The document provides information on the facial nerve (cranial nerve VII), including its embryology, nuclei, course, branches, landmarks, neurophysiology, causes of damage, and grading systems for facial palsy. It describes the facial nerve's development during gestation, its motor, sensory and parasympathetic functions. Key points along its intra- and extracranial course are identified. Variations, injuries, and resulting functional deficits are also discussed.
1. Sternberg's canal also known as lateral craniopharyngeal canal
2. lateral craniopharyngeal canal
3. temporal lobe encephalocele in sphenoid sinus
4. its is as differential diagnosis for Arachnoid Pit and Extensive Sinus Pneumatization as the Cause of Spontaneous Lateral Intra sphenoidal Encephalocele
Bony orbits are Quadrangular truncated pyramids with Anterior cranial fossa above and the maxillary sinuses below.
in this presentation we study the detailed anatomy of the arbit, the bones, relations of each wall, the contents, the apertures, orbital fissures and structures passing, fascia, septa and the surgical spaces of the orbit
Facial nerve traumatic injury and repairsarita pandey
knowledge of anatomy of facial nerve is essential for ENT practitioner,
the worldwide acknowledged high trauma in south africa often results in head and neck injuries, resulting in facial nerve injury
summary of the anatomy, classifications of injuries, and management principles touched upon
indepth surgical procedures out of scope of this slideshow
1. Sternberg's canal also known as lateral craniopharyngeal canal
2. lateral craniopharyngeal canal
3. temporal lobe encephalocele in sphenoid sinus
4. its is as differential diagnosis for Arachnoid Pit and Extensive Sinus Pneumatization as the Cause of Spontaneous Lateral Intra sphenoidal Encephalocele
Bony orbits are Quadrangular truncated pyramids with Anterior cranial fossa above and the maxillary sinuses below.
in this presentation we study the detailed anatomy of the arbit, the bones, relations of each wall, the contents, the apertures, orbital fissures and structures passing, fascia, septa and the surgical spaces of the orbit
Facial nerve traumatic injury and repairsarita pandey
knowledge of anatomy of facial nerve is essential for ENT practitioner,
the worldwide acknowledged high trauma in south africa often results in head and neck injuries, resulting in facial nerve injury
summary of the anatomy, classifications of injuries, and management principles touched upon
indepth surgical procedures out of scope of this slideshow
A seminar on nerve supply of head and neck.
Hi, I am Dr Komal Ghiya, pediatric dentist by profession, I am here to share some of my own presentations for educational purposes. I hope you a presentation on NERVE SUPPLY OF HEAD AND NECK will be useful for all the dental and medical students. Comments are welcome if you like the presentations and if not please suggest some ways I could make them better for you. All the best
osteology of head and neck and its applied aspectsSwetha Srivani
knowing the correct anatomy and applied aspect of osteology helps in accurate diagnosis.this ppt provides insight into different bones of head and neck and their applied aspects through images.
Details about the anatomy with clinical importance. An easy guide for understanding the walls, surgical spaces, orbital contents, venous and arterial supply. Hope its helpful for your examinations too!!
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
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3. INTRODUCTION
• THE FACIAL NERVE IS THE VII
CRANIAL NERVE.
• IT IS THE NERVE OF THE SECOND
BRANCHIAL ARCH.
4. EMBRYOLOGY
MAIN PATTERN OF THE NERVE’S COMPLEX COURSE, BRANCHING PATTERN
AND RELATIONSHIPS ARE ESTABLISHED DURING THE FIRST THREE MONTHS
OF PRENATAL LIFE.
DURING THIS PERIOD, THE MUSCLES OF EXPRESSION DIFFERENTIATE,
BECOME FUNCTIONAL AND ACTIVELY CONTRACT.
