Brief notes on the inflammation of Alveolar bone that surrounds a tooth that has recently been extracted. It occurs as a complication of tooth extraction.
Brief notes on the inflammation of Alveolar bone that surrounds a tooth that has recently been extracted. It occurs as a complication of tooth extraction.
Gingivitis is a form of gum disease characterised by reversible gingival inflammation without destruction of tooth-supporting tissues, periodontal ligament or bone
Halitosis is derived from a Latin word which means unpleasant breath. If not treated, it could affect your social life. Majority of the cases of halitosis have oral origin. Therefore, appropriate dental treatment eliminates the cause.
clinical assessment and treatment of oral malodour and halitosis.
includes microorganisms causing halitosis, volatile sulphur compounds and its relation to periodontal destruction.
The Gold Standard Antiseptic in Dentistry. Its composition, mechanism of action, available forms, uses, disadvantages. Its role in Periodontics. Done by : Ivan Obadiah (CRI) Guided by : Dr. Veejay Chandran (MDS).
Periodontitis is a chronic, slowly progressing disease which mainly results in the destruction of tooth supporting apparatus. Earlier it was classified as Chronic and Aggressive periodontitis with different clinical features and etiology. Current classification ( 2017) of periodontal disease involves periodontitis with is further divided into 4 stages and 3 grades depending on severity and rate of disease progression respectively. Diabetes meelitus and smoking are the validated risk factors for the progression of periodontitis.
common oral baits like tongue thrusting,nail biting,thumb sucking, lip biting, mouth breathing have been described in detail with their clinical features,oral manifestations and treatment and prevention part. removable and fixed appliances have been described in brief for various habits.
Misconceptions exist in relation to halitosis, including:
Low prevalence
Aetiology: gastrointestinal origin
No reference practitioner exists
It has no solution or treatment
In this presentation we will debunk these misconceptions...
Gingivitis is a form of gum disease characterised by reversible gingival inflammation without destruction of tooth-supporting tissues, periodontal ligament or bone
Halitosis is derived from a Latin word which means unpleasant breath. If not treated, it could affect your social life. Majority of the cases of halitosis have oral origin. Therefore, appropriate dental treatment eliminates the cause.
clinical assessment and treatment of oral malodour and halitosis.
includes microorganisms causing halitosis, volatile sulphur compounds and its relation to periodontal destruction.
The Gold Standard Antiseptic in Dentistry. Its composition, mechanism of action, available forms, uses, disadvantages. Its role in Periodontics. Done by : Ivan Obadiah (CRI) Guided by : Dr. Veejay Chandran (MDS).
Periodontitis is a chronic, slowly progressing disease which mainly results in the destruction of tooth supporting apparatus. Earlier it was classified as Chronic and Aggressive periodontitis with different clinical features and etiology. Current classification ( 2017) of periodontal disease involves periodontitis with is further divided into 4 stages and 3 grades depending on severity and rate of disease progression respectively. Diabetes meelitus and smoking are the validated risk factors for the progression of periodontitis.
common oral baits like tongue thrusting,nail biting,thumb sucking, lip biting, mouth breathing have been described in detail with their clinical features,oral manifestations and treatment and prevention part. removable and fixed appliances have been described in brief for various habits.
Misconceptions exist in relation to halitosis, including:
Low prevalence
Aetiology: gastrointestinal origin
No reference practitioner exists
It has no solution or treatment
In this presentation we will debunk these misconceptions...
Now-a-days bad breath affects a large proportion of population. Inorder to overcome bad breath, people should practice good oral hygiene habits. Through this video one can know about the cause and treatment for bad breath.
