Chemical eye trauma
•Download as PPTX, PDF•
1 like•785 views
Chemical eye trauma can occur from exposure to strong acids or alkalis and represents 11.5-22% of ocular injuries. Acids like sulfuric acid and hydrochloric acid, as well as alkalis like potassium hydroxide and sodium hydroxide, can cause chemical burns to the eye depending on the concentration, duration of exposure, and type of chemical. Clinical signs include lid edema, corneal scarring, uveitis, and glaucoma. Treatment involves immediate irrigation of the eye, antibiotic eye drops, atropine, steroid drops to reduce inflammation, and surgery for complications like symblepharon or glaucoma. Proper first aid through irrigation can help minimize tissue damage from chemical burns.
Report
Share
Report
Share
Recommended
Glaucoma and cataract include treatment
Glaucoma is a group of eye diseases characterized by increased intraocular pressure, optic nerve damage, and visual field loss. It is a leading cause of blindness worldwide. Cataracts are cloudy lenses that reduce vision and are most often age-related. They occur when the lens becomes less transparent over time. Risk factors for cataracts include diabetes, corticosteroid use, and glaucoma. Both conditions are generally treated surgically by removing the opaque lens or reducing eye pressure, but vision lost from glaucoma cannot be recovered. A combined study found diabetes significantly increases cataract risk while glaucoma is also an independent risk factor, potentially causing 5% of all cataracts.
Glaucoma
Glaucoma presentation for ophthalmology course, presented as a student seminar. Class location: ophthalmology unit, An-Najah National University Hospital.
Keratitis
Keratitis is an inflammatory condition that affects the cornea of your eye.
The cornea is the clear part that covers both the iris and the pupil.
Keratitis can be caused by an infection or injury to the eye.
Keratitis is a common condition.
People who wear contact lenses may experience keratitis more frequently than people who don’t wear contacts. In either case, you can take steps to help prevent this condition.
If you do develop keratitis, see your doctor right away.
Eye trauma
This document provides information on eye trauma, including causes, assessments, interventions, and nursing care. Key points include:
- Eye injuries are often preventable through safety measures. Common causes include accidents, assaults, and work or sports injuries.
- Assessment of eye trauma involves evaluating visual acuity, eye movements, pupil response, and checking for foreign bodies or orbital fractures. History should include injury details.
- Interventions depend on injury type but may include wound irrigation, patching, antibiotics, surgery for foreign bodies or fractures, and corticosteroids to reduce inflammation. Goals are to prevent infection and further damage.
- Nursing care focuses on monitoring for complications, educating on activity restrictions, and
Chemical injury to eye
This document provides information on chemical eye burns, including:
- Alkali burns are more dangerous than acid burns as alkalis penetrate rapidly into the eye.
- Immediate irrigation with saline or water for at least 20 minutes is essential to remove the chemical from the eye.
- The Dua classification system is useful for assessing prognosis based on limbal and conjunctival involvement.
- Medical management focuses on removing the chemical, promoting healing, controlling inflammation and infection, and managing intraocular pressure.
Dry eye presentation latest-Dr Dildar Singh
This document reviews diagnostic approaches and treatments for dry eye disease. It begins by defining dry eye as a multifactorial disease resulting from tear film instability and inflammation of the ocular surface. Diagnostic assessments include both subjective evaluations using questionnaires and objective evaluations of the tear film, ocular surface, and inflammation using tests such as tear osmolarity, tear breakup time, corneal staining, and meibomian gland evaluation. Treatments discussed include anti-inflammatory therapies like cyclosporine A and corticosteroids, supplements like essential fatty acids and nerve growth factor, and procedures like punctal occlusion and artificial tear implants.
Ocular chemical injury
chemical injury to eye by alkali, acids and irritants.
pathophysiology and management.
recent advances in management.
ITS A TRUE EMERGENCY IN OPHTHALMOLOGY
Laso corneal ulcer presentation
The document discusses corneal ulcer, beginning with the applied anatomy of the cornea. It defines corneal ulcer as the discontinuation of the normal epithelial surface. Causes can be infectious from bacteria, viruses, fungi, or protozoa, or noninfectious such as allergic, trophic, traumatic, idiopathic, vitamin A deficiency, or drug-induced. Clinical presentation includes pain, photophobia, tearing, redness, blurry vision, discharge, and foreign body sensation. Management involves investigations like cultures and scrapings to identify causative agents, followed by specific treatment targeting the agent and nonspecific treatments like antibiotics, cycloplegics, analgesics, and vitamins to aid healing. Complications can include
Recommended
Glaucoma and cataract include treatment
Glaucoma is a group of eye diseases characterized by increased intraocular pressure, optic nerve damage, and visual field loss. It is a leading cause of blindness worldwide. Cataracts are cloudy lenses that reduce vision and are most often age-related. They occur when the lens becomes less transparent over time. Risk factors for cataracts include diabetes, corticosteroid use, and glaucoma. Both conditions are generally treated surgically by removing the opaque lens or reducing eye pressure, but vision lost from glaucoma cannot be recovered. A combined study found diabetes significantly increases cataract risk while glaucoma is also an independent risk factor, potentially causing 5% of all cataracts.
Glaucoma
Glaucoma presentation for ophthalmology course, presented as a student seminar. Class location: ophthalmology unit, An-Najah National University Hospital.
Keratitis
Keratitis is an inflammatory condition that affects the cornea of your eye.
The cornea is the clear part that covers both the iris and the pupil.
Keratitis can be caused by an infection or injury to the eye.
Keratitis is a common condition.
People who wear contact lenses may experience keratitis more frequently than people who don’t wear contacts. In either case, you can take steps to help prevent this condition.
If you do develop keratitis, see your doctor right away.
Eye trauma
This document provides information on eye trauma, including causes, assessments, interventions, and nursing care. Key points include:
- Eye injuries are often preventable through safety measures. Common causes include accidents, assaults, and work or sports injuries.
- Assessment of eye trauma involves evaluating visual acuity, eye movements, pupil response, and checking for foreign bodies or orbital fractures. History should include injury details.
- Interventions depend on injury type but may include wound irrigation, patching, antibiotics, surgery for foreign bodies or fractures, and corticosteroids to reduce inflammation. Goals are to prevent infection and further damage.
- Nursing care focuses on monitoring for complications, educating on activity restrictions, and
Chemical injury to eye
This document provides information on chemical eye burns, including:
- Alkali burns are more dangerous than acid burns as alkalis penetrate rapidly into the eye.
- Immediate irrigation with saline or water for at least 20 minutes is essential to remove the chemical from the eye.
- The Dua classification system is useful for assessing prognosis based on limbal and conjunctival involvement.
- Medical management focuses on removing the chemical, promoting healing, controlling inflammation and infection, and managing intraocular pressure.
Dry eye presentation latest-Dr Dildar Singh
This document reviews diagnostic approaches and treatments for dry eye disease. It begins by defining dry eye as a multifactorial disease resulting from tear film instability and inflammation of the ocular surface. Diagnostic assessments include both subjective evaluations using questionnaires and objective evaluations of the tear film, ocular surface, and inflammation using tests such as tear osmolarity, tear breakup time, corneal staining, and meibomian gland evaluation. Treatments discussed include anti-inflammatory therapies like cyclosporine A and corticosteroids, supplements like essential fatty acids and nerve growth factor, and procedures like punctal occlusion and artificial tear implants.
Ocular chemical injury
chemical injury to eye by alkali, acids and irritants.
pathophysiology and management.
recent advances in management.