IMPORTANT STEPS IN FACIAL NERVE DEVELOPMENT OCCUR THROUGH OUT
GESTATION AND THE NERVE IS NOT FULLY DEVELOPED UNTIL 4 YEARS
5. TIME DURING GESTATION THAT ANATOMICAL
STRUCTURES APPEAR
• 3RD WEEK: FACIOACOUSTIC MEATUS DEVELOPS
• 4TH WEEK: FACIAL NERVE DIVIDES INTO TWO PARTS
• 5TH WEEK: GENICULATE GANGLION, NERVUS INTERMEDIUS, GREATER
PETROSAL NERVE VISIBLE
• 7TH & 8TH WEEK: MUSCLES OF FACIAL EXPRESSION DEVELOP
• 11TH WEEK: FACIAL NERVE ARBORIZED.
6. NUCLEI
• THE FIBRES OF THE NERVE ARISE FROM 4 NUCLEI SITUATED AT LOWER PONS:
1. BRACHIAL NUCLEUS (MOTOR NUCLEUS)
2. SUPERIOR SALIVATORY NUCLEUS (PARASYMPATHETIC NUCLEUS)
3. NUCLEUS OF TRACTUS SOLITARIUS (TASTE)
4. SPINAL TRIGEMINAL NUCLEUS (GENERAL SENSATION)
7. IT IS A MIXED NERVE
MOTOR: MUSCLES OF EXPRESSION, STYLOHYOID MUSCLE, POSTERIOR
BELLY OF DIGASTRIC AND STAPEDIUS MUSCLE.
PARASYMPATHETIC: SUBMANDIBULAR GLAND, SUBLINGUAL GLAND,
NASAL GLANDS, PALATINE GLANDS AND LACRIMAL GLANDS.
SPECIAL SENSORY: TASTE FROM ANTERIOR 2/3RD OF THE TONGUE.
SENSORY: SKIN AROUND THE EAR
8. BRACHIMOTOR NUCLEUS
IT IS DIVIDED INTO AN UPPER PART AND A LOWER PART
THE UPPER PART IS CONTROLLED BY THE LEFT AND THE RIGHT SIDE OF
THE BRAIN.
THE UPPER PART OF THE NUCLEUS IS RESPONSIBLE FOR SUPPLYING THE
MUSCLES OF THE UPPER PART OF THE FACE (ORBICULARIS OCULI,
FRONTALIS MUSCLE, CORRUGATOR SUPERCILI, NASALIS, LEVATOR LABII
SUPERIORIS)
THE LOWER PART OF THE NUCLEUS IS CONTROLLED BY THE OPPOSITE OF
10. COURSE AND RELATIONS
THE COURSE OF THE FACIAL NERVE IS VERY COMPLEX SINCE THERE ARE
MANY BRANCHES.
THE COURSE CAN BE DIVIDED INTO THREE PARTS:
INTRACRANIAL
INTRATEMPORAL
EXTRACRANIAL
11. INTRACRANIAL COURSE
THE NERVE ARISES IN THE PONS, AN AREA
OF THE BRAINSTEM. IT BEGINS AS TWO
ROOTS: A LARGE MOTOR ROOT AND A
SMALL SENSORY ROOT
THE TWO ROOTS TRAVEL THROUGH THE
INTERNAL ACOUSTIC MEATUS, A 1CM
LONG OPENING IN THE PETROUS PART OF
THE TEMPORAL BONE WHERE THEY ARE IN
VERY CLOSE PROXIMITY TO THE INNER
EAR.
IT TRAVELS SLIGHTLY ABOVE AND
TH
13. INTRATEMPORAL
BRANCHES
NERVE TO STAPEDIUS: ARISES FROM THE MASTOID SEGMENT
AND SUPPLIES THE STAPEDIUS MUSCLE.
CHORDA TYMPANI: ARISES FROM THE MASTOID SEGMENT,
MOVES ANTERIORLY BETWEEN INCUS & MALLEUS AND JOINS
THE LINGUAL NERVE TO SUPPLY THE ANTERIOR 2/3RD OF
THE TONGUE.
IT ALSO SUPPLIES PARASYMPATHETIC FIBRES TO
SUBMANDIBULAR AND SUBLINGUAL GLANDS.