For more information please click :: https://www.youtube.com/watch?v=HrQX29wT7EQ&t=11s
Instruments used in oral and maxillofacial surgeryCing Sian Dal
Instruments used in oral and maxillofacial surgery
Copyright (c) Prof. Dr. U Ko Ko Maung
Department of Oral and Maxillofacial Surgery
University of Dental Medicine, Yangon
Instruments used in oral and maxillofacial surgeryCing Sian Dal
Instruments used in oral and maxillofacial surgery
Copyright (c) Dr. Ko Ko Maung
Department of Oral & Maxillofacial Surgery
University of Dental Medicine, Yangon
Practical Points of View for Removable Partial DentureCing Sian Dal
Practical Points of View for Removable Partial Denture
Copyright (c) Dr. Myint Kyaw Thu
Department of Prosthodontics
University of Dental Medicine, Yangon
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
263778731218 Abortion Clinic /Pills In Harare ,sisternakatoto
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- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
2. Definition
❖ Halitosis is derived from Latin.
❖ It means breath and osis, an abnormal or diseased
conditions.
❖ Unpleasant foul offensive odour; bad breath; malodour;
fetor ex oris (ore)
3. Causes / Etiology
❖ The primary cause of halitosis is the uncontrolled growth
of gram negative anaerobic bacteria.
4. ❖ Oral microbes are known to produce volatile sulphur
compounds, namely methly mercaptan (CH3SH),
hydrogen sulphide (H2S) and dimethyl sulfide (CH3)2S.
❖ The most commonly involved bacteria are
Porphyromonas gingivalis, Prevotella intermedia /
nigrescens, Aggregatibacter actinomycetemcomitans,
Campylobacter rectus, Fusobacterium nucleatum,
Peptostreptococcus micros, Tannerella forsythia,
Eubacterium spp, and spirochetes.
5. ❖ These volatile sulphur compounds are the primary
components of oral malodour.
❖ These are formed through the putrefactive action of oral
microbes on sulphur containing amino acids, peptones or
proteins found in the mouth.
6. Production of volatile sulfur compounds causing
halitosis
Proteins in diet
Proteins in
GCF
Proteins in
saliva
Peptid
esBacteria
proteases
Host proteases Sulfur containing
amino acids
Volatile sulfur
compounds
Halitosis
Other amino
acids
Anaerobic
gram
negative
bacteria
7. ❖ Many components besides sulfide components (e.g.,
diamines) in the GCF and saliva can be malodourous.
❖ First, pH has to be above the neutrality (i.e. basic) to
cause halitosis.
❖ Conversely, an acidic pH is inhibitory to the proliferation
of gram negative, anaerobic bacteria and consequently
low pH leads to reduction of malodour.
8. ❖ Second, reduced flow of saliva allows colonization of
bacteria on the teeth and soft tissue, thereby promoting
the production of odorous gases.
❖ Other related important factors are age, genders, habits
and diurnal variation.
9. A. Local causes (Oral Origins
90%)
❖ Poor oral hygiene
❖ Periodontal diseases are strongly associated with bad
breath. The concentration of volatile sulphur compounds
increases with severity of periodontal diseases.
❖ Carious lesions (especially with food particles)
❖ Tongue coating
❖ Food impaction
❖ Poor dental prothesis hygiene
10. ❖ Surgical or extraction sites
❖ Oral infections or abscess
❖ Soft tissue lesion with ulcerations, bleeding or necrosis
❖ Ingestion of highly flavored food and beverages
❖ Heavy smoking
❖ Xerostomia
❖ Allergic conditions
❖ Oral carcinoma
11. B. Systemic causes (Non-oral
origins)
❖ Upper and lower respiratory tract diseases (e.g.,
bronchitis, bronchiectasis)
❖ Gastro-intestinal tract diseases (e.g., gastric hernia)
❖ Pathologic conditions of peri-oral structures (e.g.,
purulent sinusitis, chronic pharyngitis, post-nasal drip or
discharges)
❖ Stress or nervous tension is a major enhance of bad
breath. One major effect of stress is drying of the mouth.