ITS A TRUE EMERGENCY IN OPHTHALMOLOGY
Laso corneal ulcer presentation
The document discusses corneal ulcer, beginning with the applied anatomy of the cornea. It defines corneal ulcer as the discontinuation of the normal epithelial surface. Causes can be infectious from bacteria, viruses, fungi, or protozoa, or noninfectious such as allergic, trophic, traumatic, idiopathic, vitamin A deficiency, or drug-induced. Clinical presentation includes pain, photophobia, tearing, redness, blurry vision, discharge, and foreign body sensation. Management involves investigations like cultures and scrapings to identify causative agents, followed by specific treatment targeting the agent and nonspecific treatments like antibiotics, cycloplegics, analgesics, and vitamins to aid healing. Complications can include
Ophthalmology 5th year, 5th lecture (Dr. Ali)
Ocular trauma can cause injuries to the eyeball and surrounding structures. Common types of injuries include chemical burns, thermal burns, blunt trauma, penetrating injuries, and injuries involving retained foreign bodies. A thorough history and examination are important to classify the injury and determine appropriate treatment. Minor injuries like corneal abrasions are generally treated with antibiotics while more severe injuries involving penetration of the eye wall may require specialty care. Prevention emphasizes reducing risk factors for work, home, sports, and transportation related injuries through primary, secondary and tertiary prevention strategies.
Chemical injuries of the eye
This document discusses chemical injuries to the eye. It begins with an introduction and overview of the epidemiology, etiology, pathogenesis, classification, clinical course, features, and management of such injuries. It notes that alkali injuries are more common and can be more deleterious. The pathogenesis and healing process differs for acid versus alkali injuries. Classification systems include Hughes, Roper-Hall, and Dua's, which predicts outcomes. Management involves immediate irrigation, acute medical treatment with steroids and antibiotics, and potential surgical interventions like debridement or amniotic membrane transplantation. Long-term goals are promoting healing, controlling inflammation and preventing complications.
Glaucoma for undergraduates
This document discusses glaucoma, including its definition, anatomy, types, risk factors, examination techniques, and classification. Specifically:
- Glaucoma is a disease of the optic nerve where damage to optic nerve fibers causes blind spots and vision loss.
- It involves abnormal drainage of the aqueous humor in the front of the eye, leading to increased intraocular pressure that damages the optic nerve over time.
- The main types are open-angle and closed-angle glaucoma. Risk factors include high eye pressure, family history, age, and certain medical conditions.
- Examination techniques to detect glaucoma include tonometry to measure intraocular pressure, fundoscopy to examine the optic disc
Penetrating Oc Injury
This document discusses penetrating ocular injuries, including their causes, effects, diagnosis, and treatment. Penetrating injuries can be caused by sharp objects and usually result in full thickness wounds. They can lead to mechanical effects like lacerations and infections. Diagnosis involves external examination, tonometry, ophthalmoscopy, and sometimes ultrasound or CT scans. Treatment depends on the type and severity of injury but may include antibiotics, pressure reduction, wound suturing, and repair of ruptures or lacerations.
Conjunctivitis
This document defines and describes various types of conjunctivitis, including infective bacterial conjunctivitis caused by organisms like Staphylococcus aureus and transmitted through flies or poor hygiene. Viral conjunctivitis is common and often caused by adenovirus. Ophthalmia neonatorum describes bilateral inflammation in infants less than 30 days old which can be caused by gonorrhea, chlamydia, or chemicals. Allergic conjunctivitis includes seasonal forms related to airborne allergens. Granulomatous conjunctivitis involves proliferative lesions possibly from tuberculosis, sarcoidosis, or leprosy. Cicatricial conjunctivitis can arise from conditions like
OPHTHALMIC NURSING.ppt
This document provides information on various topics in ophthalmic nursing including dry eye syndrome, conjunctivitis, uveitis, and orbital cellulitis. It discusses the anatomy, clinical manifestations, types, management, and prevention of these common eye conditions. Dry eye syndrome causes discomfort and is treated with artificial tears and ointments. Conjunctivitis can be caused by bacteria, viruses, allergies, or irritants and usually resolves on its own or with antibiotics. Uveitis and orbital cellulitis involve inflammation inside the eye and surrounding tissues.
Vernal keratoconjunctivitis
VKC is a chronic allergic inflammation of the ocular surface that is more common in children under 10 years of age. It involves the conjunctiva and cornea and causes symptoms like itching, redness, watering and sensitivity to light. Signs include thick mucus, transient yellow-white deposits called Horner-Tranta's dots, and large papillae on the conjunctiva that are graded based on their size. Corneal involvement can lead to punctate keratitis, erosions, ulcers and scarring if left untreated. The disease is seasonal in onset but may become perennial over time.
Allergic conjunctivitis
Allergic conjunctivitis is inflammation of the conjunctiva (the membrane covering the white part of the eye) due to allergy.Although allergens differ among patients, the most common cause is hay fever. Symptoms consist of redness (mainly due to vasodilation of the peripheral small blood vessels), edema (swelling) of the conjunctiva, itching, and increased lacrimation (production of tears). If this is combined with rhinitis, the condition is termed allergic rhinoconjunctivitis.
The symptoms are due to release of histamine and other active substances by mast cells, which stimulate dilation of blood vessels, irritate nerve endings, and increase secretion of tears.
Treatment of allergic conjunctivitis is by avoiding the allergen (e.g., avoiding grass in bloom during "hay fever season") and treatment with antihistamines, either topical (in the form of eye drops), or systemic (in the form of tablets). Antihistamines, medications that stabilize mast cells, and nonsteroidal anti-inflammatory drugs (NSAIDs) are generally safe and usually effective.
Chemical burns
This document discusses chemical burns to the eye. It notes that both thermal and chemical burns can cause blindness. Common causes of chemical burns are cleaning products, fertilizers, cement, fireworks, battery acid, and bleach. Alkaline agents like these cause more damage than acids as they penetrate cell membranes and disrupt collagen. A classification system grades injuries based on extent of limbal ischemia and corneal haze. Treatment involves immediate and copious irrigation, controlling inflammation with steroids, promoting healing with artificial tears and ascorbate, and managing intraocular pressure. The goal is to remove the chemical, promote healing, prevent infection, and control inflammation.
Posner schlossmann syndrome
Posner-Schlossman syndrome is characterized by recurrent unilateral attacks of ocular hypertension accompanied by mild anterior chamber inflammation. It was first described in 1948 and affects people aged 20-50, with males and females equally impacted. The exact pathogenesis is unknown but may involve trabecular meshwork obstruction by mononuclear cells. Symptoms include blurred vision and eye discomfort. Signs include elevated IOP out of proportion to mild inflammation and open iridocorneal angles. While usually self-limiting, long-term damage from recurrent attacks can occur, so treatment focuses on IOP control and inflammation management medically or surgically if needed.
Post operative endophthalmitis
1. Post-operative endophthalmitis is an intraocular inflammation that can occur after eye surgery or trauma as a complication. It involves inflammation in the anterior and posterior segments of the eye.
2. It is classified as infectious or non-infectious. Infectious endophthalmitis can be exogenous from surgery or trauma, or endogenous from hematogenous spread. Acute infectious endophthalmitis typically occurs within 6 weeks of surgery.
3. Management involves medical therapy including intravitreal and systemic antibiotics, as well as vitrectomy in some cases depending on visual acuity. The Endophthalmitis Vitrectomy Study showed vitrectomy is beneficial for patients
PTERYGIUM.pptx
Pterygium is a triangular growth of conjunctival tissue that extends onto the cornea. It is caused by prolonged sun exposure and is more common in older males working outdoors. Symptoms include irritation, foreign body sensation, and decreased vision. Treatment options include conservative measures like artificial tears or surgical excision with conjunctival autograft to prevent recurrence. Surgery involves removing the pterygium tissue from the cornea and replacing it with a conjunctival graft taken from the patient's eye. Post-operative care and monitoring for recurrence is important.