SENSORY FIBRES: JOINS THE AURICULAR BRANCH OF VAGUS
14. MORPHOLOGIC PECULIARITIES (GERRIER
1977)
a) INDIVIDUAL SHEATH OF PIA MATER CURVES UP AND CONTINUES
WITH ARACHINOID.
b) SLIGHT CONSTRICTION OF THE NERVE JUST PRIOR TO THE
LABYRINTH SEGMENT WHICH IS A NORMAL CONSTRICTION.
c) CHANGE IN THE DIRECTION OF THE NERVE THAT PRODUCES AN
ANGLE OF 132°
15. THE NERVE THEN EMERGES OUT OF THE STYLOMASTOID FORAMEN
IN A NEWBORN, THE STYLOMASTOID FORAMEN IS AT A HIGHER LEVEL
WITH THE FACIAL NERVE EMERGING AT THE LEVEL OF THE MASTOID
ANTRUM
16. EXTRACRANIAL COURSE
AFTER EXITING THE FORAMEN, IT GIVES RISE TO THE:
POSTERIOR AURICULAR NERVE: WHICH ASCENDS IN FRONT OF THE MASTOID PROCESS AND
PROVIDES MOTOR INNERVATION TO THE INTRINSIC AND EXTRINSIC MUSCLES OF THE OUTER
EAR. IT ALSO SUPPLIES THE OCCIPITAL PART OF THE OCCIPITOFRONTALIS MUSCLE.
NERVE TO THE POSTERIOR BELLY OF THE DIGASTRIC MUSCLE: INNERVATES THE POSTERIOR
BELLY OF THE DIGASTRIC MUSCLE WHICH IS RESPONSIBLE FOR RAISING THE HYOID BONE.
NERVE TO THE STYLOHYOID MUSCLE: INNERVATES THE STYLOHYOID MUSCLE WHICH IS
RESPONSIBLE FOR RAISING THE HYOID BONE.
17. THE MAIN TRUNK OF THE NERVE CONTINUES ANTERIORLY AND INFERIORLY INTO THE PAROTID
GLAND AND TERMINATES BY SPLITTING INTO FIVE BRANCHES:
TEMPORAL BRANCH: INNERVATES THE FRONTALIS, ORBICULARIS OCULI AND CORRUGATOR
SUPERCILII
ZYGOMATIC BRANCH: INNERVATES THE ORBICULARIS OCULI.
BUCCAL BRANCH: INNERVATES THE ORBICULARIS ORIS, BUCCINATOR
AND ZYGOMATICUS MUSCLES.
MARGINAL MANDIBULAR BRANCH: INNERVATES THE MENTALIS MUSCLE.
CERVICAL BRANCH: INNERVATES THE PLATYSMA
18. SUMMARY OF BRANCHES OF FACIAL NERVE
A: WITHIN THE FACIAL CANAL:
1. GREATER PETROSAL NERVE
2. NERVE TO STAPEDIUS
3. THE CHORDA TYMPANI
19. B: AT ITS EXIT FROM STYLOMASTOID FORAMEN
1. POSTERIOR AURICULAR NERVE
2. DIGASTRIC
3. STYLOHYOID
20. C: TERMINAL BRANCHES WITHIN THE PAROTID GLAND
1. TEMPORAL
2. ZYGOMATIC
3. BUCCAL
4. MARGINAL MANDIBULAR
5. CERVICAL
21. VASCULAR SUPPLY
THE FACIAL NERVE GETS IT’S BLOOD SUPPLY FROM:
1. ANTERIOR INFERIOR CEREBELLAR ARTERY – AT THE
CEREBELLOPONTINE ANGLE
2. LABYRINTHINE ARTERY (BRANCH OF ANTERIOR INFERIOR CEREBELLAR
ARTERY) – WITHIN INTERNAL ACOUSTIC MEATUS
3. SUPERFICIAL PETROSAL ARTERY (BRANCH OF MIDDLE MENINGEAL
ARTERY) – GENICULATE GANGLION AND NEARBY PARTS
22. 4. STYLOMASTOID ARTERY (BRANCH OF POSTERIOR AURICULAR
ARTERY) – MASTOID SEGMENT
5. POSTERIOR AURICULAR ARTERY SUPPLIES THE FACIAL NERVE
DISTAL TO STYLOMASTOID FORAMEN.
