1. The document discusses various occupational lung diseases caused by the inhalation of mineral and organic dusts. 2. It describes diseases such as coal worker's pneumoconiosis, silicosis, asbestosis, and berylliosis which are caused by inorganic dusts like coal, silica, asbestos, and beryllium. Diseases from organic dusts mentioned include byssinosis, farmer's lung, and bagassosis. 3. The signs and symptoms, diagnosis, and chest x-ray findings of some of these diseases are outlined including symptoms like cough, dyspnea, and fibrosis seen in conditions like silicosis, asbestosis and coal worker's pneumocon

1. CHEST X-RAY SEMINAR
By
Sailee Kamat Bambolkar
Karrishma Karampure
Mansi Karnik

2. Pneumoconiosis
It can be defined as a permanent alteration of lung
structure due to the inhalation of mineral dust and
the tissue reactions of the lung to its presence.
Strictly speaking the term ‘pneumoconiosis
includes those occupational lung diseases which
are due to exposure to inorganic or mineral dust
that is retained in the lung parenchyma and
incites fibrosis.’

3. Diseases due to
inorganic dust
Coal worker’s
pneumoconiosis
Silicosis
Asbestosis
Siderosis
Berylliosis
Diseases due to organic
dust
Bysinosis
Farmer’s lung
Malt worker’s lung
Mushroom worker’s
lung
Bagassosis
Aetiology

5. 1.
2.
3.
The type of lung disease varies according to the nature of the
inhaled dust. Some dusts are inert and cause no reaction at all
while the others cause immunologic damage and predispose to
tuberculosis or neoplasia.
The tissue response to the inhaled dust may be one of the following
3 types;
Fibrous nodules e.g. in coal worker’s pneumoconiosis and silicosis
Interstitial fibrosis e.g. Asbestosis
Hypersensitivity reaction e.g. Berylliosis.

6. Signs and symptoms
1.
2.
1.
2.
1.
2.
1.
2.
Simple Coal Worker’s Pneumoconiosis
Develops after prolonged exposure (15-20 years) to coal dust
Patients present with cough and dyspnoea.
Progressive Massive Fibrosis
This condition progresses even after the miner leaves the industry.
Clinically presence of cough with blackish expectoration and progressive
breathlessness.
Silicosis-Chronic or Simple Silicosis
Develops after many decades of exposure to relatively low levels of silica.
Upper lobes mostly involved, presence of gradually progressive dyspnoea dry
cough.
Acute Silicosis
Occurs after exposure to very high levels of dust over few months and fatal
within years.
Characterised by pulmonary oedema interstitial inflammation and
accumulation of proteinaceous surfactant like fluid within alveoli.

7. 1.
2.
1.
Asbestosis
Moderate to severe exposure for at least 10 years.
Clinically presence of progressive exertion dyspnoea, finger
clubbing and end inspiratory crepitations over the lung bases
Berylliosis
Presence of cough, progressive breathlessness, night sweats and
anthralgia.

8. Diagnosis
X-ray
CT scan
HRCT
Pulmonary Function Tests
Lung Biopsy
Specialised testing of lymphocyte function

9. (a) INORGANIC
SILICOSIS

11. ASBESTOSIS

12. COAL WORKERS PNEUMOCONIOSIS

13. Simple coal workers pneumoconios

14. BERYLLIOSS

15. (b) ORGANIC
BAGASOSIS

16. THANKYOU