Conjunctivitis
conjunctivitis is an eye disorder .this is all so known as pink eye .in the India south side people it is called as madras eye ..in this slide mentioned about .definition, review anatomy and physiology, types including causes , pathophysiology, signs and symptoms, treatment, nursing management, and prevention are there. the client who is affected with this use separate towels, wash your hands daily, eat eye healthy foods like vitamin a , e,omega 3 fatty acid, this will help the eyes verywell.eye exercise very helpful for eye health.if eye sensitive is there use sun glass that is very help full for the eye , eye staraning should be avioded.if the school student is affected means student should not go to the school for one week.
Ocular changes in pregnancy
This document summarizes ocular changes that can occur during pregnancy. It discusses both physiological changes like increased pigmentation and pathological changes like worsening of diabetic retinopathy. Physiological changes include lowered intraocular pressure and darkened skin around the eyes. Pathological changes may affect structures like the retina, optic nerve and lens. Conditions like preeclampsia can cause retinal vascular changes. Managing preexisting eye conditions and monitoring for complications are important during pregnancy.
Keratitis
This document discusses various types of microbial keratitis, including bacterial, fungal, and viral keratitis. It describes common pathogens that can cause bacterial keratitis and their characteristic clinical features. The management of bacterial keratitis involves identifying the causative organism, administering topical antibiotics, and addressing complications. Fungal keratitis requires reculturing, antifungal therapy, and sometimes keratoplasty. Viral keratitis from herpes simplex virus can cause dendritic ulcers. Corneal ectasias like keratoconus and keratoglobus are also briefly mentioned.
Ocular Trauma
This document summarizes ocular trauma including extraocular foreign bodies, blunt trauma, penetrating trauma, and chemical injuries. It describes signs, symptoms, complications, investigations, and treatments for various types of eye injuries including hyphema, blowout fractures, iris tears, retinal detachment, and intraocular foreign bodies. Surgical management aims to repair wounds, remove foreign bodies or infected tissue, and prevent infections like endophthalmitis or sympathetic ophthalmia which can spread between eyes. Prognosis depends on severity and timing of treatment for the initial trauma.
Congenital anomalies in the eye
This document discusses various congenital anomalies that can occur in the eye. It covers anomalies of the eyelid, cornea, lens, uveal tract, retina, choroid, lacrimal apparatus, orbit, and optic nerve. For each structure, it provides examples of specific anomalies such as ptosis, coloboma, aniridia, persistent pupillary membrane, optic nerve hypoplasia, and more. It describes the signs, causes, and other clinical features of each congenital ocular anomaly.
Lens Induced Uveitis
This document summarizes lens-induced uveitis, which occurs when the lens capsule is ruptured or degraded, exposing lens proteins. This can trigger an immune reaction. There are two main types - phacoanaphylactic uveitis, which is a severe granulomatous anterior uveitis, and phacotoxic uveitis, a mild iridocyclitis. Lens-induced uveitis is usually caused by trauma or surgery that disrupts the lens capsule. Treatment involves topical corticosteroids and cycloplegics, with surgical removal of lens fragments if inflammation cannot be controlled. Complications can include cystoid macular edema, secondary glaucoma, and retinal detachment
Strabismus
Strabismus is a visual problem where the eyes are not properly aligned and point in different directions. It can be caused by disorders affecting the brain like cerebral palsy. The main symptom is an eye that is not straight. Diagnosis involves an eye exam. Treatment may include eyeglasses, prism, patching, injections, or surgery to correct the imbalanced eye muscles. Surgery involves weakening, strengthening, or moving eye muscles to change their action and align the eyes. Post-surgery, double vision may occur temporarily and activities are restricted for a few weeks to heal.
Ophthalmologic approach to chemical burns
Chemical burns are one of the true ophthalmologic emergencies. The ophthalmologist and general practitioner thus needs to be aware of the management of this type of eye injury.
CHEMICAL INJURIES OF EYEnew.p dr manuptx
This document discusses chemical eye injuries, including common causes, pathophysiology, clinical presentation, grading, complications, and management. Some key points:
- Chemical exposures can cause ocular trauma ranging from mild irritation to severe damage and vision loss. Common causes include household cleaners, industrial chemicals, and agricultural chemicals.
- Injuries are classified based on extent of corneal and limbal damage. Grade 1-2 injuries often recover with medical treatment while grade 3-4 generally require surgery in addition to medical management.
- Immediate irrigation is critical. Examination evaluates clarity, ischemia, IOP, and debridement may be needed. Treatment goals are removing the chemical, promoting healing, controlling inflammation,
Management of ocular chemical injuries
1. Chemical injuries to the eye represent one of the true ophthalmic emergencies. Alkali injuries are more common and severe than acid injuries due to their ability to rapidly
More Related Content
What's hot
Ophthalmology 5th year, 5th lecture (Dr. Ali)
Ocular trauma can cause injuries to the eyeball and surrounding structures. Common types of injuries include chemical burns, thermal burns, blunt trauma, penetrating injuries, and injuries involving retained foreign bodies. A thorough history and examination are important to classify the injury and determine appropriate treatment. Minor injuries like corneal abrasions are generally treated with antibiotics while more severe injuries involving penetration of the eye wall may require specialty care. Prevention emphasizes reducing risk factors for work, home, sports, and transportation related injuries through primary, secondary and tertiary prevention strategies.
Chemical injuries of the eye
This document discusses chemical injuries to the eye. It begins with an introduction and overview of the epidemiology, etiology, pathogenesis, classification, clinical course, features, and management of such injuries. It notes that alkali injuries are more common and can be more deleterious. The pathogenesis and healing process differs for acid versus alkali injuries. Classification systems include Hughes, Roper-Hall, and Dua's, which predicts outcomes. Management involves immediate irrigation, acute medical treatment with steroids and antibiotics, and potential surgical interventions like debridement or amniotic membrane transplantation. Long-term goals are promoting healing, controlling inflammation and preventing complications.
Glaucoma for undergraduates
This document discusses glaucoma, including its definition, anatomy, types, risk factors, examination techniques, and classification. Specifically:
- Glaucoma is a disease of the optic nerve where damage to optic nerve fibers causes blind spots and vision loss.
- It involves abnormal drainage of the aqueous humor in the front of the eye, leading to increased intraocular pressure that damages the optic nerve over time.
- The main types are open-angle and closed-angle glaucoma. Risk factors include high eye pressure, family history, age, and certain medical conditions.
- Examination techniques to detect glaucoma include tonometry to measure intraocular pressure, fundoscopy to examine the optic disc
Penetrating Oc Injury
This document discusses penetrating ocular injuries, including their causes, effects, diagnosis, and treatment. Penetrating injuries can be caused by sharp objects and usually result in full thickness wounds. They can lead to mechanical effects like lacerations and infections. Diagnosis involves external examination, tonometry, ophthalmoscopy, and sometimes ultrasound or CT scans. Treatment depends on the type and severity of injury but may include antibiotics, pressure reduction, wound suturing, and repair of ruptures or lacerations.