12. ❖ Onset of menstruation
❖ Other systemic causes of breath malodour includes renal
(uremia / fishy), pancreatic (acetone) and liver
(ammonium)
15. Genuine halitosis
❖ Genuine halitosis is the term that is used when the
breath malodour really exists and can be diagnosed
organoleptically or by measurement of the responsible
compounds.
16. Psedo-halitosis
❖ When an obvious breath malodour cannot be perceived,
but the patient is convinced that he is she suffers from it,
this is called pseudo-halitosis.
17. Halitophobia
❖ If the patient still believes that there is bad breath after
treatment of genuine halitosis or diagnosis of pseudo-
halitosis, one considers halitophobia, which is a
recognized psychiatric condition.
19. A. Diagnosis
i. Patient history
❖ There is a saying “Listening to the patient and he will tell
you the diagnosis”
20. ❖ The common questions are about the frequency of odor,
the time of appearance within the day, the time of
appearance within the day, whether others have
identified the problems, what kind of medications are
taken and whether dryness of the mouth noticed.
21. ii. Clinical and Laboratory
exmination
1. Gas chromatography or combined with mass
spectroscopy
2. Sulphide monitor system (Helimeter)
3. Organoleptic measurement
22. B. Treatment
❖ Local measures
❖ Systemic measures - specific treatment of systemic
cause if present
❖ Psychotherapy (if needed)
23. Classification of halitosis on Treatment
need (TN)
No Classification Treatment Needs
I Genuine halitosis
A. Physiologic halitosis TN 1
B. Pathologic halitosis
a. Oral Pathologic halitosis TN-1 and TN-2
b. Extra Oral Pathologic halitosis TN-1 and TN-3
II Pseudo halitosis TN-1 and TN-4
III Halitophobia TN-1 and TN-5
24. Treatment Needs (TN) for
breath odor
Category Description
TN-1
Explanation of halitosis and instructions for oral hygiene
(Support and reinforcement of patient’s own self care for further
improvement in their oral hygiene)
TN-2
Oral prophylaxis, professional cleaning and treatments for oral
diseases
TN-3 Referral to a physician or medical specialist
TN-4
Explanation of examination date, further professional instruction,
education and reassurance
TN-5
Referral to a clinical psychologist or other psychological
specialists
25. Local measures
❖ An etiologic treatment is to be preferred.
❖ The treatment of oral malodour consists of the elimination of the local
pathology / cause present.
❖ If underlying disease is suspected, the patient should be referred to
the respective specialists.
❖ Thorough plaque control to remove plaque and food particle is a
daily necessity.
❖ Regular check up to correct problems areas; common oral diseases,
faulty restorations, leaking crowns, all of which causes food trap is a
must.
26. ❖ Instructions on proper oral hygiene be reinforced and
professional oral prophylaxis are essential.
❖ Inter-proximal cleaning is relatively more important
because toothbrush bristles do not gain access to inter-
proximal areas.
❖ Further, the tongue is probably some of the important
reservoirs of bacteria involved in halitosis and tongue
scraping should be an important component of the
treatment.
27. ❖ Mouth rinses are valuable adjuncts but do not provide the best
sole treatment.
❖ Chlorhexidine is the mouth rinse that demonstrates
considerable reduction of malodour.
❖ It is well established that zinc containing mouth rinses have the
property of the complex the divalent sulphur radicals, reducing
the important cause of malodour.
❖ (Zinc can block the biologic effects of volatile sulphur
compounds on protein synthesis)
❖ The use of hydrogen peroxide rinse also offers positive
perspectives, mouth rinses are best used before bed time.
28. ❖ Any treatment that promotes increased level of saliva
and tongue action will be of help to reduce malodour
especially when oral dryness is at stage.
❖ This can mean a proper fluid intake or the use of
sugarless chewing gum, candy or the fibrous vegetables.
❖ Vit C deficiency may be a cofactor on bad breath.
❖ Smokers especially should take regular supplements of
Vit C since the nicotine in the cigarettes destroys Vit C.