6. VENOUS DRAINAGE PARALLELS THE ARTERIAL BLOOD SUPPLY
24. LANDMARKS FOR EXTRATEMPORAL PART
TRAGAL POINTER OF CONLEY: THE NERVE IS
LOCATED MEDIAL AND ABOUT 1 CM
INFERIOR TO THE TRAGAL CARTILAGE
TYMPANOMASTOID SUTURE: THIS IS LOCATED
AT THE APEX OF THE VAGINO-MASTOID ANGLE
OR VALLEY OF THE NERVE .
THE FACIAL NERVE RUNS DEEP TO THIS SUTURE.
25. STYLOID PROCESS: THE NERVE PASSES LATERAL
TO THE STYLOID.
TENDON OF POSTERIOR BELLY OF DIGASTRIC
MUSCLE: THE MAIN TRUNK OF THE NERVE CAN BE
FOUND MIDWAY BETWEEN THE CARTILAGINOUS POINTER
OF THE EXTERNAL ACCOUSTIC MEATUS AND THE POSTERIOR
BELLY OF DIGASTRIC MUSCLE.
POSTERIOR AURICULAR VEIN OR THE RETROMANDIBULAR VEIN
26. LANDMARKS IN THE MASTOID AND MIDDLE EAR
THE COG: IT IS A BONY RIDGE WHICH HANGS FROM THE TEGMEN,
ANTERIOR TO THE HEAD OF THE MALLEUS AND IS USEFUL FOR
IDENTIFYING THE
FIRST GENU
COCHLEARIFORM PROCESS: IT IS INFERIOR TO THE ANTERIOR
PORTION OF THE TYMPANIC SEGMENT OF THE FACIAL NERVE.
27. OVAL WINDOW: IT IS A USEFUL GUIDE TO THE POSTERIOR PORTION OF THE HORIZONTAL
SEGMENT OF THE NERVE. THE NERVE LIES ABOVE THE OVAL WINDOW
LATERAL SEMICIRCULAR CANAL: IT LIES POSTERIOR-
SUPERIOR TO THE SECOND GENU AND IS A
CONSTANT LANDMARK.
RETROFACIAL AIR CELLS: IT HELPS IN DELINEATING
THE MEDIAL ASPECT OF THE VERTICAL SEGMENT
OF THE FACIAL NERVE.
28. LANDMARKS IN THE MIDDLE CRANIAL FOSSA
THE GREATER PETROSAL NERVE IS IDENTIFIED AND FOLLOWED BACKWARDS
TO THE GENICUALATE GANGLION AND FACIAL NERVE IS IDENTIFIED.
IDENTIFICATION OF THE INTERNAL AUDITORY CANAL SINCE IT LIES IN THE
SAME CORONAL PLANE WITH THE EXTERNAL AUDITORY CANAL.
THE BULGE OF THE SEMICIRCULAR CANAL CAN BE IDENTIFIED AND
SUBSEQUENTLY THE INTERNAL AUDITORY CANAL IS IDENTIFIED.
30. • VARIATION 1: FACIAL NERVE GOES
ABOVE THE LATERAL
SEMICIRCULAR CANAL
• VARIATION 2: FACIAL NERVE GOES
ABOVE THE FOOT PLATE OF STAPES
• VARIATION 3: GOES IN BETWEEN
THE COURA OF THE STAPES.