Conjunctivitis
This document defines and describes various types of conjunctivitis, including infective bacterial conjunctivitis caused by organisms like Staphylococcus aureus and transmitted through flies or poor hygiene. Viral conjunctivitis is common and often caused by adenovirus. Ophthalmia neonatorum describes bilateral inflammation in infants less than 30 days old which can be caused by gonorrhea, chlamydia, or chemicals. Allergic conjunctivitis includes seasonal forms related to airborne allergens. Granulomatous conjunctivitis involves proliferative lesions possibly from tuberculosis, sarcoidosis, or leprosy. Cicatricial conjunctivitis can arise from conditions like
OPHTHALMIC NURSING.ppt
This document provides information on various topics in ophthalmic nursing including dry eye syndrome, conjunctivitis, uveitis, and orbital cellulitis. It discusses the anatomy, clinical manifestations, types, management, and prevention of these common eye conditions. Dry eye syndrome causes discomfort and is treated with artificial tears and ointments. Conjunctivitis can be caused by bacteria, viruses, allergies, or irritants and usually resolves on its own or with antibiotics. Uveitis and orbital cellulitis involve inflammation inside the eye and surrounding tissues.
Vernal keratoconjunctivitis
VKC is a chronic allergic inflammation of the ocular surface that is more common in children under 10 years of age. It involves the conjunctiva and cornea and causes symptoms like itching, redness, watering and sensitivity to light. Signs include thick mucus, transient yellow-white deposits called Horner-Tranta's dots, and large papillae on the conjunctiva that are graded based on their size. Corneal involvement can lead to punctate keratitis, erosions, ulcers and scarring if left untreated. The disease is seasonal in onset but may become perennial over time.
Allergic conjunctivitis
Allergic conjunctivitis is inflammation of the conjunctiva (the membrane covering the white part of the eye) due to allergy.Although allergens differ among patients, the most common cause is hay fever. Symptoms consist of redness (mainly due to vasodilation of the peripheral small blood vessels), edema (swelling) of the conjunctiva, itching, and increased lacrimation (production of tears). If this is combined with rhinitis, the condition is termed allergic rhinoconjunctivitis.
The symptoms are due to release of histamine and other active substances by mast cells, which stimulate dilation of blood vessels, irritate nerve endings, and increase secretion of tears.
Treatment of allergic conjunctivitis is by avoiding the allergen (e.g., avoiding grass in bloom during "hay fever season") and treatment with antihistamines, either topical (in the form of eye drops), or systemic (in the form of tablets). Antihistamines, medications that stabilize mast cells, and nonsteroidal anti-inflammatory drugs (NSAIDs) are generally safe and usually effective.
Chemical burns
This document discusses chemical burns to the eye. It notes that both thermal and chemical burns can cause blindness. Common causes of chemical burns are cleaning products, fertilizers, cement, fireworks, battery acid, and bleach. Alkaline agents like these cause more damage than acids as they penetrate cell membranes and disrupt collagen. A classification system grades injuries based on extent of limbal ischemia and corneal haze. Treatment involves immediate and copious irrigation, controlling inflammation with steroids, promoting healing with artificial tears and ascorbate, and managing intraocular pressure. The goal is to remove the chemical, promote healing, prevent infection, and control inflammation.
Posner schlossmann syndrome
Posner-Schlossman syndrome is characterized by recurrent unilateral attacks of ocular hypertension accompanied by mild anterior chamber inflammation. It was first described in 1948 and affects people aged 20-50, with males and females equally impacted. The exact pathogenesis is unknown but may involve trabecular meshwork obstruction by mononuclear cells. Symptoms include blurred vision and eye discomfort. Signs include elevated IOP out of proportion to mild inflammation and open iridocorneal angles. While usually self-limiting, long-term damage from recurrent attacks can occur, so treatment focuses on IOP control and inflammation management medically or surgically if needed.
Post operative endophthalmitis
1. Post-operative endophthalmitis is an intraocular inflammation that can occur after eye surgery or trauma as a complication. It involves inflammation in the anterior and posterior segments of the eye.
2. It is classified as infectious or non-infectious. Infectious endophthalmitis can be exogenous from surgery or trauma, or endogenous from hematogenous spread. Acute infectious endophthalmitis typically occurs within 6 weeks of surgery.
3. Management involves medical therapy including intravitreal and systemic antibiotics, as well as vitrectomy in some cases depending on visual acuity. The Endophthalmitis Vitrectomy Study showed vitrectomy is beneficial for patients
PTERYGIUM.pptx
Pterygium is a triangular growth of conjunctival tissue that extends onto the cornea. It is caused by prolonged sun exposure and is more common in older males working outdoors. Symptoms include irritation, foreign body sensation, and decreased vision. Treatment options include conservative measures like artificial tears or surgical excision with conjunctival autograft to prevent recurrence. Surgery involves removing the pterygium tissue from the cornea and replacing it with a conjunctival graft taken from the patient's eye. Post-operative care and monitoring for recurrence is important.
Conjunctivitis
conjunctivitis is an eye disorder .this is all so known as pink eye .in the India south side people it is called as madras eye ..in this slide mentioned about .definition, review anatomy and physiology, types including causes , pathophysiology, signs and symptoms, treatment, nursing management, and prevention are there. the client who is affected with this use separate towels, wash your hands daily, eat eye healthy foods like vitamin a , e,omega 3 fatty acid, this will help the eyes verywell.eye exercise very helpful for eye health.if eye sensitive is there use sun glass that is very help full for the eye , eye staraning should be avioded.if the school student is affected means student should not go to the school for one week.
Ocular changes in pregnancy
This document summarizes ocular changes that can occur during pregnancy. It discusses both physiological changes like increased pigmentation and pathological changes like worsening of diabetic retinopathy. Physiological changes include lowered intraocular pressure and darkened skin around the eyes. Pathological changes may affect structures like the retina, optic nerve and lens. Conditions like preeclampsia can cause retinal vascular changes. Managing preexisting eye conditions and monitoring for complications are important during pregnancy.
Keratitis
This document discusses various types of microbial keratitis, including bacterial, fungal, and viral keratitis. It describes common pathogens that can cause bacterial keratitis and their characteristic clinical features. The management of bacterial keratitis involves identifying the causative organism, administering topical antibiotics, and addressing complications. Fungal keratitis requires reculturing, antifungal therapy, and sometimes keratoplasty. Viral keratitis from herpes simplex virus can cause dendritic ulcers. Corneal ectasias like keratoconus and keratoglobus are also briefly mentioned.
Ocular Trauma
This document summarizes ocular trauma including extraocular foreign bodies, blunt trauma, penetrating trauma, and chemical injuries. It describes signs, symptoms, complications, investigations, and treatments for various types of eye injuries including hyphema, blowout fractures, iris tears, retinal detachment, and intraocular foreign bodies. Surgical management aims to repair wounds, remove foreign bodies or infected tissue, and prevent infections like endophthalmitis or sympathetic ophthalmia which can spread between eyes. Prognosis depends on severity and timing of treatment for the initial trauma.
Congenital anomalies in the eye
This document discusses various congenital anomalies that can occur in the eye. It covers anomalies of the eyelid, cornea, lens, uveal tract, retina, choroid, lacrimal apparatus, orbit, and optic nerve. For each structure, it provides examples of specific anomalies such as ptosis, coloboma, aniridia, persistent pupillary membrane, optic nerve hypoplasia, and more. It describes the signs, causes, and other clinical features of each congenital ocular anomaly.
Lens Induced Uveitis
This document summarizes lens-induced uveitis, which occurs when the lens capsule is ruptured or degraded, exposing lens proteins. This can trigger an immune reaction. There are two main types - phacoanaphylactic uveitis, which is a severe granulomatous anterior uveitis, and phacotoxic uveitis, a mild iridocyclitis. Lens-induced uveitis is usually caused by trauma or surgery that disrupts the lens capsule. Treatment involves topical corticosteroids and cycloplegics, with surgical removal of lens fragments if inflammation cannot be controlled. Complications can include cystoid macular edema, secondary glaucoma, and retinal detachment
Strabismus
Strabismus is a visual problem where the eyes are not properly aligned and point in different directions. It can be caused by disorders affecting the brain like cerebral palsy. The main symptom is an eye that is not straight. Diagnosis involves an eye exam. Treatment may include eyeglasses, prism, patching, injections, or surgery to correct the imbalanced eye muscles. Surgery involves weakening, strengthening, or moving eye muscles to change their action and align the eyes. Post-surgery, double vision may occur temporarily and activities are restricted for a few weeks to heal.
