medicines will block the
movement of extra cellular calcium into the cells and causing
vasodilation and decreased heart rate.
Eg: Amlodipine, Verapamil
 Alternative therapies which are helpful to regulate blood
pressure includes acupuncture, relaxation techniques and
diversional therapies.
NURSING MANAGEMENT
 Proper history collection should be done which includes
family history also.
 Dietary habits should be assessed
 Identify the medical history such as diabetes, CAD, renal
disease etc…
 Instruct the patient to avoid smoking and alcoholism
 Auscultate heart rate and palpate peripheral pulses.
 Identify the use of medications such as contraceptives,
steroids, NSAID etc…
 Monitor vital signs frequently
 Provide diet which is low in sodium and rich with fruits
and vegetables.
 Monitor the blood cholesterol level frequently
Hypertension

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Hypertension

  • 2. DEFINITION  Hypertension (HTN or HT), also known as high blood pressure (HBP), is a long term medical condition in which the blood pressure in the arteries is persistently elevated.  The SBP will be more than or equal of 140 mmHg and DBP will be more than or equal of 90 mmHg
  • 3. TYPES  Pre hypertension: SBP: 120-139 mmHg DBP: 80-89 mmHg  Hypertension stage I: SBP: 140-159 mmHg DBP: 90-99 mmHg  Hypertension stage II: SBP: More or equal to 160 mmHg DBP: More or equal to 100 mmHg  Pregnancy induced HTN: because of increased production of hormones and enzymes during pregnancy.
  • 4. ETIOLOGY  Primary HTN: it is the elevation in BP without an identified cause.  Secondary HTN: it is the elevation in BP with an exact cause. This type is account for 5-10% of total cases.  The causes of Secondary HTN includes  Congenital narrowing of aorta
  • 5.  Renal disease  Endocrine disorders like cushing’s syndrome  Neurological disorders like brain tumors and head injury  Sleep apnea  Medications like oral contraceptive pills, NSAID, and coccaine  Cirrhosis of liver
  • 6. RISK FACTORS  Age: chance of CAD after 50 yrs of age  Alcohol, smoking and DM  Excessive dietary intake of sodium  Gender  Family history  Obesity  Sedentary life style  stress
  • 7. PATHOPHYSIOLOGY  The normal blood pressure is maintained by four mechanisms  Sympathetic nervous system activities  Activities of vascular endothelium  Activities of renal system  Activities of endocrine system
  • 8. SYMPATHETIC NERVOUS SYSTEM ACTIVITIES  When the BP is decreasing the activation of SNS will occur. The increased SNS activity increases the heart rate and cardiac contraction.  The increased the heart rate and cardiac contraction produce vasoconstriction in the peripheral arterioles and promotes the release of renin from kidney.  The net effect of SNS activation is to increase the arterial blood pressure by increasing cardiac output and systemic vascular resistance. BP=CO X SVR
  • 9. ACTIVITIES OF VASCULAR ENDOTHELIUM  The vascular endothelium is a single cell layer that lines the blood vessal.  It will produce vasoactive substances and growth factors like nitric acid, endothelin etc..  These substances are potent vasoconstrictors and causes increases blood pressure level.
  • 11. ACTIVITIES OF ENDOCRINE SYSTEM  When the angiotensin-II is stimulated in the adrenal cortex, it will secrete aldosterone.  The aldosterone will stimulate the kidneys to retain sodium and water. Thus the BP and cardiac output will get increased.
  • 12. CLINICAL FEATURES  Some times the high blood pressure does not causes any symptoms, so that it is known as silent killer disease.  In some patients the symptoms will develop like,
  • 13.  Severe head ache  Blurred vision  Dizziness  Nausea  Vomiting  Fatigue  Confusion epistaxis  Chest pain  Shortness of breath  Irregular heart beat  papilledema
  • 14. DIAGNOSTIC EVALUATIONS  History collection and physical examination  Medical history of diabetes mellitus  Complete blood count  Chest x-ray  ECG
  • 15. MANAGEMENT  Mainly the management of hypertension is possible by two ways, which include  Life style modification  Pharmacological therapy
  • 16. LIFE STYLE MODIFICATION The life style modification measures mainly includes,  Weight reduction  DASH Diet (Dietary approaches to stop hypertension)  Dietary sodium reduction  Reduce alcohol  Exercise  Stress management
  • 17. PHARMACOLOGICAL THERAPY Various groups of drugs are used for the treatment of hypertension, collectively these drugs are called as anti-hypertensive drugs, which includes,  Diuretics: it helps the kidneys to inhibit the sodium reabsorption in the distal convoluted tubules, ascending limb and loop of henle. Eg: chlorothiazide, furosemide
  • 18.  Beta blockers: These medications reduces the workload of the heart and blood vessal and causing the heart to beat slowly and with less force. Eg: Atenolol, propanolol  Alpha blockers: These medications causes the peripheral vasodilation of blood vessals. Eg: Prazosin
  • 19.  Vasodilators: These medications acting directly on the muscles in the wall of arteries and preventing the muscles from tightening and arteries from narrowing. Eg: Nitroglycerin, Sodium nitro prusside  ACE Inhibitors: This group of medication will reduce the conversion of A-I to A-II and prevents vasoconstriction. Eg: Captopril, Ramipril
  • 20.  Calcium channel blockers: These medicines will block the movement of extra cellular calcium into the cells and causing vasodilation and decreased heart rate. Eg: Amlodipine, Verapamil
  • 21.  Alternative therapies which are helpful to regulate blood pressure includes acupuncture, relaxation techniques and diversional therapies.
  • 22. NURSING MANAGEMENT  Proper history collection should be done which includes family history also.  Dietary habits should be assessed  Identify the medical history such as diabetes, CAD, renal disease etc…  Instruct the patient to avoid smoking and alcoholism  Auscultate heart rate and palpate peripheral pulses.
  • 23.  Identify the use of medications such as contraceptives, steroids, NSAID etc…  Monitor vital signs frequently  Provide diet which is low in sodium and rich with fruits and vegetables.  Monitor the blood cholesterol level frequently