VARIATIONS IN TYMPANIC
SEGMENT
31. • VARIATION 4: GOES INFERIOR TO
THE FOOT PLATE OF STAPES
• VARIATION 5: GOES IN BETWEEN THE
OVAL WINDOW & ROUND WINDOW
• VARIATION 6: GOES BELOW ROUND
WINDOW
• VARIATION 7: NERVE SPLITS
32. VARIATIONS IN MASTOID
SEGMENT
• VARIATION 1: (DORSAL HUMP): HUMPS BELOW
LATERAL
SEMICIRCULAR CANAL
• VARIATION 2: CURVES ANTERIORLY AND
CONTINUES
ABOVE THE FOOT PLATE
• VARIATION 3: VERTICAL PART OF THE NERVE
33. • VARIATION 4: VERTICAL PART OF THE FACIAL
NERVE GOES VERY POSTERIORLY AND STAYS
ABOVE THE SIGMOID SINUS
• VARIATION 5: BIFURCATION OR TRIFURCATION
• VARIATION 6: HYPOPLASIA OF VERTICAL PART
OF FACIAL NERVE
35. NEUROPHYSIOLOGY OF FACIAL NERVE
FACIAL EXPRESSION DEPENDS ON 7000 MOTOR FIBRES OF THE FACIAL
NERVE FIRING IN UNISON TO BRING ABOUT MUSCULAR CONTRACTION
36. DEGREES OF NERVE INJURY
SEDDON (1943) DESCRIBED THREE TYPES OF NERVE INJURY:
NEUROPRAXIA: PRESSURE ON A PERIPHERAL NERVE CAN BLOCK THE
TRANSMISSION OF THE IMPULSES WITHOUT DEATH AND DEGENERATION OF
THE AXON BEYOND THE SITE OF PRESSURE.
ASSOCIATED WITH LOSS OF MYELIN. THIS IS A REVERSIBLE CONDUCTION
BLOCK.
AXONTEMESIS: SECTIONING OF AN AXON AND RESULTS IN DEATH OF THE
BLOCKED DISTAL SEGMENT.
NEURONTEMESIS: SECTIONING OF THE ENTIRE NERVE TRUNK
37. SUNDERLAND (1978) CLASSIFIED NERVE INJURIES INTO 5 DEGREES.
FIRST DEGREE: INDICATES COMPRESSION OF THE NERVE. IT IS REVERSIBLE.
SECOND DEGREE: THERE IS INTERRUPTION OF THE AXON AND MYELIN.
OCCURS WHEN THE COMPRESSION PERSISTS. HERE, THERE IS LOSS OF AXON
BUT ENDONEURIUM IS INTACT. RECOVERY MAY TAKE MORE THAN 1 – 2
MONTHS.
THIRD DEGREE: THERE IS LOSS OF MYELIN TUBES DUE TO INCREASED
INTRANEURAL PRESSURE. RECOVERY TAKES ABOUT 2 – 4 MONTHS. THERE
MAY NOT BE COMPLETE RECOVERY
FOURTH DEGREE: PARTIAL TRANSECTION OF THE NERVE. RECOVERY IS
POOR.
38. HOUSE AND BRACKMAN FACIAL NERVE GRADING
SYSTEM
APPROVED BY AMERICAN ACADEMY OF OTOLARYNGOLOGY
GRADE I: NORMAL
GRADE II: MILD DYSFUNCTION
FOREHEAD MOTION IS MODERATE TO GOOD.
SLIGHT ASYMMETRY OF THE MOUTH.
EYE CLOSURE COMPLETE
GRADE III: MODERATE DYSFUNCTION
FOREHEAD MOTION IS SLIGHT TO MODERATE.
WEAKNESS OF ANGLE OF THE MOUTH ON MAXIMAL EFFORT
EYE CLOSURE IS INCOMPLETE WITH MAXIMAL EFFORT
39. GRADE IV: MODERATELY SEVERE DYSFUNCTION
NO FOREHEAD MOTION
MOUTH MOVEMENT IS COMPLETE ON MAXIMAL EFFORT
EYE CLOSURE IS INCOMPLETE WITH MAXIMAL EFFORT
GRADE V: SEVERE DYSFUNCTION
NO FOREHEAD MOTION
VERY SLIGHT MOUTH MOVEMENT
EYE CLOSURE IS INCOMPLETE
GRADE VI: TOTAL PARALYSIS
41. DAMAGE TO THE FACIAL NERVE
THE FACIAL NERVE HAS A WIDE RANGE OF FUNCTIONS. THUS,
DAMAGE TO THE NERVE CAN PRODUCE A VARIED SET OF
SYMPTOMS, DEPENDING ON THE SITE OF THE LESION.