Ophthalmologic approach to chemical burns
Chemical burns are one of the true ophthalmologic emergencies. The ophthalmologist and general practitioner thus needs to be aware of the management of this type of eye injury.
What's hot (20)
Similar to Chemical eye trauma
CHEMICAL INJURIES OF EYEnew.p dr manuptx
This document discusses chemical eye injuries, including common causes, pathophysiology, clinical presentation, grading, complications, and management. Some key points:
- Chemical exposures can cause ocular trauma ranging from mild irritation to severe damage and vision loss. Common causes include household cleaners, industrial chemicals, and agricultural chemicals.
- Injuries are classified based on extent of corneal and limbal damage. Grade 1-2 injuries often recover with medical treatment while grade 3-4 generally require surgery in addition to medical management.
- Immediate irrigation is critical. Examination evaluates clarity, ischemia, IOP, and debridement may be needed. Treatment goals are removing the chemical, promoting healing, controlling inflammation,
Management of ocular chemical injuries
1. Chemical injuries to the eye represent one of the true ophthalmic emergencies. Alkali injuries are more common and severe than acid injuries due to their ability to rapidly
Chemical injury of the eye
1) Chemical injuries to the eye are common, ranging from mild irritation to complete destruction of ocular tissues. Alkali agents are more damaging than acids due to their ability to penetrate tissues.
2) Treatment depends on severity but generally involves immediate irrigation, antibiotics, steroids, and artificial tears. More severe injuries may require amniotic membrane transplantation or limbal stem cell transplant.
3) Even with treatment, complications can include dry eye, glaucoma, eyelid scarring, and vision loss. Patients require long-term follow up due to risk of these complications.
CHEMICAL INJURIES_DEEEKSHA k.pptx
- Chemical injuries to the eyes are a type of ocular emergency that can cause significant damage. Alkali injuries are more common and severe than acid injuries.
- Proper management involves immediate irrigation to remove the chemical and restore pH, along with medical treatment including antibiotics, steroids, and cycloplegics to promote healing and control inflammation. More severe injuries may require surgical interventions.
- Long term follow up is important to monitor for complications like glaucoma and dry eye. Protection of the eyes is also critical in environments where chemical exposures are possible through the use of goggles, face shields, and other protective equipment.
Occupational Eye Disorders
This document discusses occupational eye disorders and vision screening. It notes that in the US in 2002, 47% of all occupational head injuries were eye-related, with ocular foreign bodies and contusions/abrasions being the most common. There are two types of vision assessments: examinations which diagnose issues, and screenings which identify risks and provide referrals. Screenings evaluate factors like visual acuity, fields, color vision, depth perception and contrast sensitivity. The document also discusses hazards like chemicals, radiation, physical trauma and lasers that can threaten vision in the workplace. Proper screening and treatment can help prevent occupational eye diseases and injuries.
Seminar chemical injuries
Chemical injuries to the eye are one of the true ophthalmic emergencies and can result in significant visual impairment or disability. Alkali injuries are more common and tend to cause more severe damage than acids. The severity of injury depends on the type, concentration, and duration of exposure to the chemical agent. Several classification systems have been developed to describe the extent of injury, with the Duas classification providing the most detailed assessment of limbal and conjunctival involvement. The pathophysiology involves acute damage to ocular tissues, followed by inflammation and attempts at epithelial regeneration and stromal repair which can sometimes result in complications like corneal ulceration if the balance tips too far toward degradation.
Chemical injury
This document discusses chemical eye injuries, which are ophthalmic emergencies requiring immediate treatment to prevent vision loss. It describes the types of chemicals that can cause eye injuries, including acids, alkalis, and neutrals, and how their pH levels affect injury severity. Alkalis like ammonia and sodium hydroxide are particularly dangerous as they penetrate rapidly. The document also outlines classifications of ocular burns and recommends initial management steps like irrigation before discussing longer-term treatment approaches.
Chemical Injuries.pptx
This document discusses chemical injuries to the eye. It describes the types of chemicals that can cause injuries, including acids and alkalis found in industrial and domestic settings. The pathology of chemical injuries is explained, noting how alkalis and acids damage ocular tissues differently. Clinical features include ulceration, limbal ischemia, and complications like symblepharon. Treatment involves immediate irrigation at the injury site, then continued irrigation, antibiotics, and steroids in the hospital. Promoting epithelialization and limiting further damage are priorities in management. Prevention emphasizes public education, industrial safety precautions, and identifying the chemical involved.
Ocular chemical injuries
Chemical injuries to the eye can be caused by either strongly basic (alkaline) compounds or acidic compounds. Alkali injuries are more common and tend to be more severe, damaging the eye through penetration and interaction with ocular tissues. The severity of injury depends on the chemical agent, concentration, duration of exposure, and degree of penetration. Various classification systems aim to describe chemical injuries and predict visual prognosis, but newer systems provide more accurate assessment of limbal involvement to better determine treatment approach and expected outcomes.
Chemical injuries
This document summarizes the pathophysiology and management of chemical eye injuries. It discusses that chemical injuries can cause significant damage through alkali or acid exposure. Treatment involves immediate irrigation, debridement if needed, and aggressive medical therapy including corticosteroids, ascorbate, antibiotics and citrate to promote reepithelialization during the acute and early repair phases. More severe injuries may require surgical interventions like limbal stem cell transplantation or amniotic membrane transplantation depending on the degree of limbal involvement and prognosis.
Hydrofluoric Acid #3
This document provides information on the hazards and safe handling of hydrofluoric acid (HF). HF is a highly hazardous liquid and vapor that causes severe burns. It can penetrate the skin and bones, and exposure may be fatal if swallowed or inhaled. The summary describes first aid measures for different exposure routes and symptoms of HF poisoning including burns and hypocalcemia. Firefighting procedures are outlined, and uses of HF in various industries are listed, along with examples of HF accidents and pollution incidents.
Dry eye
Dry eye is a disease of the ocular surface caused by disturbances in the tear film. The normal tear film consists of an inner mucin layer, middle aqueous layer, and outer lipid layer, which work together to form a stable tear film. Disruptions to the tear film components or their functions can lead to dry eye. Common tests to diagnose dry eye include tear break-up time, fluorescein clearance, and tear osmolarity measurement, which help identify tear film instability and inflammation associated with the condition.
Ocular Emergencies
This document discusses ocular emergencies, including trauma and non-trauma related conditions. It describes various types of blunt and penetrating trauma such as corneal abrasions, hyphema, lid lacerations, and chemical burns. Signs and symptoms of conditions like subconjunctival hemorrhage, corneal ulcers, acute angle closure glaucoma, and orbital cellulitis are provided. Priority levels ranging from urgent to emergency are assigned based on the acute nature and visual prognosis of different eye conditions. Treatment methods like irrigation are also outlined for select emergencies.
Chemical eye trauma, By Dr. Safaa Refaat
1) Chemical eye injuries can occur in domestic, industrial, or military settings and are among the most destructive traumatic eye injuries. Alkalis generally penetrate further than acids due to their mechanisms of tissue damage.
2) Immediate and effective irrigation is crucial for chemical eye injuries, with the amount of fluid and time of irrigation depending on the substance and pH level. Neutralizing the pH through irrigation is critical even before a full examination.
