HYDROCEPHALUS
Presented by : Dr. Vishal
kr. Kandhway
1
Introduction
 Derived from Greek word “Hydro” meaning
“Water”, and “Cephalus”, meaning “Head”.
 Excessive accumulation of cerebrospinal
fluid (CSF) resulting in abnormal widening
of the spaces in the brain.
 The normal volume of CSF is 140 ml. CSF is
produced by the choroid plexus in the
ventricles at a rate of about 500 ml in 24 hours.
2
3
Ventricles of The Brain
 Lateral Ventricle
 Third Ventricle
 Fourth Ventricle
4
Circulation of CSF
 Normally, CSF flows continually from the
interior cavities in the brain (ventricles) to
the thin subarachnoid space that
surrounds the brain and spinal cord.
5
6
Circulation of CSF
 Normally, CSF flows through the ventricles,
exits into cisterns (closed spaces that
serve as reservoirs) at the base of the
brain, bathes the surfaces of the brain and
spinal cord, and then reabsorbs into the
bloodstream.
7
Circulation of CSF
The major clinical features in infants are:
 failure to thrive
 increased skull circumference
 tense anterior fontanelle
 ‘cracked pot’ sound on skull percussion
 transillumination of cranial cavity with strong light
 when severe, impaired conscious level and
vomiting
 ‘setting sun’ appearance due to lid retraction and
impaired upward gaze from 3rd ventricular
pressure on the midbrain tectum
 thin scalp with dilated veins. 8
 The balance between production and
absorption of CSF is critically important.
Because CSF is made continuously,
medical conditions that block its normal
flow or absorption will result in an over-
accumulation of CSF. The resulting
pressure of the fluid against brain tissue is
what causes Hydrocephalus.
9
Cause of Hydrocephalus
Causes of Hydrocephalus
10
Types of Hydrocephalus
 There are several different types of
Hydrocephalus:
 Congenital hydrocephalus
 Acquired hydrocephalus
 Communicating hydrocephalus
 Non-communicating hydrocephalus
11
Congenital Hydrocephalus
 Congenital hydrocephalus is present at
birth and may be caused by environmental
influences during fetal development or by
genetic factors.
 Causes are:
 Aqueduct Stenosis
 Colloid Cyst
12
Acquired Hydrocephalus
 Develops at the time of birth or at some
point afterward. This type of hydrocephalus
can affect individuals of all ages and may
be caused by injury or disease.
13
 Acquired Hydrocephalus is due to:
 Tumors
 Hemorrhage
 Ventriculitis
Acquired Hydrocephalus
14
Communicating Hydrocephalus
 Communicating hydrocephalus occurs
when the flow of cerebrospinal fluid (CSF)
is blocked after it exits the ventricles. This
form is called communicating, because the
CSF can still flow between the ventricles,
which remain open.
15
 Causes are due to:
 Post-hemorrhage
 Bacterial Meningitis
 Malignant Meningitis
 Increased Venous Pressure
Communicating Hydrocephalus
16
Non-Communicating Hydrocephalus
 Non-communicating hydrocephalus, also
called "obstructive" hydrocephalus, occurs
when the flow of CSF is blocked along one
or more of the narrow pathways connecting
the ventricles.
 Causes include:
 Congenital
 Acquired
17
Treatment
 In general, the treatment of hydrocephalus is a CSF
shunt or a 3rd ventriculostomy. If there has been
rapid neurological deterioration this will need to be
performed as an emergency.
 If the hydrocephalus is due to an obstructing tumour
that is surgically accessible, resection of the mass may
lead to resolution of the hydrocephalus and a shunt
might not be necessary.
CSF shunt
 The usual method of CSF diversion is a
ventriculoperitoneal shunt, in which a catheter is
placed into the lateral ventricle and is connected
to a subcutaneous unidirectional pressure-
regulated valve which is attached to a catheter
threaded subcutaneously down to the abdomen
and inserted into the peritoneal cavity.
 Modern valves can have their draining pressures
adjusted percutaneously and shunts are being
developed allowing intracranial pressure to be
monitored percutaneously.
Complications of ventriculoperitoneal shunt
The major possible complications are:
• infection of the shunt
• obstruction of the shunt
• intracranial haemorrhage.
Other CSF shunts :
 ventriculoatrial shunts
 ventriculopleural shunts
 lumboperitoneal shunts.
Anesthetic consideration for VP Shunt
 The child should be carefully monitored whilst awaiting
surgery for signs of neurological deterioration as this
can occur rapidly.
 If patient suffers resp. arrest or sudden deterioration in
neurological ststus due to raised ICP & tonsillar
herniation, ET intubation & hyperventilation with with
100% O2 should be carried out.
 The goal for induction is to avoid any further rise in ICP
& avoid hypotension. IV or Inhalational induction can
be used in child with normal ICP.
 IV induction allows rapid control of airway in
emergency situations(raised ICP). 22
 Gaseous induction is an acceptable alternative with a
non-irritant volatile anesthetic agent such as
sevoflurane or halothane especially when IV cannula is
not in place. However IV induction should be preferred
if child has signs of raised ICP, stupor or delayed
gastric emptying.
 Muscle relaxation can be achieved with the use of a
neuromuscular blocking agent.
 In case of very large head, intubation can be very
difficult. Proper positioning using pad/bolster below the
shoulder can help overcome the problem.
 Anethesia can be maintained with volatile agent & a
mixture of O2 + air.
 Core temperature should be monitored to maintain
normothermia. 23
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Hydrocephalus

  • 1.