42. INTRACRANIAL LESIONS
• THE MUSCLES OF FACIAL EXPRESSION WILL BE PARALYZED OR SEVERELY
WEAKENED.
• CHORDA TYMPANI – REDUCED SALIVATION AND LOSS OF TASTE ON THE
IPSILATERAL 2/3 OF THE TONGUE.
• NERVE TO STAPEDIUS – IPSILATERAL HYPERACUSIS (HYPERSENSITIVE TO
SOUND).
• GREATER PETROSAL NERVE – IPSILATERAL REDUCED LACRIMAL FLUID
PRODUCTION.
43. EXTRACRANIAL LESIONS
• PAROTID GLAND PATHOLOGY – E.G. A TUMOR, PAROTITIS,
SURGERY.
• INFECTION OF THE NERVE – PARTICULARLY BY THE HERPES VIRUS.
• COMPRESSION DURING FORCEPS DELIVERY – THE NEONATAL
MASTOID PROCESS IS NOT FULLY DEVELOPED, AND DOES NOT
PROVIDE COMPLETE PROTECTION OF THE NERVE.
47. SCHIRMER’S TEST
• A STRIP OF PAPER OF 5 CM X 0.5 CM IS PLACED ON THE LOWER
CONJUNCTIVAL FORNIX OF EACH EYE AND THE PATIENT IS
INTRODUCED TO INHALATION AMMONIA TO ENHANCE LACRIMATION.
• A REDUCED LACRIMATION BY 30% COMPARED TO THE NORMAL SIDE
IS SIGNIFICANT.
SALIVARY FLOW TEST
• A NO. 50/60 POLYETHYLENE CATHETER IS INTRODUCED TO BOTH WHARTON’S
PAPILLAE FOR ABOUT 3 MM.
• THE PATIENT IS GIVEN A FEW DROPS OF LEMON, AND THE NUMBER OF DROPS OF
SALIVA OVER ONE TO FIVE MINUTES IS MONITORED.
• A 25% REDUCTION BETWEEN THE SIDES IS SIGNIFICANT
48. TASTE SENSATION OF THE ANTERIOR 2/3RD OF THE TONGUE
• IT IS BEST ASSESSED USING A ELECTROGUSTOMETRY, HOWEVER, IT IS NOT
PROVED TO BE A USEFUL DIAGNOSTIC TOOL.
STAPEDIAL REFLEX
• THIS TEST ASSESSES THE STAPEDIAL MOVEMENTS BY TYMPANOMETRY.
NERVE CONDUCTION TIME
• IT IS USED TO TEST THE LATENCY RESPONSE OF A MUSCLE (INNERVATED BY
FACIAL NERVE) ON ELECTRICAL STIMULATION.
NERVE EXCITABILITY TEST
• PERFORMED BY STIMULATING THE NERVE AT THE STYLOMASTOID FORAMEN
AND IS DETERMINED BY A TWITCH RESPONSE IN THE FACIAL MUSCULATURE.
BLINK TEST
• THIS TEST IS DONE BY ELECTRICALLY STIMULATING THE NERVE AT THE
SUPRAORBITAL FORAMEN.
51. BELL’S PALSY
DESCRIBED BY SIR CHARLES BELL (1829)
IT IS AN ACUTE IDIOPATHIC LOWER MOTOR NEURON PALSY OF
THE FACIAL NERVE THAT IS UNILATERAL, SELF LIMITING, NON-
PROGRESSIVE, NON LIFE THREATENING AND SPONTANEOUSLY
REMITS BY 4-6 MONTHS AND ALWAYS BY 1 YEAR.
52. THEORIES
VASCULAR ISCHEMIC THEORY
IN THIS THEORY, THERE IS DECREASE IN CIRCULATION TO THE FACIAL
NERVE WHICH IS BELIEVED TO BE DUE TO THE INTERRUPTION OF A NUTRIENT
VESSEL.