3) Prognosis depends on factors like pH, duration of contact, extent of corneal involvement and limbal ischemia. More severe injuries may require long-term treatments like conjunctival grafts or keratoplasty to attempt to save vision.
occupational, household & sports hazards
This document discusses common eye injuries, their causes, and symptoms. It covers injuries that can occur in workplace, household, and sports settings from flying debris, chemicals, heat, dust, radiation, and blunt impacts. Specific risks are mentioned for various industries and activities. Eye injuries range from minor abrasions to serious issues like burns, ulcers, and blindness. Prompt treatment is important for penetrating injuries. Workplace hazards include rotating machinery, welding, grinding, and toxic chemicals. Sports with eye injury risks include baseball, boxing, racquet sports, and bicycling.
Directory of Toxic Ingredient in Cosmetics & Body Care Products
Directory of Toxic Ingredient in Cosmetics & Body Care Products - Resources for Healthy Children www.scribd.com/doc/254613619 - For more information, Please see Organic Edible Schoolyards & Gardening with Children www.scribd.com/doc/254613963 - Gardening with Volcanic Rock Dust www.scribd.com/doc/254613846 - Double Food Production from your School Garden with Organic Tech www.scribd.com/doc/254613765 - Free School Gardening Art Posters www.scribd.com/doc/254613694 - Increase Food Production with Companion Planting in your School Garden www.scribd.com/doc/254609890 - Healthy Foods Dramatically Improves Student Academic Success www.scribd.com/doc/254613619 - City Chickens for your Organic School Garden www.scribd.com/doc/254613553 - Huerto Ecológico, Tecnologías Sostenibles, Agricultura Organica www.scribd.com/doc/254613494 - Simple Square Foot Gardening for Schools - Teacher Guide www.scribd.com/doc/254613410 - Free Organic Gardening Publications www.scribd.com/doc/254609890 ~
The ocular surface chemical burns
The document summarizes the effects of chemical burns on the ocular surface and the different treatment phases. Basic substances penetrate the eye more rapidly than acids, lysing cell membranes and triggering inflammation. Acids cause protein coagulation in the epithelium. Factors like pH, temperature, and concentration determine damage. The reparative phases are immediate, acute, early, and late. Early signs include epithelial defects and conjunctival inflammation. The acute phase involves reestablishing the corneal epithelium and potential glaucoma. Later phases involve chronic inflammation, scarring, and secondary complications like glaucoma. Emergency therapy focuses on irrigation, lubricants, and antibiotics. Surgical management includes debridement, amniotic membrane transplantation
Chemical disaster tam 2014-10
Chemical disasters can harm human health and the environment through the unintentional release of hazardous substances. The document discusses the 1984 Bhopal disaster in India, where a leak of methyl isocyanate (MIC) gas from a Union Carbide pesticide plant killed thousands. MIC is an intermediate chemical used to produce carbaryl insecticide. Exposure to the toxic gas caused many deaths in the days following and ongoing health issues. The disaster highlights the dangers of chemical accidents and their long-term human and environmental impacts.
Heavy metals in Cosmetics
Cosmetics are used by most of women all over the world,So taking care of their composition is the most critical point to avoid the harm of heavy metals and their concentration in them.
Contact dermatitis
Contact dermatitis can be caused by allergic reactions (allergic contact dermatitis) or irritants (irritant contact dermatitis). Allergic contact dermatitis is a type IV delayed hypersensitivity reaction that occurs when a sensitized individual is re-exposed to an allergen. It accounts for about 20% of contact dermatitis cases. Patch testing is needed to identify the specific allergen causing allergic contact dermatitis. Irritant contact dermatitis results from direct skin damage caused by chemicals, metals, fabrics, or other irritating substances. Avoiding the irritating or allergenic substance is the primary treatment approach for both types of contact dermatitis.
Similar to Chemical eye trauma (20)
Directory of Toxic Ingredient in Cosmetics & Body Care Products
Directory of Toxic Ingredient in Cosmetics & Body Care Products
More from Amr Mounir
Corneal Causes of Decreased Vision.pptx
This document discusses various corneal causes of decreased vision. It begins with an introduction to the anatomy and function of the cornea. There are then several sections covering common corneal conditions like injuries, keratitis, dry eye, and corneal dystrophies. Specific causes of decreased vision are explored such as infections, nutritional deficiencies, inflammation, inherited conditions, degeneration, and trauma. Important corneal conditions like keratitis, trachoma, xerophthalmia, peripheral ulcerative keratitis, and corneal dystrophies are described in more detail. The document concludes with a section on diagnostic approaches for corneal conditions.
Secondary Corneal Ectasia.pptx
1) Secondary ectasia refers to corneal shape changes caused by external factors like previous refractive surgery or corneal transplantation.
2) Post-LASIK ectasia is a rare complication characterized by progressive myopia and astigmatism due to thinning and protrusion of the cornea. Risk factors include young age and pre-existing thin corneas.
3) On Pentacam, early post-LASIK ectasia shows mild corneal thinning and elevation changes mostly in the inferior cornea, while advanced cases resemble keratoconus with significant steepening and irregularity.
Refractive surgery for beginners which surgery for which patien.pptx
This document discusses different types of refractive surgery, including surface ablation techniques like PRK as well as LASIK. It notes that while LASIK became popular initially due to faster recovery times, surface ablation procedures have advantages in safety and corneal stability since they do not create a flap. The document outlines some risks of LASIK like flap complications and long term safety issues. It also discusses problems with early surface ablation like haze and healing issues. More advanced surface ablation techniques like LASEK and Epi-LASIK are highlighted as safer alternatives to LASIK that avoid flap risks. The use of femtosecond lasers to create thinner, more precise flaps for LASIK is also discussed as a way to increase its safety profile.
References & Legends.pptx
This document discusses referencing styles, citations, and figure and table legends. It provides examples of references in APA, MLA, Harvard, and ACS styles. Key points covered include:
- References should provide necessary information for readers to identify cited works and be accurate and complete.
- Common referencing styles are MLA, APA, Harvard and MHRA.
- Figure and table legends should include a title, methods, results, and any necessary definitions to explain the materials, methods and key findings shown. Legends are usually short phrases placed above tables.
H-index.pptx
The h-index is a metric developed by J.E. Hirsch to quantify an individual researcher's scientific impact. It is calculated based on the number of papers they have published and the number of citations received, with the h-index being the number of papers that have been cited at least that same number of times. Specifically, the h-index looks at a researcher's most cited papers ranked in descending order by citations, and the h-index value is equal to the number of papers that have received that same number or more citations.
Management of Cataract.pptx
This document provides an overview of cataract management, including causes, types, preoperative preparation, surgical techniques, and complications. A cataract is a cloudy area in the lens that decreases vision, with common symptoms including blurred or double vision. Causes include age, trauma, medications, and diseases. Preoperative preparation involves visual acuity testing, slit lamp examination, and imaging of the eye and lens. Surgical techniques include phacoemulsification, the most common, manual small incision surgery, extracapsular extraction, and intracapsular extraction. Potential complications are complications of anesthesia, posterior capsular tear, endophthalmitis, toxic anterior segment syndrome, and retinal detachment.