    HYDROCEPHALUS Presented by :Dr. Vishal kr. Kandhway 1
  • 2.
    Introduction  Derived fromGreek word “Hydro” meaning “Water”, and “Cephalus”, meaning “Head”.  Excessive accumulation of cerebrospinal fluid (CSF) resulting in abnormal widening of the spaces in the brain.  The normal volume of CSF is 140 ml. CSF is produced by the choroid plexus in the ventricles at a rate of about 500 ml in 24 hours. 2
  • 3.
  • 4.
    Ventricles of TheBrain  Lateral Ventricle  Third Ventricle  Fourth Ventricle 4
  • 5.
    Circulation of CSF Normally, CSF flows continually from the interior cavities in the brain (ventricles) to the thin subarachnoid space that surrounds the brain and spinal cord. 5
  • 6.
  • 7.
     Normally, CSFflows through the ventricles, exits into cisterns (closed spaces that serve as reservoirs) at the base of the brain, bathes the surfaces of the brain and spinal cord, and then reabsorbs into the bloodstream. 7 Circulation of CSF
  • 8.
    The major clinicalfeatures in infants are:  failure to thrive  increased skull circumference  tense anterior fontanelle  ‘cracked pot’ sound on skull percussion  transillumination of cranial cavity with strong light  when severe, impaired conscious level and vomiting  ‘setting sun’ appearance due to lid retraction and impaired upward gaze from 3rd ventricular pressure on the midbrain tectum  thin scalp with dilated veins. 8
  • 9.
     The balancebetween production and absorption of CSF is critically important. Because CSF is made continuously, medical conditions that block its normal flow or absorption will result in an over- accumulation of CSF. The resulting pressure of the fluid against brain tissue is what causes Hydrocephalus. 9 Cause of Hydrocephalus
  • 10.
  • 11.
    Types of Hydrocephalus There are several different types of Hydrocephalus:  Congenital hydrocephalus  Acquired hydrocephalus  Communicating hydrocephalus  Non-communicating hydrocephalus 11
  • 12.
    Congenital Hydrocephalus  Congenitalhydrocephalus is present at birth and may be caused by environmental influences during fetal development or by genetic factors.  Causes are:  Aqueduct Stenosis  Colloid Cyst 12
  • 13.
    Acquired Hydrocephalus  Developsat the time of birth or at some point afterward. This type of hydrocephalus can affect individuals of all ages and may be caused by injury or disease. 13
  • 14.
     Acquired Hydrocephalusis due to:  Tumors  Hemorrhage  Ventriculitis Acquired Hydrocephalus 14
  • 15.
    Communicating Hydrocephalus  Communicatinghydrocephalus occurs when the flow of cerebrospinal fluid (CSF) is blocked after it exits the ventricles. This form is called communicating, because the CSF can still flow between the ventricles, which remain open. 15
  • 16.
     Causes aredue to:  Post-hemorrhage  Bacterial Meningitis  Malignant Meningitis  Increased Venous Pressure Communicating Hydrocephalus 16
  • 17.
    Non-Communicating Hydrocephalus  Non-communicatinghydrocephalus, also called "obstructive" hydrocephalus, occurs when the flow of CSF is blocked along one or more of the narrow pathways connecting the ventricles.  Causes include:  Congenital  Acquired 17
  • 18.
    Treatment  In general,the treatment of hydrocephalus is a CSF shunt or a 3rd ventriculostomy. If there has been rapid neurological deterioration this will need to be performed as an emergency.  If the hydrocephalus is due to an obstructing tumour that is surgically accessible, resection of the mass may lead to resolution of the hydrocephalus and a shunt might not be necessary.
  • 19.
    CSF shunt  Theusual method of CSF diversion is a ventriculoperitoneal shunt, in which a catheter is placed into the lateral ventricle and is connected to a subcutaneous unidirectional pressure- regulated valve which is attached to a catheter threaded subcutaneously down to the abdomen and inserted into the peritoneal cavity.  Modern valves can have their draining pressures adjusted percutaneously and shunts are being developed allowing intracranial pressure to be monitored percutaneously.
  • 20.
    Complications of ventriculoperitonealshunt The major possible complications are: • infection of the shunt • obstruction of the shunt • intracranial haemorrhage.
  • 21.
    Other CSF shunts:  ventriculoatrial shunts  ventriculopleural shunts  lumboperitoneal shunts.
  • 22.
    Anesthetic consideration forVP Shunt  The child should be carefully monitored whilst awaiting surgery for signs of neurological deterioration as this can occur rapidly.  If patient suffers resp. arrest or sudden deterioration in neurological ststus due to raised ICP & tonsillar herniation, ET intubation & hyperventilation with with 100% O2 should be carried out.  The goal for induction is to avoid any further rise in ICP & avoid hypotension. IV or Inhalational induction can be used in child with normal ICP.  IV induction allows rapid control of airway in emergency situations(raised ICP). 22
  • 23.
     Gaseous inductionis an acceptable alternative with a non-irritant volatile anesthetic agent such as sevoflurane or halothane especially when IV cannula is not in place. However IV induction should be preferred if child has signs of raised ICP, stupor or delayed gastric emptying.  Muscle relaxation can be achieved with the use of a neuromuscular blocking agent.  In case of very large head, intubation can be very difficult. Proper positioning using pad/bolster below the shoulder can help overcome the problem.  Anethesia can be maintained with volatile agent & a mixture of O2 + air.  Core temperature should be monitored to maintain normothermia. 23
  • 24.