VIRAL THEORY
IT IS CONCLUDED THAT BELL’S PALSY IS AN ACUTE BENIGN CRANIAL
POLYNEURITIS CAUSED BY THE REACTIVATION OF HERPES SIMPLEX VIRUS.
THE VIRUS REPLICATES IN THE GANGLION CELLS CAUSING LOCAL DAMAGE
AND HYPOFUNCTION OF THE NERVES THEREFORE PREVENTING
53. HEREDITARY THEORY
A FAMILIAL ANATOMIC VARIATION IN THE FACIAL CANAL (BONY
CONSTRICTION) MAY ACCOUNT FOR A GREATER TENDENCY TOWARDS
THE DEVELOPMENT OF FACIAL PALSY.
IT IS BELIEVED TO BE A RECESSIVE TRAIT.
54. EXAMINATION
DIFFERENTIATE BETWEEN UPPER MOTOR LESION (UML) OR LOWER MOTOR MOTOR
NEURON LESION (LML)
IN UML, THE FRONTALIS MUSCLE IS SPARED ALLOWING NORMAL FURROWING OF
EYE BROW AND EYE BLINKING.
IN LML, ALL THE MUSCLES OF FACIAL EXPRESSION ARE
AFFECTED ON ONE SIDE.
EYE CLOSURE (ORBICULARIS OCULI)
WIDE SMILE
BLOWING (BUCCINATOR, ORBICULARIS ORIS)
55. CLINICAL FEATURES
• RAPID ONSET OF MILD WEAKNESS TO TOTAL PARALYSIS ON ONE SIDE OF THE FACE.
• FACIAL DROOP AND DIFFICULTY IN MAKING EXPRESSIONS.
• INCREASED SENSITIVITY TO SOUND.
• DECREASED ABILITY TO TASTE.
• DRIPPING OF SALIVA (WEIR, PENTLAND & MURRAY, 1993)
• ACCUMULATION OF FOOD INSIDE THE CHEEK.
• PAIN AROUND THE JAW AND/OR BEHIND EAR OF THE AFFECTED SIDE.
• FLATTENING OF THE NASOLABIAL FOLD.
56. MANAGEMENT
MEDICAL MANAGEMENT
PREDNISOLONE 1MG/KG/DAY OR 60 MG GIVEN ORALLY
ACYCLOVIR 200 TO 400 MG GIVEN ORALLY
VASODILATORS LIKE XANITOL
ASCORBIC ACID
MULTIVITAMINS LIKE B1, B6 AND B12
SURGICAL TREATMENT
FACIAL NERVE DECOMPRESSION IN CASE OF NO IMPROVEMENT AFTER 3 WEEKS OF
MEDICAL TREATMENT.
57. CONCLUSION
SURGEONS HAVE TO PAY ATTENTION TO MINIMIZE THE RISK OF COMPLICATION
DURING PAROTIDECTOMY.
THE BEST WAY TO REDUCE IATROGENIC FACIAL NERVE INJURY IS TO HAVE A CLEAR
UNDERSTANDING OF THE ANATOMY, GOOD SURGICAL TECHNIQUE AND USE OF
MULTIPLE LANDMARKS.
THE PATIENT HAS TO BE INFORMED ABOUT THE COSMETIC SEQUELAE OF THE
INCISION AND ALL PATIENTS HAVE TO BE TOLD THAT FACIAL NERVE PARALYSIS IS
POSSIBLE AND CAN BE PARTIAL OR TOTAL, TEMPORARY OR PERMANENT.
Immediately distal to this, motor branches are sent to the posterior belly of the digastric muscle and to the stylohyoidmuscle.
parotid gland is innervated by the glossopharyngeal nerve).
The term facial palsy generally refers to weakness of the facial muscles, mainly resulting from temporary or permanent damage to the facial nerve
In this theory, there is decrease in circulation to the facial nerve which is believed to be due to the interruption of a nutrient vessel which could be due to compression of the facial nerve in the facial canal or due to presence of a thickened fibrous sheath.