Noncontact Meibography.pptx
This document discusses non-contact meibography, a technique used to image the meibomian glands of the eyelid. It can be performed easily using a slit lamp equipped with an infrared camera. Meibography allows visualization of the glands and quantification of gland dropout. Normal glands appear as hypoilluminant grape-like clusters while dropout glands do not traverse the entire tarsal plate. The technique has advantages of being non-invasive while allowing documentation over time. Grading systems have also been developed to objectively analyze meibography images. The document outlines several studies where meibography was used to evaluate gland changes in various ocular surface disorders like blepharitis, contact lens use, and
Discussion.pptx
This document provides guidance on writing the discussion section of a research article. The discussion section analyzes the meaning and importance of results, relating them to the literature review and research questions. It should include interpretation of results, implications, limitations, and recommendations. When interpreting results, the author should identify patterns in the data, compare findings to hypotheses and previous research, and explain unexpected results. The discussion should also discuss implications for theory and practice, acknowledge limitations, and make recommendations for further research based on results. The author is advised to summarize key findings, interpret results in light of other research without exaggerating claims, and avoid introducing new results or undermining their own research.
Retinal detachment
This document discusses retinal detachment, which occurs when the retina separates from the underlying tissue. The most common type, rhegmatogenous retinal detachment, results from a tear or break in the retina that allows fluid to seep in. Symptoms include floaters, visual field defects, and flashes of light. Diagnosis involves examinations of the eye and ultrasound. Treatment depends on the type but generally aims to seal retinal breaks, relieve traction, and reattach the retina using procedures like cryotherapy, laser photocoagulation, pneumatic retinopexy using gas bubbles, or vitrectomy surgery to remove the vitreous gel.
Eyelid malposition
This document discusses various eyelid malpositions including entropion, ectropion, ptosis, and trichiasis. Entropion is when the eyelid rolls inward causing eyelashes to rub the eye, potentially damaging the cornea. Ectropion is when the eyelid rolls outward. Causes, symptoms, evaluations, and treatments are described for each condition. Conservative treatments include taping, lubricants, and botulinum toxin injections. Surgical treatments aim to tighten or reposition the eyelid depending on the specific malposition and its underlying cause. Prompt treatment is important to prevent corneal complications.
Blepharitis
Blepharitis is a common eye condition characterized by inflamed, scaly, red eyelids. It is caused by bacteria, dandruff, or other skin conditions. There are different types of blepharitis that affect the front or back of the eyelids. Symptoms include gritty or burning eyes, excessive tearing, and eyelid crusting. While blepharitis itself is not sight-threatening, it can lead to permanent eyelid changes if left untreated. Treatment focuses on keeping eyelids clean through warm compresses and scrubbing, with antibiotics or steroids possibly prescribed depending on the underlying cause.
Eye and covid 19
This document outlines ophthalmic manifestations of COVID-19 reported in the literature. It discusses how SARS-CoV-2 interacts with ACE2 receptors in ocular tissues, potentially causing immune-mediated damage and coagulopathy. Common anterior segment manifestations include conjunctivitis (in up to 8% of patients), keratoconjunctivitis, hemorrhagic conjunctivitis, and episcleritis. Posterior segment and neuro-ophthalmic manifestations as well as orbital involvement are also reviewed. The document examines evidence that the eye may serve as a route of COVID-19 infection transmission and discusses evaluations and management according to clinical presentation.
Ptosis
Ptosis, or drooping of the upper eyelid, can have several causes including paralysis or disease (myogenic, neurogenic), or be congenital (aponeurotic). Myogenic ptosis may be congenital due to poor levator muscle contraction/relaxation, or acquired like in myasthenia gravis where the myoneural junction is defective. Neurogenic ptosis includes third nerve palsy or Horner's syndrome. Aponeurotic ptosis includes senile or postoperative changes. Treatment depends on the cause and degree of ptosis, and may involve frontal sling surgery using the frontalis muscle, levator resection/advancement for moderate ptosis, or Whitnall sling or
Chalazion
Chalazion is a chronic inflammatory nodule of the meibomian gland in the eyelid. It is the most common benign eyelid lesion, occurring more often in adults. Chalazions start as small, tender swellings and are caused by obstruction and retention of secretions in the meibomian gland. On pathology, they show chronic inflammation with lipid-filled giant cells. Clinically, they appear as painless, hard swellings of the eyelid that can cause blurry vision. Treatment involves warm compresses, antibiotics, steroids, or incision and curettage for larger lesions. Complications include infection, cyst formation, or recurrence.
Non infectious corneal ulcers
Non-infectious corneal ulcers can be caused by local factors like punctate marginal keratitis due to hypersensitivity to medications or peripheral keratitis associated with blepharitis, or systemic immune-mediated diseases like rheumatoid arthritis. Marginal keratitis is characterized by chronic blepharitis and subepithelial marginal infiltrates separated from the limbus by a clear zone. Mooren's ulcer is a rare autoimmune disease seen as progressive peripheral circumferential stromal ulceration. Terrien marginal degeneration is an idiopathic thinning of the peripheral cornea. Peripheral ulcerative keratitis can be associated with underlying systemic autoimmune diseases like rheumatoid arthritis.
Keratoconus mangment
This presentation describes all the clinical aspects of keratoconus management
You can watch the illustrated presentation in this link :
https://www.youtube.com/watch?v=pYxwZPGm7e4&list=PLZ_mM13I_TrhWavjTmE9NjW1O5bGxkONO&index=13
Infectious corneal ulcers
This presentation describes all clinical aspects of infectious corneal ulcers
You can watch the illustrated presentation in this link :
https://www.youtube.com/watch?v=okWDPG3C34g&list=PLZ_mM13I_TrhwqZuGjB6M9Z3n7MntrURd
Glaucoma medication
This presentation describes all clinical aspects of glaucoma medications.....you can watch this presentation in video form at the following link
https://www.youtube.com/watch?v=92xurWP41dA
Congenital glaucoma
This presentation describes all clinical aspects of congenital glaucoma....you can watch this presentation in video form at the following link
https://www.youtube.com/watch?v=Y5YA2CYzb5c
Femtolasik...Indications and limitations
This presentation describes all indications and limitations of femtosecond laser surgery.....you can watch the illustrated video presentation in the following link:
https://youtu.be/vCwu-_hpWxA
More from Amr Mounir (20)
Refractive surgery for beginners which surgery for which patien.pptx
Refractive surgery for beginners which surgery for which patien.pptx
Recently uploaded
The Best Ayurvedic Antacid Tablets in India
Treat the symptoms of indigestion, heartburn and stomach reflux with the 10 Best Ayurvedic Antacid Tablets in India.
Best Ayurvedic medicine for Gas and Indigestion
Here is the updated list of Top Best Ayurvedic medicine for Gas and Indigestion and those are Gas-O-Go Syp for Dyspepsia | Lavizyme Syrup for Acidity | Yumzyme Hepatoprotective Capsules etc
Cell Therapy Expansion and Challenges in Autoimmune Disease
There is increasing confidence that cell therapies will soon play a role in the treatment of autoimmune disorders, but the extent of this impact remains to be seen. Early readouts on autologous CAR-Ts in lupus are encouraging, but manufacturing and cost limitations are likely to restrict access to highly refractory patients. Allogeneic CAR-Ts have the potential to broaden access to earlier lines of treatment due to their inherent cost benefits, however they will need to demonstrate comparable or improved efficacy to established modalities.
In addition to infrastructure and capacity constraints, CAR-Ts face a very different risk-benefit dynamic in autoimmune compared to oncology, highlighting the need for tolerable therapies with low adverse event risk. CAR-NK and Treg-based therapies are also being developed in certain autoimmune disorders and may demonstrate favorable safety profiles. Several novel non-cell therapies such as bispecific antibodies, nanobodies, and RNAi drugs, may also offer future alternative competitive solutions with variable value propositions.
Widespread adoption of cell therapies will not only require strong efficacy and safety data, but also adapted pricing and access strategies. At oncology-based price points, CAR-Ts are unlikely to achieve broad market access in autoimmune disorders, with eligible patient populations that are potentially orders of magnitude greater than the number of currently addressable cancer patients. Developers have made strides towards reducing cell therapy COGS while improving manufacturing efficiency, but payors will inevitably restrict access until more sustainable pricing is achieved.
Despite these headwinds, industry leaders and investors remain confident that cell therapies are poised to address significant unmet need in patients suffering from autoimmune disorders. However, the extent of this impact on the treatment landscape remains to be seen, as the industry rapidly approaches an inflection point.
ABDOMINAL TRAUMA in pediatrics part one.
Abdominal trauma in pediatrics refers to injuries or damage to the abdominal organs in children. It can occur due to various causes such as falls, motor vehicle accidents, sports-related injuries, and physical abuse. Children are more vulnerable to abdominal trauma due to their unique anatomical and physiological characteristics. Signs and symptoms include abdominal pain, tenderness, distension, vomiting, and signs of shock. Diagnosis involves physical examination, imaging studies, and laboratory tests. Management depends on the severity and may involve conservative treatment or surgical intervention. Prevention is crucial in reducing the incidence of abdominal trauma in children.
A Classical Text Review on Basavarajeeyam
Basavarajeeyam is a Sreshta Sangraha grantha (Compiled book ), written by Neelkanta kotturu Basavaraja Virachita. It contains 25 Prakaranas, First 24 Chapters related to Rogas& 25th to Rasadravyas.
Phone Us ❤8107221448❤ #ℂall #gIRLS In Dehradun By Dehradun @ℂall @Girls Hotel...
Phone Us ❤8107221448❤ #ℂall #gIRLS In Dehradun By Dehradun @ℂall @Girls Hotel...chandankumarsmartiso
Phone Us ❤8107221448❤ #ℂall #gIRLS In Dehradun By Dehradun @ℂall @Girls Hotel With 100% Satisfaction
Journal Article Review on Rasamanikya
Rasamanikya is a excellent preparation in the field of Rasashastra, it is used in various Kushtha Roga, Shwasa, Vicharchika, Bhagandara, Vatarakta, and Phiranga Roga. In this article Preparation& Comparative analytical profile for both Formulationon i.e Rasamanikya prepared by Kushmanda swarasa & Churnodhaka Shodita Haratala. The study aims to provide insights into the comparative efficacy and analytical aspects of these formulations for enhanced therapeutic outcomes.
Osteoporosis - Definition , Evaluation and Management .pdf
Osteoporosis is an increasing cause of morbidity among the elderly.
In this document , a brief outline of osteoporosis is given , including the risk factors of osteoporosis fractures , the indications for testing bone mineral density and the management of osteoporosis
Role of Mukta Pishti in the Management of Hyperthyroidism
Muktapishti is a traditional Ayurvedic preparation made from Shoditha Mukta (Purified Pearl), is believed to help regulate thyroid function and reduce symptoms of hyperthyroidism due to its cooling and balancing properties. Clinical evidence on its efficacy remains limited, necessitating further research to validate its therapeutic benefits.
NVBDCP.pptx Nation vector borne disease control program
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
Top-Vitamin-Supplement-Brands-in-India List
Swisschem Dermacare provides the Top 10 Vitamin Supplement Brands in India. To know more about us give us call at our official number
Does Over-Masturbation Contribute to Chronic Prostatitis.pptx
In some case, your chronic prostatitis may be related to over-masturbation. Generally, natural medicine Diuretic and Anti-inflammatory Pill can help mee get a cure.
Recently uploaded (20)
Ear and its clinical correlations By Dr. Rabia Inam Gandapore.pptx
Ear and its clinical correlations By Dr. Rabia Inam Gandapore.pptx
Thyroid Gland- Gross Anatomy by Dr. Rabia Inam Gandapore.pptx
Thyroid Gland- Gross Anatomy by Dr. Rabia Inam Gandapore.pptx
Tests for analysis of different pharmaceutical.pptx
Tests for analysis of different pharmaceutical.pptx
CHEMOTHERAPY_RDP_CHAPTER 6_Anti Malarial Drugs.pdf
CHEMOTHERAPY_RDP_CHAPTER 6_Anti Malarial Drugs.pdf
Cell Therapy Expansion and Challenges in Autoimmune Disease
Cell Therapy Expansion and Challenges in Autoimmune Disease
Phone Us ❤8107221448❤ #ℂall #gIRLS In Dehradun By Dehradun @ℂall @Girls Hotel...
Phone Us ❤8107221448❤ #ℂall #gIRLS In Dehradun By Dehradun @ℂall @Girls Hotel...
Osteoporosis - Definition , Evaluation and Management .pdf
Osteoporosis - Definition , Evaluation and Management .pdf
Role of Mukta Pishti in the Management of Hyperthyroidism
Role of Mukta Pishti in the Management of Hyperthyroidism
NVBDCP.pptx Nation vector borne disease control program
NVBDCP.pptx Nation vector borne disease control program
Does Over-Masturbation Contribute to Chronic Prostatitis.pptx
Does Over-Masturbation Contribute to Chronic Prostatitis.pptx
Chemical eye trauma
- 1. Chemical Eye Trauma By: Dr. Amr Mounir Elham Mahmoud Ahmed
- 3. Epidemiology Chemical injuries to the eye represent about 11,5- 22%of ocular trauma,more in young males,and at work and homes Causes: Duo to exposure to chemicals as acid,alkali ,gases.
- 4. Types Strong acid Sulphoric acid in car battery. Hydrochloric acid in swimming pool. Strong alkali Potassium hydroxide in caustic potash. Sodium hydroxide in France cleaner. Other chemicals Lime, Aniline dye,iodine.
- 5. The effect depends on:- *Concentration *Duration:- interval between chemical trauma&1st aid *Pathogenesis:- it deffers according to the type as: 1-Alkali agents are lipophilic so it penetrate tissues by saponifing the fatty acids of cell membrans and causing pentration to the corneal stroma. 2-Acide agents coagulates and participate protiens at the epithelium forming a physical barrier with less penetrating effect.
- 6. Clinical picture *Symptoms :- Pain, lacrimation, blepharospasm,photophobia and drop of vision.
- 7. Signs 1-Lid Edema Triciasis Corneal ulcer Cicatricial ectropion , Entropion, Lagophthalmos 2-Ocular surface Dry eye Corneal scaring,opacity,corneal melt Symblepharon Anterior uveitis,iridocyclitis,atrophia bulbi Conjunctival ulcer,injection,chemosis,pseudopterygium 3-Elevated IOP Secondary glucoma
- 12. Treatment 1-First Aid ttt Immediate irrigation of the eye with water or physiologic saline for 30 minutes then removal of partcle and deprdment of necrotic tissue. A-If the nature of the chemical substance is known : wash the eye with proper antidote as : -acids, NaHCO3 3% -alkali,Boric acid lotion 4% -iodine, starch solution or milk -Aniline,Alcohol 10%then glycerin 10% -Lime,use EDTA 0,1% but water not used B-If the nature of the chemical substance is unknown: wash the eye with either,-Tap water -Milk
- 13. 2-Local treatment -Antibiotic: tetracycline -Atropine : for corneal ulceration&iritis -Steroid :to decrease inflammation,corneal edema, vascularization,scaring (1st7days ) 3-ttt of complication A -Prevention of symblepharon: *Glass rod coated with antibiotic ointment *Cortisone ointment *Contact shell *Soft CL *Limbal stem cell transplantation,help epitheliazation *AMG,supress fibrosis
- 14. B-2ry glaucoma *Diamox *Trabeculectomy in late cases C-Lagophthalmos , D-Corneal opacity: *Keratoplasty E-Dry eye treatment F-Surgical ttt ot trichiasis & lid
- 15. Thank you